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Sex and Racial Differences in High-Density Lipoprotein Levels in Acute Coronary Syndromes.

The American journal of the medical sciences

Ozaki A, Bravo-Jaimes K, Smotherman C, Kraemer DF, Samura A, Velarde GP.
PMID: 33961844
Am J Med Sci. 2021 Nov;362(5):435-441. doi: 10.1016/j.amjms.2021.04.013. Epub 2021 May 04.

BACKGROUND: This study aimed to assess sex and racial differences related to high-density lipoprotein cholesterol (HDL-C) levels in those presenting with acute coronary syndromes (ACS).METHODS: Records from patients with ACS presenting to the Emergency Department of University of Florida...

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Ozaki A, Bravo-Jaimes K, Smotherman C, et al. Sex and Racial Differences in High-Density Lipoprotein Levels in Acute Coronary Syndromes. Am J Med Sci. 2021;362(5):435-441doi: 10.1016/j.amjms.2021.04.013.
Ozaki, A., Bravo-Jaimes, K., Smotherman, C., Kraemer, D. F., Samura, A., & Velarde, G. P. (2021). Sex and Racial Differences in High-Density Lipoprotein Levels in Acute Coronary Syndromes. The American journal of the medical sciences, 362(5), 435-441. https://doi.org/10.1016/j.amjms.2021.04.013
Ozaki, Asuka, et al. "Sex and Racial Differences in High-Density Lipoprotein Levels in Acute Coronary Syndromes." The American journal of the medical sciences vol. 362,5 (2021): 435-441. doi: https://doi.org/10.1016/j.amjms.2021.04.013
Ozaki A, Bravo-Jaimes K, Smotherman C, Kraemer DF, Samura A, Velarde GP. Sex and Racial Differences in High-Density Lipoprotein Levels in Acute Coronary Syndromes. Am J Med Sci. 2021 Nov;362(5):435-441. doi: 10.1016/j.amjms.2021.04.013. Epub 2021 May 04. PMID: 33961844.
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SNAP25 mutation disrupts metabolic homeostasis, steroid hormone production and central neurobehavior.

Biochimica et biophysica acta. Molecular basis of disease

Hao X, Zhu B, Yang P, Dong D, Sahbaie P, Oliver PL, Shen WJ, Azhar S, Kraemer FB.
PMID: 34826585
Biochim Biophys Acta Mol Basis Dis. 2021 Nov 24;1868(2):166304. doi: 10.1016/j.bbadis.2021.166304. Epub 2021 Nov 24.

OBJECTIVE: SNAP-25 is one of the key proteins involved in formation of soluble N-ethylmaleimide-sensitive factor attachment protein receptor (SNARE) complexes that are at the core of hormonal secretion and synaptic transmission. Altered expression or function of SNAP-25 can contribute...

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Hao X, Zhu B, Yang P, et al. SNAP25 mutation disrupts metabolic homeostasis, steroid hormone production and central neurobehavior. Biochim Biophys Acta Mol Basis Dis. 2021;1868(2):166304doi: 10.1016/j.bbadis.2021.166304.
Hao, X., Zhu, B., Yang, P., Dong, D., Sahbaie, P., Oliver, P. L., Shen, W. J., Azhar, S., & Kraemer, F. B. (2021). SNAP25 mutation disrupts metabolic homeostasis, steroid hormone production and central neurobehavior. Biochimica et biophysica acta. Molecular basis of disease, 1868(2), 166304. https://doi.org/10.1016/j.bbadis.2021.166304
Hao, Xiao, et al. "SNAP25 mutation disrupts metabolic homeostasis, steroid hormone production and central neurobehavior." Biochimica et biophysica acta. Molecular basis of disease vol. 1868,2 (2021): 166304. doi: https://doi.org/10.1016/j.bbadis.2021.166304
Hao X, Zhu B, Yang P, Dong D, Sahbaie P, Oliver PL, Shen WJ, Azhar S, Kraemer FB. SNAP25 mutation disrupts metabolic homeostasis, steroid hormone production and central neurobehavior. Biochim Biophys Acta Mol Basis Dis. 2021 Nov 24;1868(2):166304. doi: 10.1016/j.bbadis.2021.166304. Epub 2021 Nov 24. PMID: 34826585.
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Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans.

G3 (Bethesda, Md.)

Saxton AD, Kraemer BC.
PMID: 33963840
G3 (Bethesda). 2021 Aug 07;11(8). doi: 10.1093/g3journal/jkab158.

Amyotrophic lateral sclerosis (ALS) is a debilitating, fatal neurodegenerative disease that causes rapid muscle wasting. It shares a spectrum of symptoms and pathology with frontotemporal lobar degeneration (FTLD). These diseases are caused by aberrant activity of a set of...

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Saxton AD, Kraemer BC. Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans. G3 (Bethesda). 2021;11(8)doi: 10.1093/g3journal/jkab158.
Saxton, A. D., & Kraemer, B. C. (2021). Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans. G3 (Bethesda, Md.), 11(8), . https://doi.org/10.1093/g3journal/jkab158
Saxton, Aleen D, and Kraemer, Brian C. "Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans." G3 (Bethesda, Md.) vol. 11,8 (2021). doi: https://doi.org/10.1093/g3journal/jkab158
Saxton AD, Kraemer BC. Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans. G3 (Bethesda). 2021 Aug 07;11(8). doi: 10.1093/g3journal/jkab158. PMID: 33963840; PMCID: PMC8496285.
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Perceptions of Resident Autonomy in Internal Medicine, Pediatrics, and Internal Medicine-Pediatrics.

Cureus

Mieczkowski AE, Gonzaga AMR, Kraemer K, Habicht R, Friedland AR, Rubio D, Van Deusen R.
PMID: 33842175
Cureus. 2021 Mar 10;13(3):e13805. doi: 10.7759/cureus.13805.

BACKGROUND: Although graduated autonomy is an essential component of residency training, we have an incomplete understanding of resident and attending faculty perceptions of autonomy.OBJECTIVE: In this study, we assessed differences in perceived autonomy among residents and faculty in pediatrics,...

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Mieczkowski AE, Gonzaga AMR, Kraemer K, et al. Perceptions of Resident Autonomy in Internal Medicine, Pediatrics, and Internal Medicine-Pediatrics. Cureus. 2021;13(3):e13805doi: 10.7759/cureus.13805.
Mieczkowski, A. E., Gonzaga, A. M. R., Kraemer, K., Habicht, R., Friedland, A. R., Rubio, D., & Van Deusen, R. (2021). Perceptions of Resident Autonomy in Internal Medicine, Pediatrics, and Internal Medicine-Pediatrics. Cureus, 13(3), e13805. https://doi.org/10.7759/cureus.13805
Mieczkowski, Alexandra E, et al. "Perceptions of Resident Autonomy in Internal Medicine, Pediatrics, and Internal Medicine-Pediatrics." Cureus vol. 13,3 (2021): e13805. doi: https://doi.org/10.7759/cureus.13805
Mieczkowski AE, Gonzaga AMR, Kraemer K, Habicht R, Friedland AR, Rubio D, Van Deusen R. Perceptions of Resident Autonomy in Internal Medicine, Pediatrics, and Internal Medicine-Pediatrics. Cureus. 2021 Mar 10;13(3):e13805. doi: 10.7759/cureus.13805. PMID: 33842175; PMCID: PMC8027953.
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