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Showing 193 to 198 of 198 entries
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Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts.

BioEssays : news and reviews in molecular, cellular and developmental biology

Kruglikov IL, Zhang Z, Scherer PE.
PMID: 34766637
Bioessays. 2022 Jan;44(1):e2100207. doi: 10.1002/bies.202100207. Epub 2021 Nov 12.

Emerging data connects the aging process in dermal fibroblasts with metabolic reprogramming, provided by enhanced fatty acid oxidation and reduced glycolysis. This switch may be caused by a significant expansion of the dermal white adipose tissue (dWAT) layer in...

Cite
Kruglikov IL, Zhang Z, Scherer PE. Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts. Bioessays. 2021;44(1):e2100207doi: 10.1002/bies.202100207.
Kruglikov, I. L., Zhang, Z., & Scherer, P. E. (2022). Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts. BioEssays : news and reviews in molecular, cellular and developmental biology, 44(1), e2100207. https://doi.org/10.1002/bies.202100207
Kruglikov, Ilja L, et al. "Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts." BioEssays : news and reviews in molecular, cellular and developmental biology vol. 44,1 (2022): e2100207. doi: https://doi.org/10.1002/bies.202100207
Kruglikov IL, Zhang Z, Scherer PE. Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts. Bioessays. 2022 Jan;44(1):e2100207. doi: 10.1002/bies.202100207. Epub 2021 Nov 12. PMID: 34766637; PMCID: PMC8688300.
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The metabolic syndrome, thiazolidinediones, and implications for intersection of chronic and inflammatory disease.

Molecular metabolism

Colca JR, Scherer PE.
PMID: 34863942
Mol Metab. 2021 Dec 01;55:101409. doi: 10.1016/j.molmet.2021.101409. Epub 2021 Dec 01.

BACKGROUND: Chronic disease appears connected to obesity. However, evidence suggests that chronic metabolic diseases are more specifically related to adipose dysfunction rather than to body weight itself.SCOPE OF REVIEW: Further study of the first generation "insulin sensitizer" pioglitazone and...

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Colca JR, Scherer PE. The metabolic syndrome, thiazolidinediones, and implications for intersection of chronic and inflammatory disease. Mol Metab. 2021;55:101409doi: 10.1016/j.molmet.2021.101409.
Colca, J. R., & Scherer, P. E. (2021). The metabolic syndrome, thiazolidinediones, and implications for intersection of chronic and inflammatory disease. Molecular metabolism, 55101409. https://doi.org/10.1016/j.molmet.2021.101409
Colca, Jerry R, and Scherer, Philipp E. "The metabolic syndrome, thiazolidinediones, and implications for intersection of chronic and inflammatory disease." Molecular metabolism vol. 55 (2021): 101409. doi: https://doi.org/10.1016/j.molmet.2021.101409
Colca JR, Scherer PE. The metabolic syndrome, thiazolidinediones, and implications for intersection of chronic and inflammatory disease. Mol Metab. 2021 Dec 01;55:101409. doi: 10.1016/j.molmet.2021.101409. Epub 2021 Dec 01. PMID: 34863942.
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Neurologic Effects of Gadolinium Retention in the Brain after Gadolinium-based Contrast Agent Administration.

Radiology

Ayers-Ringler J, McDonald JS, Connors MA, Fisher CR, Han S, Jakaitis DR, Scherer B, Tutor G, Wininger KM, Dai D, Choi DS, Salisbury JL, Jannetto PJ, Bornhorst JA, Kadirvel R, Kallmes DF, McDonald RJ.
PMID: 34931861
Radiology. 2021 Dec 21;210559. doi: 10.1148/radiol.210559. Epub 2021 Dec 21.

Background Concerns over the neurotoxic potential of retained gadolinium in brain tissues after intravenous gadolinium-based contrast agent (GBCA) administration have led to pronounced worldwide use changes, yet the clinical sequelae of gadolinium retention remain undefined. Purpose To assess clinical...

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Ayers-Ringler J, McDonald JS, Connors MA, et al. Neurologic Effects of Gadolinium Retention in the Brain after Gadolinium-based Contrast Agent Administration. Radiology. 2021;210559doi: 10.1148/radiol.210559.
Ayers-Ringler, J., McDonald, J. S., Connors, M. A., Fisher, C. R., Han, S., Jakaitis, D. R., Scherer, B., Tutor, G., Wininger, K. M., Dai, D., Choi, D. S., Salisbury, J. L., Jannetto, P. J., Bornhorst, J. A., Kadirvel, R., Kallmes, D. F., & McDonald, R. J. (2021). Neurologic Effects of Gadolinium Retention in the Brain after Gadolinium-based Contrast Agent Administration. Radiology, 210559. https://doi.org/10.1148/radiol.210559
Ayers-Ringler, Jennifer, et al. "Neurologic Effects of Gadolinium Retention in the Brain after Gadolinium-based Contrast Agent Administration." Radiology vol. (2021): 210559. doi: https://doi.org/10.1148/radiol.210559
Ayers-Ringler J, McDonald JS, Connors MA, Fisher CR, Han S, Jakaitis DR, Scherer B, Tutor G, Wininger KM, Dai D, Choi DS, Salisbury JL, Jannetto PJ, Bornhorst JA, Kadirvel R, Kallmes DF, McDonald RJ. Neurologic Effects of Gadolinium Retention in the Brain after Gadolinium-based Contrast Agent Administration. Radiology. 2021 Dec 21;210559. doi: 10.1148/radiol.210559. Epub 2021 Dec 21. PMID: 34931861.
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Age differences in social-cognitive abilities across the stages of adulthood and path model investigation of adult social cognition.

Neuropsychology, development, and cognition. Section B, Aging, neuropsychology and cognition

Gourlay C, Collin P, D'Auteuil C, Jacques M, Caron PO, Scherzer PB.
PMID: 34355998
Neuropsychol Dev Cogn B Aging Neuropsychol Cogn. 2021 Aug 06;1-35. doi: 10.1080/13825585.2021.1962789. Epub 2021 Aug 06.

Accumulating evidence points toward an association between older age and performance decrements in social cognition (SC). We explored age-related variations in four components of SC: emotion recognition, theory of mind, social judgment, and blame attributions. A total of 120...

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Gourlay C, Collin P, D'Auteuil C, et al. Age differences in social-cognitive abilities across the stages of adulthood and path model investigation of adult social cognition. Neuropsychol Dev Cogn B Aging Neuropsychol Cogn. 2021;1-35doi: 10.1080/13825585.2021.1962789.
Gourlay, C., Collin, P., D'Auteuil, C., Jacques, M., Caron, P. O., & Scherzer, P. B. (2021). Age differences in social-cognitive abilities across the stages of adulthood and path model investigation of adult social cognition. Neuropsychology, development, and cognition. Section B, Aging, neuropsychology and cognition, 1-35. https://doi.org/10.1080/13825585.2021.1962789
Gourlay, Catherine, et al. "Age differences in social-cognitive abilities across the stages of adulthood and path model investigation of adult social cognition." Neuropsychology, development, and cognition. Section B, Aging, neuropsychology and cognition vol. (2021): 1-35. doi: https://doi.org/10.1080/13825585.2021.1962789
Gourlay C, Collin P, D'Auteuil C, Jacques M, Caron PO, Scherzer PB. Age differences in social-cognitive abilities across the stages of adulthood and path model investigation of adult social cognition. Neuropsychol Dev Cogn B Aging Neuropsychol Cogn. 2021 Aug 06;1-35. doi: 10.1080/13825585.2021.1962789. Epub 2021 Aug 06. PMID: 34355998.
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Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts.

BioEssays : news and reviews in molecular, cellular and developmental biology

Kruglikov IL, Zhang Z, Scherer PE.
PMID: 34766637
Bioessays. 2022 Jan;44(1):e2100207. doi: 10.1002/bies.202100207. Epub 2021 Nov 12.

Emerging data connects the aging process in dermal fibroblasts with metabolic reprogramming, provided by enhanced fatty acid oxidation and reduced glycolysis. This switch may be caused by a significant expansion of the dermal white adipose tissue (dWAT) layer in...

Cite
Kruglikov IL, Zhang Z, Scherer PE. Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts. Bioessays. 2021;44(1):e2100207doi: 10.1002/bies.202100207.
Kruglikov, I. L., Zhang, Z., & Scherer, P. E. (2022). Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts. BioEssays : news and reviews in molecular, cellular and developmental biology, 44(1), e2100207. https://doi.org/10.1002/bies.202100207
Kruglikov, Ilja L, et al. "Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts." BioEssays : news and reviews in molecular, cellular and developmental biology vol. 44,1 (2022): e2100207. doi: https://doi.org/10.1002/bies.202100207
Kruglikov IL, Zhang Z, Scherer PE. Skin aging: Dermal adipocytes metabolically reprogram dermal fibroblasts. Bioessays. 2022 Jan;44(1):e2100207. doi: 10.1002/bies.202100207. Epub 2021 Nov 12. PMID: 34766637.
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Integrated Stress Response Couples Mitochondrial Protein Translation With Oxidative Stress Control.

Circulation

Zhang G, Wang X, Li C, Li Q, An YA, Luo X, Deng Y, Gillette TG, Scherer PE, Wang ZV.
PMID: 34583519
Circulation. 2021 Nov 02;144(18):1500-1515. doi: 10.1161/CIRCULATIONAHA.120.053125. Epub 2021 Sep 29.

BACKGROUND: The integrated stress response (ISR) is an evolutionarily conserved process to cope with intracellular and extracellular disturbances. Myocardial infarction is a leading cause of death worldwide. Coronary artery reperfusion, the most effective means to mitigate cardiac damage of...

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Zhang G, Wang X, Li C, et al. Integrated Stress Response Couples Mitochondrial Protein Translation With Oxidative Stress Control. Circulation. 2021;144(18):1500-1515doi: 10.1161/CIRCULATIONAHA.120.053125.
Zhang, G., Wang, X., Li, C., Li, Q., An, Y. A., Luo, X., Deng, Y., Gillette, T. G., Scherer, P. E., & Wang, Z. V. (2021). Integrated Stress Response Couples Mitochondrial Protein Translation With Oxidative Stress Control. Circulation, 144(18), 1500-1515. https://doi.org/10.1161/CIRCULATIONAHA.120.053125
Zhang, Guangyu, et al. "Integrated Stress Response Couples Mitochondrial Protein Translation With Oxidative Stress Control." Circulation vol. 144,18 (2021): 1500-1515. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.053125
Zhang G, Wang X, Li C, Li Q, An YA, Luo X, Deng Y, Gillette TG, Scherer PE, Wang ZV. Integrated Stress Response Couples Mitochondrial Protein Translation With Oxidative Stress Control. Circulation. 2021 Nov 02;144(18):1500-1515. doi: 10.1161/CIRCULATIONAHA.120.053125. Epub 2021 Sep 29. PMID: 34583519; PMCID: PMC8563444.
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Showing 193 to 198 of 198 entries