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Potential independent action of sigma receptor ligands through inhibition of the Kv2.1 channel.

Oncotarget

Liu X, Fu Y, Yang H, Mavlyutov T, Li J, McCurdy CR, Guo LW, Pattnaik BR.
PMID: 28938641
Oncotarget. 2017 Jul 26;8(35):59345-59358. doi: 10.18632/oncotarget.19581. eCollection 2017 Aug 29.

The sigma-1 receptor (σ1-R) and sigma-2 receptor (σ2-R) are potential drug targets for treatment of cancer, pain, depression, retinal degeneration and other neuronal diseases. Previous reports show that sigma-1 receptor modulates the activities of multiple channels. We are interested...

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Liu X, Fu Y, Yang H, et al. Potential independent action of sigma receptor ligands through inhibition of the Kv2.1 channel. Oncotarget. 2017;8(35):59345-59358doi: 10.18632/oncotarget.19581.
Liu, X., Fu, Y., Yang, H., Mavlyutov, T., Li, J., McCurdy, C. R., Guo, L. W., & Pattnaik, B. R. (2017). Potential independent action of sigma receptor ligands through inhibition of the Kv2.1 channel. Oncotarget, 8(35), 59345-59358. https://doi.org/10.18632/oncotarget.19581
Liu, Xinying, et al. "Potential independent action of sigma receptor ligands through inhibition of the Kv2.1 channel." Oncotarget vol. 8,35 (2017): 59345-59358. doi: https://doi.org/10.18632/oncotarget.19581
Liu X, Fu Y, Yang H, Mavlyutov T, Li J, McCurdy CR, Guo LW, Pattnaik BR. Potential independent action of sigma receptor ligands through inhibition of the Kv2.1 channel. Oncotarget. 2017 Jul 26;8(35):59345-59358. doi: 10.18632/oncotarget.19581. eCollection 2017 Aug 29. PMID: 28938641; PMCID: PMC5601737.
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APEX2-enhanced electron microscopy distinguishes sigma-1 receptor localization in the nucleoplasmic reticulum.

Oncotarget

Mavlyutov TA, Yang H, Epstein ML, Ruoho AE, Yang J, Guo LW.
PMID: 28881650
Oncotarget. 2017 May 16;8(31):51317-51330. doi: 10.18632/oncotarget.17906. eCollection 2017 Aug 01.

The sigma-1 receptor (Sig1R) is an endoplasmic reticulum chaperonin that is attracting tremendous interest as a potential anti-neurodegenerative target. While this membrane protein is known to reside in the inner nuclear envelope (NE) and influences transcription, apparent Sig1R presence...

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Mavlyutov TA, Yang H, Epstein ML, et al. APEX2-enhanced electron microscopy distinguishes sigma-1 receptor localization in the nucleoplasmic reticulum. Oncotarget. 2017;8(31):51317-51330doi: 10.18632/oncotarget.17906.
Mavlyutov, T. A., Yang, H., Epstein, M. L., Ruoho, A. E., Yang, J., & Guo, L. W. (2017). APEX2-enhanced electron microscopy distinguishes sigma-1 receptor localization in the nucleoplasmic reticulum. Oncotarget, 8(31), 51317-51330. https://doi.org/10.18632/oncotarget.17906
Mavlyutov, Timur A, et al. "APEX2-enhanced electron microscopy distinguishes sigma-1 receptor localization in the nucleoplasmic reticulum." Oncotarget vol. 8,31 (2017): 51317-51330. doi: https://doi.org/10.18632/oncotarget.17906
Mavlyutov TA, Yang H, Epstein ML, Ruoho AE, Yang J, Guo LW. APEX2-enhanced electron microscopy distinguishes sigma-1 receptor localization in the nucleoplasmic reticulum. Oncotarget. 2017 May 16;8(31):51317-51330. doi: 10.18632/oncotarget.17906. eCollection 2017 Aug 01. PMID: 28881650; PMCID: PMC5584251.
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Extracting entangled qubits from Majorana fermions in quantum dot chains through the measurement of parity.

Scientific reports

Dai L, Kuo W, Chung MC.
PMID: 26062033
Sci Rep. 2015 Jun 10;5:11188. doi: 10.1038/srep11188.

We propose a scheme for extracting entangled charge qubits from quantum-dot chains that support zero-energy edge modes. The edge mode is composed of Majorana fermions localized at the ends of each chain. The qubit, logically encoded in double quantum...

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Dai L, Kuo W, Chung MC. Extracting entangled qubits from Majorana fermions in quantum dot chains through the measurement of parity. Sci Rep. 2015;5:11188doi: 10.1038/srep11188.
Dai, L., Kuo, W., & Chung, M. C. (2015). Extracting entangled qubits from Majorana fermions in quantum dot chains through the measurement of parity. Scientific reports, 511188. https://doi.org/10.1038/srep11188
Dai, Li, et al. "Extracting entangled qubits from Majorana fermions in quantum dot chains through the measurement of parity." Scientific reports vol. 5 (2015): 11188. doi: https://doi.org/10.1038/srep11188
Dai L, Kuo W, Chung MC. Extracting entangled qubits from Majorana fermions in quantum dot chains through the measurement of parity. Sci Rep. 2015 Jun 10;5:11188. doi: 10.1038/srep11188. PMID: 26062033; PMCID: PMC5395956.
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Endovascular Declotting of Wall-Adherent Thrombi in Hemodialysis Vascular Access.

Acta Cardiologica Sinica

Hung CW, Lai CL, Hsieh MY, Kuo RC, Tsai KC, Lin L, Wu CC.
PMID: 27122779
Acta Cardiol Sin. 2014 Mar;30(2):128-35.

BACKGROUND: To investigate the efficacy, safety, and patency following treatment of wall-adherent thrombus in hemodialysis vascular access with a wall-contact device, the Arrow-Trerotola percutaneous thrombolytic device (PTD).METHODS: We retrospectively reviewed an existing database of procedures fulfilling the following criteria:...

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Hung CW, Lai CL, Hsieh MY, et al. Endovascular Declotting of Wall-Adherent Thrombi in Hemodialysis Vascular Access. Acta Cardiol Sin. 2014;30(2):128-35
Hung, C. W., Lai, C. L., Hsieh, M. Y., Kuo, R. C., Tsai, K. C., Lin, L., & Wu, C. C. (2014). Endovascular Declotting of Wall-Adherent Thrombi in Hemodialysis Vascular Access. Acta Cardiologica Sinica, 30(2), 128-35.
Hung, Chih-Wei, et al. "Endovascular Declotting of Wall-Adherent Thrombi in Hemodialysis Vascular Access." Acta Cardiologica Sinica vol. 30,2 (2014): 128-35.
Hung CW, Lai CL, Hsieh MY, Kuo RC, Tsai KC, Lin L, Wu CC. Endovascular Declotting of Wall-Adherent Thrombi in Hemodialysis Vascular Access. Acta Cardiol Sin. 2014 Mar;30(2):128-35. PMID: 27122779; PMCID: PMC4805018.
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Ocular adnexal lymphoma: Five case reports and a literature review.

Taiwan journal of ophthalmology

Tsen CL, Lin MC, Bee YS, Chen JL, Kuo NW, Sheu SJ.
PMID: 29018677
Taiwan J Ophthalmol. 2015 Apr-Jun;5(2):99-102. doi: 10.1016/j.tjo.2014.05.007. Epub 2014 Aug 01.

This article reports the clinical course and treatment of ocular adnexal lymphoma based on a retrospective review of five cases with a histologically approved ocular adnexal lymphoma at Kaohsiung Veterans General Hospital over 10 years. Extranodal B-cell lymphoma in...

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Tsen CL, Lin MC, Bee YS, et al. Ocular adnexal lymphoma: Five case reports and a literature review. Taiwan J Ophthalmol. 2014;5(2):99-102doi: 10.1016/j.tjo.2014.05.007.
Tsen, C. L., Lin, M. C., Bee, Y. S., Chen, J. L., Kuo, N. W., & Sheu, S. J. (2015). Ocular adnexal lymphoma: Five case reports and a literature review. Taiwan journal of ophthalmology, 5(2), 99-102. https://doi.org/10.1016/j.tjo.2014.05.007
Tsen, Chui-Lien, et al. "Ocular adnexal lymphoma: Five case reports and a literature review." Taiwan journal of ophthalmology vol. 5,2 (2015): 99-102. doi: https://doi.org/10.1016/j.tjo.2014.05.007
Tsen CL, Lin MC, Bee YS, Chen JL, Kuo NW, Sheu SJ. Ocular adnexal lymphoma: Five case reports and a literature review. Taiwan J Ophthalmol. 2015 Apr-Jun;5(2):99-102. doi: 10.1016/j.tjo.2014.05.007. Epub 2014 Aug 01. PMID: 29018677; PMCID: PMC5602737.
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Dung-Shen Downregulates the Synergistic Apoptotic Effects of Angiotensin II Plus Leu 27-IGF II on Cardiomyoblasts.

Acta Cardiologica Sinica

Chang KS, Lee NH, Kuo WW, Hu WS, Chang MH, Tsai FJ, Tsai KH, Yang YS, Chen TS, Huang CY.
PMID: 27122769
Acta Cardiol Sin. 2014 Jan;30(1):56-66.

BACKGROUND: Insulin growth factor II (IGFII) is expressed after ischemic stress in pig hearts and after myocardial infarction in humans. However, its receptor (IGFIIR) cannot be found in normal adult hearts. Moreover, a mouse IGFII overexpression model showed a...

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Chang KS, Lee NH, Kuo WW, et al. Dung-Shen Downregulates the Synergistic Apoptotic Effects of Angiotensin II Plus Leu 27-IGF II on Cardiomyoblasts. Acta Cardiol Sin. 2014;30(1):56-66
Chang, K. S., Lee, N. H., Kuo, W. W., Hu, W. S., Chang, M. H., Tsai, F. J., Tsai, K. H., Yang, Y. S., Chen, T. S., & Huang, C. Y. (2014). Dung-Shen Downregulates the Synergistic Apoptotic Effects of Angiotensin II Plus Leu 27-IGF II on Cardiomyoblasts. Acta Cardiologica Sinica, 30(1), 56-66.
Chang, Ko-Shih, et al. "Dung-Shen Downregulates the Synergistic Apoptotic Effects of Angiotensin II Plus Leu 27-IGF II on Cardiomyoblasts." Acta Cardiologica Sinica vol. 30,1 (2014): 56-66.
Chang KS, Lee NH, Kuo WW, Hu WS, Chang MH, Tsai FJ, Tsai KH, Yang YS, Chen TS, Huang CY. Dung-Shen Downregulates the Synergistic Apoptotic Effects of Angiotensin II Plus Leu 27-IGF II on Cardiomyoblasts. Acta Cardiol Sin. 2014 Jan;30(1):56-66. PMID: 27122769; PMCID: PMC4804822.
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In-Situ Measurement of High-Temperature Proton Exchange Membrane Fuel Cell Stack Using Flexible Five-in-One Micro-Sensor.

Sensors (Basel, Switzerland)

Lee CY, Weng FB, Kuo YW, Tsai CH, Cheng YT, Cheng CK, Lin JT.
PMID: 27763559
Sensors (Basel). 2016 Oct 18;16(10). doi: 10.3390/s16101731.

In the chemical reaction that proceeds in a high-temperature proton exchange membrane fuel cell stack (HT-PEMFC stack), the internal local temperature, voltage, pressure, flow and current nonuniformity may cause poor membrane material durability and nonuniform fuel distribution, thus influencing...

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Lee CY, Weng FB, Kuo YW, et al. In-Situ Measurement of High-Temperature Proton Exchange Membrane Fuel Cell Stack Using Flexible Five-in-One Micro-Sensor. Sensors (Basel). 2016;16(10)doi: 10.3390/s16101731.
Lee, C. Y., Weng, F. B., Kuo, Y. W., Tsai, C. H., Cheng, Y. T., Cheng, C. K., & Lin, J. T. (2016). In-Situ Measurement of High-Temperature Proton Exchange Membrane Fuel Cell Stack Using Flexible Five-in-One Micro-Sensor. Sensors (Basel, Switzerland), 16(10), . https://doi.org/10.3390/s16101731
Lee, Chi-Yuan, et al. "In-Situ Measurement of High-Temperature Proton Exchange Membrane Fuel Cell Stack Using Flexible Five-in-One Micro-Sensor." Sensors (Basel, Switzerland) vol. 16,10 (2016). doi: https://doi.org/10.3390/s16101731
Lee CY, Weng FB, Kuo YW, Tsai CH, Cheng YT, Cheng CK, Lin JT. In-Situ Measurement of High-Temperature Proton Exchange Membrane Fuel Cell Stack Using Flexible Five-in-One Micro-Sensor. Sensors (Basel). 2016 Oct 18;16(10). doi: 10.3390/s16101731. PMID: 27763559; PMCID: PMC5087516.
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MicroRNA160 Modulates Plant Development and Heat Shock Protein Gene Expression to Mediate Heat Tolerance in .

Frontiers in plant science

Lin JS, Kuo CC, Yang IC, Tsai WA, Shen YH, Lin CC, Liang YC, Li YC, Kuo YW, King YC, Lai HM, Jeng ST.
PMID: 29449855
Front Plant Sci. 2018 Feb 01;9:68. doi: 10.3389/fpls.2018.00068. eCollection 2018.

Global warming is causing a negative impact on plant growth and adversely impacts on crop yield. MicroRNAs (miRNAs) are critical in regulating the expression of genes involved in plant development as well as defense responses. The effects of miRNAs...

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Lin JS, Kuo CC, Yang IC, et al. MicroRNA160 Modulates Plant Development and Heat Shock Protein Gene Expression to Mediate Heat Tolerance in . Front Plant Sci. 2018;9:68doi: 10.3389/fpls.2018.00068.
Lin, J. S., Kuo, C. C., Yang, I. C., Tsai, W. A., Shen, Y. H., Lin, C. C., Liang, Y. C., Li, Y. C., Kuo, Y. W., King, Y. C., Lai, H. M., & Jeng, S. T. (2018). MicroRNA160 Modulates Plant Development and Heat Shock Protein Gene Expression to Mediate Heat Tolerance in . Frontiers in plant science, 968. https://doi.org/10.3389/fpls.2018.00068
Lin, Jeng-Shane, et al. "MicroRNA160 Modulates Plant Development and Heat Shock Protein Gene Expression to Mediate Heat Tolerance in ." Frontiers in plant science vol. 9 (2018): 68. doi: https://doi.org/10.3389/fpls.2018.00068
Lin JS, Kuo CC, Yang IC, Tsai WA, Shen YH, Lin CC, Liang YC, Li YC, Kuo YW, King YC, Lai HM, Jeng ST. MicroRNA160 Modulates Plant Development and Heat Shock Protein Gene Expression to Mediate Heat Tolerance in . Front Plant Sci. 2018 Feb 01;9:68. doi: 10.3389/fpls.2018.00068. eCollection 2018. PMID: 29449855; PMCID: PMC5799662.
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Lipid Nanoparticle Delivery of siRNA to Silence Neuronal Gene Expression in the Brain.

Molecular therapy. Nucleic acids

Rungta RL, Choi HB, Lin PJ, Ko RW, Ashby D, Nair J, Manoharan M, Cullis PR, Macvicar BA.
PMID: 24301867
Mol Ther Nucleic Acids. 2013 Dec 03;2:e136. doi: 10.1038/mtna.2013.65.

Manipulation of gene expression in the brain is fundamental for understanding the function of proteins involved in neuronal processes. In this article, we show a method for using small interfering RNA (siRNA) in lipid nanoparticles (LNPs) to efficiently silence...

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Rungta RL, Choi HB, Lin PJ, et al. Lipid Nanoparticle Delivery of siRNA to Silence Neuronal Gene Expression in the Brain. Mol Ther Nucleic Acids. 2013;2:e136doi: 10.1038/mtna.2013.65.
Rungta, R. L., Choi, H. B., Lin, P. J., Ko, R. W., Ashby, D., Nair, J., Manoharan, M., Cullis, P. R., & Macvicar, B. A. (2013). Lipid Nanoparticle Delivery of siRNA to Silence Neuronal Gene Expression in the Brain. Molecular therapy. Nucleic acids, 2e136. https://doi.org/10.1038/mtna.2013.65
Rungta, Ravi L, et al. "Lipid Nanoparticle Delivery of siRNA to Silence Neuronal Gene Expression in the Brain." Molecular therapy. Nucleic acids vol. 2 (2013): e136. doi: https://doi.org/10.1038/mtna.2013.65
Rungta RL, Choi HB, Lin PJ, Ko RW, Ashby D, Nair J, Manoharan M, Cullis PR, Macvicar BA. Lipid Nanoparticle Delivery of siRNA to Silence Neuronal Gene Expression in the Brain. Mol Ther Nucleic Acids. 2013 Dec 03;2:e136. doi: 10.1038/mtna.2013.65. PMID: 24301867; PMCID: PMC3889191.
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[No title available]

International journal of systematic and evolutionary microbiology

Li YQ, Li L, Fu YS, Cui ZQ, Duan YQ, Salam N, Guo JW, Chen W, Li WJ.
PMID: 26739161
Int J Syst Evol Microbiol. 2016 Mar;66(3):1287-1292. doi: 10.1099/ijsem.0.000876. Epub 2016 Jan 05.

A Gram-stain-positive, aerobic, rod-shaped, non-motile actinomycete strain, designated EGI 60007

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Li YQ, Li L, Fu YS, et al. . Int J Syst Evol Microbiol. 2016;66(3):1287-1292doi: 10.1099/ijsem.0.000876.
Li, Y. Q., Li, L., Fu, Y. S., Cui, Z. Q., Duan, Y. Q., Salam, N., Guo, J. W., Chen, W., & Li, W. J. (2016). . International journal of systematic and evolutionary microbiology, 66(3), 1287-1292. https://doi.org/10.1099/ijsem.0.000876
Li, Yan-Qiong, et al. "." International journal of systematic and evolutionary microbiology vol. 66,3 (2016): 1287-1292. doi: https://doi.org/10.1099/ijsem.0.000876
Li YQ, Li L, Fu YS, Cui ZQ, Duan YQ, Salam N, Guo JW, Chen W, Li WJ. . Int J Syst Evol Microbiol. 2016 Mar;66(3):1287-1292. doi: 10.1099/ijsem.0.000876. Epub 2016 Jan 05. PMID: 26739161.
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Inhibition of HDAC3- and HDAC6-Promoted Survivin Expression Plays an Important Role in SAHA-Induced Autophagy and Viability Reduction in Breast Cancer Cells.

Frontiers in pharmacology

Lee JY, Kuo CW, Tsai SL, Cheng SM, Chen SH, Chan HH, Lin CH, Lin KY, Li CF, Kanwar JR, Leung EY, Cheung CC, Huang WJ, Wang YC, Cheung CH.
PMID: 27065869
Front Pharmacol. 2016 Mar 31;7:81. doi: 10.3389/fphar.2016.00081. eCollection 2016.

SAHA is a class I HDAC/HDAC6 co-inhibitor and an autophagy inducer currently undergoing clinical investigations in breast cancer patients. However, the molecular mechanism of action of SAHA in breast cancer cells remains unclear. In this study, we found that...

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Lee JY, Kuo CW, Tsai SL, et al. Inhibition of HDAC3- and HDAC6-Promoted Survivin Expression Plays an Important Role in SAHA-Induced Autophagy and Viability Reduction in Breast Cancer Cells. Front Pharmacol. 2016;7:81doi: 10.3389/fphar.2016.00081.
Lee, J. Y., Kuo, C. W., Tsai, S. L., Cheng, S. M., Chen, S. H., Chan, H. H., Lin, C. H., Lin, K. Y., Li, C. F., Kanwar, J. R., Leung, E. Y., Cheung, C. C., Huang, W. J., Wang, Y. C., & Cheung, C. H. (2016). Inhibition of HDAC3- and HDAC6-Promoted Survivin Expression Plays an Important Role in SAHA-Induced Autophagy and Viability Reduction in Breast Cancer Cells. Frontiers in pharmacology, 781. https://doi.org/10.3389/fphar.2016.00081
Lee, Jane Ying-Chieh, et al. "Inhibition of HDAC3- and HDAC6-Promoted Survivin Expression Plays an Important Role in SAHA-Induced Autophagy and Viability Reduction in Breast Cancer Cells." Frontiers in pharmacology vol. 7 (2016): 81. doi: https://doi.org/10.3389/fphar.2016.00081
Lee JY, Kuo CW, Tsai SL, Cheng SM, Chen SH, Chan HH, Lin CH, Lin KY, Li CF, Kanwar JR, Leung EY, Cheung CC, Huang WJ, Wang YC, Cheung CH. Inhibition of HDAC3- and HDAC6-Promoted Survivin Expression Plays an Important Role in SAHA-Induced Autophagy and Viability Reduction in Breast Cancer Cells. Front Pharmacol. 2016 Mar 31;7:81. doi: 10.3389/fphar.2016.00081. eCollection 2016. PMID: 27065869; PMCID: PMC4814469.
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Anthocyanin Attenuates Doxorubicin-Induced Cardiomyotoxicity via Estrogen Receptor-α/β and Stabilizes HSF1 to Inhibit the IGF-IIR Apoptotic Pathway.

International journal of molecular sciences

Huang PC, Kuo WW, Shen CY, Chen YF, Lin YM, Ho TJ, Padma VV, Lo JF, Huang CY, Huang CY.
PMID: 27657062
Int J Mol Sci. 2016 Sep 21;17(9). doi: 10.3390/ijms17091588.

Doxorubicin (Dox) is extensively used for chemotherapy in different types of cancer, but its use is limited to because of its cardiotoxicity. Our previous studies found that doxorubicin-induced insulin-like growth factor II receptor (IGF-IIR) accumulation causes cardiomyocytes apoptosis via...

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Huang PC, Kuo WW, Shen CY, et al. Anthocyanin Attenuates Doxorubicin-Induced Cardiomyotoxicity via Estrogen Receptor-α/β and Stabilizes HSF1 to Inhibit the IGF-IIR Apoptotic Pathway. Int J Mol Sci. 2016;17(9)doi: 10.3390/ijms17091588.
Huang, P. C., Kuo, W. W., Shen, C. Y., Chen, Y. F., Lin, Y. M., Ho, T. J., Padma, V. V., Lo, J. F., Huang, C. Y., & Huang, C. Y. (2016). Anthocyanin Attenuates Doxorubicin-Induced Cardiomyotoxicity via Estrogen Receptor-α/β and Stabilizes HSF1 to Inhibit the IGF-IIR Apoptotic Pathway. International journal of molecular sciences, 17(9), . https://doi.org/10.3390/ijms17091588
Huang, Pei-Chen, et al. "Anthocyanin Attenuates Doxorubicin-Induced Cardiomyotoxicity via Estrogen Receptor-α/β and Stabilizes HSF1 to Inhibit the IGF-IIR Apoptotic Pathway." International journal of molecular sciences vol. 17,9 (2016). doi: https://doi.org/10.3390/ijms17091588
Huang PC, Kuo WW, Shen CY, Chen YF, Lin YM, Ho TJ, Padma VV, Lo JF, Huang CY, Huang CY. Anthocyanin Attenuates Doxorubicin-Induced Cardiomyotoxicity via Estrogen Receptor-α/β and Stabilizes HSF1 to Inhibit the IGF-IIR Apoptotic Pathway. Int J Mol Sci. 2016 Sep 21;17(9). doi: 10.3390/ijms17091588. PMID: 27657062; PMCID: PMC5037853.
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