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Showing 13 to 24 of 97 entries
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A dual-time-window protocol to reduce acquisition time of dynamic tau PET imaging using [.

EJNMMI research

Kolinger GD, Vállez García D, Lohith TG, Hostetler ED, Sur C, Struyk A, Boellaard R, Koole M.
PMID: 34046730
EJNMMI Res. 2021 May 27;11(1):49. doi: 10.1186/s13550-021-00790-x.

BACKGROUND: [RESULTS: It was found that DVR from a DTW protocol with a 60-min break between two 30-min dynamic scans closely approximates the DVR from the uninterrupted TACCONCLUSIONS: Therefore, such DTW protocol has the potential to increase patient comfort...

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Kolinger GD, Vállez García D, Lohith TG, et al. A dual-time-window protocol to reduce acquisition time of dynamic tau PET imaging using [. EJNMMI Res. 2021;11(1):49doi: 10.1186/s13550-021-00790-x.
Kolinger, G. D., Vállez García, D., Lohith, T. G., Hostetler, E. D., Sur, C., Struyk, A., Boellaard, R., & Koole, M. (2021). A dual-time-window protocol to reduce acquisition time of dynamic tau PET imaging using [. EJNMMI research, 11(1), 49. https://doi.org/10.1186/s13550-021-00790-x
Kolinger, Guilherme D, et al. "A dual-time-window protocol to reduce acquisition time of dynamic tau PET imaging using [." EJNMMI research vol. 11,1 (2021): 49. doi: https://doi.org/10.1186/s13550-021-00790-x
Kolinger GD, Vállez García D, Lohith TG, Hostetler ED, Sur C, Struyk A, Boellaard R, Koole M. A dual-time-window protocol to reduce acquisition time of dynamic tau PET imaging using [. EJNMMI Res. 2021 May 27;11(1):49. doi: 10.1186/s13550-021-00790-x. PMID: 34046730; PMCID: PMC8160074.
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Antepartum Hemorrhage and Outcome of Very Low Birth Weight, Very Preterm Infants: A Population-Based Study.

American journal of perinatology

Klinger G, Bromiker R, Zaslavsky-Paltiel I, Sokolover N, Lerner-Geva L, Yogev Y, Reichman B.
PMID: 32446258
Am J Perinatol. 2021 Sep;38(11):1134-1141. doi: 10.1055/s-0040-1710353. Epub 2020 May 23.

OBJECTIVE: We aimed to determine the independent effect of maternal antepartum hemorrhage (APH) on mortality and major neonatal morbidities among very low birth weight (VLBW), very preterm infants.STUDY DESIGN: A population-based cohort study of VLBW singleton infants born at...

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Klinger G, Bromiker R, Zaslavsky-Paltiel I, et al. Antepartum Hemorrhage and Outcome of Very Low Birth Weight, Very Preterm Infants: A Population-Based Study. Am J Perinatol. 2020;38(11):1134-1141doi: 10.1055/s-0040-1710353.
Klinger, G., Bromiker, R., Zaslavsky-Paltiel, I., Sokolover, N., Lerner-Geva, L., Yogev, Y., Reichman, B. (2021). Antepartum Hemorrhage and Outcome of Very Low Birth Weight, Very Preterm Infants: A Population-Based Study. American journal of perinatology, 38(11), 1134-1141. https://doi.org/10.1055/s-0040-1710353
Klinger, Gil, et al. "Antepartum Hemorrhage and Outcome of Very Low Birth Weight, Very Preterm Infants: A Population-Based Study." American journal of perinatology vol. 38,11 (2021): 1134-1141. doi: https://doi.org/10.1055/s-0040-1710353
Klinger G, Bromiker R, Zaslavsky-Paltiel I, Sokolover N, Lerner-Geva L, Yogev Y, Reichman B. Antepartum Hemorrhage and Outcome of Very Low Birth Weight, Very Preterm Infants: A Population-Based Study. Am J Perinatol. 2021 Sep;38(11):1134-1141. doi: 10.1055/s-0040-1710353. Epub 2020 May 23. PMID: 32446258.
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Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype.

American journal of human genetics

Senum SR, Li YSM, Benson KA, Joli G, Olinger E, Lavu S, Madsen CD, Gregory AV, Neatu R, Kline TL, Audrézet MP, Outeda P, Nau CB, Meijer E, Ali H, Steinman TI, Mrug M, Phelan PJ, Watnick TJ, Peters DJM, Ong ACM, Conlon PJ, Perrone RD, Cornec-Le Gall E, Hogan MC, Torres VE, Sayer JA, Harris PC.
PMID: 34890546
Am J Hum Genet. 2021 Dec 07; doi: 10.1016/j.ajhg.2021.11.016. Epub 2021 Dec 07.

Autosomal dominant polycystic kidney disease (ADPKD), characterized by progressive cyst formation/expansion, results in enlarged kidneys and often end stage kidney disease. ADPKD is genetically heterogeneous; PKD1 and PKD2 are the common loci (∼78% and ∼15% of families) and GANAB,...

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Senum SR, Li YSM, Benson KA, et al. Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype. Am J Hum Genet. 2021;doi: 10.1016/j.ajhg.2021.11.016.
Senum, S. R., Li, Y. S. M., Benson, K. A., Joli, G., Olinger, E., Lavu, S., Madsen, C. D., Gregory, A. V., Neatu, R., Kline, T. L., Audrézet, M. P., Outeda, P., Nau, C. B., Meijer, E., Ali, H., Steinman, T. I., Mrug, M., Phelan, P. J., Watnick, T. J., Peters, D. J. M., Ong, A. C. M., Conlon, P. J., Perrone, R. D., Cornec-Le Gall, E., Hogan, M. C., Torres, V. E., Sayer, J. A., & Harris, P. C. (2021). Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype. American journal of human genetics, . https://doi.org/10.1016/j.ajhg.2021.11.016
Senum, Sarah R, et al. "Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype." American journal of human genetics vol. (2021). doi: https://doi.org/10.1016/j.ajhg.2021.11.016
Senum SR, Li YSM, Benson KA, Joli G, Olinger E, Lavu S, Madsen CD, Gregory AV, Neatu R, Kline TL, Audrézet MP, Outeda P, Nau CB, Meijer E, Ali H, Steinman TI, Mrug M, Phelan PJ, Watnick TJ, Peters DJM, Ong ACM, Conlon PJ, Perrone RD, Cornec-Le Gall E, Hogan MC, Torres VE, Sayer JA, Harris PC. Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype. Am J Hum Genet. 2021 Dec 07; doi: 10.1016/j.ajhg.2021.11.016. Epub 2021 Dec 07. PMID: 34890546.
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An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth.

Neuroscience and biobehavioral reviews

Slinger G, Otte WM, Braun KPJ, van Diessen E.
PMID: 34813826
Neurosci Biobehav Rev. 2021 Nov 20;132:211-223. doi: 10.1016/j.neubiorev.2021.11.028. Epub 2021 Nov 20.

Abnormalities of the brain network organization in focal epilepsy have been extensively quantified. However, the extent and directionality of abnormalities are highly variable and subtype insensitive. We conducted meta-analyses to obtain a more accurate and epilepsy type-specific quantification of...

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Slinger G, Otte WM, Braun KPJ, et al. An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth. Neurosci Biobehav Rev. 2021;132:211-223doi: 10.1016/j.neubiorev.2021.11.028.
Slinger, G., Otte, W. M., Braun, K. P. J., & van Diessen, E. (2021). An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth. Neuroscience and biobehavioral reviews, 132211-223. https://doi.org/10.1016/j.neubiorev.2021.11.028
Slinger, Geertruida, et al. "An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth." Neuroscience and biobehavioral reviews vol. 132 (2021): 211-223. doi: https://doi.org/10.1016/j.neubiorev.2021.11.028
Slinger G, Otte WM, Braun KPJ, van Diessen E. An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth. Neurosci Biobehav Rev. 2021 Nov 20;132:211-223. doi: 10.1016/j.neubiorev.2021.11.028. Epub 2021 Nov 20. PMID: 34813826.
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Repeatability of [.

EJNMMI research

Kolinger GD, Vállez García D, Kramer GM, Frings V, Smit EF, de Langen AJ, Dierckx RAJO, Hoekstra OS, Boellaard R.
PMID: 30734113
EJNMMI Res. 2019 Feb 07;9(1):14. doi: 10.1186/s13550-019-0481-1.

BACKGROUND: Total metabolic active tumour volume (TMATV) and total tumour burden (TTB) are increasingly studied as prognostic and predictive factors in non-small cell lung cancer (NSCLC) patients. In this study, we investigated the repeatability of TMATV and TTB as...

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Kolinger GD, Vállez García D, Kramer GM, et al. Repeatability of [. EJNMMI Res. 2019;9(1):14doi: 10.1186/s13550-019-0481-1.
Kolinger, G. D., Vállez García, D., Kramer, G. M., Frings, V., Smit, E. F., de Langen, A. J., Dierckx, R. A. J. O., Hoekstra, O. S., & Boellaard, R. (2019). Repeatability of [. EJNMMI research, 9(1), 14. https://doi.org/10.1186/s13550-019-0481-1
Kolinger, Guilherme D, et al. "Repeatability of [." EJNMMI research vol. 9,1 (2019): 14. doi: https://doi.org/10.1186/s13550-019-0481-1
Kolinger GD, Vállez García D, Kramer GM, Frings V, Smit EF, de Langen AJ, Dierckx RAJO, Hoekstra OS, Boellaard R. Repeatability of [. EJNMMI Res. 2019 Feb 07;9(1):14. doi: 10.1186/s13550-019-0481-1. PMID: 30734113; PMCID: PMC6367490.
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The Convergence of High-Consequence Livestock and Human Pathogen Research and Development: A Paradox of Zoonotic Disease.

Tropical medicine and infectious disease

Michelotti JM, Yeh KB, Beckham TR, Colby MM, Dasgupta D, Zuelke KA, Olinger GG.
PMID: 30274451
Trop Med Infect Dis. 2018 May 30;3(2). doi: 10.3390/tropicalmed3020055.

The World Health Organization (WHO) estimates that zoonotic diseases transmitted from animals to humans account for 75 percent of new and emerging infectious diseases. Globally, high-consequence pathogens that impact livestock and have the potential for human transmission create research...

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Michelotti JM, Yeh KB, Beckham TR, et al. The Convergence of High-Consequence Livestock and Human Pathogen Research and Development: A Paradox of Zoonotic Disease. Trop Med Infect Dis. 2018;3(2)doi: 10.3390/tropicalmed3020055.
Michelotti, J. M., Yeh, K. B., Beckham, T. R., Colby, M. M., Dasgupta, D., Zuelke, K. A., & Olinger, G. G. (2018). The Convergence of High-Consequence Livestock and Human Pathogen Research and Development: A Paradox of Zoonotic Disease. Tropical medicine and infectious disease, 3(2), . https://doi.org/10.3390/tropicalmed3020055
Michelotti, Julia M, et al. "The Convergence of High-Consequence Livestock and Human Pathogen Research and Development: A Paradox of Zoonotic Disease." Tropical medicine and infectious disease vol. 3,2 (2018). doi: https://doi.org/10.3390/tropicalmed3020055
Michelotti JM, Yeh KB, Beckham TR, Colby MM, Dasgupta D, Zuelke KA, Olinger GG. The Convergence of High-Consequence Livestock and Human Pathogen Research and Development: A Paradox of Zoonotic Disease. Trop Med Infect Dis. 2018 May 30;3(2). doi: 10.3390/tropicalmed3020055. PMID: 30274451; PMCID: PMC6073383.
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Physicians' perceptions of reimbursement as a barrier to comprehensive diabetes care.

American health & drug benefits

Pozniak A, Olinger L, Shier V.
PMID: 25126307
Am Health Drug Benefits. 2010 Jan;3(1):31-40.

BACKGROUND: As the incidence of diabetes increases, there is growing concern about the adequacy of reimbursement levels for delivering comprehensive diabetes care.OBJECTIVE: To investigate physicians' perceptions of the adequacy of reimbursement, as well as resources (eg, staff, facilities, materials),...

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Pozniak A, Olinger L, Shier V. Physicians' perceptions of reimbursement as a barrier to comprehensive diabetes care. Am Health Drug Benefits. 2010;3(1):31-40
Pozniak, A., Olinger, L., & Shier, V. (2010). Physicians' perceptions of reimbursement as a barrier to comprehensive diabetes care. American health & drug benefits, 3(1), 31-40.
Pozniak, Alyssa, et al. "Physicians' perceptions of reimbursement as a barrier to comprehensive diabetes care." American health & drug benefits vol. 3,1 (2010): 31-40.
Pozniak A, Olinger L, Shier V. Physicians' perceptions of reimbursement as a barrier to comprehensive diabetes care. Am Health Drug Benefits. 2010 Jan;3(1):31-40. PMID: 25126307; PMCID: PMC4106549.
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High-pressure polymorphism of titanium dioxide.

Science (New York, N.Y.)

Jamieson JC, Olinger B.
PMID: 17812118
Science. 1968 Aug 30;161(3844):893-5. doi: 10.1126/science.161.3844.893.

X-ray diffraction studies made in situ under conditions of high pressure and high temperature revealed the direct transition of rutile to the alpha lead dioxide form in titanium dioxide. Compressibility studies of this alpha lead dioxide form at room...

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Jamieson JC, Olinger B. High-pressure polymorphism of titanium dioxide. Science. 1968;161(3844):893-5doi: 10.1126/science.161.3844.893.
Jamieson, J. C., & Olinger, B. (1968). High-pressure polymorphism of titanium dioxide. Science (New York, N.Y.), 161(3844), 893-5. https://doi.org/10.1126/science.161.3844.893
Jamieson, J C, and Olinger, B. "High-pressure polymorphism of titanium dioxide." Science (New York, N.Y.) vol. 161,3844 (1968): 893-5. doi: https://doi.org/10.1126/science.161.3844.893
Jamieson JC, Olinger B. High-pressure polymorphism of titanium dioxide. Science. 1968 Aug 30;161(3844):893-5. doi: 10.1126/science.161.3844.893. PMID: 17812118.
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An Emergent Research and Policy Framework for Telehealth.

EGEMS (Washington, DC)

Edmunds M, Tuckson R, Lewis J, Atchinson B, Rheuban K, Fanberg H, Olinger L, Rosati R, Austein-Casnoff C, Capistrant G, Thomas L.
PMID: 28459085
EGEMS (Wash DC). 2017 Mar 29;5(2):1303. doi: 10.13063/2327-9214.1303. eCollection 2017.

CONTEXT: Telehealth is a fast-growing sector in health care, using a variety of technologies to exchange information across locations and to improve access, quality, and outcomes across the continuum of care. Thousands of studies and hundreds of systematic reviews...

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Edmunds M, Tuckson R, Lewis J, et al. An Emergent Research and Policy Framework for Telehealth. EGEMS (Wash DC). 2017;5(2):1303doi: 10.13063/2327-9214.1303.
Edmunds, M., Tuckson, R., Lewis, J., Atchinson, B., Rheuban, K., Fanberg, H., Olinger, L., Rosati, R., Austein-Casnoff, C., Capistrant, G., & Thomas, L. (2017). An Emergent Research and Policy Framework for Telehealth. EGEMS (Washington, DC), 5(2), 1303. https://doi.org/10.13063/2327-9214.1303
Edmunds, Margo, et al. "An Emergent Research and Policy Framework for Telehealth." EGEMS (Washington, DC) vol. 5,2 (2017): 1303. doi: https://doi.org/10.13063/2327-9214.1303
Edmunds M, Tuckson R, Lewis J, Atchinson B, Rheuban K, Fanberg H, Olinger L, Rosati R, Austein-Casnoff C, Capistrant G, Thomas L. An Emergent Research and Policy Framework for Telehealth. EGEMS (Wash DC). 2017 Mar 29;5(2):1303. doi: 10.13063/2327-9214.1303. eCollection 2017. PMID: 28459085; PMCID: PMC5389433.
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An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth.

Neuroscience and biobehavioral reviews

Slinger G, Otte WM, Braun KPJ, van Diessen E.
PMID: 34813826
Neurosci Biobehav Rev. 2021 Nov 20;132:211-223. doi: 10.1016/j.neubiorev.2021.11.028. Epub 2021 Nov 20.

Abnormalities of the brain network organization in focal epilepsy have been extensively quantified. However, the extent and directionality of abnormalities are highly variable and subtype insensitive. We conducted meta-analyses to obtain a more accurate and epilepsy type-specific quantification of...

Cite
Slinger G, Otte WM, Braun KPJ, et al. An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth. Neurosci Biobehav Rev. 2021;132:211-223doi: 10.1016/j.neubiorev.2021.11.028.
Slinger, G., Otte, W. M., Braun, K. P. J., & van Diessen, E. (2021). An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth. Neuroscience and biobehavioral reviews, 132211-223. https://doi.org/10.1016/j.neubiorev.2021.11.028
Slinger, Geertruida, et al. "An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth." Neuroscience and biobehavioral reviews vol. 132 (2021): 211-223. doi: https://doi.org/10.1016/j.neubiorev.2021.11.028
Slinger G, Otte WM, Braun KPJ, van Diessen E. An updated systematic review and meta-analysis of brain network organization in focal epilepsy: Looking back and forth. Neurosci Biobehav Rev. 2021 Nov 20;132:211-223. doi: 10.1016/j.neubiorev.2021.11.028. Epub 2021 Nov 20. PMID: 34813826.
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Update of genetic variants in CEP120 and CC2D2A-With an emphasis on genotype-phenotype correlations, tissue specific transcripts and exploring mutation specific exon skipping therapies.

Molecular genetics & genomic medicine

Barroso-Gil M, Olinger E, Ramsbottom SA, Molinari E, Miles CG, Sayer JA.
PMID: 33486889
Mol Genet Genomic Med. 2021 Jan 24;e1603. doi: 10.1002/mgg3.1603. Epub 2021 Jan 24.

BACKGROUND: Mutations in ciliary genes cause a spectrum of both overlapping and distinct clinical syndromes (ciliopathies). CEP120 and CC2D2A are paradigmatic examples for this genetic heterogeneity and pleiotropy as mutations in both cause Joubert syndrome but are also associated...

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Barroso-Gil M, Olinger E, Ramsbottom SA, et al. Update of genetic variants in CEP120 and CC2D2A-With an emphasis on genotype-phenotype correlations, tissue specific transcripts and exploring mutation specific exon skipping therapies. Mol Genet Genomic Med. 2021;e1603doi: 10.1002/mgg3.1603.
Barroso-Gil, M., Olinger, E., Ramsbottom, S. A., Molinari, E., Miles, C. G., & Sayer, J. A. (2021). Update of genetic variants in CEP120 and CC2D2A-With an emphasis on genotype-phenotype correlations, tissue specific transcripts and exploring mutation specific exon skipping therapies. Molecular genetics & genomic medicine, e1603. https://doi.org/10.1002/mgg3.1603
Barroso-Gil, Miguel, et al. "Update of genetic variants in CEP120 and CC2D2A-With an emphasis on genotype-phenotype correlations, tissue specific transcripts and exploring mutation specific exon skipping therapies." Molecular genetics & genomic medicine vol. (2021): e1603. doi: https://doi.org/10.1002/mgg3.1603
Barroso-Gil M, Olinger E, Ramsbottom SA, Molinari E, Miles CG, Sayer JA. Update of genetic variants in CEP120 and CC2D2A-With an emphasis on genotype-phenotype correlations, tissue specific transcripts and exploring mutation specific exon skipping therapies. Mol Genet Genomic Med. 2021 Jan 24;e1603. doi: 10.1002/mgg3.1603. Epub 2021 Jan 24. PMID: 33486889.
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Antibiotic Discontinuation 24 h After Neonatal Late-Onset Sepsis Work-Up-A Validated Decision Tree Model.

Frontiers in pediatrics

Goldberg O, Sokolover N, Bromiker R, Amitai N, Chodick G, Scheuerman O, Ben-Zvi H, Klinger G.
PMID: 34490157
Front Pediatr. 2021 Aug 19;9:693882. doi: 10.3389/fped.2021.693882. eCollection 2021.

No abstract available.

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Goldberg O, Sokolover N, Bromiker R, et al. Antibiotic Discontinuation 24 h After Neonatal Late-Onset Sepsis Work-Up-A Validated Decision Tree Model. Front Pediatr. 2021;9:693882doi: 10.3389/fped.2021.693882.
Goldberg, O., Sokolover, N., Bromiker, R., Amitai, N., Chodick, G., Scheuerman, O., Ben-Zvi, H., & Klinger, G. (2021). Antibiotic Discontinuation 24 h After Neonatal Late-Onset Sepsis Work-Up-A Validated Decision Tree Model. Frontiers in pediatrics, 9693882. https://doi.org/10.3389/fped.2021.693882
Goldberg, Ori, et al. "Antibiotic Discontinuation 24 h After Neonatal Late-Onset Sepsis Work-Up-A Validated Decision Tree Model." Frontiers in pediatrics vol. 9 (2021): 693882. doi: https://doi.org/10.3389/fped.2021.693882
Goldberg O, Sokolover N, Bromiker R, Amitai N, Chodick G, Scheuerman O, Ben-Zvi H, Klinger G. Antibiotic Discontinuation 24 h After Neonatal Late-Onset Sepsis Work-Up-A Validated Decision Tree Model. Front Pediatr. 2021 Aug 19;9:693882. doi: 10.3389/fped.2021.693882. eCollection 2021. PMID: 34490157; PMCID: PMC8417412.
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Showing 13 to 24 of 97 entries