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Women with chronic constipation have more bothersome urogenital symptoms.

Techniques in coloproctology

Ortega MV, Kim Y, Hung K, James K, Savitt L, Von Bargen E, Bordeianou LG, Weinstein MM.
PMID: 34820751
Tech Coloproctol. 2022 Jan;26(1):29-34. doi: 10.1007/s10151-021-02518-4. Epub 2021 Nov 24.

BACKGROUND: The aim of our study was to characterize urogenital symptoms in women with and without constipation, and by severity of constipation.METHODS: This was a retrospective cohort study conducted at a pelvic floor disorder center in a tertiary healthcare...

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Ortega MV, Kim Y, Hung K, et al. Women with chronic constipation have more bothersome urogenital symptoms. Tech Coloproctol. 2021;26(1):29-34doi: 10.1007/s10151-021-02518-4.
Ortega, M. V., Kim, Y., Hung, K., James, K., Savitt, L., Von Bargen, E., Bordeianou, L. G., & Weinstein, M. M. (2022). Women with chronic constipation have more bothersome urogenital symptoms. Techniques in coloproctology, 26(1), 29-34. https://doi.org/10.1007/s10151-021-02518-4
Ortega, M V, et al. "Women with chronic constipation have more bothersome urogenital symptoms." Techniques in coloproctology vol. 26,1 (2022): 29-34. doi: https://doi.org/10.1007/s10151-021-02518-4
Ortega MV, Kim Y, Hung K, James K, Savitt L, Von Bargen E, Bordeianou LG, Weinstein MM. Women with chronic constipation have more bothersome urogenital symptoms. Tech Coloproctol. 2022 Jan;26(1):29-34. doi: 10.1007/s10151-021-02518-4. Epub 2021 Nov 24. PMID: 34820751.
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Low Glucose Mediated Fluconazole Tolerance in .

Journal of fungi (Basel, Switzerland)

Bhattacharya S, Oliveira NK, Savitt AG, Silva VKA, Krausert RB, Ghebrehiwet B, Fries BC.
PMID: 34207384
J Fungi (Basel). 2021 Jun 18;7(6). doi: 10.3390/jof7060489.

Chronic meningoencephalitis is caused by

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Bhattacharya S, Oliveira NK, Savitt AG, et al. Low Glucose Mediated Fluconazole Tolerance in . J Fungi (Basel). 2021;7(6)doi: 10.3390/jof7060489.
Bhattacharya, S., Oliveira, N. K., Savitt, A. G., Silva, V. K. A., Krausert, R. B., Ghebrehiwet, B., & Fries, B. C. (2021). Low Glucose Mediated Fluconazole Tolerance in . Journal of fungi (Basel, Switzerland), 7(6), . https://doi.org/10.3390/jof7060489
Bhattacharya, Somanon, et al. "Low Glucose Mediated Fluconazole Tolerance in ." Journal of fungi (Basel, Switzerland) vol. 7,6 (2021). doi: https://doi.org/10.3390/jof7060489
Bhattacharya S, Oliveira NK, Savitt AG, Silva VKA, Krausert RB, Ghebrehiwet B, Fries BC. Low Glucose Mediated Fluconazole Tolerance in . J Fungi (Basel). 2021 Jun 18;7(6). doi: 10.3390/jof7060489. PMID: 34207384; PMCID: PMC8233753.
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Fibers Generated by Plasma Des-AA Fibrin Monomers and Protofibril/Fibrinogen Clusters Bind Platelets: Clinical and Nonclinical Implications.

TH open : companion journal to thrombosis and haemostasis

Galanakis DK, Protopopova A, Zhang L, Li K, Marmorat C, Scheiner T, Koo J, Savitt AG, Rafailovich M, Weisel J.
PMID: 34240000
TH Open. 2021 Jul 06;5(3):e273-e285. doi: 10.1055/s-0041-1725976. eCollection 2021 Jul.

No abstract available.

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Galanakis DK, Protopopova A, Zhang L, et al. Fibers Generated by Plasma Des-AA Fibrin Monomers and Protofibril/Fibrinogen Clusters Bind Platelets: Clinical and Nonclinical Implications. TH Open. 2021;5(3):e273-e285doi: 10.1055/s-0041-1725976.
Galanakis, D. K., Protopopova, A., Zhang, L., Li, K., Marmorat, C., Scheiner, T., Koo, J., Savitt, A. G., Rafailovich, M., & Weisel, J. (2021). Fibers Generated by Plasma Des-AA Fibrin Monomers and Protofibril/Fibrinogen Clusters Bind Platelets: Clinical and Nonclinical Implications. TH open : companion journal to thrombosis and haemostasis, 5(3), e273-e285. https://doi.org/10.1055/s-0041-1725976
Galanakis, Dennis K, et al. "Fibers Generated by Plasma Des-AA Fibrin Monomers and Protofibril/Fibrinogen Clusters Bind Platelets: Clinical and Nonclinical Implications." TH open : companion journal to thrombosis and haemostasis vol. 5,3 (2021): e273-e285. doi: https://doi.org/10.1055/s-0041-1725976
Galanakis DK, Protopopova A, Zhang L, Li K, Marmorat C, Scheiner T, Koo J, Savitt AG, Rafailovich M, Weisel J. Fibers Generated by Plasma Des-AA Fibrin Monomers and Protofibril/Fibrinogen Clusters Bind Platelets: Clinical and Nonclinical Implications. TH Open. 2021 Jul 06;5(3):e273-e285. doi: 10.1055/s-0041-1725976. eCollection 2021 Jul. PMID: 34240000; PMCID: PMC8260279.
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GTP binding to translation factor eIF2B stimulates its guanine nucleotide exchange activity.

iScience

Kershaw CJ, Jennings MD, Cortopassi F, Guaita M, Al-Ghafli H, Pavitt GD.
PMID: 34877508
iScience. 2021 Nov 14;24(12):103454. doi: 10.1016/j.isci.2021.103454. eCollection 2021 Dec 17.

eIF2B is the guanine nucleotide exchange factor (GEF) required for cytoplasmic protein synthesis initiation in eukaryotes and its regulation within the integrated stress response (ISR). It activates its partner factor eIF2, thereby promoting translation initiation. Here we provide evidence...

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Kershaw CJ, Jennings MD, Cortopassi F, et al. GTP binding to translation factor eIF2B stimulates its guanine nucleotide exchange activity. iScience. 2021;24(12):103454doi: 10.1016/j.isci.2021.103454.
Kershaw, C. J., Jennings, M. D., Cortopassi, F., Guaita, M., Al-Ghafli, H., & Pavitt, G. D. (2021). GTP binding to translation factor eIF2B stimulates its guanine nucleotide exchange activity. iScience, 24(12), 103454. https://doi.org/10.1016/j.isci.2021.103454
Kershaw, Christopher J, et al. "GTP binding to translation factor eIF2B stimulates its guanine nucleotide exchange activity." iScience vol. 24,12 (2021): 103454. doi: https://doi.org/10.1016/j.isci.2021.103454
Kershaw CJ, Jennings MD, Cortopassi F, Guaita M, Al-Ghafli H, Pavitt GD. GTP binding to translation factor eIF2B stimulates its guanine nucleotide exchange activity. iScience. 2021 Nov 14;24(12):103454. doi: 10.1016/j.isci.2021.103454. eCollection 2021 Dec 17. PMID: 34877508; PMCID: PMC8633983.
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Corrigendum: Mutations in SNORD118 cause the cerebral microangiopathy leukoencephalopathy with calcifications and cysts.

Nature genetics

Jenkinson EM, Rodero MP, Kasher PR, Uggenti C, Oojageer A, Goosey LC, Rose Y, Kershaw CJ, Urquhart JE, Williams SG, Bhaskar SS, O'Sullivan J, Baerlocher GM, Haubitz M, Aubert G, Barañano KW, Barnicoat AJ, Battini R, Berger A, Blair EM, Brunstrom-Hernandez JE, Buckard JA, Cassiman DM, Caumes R, Cordelli DM, De Waele LM, Fay AJ, Ferreira P, Fletcher NA, Fryer AE, Goel H, Hemingway CA, Henneke M, Hughes I, Jefferson RJ, Kumar R, Lagae L, Landrieu PG, Lourenço CM, Malpas TJ, Mehta SG, Metz I, Naidu S, Õunap K, Panzer A, Prabhakar P, Quaghebeur G, Schiffmann R, Sherr EH, Sinnathuray KR, Soh C, Stewart HS, Stone J, Van Esch H, Van Mol CE, Vanderver A, Wakeling EL, Whitney A, Pavitt GD, Griffiths-Jones S, Rice GI, Revy P, van der Knaap MS, Livingston JH, O'Keefe RT, Crow YJ.
PMID: 28138155
Nat Genet. 2017 Jan 31;49(2):317. doi: 10.1038/ng0217-317b.

No abstract available.

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Jenkinson EM, Rodero MP, Kasher PR, et al. Corrigendum: Mutations in SNORD118 cause the cerebral microangiopathy leukoencephalopathy with calcifications and cysts. Nat Genet. 2017;49(2):317doi: 10.1038/ng0217-317b.
Jenkinson, E. M., Rodero, M. P., Kasher, P. R., Uggenti, C., Oojageer, A., Goosey, L. C., Rose, Y., Kershaw, C. J., Urquhart, J. E., Williams, S. G., Bhaskar, S. S., O'Sullivan, J., Baerlocher, G. M., Haubitz, M., Aubert, G., Barañano, K. W., Barnicoat, A. J., Battini, R., Berger, A., Blair, E. M., Brunstrom-Hernandez, J. E., Buckard, J. A., Cassiman, D. M., Caumes, R., Cordelli, D. M., De Waele, L. M., Fay, A. J., Ferreira, P., Fletcher, N. A., Fryer, A. E., Goel, H., Hemingway, C. A., Henneke, M., Hughes, I., Jefferson, R. J., Kumar, R., Lagae, L., Landrieu, P. G., Lourenço, C. M., Malpas, T. J., Mehta, S. G., Metz, I., Naidu, S., Õunap, K., Panzer, A., Prabhakar, P., Quaghebeur, G., Schiffmann, R., Sherr, E. H., Sinnathuray, K. R., Soh, C., Stewart, H. S., Stone, J., Van Esch, H., Van Mol, C. E., Vanderver, A., Wakeling, E. L., Whitney, A., Pavitt, G. D., Griffiths-Jones, S., Rice, G. I., Revy, P., van der Knaap, M. S., Livingston, J. H., O'Keefe, R. T., & Crow, Y. J. (2017). Corrigendum: Mutations in SNORD118 cause the cerebral microangiopathy leukoencephalopathy with calcifications and cysts. Nature genetics, 49(2), 317. https://doi.org/10.1038/ng0217-317b
Jenkinson, Emma M, et al. "Corrigendum: Mutations in SNORD118 cause the cerebral microangiopathy leukoencephalopathy with calcifications and cysts." Nature genetics vol. 49,2 (2017): 317. doi: https://doi.org/10.1038/ng0217-317b
Jenkinson EM, Rodero MP, Kasher PR, Uggenti C, Oojageer A, Goosey LC, Rose Y, Kershaw CJ, Urquhart JE, Williams SG, Bhaskar SS, O'Sullivan J, Baerlocher GM, Haubitz M, Aubert G, Barañano KW, Barnicoat AJ, Battini R, Berger A, Blair EM, Brunstrom-Hernandez JE, Buckard JA, Cassiman DM, Caumes R, Cordelli DM, De Waele LM, Fay AJ, Ferreira P, Fletcher NA, Fryer AE, Goel H, Hemingway CA, Henneke M, Hughes I, Jefferson RJ, Kumar R, Lagae L, Landrieu PG, Lourenço CM, Malpas TJ, Mehta SG, Metz I, Naidu S, Õunap K, Panzer A, Prabhakar P, Quaghebeur G, Schiffmann R, Sherr EH, Sinnathuray KR, Soh C, Stewart HS, Stone J, Van Esch H, Van Mol CE, Vanderver A, Wakeling EL, Whitney A, Pavitt GD, Griffiths-Jones S, Rice GI, Revy P, van der Knaap MS, Livingston JH, O'Keefe RT, Crow YJ. Corrigendum: Mutations in SNORD118 cause the cerebral microangiopathy leukoencephalopathy with calcifications and cysts. Nat Genet. 2017 Jan 31;49(2):317. doi: 10.1038/ng0217-317b. PMID: 28138155.
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Growth and Clinical Outcomes of Very Low-Birth-Weight Infants Receiving Acidified vs Nonacidified Liquid Human Milk Fortifiers.

Nutrition in clinical practice : official publication of the American Society for Parenteral and Enteral Nutrition

Cordova EG, Soldateli B, Rosner B, Drouin K, Davitt E, Pepin HL, Ellard DM, Yu A, Bell K, Belfort MB.
PMID: 32840926
Nutr Clin Pract. 2021 Dec;36(6):1304-1311. doi: 10.1002/ncp.10569. Epub 2020 Aug 25.

BACKGROUND: Liquid human milk fortifiers are used commonly in neonatal intensive care. Use of an acidified HMF (A-HMF) is associated with transient metabolic acidosis, but whether growth outcomes differ between infants fed A-HMF vs nonacidified HMF (NA-HMF) remains unknown.METHODS:...

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Cordova EG, Soldateli B, Rosner B, et al. Growth and Clinical Outcomes of Very Low-Birth-Weight Infants Receiving Acidified vs Nonacidified Liquid Human Milk Fortifiers. Nutr Clin Pract. 2020;36(6):1304-1311doi: 10.1002/ncp.10569.
Cordova, E. G., Soldateli, B., Rosner, B., Drouin, K., Davitt, E., Pepin, H. L., Ellard, D. M., Yu, A., Bell, K., & Belfort, M. B. (2021). Growth and Clinical Outcomes of Very Low-Birth-Weight Infants Receiving Acidified vs Nonacidified Liquid Human Milk Fortifiers. Nutrition in clinical practice : official publication of the American Society for Parenteral and Enteral Nutrition, 36(6), 1304-1311. https://doi.org/10.1002/ncp.10569
Cordova, Erika G, et al. "Growth and Clinical Outcomes of Very Low-Birth-Weight Infants Receiving Acidified vs Nonacidified Liquid Human Milk Fortifiers." Nutrition in clinical practice : official publication of the American Society for Parenteral and Enteral Nutrition vol. 36,6 (2021): 1304-1311. doi: https://doi.org/10.1002/ncp.10569
Cordova EG, Soldateli B, Rosner B, Drouin K, Davitt E, Pepin HL, Ellard DM, Yu A, Bell K, Belfort MB. Growth and Clinical Outcomes of Very Low-Birth-Weight Infants Receiving Acidified vs Nonacidified Liquid Human Milk Fortifiers. Nutr Clin Pract. 2021 Dec;36(6):1304-1311. doi: 10.1002/ncp.10569. Epub 2020 Aug 25. PMID: 32840926.
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Transdermal microneedles for the programmable burst release of multiple vaccine payloads.

Nature biomedical engineering

Tran KTM, Gavitt TD, Farrell NJ, Curry EJ, Mara AB, Patel A, Brown L, Kilpatrick S, Piotrowska R, Mishra N, Szczepanek SM, Nguyen TD.
PMID: 33230304
Nat Biomed Eng. 2021 Sep;5(9):998-1007. doi: 10.1038/s41551-020-00650-4. Epub 2020 Nov 23.

Repeated bolus injections are associated with higher costs and poor compliance and can hinder the implementation of global immunization campaigns. Here, we report the development and preclinical testing of patches of transdermal core-shell microneedles-which were fabricated by the micromoulding...

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Tran KTM, Gavitt TD, Farrell NJ, et al. Transdermal microneedles for the programmable burst release of multiple vaccine payloads. Nat Biomed Eng. 2020;5(9):998-1007doi: 10.1038/s41551-020-00650-4.
Tran, K. T. M., Gavitt, T. D., Farrell, N. J., Curry, E. J., Mara, A. B., Patel, A., Brown, L., Kilpatrick, S., Piotrowska, R., Mishra, N., Szczepanek, S. M., & Nguyen, T. D. (2021). Transdermal microneedles for the programmable burst release of multiple vaccine payloads. Nature biomedical engineering, 5(9), 998-1007. https://doi.org/10.1038/s41551-020-00650-4
Tran, Khanh T M, et al. "Transdermal microneedles for the programmable burst release of multiple vaccine payloads." Nature biomedical engineering vol. 5,9 (2021): 998-1007. doi: https://doi.org/10.1038/s41551-020-00650-4
Tran KTM, Gavitt TD, Farrell NJ, Curry EJ, Mara AB, Patel A, Brown L, Kilpatrick S, Piotrowska R, Mishra N, Szczepanek SM, Nguyen TD. Transdermal microneedles for the programmable burst release of multiple vaccine payloads. Nat Biomed Eng. 2021 Sep;5(9):998-1007. doi: 10.1038/s41551-020-00650-4. Epub 2020 Nov 23. PMID: 33230304.
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In the Journal of the National Medical Association 100 years ago: salutatory.

Journal of the National Medical Association

Savitt TL.
PMID: 19998647
J Natl Med Assoc. 2009 Nov;101(11):1173-5. doi: 10.1016/s0027-9684(15)31114-7.

No abstract available.

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Savitt TL. In the Journal of the National Medical Association 100 years ago: salutatory. J Natl Med Assoc. 2009;101(11):1173-5doi: 10.1016/s0027-9684(15)31114-7.
Savitt, T. L. (2009). In the Journal of the National Medical Association 100 years ago: salutatory. Journal of the National Medical Association, 101(11), 1173-5. https://doi.org/10.1016/s0027-9684(15)31114-7
Savitt, Todd L. "In the Journal of the National Medical Association 100 years ago: salutatory." Journal of the National Medical Association vol. 101,11 (2009): 1173-5. doi: https://doi.org/10.1016/s0027-9684(15)31114-7
Savitt TL. In the Journal of the National Medical Association 100 years ago: salutatory. J Natl Med Assoc. 2009 Nov;101(11):1173-5. doi: 10.1016/s0027-9684(15)31114-7. PMID: 19998647.
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Practical observations on the diagnosis and treatment of syphilis.

Annals of western medicine and surgery

SAVITT LE.
PMID: 15394621
Ann West Med Surg. 1949 Nov;3(11):401-4.

No abstract available.

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SAVITT LE. Practical observations on the diagnosis and treatment of syphilis. Ann West Med Surg. 1949;3(11):401-4
SAVITT, L. E. (1949). Practical observations on the diagnosis and treatment of syphilis. Annals of western medicine and surgery, 3(11), 401-4.
SAVITT, L E. "Practical observations on the diagnosis and treatment of syphilis." Annals of western medicine and surgery vol. 3,11 (1949): 401-4.
SAVITT LE. Practical observations on the diagnosis and treatment of syphilis. Ann West Med Surg. 1949 Nov;3(11):401-4. PMID: 15394621.
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Use of the Word "Crisis" in Sickle Cell Disease: The Language of Sickle Cell.

Journal of the National Medical Association

Savitt TL, Smith WR, Haywood C, Creary MS.
PMID: 26744112
J Natl Med Assoc. Summer 2014;106(1):23-30. doi: 10.1016/S0027-9684(15)30067-5.

Language matters. The words used to name and describe disease phenomena are a reflection of society. The authors address the use of the word "crisis" in SCD from sociological, historical, medical, and patient perspectives. The term "crisis" became associated...

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Savitt TL, Smith WR, Haywood C, et al. Use of the Word "Crisis" in Sickle Cell Disease: The Language of Sickle Cell. J Natl Med Assoc. 0;106(1):23-30doi: 10.1016/S0027-9684(15)30067-5.
Savitt, T. L., Smith, W. R., Haywood, C., & Creary, M. S. (0). Use of the Word "Crisis" in Sickle Cell Disease: The Language of Sickle Cell. Journal of the National Medical Association, 106(1), 23-30. https://doi.org/10.1016/S0027-9684(15)30067-5
Savitt, Todd L, et al. "Use of the Word "Crisis" in Sickle Cell Disease: The Language of Sickle Cell." Journal of the National Medical Association vol. 106,1 (0): 23-30. doi: https://doi.org/10.1016/S0027-9684(15)30067-5
Savitt TL, Smith WR, Haywood C, Creary MS. Use of the Word "Crisis" in Sickle Cell Disease: The Language of Sickle Cell. J Natl Med Assoc. Summer 2014;106(1):23-30. doi: 10.1016/S0027-9684(15)30067-5. PMID: 26744112.
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Oleanolic Acid Prevents Increase in Blood Pressure and Nephrotoxicity in Nitric Oxide Dependent Type of Hypertension in Rats.

Pharmacognosy research

Bachhav SS, Bhutada MS, Patil SP, Sharma KS, Patil SD.
PMID: 26692754
Pharmacognosy Res. 2014 Oct-Dec;7(4):385-92. doi: 10.4103/0974-8490.159575.

BACKGROUND: Recently, we have reported antihypertensive activity of oleanolic acid (OA) in glucocorticoid-induced hypertension with restoration of nitric oxide (NO) level. However, the involvement of NO-releasing action of OA was unclear.OBJECTIVE: To explore antihypertensive activity of OA in N(ω)-nitro-L-arginine...

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Bachhav SS, Bhutada MS, Patil SP, et al. Oleanolic Acid Prevents Increase in Blood Pressure and Nephrotoxicity in Nitric Oxide Dependent Type of Hypertension in Rats. Pharmacognosy Res. 2014;7(4):385-92doi: 10.4103/0974-8490.159575.
Bachhav, S. S., Bhutada, M. S., Patil, S. P., Sharma, K. S., & Patil, S. D. (2014). Oleanolic Acid Prevents Increase in Blood Pressure and Nephrotoxicity in Nitric Oxide Dependent Type of Hypertension in Rats. Pharmacognosy research, 7(4), 385-92. https://doi.org/10.4103/0974-8490.159575
Bachhav, Sagar S, et al. "Oleanolic Acid Prevents Increase in Blood Pressure and Nephrotoxicity in Nitric Oxide Dependent Type of Hypertension in Rats." Pharmacognosy research vol. 7,4 (2014): 385-92. doi: https://doi.org/10.4103/0974-8490.159575
Bachhav SS, Bhutada MS, Patil SP, Sharma KS, Patil SD. Oleanolic Acid Prevents Increase in Blood Pressure and Nephrotoxicity in Nitric Oxide Dependent Type of Hypertension in Rats. Pharmacognosy Res. 2014 Oct-Dec;7(4):385-92. doi: 10.4103/0974-8490.159575. PMID: 26692754; PMCID: PMC4660519.
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Parallel High-Throughput Automated Assays to Measure Cell Growth and Beta-Galactosidase Reporter Gene Expression in the Yeast Saccharomyces cerevisiae.

Current protocols in chemical biology

Napper AD, Motlekar N, de Almeida RA, Pavitt GD.
PMID: 23836585
Curr Protoc Chem Biol. 2011 Mar 01;3(1):1-14. doi: 10.1002/9780470559277.ch100119.

Parallel high-throughput automated assays are described for the measurement of cell growth and β-galactosidase reporter gene expression from a single culture of the yeast S. cerevisiae. The dual assay measures the effect of test compounds on expression of a...

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Napper AD, Motlekar N, de Almeida RA, et al. Parallel High-Throughput Automated Assays to Measure Cell Growth and Beta-Galactosidase Reporter Gene Expression in the Yeast Saccharomyces cerevisiae. Curr Protoc Chem Biol. 2011;3(1):1-14doi: 10.1002/9780470559277.ch100119.
Napper, A. D., Motlekar, N., de Almeida, R. A., & Pavitt, G. D. (2011). Parallel High-Throughput Automated Assays to Measure Cell Growth and Beta-Galactosidase Reporter Gene Expression in the Yeast Saccharomyces cerevisiae. Current protocols in chemical biology, 3(1), 1-14. https://doi.org/10.1002/9780470559277.ch100119
Napper, Andrew D, et al. "Parallel High-Throughput Automated Assays to Measure Cell Growth and Beta-Galactosidase Reporter Gene Expression in the Yeast Saccharomyces cerevisiae." Current protocols in chemical biology vol. 3,1 (2011): 1-14. doi: https://doi.org/10.1002/9780470559277.ch100119
Napper AD, Motlekar N, de Almeida RA, Pavitt GD. Parallel High-Throughput Automated Assays to Measure Cell Growth and Beta-Galactosidase Reporter Gene Expression in the Yeast Saccharomyces cerevisiae. Curr Protoc Chem Biol. 2011 Mar 01;3(1):1-14. doi: 10.1002/9780470559277.ch100119. PMID: 23836585; PMCID: PMC4250931.
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