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Showing 205 to 211 of 211 entries
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Integration of spatial and single-cell transcriptomics localizes epithelial cell-immune cross-talk in kidney injury.

JCI insight

Melo Ferreira R, Sabo AR, Winfree S, Collins KS, Janosevic D, Gulbronson CJ, Cheng YH, Casbon L, Barwinska D, Ferkowicz MJ, Xuei X, Zhang C, Dunn KW, Kelly KJ, Sutton TA, Hato T, Dagher PC, El-Achkar TM, Eadon MT.
PMID: 34003797
JCI Insight. 2021 Jun 22;6(12). doi: 10.1172/jci.insight.147703.

Single-cell sequencing studies have characterized the transcriptomic signature of cell types within the kidney. However, the spatial distribution of acute kidney injury (AKI) is regional and affects cells heterogeneously. We first optimized coordination of spatial transcriptomics and single-nuclear sequencing...

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Melo Ferreira R, Sabo AR, Winfree S, et al. Integration of spatial and single-cell transcriptomics localizes epithelial cell-immune cross-talk in kidney injury. JCI Insight. 2021;6(12)doi: 10.1172/jci.insight.147703.
Melo Ferreira, R., Sabo, A. R., Winfree, S., Collins, K. S., Janosevic, D., Gulbronson, C. J., Cheng, Y. H., Casbon, L., Barwinska, D., Ferkowicz, M. J., Xuei, X., Zhang, C., Dunn, K. W., Kelly, K. J., Sutton, T. A., Hato, T., Dagher, P. C., El-Achkar, T. M., & Eadon, M. T. (2021). Integration of spatial and single-cell transcriptomics localizes epithelial cell-immune cross-talk in kidney injury. JCI insight, 6(12), . https://doi.org/10.1172/jci.insight.147703
Melo Ferreira, Ricardo, et al. "Integration of spatial and single-cell transcriptomics localizes epithelial cell-immune cross-talk in kidney injury." JCI insight vol. 6,12 (2021). doi: https://doi.org/10.1172/jci.insight.147703
Melo Ferreira R, Sabo AR, Winfree S, Collins KS, Janosevic D, Gulbronson CJ, Cheng YH, Casbon L, Barwinska D, Ferkowicz MJ, Xuei X, Zhang C, Dunn KW, Kelly KJ, Sutton TA, Hato T, Dagher PC, El-Achkar TM, Eadon MT. Integration of spatial and single-cell transcriptomics localizes epithelial cell-immune cross-talk in kidney injury. JCI Insight. 2021 Jun 22;6(12). doi: 10.1172/jci.insight.147703. PMID: 34003797; PMCID: PMC8262485.
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Generation and basic characterization of a gene-trap knockout mouse model of Scn2a with a substantial reduction of voltage-gated sodium channel Na.

Genes, brain, and behavior

Eaton M, Zhang J, Ma Z, Park AC, Lietzke E, Romero CM, Liu Y, Coleman ER, Chen X, Xiao T, Que Z, Lai S, Wu J, Lee JH, Palant S, Nguyen HP, Huang Z, Skarnes WC, Koss WA, Yang Y.
PMID: 33369088
Genes Brain Behav. 2021 Apr;20(4):e12725. doi: 10.1111/gbb.12725. Epub 2021 Jan 18.

Large-scale genetic studies revealed SCN2A as one of the most frequently mutated genes in patients with neurodevelopmental disorders. SCN2A encodes for the voltage-gated sodium channel isoform 1.2 (Na

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Eaton M, Zhang J, Ma Z, et al. Generation and basic characterization of a gene-trap knockout mouse model of Scn2a with a substantial reduction of voltage-gated sodium channel Na. Genes Brain Behav. 2021;20(4):e12725doi: 10.1111/gbb.12725.
Eaton, M., Zhang, J., Ma, Z., Park, A. C., Lietzke, E., Romero, C. M., Liu, Y., Coleman, E. R., Chen, X., Xiao, T., Que, Z., Lai, S., Wu, J., Lee, J. H., Palant, S., Nguyen, H. P., Huang, Z., Skarnes, W. C., Koss, W. A., & Yang, Y. (2021). Generation and basic characterization of a gene-trap knockout mouse model of Scn2a with a substantial reduction of voltage-gated sodium channel Na. Genes, brain, and behavior, 20(4), e12725. https://doi.org/10.1111/gbb.12725
Eaton, Muriel, et al. "Generation and basic characterization of a gene-trap knockout mouse model of Scn2a with a substantial reduction of voltage-gated sodium channel Na." Genes, brain, and behavior vol. 20,4 (2021): e12725. doi: https://doi.org/10.1111/gbb.12725
Eaton M, Zhang J, Ma Z, Park AC, Lietzke E, Romero CM, Liu Y, Coleman ER, Chen X, Xiao T, Que Z, Lai S, Wu J, Lee JH, Palant S, Nguyen HP, Huang Z, Skarnes WC, Koss WA, Yang Y. Generation and basic characterization of a gene-trap knockout mouse model of Scn2a with a substantial reduction of voltage-gated sodium channel Na. Genes Brain Behav. 2021 Apr;20(4):e12725. doi: 10.1111/gbb.12725. Epub 2021 Jan 18. PMID: 33369088.
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Clinical, histopathologic and molecular features of idiopathic and diabetic nodular mesangial sclerosis in humans.

Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association

Eadon MT, Lampe S, Baig MM, Collins KS, Melo Ferreira R, Mang H, Cheng YH, Barwinska D, El-Achkar TM, Schwantes-An TH, Winfree S, Temm CJ, Ferkowicz MJ, Dunn KW, Kelly KJ, Sutton TA, Moe SM, Moorthi RN, Phillips CL, Dagher PC.
PMID: 33537765
Nephrol Dial Transplant. 2021 Dec 31;37(1):72-84. doi: 10.1093/ndt/gfaa331.

BACKGROUND: Idiopathic nodular mesangial sclerosis, also called idiopathic nodular glomerulosclerosis (ING), is a rare clinical entity with an unclear pathogenesis. The hallmark of this disease is the presence of nodular mesangial sclerosis on histology without clinical evidence of diabetes...

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Eadon MT, Lampe S, Baig MM, et al. Clinical, histopathologic and molecular features of idiopathic and diabetic nodular mesangial sclerosis in humans. Nephrol Dial Transplant. 2021;37(1):72-84doi: 10.1093/ndt/gfaa331.
Eadon, M. T., Lampe, S., Baig, M. M., Collins, K. S., Melo Ferreira, R., Mang, H., Cheng, Y. H., Barwinska, D., El-Achkar, T. M., Schwantes-An, T. H., Winfree, S., Temm, C. J., Ferkowicz, M. J., Dunn, K. W., Kelly, K. J., Sutton, T. A., Moe, S. M., Moorthi, R. N., Phillips, C. L., Dagher, P. C. (2021). Clinical, histopathologic and molecular features of idiopathic and diabetic nodular mesangial sclerosis in humans. Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, 37(1), 72-84. https://doi.org/10.1093/ndt/gfaa331
Eadon, Michael T, et al. "Clinical, histopathologic and molecular features of idiopathic and diabetic nodular mesangial sclerosis in humans." Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association vol. 37,1 (2021): 72-84. doi: https://doi.org/10.1093/ndt/gfaa331
Eadon MT, Lampe S, Baig MM, Collins KS, Melo Ferreira R, Mang H, Cheng YH, Barwinska D, El-Achkar TM, Schwantes-An TH, Winfree S, Temm CJ, Ferkowicz MJ, Dunn KW, Kelly KJ, Sutton TA, Moe SM, Moorthi RN, Phillips CL, Dagher PC. Clinical, histopathologic and molecular features of idiopathic and diabetic nodular mesangial sclerosis in humans. Nephrol Dial Transplant. 2021 Dec 31;37(1):72-84. doi: 10.1093/ndt/gfaa331. PMID: 33537765.
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Decommissioning normal: COVID-19 as a disruptor of school norms for young people with learning disabilities.

British journal of learning disabilities

Beaton MC, Codina GN, Wharton JC.
PMID: 34220278
Br J Learn Disabil. 2021 Jun 02; doi: 10.1111/bld.12399. Epub 2021 Jun 02.

BACKGROUND: To slow the spread of COVID-19, on 20 March 2020, nurseries, schools and colleges across England were closed to all learners, apart from those who were children of key workers or were considered "vulnerable." As young people with...

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Beaton MC, Codina GN, Wharton JC. Decommissioning normal: COVID-19 as a disruptor of school norms for young people with learning disabilities. Br J Learn Disabil. 2021;doi: 10.1111/bld.12399.
Beaton, M. C., Codina, G. N., & Wharton, J. C. (2021). Decommissioning normal: COVID-19 as a disruptor of school norms for young people with learning disabilities. British journal of learning disabilities, . https://doi.org/10.1111/bld.12399
Beaton, Mhairi C, et al. "Decommissioning normal: COVID-19 as a disruptor of school norms for young people with learning disabilities." British journal of learning disabilities vol. (2021). doi: https://doi.org/10.1111/bld.12399
Beaton MC, Codina GN, Wharton JC. Decommissioning normal: COVID-19 as a disruptor of school norms for young people with learning disabilities. Br J Learn Disabil. 2021 Jun 02; doi: 10.1111/bld.12399. Epub 2021 Jun 02. PMID: 34220278; PMCID: PMC8239902.
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PROMISE I: Early feasibility study of the LimFlow System for percutaneous deep vein arterialization in no-option chronic limb-threatening ischemia: 12-month results.

Journal of vascular surgery

Clair DG, Mustapha JA, Shishehbor MH, Schneider PA, Henao S, Bernardo NN, Deaton DH.
PMID: 34019990
J Vasc Surg. 2021 Nov;74(5):1626-1635. doi: 10.1016/j.jvs.2021.04.057. Epub 2021 May 18.

OBJECTIVE: We report the 6- and 12-month outcomes of the PROMISE I early feasibility study after treatment of no-option chronic limb-threatening ischemia (CLTI) with percutaneous deep vein arterialization (pDVA) using the LimFlow System.METHODS: Thirty-two patients with no-option CLTI, previously...

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Clair DG, Mustapha JA, Shishehbor MH, et al. PROMISE I: Early feasibility study of the LimFlow System for percutaneous deep vein arterialization in no-option chronic limb-threatening ischemia: 12-month results. J Vasc Surg. 2021;74(5):1626-1635doi: 10.1016/j.jvs.2021.04.057.
Clair, D. G., Mustapha, J. A., Shishehbor, M. H., Schneider, P. A., Henao, S., Bernardo, N. N., & Deaton, D. H. (2021). PROMISE I: Early feasibility study of the LimFlow System for percutaneous deep vein arterialization in no-option chronic limb-threatening ischemia: 12-month results. Journal of vascular surgery, 74(5), 1626-1635. https://doi.org/10.1016/j.jvs.2021.04.057
Clair, Daniel G, et al. "PROMISE I: Early feasibility study of the LimFlow System for percutaneous deep vein arterialization in no-option chronic limb-threatening ischemia: 12-month results." Journal of vascular surgery vol. 74,5 (2021): 1626-1635. doi: https://doi.org/10.1016/j.jvs.2021.04.057
Clair DG, Mustapha JA, Shishehbor MH, Schneider PA, Henao S, Bernardo NN, Deaton DH. PROMISE I: Early feasibility study of the LimFlow System for percutaneous deep vein arterialization in no-option chronic limb-threatening ischemia: 12-month results. J Vasc Surg. 2021 Nov;74(5):1626-1635. doi: 10.1016/j.jvs.2021.04.057. Epub 2021 May 18. PMID: 34019990.
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L-DOPA Uptake in Astrocytic Endfeet Enwrapping Blood Vessels in Rat Brain.

Parkinson's disease

Inyushin MY, Huertas A, Kucheryavykh YV, Kucheryavykh LY, Tsydzik V, Sanabria P, Eaton MJ, Skatchkov SN, Rojas LV, Wessinger WD.
PMID: 22888467
Parkinsons Dis. 2012;2012:321406. doi: 10.1155/2012/321406. Epub 2012 Jul 24.

Astrocyte endfeet surround brain blood vessels and can play a role in the delivery of therapeutic drugs for Parkinson's disease. However, there is no previous evidence of the presence of LAT transporter for L-DOPA in brain astrocytes except in...

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Inyushin MY, Huertas A, Kucheryavykh YV, et al. L-DOPA Uptake in Astrocytic Endfeet Enwrapping Blood Vessels in Rat Brain. Parkinsons Dis. 2012;2012:321406doi: 10.1155/2012/321406.
Inyushin, M. Y., Huertas, A., Kucheryavykh, Y. V., Kucheryavykh, L. Y., Tsydzik, V., Sanabria, P., Eaton, M. J., Skatchkov, S. N., Rojas, L. V., & Wessinger, W. D. (2012). L-DOPA Uptake in Astrocytic Endfeet Enwrapping Blood Vessels in Rat Brain. Parkinson's disease, 2012321406. https://doi.org/10.1155/2012/321406
Inyushin, M Y, et al. "L-DOPA Uptake in Astrocytic Endfeet Enwrapping Blood Vessels in Rat Brain." Parkinson's disease vol. 2012 (2012): 321406. doi: https://doi.org/10.1155/2012/321406
Inyushin MY, Huertas A, Kucheryavykh YV, Kucheryavykh LY, Tsydzik V, Sanabria P, Eaton MJ, Skatchkov SN, Rojas LV, Wessinger WD. L-DOPA Uptake in Astrocytic Endfeet Enwrapping Blood Vessels in Rat Brain. Parkinsons Dis. 2012;2012:321406. doi: 10.1155/2012/321406. Epub 2012 Jul 24. PMID: 22888467; PMCID: PMC3409556.
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Hyperexcitability and Pharmacological Responsiveness of Cortical Neurons Derived from Human iPSCs Carrying Epilepsy-Associated Sodium Channel Nav1.2-L1342P Genetic Variant.

The Journal of neuroscience : the official journal of the Society for Neuroscience

Que Z, Olivero-Acosta MI, Zhang J, Eaton M, Tukker AM, Chen X, Wu J, Xie J, Xiao T, Wettschurack K, Yunis L, Shafer JM, Schaber JA, Rochet JC, Bowman AB, Yuan C, Huang Z, Hu CD, Trader DJ, Skarnes WC, Yang Y.
PMID: 34716231
J Neurosci. 2021 Dec 08;41(49):10194-10208. doi: 10.1523/JNEUROSCI.0564-21.2021. Epub 2021 Oct 29.

With the wide adoption of genomic sequencing in children having seizures, an increasing number of

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Que Z, Olivero-Acosta MI, Zhang J, et al. Hyperexcitability and Pharmacological Responsiveness of Cortical Neurons Derived from Human iPSCs Carrying Epilepsy-Associated Sodium Channel Nav1.2-L1342P Genetic Variant. J Neurosci. 2021;41(49):10194-10208doi: 10.1523/JNEUROSCI.0564-21.2021.
Que, Z., Olivero-Acosta, M. I., Zhang, J., Eaton, M., Tukker, A. M., Chen, X., Wu, J., Xie, J., Xiao, T., Wettschurack, K., Yunis, L., Shafer, J. M., Schaber, J. A., Rochet, J. C., Bowman, A. B., Yuan, C., Huang, Z., Hu, C. D., Trader, D. J., Skarnes, W. C., & Yang, Y. (2021). Hyperexcitability and Pharmacological Responsiveness of Cortical Neurons Derived from Human iPSCs Carrying Epilepsy-Associated Sodium Channel Nav1.2-L1342P Genetic Variant. The Journal of neuroscience : the official journal of the Society for Neuroscience, 41(49), 10194-10208. https://doi.org/10.1523/JNEUROSCI.0564-21.2021
Que, Zhefu, et al. "Hyperexcitability and Pharmacological Responsiveness of Cortical Neurons Derived from Human iPSCs Carrying Epilepsy-Associated Sodium Channel Nav1.2-L1342P Genetic Variant." The Journal of neuroscience : the official journal of the Society for Neuroscience vol. 41,49 (2021): 10194-10208. doi: https://doi.org/10.1523/JNEUROSCI.0564-21.2021
Que Z, Olivero-Acosta MI, Zhang J, Eaton M, Tukker AM, Chen X, Wu J, Xie J, Xiao T, Wettschurack K, Yunis L, Shafer JM, Schaber JA, Rochet JC, Bowman AB, Yuan C, Huang Z, Hu CD, Trader DJ, Skarnes WC, Yang Y. Hyperexcitability and Pharmacological Responsiveness of Cortical Neurons Derived from Human iPSCs Carrying Epilepsy-Associated Sodium Channel Nav1.2-L1342P Genetic Variant. J Neurosci. 2021 Dec 08;41(49):10194-10208. doi: 10.1523/JNEUROSCI.0564-21.2021. Epub 2021 Oct 29. PMID: 34716231; PMCID: PMC8660047.
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Showing 205 to 211 of 211 entries