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A Dual-Functional Fibrous Skeleton Implanted with Single-Atomic Co-N.

ACS nano

Huang T, Sun Y, Wu J, Jin J, Wei C, Shi Z, Wang M, Cai J, An XT, Wang P, Su C, Li YY, Sun J.
PMID: 34351143
ACS Nano. 2021 Sep 28;15(9):14105-14115. doi: 10.1021/acsnano.1c04642. Epub 2021 Aug 05.

Although lithium-sulfur (Li-S) batteries have long been touted as next-generation energy storage devices, the rampant dendrite growth at the anode side and sluggish redox kinetics at the cathode side drastically impede their practical application. Herein, a dual-functional fibrous skeleton...

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Huang T, Sun Y, Wu J, et al. A Dual-Functional Fibrous Skeleton Implanted with Single-Atomic Co-N. ACS Nano. 2021;15(9):14105-14115doi: 10.1021/acsnano.1c04642.
Huang, T., Sun, Y., Wu, J., Jin, J., Wei, C., Shi, Z., Wang, M., Cai, J., An, X. T., Wang, P., Su, C., Li, Y. Y., & Sun, J. (2021). A Dual-Functional Fibrous Skeleton Implanted with Single-Atomic Co-N. ACS nano, 15(9), 14105-14115. https://doi.org/10.1021/acsnano.1c04642
Huang, Ting, et al. "A Dual-Functional Fibrous Skeleton Implanted with Single-Atomic Co-N." ACS nano vol. 15,9 (2021): 14105-14115. doi: https://doi.org/10.1021/acsnano.1c04642
Huang T, Sun Y, Wu J, Jin J, Wei C, Shi Z, Wang M, Cai J, An XT, Wang P, Su C, Li YY, Sun J. A Dual-Functional Fibrous Skeleton Implanted with Single-Atomic Co-N. ACS Nano. 2021 Sep 28;15(9):14105-14115. doi: 10.1021/acsnano.1c04642. Epub 2021 Aug 05. PMID: 34351143.
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Effect of Antifungal-Treated Host Macrophages on .

The Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale

Li HB, Li N, Wen SR, Qiang MY, Yang ZH, Dong TX, Li YY, Kuang YQ.
PMID: 33680221
Can J Infect Dis Med Microbiol. 2021 Feb 18;2021:8838444. doi: 10.1155/2021/8838444. eCollection 2021.

METHODS: RAW264.1 macrophages were treated with MCF, ICZ, or AmB and then challenged with RESULTS: The activity of SOD was significantly increased in RAW264.1 cells that phagocytized CONCLUSION: Echinocandins may increase their antifungal efficacy by altering the innate immune...

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Li HB, Li N, Wen SR, et al. Effect of Antifungal-Treated Host Macrophages on . Can J Infect Dis Med Microbiol. 2021;2021:8838444doi: 10.1155/2021/8838444.
Li, H. B., Li, N., Wen, S. R., Qiang, M. Y., Yang, Z. H., Dong, T. X., Li, Y. Y., & Kuang, Y. Q. (2021). Effect of Antifungal-Treated Host Macrophages on . The Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale, 20218838444. https://doi.org/10.1155/2021/8838444
Li, Hong-Bin, et al. "Effect of Antifungal-Treated Host Macrophages on ." The Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale vol. 2021 (2021): 8838444. doi: https://doi.org/10.1155/2021/8838444
Li HB, Li N, Wen SR, Qiang MY, Yang ZH, Dong TX, Li YY, Kuang YQ. Effect of Antifungal-Treated Host Macrophages on . Can J Infect Dis Med Microbiol. 2021 Feb 18;2021:8838444. doi: 10.1155/2021/8838444. eCollection 2021. PMID: 33680221; PMCID: PMC7906804.
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Analysis of m6A-Related lncRNAs for Prognosis Value and Response to Immune Checkpoint Inhibitors Therapy in Hepatocellular Carcinoma.

Cancer management and research

Wang Y, Li N, Tian D, Zhou CW, Wang YH, Yang C, Zeng MS.
PMID: 34429653
Cancer Manag Res. 2021 Aug 16;13:6451-6471. doi: 10.2147/CMAR.S322179. eCollection 2021.

INTRODUCTION: N6-methyladenosine (m6A) modification and long non-coding RNAs (lncRNAs) play pivotal roles in the progression of hepatocellular carcinoma (HCC). However, how their interaction is involved in the prognostic value of HCC and immune checkpoint inhibitors (ICIs) therapy remains unclear.METHODS:...

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Wang Y, Li N, Tian D, et al. Analysis of m6A-Related lncRNAs for Prognosis Value and Response to Immune Checkpoint Inhibitors Therapy in Hepatocellular Carcinoma. Cancer Manag Res. 2021;13:6451-6471doi: 10.2147/CMAR.S322179.
Wang, Y., Li, N., Tian, D., Zhou, C. W., Wang, Y. H., Yang, C., & Zeng, M. S. (2021). Analysis of m6A-Related lncRNAs for Prognosis Value and Response to Immune Checkpoint Inhibitors Therapy in Hepatocellular Carcinoma. Cancer management and research, 136451-6471. https://doi.org/10.2147/CMAR.S322179
Wang, Yi, et al. "Analysis of m6A-Related lncRNAs for Prognosis Value and Response to Immune Checkpoint Inhibitors Therapy in Hepatocellular Carcinoma." Cancer management and research vol. 13 (2021): 6451-6471. doi: https://doi.org/10.2147/CMAR.S322179
Wang Y, Li N, Tian D, Zhou CW, Wang YH, Yang C, Zeng MS. Analysis of m6A-Related lncRNAs for Prognosis Value and Response to Immune Checkpoint Inhibitors Therapy in Hepatocellular Carcinoma. Cancer Manag Res. 2021 Aug 16;13:6451-6471. doi: 10.2147/CMAR.S322179. eCollection 2021. PMID: 34429653; PMCID: PMC8379396.
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Clinical Comparison of Three Sample-to-Answer Systems for Detecting SARS-CoV-2 in B.1.1.7 Lineage Emergence.

Infection and drug resistance

Jian MJ, Chung HY, Chang CK, Lin JC, Yeh KM, Chen CW, Li SY, Hsieh SS, Liu MT, Yang JR, Tang SH, Perng CL, Chang FY, Shang HS.
PMID: 34429623
Infect Drug Resist. 2021 Aug 17;14:3255-3261. doi: 10.2147/IDR.S328327. eCollection 2021.

PURPOSE: Accurate molecular diagnostic assays for detecting severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the causative agent of COVID-19, are needed for epidemiology studies and to support infection-control measures. We evaluated the analytical sensitivity and clinical performance of three...

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Jian MJ, Chung HY, Chang CK, et al. Clinical Comparison of Three Sample-to-Answer Systems for Detecting SARS-CoV-2 in B.1.1.7 Lineage Emergence. Infect Drug Resist. 2021;14:3255-3261doi: 10.2147/IDR.S328327.
Jian, M. J., Chung, H. Y., Chang, C. K., Lin, J. C., Yeh, K. M., Chen, C. W., Li, S. Y., Hsieh, S. S., Liu, M. T., Yang, J. R., Tang, S. H., Perng, C. L., Chang, F. Y., & Shang, H. S. (2021). Clinical Comparison of Three Sample-to-Answer Systems for Detecting SARS-CoV-2 in B.1.1.7 Lineage Emergence. Infection and drug resistance, 143255-3261. https://doi.org/10.2147/IDR.S328327
Jian, Ming-Jr, et al. "Clinical Comparison of Three Sample-to-Answer Systems for Detecting SARS-CoV-2 in B.1.1.7 Lineage Emergence." Infection and drug resistance vol. 14 (2021): 3255-3261. doi: https://doi.org/10.2147/IDR.S328327
Jian MJ, Chung HY, Chang CK, Lin JC, Yeh KM, Chen CW, Li SY, Hsieh SS, Liu MT, Yang JR, Tang SH, Perng CL, Chang FY, Shang HS. Clinical Comparison of Three Sample-to-Answer Systems for Detecting SARS-CoV-2 in B.1.1.7 Lineage Emergence. Infect Drug Resist. 2021 Aug 17;14:3255-3261. doi: 10.2147/IDR.S328327. eCollection 2021. PMID: 34429623; PMCID: PMC8380303.
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NAMPT promotes hepatitis B virus replication and liver cancer cell proliferation through the regulation of aerobic glycolysis.

Oncology letters

Guo HJ, Li HY, Chen ZH, Zhou WJ, Li JJ, Zhang JY, Wang J, Luo XY, Zeng T, Shi Z, Mo CF.
PMID: 33777213
Oncol Lett. 2021 May;21(5):390. doi: 10.3892/ol.2021.12651. Epub 2021 Mar 18.

Nicotinamide phosphoribosyltransferase (NAMPT) is a critical rate-limiting enzyme involved in NAD synthesis that has been shown to contribute to the progression of liver cancer. However, the potential role and mechanism of NAMPT in hepatitis B virus (HBV)-associated liver cancer...

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Guo HJ, Li HY, Chen ZH, et al. NAMPT promotes hepatitis B virus replication and liver cancer cell proliferation through the regulation of aerobic glycolysis. Oncol Lett. 2021;21(5):390doi: 10.3892/ol.2021.12651.
Guo, H. J., Li, H. Y., Chen, Z. H., Zhou, W. J., Li, J. J., Zhang, J. Y., Wang, J., Luo, X. Y., Zeng, T., Shi, Z., & Mo, C. F. (2021). NAMPT promotes hepatitis B virus replication and liver cancer cell proliferation through the regulation of aerobic glycolysis. Oncology letters, 21(5), 390. https://doi.org/10.3892/ol.2021.12651
Guo, Hui-Jie, et al. "NAMPT promotes hepatitis B virus replication and liver cancer cell proliferation through the regulation of aerobic glycolysis." Oncology letters vol. 21,5 (2021): 390. doi: https://doi.org/10.3892/ol.2021.12651
Guo HJ, Li HY, Chen ZH, Zhou WJ, Li JJ, Zhang JY, Wang J, Luo XY, Zeng T, Shi Z, Mo CF. NAMPT promotes hepatitis B virus replication and liver cancer cell proliferation through the regulation of aerobic glycolysis. Oncol Lett. 2021 May;21(5):390. doi: 10.3892/ol.2021.12651. Epub 2021 Mar 18. PMID: 33777213; PMCID: PMC7988713.
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CD169-positive macrophages enhance abscopal effect of radiofrequency ablation therapy in liver cancer.

Translational oncology

Song X, Li N, Liu Y, Wang Z, Wang T, Tan S, Li C, Qiu C, Gao L, Asano K, Tanaka M, Liang X, Liu X, Ma C.
PMID: 34883446
Transl Oncol. 2022 Jan;15(1):101306. doi: 10.1016/j.tranon.2021.101306. Epub 2021 Dec 06.

Radiofrequency ablation (RFA) is a widely used and effective treatment for primary or metastatic liver cancer with small-size lesions. However, the therapeutic effectiveness of RFA in controlling metastatic lesion or recurrence is still limited. As the major cell population...

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Song X, Li N, Liu Y, et al. CD169-positive macrophages enhance abscopal effect of radiofrequency ablation therapy in liver cancer. Transl Oncol. 2021;15(1):101306doi: 10.1016/j.tranon.2021.101306.
Song, X., Li, N., Liu, Y., Wang, Z., Wang, T., Tan, S., Li, C., Qiu, C., Gao, L., Asano, K., Tanaka, M., Liang, X., Liu, X., & Ma, C. (2022). CD169-positive macrophages enhance abscopal effect of radiofrequency ablation therapy in liver cancer. Translational oncology, 15(1), 101306. https://doi.org/10.1016/j.tranon.2021.101306
Song, Xiaojia, et al. "CD169-positive macrophages enhance abscopal effect of radiofrequency ablation therapy in liver cancer." Translational oncology vol. 15,1 (2022): 101306. doi: https://doi.org/10.1016/j.tranon.2021.101306
Song X, Li N, Liu Y, Wang Z, Wang T, Tan S, Li C, Qiu C, Gao L, Asano K, Tanaka M, Liang X, Liu X, Ma C. CD169-positive macrophages enhance abscopal effect of radiofrequency ablation therapy in liver cancer. Transl Oncol. 2022 Jan;15(1):101306. doi: 10.1016/j.tranon.2021.101306. Epub 2021 Dec 06. PMID: 34883446.
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RND3 attenuates oxidative stress and vascular remodeling in spontaneously hypertensive rat via inhibiting ROCK1 signaling.

Redox biology

Wu N, Zheng F, Li N, Han Y, Xiong XQ, Wang JJ, Chen Q, Li YH, Zhu GQ, Zhou YB.
PMID: 34883403
Redox Biol. 2021 Dec 06;48:102204. doi: 10.1016/j.redox.2021.102204. Epub 2021 Dec 06.

Superoxide and vascular smooth muscle cells (VSMCs) migration and proliferation play crucial roles in the vascular remodeling. Vascular remodeling contributes to the development and complications of hypertension. Rho family GTPase 3 (RND3 or RhoE), an atypical small Rho-GTPase, is...

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Wu N, Zheng F, Li N, et al. RND3 attenuates oxidative stress and vascular remodeling in spontaneously hypertensive rat via inhibiting ROCK1 signaling. Redox Biol. 2021;48:102204doi: 10.1016/j.redox.2021.102204.
Wu, N., Zheng, F., Li, N., Han, Y., Xiong, X. Q., Wang, J. J., Chen, Q., Li, Y. H., Zhu, G. Q., & Zhou, Y. B. (2021). RND3 attenuates oxidative stress and vascular remodeling in spontaneously hypertensive rat via inhibiting ROCK1 signaling. Redox biology, 48102204. https://doi.org/10.1016/j.redox.2021.102204
Wu, Nan, et al. "RND3 attenuates oxidative stress and vascular remodeling in spontaneously hypertensive rat via inhibiting ROCK1 signaling." Redox biology vol. 48 (2021): 102204. doi: https://doi.org/10.1016/j.redox.2021.102204
Wu N, Zheng F, Li N, Han Y, Xiong XQ, Wang JJ, Chen Q, Li YH, Zhu GQ, Zhou YB. RND3 attenuates oxidative stress and vascular remodeling in spontaneously hypertensive rat via inhibiting ROCK1 signaling. Redox Biol. 2021 Dec 06;48:102204. doi: 10.1016/j.redox.2021.102204. Epub 2021 Dec 06. PMID: 34883403.
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Bisphenol A-induced DNA damages promote to lymphoma progression in human lymphoblastoid cells through aberrant CTNNB1 signaling pathway.

iScience

Chen YK, Tan YY, Yao M, Lin HC, Tsai MH, Li YY, Hsu YJ, Huang TT, Chang CW, Cheng CM, Chuang CY.
PMID: 34401669
iScience. 2021 Jul 21;24(8):102888. doi: 10.1016/j.isci.2021.102888. eCollection 2021 Aug 20.

Lymphoma is a group of blood cancers that develop from the immune system, and one of the main risk factors is associated with exposure to environmental chemicals. Bisphenol A (BPA) is a common chemical used in the manufacture of...

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Chen YK, Tan YY, Yao M, et al. Bisphenol A-induced DNA damages promote to lymphoma progression in human lymphoblastoid cells through aberrant CTNNB1 signaling pathway. iScience. 2021;24(8):102888doi: 10.1016/j.isci.2021.102888.
Chen, Y. K., Tan, Y. Y., Yao, M., Lin, H. C., Tsai, M. H., Li, Y. Y., Hsu, Y. J., Huang, T. T., Chang, C. W., Cheng, C. M., & Chuang, C. Y. (2021). Bisphenol A-induced DNA damages promote to lymphoma progression in human lymphoblastoid cells through aberrant CTNNB1 signaling pathway. iScience, 24(8), 102888. https://doi.org/10.1016/j.isci.2021.102888
Chen, Yin-Kai, et al. "Bisphenol A-induced DNA damages promote to lymphoma progression in human lymphoblastoid cells through aberrant CTNNB1 signaling pathway." iScience vol. 24,8 (2021): 102888. doi: https://doi.org/10.1016/j.isci.2021.102888
Chen YK, Tan YY, Yao M, Lin HC, Tsai MH, Li YY, Hsu YJ, Huang TT, Chang CW, Cheng CM, Chuang CY. Bisphenol A-induced DNA damages promote to lymphoma progression in human lymphoblastoid cells through aberrant CTNNB1 signaling pathway. iScience. 2021 Jul 21;24(8):102888. doi: 10.1016/j.isci.2021.102888. eCollection 2021 Aug 20. PMID: 34401669; PMCID: PMC8350018.
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Bepridil, a class IV antiarrhythmic agent, can block the TREK-1 potassium channel.

Annals of translational medicine

Wang Y, Fu Z, Ma Z, Li N, Shang H.
PMID: 34430564
Ann Transl Med. 2021 Jul;9(14):1123. doi: 10.21037/atm-20-7971.

BACKGROUND: The TWIK-related potassium channel (TREK-1) can be regulated by different stimuli. However, it is not clear whether some antiarrhythmics affect the activity of TREK-1. In the present study, the effect of bepridil on the TREK-1 currents is investigated.METHODS:...

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Wang Y, Fu Z, Ma Z, et al. Bepridil, a class IV antiarrhythmic agent, can block the TREK-1 potassium channel. Ann Transl Med. 2021;9(14):1123doi: 10.21037/atm-20-7971.
Wang, Y., Fu, Z., Ma, Z., Li, N., & Shang, H. (2021). Bepridil, a class IV antiarrhythmic agent, can block the TREK-1 potassium channel. Annals of translational medicine, 9(14), 1123. https://doi.org/10.21037/atm-20-7971
Wang, Ying, et al. "Bepridil, a class IV antiarrhythmic agent, can block the TREK-1 potassium channel." Annals of translational medicine vol. 9,14 (2021): 1123. doi: https://doi.org/10.21037/atm-20-7971
Wang Y, Fu Z, Ma Z, Li N, Shang H. Bepridil, a class IV antiarrhythmic agent, can block the TREK-1 potassium channel. Ann Transl Med. 2021 Jul;9(14):1123. doi: 10.21037/atm-20-7971. PMID: 34430564; PMCID: PMC8350656.
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Showing 7285 to 7293 of 7293 entries