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Spatial and time domain analysis of eye-tracking data during screening of brain magnetic resonance images.

PloS one

Suman AA, Russo C, Carrigan A, Nalepka P, Liquet-Weiland B, Newport RA, Kumari P, Di Ieva A.
PMID: 34855867
PLoS One. 2021 Dec 02;16(12):e0260717. doi: 10.1371/journal.pone.0260717. eCollection 2021.

INTRODUCTION: Eye-tracking research has been widely used in radiology applications. Prior studies exclusively analysed either temporal or spatial eye-tracking features, both of which alone do not completely characterise the spatiotemporal dynamics of radiologists' gaze features.PURPOSE: Our research aims to...

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Suman AA, Russo C, Carrigan A, et al. Spatial and time domain analysis of eye-tracking data during screening of brain magnetic resonance images. PLoS One. 2021;16(12):e0260717doi: 10.1371/journal.pone.0260717.
Suman, A. A., Russo, C., Carrigan, A., Nalepka, P., Liquet-Weiland, B., Newport, R. A., Kumari, P., & Di Ieva, A. (2021). Spatial and time domain analysis of eye-tracking data during screening of brain magnetic resonance images. PloS one, 16(12), e0260717. https://doi.org/10.1371/journal.pone.0260717
Suman, Abdulla Al, et al. "Spatial and time domain analysis of eye-tracking data during screening of brain magnetic resonance images." PloS one vol. 16,12 (2021): e0260717. doi: https://doi.org/10.1371/journal.pone.0260717
Suman AA, Russo C, Carrigan A, Nalepka P, Liquet-Weiland B, Newport RA, Kumari P, Di Ieva A. Spatial and time domain analysis of eye-tracking data during screening of brain magnetic resonance images. PLoS One. 2021 Dec 02;16(12):e0260717. doi: 10.1371/journal.pone.0260717. eCollection 2021. PMID: 34855867; PMCID: PMC8639086.
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Impairment of lysosomal function by Clostridioides difficile TcdB.

Molecular microbiology

Klepka C, Sandmann M, Tatge H, Mangan M, Arens A, Henkel D, Gerhard R.
PMID: 34931374
Mol Microbiol. 2021 Dec 20; doi: 10.1111/mmi.14864. Epub 2021 Dec 20.

TcdB is a potent cytotoxin produced by pathogenic Clostridioides difficile that inhibits Rho GTPases by mono-glucosylation. TcdB enters cells via receptor-mediated endocytosis. The pathogenic glucosyltransferase domain (GTD) egresses endosomes by pH-mediated conformational changes, and is subsequently released in an...

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Klepka C, Sandmann M, Tatge H, et al. Impairment of lysosomal function by Clostridioides difficile TcdB. Mol Microbiol. 2021;doi: 10.1111/mmi.14864.
Klepka, C., Sandmann, M., Tatge, H., Mangan, M., Arens, A., Henkel, D., & Gerhard, R. (2021). Impairment of lysosomal function by Clostridioides difficile TcdB. Molecular microbiology, . https://doi.org/10.1111/mmi.14864
Klepka, Carmen, et al. "Impairment of lysosomal function by Clostridioides difficile TcdB." Molecular microbiology vol. (2021). doi: https://doi.org/10.1111/mmi.14864
Klepka C, Sandmann M, Tatge H, Mangan M, Arens A, Henkel D, Gerhard R. Impairment of lysosomal function by Clostridioides difficile TcdB. Mol Microbiol. 2021 Dec 20; doi: 10.1111/mmi.14864. Epub 2021 Dec 20. PMID: 34931374.
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SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA-deficient cells.

Molecular oncology

Chan KK, Abdul-Sater Z, Sheth A, Mitchell DK, Sharma R, Edwards DM, He Y, Nalepa G, Rhodes SD, Clapp DW, Sierra Potchanant EA.
PMID: 34058059
Mol Oncol. 2021 May 31; doi: 10.1002/1878-0261.13027. Epub 2021 May 31.

The Fanconi anemia (FA) pathway safeguards genomic stability through cell cycle regulation and DNA damage repair. The canonical tumor suppressive role of FA proteins in the repair of DNA damage during interphase is well established, but their function in...

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Chan KK, Abdul-Sater Z, Sheth A, et al. SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA-deficient cells. Mol Oncol. 2021;doi: 10.1002/1878-0261.13027.
Chan, K. K., Abdul-Sater, Z., Sheth, A., Mitchell, D. K., Sharma, R., Edwards, D. M., He, Y., Nalepa, G., Rhodes, S. D., Clapp, D. W., & Sierra Potchanant, E. A. (2021). SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA-deficient cells. Molecular oncology, . https://doi.org/10.1002/1878-0261.13027
Chan, Ka-Kui, et al. "SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA-deficient cells." Molecular oncology vol. (2021). doi: https://doi.org/10.1002/1878-0261.13027
Chan KK, Abdul-Sater Z, Sheth A, Mitchell DK, Sharma R, Edwards DM, He Y, Nalepa G, Rhodes SD, Clapp DW, Sierra Potchanant EA. SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA-deficient cells. Mol Oncol. 2021 May 31; doi: 10.1002/1878-0261.13027. Epub 2021 May 31. PMID: 34058059.
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Reconceptualization of Hormetic Responses in the Frame of Redox Toxicology.

International journal of molecular sciences

Skaperda Z, Tekos F, Vardakas P, Nepka C, Kouretas D.
PMID: 35008472
Int J Mol Sci. 2021 Dec 21;23(1). doi: 10.3390/ijms23010049.

Cellular adaptive mechanisms emerging after exposure to low levels of toxic agents or stressful stimuli comprise an important biological feature that has gained considerable scientific interest. Investigations of low-dose exposures to diverse chemical compounds signify the non-linear mode of...

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Skaperda Z, Tekos F, Vardakas P, et al. Reconceptualization of Hormetic Responses in the Frame of Redox Toxicology. Int J Mol Sci. 2021;23(1)doi: 10.3390/ijms23010049.
Skaperda, Z., Tekos, F., Vardakas, P., Nepka, C., & Kouretas, D. (2021). Reconceptualization of Hormetic Responses in the Frame of Redox Toxicology. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010049
Skaperda, Zoi, et al. "Reconceptualization of Hormetic Responses in the Frame of Redox Toxicology." International journal of molecular sciences vol. 23,1 (2021). doi: https://doi.org/10.3390/ijms23010049
Skaperda Z, Tekos F, Vardakas P, Nepka C, Kouretas D. Reconceptualization of Hormetic Responses in the Frame of Redox Toxicology. Int J Mol Sci. 2021 Dec 21;23(1). doi: 10.3390/ijms23010049. PMID: 35008472.
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Showing 49 to 52 of 52 entries