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The N terminus of Orai1 couples to the AKAP79 signaling complex to drive NFAT1 activation by local Ca.

Proceedings of the National Academy of Sciences of the United States of America

Kar P, Lin YP, Bhardwaj R, Tucker CJ, Bird GS, Hediger MA, Monico C, Amin N, Parekh AB.
PMID: 33941685
Proc Natl Acad Sci U S A. 2021 May 11;118(19). doi: 10.1073/pnas.2012908118.

To avoid conflicting and deleterious outcomes, eukaryotic cells often confine second messengers to spatially restricted subcompartments. The smallest signaling unit is the Ca

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Kar P, Lin YP, Bhardwaj R, et al. The N terminus of Orai1 couples to the AKAP79 signaling complex to drive NFAT1 activation by local Ca. Proc Natl Acad Sci U S A. 2021;118(19)doi: 10.1073/pnas.2012908118.
Kar, P., Lin, Y. P., Bhardwaj, R., Tucker, C. J., Bird, G. S., Hediger, M. A., Monico, C., Amin, N., & Parekh, A. B. (2021). The N terminus of Orai1 couples to the AKAP79 signaling complex to drive NFAT1 activation by local Ca. Proceedings of the National Academy of Sciences of the United States of America, 118(19), . https://doi.org/10.1073/pnas.2012908118
Kar, Pulak, et al. "The N terminus of Orai1 couples to the AKAP79 signaling complex to drive NFAT1 activation by local Ca." Proceedings of the National Academy of Sciences of the United States of America vol. 118,19 (2021). doi: https://doi.org/10.1073/pnas.2012908118
Kar P, Lin YP, Bhardwaj R, Tucker CJ, Bird GS, Hediger MA, Monico C, Amin N, Parekh AB. The N terminus of Orai1 couples to the AKAP79 signaling complex to drive NFAT1 activation by local Ca. Proc Natl Acad Sci U S A. 2021 May 11;118(19). doi: 10.1073/pnas.2012908118. PMID: 33941685; PMCID: PMC8126794.
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The Distinct Immune Nature of the Fetal Inflammatory Response Syndrome Type I and Type II.

ImmunoHorizons

Para R, Romero R, Miller D, Galaz J, Done B, Peyvandipour A, Gershater M, Tao L, Motomura K, Ruden DM, Isherwood J, Jung E, Kanninen T, Pique-Regi R, Tarca AL, Gomez-Lopez N.
PMID: 34521696
Immunohorizons. 2021 Sep 14;5(9):735-751. doi: 10.4049/immunohorizons.2100047.

Fetal inflammatory response syndrome (FIRS) is strongly associated with neonatal morbidity and mortality and can be classified as type I or type II. Clinically, FIRS type I and type II are considered as distinct syndromes, yet the molecular underpinnings...

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Para R, Romero R, Miller D, et al. The Distinct Immune Nature of the Fetal Inflammatory Response Syndrome Type I and Type II. Immunohorizons. 2021;5(9):735-751doi: 10.4049/immunohorizons.2100047.
Para, R., Romero, R., Miller, D., Galaz, J., Done, B., Peyvandipour, A., Gershater, M., Tao, L., Motomura, K., Ruden, D. M., Isherwood, J., Jung, E., Kanninen, T., Pique-Regi, R., Tarca, A. L., & Gomez-Lopez, N. (2021). The Distinct Immune Nature of the Fetal Inflammatory Response Syndrome Type I and Type II. ImmunoHorizons, 5(9), 735-751. https://doi.org/10.4049/immunohorizons.2100047
Para, Robert, et al. "The Distinct Immune Nature of the Fetal Inflammatory Response Syndrome Type I and Type II." ImmunoHorizons vol. 5,9 (2021): 735-751. doi: https://doi.org/10.4049/immunohorizons.2100047
Para R, Romero R, Miller D, Galaz J, Done B, Peyvandipour A, Gershater M, Tao L, Motomura K, Ruden DM, Isherwood J, Jung E, Kanninen T, Pique-Regi R, Tarca AL, Gomez-Lopez N. The Distinct Immune Nature of the Fetal Inflammatory Response Syndrome Type I and Type II. Immunohorizons. 2021 Sep 14;5(9):735-751. doi: 10.4049/immunohorizons.2100047. PMID: 34521696.
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Human oral mucosa cell atlas reveals a stromal-neutrophil axis regulating tissue immunity.

Cell

Williams DW, Greenwell-Wild T, Brenchley L, Dutzan N, Overmiller A, Sawaya AP, Webb S, Martin D, Hajishengallis G, Divaris K, Morasso M, Haniffa M, Moutsopoulos NM.
PMID: 34129837
Cell. 2021 Jul 22;184(15):4090-4104.e15. doi: 10.1016/j.cell.2021.05.013. Epub 2021 Jun 14.

The oral mucosa remains an understudied barrier tissue. This is a site of rich exposure to antigens and commensals, and a tissue susceptible to one of the most prevalent human inflammatory diseases, periodontitis. To aid in understanding tissue-specific pathophysiology,...

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Williams DW, Greenwell-Wild T, Brenchley L, et al. Human oral mucosa cell atlas reveals a stromal-neutrophil axis regulating tissue immunity. Cell. 2021;184(15):4090-4104.e15doi: 10.1016/j.cell.2021.05.013.
Williams, D. W., Greenwell-Wild, T., Brenchley, L., Dutzan, N., Overmiller, A., Sawaya, A. P., Webb, S., Martin, D., Hajishengallis, G., Divaris, K., Morasso, M., Haniffa, M., & Moutsopoulos, N. M. (2021). Human oral mucosa cell atlas reveals a stromal-neutrophil axis regulating tissue immunity. Cell, 184(15), 4090-4104.e15. https://doi.org/10.1016/j.cell.2021.05.013
Williams, Drake Winslow, et al. "Human oral mucosa cell atlas reveals a stromal-neutrophil axis regulating tissue immunity." Cell vol. 184,15 (2021): 4090-4104.e15. doi: https://doi.org/10.1016/j.cell.2021.05.013
Williams DW, Greenwell-Wild T, Brenchley L, Dutzan N, Overmiller A, Sawaya AP, Webb S, Martin D, Hajishengallis G, Divaris K, Morasso M, Haniffa M, Moutsopoulos NM. Human oral mucosa cell atlas reveals a stromal-neutrophil axis regulating tissue immunity. Cell. 2021 Jul 22;184(15):4090-4104.e15. doi: 10.1016/j.cell.2021.05.013. Epub 2021 Jun 14. PMID: 34129837; PMCID: PMC8359928.
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Editorial: Thymic Epithelial Cells: New Insights Into the Essential Driving Force of T-Cell Differentiation.

Frontiers in immunology

Bosticardo M, Ohigashi I, Cowan JE, Alves NL.
PMID: 34484248
Front Immunol. 2021 Aug 13;12:744623. doi: 10.3389/fimmu.2021.744623. eCollection 2021.

No abstract available.

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Bosticardo M, Ohigashi I, Cowan JE, et al. Editorial: Thymic Epithelial Cells: New Insights Into the Essential Driving Force of T-Cell Differentiation. Front Immunol. 2021;12:744623doi: 10.3389/fimmu.2021.744623.
Bosticardo, M., Ohigashi, I., Cowan, J. E., & Alves, N. L. (2021). Editorial: Thymic Epithelial Cells: New Insights Into the Essential Driving Force of T-Cell Differentiation. Frontiers in immunology, 12744623. https://doi.org/10.3389/fimmu.2021.744623
Bosticardo, Marita, et al. "Editorial: Thymic Epithelial Cells: New Insights Into the Essential Driving Force of T-Cell Differentiation." Frontiers in immunology vol. 12 (2021): 744623. doi: https://doi.org/10.3389/fimmu.2021.744623
Bosticardo M, Ohigashi I, Cowan JE, Alves NL. Editorial: Thymic Epithelial Cells: New Insights Into the Essential Driving Force of T-Cell Differentiation. Front Immunol. 2021 Aug 13;12:744623. doi: 10.3389/fimmu.2021.744623. eCollection 2021. PMID: 34484248; PMCID: PMC8414565.
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Complete vertebrate mitogenomes reveal widespread repeats and gene duplications.

Genome biology

Formenti G, Rhie A, Balacco J, Haase B, Mountcastle J, Fedrigo O, Brown S, Capodiferro MR, Al-Ajli FO, Ambrosini R, Houde P, Koren S, Oliver K, Smith M, Skelton J, Betteridge E, Dolucan J, Corton C, Bista I, Torrance J, Tracey A, Wood J, Uliano-Silva M, Howe K, McCarthy S, Winkler S, Kwak W, Korlach J, Fungtammasan A, Fordham D, Costa V, Mayes S, Chiara M, Horner DS, Myers E, Durbin R, Achilli A, Braun EL, Phillippy AM, Jarvis ED.
PMID: 33910595
Genome Biol. 2021 Apr 29;22(1):120. doi: 10.1186/s13059-021-02336-9.

BACKGROUND: Modern sequencing technologies should make the assembly of the relatively small mitochondrial genomes an easy undertaking. However, few tools exist that address mitochondrial assembly directly.RESULTS: As part of the Vertebrate Genomes Project (VGP) we develop mitoVGP, a fully...

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Formenti G, Rhie A, Balacco J, et al. Complete vertebrate mitogenomes reveal widespread repeats and gene duplications. Genome Biol. 2021;22(1):120doi: 10.1186/s13059-021-02336-9.
Formenti, G., Rhie, A., Balacco, J., Haase, B., Mountcastle, J., Fedrigo, O., Brown, S., Capodiferro, M. R., Al-Ajli, F. O., Ambrosini, R., Houde, P., Koren, S., Oliver, K., Smith, M., Skelton, J., Betteridge, E., Dolucan, J., Corton, C., Bista, I., Torrance, J., Tracey, A., Wood, J., Uliano-Silva, M., Howe, K., McCarthy, S., Winkler, S., Kwak, W., Korlach, J., Fungtammasan, A., Fordham, D., Costa, V., Mayes, S., Chiara, M., Horner, D. S., Myers, E., Durbin, R., Achilli, A., Braun, E. L., Phillippy, A. M., Jarvis, E. D. (2021). Complete vertebrate mitogenomes reveal widespread repeats and gene duplications. Genome biology, 22(1), 120. https://doi.org/10.1186/s13059-021-02336-9
Formenti, Giulio, et al. "Complete vertebrate mitogenomes reveal widespread repeats and gene duplications." Genome biology vol. 22,1 (2021): 120. doi: https://doi.org/10.1186/s13059-021-02336-9
Formenti G, Rhie A, Balacco J, Haase B, Mountcastle J, Fedrigo O, Brown S, Capodiferro MR, Al-Ajli FO, Ambrosini R, Houde P, Koren S, Oliver K, Smith M, Skelton J, Betteridge E, Dolucan J, Corton C, Bista I, Torrance J, Tracey A, Wood J, Uliano-Silva M, Howe K, McCarthy S, Winkler S, Kwak W, Korlach J, Fungtammasan A, Fordham D, Costa V, Mayes S, Chiara M, Horner DS, Myers E, Durbin R, Achilli A, Braun EL, Phillippy AM, Jarvis ED. Complete vertebrate mitogenomes reveal widespread repeats and gene duplications. Genome Biol. 2021 Apr 29;22(1):120. doi: 10.1186/s13059-021-02336-9. PMID: 33910595; PMCID: PMC8082918.
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Recurrent Nodular Regenerative Hyperplasia Following Liver Transplantation in Common Variable Immunodeficiency.

Hepatology (Baltimore, Md.)

Hercun J, Parikh E, Kleiner DE, Fuss I, Uzel G, Strober W, Koh C, Holland SM, Heller T.
PMID: 33638875
Hepatology. 2021 Sep;74(3):1698-1701. doi: 10.1002/hep.31775. Epub 2021 Aug 26.

No abstract available.

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Hercun J, Parikh E, Kleiner DE, et al. Recurrent Nodular Regenerative Hyperplasia Following Liver Transplantation in Common Variable Immunodeficiency. Hepatology. 2021;74(3):1698-1701doi: 10.1002/hep.31775.
Hercun, J., Parikh, E., Kleiner, D. E., Fuss, I., Uzel, G., Strober, W., Koh, C., Holland, S. M., & Heller, T. (2021). Recurrent Nodular Regenerative Hyperplasia Following Liver Transplantation in Common Variable Immunodeficiency. Hepatology (Baltimore, Md.), 74(3), 1698-1701. https://doi.org/10.1002/hep.31775
Hercun, Julian, et al. "Recurrent Nodular Regenerative Hyperplasia Following Liver Transplantation in Common Variable Immunodeficiency." Hepatology (Baltimore, Md.) vol. 74,3 (2021): 1698-1701. doi: https://doi.org/10.1002/hep.31775
Hercun J, Parikh E, Kleiner DE, Fuss I, Uzel G, Strober W, Koh C, Holland SM, Heller T. Recurrent Nodular Regenerative Hyperplasia Following Liver Transplantation in Common Variable Immunodeficiency. Hepatology. 2021 Sep;74(3):1698-1701. doi: 10.1002/hep.31775. Epub 2021 Aug 26. PMID: 33638875; PMCID: PMC8390566.
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Red Cell Distribution Width and Dementia Among Rural-Dwelling Older Adults: The MIND-China Study.

Journal of Alzheimer's disease : JAD

Jiang Z, Han X, Wang Y, Hou T, Cong L, Tang S, Han X, Ngandu T, Kivipelto M, Winblad B, Launer LJ, Du Y, Qiu C.
PMID: 34420964
J Alzheimers Dis. 2021;83(3):1187-1198. doi: 10.3233/JAD-210517.

BACKGROUND: Evidence has emerged that anemia is associated with dementia, but data on the relationships of red blood cell distribution width (RDW) with dementia and cognitive function in older adults are sparse.OBJECTIVE: We sought to investigate the associations of...

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Jiang Z, Han X, Wang Y, et al. Red Cell Distribution Width and Dementia Among Rural-Dwelling Older Adults: The MIND-China Study. J Alzheimers Dis. 2021;83(3):1187-1198doi: 10.3233/JAD-210517.
Jiang, Z., Han, X., Wang, Y., Hou, T., Cong, L., Tang, S., Han, X., Ngandu, T., Kivipelto, M., Winblad, B., Launer, L. J., Du, Y., & Qiu, C. (2021). Red Cell Distribution Width and Dementia Among Rural-Dwelling Older Adults: The MIND-China Study. Journal of Alzheimer's disease : JAD, 83(3), 1187-1198. https://doi.org/10.3233/JAD-210517
Jiang, Ziying, et al. "Red Cell Distribution Width and Dementia Among Rural-Dwelling Older Adults: The MIND-China Study." Journal of Alzheimer's disease : JAD vol. 83,3 (2021): 1187-1198. doi: https://doi.org/10.3233/JAD-210517
Jiang Z, Han X, Wang Y, Hou T, Cong L, Tang S, Han X, Ngandu T, Kivipelto M, Winblad B, Launer LJ, Du Y, Qiu C. Red Cell Distribution Width and Dementia Among Rural-Dwelling Older Adults: The MIND-China Study. J Alzheimers Dis. 2021;83(3):1187-1198. doi: 10.3233/JAD-210517. PMID: 34420964; PMCID: PMC8543291.
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Microbiota triggers STING-type I IFN-dependent monocyte reprogramming of the tumor microenvironment.

Cell

Lam KC, Araya RE, Huang A, Chen Q, Di Modica M, Rodrigues RR, Lopès A, Johnson SB, Schwarz B, Bohrnsen E, Cogdill AP, Bosio CM, Wargo JA, Lee MP, Goldszmid RS.
PMID: 34624222
Cell. 2021 Oct 14;184(21):5338-5356.e21. doi: 10.1016/j.cell.2021.09.019. Epub 2021 Oct 07.

The tumor microenvironment (TME) influences cancer progression and therapy response. Therefore, understanding what regulates the TME immune compartment is vital. Here we show that microbiota signals program mononuclear phagocytes in the TME toward immunostimulatory monocytes and dendritic cells (DCs)....

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Lam KC, Araya RE, Huang A, et al. Microbiota triggers STING-type I IFN-dependent monocyte reprogramming of the tumor microenvironment. Cell. 2021;184(21):5338-5356.e21doi: 10.1016/j.cell.2021.09.019.
Lam, K. C., Araya, R. E., Huang, A., Chen, Q., Di Modica, M., Rodrigues, R. R., Lopès, A., Johnson, S. B., Schwarz, B., Bohrnsen, E., Cogdill, A. P., Bosio, C. M., Wargo, J. A., Lee, M. P., & Goldszmid, R. S. (2021). Microbiota triggers STING-type I IFN-dependent monocyte reprogramming of the tumor microenvironment. Cell, 184(21), 5338-5356.e21. https://doi.org/10.1016/j.cell.2021.09.019
Lam, Khiem C, et al. "Microbiota triggers STING-type I IFN-dependent monocyte reprogramming of the tumor microenvironment." Cell vol. 184,21 (2021): 5338-5356.e21. doi: https://doi.org/10.1016/j.cell.2021.09.019
Lam KC, Araya RE, Huang A, Chen Q, Di Modica M, Rodrigues RR, Lopès A, Johnson SB, Schwarz B, Bohrnsen E, Cogdill AP, Bosio CM, Wargo JA, Lee MP, Goldszmid RS. Microbiota triggers STING-type I IFN-dependent monocyte reprogramming of the tumor microenvironment. Cell. 2021 Oct 14;184(21):5338-5356.e21. doi: 10.1016/j.cell.2021.09.019. Epub 2021 Oct 07. PMID: 34624222; PMCID: PMC8650838.
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Novel Humanized Mesothelin-Expressing Genetically Engineered Mouse Models Underscore Challenges in Delivery of Complex Therapeutics to Pancreatic Cancers.

Molecular cancer therapeutics

Hagerty B, O'Sullivan TN, Zhang X, Collins NK, Custer Lawrence W, Bassel LL, Pate N, Xu J, Guerin TM, Kozlov S, Alewine C.
PMID: 34315768
Mol Cancer Ther. 2021 Oct;20(10):2082-2092. doi: 10.1158/1535-7163.MCT-21-0017. Epub 2021 Jul 26.

Antibody-based therapies designed for human use frequently fail to cross-react with the murine isoform of their target. Because of this problem, preclinical studies of antibody-based mesothelin (Msl)-targeted therapeutics in immunocompetent systems have been limited by the lack of suitable...

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Hagerty B, O'Sullivan TN, Zhang X, et al. Novel Humanized Mesothelin-Expressing Genetically Engineered Mouse Models Underscore Challenges in Delivery of Complex Therapeutics to Pancreatic Cancers. Mol Cancer Ther. 2021;20(10):2082-2092doi: 10.1158/1535-7163.MCT-21-0017.
Hagerty, B., O'Sullivan, T. N., Zhang, X., Collins, N. K., Custer Lawrence, W., Bassel, L. L., Pate, N., Xu, J., Guerin, T. M., Kozlov, S., & Alewine, C. (2021). Novel Humanized Mesothelin-Expressing Genetically Engineered Mouse Models Underscore Challenges in Delivery of Complex Therapeutics to Pancreatic Cancers. Molecular cancer therapeutics, 20(10), 2082-2092. https://doi.org/10.1158/1535-7163.MCT-21-0017
Hagerty, Brendan, et al. "Novel Humanized Mesothelin-Expressing Genetically Engineered Mouse Models Underscore Challenges in Delivery of Complex Therapeutics to Pancreatic Cancers." Molecular cancer therapeutics vol. 20,10 (2021): 2082-2092. doi: https://doi.org/10.1158/1535-7163.MCT-21-0017
Hagerty B, O'Sullivan TN, Zhang X, Collins NK, Custer Lawrence W, Bassel LL, Pate N, Xu J, Guerin TM, Kozlov S, Alewine C. Novel Humanized Mesothelin-Expressing Genetically Engineered Mouse Models Underscore Challenges in Delivery of Complex Therapeutics to Pancreatic Cancers. Mol Cancer Ther. 2021 Oct;20(10):2082-2092. doi: 10.1158/1535-7163.MCT-21-0017. Epub 2021 Jul 26. PMID: 34315768; PMCID: PMC8492545.
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The effects of housing density on mouse thermal physiology depend on sex and ambient temperature.

Molecular metabolism

Škop V, Xiao C, Liu N, Gavrilova O, Reitman ML.
PMID: 34478905
Mol Metab. 2021 Nov;53:101332. doi: 10.1016/j.molmet.2021.101332. Epub 2021 Sep 01.

OBJECTIVE: To improve understanding of mouse energy homeostasis and its applicability to humans, we quantitated the effects of housing density on mouse thermal physiology in both sexes.METHODS: Littermate wild type and Brs3-null mice were single- or group- (three per...

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Škop V, Xiao C, Liu N, et al. The effects of housing density on mouse thermal physiology depend on sex and ambient temperature. Mol Metab. 2021;53:101332doi: 10.1016/j.molmet.2021.101332.
Škop, V., Xiao, C., Liu, N., Gavrilova, O., & Reitman, M. L. (2021). The effects of housing density on mouse thermal physiology depend on sex and ambient temperature. Molecular metabolism, 53101332. https://doi.org/10.1016/j.molmet.2021.101332
Škop, Vojtěch, et al. "The effects of housing density on mouse thermal physiology depend on sex and ambient temperature." Molecular metabolism vol. 53 (2021): 101332. doi: https://doi.org/10.1016/j.molmet.2021.101332
Škop V, Xiao C, Liu N, Gavrilova O, Reitman ML. The effects of housing density on mouse thermal physiology depend on sex and ambient temperature. Mol Metab. 2021 Nov;53:101332. doi: 10.1016/j.molmet.2021.101332. Epub 2021 Sep 01. PMID: 34478905; PMCID: PMC8463779.
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Luminal microvesicles uniquely influence translocating bacteria after SIV infection.

Mucosal immunology

Flynn JK, Langner CA, Karmele EP, Baker PJ, Pei L, Gorfu EG, Bochart RM, Santiana M, Smelkinson MG, Nutman TB, Altan-Bonnet N, Bosinger SE, Kelsall BL, Brenchley JM, Ortiz AM.
PMID: 33731830
Mucosal Immunol. 2021 Jul;14(4):937-948. doi: 10.1038/s41385-021-00393-8. Epub 2021 Mar 17.

Microbial translocation contributes to persistent inflammation in both treated and untreated HIV infection. Although translocation is due in part to a disintegration of the intestinal epithelial barrier, there is a bias towards the translocation of Proteobacteria. We hypothesized that...

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Flynn JK, Langner CA, Karmele EP, et al. Luminal microvesicles uniquely influence translocating bacteria after SIV infection. Mucosal Immunol. 2021;14(4):937-948doi: 10.1038/s41385-021-00393-8.
Flynn, J. K., Langner, C. A., Karmele, E. P., Baker, P. J., Pei, L., Gorfu, E. G., Bochart, R. M., Santiana, M., Smelkinson, M. G., Nutman, T. B., Altan-Bonnet, N., Bosinger, S. E., Kelsall, B. L., Brenchley, J. M., & Ortiz, A. M. (2021). Luminal microvesicles uniquely influence translocating bacteria after SIV infection. Mucosal immunology, 14(4), 937-948. https://doi.org/10.1038/s41385-021-00393-8
Flynn, Jacob K, et al. "Luminal microvesicles uniquely influence translocating bacteria after SIV infection." Mucosal immunology vol. 14,4 (2021): 937-948. doi: https://doi.org/10.1038/s41385-021-00393-8
Flynn JK, Langner CA, Karmele EP, Baker PJ, Pei L, Gorfu EG, Bochart RM, Santiana M, Smelkinson MG, Nutman TB, Altan-Bonnet N, Bosinger SE, Kelsall BL, Brenchley JM, Ortiz AM. Luminal microvesicles uniquely influence translocating bacteria after SIV infection. Mucosal Immunol. 2021 Jul;14(4):937-948. doi: 10.1038/s41385-021-00393-8. Epub 2021 Mar 17. PMID: 33731830; PMCID: PMC8225551.
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The long and short of it: long noncoding RNAs in neural development and diseases.

Frontiers in bioscience (Landmark edition)

Zhang J.
PMID: 34455757
Front Biosci (Landmark Ed). 2021 Aug 30;26(8):258-261. doi: 10.52586/4941.

No abstract present.

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Zhang J. The long and short of it: long noncoding RNAs in neural development and diseases. Front Biosci (Landmark Ed). 2021;26(8):258-261doi: 10.52586/4941.
Zhang, J. (2021). The long and short of it: long noncoding RNAs in neural development and diseases. Frontiers in bioscience (Landmark edition), 26(8), 258-261. https://doi.org/10.52586/4941
Zhang, Jinwei. "The long and short of it: long noncoding RNAs in neural development and diseases." Frontiers in bioscience (Landmark edition) vol. 26,8 (2021): 258-261. doi: https://doi.org/10.52586/4941
Zhang J. The long and short of it: long noncoding RNAs in neural development and diseases. Front Biosci (Landmark Ed). 2021 Aug 30;26(8):258-261. doi: 10.52586/4941. PMID: 34455757.
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