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Percutaneous reconstruction of aortic isthmus atresia using coronary total occlusion technique.

Journal of cardiology cases

Goel PK, Syal SK.
PMID: 30534221
J Cardiol Cases. 2014 Jul 22;10(4):121-124. doi: 10.1016/j.jccase.2014.06.003. eCollection 2014 Oct.

Aortic isthmus atresia is an extreme form of coarctation presenting in adults and usually calls for surgical correction. In this report, we present our initial experience with the first four cases successfully treated percutaneously using dedicated coronary total occlusion...

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Goel PK, Syal SK. Percutaneous reconstruction of aortic isthmus atresia using coronary total occlusion technique. J Cardiol Cases. 2014;10(4):121-124doi: 10.1016/j.jccase.2014.06.003.
Goel, P. K., & Syal, S. K. (2014). Percutaneous reconstruction of aortic isthmus atresia using coronary total occlusion technique. Journal of cardiology cases, 10(4), 121-124. https://doi.org/10.1016/j.jccase.2014.06.003
Goel, Pravin K, and Syal, Sanjeev K. "Percutaneous reconstruction of aortic isthmus atresia using coronary total occlusion technique." Journal of cardiology cases vol. 10,4 (2014): 121-124. doi: https://doi.org/10.1016/j.jccase.2014.06.003
Goel PK, Syal SK. Percutaneous reconstruction of aortic isthmus atresia using coronary total occlusion technique. J Cardiol Cases. 2014 Jul 22;10(4):121-124. doi: 10.1016/j.jccase.2014.06.003. eCollection 2014 Oct. PMID: 30534221; PMCID: PMC6279530.
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Non-multiple-Sclerosis-Related Typical and Atypical White Matter Disorders: Our Experience in the Last 2 Years in Both Children and Adults From a Tertiary Care Center in India.

Journal of pediatric neurosciences

Chandra SR, Ramanujam CN, Kalya Vyasaraj K, Christopher R, Padmanabha H, Jagadish A, Arshad F, Gohel A.
PMID: 31316639
J Pediatr Neurosci. 2019 Jan-Mar;14(1):20-29. doi: 10.4103/jpn.JPN_37_19.

INTRODUCTION: White matter signal changes are common in wide spectrum of disorders other than primary demyelinating diseases. Knowledge into their characteristics is of great relevance as treatment options are available in most cases, if diagnosed early.PATIENT AND METHODS: Patients...

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Chandra SR, Ramanujam CN, Kalya Vyasaraj K, et al. Non-multiple-Sclerosis-Related Typical and Atypical White Matter Disorders: Our Experience in the Last 2 Years in Both Children and Adults From a Tertiary Care Center in India. J Pediatr Neurosci. 2019;14(1):20-29doi: 10.4103/jpn.JPN_37_19.
Chandra, S. R., Ramanujam, C. N., Kalya Vyasaraj, K., Christopher, R., Padmanabha, H., Jagadish, A., Arshad, F., & Gohel, A. (2019). Non-multiple-Sclerosis-Related Typical and Atypical White Matter Disorders: Our Experience in the Last 2 Years in Both Children and Adults From a Tertiary Care Center in India. Journal of pediatric neurosciences, 14(1), 20-29. https://doi.org/10.4103/jpn.JPN_37_19
Chandra, Sadanandavalli Retnaswami, et al. "Non-multiple-Sclerosis-Related Typical and Atypical White Matter Disorders: Our Experience in the Last 2 Years in Both Children and Adults From a Tertiary Care Center in India." Journal of pediatric neurosciences vol. 14,1 (2019): 20-29. doi: https://doi.org/10.4103/jpn.JPN_37_19
Chandra SR, Ramanujam CN, Kalya Vyasaraj K, Christopher R, Padmanabha H, Jagadish A, Arshad F, Gohel A. Non-multiple-Sclerosis-Related Typical and Atypical White Matter Disorders: Our Experience in the Last 2 Years in Both Children and Adults From a Tertiary Care Center in India. J Pediatr Neurosci. 2019 Jan-Mar;14(1):20-29. doi: 10.4103/jpn.JPN_37_19. PMID: 31316639; PMCID: PMC6601113.
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Reply to 'The link between pulmonary hypertension and adverse renal transplant outcome may be renal venous hypertension'.

Indian journal of anaesthesia

Solanki SL, Goyal VK, Baj B.
PMID: 29720766
Indian J Anaesth. 2018 Apr;62(4):325. doi: 10.4103/ija.IJA_179_18.

No abstract available.

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Solanki SL, Goyal VK, Baj B. Reply to 'The link between pulmonary hypertension and adverse renal transplant outcome may be renal venous hypertension'. Indian J Anaesth. 2018;62(4):325doi: 10.4103/ija.IJA_179_18.
Solanki, S. L., Goyal, V. K., & Baj, B. (2018). Reply to 'The link between pulmonary hypertension and adverse renal transplant outcome may be renal venous hypertension'. Indian journal of anaesthesia, 62(4), 325. https://doi.org/10.4103/ija.IJA_179_18
Solanki, Sohan Lal, et al. "Reply to 'The link between pulmonary hypertension and adverse renal transplant outcome may be renal venous hypertension'." Indian journal of anaesthesia vol. 62,4 (2018): 325. doi: https://doi.org/10.4103/ija.IJA_179_18
Solanki SL, Goyal VK, Baj B. Reply to 'The link between pulmonary hypertension and adverse renal transplant outcome may be renal venous hypertension'. Indian J Anaesth. 2018 Apr;62(4):325. doi: 10.4103/ija.IJA_179_18. PMID: 29720766; PMCID: PMC5907446.
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Biomechanics of the Sacroiliac Joint: Surgical Treatments.

International journal of spine surgery

Joukar A, Kiapour A, Elgafy H, Erbulut DU, Agarwal AK, Goel VK.
PMID: 32699758
Int J Spine Surg. 2020 Jun 30;14(3):355-367. doi: 10.14444/7047. eCollection 2020 Jun.

BACKGROUND: Fixation is one of the most common surgical techniques for the treatment of chronic pain originating from the sacroiliac joint (SIJ). Many studies have investigated the clinical outcomes and biomechanics of various SIJ surgical procedures. However, the biomechanical...

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Joukar A, Kiapour A, Elgafy H, et al. Biomechanics of the Sacroiliac Joint: Surgical Treatments. Int J Spine Surg. 2020;14(3):355-367doi: 10.14444/7047.
Joukar, A., Kiapour, A., Elgafy, H., Erbulut, D. U., Agarwal, A. K., & Goel, V. K. (2020). Biomechanics of the Sacroiliac Joint: Surgical Treatments. International journal of spine surgery, 14(3), 355-367. https://doi.org/10.14444/7047
Joukar, Amin, et al. "Biomechanics of the Sacroiliac Joint: Surgical Treatments." International journal of spine surgery vol. 14,3 (2020): 355-367. doi: https://doi.org/10.14444/7047
Joukar A, Kiapour A, Elgafy H, Erbulut DU, Agarwal AK, Goel VK. Biomechanics of the Sacroiliac Joint: Surgical Treatments. Int J Spine Surg. 2020 Jun 30;14(3):355-367. doi: 10.14444/7047. eCollection 2020 Jun. PMID: 32699758; PMCID: PMC7343255.
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Bilateral Extracranial Internal Carotid Artery Aneurysms in a Patient with Marfan Syndrome: Case Report and Review of the Literature.

EJVES short reports

Singh AA, Velineni R, Varty K, Gohel MS.
PMID: 31650048
EJVES Short Rep. 2019 Sep 25;45:14-16. doi: 10.1016/j.ejvssr.2019.08.003. eCollection 2019.

INTRODUCTION: Aneurysms of the extracranial carotid artery are rare. They are often asymptomatic but can lead to disabling stroke. This report describes a case of bilateral internal carotid artery aneurysms in a patient with Marfan syndrome.REPORT: A 65 year...

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Singh AA, Velineni R, Varty K, et al. Bilateral Extracranial Internal Carotid Artery Aneurysms in a Patient with Marfan Syndrome: Case Report and Review of the Literature. EJVES Short Rep. 2019;45:14-16doi: 10.1016/j.ejvssr.2019.08.003.
Singh, A. A., Velineni, R., Varty, K., & Gohel, M. S. (2019). Bilateral Extracranial Internal Carotid Artery Aneurysms in a Patient with Marfan Syndrome: Case Report and Review of the Literature. EJVES short reports, 4514-16. https://doi.org/10.1016/j.ejvssr.2019.08.003
Singh, Aminder Anthony, et al. "Bilateral Extracranial Internal Carotid Artery Aneurysms in a Patient with Marfan Syndrome: Case Report and Review of the Literature." EJVES short reports vol. 45 (2019): 14-16. doi: https://doi.org/10.1016/j.ejvssr.2019.08.003
Singh AA, Velineni R, Varty K, Gohel MS. Bilateral Extracranial Internal Carotid Artery Aneurysms in a Patient with Marfan Syndrome: Case Report and Review of the Literature. EJVES Short Rep. 2019 Sep 25;45:14-16. doi: 10.1016/j.ejvssr.2019.08.003. eCollection 2019. PMID: 31650048; PMCID: PMC6804523.
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First report of pulmonary alveolar microlithiasis diagnosed by cryobiopsy.

Lung India : official organ of Indian Chest Society

Goel MK, Kumar A, Maitra G.
PMID: 32108613
Lung India. 2020 Mar-Apr;37(2):183-185. doi: 10.4103/lungindia.lungindia_497_19.

No abstract available.

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Goel MK, Kumar A, Maitra G. First report of pulmonary alveolar microlithiasis diagnosed by cryobiopsy. Lung India. 2020;37(2):183-185doi: 10.4103/lungindia.lungindia_497_19.
Goel, M. K., Kumar, A., & Maitra, G. (2020). First report of pulmonary alveolar microlithiasis diagnosed by cryobiopsy. Lung India : official organ of Indian Chest Society, 37(2), 183-185. https://doi.org/10.4103/lungindia.lungindia_497_19
Goel, Manoj Kumar, et al. "First report of pulmonary alveolar microlithiasis diagnosed by cryobiopsy." Lung India : official organ of Indian Chest Society vol. 37,2 (2020): 183-185. doi: https://doi.org/10.4103/lungindia.lungindia_497_19
Goel MK, Kumar A, Maitra G. First report of pulmonary alveolar microlithiasis diagnosed by cryobiopsy. Lung India. 2020 Mar-Apr;37(2):183-185. doi: 10.4103/lungindia.lungindia_497_19. PMID: 32108613; PMCID: PMC7065543.
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Connecting copper and cancer: from transition metal signalling to metalloplasia.

Nature reviews. Cancer

Ge EJ, Bush AI, Casini A, Cobine PA, Cross JR, DeNicola GM, Dou QP, Franz KJ, Gohil VM, Gupta S, Kaler SG, Lutsenko S, Mittal V, Petris MJ, Polishchuk R, Ralle M, Schilsky ML, Tonks NK, Vahdat LT, Van Aelst L, Xi D, Yuan P, Brady DC, Chang CJ.
PMID: 34764459
Nat Rev Cancer. 2021 Nov 11; doi: 10.1038/s41568-021-00417-2. Epub 2021 Nov 11.

Copper is an essential nutrient whose redox properties make it both beneficial and toxic to the cell. Recent progress in studying transition metal signalling has forged new links between researchers of different disciplines that can help translate basic research...

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Ge EJ, Bush AI, Casini A, et al. Connecting copper and cancer: from transition metal signalling to metalloplasia. Nat Rev Cancer. 2021;doi: 10.1038/s41568-021-00417-2.
Ge, E. J., Bush, A. I., Casini, A., Cobine, P. A., Cross, J. R., DeNicola, G. M., Dou, Q. P., Franz, K. J., Gohil, V. M., Gupta, S., Kaler, S. G., Lutsenko, S., Mittal, V., Petris, M. J., Polishchuk, R., Ralle, M., Schilsky, M. L., Tonks, N. K., Vahdat, L. T., Van Aelst, L., Xi, D., Yuan, P., Brady, D. C., & Chang, C. J. (2021). Connecting copper and cancer: from transition metal signalling to metalloplasia. Nature reviews. Cancer, . https://doi.org/10.1038/s41568-021-00417-2
Ge, Eva J, et al. "Connecting copper and cancer: from transition metal signalling to metalloplasia." Nature reviews. Cancer vol. (2021). doi: https://doi.org/10.1038/s41568-021-00417-2
Ge EJ, Bush AI, Casini A, Cobine PA, Cross JR, DeNicola GM, Dou QP, Franz KJ, Gohil VM, Gupta S, Kaler SG, Lutsenko S, Mittal V, Petris MJ, Polishchuk R, Ralle M, Schilsky ML, Tonks NK, Vahdat LT, Van Aelst L, Xi D, Yuan P, Brady DC, Chang CJ. Connecting copper and cancer: from transition metal signalling to metalloplasia. Nat Rev Cancer. 2021 Nov 11; doi: 10.1038/s41568-021-00417-2. Epub 2021 Nov 11. PMID: 34764459.
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Zotatifin, an eIF4A-Selective Inhibitor, Blocks Tumor Growth in Receptor Tyrosine Kinase Driven Tumors.

Frontiers in oncology

Gerson-Gurwitz A, Young NP, Goel VK, Eam B, Stumpf CR, Chen J, Fish S, Barrera M, Sung E, Staunton J, Chiang GG, Webster KR, Thompson PA.
PMID: 34900714
Front Oncol. 2021 Nov 24;11:766298. doi: 10.3389/fonc.2021.766298. eCollection 2021.

Oncoprotein expression is controlled at the level of mRNA translation and is regulated by the eukaryotic translation initiation factor 4F (eIF4F) complex. eIF4A, a component of eIF4F, catalyzes the unwinding of secondary structure in the 5'-untranslated region (5'-UTR) of...

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Gerson-Gurwitz A, Young NP, Goel VK, et al. Zotatifin, an eIF4A-Selective Inhibitor, Blocks Tumor Growth in Receptor Tyrosine Kinase Driven Tumors. Front Oncol. 2021;11:766298doi: 10.3389/fonc.2021.766298.
Gerson-Gurwitz, A., Young, N. P., Goel, V. K., Eam, B., Stumpf, C. R., Chen, J., Fish, S., Barrera, M., Sung, E., Staunton, J., Chiang, G. G., Webster, K. R., & Thompson, P. A. (2021). Zotatifin, an eIF4A-Selective Inhibitor, Blocks Tumor Growth in Receptor Tyrosine Kinase Driven Tumors. Frontiers in oncology, 11766298. https://doi.org/10.3389/fonc.2021.766298
Gerson-Gurwitz, Adina, et al. "Zotatifin, an eIF4A-Selective Inhibitor, Blocks Tumor Growth in Receptor Tyrosine Kinase Driven Tumors." Frontiers in oncology vol. 11 (2021): 766298. doi: https://doi.org/10.3389/fonc.2021.766298
Gerson-Gurwitz A, Young NP, Goel VK, Eam B, Stumpf CR, Chen J, Fish S, Barrera M, Sung E, Staunton J, Chiang GG, Webster KR, Thompson PA. Zotatifin, an eIF4A-Selective Inhibitor, Blocks Tumor Growth in Receptor Tyrosine Kinase Driven Tumors. Front Oncol. 2021 Nov 24;11:766298. doi: 10.3389/fonc.2021.766298. eCollection 2021. PMID: 34900714; PMCID: PMC8663026.
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Actionable Cytopathogenic Host Responses of Human Alveolar Type 2 Cells to SARS-CoV-2.

Molecular cell

Hekman RM, Hume AJ, Goel RK, Abo KM, Huang J, Blum BC, Werder RB, Suder EL, Paul I, Phanse S, Youssef A, Alysandratos KD, Padhorny D, Ojha S, Mora-Martin A, Kretov D, Ash PEA, Verma M, Zhao J, Patten JJ, Villacorta-Martin C, Bolzan D, Perea-Resa C, Bullitt E, Hinds A, Tilston-Lunel A, Varelas X, Farhangmehr S, Braunschweig U, Kwan JH, McComb M, Basu A, Saeed M, Perissi V, Burks EJ, Layne MD, Connor JH, Davey R, Cheng JX, Wolozin BL, Blencowe BJ, Wuchty S, Lyons SM, Kozakov D, Cifuentes D, Blower M, Kotton DN, Wilson AA, Mühlberger E, Emili A.
PMID: 33417854
Mol Cell. 2021 Jan 07;81(1):212. doi: 10.1016/j.molcel.2020.12.028.

No abstract available.

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Hekman RM, Hume AJ, Goel RK, et al. Actionable Cytopathogenic Host Responses of Human Alveolar Type 2 Cells to SARS-CoV-2. Mol Cell. 2021;81(1):212doi: 10.1016/j.molcel.2020.12.028.
Hekman, R. M., Hume, A. J., Goel, R. K., Abo, K. M., Huang, J., Blum, B. C., Werder, R. B., Suder, E. L., Paul, I., Phanse, S., Youssef, A., Alysandratos, K. D., Padhorny, D., Ojha, S., Mora-Martin, A., Kretov, D., Ash, P. E. A., Verma, M., Zhao, J., Patten, J. J., Villacorta-Martin, C., Bolzan, D., Perea-Resa, C., Bullitt, E., Hinds, A., Tilston-Lunel, A., Varelas, X., Farhangmehr, S., Braunschweig, U., Kwan, J. H., McComb, M., Basu, A., Saeed, M., Perissi, V., Burks, E. J., Layne, M. D., Connor, J. H., Davey, R., Cheng, J. X., Wolozin, B. L., Blencowe, B. J., Wuchty, S., Lyons, S. M., Kozakov, D., Cifuentes, D., Blower, M., Kotton, D. N., Wilson, A. A., Mühlberger, E., & Emili, A. (2021). Actionable Cytopathogenic Host Responses of Human Alveolar Type 2 Cells to SARS-CoV-2. Molecular cell, 81(1), 212. https://doi.org/10.1016/j.molcel.2020.12.028
Hekman, Ryan M, et al. "Actionable Cytopathogenic Host Responses of Human Alveolar Type 2 Cells to SARS-CoV-2." Molecular cell vol. 81,1 (2021): 212. doi: https://doi.org/10.1016/j.molcel.2020.12.028
Hekman RM, Hume AJ, Goel RK, Abo KM, Huang J, Blum BC, Werder RB, Suder EL, Paul I, Phanse S, Youssef A, Alysandratos KD, Padhorny D, Ojha S, Mora-Martin A, Kretov D, Ash PEA, Verma M, Zhao J, Patten JJ, Villacorta-Martin C, Bolzan D, Perea-Resa C, Bullitt E, Hinds A, Tilston-Lunel A, Varelas X, Farhangmehr S, Braunschweig U, Kwan JH, McComb M, Basu A, Saeed M, Perissi V, Burks EJ, Layne MD, Connor JH, Davey R, Cheng JX, Wolozin BL, Blencowe BJ, Wuchty S, Lyons SM, Kozakov D, Cifuentes D, Blower M, Kotton DN, Wilson AA, Mühlberger E, Emili A. Actionable Cytopathogenic Host Responses of Human Alveolar Type 2 Cells to SARS-CoV-2. Mol Cell. 2021 Jan 07;81(1):212. doi: 10.1016/j.molcel.2020.12.028. PMID: 33417854; PMCID: PMC7831449.
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Reversal of Acute Lead Encephalopathy in a Child.

Cureus

Keshri S, Goel AK, Garg AK.
PMID: 34178489
Cureus. 2021 May 21;13(5):e15155. doi: 10.7759/cureus.15155.

Lead poisoning, fairly common in the 20th century, has decreased drastically in the last decade. Severe lead poisoning in the form of encephalopathy has a fatality rate of 28% to 45% and neurological sequelae in about 82%. We present...

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Keshri S, Goel AK, Garg AK. Reversal of Acute Lead Encephalopathy in a Child. Cureus. 2021;13(5):e15155doi: 10.7759/cureus.15155.
Keshri, S., Goel, A. K., & Garg, A. K. (2021). Reversal of Acute Lead Encephalopathy in a Child. Cureus, 13(5), e15155. https://doi.org/10.7759/cureus.15155
Keshri, Swasti, et al. "Reversal of Acute Lead Encephalopathy in a Child." Cureus vol. 13,5 (2021): e15155. doi: https://doi.org/10.7759/cureus.15155
Keshri S, Goel AK, Garg AK. Reversal of Acute Lead Encephalopathy in a Child. Cureus. 2021 May 21;13(5):e15155. doi: 10.7759/cureus.15155. PMID: 34178489; PMCID: PMC8216578.
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TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1.

Cellular signalling

Roy M, Singh K, Shinde A, Singh J, Mane M, Bedekar S, Tailor Y, Gohel D, Vasiyani H, Currim F, Singh R.
PMID: 34871740
Cell Signal. 2021 Dec 03;91:110210. doi: 10.1016/j.cellsig.2021.110210. Epub 2021 Dec 03.

Ubiquitin E3-ligases are recruited at different steps of TNF-α-induced NF-κB activation; however, their role in temporal regulation of the pathway remains elusive. The study systematically identified TRIMs as potential feedback regulators of the TNF-α-induced NF-κB pathway. We further observed...

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Roy M, Singh K, Shinde A, et al. TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1. Cell Signal. 2021;91:110210doi: 10.1016/j.cellsig.2021.110210.
Roy, M., Singh, K., Shinde, A., Singh, J., Mane, M., Bedekar, S., Tailor, Y., Gohel, D., Vasiyani, H., Currim, F., & Singh, R. (2021). TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1. Cellular signalling, 91110210. https://doi.org/10.1016/j.cellsig.2021.110210
Roy, Milton, et al. "TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1." Cellular signalling vol. 91 (2021): 110210. doi: https://doi.org/10.1016/j.cellsig.2021.110210
Roy M, Singh K, Shinde A, Singh J, Mane M, Bedekar S, Tailor Y, Gohel D, Vasiyani H, Currim F, Singh R. TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1. Cell Signal. 2021 Dec 03;91:110210. doi: 10.1016/j.cellsig.2021.110210. Epub 2021 Dec 03. PMID: 34871740.
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TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1.

Cellular signalling

Roy M, Singh K, Shinde A, Singh J, Mane M, Bedekar S, Tailor Y, Gohel D, Vasiyani H, Currim F, Singh R.
PMID: 34871740
Cell Signal. 2021 Dec 03;110210. doi: 10.1016/j.cellsig.2021.110210. Epub 2021 Dec 03.

Ubiquitin E3-ligases are recruited at different steps of TNF-α-induced NF-κB activation; however, their role in temporal regulation of the pathway remains elusive. The study systematically identified TRIMs as potential feedback regulators of the TNF-α-induced NF-κB pathway. We further observed...

Cite
Roy M, Singh K, Shinde A, et al. TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1. Cell Signal. 2021;110210doi: 10.1016/j.cellsig.2021.110210.
Roy, M., Singh, K., Shinde, A., Singh, J., Mane, M., Bedekar, S., Tailor, Y., Gohel, D., Vasiyani, H., Currim, F., & Singh, R. (2021). TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1. Cellular signalling, 110210. https://doi.org/10.1016/j.cellsig.2021.110210
Roy, Milton, et al. "TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1." Cellular signalling vol. (2021): 110210. doi: https://doi.org/10.1016/j.cellsig.2021.110210
Roy M, Singh K, Shinde A, Singh J, Mane M, Bedekar S, Tailor Y, Gohel D, Vasiyani H, Currim F, Singh R. TNF-α-induced E3 ligase, TRIM15 inhibits TNF-α-regulated NF-κB pathway by promoting turnover of K63 linked ubiquitination of TAK1. Cell Signal. 2021 Dec 03;110210. doi: 10.1016/j.cellsig.2021.110210. Epub 2021 Dec 03. PMID: 34871740.
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