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3D Printing Custom Bioactive and Absorbable Surgical Screws, Pins, and Bone Plates for Localized Drug Delivery.

Journal of functional biomaterials

Tappa K, Jammalamadaka U, Weisman JA, Ballard DH, Wolford DD, Pascual-Garrido C, Wolford LM, Woodard PK, Mills DK.
PMID: 30939719
J Funct Biomater. 2019 Apr 01;10(2). doi: 10.3390/jfb10020017.

Additive manufacturing has great potential for personalized medicine in osseous fixation surgery, including maxillofacial and orthopedic applications. The purpose of this study was to demonstrate 3D printing methods for the fabrication of patient-specific fixation implants that allow for localized...

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Tappa K, Jammalamadaka U, Weisman JA, et al. 3D Printing Custom Bioactive and Absorbable Surgical Screws, Pins, and Bone Plates for Localized Drug Delivery. J Funct Biomater. 2019;10(2)doi: 10.3390/jfb10020017.
Tappa, K., Jammalamadaka, U., Weisman, J. A., Ballard, D. H., Wolford, D. D., Pascual-Garrido, C., Wolford, L. M., Woodard, P. K., & Mills, D. K. (2019). 3D Printing Custom Bioactive and Absorbable Surgical Screws, Pins, and Bone Plates for Localized Drug Delivery. Journal of functional biomaterials, 10(2), . https://doi.org/10.3390/jfb10020017
Tappa, Karthik, et al. "3D Printing Custom Bioactive and Absorbable Surgical Screws, Pins, and Bone Plates for Localized Drug Delivery." Journal of functional biomaterials vol. 10,2 (2019). doi: https://doi.org/10.3390/jfb10020017
Tappa K, Jammalamadaka U, Weisman JA, Ballard DH, Wolford DD, Pascual-Garrido C, Wolford LM, Woodard PK, Mills DK. 3D Printing Custom Bioactive and Absorbable Surgical Screws, Pins, and Bone Plates for Localized Drug Delivery. J Funct Biomater. 2019 Apr 01;10(2). doi: 10.3390/jfb10020017. PMID: 30939719; PMCID: PMC6616894.
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Corrigendum to "Klinefelter's syndrome (47,XXY) is in excess among men with Sjögren's syndrome" [Clin. Immunol. 168 (2016) 25-29].

Clinical immunology (Orlando, Fla.)

Harris VM, Sharma R, Cavett J, Kurien BT, Liu K, Koelsch KA, Rasmussen A, Radfar L, Lewis D, Stone DU, Kaufman CE, Li S, Segal B, Wallace DJ, Weisman MH, Venuturupalli S, Kelly JA, Alarcon-Riquelme ME, Pons-Estel B, Jonsson R, Lu X, Gottenberg JE, Anaya JM, Cunninghame-Graham DS, Huang AJW, Brennan MT, Hughes P, Alevizos I, Miceli-Richard C, Keystone EC, Bykerk VP, Hirschfield G, Xie G, Siminovitch KA, Ng WF, Nordmark G, Bucher SM, Eriksson P, Omdal R, Rhodus NL, Rischmueller M, Rohrer M, Wahren-Herlenius M, Witte T, Mariette X, Lessard CJ, Harley JB, Sivils KL, Scofield RH.
PMID: 29195081
Clin Immunol. 2018 Feb;187:137-138. doi: 10.1016/j.clim.2017.11.012. Epub 2017 Dec 07.

No abstract available.

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Harris VM, Sharma R, Cavett J, et al. Corrigendum to "Klinefelter's syndrome (47,XXY) is in excess among men with Sjögren's syndrome" [Clin. Immunol. 168 (2016) 25-29]. Clin Immunol. 2017;187:137-138doi: 10.1016/j.clim.2017.11.012.
Harris, V. M., Sharma, R., Cavett, J., Kurien, B. T., Liu, K., Koelsch, K. A., Rasmussen, A., Radfar, L., Lewis, D., Stone, D. U., Kaufman, C. E., Li, S., Segal, B., Wallace, D. J., Weisman, M. H., Venuturupalli, S., Kelly, J. A., Alarcon-Riquelme, M. E., Pons-Estel, B., Jonsson, R., Lu, X., Gottenberg, J. E., Anaya, J. M., Cunninghame-Graham, D. S., Huang, A. J. W., Brennan, M. T., Hughes, P., Alevizos, I., Miceli-Richard, C., Keystone, E. C., Bykerk, V. P., Hirschfield, G., Xie, G., Siminovitch, K. A., Ng, W. F., Nordmark, G., Bucher, S. M., Eriksson, P., Omdal, R., Rhodus, N. L., Rischmueller, M., Rohrer, M., Wahren-Herlenius, M., Witte, T., Mariette, X., Lessard, C. J., Harley, J. B., Sivils, K. L., & Scofield, R. H. (2018). Corrigendum to "Klinefelter's syndrome (47,XXY) is in excess among men with Sjögren's syndrome" [Clin. Immunol. 168 (2016) 25-29]. Clinical immunology (Orlando, Fla.), 187137-138. https://doi.org/10.1016/j.clim.2017.11.012
Harris, Valerie M, et al. "Corrigendum to "Klinefelter's syndrome (47,XXY) is in excess among men with Sjögren's syndrome" [Clin. Immunol. 168 (2016) 25-29]." Clinical immunology (Orlando, Fla.) vol. 187 (2018): 137-138. doi: https://doi.org/10.1016/j.clim.2017.11.012
Harris VM, Sharma R, Cavett J, Kurien BT, Liu K, Koelsch KA, Rasmussen A, Radfar L, Lewis D, Stone DU, Kaufman CE, Li S, Segal B, Wallace DJ, Weisman MH, Venuturupalli S, Kelly JA, Alarcon-Riquelme ME, Pons-Estel B, Jonsson R, Lu X, Gottenberg JE, Anaya JM, Cunninghame-Graham DS, Huang AJW, Brennan MT, Hughes P, Alevizos I, Miceli-Richard C, Keystone EC, Bykerk VP, Hirschfield G, Xie G, Siminovitch KA, Ng WF, Nordmark G, Bucher SM, Eriksson P, Omdal R, Rhodus NL, Rischmueller M, Rohrer M, Wahren-Herlenius M, Witte T, Mariette X, Lessard CJ, Harley JB, Sivils KL, Scofield RH. Corrigendum to "Klinefelter's syndrome (47,XXY) is in excess among men with Sjögren's syndrome" [Clin. Immunol. 168 (2016) 25-29]. Clin Immunol. 2018 Feb;187:137-138. doi: 10.1016/j.clim.2017.11.012. Epub 2017 Dec 07. PMID: 29195081.
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Gene Evolution: Getting Something from Nothing.

Current biology : CB

Weisman CM, Eddy SR.
PMID: 28697368
Curr Biol. 2017 Jul 10;27(13):R661-R663. doi: 10.1016/j.cub.2017.05.056.

New genes arise from pre-existing genes, but some de novo origin from non-genic sequence also seems plausible. A new study has surprisingly concluded that 25% of random DNA sequences yield beneficial products when expressed in bacteria.

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Weisman CM, Eddy SR. Gene Evolution: Getting Something from Nothing. Curr Biol. 2017;27(13):R661-R663doi: 10.1016/j.cub.2017.05.056.
Weisman, C. M., & Eddy, S. R. (2017). Gene Evolution: Getting Something from Nothing. Current biology : CB, 27(13), R661-R663. https://doi.org/10.1016/j.cub.2017.05.056
Weisman, Caroline M, and Eddy, Sean R. "Gene Evolution: Getting Something from Nothing." Current biology : CB vol. 27,13 (2017): R661-R663. doi: https://doi.org/10.1016/j.cub.2017.05.056
Weisman CM, Eddy SR. Gene Evolution: Getting Something from Nothing. Curr Biol. 2017 Jul 10;27(13):R661-R663. doi: 10.1016/j.cub.2017.05.056. PMID: 28697368.
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P2 Receptors as Therapeutic Targets in the Salivary Gland: From Physiology to Dysfunction.

Frontiers in pharmacology

Khalafalla MG, Woods LT, Jasmer KJ, Forti KM, Camden JM, Jensen JL, Limesand KH, Galtung HK, Weisman GA.
PMID: 32231563
Front Pharmacol. 2020 Mar 13;11:222. doi: 10.3389/fphar.2020.00222. eCollection 2020.

Although often overlooked in our daily lives, saliva performs a host of necessary physiological functions, including lubricating and protecting the oral cavity, facilitating taste sensation and digestion and maintaining tooth enamel. Therefore, salivary gland dysfunction and hyposalivation, often resulting...

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Khalafalla MG, Woods LT, Jasmer KJ, et al. P2 Receptors as Therapeutic Targets in the Salivary Gland: From Physiology to Dysfunction. Front Pharmacol. 2020;11:222doi: 10.3389/fphar.2020.00222.
Khalafalla, M. G., Woods, L. T., Jasmer, K. J., Forti, K. M., Camden, J. M., Jensen, J. L., Limesand, K. H., Galtung, H. K., & Weisman, G. A. (2020). P2 Receptors as Therapeutic Targets in the Salivary Gland: From Physiology to Dysfunction. Frontiers in pharmacology, 11222. https://doi.org/10.3389/fphar.2020.00222
Khalafalla, Mahmoud G, et al. "P2 Receptors as Therapeutic Targets in the Salivary Gland: From Physiology to Dysfunction." Frontiers in pharmacology vol. 11 (2020): 222. doi: https://doi.org/10.3389/fphar.2020.00222
Khalafalla MG, Woods LT, Jasmer KJ, Forti KM, Camden JM, Jensen JL, Limesand KH, Galtung HK, Weisman GA. P2 Receptors as Therapeutic Targets in the Salivary Gland: From Physiology to Dysfunction. Front Pharmacol. 2020 Mar 13;11:222. doi: 10.3389/fphar.2020.00222. eCollection 2020. PMID: 32231563; PMCID: PMC7082426.
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P2Y.

Oral oncology

Woods LT, Jasmer KJ, Muñoz Forti K, Shanbhag VC, Camden JM, Erb L, Petris MJ, Weisman GA.
PMID: 32540611
Oral Oncol. 2020 Jun 12;109:104808. doi: 10.1016/j.oraloncology.2020.104808. Epub 2020 Jun 12.

OBJECTIVES: To assess functional expression of the P2YMATERIALS AND METHODS: In human HNSCC cell lines CAL27 and FaDu and the mouse oral cancer cell line MOC2, P2YRESULTS: P2YCONCLUSION: P2Y

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Woods LT, Jasmer KJ, Muñoz Forti K, et al. P2Y. Oral Oncol. 2020;109:104808doi: 10.1016/j.oraloncology.2020.104808.
Woods, L. T., Jasmer, K. J., Muñoz Forti, K., Shanbhag, V. C., Camden, J. M., Erb, L., Petris, M. J., & Weisman, G. A. (2020). P2Y. Oral oncology, 109104808. https://doi.org/10.1016/j.oraloncology.2020.104808
Woods, Lucas T, et al. "P2Y." Oral oncology vol. 109 (2020): 104808. doi: https://doi.org/10.1016/j.oraloncology.2020.104808
Woods LT, Jasmer KJ, Muñoz Forti K, Shanbhag VC, Camden JM, Erb L, Petris MJ, Weisman GA. P2Y. Oral Oncol. 2020 Jun 12;109:104808. doi: 10.1016/j.oraloncology.2020.104808. Epub 2020 Jun 12. PMID: 32540611; PMCID: PMC7736485.
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Foreword.

Rheumatic diseases clinics of North America

Weisman MH.
PMID: 34042063
Rheum Dis Clin North Am. 2021 Feb;47(1):xi-xii. doi: 10.1016/j.rdc.2020.10.002. Epub 2020 Oct 27.

No abstract available.

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Weisman MH. Foreword. Rheum Dis Clin North Am. 2020;47(1):xi-xiidoi: 10.1016/j.rdc.2020.10.002.
Weisman, M. H. (2021). Foreword. Rheumatic diseases clinics of North America, 47(1), xi-xii. https://doi.org/10.1016/j.rdc.2020.10.002
Weisman, Michael H. "Foreword." Rheumatic diseases clinics of North America vol. 47,1 (2021): xi-xii. doi: https://doi.org/10.1016/j.rdc.2020.10.002
Weisman MH. Foreword. Rheum Dis Clin North Am. 2021 Feb;47(1):xi-xii. doi: 10.1016/j.rdc.2020.10.002. Epub 2020 Oct 27. PMID: 34042063.
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The Interplay Between COVID-19 and Spondyloarthritis or Its Treatment.

The Journal of rheumatology

Rosenbaum JT, Weisman MH, Hamilton H, Shafer C, Aslanyan E, Howard RA, Ogle K, Reveille JD, Winthrop KL, Choi D.
PMID: 34599048
J Rheumatol. 2021 Oct 01; doi: 10.3899/jrheum.210742. Epub 2021 Oct 01.

OBJECTIVE: The coronavirus disease 2019 (COVID-19) pandemic has created multiple uncertainties regarding rheumatic diseases or their treatment, with regard to the susceptibility to or severity of the viral disease. We aimed to address these questions as they relate to...

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Rosenbaum JT, Weisman MH, Hamilton H, et al. The Interplay Between COVID-19 and Spondyloarthritis or Its Treatment. J Rheumatol. 2021;doi: 10.3899/jrheum.210742.
Rosenbaum, J. T., Weisman, M. H., Hamilton, H., Shafer, C., Aslanyan, E., Howard, R. A., Ogle, K., Reveille, J. D., Winthrop, K. L., & Choi, D. (2021). The Interplay Between COVID-19 and Spondyloarthritis or Its Treatment. The Journal of rheumatology, . https://doi.org/10.3899/jrheum.210742
Rosenbaum, James T, et al. "The Interplay Between COVID-19 and Spondyloarthritis or Its Treatment." The Journal of rheumatology vol. (2021). doi: https://doi.org/10.3899/jrheum.210742
Rosenbaum JT, Weisman MH, Hamilton H, Shafer C, Aslanyan E, Howard RA, Ogle K, Reveille JD, Winthrop KL, Choi D. The Interplay Between COVID-19 and Spondyloarthritis or Its Treatment. J Rheumatol. 2021 Oct 01; doi: 10.3899/jrheum.210742. Epub 2021 Oct 01. PMID: 34599048.
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Signaling through FcγRIIA and the C5a-C5aR pathway mediates platelet hyperactivation in COVID-19.

bioRxiv : the preprint server for biology

Apostolidis SA, Sarkar A, Giannini HM, Goel RR, Mathew D, Suzuki A, Baxter AE, Greenplate AR, Alanio C, Abdel-Hakeem M, Oldridge DA, Giles J, Wu JE, Chen Z, Huang YJ, Pattekar A, Manne S, Kuthuru O, Dougherty J, Weiderhold B, Weisman AR, Ittner CAG, Gouma S, Dunbar D, Frank I, Huang AC, Vella LA, Reilly JP, Hensley SE, Rauova L, Zhao L, Meyer NJ, Poncz M, Abrams CS, Wherry EJ.
PMID: 33972943
bioRxiv. 2021 May 03; doi: 10.1101/2021.05.01.442279.

ONE-SENTENCE SUMMARY: The FcγRIIA and C5a-C5aR pathways mediate platelet hyperactivation in COVID-19.

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Apostolidis SA, Sarkar A, Giannini HM, et al. Signaling through FcγRIIA and the C5a-C5aR pathway mediates platelet hyperactivation in COVID-19. bioRxiv. 2021;doi: 10.1101/2021.05.01.442279.
Apostolidis, S. A., Sarkar, A., Giannini, H. M., Goel, R. R., Mathew, D., Suzuki, A., Baxter, A. E., Greenplate, A. R., Alanio, C., Abdel-Hakeem, M., Oldridge, D. A., Giles, J., Wu, J. E., Chen, Z., Huang, Y. J., Pattekar, A., Manne, S., Kuthuru, O., Dougherty, J., Weiderhold, B., Weisman, A. R., Ittner, C. A. G., Gouma, S., Dunbar, D., Frank, I., Huang, A. C., Vella, L. A., Reilly, J. P., Hensley, S. E., Rauova, L., Zhao, L., Meyer, N. J., Poncz, M., Abrams, C. S., & Wherry, E. J. (2021). Signaling through FcγRIIA and the C5a-C5aR pathway mediates platelet hyperactivation in COVID-19. bioRxiv : the preprint server for biology, . https://doi.org/10.1101/2021.05.01.442279
Apostolidis, Sokratis A, et al. "Signaling through FcγRIIA and the C5a-C5aR pathway mediates platelet hyperactivation in COVID-19." bioRxiv : the preprint server for biology vol. (2021). doi: https://doi.org/10.1101/2021.05.01.442279
Apostolidis SA, Sarkar A, Giannini HM, Goel RR, Mathew D, Suzuki A, Baxter AE, Greenplate AR, Alanio C, Abdel-Hakeem M, Oldridge DA, Giles J, Wu JE, Chen Z, Huang YJ, Pattekar A, Manne S, Kuthuru O, Dougherty J, Weiderhold B, Weisman AR, Ittner CAG, Gouma S, Dunbar D, Frank I, Huang AC, Vella LA, Reilly JP, Hensley SE, Rauova L, Zhao L, Meyer NJ, Poncz M, Abrams CS, Wherry EJ. Signaling through FcγRIIA and the C5a-C5aR pathway mediates platelet hyperactivation in COVID-19. bioRxiv. 2021 May 03; doi: 10.1101/2021.05.01.442279. PMID: 33972943; PMCID: PMC8109205.
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Automated quantification of baseline imaging PET metrics on FDG PET/CT images of pediatric Hodgkin lymphoma patients.

EJNMMI physics

Weisman AJ, Kim J, Lee I, McCarten KM, Kessel S, Schwartz CL, Kelly KM, Jeraj R, Cho SY, Bradshaw TJ.
PMID: 33315178
EJNMMI Phys. 2020 Dec 14;7(1):76. doi: 10.1186/s40658-020-00346-3.

RESULTS: Median Dice similarity coefficient of patient contours between automated and physician contours was 0.86 (IQR 0.78-0.91). Automated SUVCONCLUSIONS: An automated method using an ensemble of multi-resolution pathway 3D CNNs was able to quantify PET imaging features of lymphoma...

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Weisman AJ, Kim J, Lee I, et al. Automated quantification of baseline imaging PET metrics on FDG PET/CT images of pediatric Hodgkin lymphoma patients. EJNMMI Phys. 2020;7(1):76doi: 10.1186/s40658-020-00346-3.
Weisman, A. J., Kim, J., Lee, I., McCarten, K. M., Kessel, S., Schwartz, C. L., Kelly, K. M., Jeraj, R., Cho, S. Y., & Bradshaw, T. J. (2020). Automated quantification of baseline imaging PET metrics on FDG PET/CT images of pediatric Hodgkin lymphoma patients. EJNMMI physics, 7(1), 76. https://doi.org/10.1186/s40658-020-00346-3
Weisman, Amy J, et al. "Automated quantification of baseline imaging PET metrics on FDG PET/CT images of pediatric Hodgkin lymphoma patients." EJNMMI physics vol. 7,1 (2020): 76. doi: https://doi.org/10.1186/s40658-020-00346-3
Weisman AJ, Kim J, Lee I, McCarten KM, Kessel S, Schwartz CL, Kelly KM, Jeraj R, Cho SY, Bradshaw TJ. Automated quantification of baseline imaging PET metrics on FDG PET/CT images of pediatric Hodgkin lymphoma patients. EJNMMI Phys. 2020 Dec 14;7(1):76. doi: 10.1186/s40658-020-00346-3. PMID: 33315178; PMCID: PMC7736382.
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Coupling of P2Y receptors to G proteins and other signaling pathways.

Wiley interdisciplinary reviews. Membrane transport and signaling

Erb L, Weisman GA.
PMID: 25774333
Wiley Interdiscip Rev Membr Transp Signal. 2012 Nov-Dec;1(6):789-803. doi: 10.1002/wmts.62.

P2Y receptors are G protein-coupled receptors (GPCRs) that are activated by adenine and uridine nucleotides and nucleotide sugars. There are eight subtypes of P2Y receptors (P2Y

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Erb L, Weisman GA. Coupling of P2Y receptors to G proteins and other signaling pathways. Wiley Interdiscip Rev Membr Transp Signal. 2012;1(6):789-803doi: 10.1002/wmts.62.
Erb, L., & Weisman, G. A. (2012). Coupling of P2Y receptors to G proteins and other signaling pathways. Wiley interdisciplinary reviews. Membrane transport and signaling, 1(6), 789-803. https://doi.org/10.1002/wmts.62
Erb, Laurie, and Weisman, Gary A. "Coupling of P2Y receptors to G proteins and other signaling pathways." Wiley interdisciplinary reviews. Membrane transport and signaling vol. 1,6 (2012): 789-803. doi: https://doi.org/10.1002/wmts.62
Erb L, Weisman GA. Coupling of P2Y receptors to G proteins and other signaling pathways. Wiley Interdiscip Rev Membr Transp Signal. 2012 Nov-Dec;1(6):789-803. doi: 10.1002/wmts.62. PMID: 25774333; PMCID: PMC4358762.
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Risk Factors for Spinal Structural Damage in a Chinese Cohort With Ankylosing Spondylitis.

Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases

Kong W, Jefferies C, Learch TJ, Gan X, Zhu F, Zhang N, Jin D, Zhang Y, Tao Q, Yan X, Ishimori ML, Weisman MH.
PMID: 33394829
J Clin Rheumatol. 2022 Jan 01;28(1):e118-e124. doi: 10.1097/RHU.0000000000001658.

OBJECTIVES: A cross-sectional study was conducted in 270 Chinese patients with ankylosing spondylitis (AS) in order to identify potential risk factors for severity of spinal structural damage.METHODS: Two hundred seventy AS patients fulfilled the Modified New York Criteria. Computed...

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Kong W, Jefferies C, Learch TJ, et al. Risk Factors for Spinal Structural Damage in a Chinese Cohort With Ankylosing Spondylitis. J Clin Rheumatol. 2022;28(1):e118-e124doi: 10.1097/RHU.0000000000001658.
Kong, W., Jefferies, C., Learch, T. J., Gan, X., Zhu, F., Zhang, N., Jin, D., Zhang, Y., Tao, Q., Yan, X., Ishimori, M. L., & Weisman, M. H. (2022). Risk Factors for Spinal Structural Damage in a Chinese Cohort With Ankylosing Spondylitis. Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases, 28(1), e118-e124. https://doi.org/10.1097/RHU.0000000000001658
Kong, Weiping, et al. "Risk Factors for Spinal Structural Damage in a Chinese Cohort With Ankylosing Spondylitis." Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases vol. 28,1 (2022): e118-e124. doi: https://doi.org/10.1097/RHU.0000000000001658
Kong W, Jefferies C, Learch TJ, Gan X, Zhu F, Zhang N, Jin D, Zhang Y, Tao Q, Yan X, Ishimori ML, Weisman MH. Risk Factors for Spinal Structural Damage in a Chinese Cohort With Ankylosing Spondylitis. J Clin Rheumatol. 2022 Jan 01;28(1):e118-e124. doi: 10.1097/RHU.0000000000001658. PMID: 33394829.
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Strong viral associations with SLE among Filipinos.

Lupus science & medicine

Vista ES, Weisman MH, Ishimori ML, Chen H, Bourn RL, Bruner BF, Hamijoyo L, Tanangunan RD, Gal NJ, Robertson JM, Harley JB, Guthridge JM, Navarra SV, James JA.
PMID: 29214036
Lupus Sci Med. 2017 Jul 28;4(1):e000214. doi: 10.1136/lupus-2017-000214. eCollection 2017.

OBJECTIVES: Epstein-Barr virus (EBV) is considered an important environmental factor in SLE aetiology, but the relationship between SLE and EBV in the Filipino population is unknown. We tested associations between SLE, lupus-associated autoantibodies and seropositivity for EBV and other...

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Vista ES, Weisman MH, Ishimori ML, et al. Strong viral associations with SLE among Filipinos. Lupus Sci Med. 2017;4(1):e000214doi: 10.1136/lupus-2017-000214.
Vista, E. S., Weisman, M. H., Ishimori, M. L., Chen, H., Bourn, R. L., Bruner, B. F., Hamijoyo, L., Tanangunan, R. D., Gal, N. J., Robertson, J. M., Harley, J. B., Guthridge, J. M., Navarra, S. V., & James, J. A. (2017). Strong viral associations with SLE among Filipinos. Lupus science & medicine, 4(1), e000214. https://doi.org/10.1136/lupus-2017-000214
Vista, Evan S, et al. "Strong viral associations with SLE among Filipinos." Lupus science & medicine vol. 4,1 (2017): e000214. doi: https://doi.org/10.1136/lupus-2017-000214
Vista ES, Weisman MH, Ishimori ML, Chen H, Bourn RL, Bruner BF, Hamijoyo L, Tanangunan RD, Gal NJ, Robertson JM, Harley JB, Guthridge JM, Navarra SV, James JA. Strong viral associations with SLE among Filipinos. Lupus Sci Med. 2017 Jul 28;4(1):e000214. doi: 10.1136/lupus-2017-000214. eCollection 2017. PMID: 29214036; PMCID: PMC5704743.
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