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Evaluating the Functional Outcomes and the Quality of Life for Pediatric Patients with Osteogenesis Imperfecta after Fracture Treatment with Intramedullary Rodding.

Children (Basel, Switzerland)

Herdea A, Ulici A, Qirjako D, Toma A, Derihaci RP, Lungu CN, Charkaoui A.
PMID: 34828779
Children (Basel). 2021 Nov 19;8(11). doi: 10.3390/children8111066.

BACKGROUND AND OBJECTIVES: Osteogenesis imperfecta is a rare pathology involving the bones and the connective tissues, generating alterations that lead to frequent fractures during childhood. When fractures occur at birth, they are associated with an impairment of walking and...

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Herdea A, Ulici A, Qirjako D, et al. Evaluating the Functional Outcomes and the Quality of Life for Pediatric Patients with Osteogenesis Imperfecta after Fracture Treatment with Intramedullary Rodding. Children (Basel). 2021;8(11)doi: 10.3390/children8111066.
Herdea, A., Ulici, A., Qirjako, D., Toma, A., Derihaci, R. P., Lungu, C. N., & Charkaoui, A. (2021). Evaluating the Functional Outcomes and the Quality of Life for Pediatric Patients with Osteogenesis Imperfecta after Fracture Treatment with Intramedullary Rodding. Children (Basel, Switzerland), 8(11), . https://doi.org/10.3390/children8111066
Herdea, Alexandru, et al. "Evaluating the Functional Outcomes and the Quality of Life for Pediatric Patients with Osteogenesis Imperfecta after Fracture Treatment with Intramedullary Rodding." Children (Basel, Switzerland) vol. 8,11 (2021). doi: https://doi.org/10.3390/children8111066
Herdea A, Ulici A, Qirjako D, Toma A, Derihaci RP, Lungu CN, Charkaoui A. Evaluating the Functional Outcomes and the Quality of Life for Pediatric Patients with Osteogenesis Imperfecta after Fracture Treatment with Intramedullary Rodding. Children (Basel). 2021 Nov 19;8(11). doi: 10.3390/children8111066. PMID: 34828779; PMCID: PMC8618628.
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Resveratrol suppresses cholestasis-induced liver injury and fibrosis in rats associated with the inhibition of TGFβ1-Smad3-miR21 axis and profibrogenic and hepatic injury biomarkers.

Clinical and experimental pharmacology & physiology

ShamsEldeen AM, Al-Ani B, Ebrahim HA, Rashed L, Badr AM, Attia A, Farag AM, Kamar SS, Haidara MA, Al Humayed S, Ali Eshra M.
PMID: 34157155
Clin Exp Pharmacol Physiol. 2021 Oct;48(10):1402-1411. doi: 10.1111/1440-1681.13546. Epub 2021 Jul 13.

Cholestasis caused by slowing or blockage of bile flow is a serious liver disease that can lead to liver fibrosis and cirrhosis. The link between transforming growth factor beta 1 (TGFβ1), Smad family member 3 (Smad3), and microRNA 21...

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ShamsEldeen AM, Al-Ani B, Ebrahim HA, et al. Resveratrol suppresses cholestasis-induced liver injury and fibrosis in rats associated with the inhibition of TGFβ1-Smad3-miR21 axis and profibrogenic and hepatic injury biomarkers. Clin Exp Pharmacol Physiol. 2021;48(10):1402-1411doi: 10.1111/1440-1681.13546.
ShamsEldeen, A. M., Al-Ani, B., Ebrahim, H. A., Rashed, L., Badr, A. M., Attia, A., Farag, A. M., Kamar, S. S., Haidara, M. A., Al Humayed, S., & Ali Eshra, M. (2021). Resveratrol suppresses cholestasis-induced liver injury and fibrosis in rats associated with the inhibition of TGFβ1-Smad3-miR21 axis and profibrogenic and hepatic injury biomarkers. Clinical and experimental pharmacology & physiology, 48(10), 1402-1411. https://doi.org/10.1111/1440-1681.13546
ShamsEldeen, Asmaa M, et al. "Resveratrol suppresses cholestasis-induced liver injury and fibrosis in rats associated with the inhibition of TGFβ1-Smad3-miR21 axis and profibrogenic and hepatic injury biomarkers." Clinical and experimental pharmacology & physiology vol. 48,10 (2021): 1402-1411. doi: https://doi.org/10.1111/1440-1681.13546
ShamsEldeen AM, Al-Ani B, Ebrahim HA, Rashed L, Badr AM, Attia A, Farag AM, Kamar SS, Haidara MA, Al Humayed S, Ali Eshra M. Resveratrol suppresses cholestasis-induced liver injury and fibrosis in rats associated with the inhibition of TGFβ1-Smad3-miR21 axis and profibrogenic and hepatic injury biomarkers. Clin Exp Pharmacol Physiol. 2021 Oct;48(10):1402-1411. doi: 10.1111/1440-1681.13546. Epub 2021 Jul 13. PMID: 34157155.
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Atypical preeclampsia before 20 weeks of gestation with multicystic placenta, hyperreactio luteinalis, and elevated sFlt-1/PlGF ratio as manifestations of fetal triploidy: A case report.

Case reports in women's health

Hayashida H, Nakamura K, Ukon K, Sato K, Mimura K, Kakuda M, Toda A, Miyake T, Hiramatsu K, Kimura T, Endo M, Kimura T.
PMID: 35024347
Case Rep Womens Health. 2021 Dec 27;33:e00379. doi: 10.1016/j.crwh.2021.e00379. eCollection 2022 Jan.

Preeclampsia is one of the most common as well as most severe complications of pregnancy, characterized by new-onset hypertension and proteinuria or other organ dysfunction. It predominantly occurs after 20 weeks of gestation. Very rarely, it can be triggered...

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Hayashida H, Nakamura K, Ukon K, et al. Atypical preeclampsia before 20 weeks of gestation with multicystic placenta, hyperreactio luteinalis, and elevated sFlt-1/PlGF ratio as manifestations of fetal triploidy: A case report. Case Rep Womens Health. 2021;33:e00379doi: 10.1016/j.crwh.2021.e00379.
Hayashida, H., Nakamura, K., Ukon, K., Sato, K., Mimura, K., Kakuda, M., Toda, A., Miyake, T., Hiramatsu, K., Kimura, T., Endo, M., & Kimura, T. (2022). Atypical preeclampsia before 20 weeks of gestation with multicystic placenta, hyperreactio luteinalis, and elevated sFlt-1/PlGF ratio as manifestations of fetal triploidy: A case report. Case reports in women's health, 33e00379. https://doi.org/10.1016/j.crwh.2021.e00379
Hayashida, Harue, et al. "Atypical preeclampsia before 20 weeks of gestation with multicystic placenta, hyperreactio luteinalis, and elevated sFlt-1/PlGF ratio as manifestations of fetal triploidy: A case report." Case reports in women's health vol. 33 (2022): e00379. doi: https://doi.org/10.1016/j.crwh.2021.e00379
Hayashida H, Nakamura K, Ukon K, Sato K, Mimura K, Kakuda M, Toda A, Miyake T, Hiramatsu K, Kimura T, Endo M, Kimura T. Atypical preeclampsia before 20 weeks of gestation with multicystic placenta, hyperreactio luteinalis, and elevated sFlt-1/PlGF ratio as manifestations of fetal triploidy: A case report. Case Rep Womens Health. 2021 Dec 27;33:e00379. doi: 10.1016/j.crwh.2021.e00379. eCollection 2022 Jan. PMID: 35024347; PMCID: PMC8728315.
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Phylogeny and ultrastructure of .

Journal of parasitic diseases : official organ of the Indian Society for Parasitology

Abou Zaid AA, Younes AM, Desouky AY, El-Seify MA.
PMID: 34475666
J Parasit Dis. 2021 Sep;45(3):831-837. doi: 10.1007/s12639-021-01364-3. Epub 2021 Mar 09.

The genus

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Abou Zaid AA, Younes AM, Desouky AY, et al. Phylogeny and ultrastructure of . J Parasit Dis. 2021;45(3):831-837doi: 10.1007/s12639-021-01364-3.
Abou Zaid, A. A., Younes, A. M., Desouky, A. Y., & El-Seify, M. A. (2021). Phylogeny and ultrastructure of . Journal of parasitic diseases : official organ of the Indian Society for Parasitology, 45(3), 831-837. https://doi.org/10.1007/s12639-021-01364-3
Abou Zaid, Attia A, et al. "Phylogeny and ultrastructure of ." Journal of parasitic diseases : official organ of the Indian Society for Parasitology vol. 45,3 (2021): 831-837. doi: https://doi.org/10.1007/s12639-021-01364-3
Abou Zaid AA, Younes AM, Desouky AY, El-Seify MA. Phylogeny and ultrastructure of . J Parasit Dis. 2021 Sep;45(3):831-837. doi: 10.1007/s12639-021-01364-3. Epub 2021 Mar 09. PMID: 34475666; PMCID: PMC8368968.
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The Longitudinal Early-onset Alzheimer's Disease Study (LEADS): Framework and methodology.

Alzheimer's & dementia : the journal of the Alzheimer's Association

Apostolova LG, Aisen P, Eloyan A, Fagan A, Fargo KN, Foroud T, Gatsonis C, Grinberg LT, Jack CR, Kramer J, Koeppe R, Kukull WA, Murray ME, Nudelman K, Rumbaugh M, Toga A, Vemuri P, Trullinger A, Iaccarino L, Day GS, Graff-Radford NR, Honig LS, Jones DT, Masdeu J, Mendez M, Musiek E, Onyike CU, Rogalski E, Salloway S, Wolk DA, Wingo TS, Carrillo MC, Dickerson BC, Rabinovici GD.
PMID: 34018654
Alzheimers Dement. 2021 Dec;17(12):2043-2055. doi: 10.1002/alz.12350. Epub 2021 May 21.

Patients with early-onset Alzheimer's disease (EOAD) are commonly excluded from large-scale observational and therapeutic studies due to their young age, atypical presentation, or absence of pathogenic mutations. The goals of the Longitudinal EOAD Study (LEADS) are to (1) define...

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Apostolova LG, Aisen P, Eloyan A, et al. The Longitudinal Early-onset Alzheimer's Disease Study (LEADS): Framework and methodology. Alzheimers Dement. 2021;17(12):2043-2055doi: 10.1002/alz.12350.
Apostolova, L. G., Aisen, P., Eloyan, A., Fagan, A., Fargo, K. N., Foroud, T., Gatsonis, C., Grinberg, L. T., Jack, C. R., Kramer, J., Koeppe, R., Kukull, W. A., Murray, M. E., Nudelman, K., Rumbaugh, M., Toga, A., Vemuri, P., Trullinger, A., Iaccarino, L., Day, G. S., Graff-Radford, N. R., Honig, L. S., Jones, D. T., Masdeu, J., Mendez, M., Musiek, E., Onyike, C. U., Rogalski, E., Salloway, S., Wolk, D. A., Wingo, T. S., Carrillo, M. C., Dickerson, B. C., Rabinovici, G. D. (2021). The Longitudinal Early-onset Alzheimer's Disease Study (LEADS): Framework and methodology. Alzheimer's & dementia : the journal of the Alzheimer's Association, 17(12), 2043-2055. https://doi.org/10.1002/alz.12350
Apostolova, Liana G, et al. "The Longitudinal Early-onset Alzheimer's Disease Study (LEADS): Framework and methodology." Alzheimer's & dementia : the journal of the Alzheimer's Association vol. 17,12 (2021): 2043-2055. doi: https://doi.org/10.1002/alz.12350
Apostolova LG, Aisen P, Eloyan A, Fagan A, Fargo KN, Foroud T, Gatsonis C, Grinberg LT, Jack CR, Kramer J, Koeppe R, Kukull WA, Murray ME, Nudelman K, Rumbaugh M, Toga A, Vemuri P, Trullinger A, Iaccarino L, Day GS, Graff-Radford NR, Honig LS, Jones DT, Masdeu J, Mendez M, Musiek E, Onyike CU, Rogalski E, Salloway S, Wolk DA, Wingo TS, Carrillo MC, Dickerson BC, Rabinovici GD. The Longitudinal Early-onset Alzheimer's Disease Study (LEADS): Framework and methodology. Alzheimers Dement. 2021 Dec;17(12):2043-2055. doi: 10.1002/alz.12350. Epub 2021 May 21. PMID: 34018654.
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Let's talk about race: changing the conversations around race in academia.

Communications biology

Miller-Kleinhenz JM, Kuzmishin Nagy AB, Majewska AA, Adebayo Michael AO, Najmi SM, Nguyen KH, Van Sciver RE, Fonkoue IT.
PMID: 34354238
Commun Biol. 2021 Aug 05;4(1):902. doi: 10.1038/s42003-021-02409-2.

No abstract available.

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Miller-Kleinhenz JM, Kuzmishin Nagy AB, Majewska AA, et al. Let's talk about race: changing the conversations around race in academia. Commun Biol. 2021;4(1):902doi: 10.1038/s42003-021-02409-2.
Miller-Kleinhenz, J. M., Kuzmishin Nagy, A. B., Majewska, A. A., Adebayo Michael, A. O., Najmi, S. M., Nguyen, K. H., Van Sciver, R. E., & Fonkoue, I. T. (2021). Let's talk about race: changing the conversations around race in academia. Communications biology, 4(1), 902. https://doi.org/10.1038/s42003-021-02409-2
Miller-Kleinhenz, Jasmine M, et al. "Let's talk about race: changing the conversations around race in academia." Communications biology vol. 4,1 (2021): 902. doi: https://doi.org/10.1038/s42003-021-02409-2
Miller-Kleinhenz JM, Kuzmishin Nagy AB, Majewska AA, Adebayo Michael AO, Najmi SM, Nguyen KH, Van Sciver RE, Fonkoue IT. Let's talk about race: changing the conversations around race in academia. Commun Biol. 2021 Aug 05;4(1):902. doi: 10.1038/s42003-021-02409-2. PMID: 34354238; PMCID: PMC8342502.
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Showing 469 to 474 of 474 entries