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Molecular imaging: a view from the inside.

Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology

Herschman HR.
PMID: 15052251
J Nucl Cardiol. 2004 Mar-Apr;11(2):210-4. doi: 10.1016/j.nuclcard.2004.01.002.

No abstract available.

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Herschman HR. Molecular imaging: a view from the inside. J Nucl Cardiol. 2004;11(2):210-4doi: 10.1016/j.nuclcard.2004.01.002.
Herschman, H. R. (2004). Molecular imaging: a view from the inside. Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology, 11(2), 210-4. https://doi.org/10.1016/j.nuclcard.2004.01.002
Herschman, Harvey R. "Molecular imaging: a view from the inside." Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology vol. 11,2 (2004): 210-4. doi: https://doi.org/10.1016/j.nuclcard.2004.01.002
Herschman HR. Molecular imaging: a view from the inside. J Nucl Cardiol. 2004 Mar-Apr;11(2):210-4. doi: 10.1016/j.nuclcard.2004.01.002. PMID: 15052251.
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Pseudomonas aeruginosa quorum-sensing systems may control virulence factor expression in the lungs of patients with cystic fibrosis.

Infection and immunity

Erickson DL, Endersby R, Kirkham A, Stuber K, Vollman DD, Rabin HR, Mitchell I, Storey DG.
PMID: 11895939
Infect Immun. 2002 Apr;70(4):1783-90. doi: 10.1128/IAI.70.4.1783-1790.2002.

Individuals with cystic fibrosis (CF) are commonly colonized with Pseudomonas aeruginosa. The chronic infections caused by P. aeruginosa are punctuated by acute exacerbations of the lung disease, which lead to significant morbidity and mortality. As regulators of virulence determinants,...

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Erickson DL, Endersby R, Kirkham A, et al. Pseudomonas aeruginosa quorum-sensing systems may control virulence factor expression in the lungs of patients with cystic fibrosis. Infect Immun. 2002;70(4):1783-90doi: 10.1128/IAI.70.4.1783-1790.2002.
Erickson, D. L., Endersby, R., Kirkham, A., Stuber, K., Vollman, D. D., Rabin, H. R., Mitchell, I., & Storey, D. G. (2002). Pseudomonas aeruginosa quorum-sensing systems may control virulence factor expression in the lungs of patients with cystic fibrosis. Infection and immunity, 70(4), 1783-90. https://doi.org/10.1128/IAI.70.4.1783-1790.2002
Erickson, David L, et al. "Pseudomonas aeruginosa quorum-sensing systems may control virulence factor expression in the lungs of patients with cystic fibrosis." Infection and immunity vol. 70,4 (2002): 1783-90. doi: https://doi.org/10.1128/IAI.70.4.1783-1790.2002
Erickson DL, Endersby R, Kirkham A, Stuber K, Vollman DD, Rabin HR, Mitchell I, Storey DG. Pseudomonas aeruginosa quorum-sensing systems may control virulence factor expression in the lungs of patients with cystic fibrosis. Infect Immun. 2002 Apr;70(4):1783-90. doi: 10.1128/IAI.70.4.1783-1790.2002. PMID: 11895939; PMCID: PMC127834.
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Perceptual and acoustic evaluation of individuals with laryngopharyngeal reflux pre- and post-treatment.

Journal of voice : official journal of the Voice Foundation

Selby JC, Gilbert HR, Lerman JW.
PMID: 14740936
J Voice. 2003 Dec;17(4):557-70. doi: 10.1067/s0892-1997(03)00017-1.

Thirteen individuals with laryngopharyngeal reflux (LPR) were studied pre- and post-treatment. The effect of treatment on perceptual ratings of voice quality and frequency and intensity measures was examined. Relationships between perceptual and acoustic parameters were assessed descriptively. Results showed...

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Selby JC, Gilbert HR, Lerman JW. Perceptual and acoustic evaluation of individuals with laryngopharyngeal reflux pre- and post-treatment. J Voice. 2003;17(4):557-70doi: 10.1067/s0892-1997(03)00017-1.
Selby, J. C., Gilbert, H. R., & Lerman, J. W. (2003). Perceptual and acoustic evaluation of individuals with laryngopharyngeal reflux pre- and post-treatment. Journal of voice : official journal of the Voice Foundation, 17(4), 557-70. https://doi.org/10.1067/s0892-1997(03)00017-1
Selby, Julia C, et al. "Perceptual and acoustic evaluation of individuals with laryngopharyngeal reflux pre- and post-treatment." Journal of voice : official journal of the Voice Foundation vol. 17,4 (2003): 557-70. doi: https://doi.org/10.1067/s0892-1997(03)00017-1
Selby JC, Gilbert HR, Lerman JW. Perceptual and acoustic evaluation of individuals with laryngopharyngeal reflux pre- and post-treatment. J Voice. 2003 Dec;17(4):557-70. doi: 10.1067/s0892-1997(03)00017-1. PMID: 14740936.
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Development of novel benzimidazole riboside compounds for treatment of cytomegalovirus disease.

Advances in experimental medicine and biology

Chulay J, Biron K, Wang L, Underwood M, Chamberlain S, Frick L, Good S, Davis M, Harvey R, Townsend L, Drach J, Koszalka G.
PMID: 10549385
Adv Exp Med Biol. 1999;458:129-34. doi: 10.1007/978-1-4615-4743-3_12.

Benzimidazole ribosides are a new class of compounds with novel mechanisms of action against CMV. One compound in this series, BDCRB, inhibits CMV DNA processing by the UL89 gene product (putative terminase), but rapid metabolism to an inactive compound...

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Chulay J, Biron K, Wang L, et al. Development of novel benzimidazole riboside compounds for treatment of cytomegalovirus disease. Adv Exp Med Biol. 1999;458:129-34doi: 10.1007/978-1-4615-4743-3_12.
Chulay, J., Biron, K., Wang, L., Underwood, M., Chamberlain, S., Frick, L., Good, S., Davis, M., Harvey, R., Townsend, L., Drach, J., & Koszalka, G. (1999). Development of novel benzimidazole riboside compounds for treatment of cytomegalovirus disease. Advances in experimental medicine and biology, 458129-34. https://doi.org/10.1007/978-1-4615-4743-3_12
Chulay, J, et al. "Development of novel benzimidazole riboside compounds for treatment of cytomegalovirus disease." Advances in experimental medicine and biology vol. 458 (1999): 129-34. doi: https://doi.org/10.1007/978-1-4615-4743-3_12
Chulay J, Biron K, Wang L, Underwood M, Chamberlain S, Frick L, Good S, Davis M, Harvey R, Townsend L, Drach J, Koszalka G. Development of novel benzimidazole riboside compounds for treatment of cytomegalovirus disease. Adv Exp Med Biol. 1999;458:129-34. doi: 10.1007/978-1-4615-4743-3_12. PMID: 10549385.
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Treatments for Alzheimer's disease.

The Practitioner

Harvey R, Rossor M.
PMID: 7659667
Practitioner. 1995 Jul;239(1552):440-3.

No abstract available.

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Harvey R, Rossor M. Treatments for Alzheimer's disease. Practitioner. 1995;239(1552):440-3
Harvey, R., & Rossor, M. (1995). Treatments for Alzheimer's disease. The Practitioner, 239(1552), 440-3.
Harvey, R, and Rossor, M. "Treatments for Alzheimer's disease." The Practitioner vol. 239,1552 (1995): 440-3.
Harvey R, Rossor M. Treatments for Alzheimer's disease. Practitioner. 1995 Jul;239(1552):440-3. PMID: 7659667.
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A high prevalence of glucokinase mutations in gestational diabetic subjects selected by clinical criteria.

Diabetologia

Ellard S, Beards F, Allen LI, Shepherd M, Ballantyne E, Harvey R, Hattersley AT.
PMID: 10753050
Diabetologia. 2000 Feb;43(2):250-3. doi: 10.1007/s001250050038.

AIMS/HYPOTHESIS: Patients with glucokinase mutations are characterised by mild, persistent fasting hyperglycaemia, a small increment in glucose in response to an oral load and a dominant family history. These patients frequently present with gestational diabetes and often require insulin...

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Ellard S, Beards F, Allen LI, et al. A high prevalence of glucokinase mutations in gestational diabetic subjects selected by clinical criteria. Diabetologia. 2000;43(2):250-3doi: 10.1007/s001250050038.
Ellard, S., Beards, F., Allen, L. I., Shepherd, M., Ballantyne, E., Harvey, R., & Hattersley, A. T. (2000). A high prevalence of glucokinase mutations in gestational diabetic subjects selected by clinical criteria. Diabetologia, 43(2), 250-3. https://doi.org/10.1007/s001250050038
Ellard, S, et al. "A high prevalence of glucokinase mutations in gestational diabetic subjects selected by clinical criteria." Diabetologia vol. 43,2 (2000): 250-3. doi: https://doi.org/10.1007/s001250050038
Ellard S, Beards F, Allen LI, Shepherd M, Ballantyne E, Harvey R, Hattersley AT. A high prevalence of glucokinase mutations in gestational diabetic subjects selected by clinical criteria. Diabetologia. 2000 Feb;43(2):250-3. doi: 10.1007/s001250050038. PMID: 10753050.
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17Beta-estradiol blocks NMDA-induced increases in regional cerebral O(2) consumption.

Brain research

Weiss HR, Doshi D, Sinha AK, Liu X, Chi OZ.
PMID: 12270495
Brain Res. 2002 Oct 04;951(2):177-82. doi: 10.1016/s0006-8993(02)03158-x.

We tested the hypothesis that 17beta-estradiol would reduce the cerebral O(2) consumption response to stimulation of N-methyl-D-aspartate (NMDA) receptors. We determined NMDA receptor density in 10 ovariectomized Wistar female rats equally divided into a control group and 17beta-estradiol (500...

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Weiss HR, Doshi D, Sinha AK, et al. 17Beta-estradiol blocks NMDA-induced increases in regional cerebral O(2) consumption. Brain Res. 2002;951(2):177-82doi: 10.1016/s0006-8993(02)03158-x.
Weiss, H. R., Doshi, D., Sinha, A. K., Liu, X., & Chi, O. Z. (2002). 17Beta-estradiol blocks NMDA-induced increases in regional cerebral O(2) consumption. Brain research, 951(2), 177-82. https://doi.org/10.1016/s0006-8993(02)03158-x
Weiss, Harvey R, et al. "17Beta-estradiol blocks NMDA-induced increases in regional cerebral O(2) consumption." Brain research vol. 951,2 (2002): 177-82. doi: https://doi.org/10.1016/s0006-8993(02)03158-x
Weiss HR, Doshi D, Sinha AK, Liu X, Chi OZ. 17Beta-estradiol blocks NMDA-induced increases in regional cerebral O(2) consumption. Brain Res. 2002 Oct 04;951(2):177-82. doi: 10.1016/s0006-8993(02)03158-x. PMID: 12270495.
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Role of glutamate delta -2 receptors in activity-dependent competition between heterologous afferent fibers.

Proceedings of the National Academy of Sciences of the United States of America

Morando L, Cesa R, Rasetti R, Harvey R, Strata P.
PMID: 11493687
Proc Natl Acad Sci U S A. 2001 Aug 14;98(17):9954-9. doi: 10.1073/pnas.171098398. Epub 2001 Aug 07.

A principle that regulates detailed architecture in the brain is that active terminals have a competitive advantage over less active terminals in establishing synaptic connections. This principle is known to apply to fibers within a single neuronal population competing...

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Morando L, Cesa R, Rasetti R, et al. Role of glutamate delta -2 receptors in activity-dependent competition between heterologous afferent fibers. Proc Natl Acad Sci U S A. 2001;98(17):9954-9doi: 10.1073/pnas.171098398.
Morando, L., Cesa, R., Rasetti, R., Harvey, R., & Strata, P. (2001). Role of glutamate delta -2 receptors in activity-dependent competition between heterologous afferent fibers. Proceedings of the National Academy of Sciences of the United States of America, 98(17), 9954-9. https://doi.org/10.1073/pnas.171098398
Morando, L, et al. "Role of glutamate delta -2 receptors in activity-dependent competition between heterologous afferent fibers." Proceedings of the National Academy of Sciences of the United States of America vol. 98,17 (2001): 9954-9. doi: https://doi.org/10.1073/pnas.171098398
Morando L, Cesa R, Rasetti R, Harvey R, Strata P. Role of glutamate delta -2 receptors in activity-dependent competition between heterologous afferent fibers. Proc Natl Acad Sci U S A. 2001 Aug 14;98(17):9954-9. doi: 10.1073/pnas.171098398. Epub 2001 Aug 07. PMID: 11493687; PMCID: PMC55559.
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Expression of IL-10 and TNF-inhibitor genes in lacrimal gland epithelial cells suppresses their ability to activate lymphocytes.

Cornea

Zhu Z, Stevenson D, Ritter T, Schechter JE, Mircheff AK, Kaslow HR, Trousdale MD.
PMID: 11862098
Cornea. 2002 Mar;21(2):210-4. doi: 10.1097/00003226-200203000-00018.

PURPOSE: To determine whether the expression of either interleukin-10 (IL-10) or tumor necrosis factor (TNF) inhibitor genes in transduced rabbit lacrimal gland epithelial cells suppresses lymphocyte proliferation in an autologous mixed cell reaction, an apparent in vitro model of...

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Zhu Z, Stevenson D, Ritter T, et al. Expression of IL-10 and TNF-inhibitor genes in lacrimal gland epithelial cells suppresses their ability to activate lymphocytes. Cornea. 2002;21(2):210-4doi: 10.1097/00003226-200203000-00018.
Zhu, Z., Stevenson, D., Ritter, T., Schechter, J. E., Mircheff, A. K., Kaslow, H. R., & Trousdale, M. D. (2002). Expression of IL-10 and TNF-inhibitor genes in lacrimal gland epithelial cells suppresses their ability to activate lymphocytes. Cornea, 21(2), 210-4. https://doi.org/10.1097/00003226-200203000-00018
Zhu, Zejin, et al. "Expression of IL-10 and TNF-inhibitor genes in lacrimal gland epithelial cells suppresses their ability to activate lymphocytes." Cornea vol. 21,2 (2002): 210-4. doi: https://doi.org/10.1097/00003226-200203000-00018
Zhu Z, Stevenson D, Ritter T, Schechter JE, Mircheff AK, Kaslow HR, Trousdale MD. Expression of IL-10 and TNF-inhibitor genes in lacrimal gland epithelial cells suppresses their ability to activate lymphocytes. Cornea. 2002 Mar;21(2):210-4. doi: 10.1097/00003226-200203000-00018. PMID: 11862098.
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Irritant dermatitis, irritancy and its role in allergic contact dermatitis.

Clinical and experimental dermatology

Smith HR, Basketter DA, McFadden JP.
PMID: 11952708
Clin Exp Dermatol. 2002 Mar;27(2):138-46. doi: 10.1046/j.1365-2230.2002.00997.x.

Irritant contact dermatitis is the clinical result of sufficient inflammation arising from release of pro-inflammatory cytokines from skin cells (principally keratinocytes) in response to (usually) chemical stimuli. Different clinical forms may arise. The three main pathophysiological changes seen are...

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Smith HR, Basketter DA, McFadden JP. Irritant dermatitis, irritancy and its role in allergic contact dermatitis. Clin Exp Dermatol. 2002;27(2):138-46doi: 10.1046/j.1365-2230.2002.00997.x.
Smith, H. R., Basketter, D. A., & McFadden, J. P. (2002). Irritant dermatitis, irritancy and its role in allergic contact dermatitis. Clinical and experimental dermatology, 27(2), 138-46. https://doi.org/10.1046/j.1365-2230.2002.00997.x
Smith, Harvey R, et al. "Irritant dermatitis, irritancy and its role in allergic contact dermatitis." Clinical and experimental dermatology vol. 27,2 (2002): 138-46. doi: https://doi.org/10.1046/j.1365-2230.2002.00997.x
Smith HR, Basketter DA, McFadden JP. Irritant dermatitis, irritancy and its role in allergic contact dermatitis. Clin Exp Dermatol. 2002 Mar;27(2):138-46. doi: 10.1046/j.1365-2230.2002.00997.x. PMID: 11952708.
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Control of bladder function by peripheral nerves: avenues for novel drug targets.

Urology

Fry CH, Ikeda Y, Harvey R, Wu C, Sui GP.
PMID: 15013649
Urology. 2004 Mar;63(3):24-31. doi: 10.1016/j.urology.2003.10.031.

The micturition reflex involves afferent nerve activation when the bladder is sufficiently full and subsequent controlled firing of parasympathetic efferent nerves to contract the detrusor muscle as part of the voiding mechanism. Alteration of the sensitivity of afferent activation...

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Fry CH, Ikeda Y, Harvey R, et al. Control of bladder function by peripheral nerves: avenues for novel drug targets. Urology. 2004;63(3):24-31doi: 10.1016/j.urology.2003.10.031.
Fry, C. H., Ikeda, Y., Harvey, R., Wu, C., & Sui, G. P. (2004). Control of bladder function by peripheral nerves: avenues for novel drug targets. Urology, 63(3), 24-31. https://doi.org/10.1016/j.urology.2003.10.031
Fry, Christopher H, et al. "Control of bladder function by peripheral nerves: avenues for novel drug targets." Urology vol. 63,3 (2004): 24-31. doi: https://doi.org/10.1016/j.urology.2003.10.031
Fry CH, Ikeda Y, Harvey R, Wu C, Sui GP. Control of bladder function by peripheral nerves: avenues for novel drug targets. Urology. 2004 Mar;63(3):24-31. doi: 10.1016/j.urology.2003.10.031. PMID: 15013649.
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Noninvasive imaging of reporter gene expression in living subjects.

Advances in cancer research

Herschman HR.
PMID: 15530556
Adv Cancer Res. 2004;92:29-80. doi: 10.1016/S0065-230X(04)92003-9.

The development of noninvasive imaging technologies designed specifically for use with small animals has provided new paradigms for cancer research. Traditional molecular biology techniques are being melded with noninvasive imaging technologies to develop a new research domain, "molecular imaging."...

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Herschman HR. Noninvasive imaging of reporter gene expression in living subjects. Adv Cancer Res. 2004;92:29-80doi: 10.1016/S0065-230X(04)92003-9.
Herschman, H. R. (2004). Noninvasive imaging of reporter gene expression in living subjects. Advances in cancer research, 9229-80. https://doi.org/10.1016/S0065-230X(04)92003-9
Herschman, Harvey R. "Noninvasive imaging of reporter gene expression in living subjects." Advances in cancer research vol. 92 (2004): 29-80. doi: https://doi.org/10.1016/S0065-230X(04)92003-9
Herschman HR. Noninvasive imaging of reporter gene expression in living subjects. Adv Cancer Res. 2004;92:29-80. doi: 10.1016/S0065-230X(04)92003-9. PMID: 15530556.
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Showing 13 to 24 of 728 entries