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A Novel Intervention Including Individualized Nutritional Recommendations Reduces Hemoglobin A1c Level, Medication Use, and Weight in Type 2 Diabetes.

JMIR diabetes

McKenzie AL, Hallberg SJ, Creighton BC, Volk BM, Link TM, Abner MK, Glon RM, McCarter JP, Volek JS, Phinney SD.
PMID: 30291062
JMIR Diabetes. 2017 Mar 07;2(1):e5. doi: 10.2196/diabetes.6981.

BACKGROUND: Type 2 diabetes (T2D) is typically managed with a reduced fat diet plus glucose-lowering medications, the latter often promoting weight gain.OBJECTIVE: We evaluated whether individuals with T2D could be taught by either on-site group or remote means to...

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McKenzie AL, Hallberg SJ, Creighton BC, et al. A Novel Intervention Including Individualized Nutritional Recommendations Reduces Hemoglobin A1c Level, Medication Use, and Weight in Type 2 Diabetes. JMIR Diabetes. 2017;2(1):e5doi: 10.2196/diabetes.6981.
McKenzie, A. L., Hallberg, S. J., Creighton, B. C., Volk, B. M., Link, T. M., Abner, M. K., Glon, R. M., McCarter, J. P., Volek, J. S., & Phinney, S. D. (2017). A Novel Intervention Including Individualized Nutritional Recommendations Reduces Hemoglobin A1c Level, Medication Use, and Weight in Type 2 Diabetes. JMIR diabetes, 2(1), e5. https://doi.org/10.2196/diabetes.6981
McKenzie, Amy L, et al. "A Novel Intervention Including Individualized Nutritional Recommendations Reduces Hemoglobin A1c Level, Medication Use, and Weight in Type 2 Diabetes." JMIR diabetes vol. 2,1 (2017): e5. doi: https://doi.org/10.2196/diabetes.6981
McKenzie AL, Hallberg SJ, Creighton BC, Volk BM, Link TM, Abner MK, Glon RM, McCarter JP, Volek JS, Phinney SD. A Novel Intervention Including Individualized Nutritional Recommendations Reduces Hemoglobin A1c Level, Medication Use, and Weight in Type 2 Diabetes. JMIR Diabetes. 2017 Mar 07;2(1):e5. doi: 10.2196/diabetes.6981. PMID: 30291062; PMCID: PMC6238887.
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Fibroblast growth factor receptor signaling plays a key role in transformation induced by the TMPRSS2/ERG fusion gene and decreased PTEN.

Oncotarget

Shao L, Wang J, Karatas OF, Feng S, Zhang Y, Creighton CJ, Ittmann M.
PMID: 29581856
Oncotarget. 2018 Feb 12;9(18):14456-14471. doi: 10.18632/oncotarget.24470. eCollection 2018 Mar 06.

Prostate cancer is the most common visceral malignancy and the second leading cause of cancer deaths in US men. Correlative studies in human prostate cancers reveal a frequent association of the TMPRSS2/ERG (TE) fusion gene with loss of PTEN...

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Shao L, Wang J, Karatas OF, et al. Fibroblast growth factor receptor signaling plays a key role in transformation induced by the TMPRSS2/ERG fusion gene and decreased PTEN. Oncotarget. 2018;9(18):14456-14471doi: 10.18632/oncotarget.24470.
Shao, L., Wang, J., Karatas, O. F., Feng, S., Zhang, Y., Creighton, C. J., & Ittmann, M. (2018). Fibroblast growth factor receptor signaling plays a key role in transformation induced by the TMPRSS2/ERG fusion gene and decreased PTEN. Oncotarget, 9(18), 14456-14471. https://doi.org/10.18632/oncotarget.24470
Shao, Longjiang, et al. "Fibroblast growth factor receptor signaling plays a key role in transformation induced by the TMPRSS2/ERG fusion gene and decreased PTEN." Oncotarget vol. 9,18 (2018): 14456-14471. doi: https://doi.org/10.18632/oncotarget.24470
Shao L, Wang J, Karatas OF, Feng S, Zhang Y, Creighton CJ, Ittmann M. Fibroblast growth factor receptor signaling plays a key role in transformation induced by the TMPRSS2/ERG fusion gene and decreased PTEN. Oncotarget. 2018 Feb 12;9(18):14456-14471. doi: 10.18632/oncotarget.24470. eCollection 2018 Mar 06. PMID: 29581856; PMCID: PMC5865682.
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Macrophages promote the progression of premalignant mammary lesions to invasive cancer.

Oncotarget

Carron EC, Homra S, Rosenberg J, Coffelt SB, Kittrell F, Zhang Y, Creighton CJ, Fuqua SA, Medina D, Machado HL.
PMID: 28881599
Oncotarget. 2017 Jan 31;8(31):50731-50746. doi: 10.18632/oncotarget.14913. eCollection 2017 Aug 01.

Breast cancer initiation, progression and metastasis rely on a complex interplay between tumor cells and their surrounding microenvironment. Infiltrating immune cells, including macrophages, promote mammary tumor progression and metastasis; however, less is known about the role of macrophages in...

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Carron EC, Homra S, Rosenberg J, et al. Macrophages promote the progression of premalignant mammary lesions to invasive cancer. Oncotarget. 2017;8(31):50731-50746doi: 10.18632/oncotarget.14913.
Carron, E. C., Homra, S., Rosenberg, J., Coffelt, S. B., Kittrell, F., Zhang, Y., Creighton, C. J., Fuqua, S. A., Medina, D., & Machado, H. L. (2017). Macrophages promote the progression of premalignant mammary lesions to invasive cancer. Oncotarget, 8(31), 50731-50746. https://doi.org/10.18632/oncotarget.14913
Carron, Emily C, et al. "Macrophages promote the progression of premalignant mammary lesions to invasive cancer." Oncotarget vol. 8,31 (2017): 50731-50746. doi: https://doi.org/10.18632/oncotarget.14913
Carron EC, Homra S, Rosenberg J, Coffelt SB, Kittrell F, Zhang Y, Creighton CJ, Fuqua SA, Medina D, Machado HL. Macrophages promote the progression of premalignant mammary lesions to invasive cancer. Oncotarget. 2017 Jan 31;8(31):50731-50746. doi: 10.18632/oncotarget.14913. eCollection 2017 Aug 01. PMID: 28881599; PMCID: PMC5584199.
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A CTC-Cluster-Specific Signature Derived from OMICS Analysis of Patient-Derived Xenograft Tumors Predicts Outcomes in Basal-Like Breast Cancer.

Journal of clinical medicine

Thangavel H, De Angelis C, Vasaikar S, Bhat R, Jolly MK, Nagi C, Creighton CJ, Chen F, Dobrolecki LE, George JT, Kumar T, Abdulkareem NM, Mao S, Nardone A, Rimawi M, Osborne CK, Lewis MT, Levine H, Zhang B, Schiff R, Giuliano M, Trivedi MV.
PMID: 31652963
J Clin Med. 2019 Oct 24;8(11). doi: 10.3390/jcm8111772.

Circulating tumor cell clusters (CTCcl) have a higher metastatic potential compared to single CTCs and predict long-term outcomes in breast cancer (BC) patients. Because of the rarity of CTCcls, molecular characterization of primary tumors that give rise to CTCcl...

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Thangavel H, De Angelis C, Vasaikar S, et al. A CTC-Cluster-Specific Signature Derived from OMICS Analysis of Patient-Derived Xenograft Tumors Predicts Outcomes in Basal-Like Breast Cancer. J Clin Med. 2019;8(11)doi: 10.3390/jcm8111772.
Thangavel, H., De Angelis, C., Vasaikar, S., Bhat, R., Jolly, M. K., Nagi, C., Creighton, C. J., Chen, F., Dobrolecki, L. E., George, J. T., Kumar, T., Abdulkareem, N. M., Mao, S., Nardone, A., Rimawi, M., Osborne, C. K., Lewis, M. T., Levine, H., Zhang, B., Schiff, R., Giuliano, M., & Trivedi, M. V. (2019). A CTC-Cluster-Specific Signature Derived from OMICS Analysis of Patient-Derived Xenograft Tumors Predicts Outcomes in Basal-Like Breast Cancer. Journal of clinical medicine, 8(11), . https://doi.org/10.3390/jcm8111772
Thangavel, Hariprasad, et al. "A CTC-Cluster-Specific Signature Derived from OMICS Analysis of Patient-Derived Xenograft Tumors Predicts Outcomes in Basal-Like Breast Cancer." Journal of clinical medicine vol. 8,11 (2019). doi: https://doi.org/10.3390/jcm8111772
Thangavel H, De Angelis C, Vasaikar S, Bhat R, Jolly MK, Nagi C, Creighton CJ, Chen F, Dobrolecki LE, George JT, Kumar T, Abdulkareem NM, Mao S, Nardone A, Rimawi M, Osborne CK, Lewis MT, Levine H, Zhang B, Schiff R, Giuliano M, Trivedi MV. A CTC-Cluster-Specific Signature Derived from OMICS Analysis of Patient-Derived Xenograft Tumors Predicts Outcomes in Basal-Like Breast Cancer. J Clin Med. 2019 Oct 24;8(11). doi: 10.3390/jcm8111772. PMID: 31652963; PMCID: PMC6912280.
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Elevated expression of the colony-stimulating factor 1 (CSF1) induces prostatic intraepithelial neoplasia dependent of epithelial-Gp130.

Oncogene

Kwon OJ, Zhang B, Jia D, Zhang L, Wei X, Zhou Z, Liu D, Huynh KT, Zhang K, Zhang Y, Labhart P, Sboner A, Barbieri C, Haffner MC, Creighton CJ, Xin L.
PMID: 34999736
Oncogene. 2022 Jan 08; doi: 10.1038/s41388-021-02169-7. Epub 2022 Jan 08.

Macrophages are increased in human benign prostatic hyperplasia and prostate cancer. We generate a Pb-Csf1 mouse model with prostate-specific overexpression of macrophage colony-stimulating factor (M-Csf/Csf1). Csf1 overexpression promotes immune cell infiltration into the prostate, modulates the macrophage polarity in...

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Kwon OJ, Zhang B, Jia D, et al. Elevated expression of the colony-stimulating factor 1 (CSF1) induces prostatic intraepithelial neoplasia dependent of epithelial-Gp130. Oncogene. 2022;doi: 10.1038/s41388-021-02169-7.
Kwon, O. J., Zhang, B., Jia, D., Zhang, L., Wei, X., Zhou, Z., Liu, D., Huynh, K. T., Zhang, K., Zhang, Y., Labhart, P., Sboner, A., Barbieri, C., Haffner, M. C., Creighton, C. J., & Xin, L. (2022). Elevated expression of the colony-stimulating factor 1 (CSF1) induces prostatic intraepithelial neoplasia dependent of epithelial-Gp130. Oncogene, . https://doi.org/10.1038/s41388-021-02169-7
Kwon, Oh-Joon, et al. "Elevated expression of the colony-stimulating factor 1 (CSF1) induces prostatic intraepithelial neoplasia dependent of epithelial-Gp130." Oncogene vol. (2022). doi: https://doi.org/10.1038/s41388-021-02169-7
Kwon OJ, Zhang B, Jia D, Zhang L, Wei X, Zhou Z, Liu D, Huynh KT, Zhang K, Zhang Y, Labhart P, Sboner A, Barbieri C, Haffner MC, Creighton CJ, Xin L. Elevated expression of the colony-stimulating factor 1 (CSF1) induces prostatic intraepithelial neoplasia dependent of epithelial-Gp130. Oncogene. 2022 Jan 08; doi: 10.1038/s41388-021-02169-7. Epub 2022 Jan 08. PMID: 34999736.
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Teprotumumab and Hearing Loss: Case Series and Proposal for Audiologic Monitoring.

Ophthalmic plastic and reconstructive surgery

Belinsky I, Creighton FX, Mahoney N, Petris CK, Callahan AB, Campbell AA, Kazim M, Lee HBH, Yoon MK, Dagi Glass LR.
PMID: 34085994
Ophthalmic Plast Reconstr Surg. 2021 Jun 04; doi: 10.1097/IOP.0000000000001995. Epub 2021 Jun 04.

PURPOSE: To present a protocol for audiologic monitoring in the setting of teprotumumab treatment of thyroid eye disease, motivated by 4 cases of significant hearing loss, and review the relevant literature.METHODS: Cases of hearing loss in the setting of...

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Belinsky I, Creighton FX, Mahoney N, et al. Teprotumumab and Hearing Loss: Case Series and Proposal for Audiologic Monitoring. Ophthalmic Plast Reconstr Surg. 2021;doi: 10.1097/IOP.0000000000001995.
Belinsky, I., Creighton, F. X., Mahoney, N., Petris, C. K., Callahan, A. B., Campbell, A. A., Kazim, M., Lee, H. B. H., Yoon, M. K., & Dagi Glass, L. R. (2021). Teprotumumab and Hearing Loss: Case Series and Proposal for Audiologic Monitoring. Ophthalmic plastic and reconstructive surgery, . https://doi.org/10.1097/IOP.0000000000001995
Belinsky, Irina, et al. "Teprotumumab and Hearing Loss: Case Series and Proposal for Audiologic Monitoring." Ophthalmic plastic and reconstructive surgery vol. (2021). doi: https://doi.org/10.1097/IOP.0000000000001995
Belinsky I, Creighton FX, Mahoney N, Petris CK, Callahan AB, Campbell AA, Kazim M, Lee HBH, Yoon MK, Dagi Glass LR. Teprotumumab and Hearing Loss: Case Series and Proposal for Audiologic Monitoring. Ophthalmic Plast Reconstr Surg. 2021 Jun 04; doi: 10.1097/IOP.0000000000001995. Epub 2021 Jun 04. PMID: 34085994.
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A Wnt-Independent LGR4-EGFR Signaling Axis in Cancer Metastasis.

Cancer research

Yue F, Jiang W, Ku AT, Young AIJ, Zhang W, Souto EP, Gao Y, Yu Z, Wang Y, Creighton CJ, Nagi C, Wang T, Hilsenbeck SG, Feng XH, Huang S, Coarfa C, Zhang XH, Liu Q, Lin X, Li Y.
PMID: 34099494
Cancer Res. 2021 Sep 01;81(17):4441-4454. doi: 10.1158/0008-5472.CAN-21-1112. Epub 2021 Jun 07.

Leucine-rich repeat-containing G protein-coupled receptors 4, 5, and 6 (LGR4/5/6) play critical roles in development and cancer. The widely accepted mechanism is that these proteins, together with their R-spondin ligands, stabilize Wnt receptors, thus potentiating Wnt signaling. Here we...

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Yue F, Jiang W, Ku AT, et al. A Wnt-Independent LGR4-EGFR Signaling Axis in Cancer Metastasis. Cancer Res. 2021;81(17):4441-4454doi: 10.1158/0008-5472.CAN-21-1112.
Yue, F., Jiang, W., Ku, A. T., Young, A. I. J., Zhang, W., Souto, E. P., Gao, Y., Yu, Z., Wang, Y., Creighton, C. J., Nagi, C., Wang, T., Hilsenbeck, S. G., Feng, X. H., Huang, S., Coarfa, C., Zhang, X. H., Liu, Q., Lin, X., & Li, Y. (2021). A Wnt-Independent LGR4-EGFR Signaling Axis in Cancer Metastasis. Cancer research, 81(17), 4441-4454. https://doi.org/10.1158/0008-5472.CAN-21-1112
Yue, Fei, et al. "A Wnt-Independent LGR4-EGFR Signaling Axis in Cancer Metastasis." Cancer research vol. 81,17 (2021): 4441-4454. doi: https://doi.org/10.1158/0008-5472.CAN-21-1112
Yue F, Jiang W, Ku AT, Young AIJ, Zhang W, Souto EP, Gao Y, Yu Z, Wang Y, Creighton CJ, Nagi C, Wang T, Hilsenbeck SG, Feng XH, Huang S, Coarfa C, Zhang XH, Liu Q, Lin X, Li Y. A Wnt-Independent LGR4-EGFR Signaling Axis in Cancer Metastasis. Cancer Res. 2021 Sep 01;81(17):4441-4454. doi: 10.1158/0008-5472.CAN-21-1112. Epub 2021 Jun 07. PMID: 34099494; PMCID: PMC8416783.
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Automated Registration-Based Temporal Bone Computed Tomography Segmentation for Applications in Neurotologic Surgery.

Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery

Ding AS, Lu A, Li Z, Galaiya D, Siewerdsen JH, Taylor RH, Creighton FX.
PMID: 34491849
Otolaryngol Head Neck Surg. 2021 Sep 07;1945998211044982. doi: 10.1177/01945998211044982. Epub 2021 Sep 07.

OBJECTIVE: This study investigates the accuracy of an automated method to rapidly segment relevant temporal bone anatomy from cone beam computed tomography (CT) images. Implementation of this segmentation pipeline has potential to improve surgical safety and decrease operative time...

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Ding AS, Lu A, Li Z, et al. Automated Registration-Based Temporal Bone Computed Tomography Segmentation for Applications in Neurotologic Surgery. Otolaryngol Head Neck Surg. 2021;1945998211044982doi: 10.1177/01945998211044982.
Ding, A. S., Lu, A., Li, Z., Galaiya, D., Siewerdsen, J. H., Taylor, R. H., & Creighton, F. X. (2021). Automated Registration-Based Temporal Bone Computed Tomography Segmentation for Applications in Neurotologic Surgery. Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery, 1945998211044982. https://doi.org/10.1177/01945998211044982
Ding, Andy S, et al. "Automated Registration-Based Temporal Bone Computed Tomography Segmentation for Applications in Neurotologic Surgery." Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery vol. (2021): 1945998211044982. doi: https://doi.org/10.1177/01945998211044982
Ding AS, Lu A, Li Z, Galaiya D, Siewerdsen JH, Taylor RH, Creighton FX. Automated Registration-Based Temporal Bone Computed Tomography Segmentation for Applications in Neurotologic Surgery. Otolaryngol Head Neck Surg. 2021 Sep 07;1945998211044982. doi: 10.1177/01945998211044982. Epub 2021 Sep 07. PMID: 34491849.
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Global molecular alterations involving recurrence or progression of pediatric brain tumors.

Neoplasia (New York, N.Y.)

Chen F, Chandrashekar DS, Scheurer ME, Varambally S, Creighton CJ.
PMID: 34864569
Neoplasia. 2021 Dec 02;24(1):22-33. doi: 10.1016/j.neo.2021.11.014. Epub 2021 Dec 02.

BACKGROUND: We aimed to identify molecular changes in recurrent or progressive pediatric brain tumors, as compared to the corresponding initial tumors from the same patients, using genomic, transcriptomic, and proteomic data from a unique and large cohort of 55...

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Chen F, Chandrashekar DS, Scheurer ME, et al. Global molecular alterations involving recurrence or progression of pediatric brain tumors. Neoplasia. 2021;24(1):22-33doi: 10.1016/j.neo.2021.11.014.
Chen, F., Chandrashekar, D. S., Scheurer, M. E., Varambally, S., & Creighton, C. J. (2021). Global molecular alterations involving recurrence or progression of pediatric brain tumors. Neoplasia (New York, N.Y.), 24(1), 22-33. https://doi.org/10.1016/j.neo.2021.11.014
Chen, Fengju, et al. "Global molecular alterations involving recurrence or progression of pediatric brain tumors." Neoplasia (New York, N.Y.) vol. 24,1 (2021): 22-33. doi: https://doi.org/10.1016/j.neo.2021.11.014
Chen F, Chandrashekar DS, Scheurer ME, Varambally S, Creighton CJ. Global molecular alterations involving recurrence or progression of pediatric brain tumors. Neoplasia. 2021 Dec 02;24(1):22-33. doi: 10.1016/j.neo.2021.11.014. Epub 2021 Dec 02. PMID: 34864569.
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Global molecular alterations involving recurrence or progression of pediatric brain tumors.

Neoplasia (New York, N.Y.)

Chen F, Chandrashekar DS, Scheurer ME, Varambally S, Creighton CJ.
PMID: 34864569
Neoplasia. 2021 Dec 02;24(1):22-33. doi: 10.1016/j.neo.2021.11.014. Epub 2021 Dec 02.

BACKGROUND: We aimed to identify molecular changes in recurrent or progressive pediatric brain tumors, as compared to the corresponding initial tumors from the same patients, using genomic, transcriptomic, and proteomic data from a unique and large cohort of 55...

Cite
Chen F, Chandrashekar DS, Scheurer ME, et al. Global molecular alterations involving recurrence or progression of pediatric brain tumors. Neoplasia. 2021;24(1):22-33doi: 10.1016/j.neo.2021.11.014.
Chen, F., Chandrashekar, D. S., Scheurer, M. E., Varambally, S., & Creighton, C. J. (2021). Global molecular alterations involving recurrence or progression of pediatric brain tumors. Neoplasia (New York, N.Y.), 24(1), 22-33. https://doi.org/10.1016/j.neo.2021.11.014
Chen, Fengju, et al. "Global molecular alterations involving recurrence or progression of pediatric brain tumors." Neoplasia (New York, N.Y.) vol. 24,1 (2021): 22-33. doi: https://doi.org/10.1016/j.neo.2021.11.014
Chen F, Chandrashekar DS, Scheurer ME, Varambally S, Creighton CJ. Global molecular alterations involving recurrence or progression of pediatric brain tumors. Neoplasia. 2021 Dec 02;24(1):22-33. doi: 10.1016/j.neo.2021.11.014. Epub 2021 Dec 02. PMID: 34864569.
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Addiction to Golgi-resident PI4P synthesis in chromosome 1q21.3-amplified lung adenocarcinoma cells.

Proceedings of the National Academy of Sciences of the United States of America

Shi L, Tan X, Liu X, Yu J, Bota-Rabassedas N, Niu Y, Luo J, Xi Y, Zong C, Creighton CJ, Glenn JS, Wang J, Kurie JM.
PMID: 34155143
Proc Natl Acad Sci U S A. 2021 Jun 22;118(25). doi: 10.1073/pnas.2023537118.

A chromosome 1q21.3 region that is frequently amplified in diverse cancer types encodes phosphatidylinositol (PI)-4 kinase IIIβ (PI4KIIIβ), a key regulator of secretory vesicle biogenesis and trafficking. Chromosome 1q21.3-amplified lung adenocarcinoma (1q-LUAD) cells rely on PI4KIIIβ for Golgi-resident PI-4-phosphate...

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Shi L, Tan X, Liu X, et al. Addiction to Golgi-resident PI4P synthesis in chromosome 1q21.3-amplified lung adenocarcinoma cells. Proc Natl Acad Sci U S A. 2021;118(25)doi: 10.1073/pnas.2023537118.
Shi, L., Tan, X., Liu, X., Yu, J., Bota-Rabassedas, N., Niu, Y., Luo, J., Xi, Y., Zong, C., Creighton, C. J., Glenn, J. S., Wang, J., & Kurie, J. M. (2021). Addiction to Golgi-resident PI4P synthesis in chromosome 1q21.3-amplified lung adenocarcinoma cells. Proceedings of the National Academy of Sciences of the United States of America, 118(25), . https://doi.org/10.1073/pnas.2023537118
Shi, Lei, et al. "Addiction to Golgi-resident PI4P synthesis in chromosome 1q21.3-amplified lung adenocarcinoma cells." Proceedings of the National Academy of Sciences of the United States of America vol. 118,25 (2021). doi: https://doi.org/10.1073/pnas.2023537118
Shi L, Tan X, Liu X, Yu J, Bota-Rabassedas N, Niu Y, Luo J, Xi Y, Zong C, Creighton CJ, Glenn JS, Wang J, Kurie JM. Addiction to Golgi-resident PI4P synthesis in chromosome 1q21.3-amplified lung adenocarcinoma cells. Proc Natl Acad Sci U S A. 2021 Jun 22;118(25). doi: 10.1073/pnas.2023537118. PMID: 34155143; PMCID: PMC8237628.
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Systematic identification of non-coding somatic single nucleotide variants associated with altered transcription and DNA methylation in adult and pediatric cancers.

NAR cancer

Chen F, Zhang Y, Creighton CJ.
PMID: 33554123
NAR Cancer. 2021 Feb 01;3(1):zcab001. doi: 10.1093/narcan/zcab001. eCollection 2021 Mar.

Whole-genome sequencing combined with transcriptomics can reveal impactful non-coding single nucleotide variants (SNVs) in cancer. Here, we developed an integrative analytical approach that, as a first step, identifies genes altered in expression or DNA methylation in association with nearby...

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Chen F, Zhang Y, Creighton CJ. Systematic identification of non-coding somatic single nucleotide variants associated with altered transcription and DNA methylation in adult and pediatric cancers. NAR Cancer. 2021;3(1):zcab001doi: 10.1093/narcan/zcab001.
Chen, F., Zhang, Y., & Creighton, C. J. (2021). Systematic identification of non-coding somatic single nucleotide variants associated with altered transcription and DNA methylation in adult and pediatric cancers. NAR cancer, 3(1), zcab001. https://doi.org/10.1093/narcan/zcab001
Chen, Fengju, et al. "Systematic identification of non-coding somatic single nucleotide variants associated with altered transcription and DNA methylation in adult and pediatric cancers." NAR cancer vol. 3,1 (2021): zcab001. doi: https://doi.org/10.1093/narcan/zcab001
Chen F, Zhang Y, Creighton CJ. Systematic identification of non-coding somatic single nucleotide variants associated with altered transcription and DNA methylation in adult and pediatric cancers. NAR Cancer. 2021 Feb 01;3(1):zcab001. doi: 10.1093/narcan/zcab001. eCollection 2021 Mar. PMID: 33554123; PMCID: PMC7849833.
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