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Showing 13 to 24 of 128 entries
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Climate Disruption and Personal Health-A Conversation About .

Creative nursing

Hanson RL, Keinan A, Berhanu D, Harris M, Habisch M.
PMID: 34903634
Creat Nurs. 2021 Nov 01;27(4):275-277. doi: 10.1891/cn-2021-0020.

In her book

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Hanson RL, Keinan A, Berhanu D, et al. Climate Disruption and Personal Health-A Conversation About . Creat Nurs. 2021;27(4):275-277doi: 10.1891/cn-2021-0020.
Hanson, R. L., Keinan, A., Berhanu, D., Harris, M., & Habisch, M. (2021). Climate Disruption and Personal Health-A Conversation About . Creative nursing, 27(4), 275-277. https://doi.org/10.1891/cn-2021-0020
Hanson, Ranae Lenor, et al. "Climate Disruption and Personal Health-A Conversation About ." Creative nursing vol. 27,4 (2021): 275-277. doi: https://doi.org/10.1891/cn-2021-0020
Hanson RL, Keinan A, Berhanu D, Harris M, Habisch M. Climate Disruption and Personal Health-A Conversation About . Creat Nurs. 2021 Nov 01;27(4):275-277. doi: 10.1891/cn-2021-0020. PMID: 34903634.
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The Rise of Populism and the Reconfiguration of the German Political Space.

Frontiers in big data

Olbrich E, Banisch S.
PMID: 34651123
Front Big Data. 2021 Sep 24;4:731349. doi: 10.3389/fdata.2021.731349. eCollection 2021.

The paper explores the notion of a reconfiguration of political space in the context of the rise of populism and its effects on the political system. We focus on Germany and the appearance of the new right wing party...

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Olbrich E, Banisch S. The Rise of Populism and the Reconfiguration of the German Political Space. Front Big Data. 2021;4:731349doi: 10.3389/fdata.2021.731349.
Olbrich, E., & Banisch, S. (2021). The Rise of Populism and the Reconfiguration of the German Political Space. Frontiers in big data, 4731349. https://doi.org/10.3389/fdata.2021.731349
Olbrich, Eckehard, and Banisch, Sven. "The Rise of Populism and the Reconfiguration of the German Political Space." Frontiers in big data vol. 4 (2021): 731349. doi: https://doi.org/10.3389/fdata.2021.731349
Olbrich E, Banisch S. The Rise of Populism and the Reconfiguration of the German Political Space. Front Big Data. 2021 Sep 24;4:731349. doi: 10.3389/fdata.2021.731349. eCollection 2021. PMID: 34651123; PMCID: PMC8508611.
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Dynamics of opinion expression.

Physical review. E

Gaisbauer F, Olbrich E, Banisch S.
PMID: 33212677
Phys Rev E. 2020 Oct;102(4):042303. doi: 10.1103/PhysRevE.102.042303.

Modeling efforts in opinion dynamics have to a large extent ignored that opinion exchange between individuals can also have an effect on how willing they are to express their opinion publicly. Here, we introduce a model of public opinion...

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Gaisbauer F, Olbrich E, Banisch S. Dynamics of opinion expression. Phys Rev E. 2020;102(4):042303doi: 10.1103/PhysRevE.102.042303.
Gaisbauer, F., Olbrich, E., & Banisch, S. (2020). Dynamics of opinion expression. Physical review. E, 102(4), 042303. https://doi.org/10.1103/PhysRevE.102.042303
Gaisbauer, Felix, et al. "Dynamics of opinion expression." Physical review. E vol. 102,4 (2020): 042303. doi: https://doi.org/10.1103/PhysRevE.102.042303
Gaisbauer F, Olbrich E, Banisch S. Dynamics of opinion expression. Phys Rev E. 2020 Oct;102(4):042303. doi: 10.1103/PhysRevE.102.042303. PMID: 33212677.
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Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistance.

Experimental hematology & oncology

Klümper T, Bruckmueller H, Diewock T, Kaehler M, Haenisch S, Pott C, Bruhn O, Cascorbi I.
PMID: 32999756
Exp Hematol Oncol. 2020 Sep 26;9:26. doi: 10.1186/s40164-020-00183-1. eCollection 2020.

BACKGROUND: Chronic myeloid leukemia (CML) is a myeloproliferative neoplasm characterized by constitutive activity of the tyrosine kinase BCR-ABL1. Although the introduction of tyrosine kinase inhibitors (TKIs) has substantially improved patients' prognosis, drug resistance remains one of the major challenges...

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Klümper T, Bruckmueller H, Diewock T, et al. Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistance. Exp Hematol Oncol. 2020;9:26doi: 10.1186/s40164-020-00183-1.
Klümper, T., Bruckmueller, H., Diewock, T., Kaehler, M., Haenisch, S., Pott, C., Bruhn, O., & Cascorbi, I. (2020). Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistance. Experimental hematology & oncology, 926. https://doi.org/10.1186/s40164-020-00183-1
Klümper, Theresa, et al. "Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistance." Experimental hematology & oncology vol. 9 (2020): 26. doi: https://doi.org/10.1186/s40164-020-00183-1
Klümper T, Bruckmueller H, Diewock T, Kaehler M, Haenisch S, Pott C, Bruhn O, Cascorbi I. Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistance. Exp Hematol Oncol. 2020 Sep 26;9:26. doi: 10.1186/s40164-020-00183-1. eCollection 2020. PMID: 32999756; PMCID: PMC7519530.
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Enzyme replacement therapy and antibodies in late-onset Pompe disease.

Molecular genetics and metabolism reports

Schneider I, Deschauer M, Hanisch F.
PMID: 27896092
Mol Genet Metab Rep. 2014 May 15;1:232-234. doi: 10.1016/j.ymgmr.2014.01.001. eCollection 2014.

No abstract available.

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Schneider I, Deschauer M, Hanisch F. Enzyme replacement therapy and antibodies in late-onset Pompe disease. Mol Genet Metab Rep. 2014;1:232-234doi: 10.1016/j.ymgmr.2014.01.001.
Schneider, I., Deschauer, M., & Hanisch, F. (2014). Enzyme replacement therapy and antibodies in late-onset Pompe disease. Molecular genetics and metabolism reports, 1232-234. https://doi.org/10.1016/j.ymgmr.2014.01.001
Schneider, I, et al. "Enzyme replacement therapy and antibodies in late-onset Pompe disease." Molecular genetics and metabolism reports vol. 1 (2014): 232-234. doi: https://doi.org/10.1016/j.ymgmr.2014.01.001
Schneider I, Deschauer M, Hanisch F. Enzyme replacement therapy and antibodies in late-onset Pompe disease. Mol Genet Metab Rep. 2014 May 15;1:232-234. doi: 10.1016/j.ymgmr.2014.01.001. eCollection 2014. PMID: 27896092; PMCID: PMC5121312.
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Porosity and distribution of water in perlite from the island of Milos, Greece.

SpringerPlus

Kaufhold S, Reese A, Schwiebacher W, Dohrmann R, Grathoff GH, Warr LN, Halisch M, Müller C, Schwarz-Schampera U, Ufer K.
PMID: 25392772
Springerplus. 2014 Oct 12;3:598. doi: 10.1186/2193-1801-3-598. eCollection 2014.

A perlite sample representative of an operating mine in Milos was investigated with respect to the type and spatial distribution of water. A set of different methods was used which finally provided a consistent view on the water at...

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Kaufhold S, Reese A, Schwiebacher W, et al. Porosity and distribution of water in perlite from the island of Milos, Greece. Springerplus. 2014;3:598doi: 10.1186/2193-1801-3-598.
Kaufhold, S., Reese, A., Schwiebacher, W., Dohrmann, R., Grathoff, G. H., Warr, L. N., Halisch, M., Müller, C., Schwarz-Schampera, U., & Ufer, K. (2014). Porosity and distribution of water in perlite from the island of Milos, Greece. SpringerPlus, 3598. https://doi.org/10.1186/2193-1801-3-598
Kaufhold, Stephan, et al. "Porosity and distribution of water in perlite from the island of Milos, Greece." SpringerPlus vol. 3 (2014): 598. doi: https://doi.org/10.1186/2193-1801-3-598
Kaufhold S, Reese A, Schwiebacher W, Dohrmann R, Grathoff GH, Warr LN, Halisch M, Müller C, Schwarz-Schampera U, Ufer K. Porosity and distribution of water in perlite from the island of Milos, Greece. Springerplus. 2014 Oct 12;3:598. doi: 10.1186/2193-1801-3-598. eCollection 2014. PMID: 25392772; PMCID: PMC4203787.
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The Biological Effect of Particulate Titanium Contaminants of Dental Implants on Human Osteoblasts and Gingival Fibroblasts.

The International journal of oral & maxillofacial implants

Happe A, Sielker S, Hanisch M, Jung S.
PMID: 31170276
Int J Oral Maxillofac Implants. 2019 Feb 26; doi: 10.11607/jomi.INSERT 4 DIGIT DOI. Epub 2019 Feb 26.

PURPOSE: To evaluate the effects of different titanium particle concentrations on viability of human calvarial osteoblasts and human gingival fibroblasts.MATERIALS AND METHODS: Primary human calvarial osteoblasts (HCO, 3H Biomedical) and human gingival fibroblasts (HGF-1, ATCC) were cultivated and allowed...

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Happe A, Sielker S, Hanisch M, et al. The Biological Effect of Particulate Titanium Contaminants of Dental Implants on Human Osteoblasts and Gingival Fibroblasts. Int J Oral Maxillofac Implants. 2019;doi: 10.11607/jomi.INSERT 4 DIGIT DOI.
Happe, A., Sielker, S., Hanisch, M., & Jung, S. (2019). The Biological Effect of Particulate Titanium Contaminants of Dental Implants on Human Osteoblasts and Gingival Fibroblasts. The International journal of oral & maxillofacial implants, . https://doi.org/10.11607/jomi.INSERT 4 DIGIT DOI
Happe, Arndt, et al. "The Biological Effect of Particulate Titanium Contaminants of Dental Implants on Human Osteoblasts and Gingival Fibroblasts." The International journal of oral & maxillofacial implants vol. (2019). doi: https://doi.org/10.11607/jomi.INSERT 4 DIGIT DOI
Happe A, Sielker S, Hanisch M, Jung S. The Biological Effect of Particulate Titanium Contaminants of Dental Implants on Human Osteoblasts and Gingival Fibroblasts. Int J Oral Maxillofac Implants. 2019 Feb 26; doi: 10.11607/jomi.INSERT 4 DIGIT DOI. Epub 2019 Feb 26. PMID: 31170276.
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Network measures of mixing.

Chaos (Woodbury, N.Y.)

Banisch R, Koltai P, Padberg-Gehle K.
PMID: 31266326
Chaos. 2019 Jun;29(6):063125. doi: 10.1063/1.5087632.

Transport and mixing processes in fluid flows can be studied directly from Lagrangian trajectory data, such as those obtained from particle tracking experiments. Recent work in this context highlights the application of graph-based approaches, where trajectories serve as nodes...

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Banisch R, Koltai P, Padberg-Gehle K. Network measures of mixing. Chaos. 2019;29(6):063125doi: 10.1063/1.5087632.
Banisch, R., Koltai, P., & Padberg-Gehle, K. (2019). Network measures of mixing. Chaos (Woodbury, N.Y.), 29(6), 063125. https://doi.org/10.1063/1.5087632
Banisch, Ralf, et al. "Network measures of mixing." Chaos (Woodbury, N.Y.) vol. 29,6 (2019): 063125. doi: https://doi.org/10.1063/1.5087632
Banisch R, Koltai P, Padberg-Gehle K. Network measures of mixing. Chaos. 2019 Jun;29(6):063125. doi: 10.1063/1.5087632. PMID: 31266326.
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ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period.

Journal of intensive care

Banjas N, Hopf HB, Hanisch E, Friedrichson B, Fichte J, Buia A.
PMID: 30598825
J Intensive Care. 2018 Dec 18;6:84. doi: 10.1186/s40560-018-0352-2. eCollection 2018.

BACKGROUND: Based on promising results over the past 10 years, the method of extracorporeal membrane oxygenation (ECMO) has developed from being used as a 'rescue therapy' to become an accepted treatment option for patients with acute lung failure (ARDS)....

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Banjas N, Hopf HB, Hanisch E, et al. ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period. J Intensive Care. 2018;6:84doi: 10.1186/s40560-018-0352-2.
Banjas, N., Hopf, H. B., Hanisch, E., Friedrichson, B., Fichte, J., & Buia, A. (2018). ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period. Journal of intensive care, 684. https://doi.org/10.1186/s40560-018-0352-2
Banjas, Norbert, et al. "ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period." Journal of intensive care vol. 6 (2018): 84. doi: https://doi.org/10.1186/s40560-018-0352-2
Banjas N, Hopf HB, Hanisch E, Friedrichson B, Fichte J, Buia A. ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period. J Intensive Care. 2018 Dec 18;6:84. doi: 10.1186/s40560-018-0352-2. eCollection 2018. PMID: 30598825; PMCID: PMC6299557.
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Laquinimod prevents cuprizone-induced demyelination independent of Toll-like receptor signaling.

Neurology(R) neuroimmunology & neuroinflammation

Kramann N, Menken L, Hayardeny L, Hanisch UK, Brück W.
PMID: 27231712
Neurol Neuroimmunol Neuroinflamm. 2016 May 17;3(3):e233. doi: 10.1212/NXI.0000000000000233. eCollection 2016 Jun.

OBJECTIVE: To test whether Toll-like receptor (TLR) signaling plays a key role for reduced nuclear factor B (NF-κB) activation after laquinimod treatment in the model of cuprizone-induced demyelination, oligodendrocyte apoptosis, inflammation, and axonal damage.METHODS: Ten-week-old C57BL/6J, TLR4(-/-), and MyD88(-/-)...

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Kramann N, Menken L, Hayardeny L, et al. Laquinimod prevents cuprizone-induced demyelination independent of Toll-like receptor signaling. Neurol Neuroimmunol Neuroinflamm. 2016;3(3):e233doi: 10.1212/NXI.0000000000000233.
Kramann, N., Menken, L., Hayardeny, L., Hanisch, U. K., & Brück, W. (2016). Laquinimod prevents cuprizone-induced demyelination independent of Toll-like receptor signaling. Neurology(R) neuroimmunology & neuroinflammation, 3(3), e233. https://doi.org/10.1212/NXI.0000000000000233
Kramann, Nadine, et al. "Laquinimod prevents cuprizone-induced demyelination independent of Toll-like receptor signaling." Neurology(R) neuroimmunology & neuroinflammation vol. 3,3 (2016): e233. doi: https://doi.org/10.1212/NXI.0000000000000233
Kramann N, Menken L, Hayardeny L, Hanisch UK, Brück W. Laquinimod prevents cuprizone-induced demyelination independent of Toll-like receptor signaling. Neurol Neuroimmunol Neuroinflamm. 2016 May 17;3(3):e233. doi: 10.1212/NXI.0000000000000233. eCollection 2016 Jun. PMID: 27231712; PMCID: PMC4871804.
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Correction to: ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period.

Journal of intensive care

Banjas N, Hopf HB, Hanisch E, Friedrichson B, Fichte J, Buia A.
PMID: 30733869
J Intensive Care. 2019 Jan 31;7:8. doi: 10.1186/s40560-019-0362-8. eCollection 2019.

[This corrects the article DOI: 10.1186/s40560-018-0352-2.].

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Banjas N, Hopf HB, Hanisch E, et al. Correction to: ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period. J Intensive Care. 2019;7:8doi: 10.1186/s40560-019-0362-8.
Banjas, N., Hopf, H. B., Hanisch, E., Friedrichson, B., Fichte, J., & Buia, A. (2019). Correction to: ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period. Journal of intensive care, 78. https://doi.org/10.1186/s40560-019-0362-8
Banjas, Norbert, et al. "Correction to: ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period." Journal of intensive care vol. 7 (2019): 8. doi: https://doi.org/10.1186/s40560-019-0362-8
Banjas N, Hopf HB, Hanisch E, Friedrichson B, Fichte J, Buia A. Correction to: ECMO-treatment in patients with acute lung failure, cardiogenic, and septic shock: mortality and ECMO-learning curve over a 6-year period. J Intensive Care. 2019 Jan 31;7:8. doi: 10.1186/s40560-019-0362-8. eCollection 2019. PMID: 30733869; PMCID: PMC6354409.
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Microglial diversity by responses and responders.

Frontiers in cellular neuroscience

Gertig U, Hanisch UK.
PMID: 24744702
Front Cell Neurosci. 2014 Apr 01;8:101. doi: 10.3389/fncel.2014.00101. eCollection 2014.

Microglia are the principal resident innate immune cells of the CNS. Their contributions to the normal development of the CNS, the maintenance and plasticity of neuronal networks and the safeguarding of proper functionality are becoming more and more evident....

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Gertig U, Hanisch UK. Microglial diversity by responses and responders. Front Cell Neurosci. 2014;8:101doi: 10.3389/fncel.2014.00101.
Gertig, U., & Hanisch, U. K. (2014). Microglial diversity by responses and responders. Frontiers in cellular neuroscience, 8101. https://doi.org/10.3389/fncel.2014.00101
Gertig, Ulla, and Hanisch, Uwe-Karsten. "Microglial diversity by responses and responders." Frontiers in cellular neuroscience vol. 8 (2014): 101. doi: https://doi.org/10.3389/fncel.2014.00101
Gertig U, Hanisch UK. Microglial diversity by responses and responders. Front Cell Neurosci. 2014 Apr 01;8:101. doi: 10.3389/fncel.2014.00101. eCollection 2014. PMID: 24744702; PMCID: PMC3978327.
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Showing 13 to 24 of 128 entries