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Showing 13 to 24 of 25 entries
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Medium-term Electrophysiologic Effects of a Cellularized Scaffold Implanted in Rats After Myocardial Infarction.

Cureus

Kolettis TM, Bagli E, Barka E, Kouroupis D, Kontonika M, Vilaeti AD, Markou M, Roumpi M, Maltabe V, La Rocca V, Agathopoulos S, Fotsis T.
PMID: 30214847
Cureus. 2018 Jul 10;10(7):e2959. doi: 10.7759/cureus.2959.

Background Cardiac repair strategies are being evaluated for myocardial infarctions, but the safety issues regarding their arrhythmogenic potential remain unresolved. By utilizing the in-vivo rat model, we have examined the medium-term electrophysiologic effects of a biomaterial scaffold that has...

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Kolettis TM, Bagli E, Barka E, et al. Medium-term Electrophysiologic Effects of a Cellularized Scaffold Implanted in Rats After Myocardial Infarction. Cureus. 2018;10(7):e2959doi: 10.7759/cureus.2959.
Kolettis, T. M., Bagli, E., Barka, E., Kouroupis, D., Kontonika, M., Vilaeti, A. D., Markou, M., Roumpi, M., Maltabe, V., La Rocca, V., Agathopoulos, S., & Fotsis, T. (2018). Medium-term Electrophysiologic Effects of a Cellularized Scaffold Implanted in Rats After Myocardial Infarction. Cureus, 10(7), e2959. https://doi.org/10.7759/cureus.2959
Kolettis, Theofilos M, et al. "Medium-term Electrophysiologic Effects of a Cellularized Scaffold Implanted in Rats After Myocardial Infarction." Cureus vol. 10,7 (2018): e2959. doi: https://doi.org/10.7759/cureus.2959
Kolettis TM, Bagli E, Barka E, Kouroupis D, Kontonika M, Vilaeti AD, Markou M, Roumpi M, Maltabe V, La Rocca V, Agathopoulos S, Fotsis T. Medium-term Electrophysiologic Effects of a Cellularized Scaffold Implanted in Rats After Myocardial Infarction. Cureus. 2018 Jul 10;10(7):e2959. doi: 10.7759/cureus.2959. PMID: 30214847; PMCID: PMC6132679.
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Formalin Versus Bouin Solution for Testis Biopsies: Which Is the Better Fixative?.

Clinical pathology (Thousand Oaks, Ventura County, Calif.)

Ellenburg JL, Kolettis P, Drwiega JC, Posey AM, Goldberg M, Mehrad M, Giannico G, Gordetsky J.
PMID: 31922127
Clin Pathol. 2020 Jan 02;13:2632010X19897262. doi: 10.1177/2632010X19897262. eCollection 2020.

PURPOSE: Some resources recommended Bouin solution for the fixation of testis biopsy specimens. We compared the histologic quality of rat testicular tissue using buffered formalin and Bouin solution as fixatives.METHODS: We prospectively compared the histologic quality of rat testicular...

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Ellenburg JL, Kolettis P, Drwiega JC, et al. Formalin Versus Bouin Solution for Testis Biopsies: Which Is the Better Fixative?. Clin Pathol. 2020;13:2632010X19897262doi: 10.1177/2632010X19897262.
Ellenburg, J. L., Kolettis, P., Drwiega, J. C., Posey, A. M., Goldberg, M., Mehrad, M., Giannico, G., & Gordetsky, J. (2020). Formalin Versus Bouin Solution for Testis Biopsies: Which Is the Better Fixative?. Clinical pathology (Thousand Oaks, Ventura County, Calif.), 132632010X19897262. https://doi.org/10.1177/2632010X19897262
Ellenburg, James L, et al. "Formalin Versus Bouin Solution for Testis Biopsies: Which Is the Better Fixative?." Clinical pathology (Thousand Oaks, Ventura County, Calif.) vol. 13 (2020): 2632010X19897262. doi: https://doi.org/10.1177/2632010X19897262
Ellenburg JL, Kolettis P, Drwiega JC, Posey AM, Goldberg M, Mehrad M, Giannico G, Gordetsky J. Formalin Versus Bouin Solution for Testis Biopsies: Which Is the Better Fixative?. Clin Pathol. 2020 Jan 02;13:2632010X19897262. doi: 10.1177/2632010X19897262. eCollection 2020. PMID: 31922127; PMCID: PMC6940599.
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Concomitant Brugada syndrome substrate ablation and epicardial abdominal cardioverter-defibrillator implantation in a child.

HeartRhythm case reports

de Asmundis C, Chierchia GB, Baltogiannis GG, Salghetti F, Sieira J, Kolettis TM, Tasi K, Vlahos A, Czapla J, Brugada P, La Meir M.
PMID: 29928586
HeartRhythm Case Rep. 2018 Mar 17;4(6):214-218. doi: 10.1016/j.hrcr.2017.12.004. eCollection 2018 Jun.

No abstract available.

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de Asmundis C, Chierchia GB, Baltogiannis GG, et al. Concomitant Brugada syndrome substrate ablation and epicardial abdominal cardioverter-defibrillator implantation in a child. HeartRhythm Case Rep. 2018;4(6):214-218doi: 10.1016/j.hrcr.2017.12.004.
de Asmundis, C., Chierchia, G. B., Baltogiannis, G. G., Salghetti, F., Sieira, J., Kolettis, T. M., Tasi, K., Vlahos, A., Czapla, J., Brugada, P., & La Meir, M. (2018). Concomitant Brugada syndrome substrate ablation and epicardial abdominal cardioverter-defibrillator implantation in a child. HeartRhythm case reports, 4(6), 214-218. https://doi.org/10.1016/j.hrcr.2017.12.004
de Asmundis, Carlo, et al. "Concomitant Brugada syndrome substrate ablation and epicardial abdominal cardioverter-defibrillator implantation in a child." HeartRhythm case reports vol. 4,6 (2018): 214-218. doi: https://doi.org/10.1016/j.hrcr.2017.12.004
de Asmundis C, Chierchia GB, Baltogiannis GG, Salghetti F, Sieira J, Kolettis TM, Tasi K, Vlahos A, Czapla J, Brugada P, La Meir M. Concomitant Brugada syndrome substrate ablation and epicardial abdominal cardioverter-defibrillator implantation in a child. HeartRhythm Case Rep. 2018 Mar 17;4(6):214-218. doi: 10.1016/j.hrcr.2017.12.004. eCollection 2018 Jun. PMID: 29928586; PMCID: PMC6007146.
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Signal-averaged electrocardiography: Past, present, and future.

Journal of arrhythmia

Gatzoulis KA, Arsenos P, Trachanas K, Dilaveris P, Antoniou C, Tsiachris D, Sideris S, Kolettis TM, Tousoulis D.
PMID: 29951136
J Arrhythm. 2018 May 28;34(3):222-229. doi: 10.1002/joa3.12062. eCollection 2018 Jun.

Signal-averaged electrocardiography records delayed depolarization of myocardial areas with slow conduction that can form the substrate for monomorphic ventricular tachycardia. This technique has been examined mostly in patients with coronary artery disease, but its use has been declined over...

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Gatzoulis KA, Arsenos P, Trachanas K, et al. Signal-averaged electrocardiography: Past, present, and future. J Arrhythm. 2018;34(3):222-229doi: 10.1002/joa3.12062.
Gatzoulis, K. A., Arsenos, P., Trachanas, K., Dilaveris, P., Antoniou, C., Tsiachris, D., Sideris, S., Kolettis, T. M., & Tousoulis, D. (2018). Signal-averaged electrocardiography: Past, present, and future. Journal of arrhythmia, 34(3), 222-229. https://doi.org/10.1002/joa3.12062
Gatzoulis, Konstantinos A, et al. "Signal-averaged electrocardiography: Past, present, and future." Journal of arrhythmia vol. 34,3 (2018): 222-229. doi: https://doi.org/10.1002/joa3.12062
Gatzoulis KA, Arsenos P, Trachanas K, Dilaveris P, Antoniou C, Tsiachris D, Sideris S, Kolettis TM, Tousoulis D. Signal-averaged electrocardiography: Past, present, and future. J Arrhythm. 2018 May 28;34(3):222-229. doi: 10.1002/joa3.12062. eCollection 2018 Jun. PMID: 29951136; PMCID: PMC6010001.
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Local conduction during acute myocardial infarction in rats: Interplay between central sympathetic activation and endothelin.

Journal of arrhythmia

Kolettis TM, Kontonika M, La Rocca V, Vlahos AP, Baltogiannis GG, Kyriakides ZS.
PMID: 28416983
J Arrhythm. 2017 Apr;33(2):144-146. doi: 10.1016/j.joa.2016.07.010. Epub 2016 Aug 31.

We investigated the effects of autonomic dysfunction and endothelin on local conduction and arrhythmogenesis during myocardial infarction. We recorded ventricular tachyarrhythmias, monophasic action potentials, and activation sequences in wild-type and ET

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Kolettis TM, Kontonika M, La Rocca V, et al. Local conduction during acute myocardial infarction in rats: Interplay between central sympathetic activation and endothelin. J Arrhythm. 2016;33(2):144-146doi: 10.1016/j.joa.2016.07.010.
Kolettis, T. M., Kontonika, M., La Rocca, V., Vlahos, A. P., Baltogiannis, G. G., & Kyriakides, Z. S. (2017). Local conduction during acute myocardial infarction in rats: Interplay between central sympathetic activation and endothelin. Journal of arrhythmia, 33(2), 144-146. https://doi.org/10.1016/j.joa.2016.07.010
Kolettis, Theofilos M, et al. "Local conduction during acute myocardial infarction in rats: Interplay between central sympathetic activation and endothelin." Journal of arrhythmia vol. 33,2 (2017): 144-146. doi: https://doi.org/10.1016/j.joa.2016.07.010
Kolettis TM, Kontonika M, La Rocca V, Vlahos AP, Baltogiannis GG, Kyriakides ZS. Local conduction during acute myocardial infarction in rats: Interplay between central sympathetic activation and endothelin. J Arrhythm. 2017 Apr;33(2):144-146. doi: 10.1016/j.joa.2016.07.010. Epub 2016 Aug 31. PMID: 28416983; PMCID: PMC5388042.
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Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats.

International journal of clinical and experimental medicine

Megalou AJ, Glava C, Oikonomidis DL, Vilaeti A, Agelaki MG, Baltogiannis GG, Papalois A, Vlahos AP, Kolettis TM.
PMID: 21072267
Int J Clin Exp Med. 2010 Oct 23;3(4):332-40.

The role of transforming growth factor-β in the pathogenesis of pulmonary arterial hypertension is unclear. We examined the effects of T9429, an antibody against transforming growth factor-β receptors, on hemodynamic, histological and functional parameters in the rat model of...

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Megalou AJ, Glava C, Oikonomidis DL, et al. Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats. Int J Clin Exp Med. 2010;3(4):332-40
Megalou, A. J., Glava, C., Oikonomidis, D. L., Vilaeti, A., Agelaki, M. G., Baltogiannis, G. G., Papalois, A., Vlahos, A. P., & Kolettis, T. M. (2010). Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats. International journal of clinical and experimental medicine, 3(4), 332-40.
Megalou, Aikaterini J, et al. "Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats." International journal of clinical and experimental medicine vol. 3,4 (2010): 332-40.
Megalou AJ, Glava C, Oikonomidis DL, Vilaeti A, Agelaki MG, Baltogiannis GG, Papalois A, Vlahos AP, Kolettis TM. Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats. Int J Clin Exp Med. 2010 Oct 23;3(4):332-40. PMID: 21072267; PMCID: PMC2971541.
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Torsade de Pointes and Persistent QTc Prolongation after Intravenous Amiodarone.

Case reports in medicine

Kotsia AP, Dimitriadis G, Baltogiannis GG, Kolettis TM.
PMID: 22474460
Case Rep Med. 2012;2012:673019. doi: 10.1155/2012/673019. Epub 2012 Mar 05.

We report a case of torsade de pointes after intravenous amiodarone and concurrent hypokalemia. Despite treatment cessation and correction of electrolyte abnormalities, excessive QTc prolongation was noted, which persisted for 14 days. This prolonged course for QTc normalization may...

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Kotsia AP, Dimitriadis G, Baltogiannis GG, et al. Torsade de Pointes and Persistent QTc Prolongation after Intravenous Amiodarone. Case Rep Med. 2012;2012:673019doi: 10.1155/2012/673019.
Kotsia, A. P., Dimitriadis, G., Baltogiannis, G. G., & Kolettis, T. M. (2012). Torsade de Pointes and Persistent QTc Prolongation after Intravenous Amiodarone. Case reports in medicine, 2012673019. https://doi.org/10.1155/2012/673019
Kotsia, Anna P, et al. "Torsade de Pointes and Persistent QTc Prolongation after Intravenous Amiodarone." Case reports in medicine vol. 2012 (2012): 673019. doi: https://doi.org/10.1155/2012/673019
Kotsia AP, Dimitriadis G, Baltogiannis GG, Kolettis TM. Torsade de Pointes and Persistent QTc Prolongation after Intravenous Amiodarone. Case Rep Med. 2012;2012:673019. doi: 10.1155/2012/673019. Epub 2012 Mar 05. PMID: 22474460; PMCID: PMC3303706.
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Transforming growth factor-β inhibition and endothelin receptor blockade in rats with monocrotaline-induced pulmonary hypertension.

Pulmonary circulation

Megalou AJ, Glava C, Vilaeti AD, Oikonomidis DL, Baltogiannis GG, Papalois A, Vlahos AP, Kolettis TM.
PMID: 23372930
Pulm Circ. 2012 Oct;2(4):461-9. doi: 10.4103/2045-8932.105034.

Transforming growth factor-β (TGF-β) inhibition is an investigational therapy for pulmonary arterial hypertension with promising results in experimental studies. The present work compared this approach with endothelin-receptor blockade and evaluated the effects of combined administration. Pulmonary arterial hypertension was...

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Megalou AJ, Glava C, Vilaeti AD, et al. Transforming growth factor-β inhibition and endothelin receptor blockade in rats with monocrotaline-induced pulmonary hypertension. Pulm Circ. 2012;2(4):461-9doi: 10.4103/2045-8932.105034.
Megalou, A. J., Glava, C., Vilaeti, A. D., Oikonomidis, D. L., Baltogiannis, G. G., Papalois, A., Vlahos, A. P., & Kolettis, T. M. (2012). Transforming growth factor-β inhibition and endothelin receptor blockade in rats with monocrotaline-induced pulmonary hypertension. Pulmonary circulation, 2(4), 461-9. https://doi.org/10.4103/2045-8932.105034
Megalou, Aikaterini J, et al. "Transforming growth factor-β inhibition and endothelin receptor blockade in rats with monocrotaline-induced pulmonary hypertension." Pulmonary circulation vol. 2,4 (2012): 461-9. doi: https://doi.org/10.4103/2045-8932.105034
Megalou AJ, Glava C, Vilaeti AD, Oikonomidis DL, Baltogiannis GG, Papalois A, Vlahos AP, Kolettis TM. Transforming growth factor-β inhibition and endothelin receptor blockade in rats with monocrotaline-induced pulmonary hypertension. Pulm Circ. 2012 Oct;2(4):461-9. doi: 10.4103/2045-8932.105034. PMID: 23372930; PMCID: PMC3555416.
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CPVT: Arrhythmogenesis, Therapeutic Management, and Future Perspectives. A Brief Review of the Literature.

Frontiers in cardiovascular medicine

Baltogiannis GG, Lysitsas DN, di Giovanni G, Ciconte G, Sieira J, Conte G, Kolettis TM, Chierchia GB, de Asmundis C, Brugada P.
PMID: 31380394
Front Cardiovasc Med. 2019 Jul 12;6:92. doi: 10.3389/fcvm.2019.00092. eCollection 2019.

Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT) is a primary electrical disease characterized by a normal resting electrocardiogram and induction of malignant arrhythmias during adrenergic stress leading to syncope or sudden cardiac death (SCD). CPVT is caused by mutations in the...

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Baltogiannis GG, Lysitsas DN, di Giovanni G, et al. CPVT: Arrhythmogenesis, Therapeutic Management, and Future Perspectives. A Brief Review of the Literature. Front Cardiovasc Med. 2019;6:92doi: 10.3389/fcvm.2019.00092.
Baltogiannis, G. G., Lysitsas, D. N., di Giovanni, G., Ciconte, G., Sieira, J., Conte, G., Kolettis, T. M., Chierchia, G. B., de Asmundis, C., & Brugada, P. (2019). CPVT: Arrhythmogenesis, Therapeutic Management, and Future Perspectives. A Brief Review of the Literature. Frontiers in cardiovascular medicine, 692. https://doi.org/10.3389/fcvm.2019.00092
Baltogiannis, Giannis G, et al. "CPVT: Arrhythmogenesis, Therapeutic Management, and Future Perspectives. A Brief Review of the Literature." Frontiers in cardiovascular medicine vol. 6 (2019): 92. doi: https://doi.org/10.3389/fcvm.2019.00092
Baltogiannis GG, Lysitsas DN, di Giovanni G, Ciconte G, Sieira J, Conte G, Kolettis TM, Chierchia GB, de Asmundis C, Brugada P. CPVT: Arrhythmogenesis, Therapeutic Management, and Future Perspectives. A Brief Review of the Literature. Front Cardiovasc Med. 2019 Jul 12;6:92. doi: 10.3389/fcvm.2019.00092. eCollection 2019. PMID: 31380394; PMCID: PMC6644488.
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Sympathetic Activation and Arrhythmogenesis after Myocardial Infarction: Where Do We Stand?.

Journal of cardiovascular development and disease

Zekios KC, Mouchtouri ET, Lekkas P, Nikas DN, Kolettis TM.
PMID: 34063477
J Cardiovasc Dev Dis. 2021 May 15;8(5). doi: 10.3390/jcdd8050057.

Myocardial infarction often leads to progressive structural and electrophysiologic remodeling of the left ventricle. Despite the widespread use of β-adrenergic blockade and implantable defibrillators, morbidity and mortality from chronic-phase ventricular tachyarrhythmias remains high, calling for further investigation on the...

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Zekios KC, Mouchtouri ET, Lekkas P, et al. Sympathetic Activation and Arrhythmogenesis after Myocardial Infarction: Where Do We Stand?. J Cardiovasc Dev Dis. 2021;8(5)doi: 10.3390/jcdd8050057.
Zekios, K. C., Mouchtouri, E. T., Lekkas, P., Nikas, D. N., & Kolettis, T. M. (2021). Sympathetic Activation and Arrhythmogenesis after Myocardial Infarction: Where Do We Stand?. Journal of cardiovascular development and disease, 8(5), . https://doi.org/10.3390/jcdd8050057
Zekios, Konstantinos C, et al. "Sympathetic Activation and Arrhythmogenesis after Myocardial Infarction: Where Do We Stand?." Journal of cardiovascular development and disease vol. 8,5 (2021). doi: https://doi.org/10.3390/jcdd8050057
Zekios KC, Mouchtouri ET, Lekkas P, Nikas DN, Kolettis TM. Sympathetic Activation and Arrhythmogenesis after Myocardial Infarction: Where Do We Stand?. J Cardiovasc Dev Dis. 2021 May 15;8(5). doi: 10.3390/jcdd8050057. PMID: 34063477; PMCID: PMC8156099.
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Endothelin-B Receptors and Left Ventricular Dysfunction after Regional versus Global Ischaemia-Reperfusion in Rat Hearts.

Cardiology research and practice

Bibli SI, Toli EV, Vilaeti AD, Varnavas VC, Baltogiannis GG, Papalois A, Kyriakides ZS, Kolettis TM.
PMID: 22844633
Cardiol Res Pract. 2012;2012:986813. doi: 10.1155/2012/986813. Epub 2012 Jul 12.

Background. Endothelin-1 (ET-1) is implicated in left ventricular dysfunction after ischaemia-reperfusion. ETA and ETB receptors mediate diverse actions, but it is unknown whether these actions depend on ischaemia type and duration. We investigated the role of ETB receptors after...

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Bibli SI, Toli EV, Vilaeti AD, et al. Endothelin-B Receptors and Left Ventricular Dysfunction after Regional versus Global Ischaemia-Reperfusion in Rat Hearts. Cardiol Res Pract. 2012;2012:986813doi: 10.1155/2012/986813.
Bibli, S. I., Toli, E. V., Vilaeti, A. D., Varnavas, V. C., Baltogiannis, G. G., Papalois, A., Kyriakides, Z. S., & Kolettis, T. M. (2012). Endothelin-B Receptors and Left Ventricular Dysfunction after Regional versus Global Ischaemia-Reperfusion in Rat Hearts. Cardiology research and practice, 2012986813. https://doi.org/10.1155/2012/986813
Bibli, Sofia-Iris, et al. "Endothelin-B Receptors and Left Ventricular Dysfunction after Regional versus Global Ischaemia-Reperfusion in Rat Hearts." Cardiology research and practice vol. 2012 (2012): 986813. doi: https://doi.org/10.1155/2012/986813
Bibli SI, Toli EV, Vilaeti AD, Varnavas VC, Baltogiannis GG, Papalois A, Kyriakides ZS, Kolettis TM. Endothelin-B Receptors and Left Ventricular Dysfunction after Regional versus Global Ischaemia-Reperfusion in Rat Hearts. Cardiol Res Pract. 2012;2012:986813. doi: 10.1155/2012/986813. Epub 2012 Jul 12. PMID: 22844633; PMCID: PMC3403336.
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Subclinical Hypothyroidism: An Overlooked Cause of Atrial Fibrillation?.

Journal of atrial fibrillation

Kolettis TM, Tsatsoulis A.
PMID: 28496796
J Atr Fibrillation. 2012 Dec 16;5(4):710. doi: 10.4022/jafib.710. eCollection 2012 Dec.

No abstract available.

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Kolettis TM, Tsatsoulis A. Subclinical Hypothyroidism: An Overlooked Cause of Atrial Fibrillation?. J Atr Fibrillation. 2012;5(4):710doi: 10.4022/jafib.710.
Kolettis, T. M., & Tsatsoulis, A. (2012). Subclinical Hypothyroidism: An Overlooked Cause of Atrial Fibrillation?. Journal of atrial fibrillation, 5(4), 710. https://doi.org/10.4022/jafib.710
Kolettis, Theofilos M, and Tsatsoulis, Agathocles. "Subclinical Hypothyroidism: An Overlooked Cause of Atrial Fibrillation?." Journal of atrial fibrillation vol. 5,4 (2012): 710. doi: https://doi.org/10.4022/jafib.710
Kolettis TM, Tsatsoulis A. Subclinical Hypothyroidism: An Overlooked Cause of Atrial Fibrillation?. J Atr Fibrillation. 2012 Dec 16;5(4):710. doi: 10.4022/jafib.710. eCollection 2012 Dec. PMID: 28496796; PMCID: PMC5153160.
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Showing 13 to 24 of 25 entries