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Microstructural Evolution in 2101 Lean Duplex Stainless Steel During Low- and Intermediate-Temperature Aging.

Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada

Maetz JY, Cazottes S, Verdu C, Danoix F, Kléber X.
PMID: 26940550
Microsc Microanal. 2016 Apr;22(2):463-73. doi: 10.1017/S1431927616000167. Epub 2016 Mar 04.

The microstructural evolution of a 2101 lean duplex stainless steel (DSS) during isothermal aging from room temperature to 470 °C was investigated using thermoelectric power (TEP) measurements to follow the kinetics, atom probe tomography, and transmission electron microscopy. Despite...

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Maetz JY, Cazottes S, Verdu C, et al. Microstructural Evolution in 2101 Lean Duplex Stainless Steel During Low- and Intermediate-Temperature Aging. Microsc Microanal. 2016;22(2):463-73doi: 10.1017/S1431927616000167.
Maetz, J. Y., Cazottes, S., Verdu, C., Danoix, F., & Kléber, X. (2016). Microstructural Evolution in 2101 Lean Duplex Stainless Steel During Low- and Intermediate-Temperature Aging. Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada, 22(2), 463-73. https://doi.org/10.1017/S1431927616000167
Maetz, Jean-Yves, et al. "Microstructural Evolution in 2101 Lean Duplex Stainless Steel During Low- and Intermediate-Temperature Aging." Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada vol. 22,2 (2016): 463-73. doi: https://doi.org/10.1017/S1431927616000167
Maetz JY, Cazottes S, Verdu C, Danoix F, Kléber X. Microstructural Evolution in 2101 Lean Duplex Stainless Steel During Low- and Intermediate-Temperature Aging. Microsc Microanal. 2016 Apr;22(2):463-73. doi: 10.1017/S1431927616000167. Epub 2016 Mar 04. PMID: 26940550.
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M23C6 carbides and Cr2N nitrides in aged duplex stainless steel: A SEM, TEM and FIB tomography investigation.

Micron (Oxford, England : 1993)

Maetz JY, Douillard T, Cazottes S, Verdu C, Kléber X.
PMID: 26925831
Micron. 2016 May;84:43-53. doi: 10.1016/j.micron.2016.01.007. Epub 2016 Feb 06.

The precipitation evolution during ageing of a 2101 lean duplex stainless steel was investigated, revealing that the precipitate type and morphology depends on the nature of the grain boundary. Triangular M23C6 carbides precipitate only at γ/δ interfaces and rod-like...

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Maetz JY, Douillard T, Cazottes S, et al. M23C6 carbides and Cr2N nitrides in aged duplex stainless steel: A SEM, TEM and FIB tomography investigation. Micron. 2016;84:43-53doi: 10.1016/j.micron.2016.01.007.
Maetz, J. Y., Douillard, T., Cazottes, S., Verdu, C., & Kléber, X. (2016). M23C6 carbides and Cr2N nitrides in aged duplex stainless steel: A SEM, TEM and FIB tomography investigation. Micron (Oxford, England : 1993), 8443-53. https://doi.org/10.1016/j.micron.2016.01.007
Maetz, J-Y, et al. "M23C6 carbides and Cr2N nitrides in aged duplex stainless steel: A SEM, TEM and FIB tomography investigation." Micron (Oxford, England : 1993) vol. 84 (2016): 43-53. doi: https://doi.org/10.1016/j.micron.2016.01.007
Maetz JY, Douillard T, Cazottes S, Verdu C, Kléber X. M23C6 carbides and Cr2N nitrides in aged duplex stainless steel: A SEM, TEM and FIB tomography investigation. Micron. 2016 May;84:43-53. doi: 10.1016/j.micron.2016.01.007. Epub 2016 Feb 06. PMID: 26925831.
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Arrhythmogenic Cardiomyopathy - New Insights into Disease Mechanisms and Drug Discovery.

Progress in pediatric cardiology

Asimaki A, Kléber AG, MacRae CA, Saffitz JE.
PMID: 25506190
Prog Pediatr Cardiol. 2014 Dec 01;37(1):3-7. doi: 10.1016/j.ppedcard.2014.10.001.

Arrhythmogenic cardiomyopathy (ACM) is a primary myocardial disorder characterized by the early appearance of ventricular arrhythmias often out of proportion to the degree of ventricular remodeling and dysfunction. ACM typically presents in adolescence or early adulthood. It accounts for...

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Asimaki A, Kléber AG, MacRae CA, et al. Arrhythmogenic Cardiomyopathy - New Insights into Disease Mechanisms and Drug Discovery. Prog Pediatr Cardiol. 2014;37(1):3-7doi: 10.1016/j.ppedcard.2014.10.001.
Asimaki, A., Kléber, A. G., MacRae, C. A., & Saffitz, J. E. (2014). Arrhythmogenic Cardiomyopathy - New Insights into Disease Mechanisms and Drug Discovery. Progress in pediatric cardiology, 37(1), 3-7. https://doi.org/10.1016/j.ppedcard.2014.10.001
Asimaki, Angeliki, et al. "Arrhythmogenic Cardiomyopathy - New Insights into Disease Mechanisms and Drug Discovery." Progress in pediatric cardiology vol. 37,1 (2014): 3-7. doi: https://doi.org/10.1016/j.ppedcard.2014.10.001
Asimaki A, Kléber AG, MacRae CA, Saffitz JE. Arrhythmogenic Cardiomyopathy - New Insights into Disease Mechanisms and Drug Discovery. Prog Pediatr Cardiol. 2014 Dec 01;37(1):3-7. doi: 10.1016/j.ppedcard.2014.10.001. PMID: 25506190; PMCID: PMC4260412.
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Role of the intercalated disc in cardiac propagation and arrhythmogenesis.

Frontiers in physiology

Kleber AG, Saffitz JE.
PMID: 25368581
Front Physiol. 2014 Oct 17;5:404. doi: 10.3389/fphys.2014.00404. eCollection 2014.

This review article discusses mechanisms underlying impulse propagation in cardiac muscle with specific emphasis on the role of the cardiac cell-to-cell junction, called the "intercalated disc."The first part of this review deals with the role of gap junction channels,...

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Kleber AG, Saffitz JE. Role of the intercalated disc in cardiac propagation and arrhythmogenesis. Front Physiol. 2014;5:404doi: 10.3389/fphys.2014.00404.
Kleber, A. G., & Saffitz, J. E. (2014). Role of the intercalated disc in cardiac propagation and arrhythmogenesis. Frontiers in physiology, 5404. https://doi.org/10.3389/fphys.2014.00404
Kleber, Andre G, and Saffitz, Jeffrey E. "Role of the intercalated disc in cardiac propagation and arrhythmogenesis." Frontiers in physiology vol. 5 (2014): 404. doi: https://doi.org/10.3389/fphys.2014.00404
Kleber AG, Saffitz JE. Role of the intercalated disc in cardiac propagation and arrhythmogenesis. Front Physiol. 2014 Oct 17;5:404. doi: 10.3389/fphys.2014.00404. eCollection 2014. PMID: 25368581; PMCID: PMC4201087.
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The works of Dirk Durrer (1918-1984).

Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation

Opthof T, Janse MJ, Kléber AG, Wellens HJ, Wilde AA, Coronel R.
PMID: 22875727
Neth Heart J. 2012 Oct;20(10):430-3. doi: 10.1007/s12471-012-0313-7.

No abstract available.

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Opthof T, Janse MJ, Kléber AG, et al. The works of Dirk Durrer (1918-1984). Neth Heart J. 2012;20(10):430-3doi: 10.1007/s12471-012-0313-7.
Opthof, T., Janse, M. J., Kléber, A. G., Wellens, H. J., Wilde, A. A., & Coronel, R. (2012). The works of Dirk Durrer (1918-1984). Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation, 20(10), 430-3. https://doi.org/10.1007/s12471-012-0313-7
Opthof, T, et al. "The works of Dirk Durrer (1918-1984)." Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation vol. 20,10 (2012): 430-3. doi: https://doi.org/10.1007/s12471-012-0313-7
Opthof T, Janse MJ, Kléber AG, Wellens HJ, Wilde AA, Coronel R. The works of Dirk Durrer (1918-1984). Neth Heart J. 2012 Oct;20(10):430-3. doi: 10.1007/s12471-012-0313-7. PMID: 22875727; PMCID: PMC3439538.
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Lentiviral transduction of neonatal rat ventricular myocytes preserves ultrastructural features of genetically modified cells.

Virology

Consonni SR, de Carvalho ACPV, Mauro AB, Franchini KG, Bajgelman MC.
PMID: 34365094
Virology. 2021 Oct;562:190-196. doi: 10.1016/j.virol.2021.07.015. Epub 2021 Jul 31.

Preserving morphological features that are important for cell function and structure is a critical parameter for in vitro experiments with rat cardiomyocytes. Lentiviral vectors are commonly used as gene transfer tool because of its high flexibility, efficiency to deliver...

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Consonni SR, de Carvalho ACPV, Mauro AB, et al. Lentiviral transduction of neonatal rat ventricular myocytes preserves ultrastructural features of genetically modified cells. Virology. 2021;562:190-196doi: 10.1016/j.virol.2021.07.015.
Consonni, S. R., de Carvalho, A. C. P. V., Mauro, A. B., Franchini, K. G., & Bajgelman, M. C. (2021). Lentiviral transduction of neonatal rat ventricular myocytes preserves ultrastructural features of genetically modified cells. Virology, 562190-196. https://doi.org/10.1016/j.virol.2021.07.015
Consonni, Sílvio R, et al. "Lentiviral transduction of neonatal rat ventricular myocytes preserves ultrastructural features of genetically modified cells." Virology vol. 562 (2021): 190-196. doi: https://doi.org/10.1016/j.virol.2021.07.015
Consonni SR, de Carvalho ACPV, Mauro AB, Franchini KG, Bajgelman MC. Lentiviral transduction of neonatal rat ventricular myocytes preserves ultrastructural features of genetically modified cells. Virology. 2021 Oct;562:190-196. doi: 10.1016/j.virol.2021.07.015. Epub 2021 Jul 31. PMID: 34365094.
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Central role for GSK3β in the pathogenesis of arrhythmogenic cardiomyopathy.

JCI insight

Chelko SP, Asimaki A, Andersen P, Bedja D, Amat-Alarcon N, DeMazumder D, Jasti R, MacRae CA, Leber R, Kleber AG, Saffitz JE, Judge DP.
PMID: 27170944
JCI Insight. 2016 Apr 21;1(5). doi: 10.1172/jci.insight.85923.

Arrhythmogenic cardiomyopathy (ACM) is characterized by redistribution of junctional proteins, arrhythmias, and progressive myocardial injury. We previously reported that SB216763 (SB2), annotated as a GSK3β inhibitor, reverses disease phenotypes in a zebrafish model of ACM. Here, we show that...

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Chelko SP, Asimaki A, Andersen P, et al. Central role for GSK3β in the pathogenesis of arrhythmogenic cardiomyopathy. JCI Insight. 2016;1(5)doi: 10.1172/jci.insight.85923.
Chelko, S. P., Asimaki, A., Andersen, P., Bedja, D., Amat-Alarcon, N., DeMazumder, D., Jasti, R., MacRae, C. A., Leber, R., Kleber, A. G., Saffitz, J. E., & Judge, D. P. (2016). Central role for GSK3β in the pathogenesis of arrhythmogenic cardiomyopathy. JCI insight, 1(5), . https://doi.org/10.1172/jci.insight.85923
Chelko, Stephen P, et al. "Central role for GSK3β in the pathogenesis of arrhythmogenic cardiomyopathy." JCI insight vol. 1,5 (2016). doi: https://doi.org/10.1172/jci.insight.85923
Chelko SP, Asimaki A, Andersen P, Bedja D, Amat-Alarcon N, DeMazumder D, Jasti R, MacRae CA, Leber R, Kleber AG, Saffitz JE, Judge DP. Central role for GSK3β in the pathogenesis of arrhythmogenic cardiomyopathy. JCI Insight. 2016 Apr 21;1(5). doi: 10.1172/jci.insight.85923. PMID: 27170944; PMCID: PMC4861310.
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ESTs from Seeds to Assist the Selective Breeding of Jatropha curcas L. for Oil and Active Compounds.

Genomics insights

Gomes KA, Almeida TC, Gesteira AS, Lôbo IP, Guimarães AC, de Miranda AB, Van Sluys MA, da Cruz RS, Cascardo JC, Carels N.
PMID: 26217103
Genomics Insights. 2010 Oct 28;3:29-56. doi: 10.4137/GEI.S4340. eCollection 2010.

We report here on the characterization of a cDNA library from seeds of Jatropha curcas L. at three stages of fruit maturation before yellowing. We sequenced a total of 2200 clones and obtained a set of 931 non-redundant sequences...

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Gomes KA, Almeida TC, Gesteira AS, et al. ESTs from Seeds to Assist the Selective Breeding of Jatropha curcas L. for Oil and Active Compounds. Genomics Insights. 2010;3:29-56doi: 10.4137/GEI.S4340.
Gomes, K. A., Almeida, T. C., Gesteira, A. S., Lôbo, I. P., Guimarães, A. C., de Miranda, A. B., Van Sluys, M. A., da Cruz, R. S., Cascardo, J. C., & Carels, N. (2010). ESTs from Seeds to Assist the Selective Breeding of Jatropha curcas L. for Oil and Active Compounds. Genomics insights, 329-56. https://doi.org/10.4137/GEI.S4340
Gomes, Kleber A, et al. "ESTs from Seeds to Assist the Selective Breeding of Jatropha curcas L. for Oil and Active Compounds." Genomics insights vol. 3 (2010): 29-56. doi: https://doi.org/10.4137/GEI.S4340
Gomes KA, Almeida TC, Gesteira AS, Lôbo IP, Guimarães AC, de Miranda AB, Van Sluys MA, da Cruz RS, Cascardo JC, Carels N. ESTs from Seeds to Assist the Selective Breeding of Jatropha curcas L. for Oil and Active Compounds. Genomics Insights. 2010 Oct 28;3:29-56. doi: 10.4137/GEI.S4340. eCollection 2010. PMID: 26217103; PMCID: PMC4510598.
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Statement of Retraction. EGFR Tyrosine Kinase Inhibitor (PD153035) Improves Glucose Tolerance and Insulin Action in High-Fat Diet-Fed Mice. Diabetes 2009;58:2910-2919. DOI: 10.2337/db08-0506. PMID: 19696185.

Diabetes

Prada PO, Ropelle ER, Mourão RH, de Souza CT, Pauli JR, Cintra DE, Schenka A, Rocco SA, Rittner R, Franchini KG, Vassallo J, Velloso LA, Carvalheira JB, Saad MJA.
PMID: 28450418
Diabetes. 2017 Jul;66(7):2059. doi: 10.2337/db17-rt07a. Epub 2017 Apr 27.

No abstract available.

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Prada PO, Ropelle ER, Mourão RH, et al. Statement of Retraction. EGFR Tyrosine Kinase Inhibitor (PD153035) Improves Glucose Tolerance and Insulin Action in High-Fat Diet-Fed Mice. Diabetes 2009;58:2910-2919. DOI: 10.2337/db08-0506. PMID: 19696185. Diabetes. 2017;66(7):2059doi: 10.2337/db17-rt07a.
Prada, P. O., Ropelle, E. R., Mourão, R. H., de Souza, C. T., Pauli, J. R., Cintra, D. E., Schenka, A., Rocco, S. A., Rittner, R., Franchini, K. G., Vassallo, J., Velloso, L. A., Carvalheira, J. B., & Saad, M. J. A. (2017). Statement of Retraction. EGFR Tyrosine Kinase Inhibitor (PD153035) Improves Glucose Tolerance and Insulin Action in High-Fat Diet-Fed Mice. Diabetes 2009;58:2910-2919. DOI: 10.2337/db08-0506. PMID: 19696185. Diabetes, 66(7), 2059. https://doi.org/10.2337/db17-rt07a
Prada, Patricia O, et al. "Statement of Retraction. EGFR Tyrosine Kinase Inhibitor (PD153035) Improves Glucose Tolerance and Insulin Action in High-Fat Diet-Fed Mice. Diabetes 2009;58:2910-2919. DOI: 10.2337/db08-0506. PMID: 19696185." Diabetes vol. 66,7 (2017): 2059. doi: https://doi.org/10.2337/db17-rt07a
Prada PO, Ropelle ER, Mourão RH, de Souza CT, Pauli JR, Cintra DE, Schenka A, Rocco SA, Rittner R, Franchini KG, Vassallo J, Velloso LA, Carvalheira JB, Saad MJA. Statement of Retraction. EGFR Tyrosine Kinase Inhibitor (PD153035) Improves Glucose Tolerance and Insulin Action in High-Fat Diet-Fed Mice. Diabetes 2009;58:2910-2919. DOI: 10.2337/db08-0506. PMID: 19696185. Diabetes. 2017 Jul;66(7):2059. doi: 10.2337/db17-rt07a. Epub 2017 Apr 27. PMID: 28450418; PMCID: PMC5482084.
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Concordance of KRAS/BRAF Mutation Status in Metastatic Colorectal Cancer before and after Anti-EGFR Therapy.

Journal of oncology

Gattenlöhner S, Etschmann B, Kunzmann V, Thalheimer A, Hack M, Kleber G, Einsele H, Germer C, Müller-Hermelink HK.
PMID: 20300583
J Oncol. 2009;2009:831626. doi: 10.1155/2009/831626. Epub 2010 Mar 10.

Anti-EGFR targeted therapy is a potent strategy in the treatment of metastatic colorectal cancer (mCRC) but activating mutations in the KRAS gene are associated with poor response to this treatment. Therefore, KRAS mutation analysis is employed in the selection...

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Gattenlöhner S, Etschmann B, Kunzmann V, et al. Concordance of KRAS/BRAF Mutation Status in Metastatic Colorectal Cancer before and after Anti-EGFR Therapy. J Oncol. 2010;2009:831626doi: 10.1155/2009/831626.
Gattenlöhner, S., Etschmann, B., Kunzmann, V., Thalheimer, A., Hack, M., Kleber, G., Einsele, H., Germer, C., & Müller-Hermelink, H. K. (2009). Concordance of KRAS/BRAF Mutation Status in Metastatic Colorectal Cancer before and after Anti-EGFR Therapy. Journal of oncology, 2009831626. https://doi.org/10.1155/2009/831626
Gattenlöhner, S, et al. "Concordance of KRAS/BRAF Mutation Status in Metastatic Colorectal Cancer before and after Anti-EGFR Therapy." Journal of oncology vol. 2009 (2009): 831626. doi: https://doi.org/10.1155/2009/831626
Gattenlöhner S, Etschmann B, Kunzmann V, Thalheimer A, Hack M, Kleber G, Einsele H, Germer C, Müller-Hermelink HK. Concordance of KRAS/BRAF Mutation Status in Metastatic Colorectal Cancer before and after Anti-EGFR Therapy. J Oncol. 2009;2009:831626. doi: 10.1155/2009/831626. Epub 2010 Mar 10. PMID: 20300583; PMCID: PMC2837901.
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An atypical presentation of gigantiform torus palatinus: A case report: Atypical tori palatine and surgical management.

International journal of surgery case reports

Bernaola-Paredes WE, Pereira AM, Albuquerque Luiz TA, Martins IS, Lima FF, Vallejo-Rosero KA.
PMID: 32919331
Int J Surg Case Rep. 2020;75:66-70. doi: 10.1016/j.ijscr.2020.08.049. Epub 2020 Sep 04.

INTRODUCTION: Torus palatinus (TP) is a slow growing maxillary exostosis, commonly found in the in the hard palate on either side of the median raphe of the palatine bone. Its aetiology is still unclear, but it has been attributed...

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Bernaola-Paredes WE, Pereira AM, Albuquerque Luiz TA, et al. An atypical presentation of gigantiform torus palatinus: A case report: Atypical tori palatine and surgical management. Int J Surg Case Rep. 2020;75:66-70doi: 10.1016/j.ijscr.2020.08.049.
Bernaola-Paredes, W. E., Pereira, A. M., Albuquerque Luiz, T. A., Martins, I. S., Lima, F. F., & Vallejo-Rosero, K. A. (2020). An atypical presentation of gigantiform torus palatinus: A case report: Atypical tori palatine and surgical management. International journal of surgery case reports, 7566-70. https://doi.org/10.1016/j.ijscr.2020.08.049
Bernaola-Paredes, Wilber E, et al. "An atypical presentation of gigantiform torus palatinus: A case report: Atypical tori palatine and surgical management." International journal of surgery case reports vol. 75 (2020): 66-70. doi: https://doi.org/10.1016/j.ijscr.2020.08.049
Bernaola-Paredes WE, Pereira AM, Albuquerque Luiz TA, Martins IS, Lima FF, Vallejo-Rosero KA. An atypical presentation of gigantiform torus palatinus: A case report: Atypical tori palatine and surgical management. Int J Surg Case Rep. 2020;75:66-70. doi: 10.1016/j.ijscr.2020.08.049. Epub 2020 Sep 04. PMID: 32919331; PMCID: PMC7490980.
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Assessing police topological efficiency in a major sting operation on the dark web.

Scientific reports

da Cunha BR, MacCarron P, Passold JF, Dos Santos LW, Oliveira KA, Gleeson JP.
PMID: 31919365
Sci Rep. 2020 Jan 09;10(1):73. doi: 10.1038/s41598-019-56704-4.

The networked nature of criminals using the dark web is poorly understood and infrequently studied, mostly due to a lack of data. Rarer still are studies on the topological effectiveness of police interventions. Between 2014 and 2016, the Brazilian...

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da Cunha BR, MacCarron P, Passold JF, et al. Assessing police topological efficiency in a major sting operation on the dark web. Sci Rep. 2020;10(1):73doi: 10.1038/s41598-019-56704-4.
da Cunha, B. R., MacCarron, P., Passold, J. F., Dos Santos, L. W., Oliveira, K. A., & Gleeson, J. P. (2020). Assessing police topological efficiency in a major sting operation on the dark web. Scientific reports, 10(1), 73. https://doi.org/10.1038/s41598-019-56704-4
da Cunha, Bruno Requião, et al. "Assessing police topological efficiency in a major sting operation on the dark web." Scientific reports vol. 10,1 (2020): 73. doi: https://doi.org/10.1038/s41598-019-56704-4
da Cunha BR, MacCarron P, Passold JF, Dos Santos LW, Oliveira KA, Gleeson JP. Assessing police topological efficiency in a major sting operation on the dark web. Sci Rep. 2020 Jan 09;10(1):73. doi: 10.1038/s41598-019-56704-4. PMID: 31919365; PMCID: PMC6952354.
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Showing 13 to 24 of 28 entries