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A Sensitization-Free Dimethyl Fumarate Prodrug, Isosorbide Di-(Methyl Fumarate), Provides a Topical Treatment Candidate for Psoriasis.

JID innovations

Bojanowski K, Ibeji CU, Singh P, Swindell WR, Chaudhuri RK.
PMID: 34909741
JID Innov. 2021 Jul 08;1(4):100040. doi: 10.1016/j.xjidi.2021.100040. eCollection 2021 Dec.

Dimethyl fumarate (DMF) is an effective oral treatment for psoriasis administered in Europe for nearly 60 years. However, its potential has been limited by contact dermatitis that prohibits topical application. This paper characterizes a DMF derivative, isosorbide DMF (IDMF),...

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Bojanowski K, Ibeji CU, Singh P, et al. A Sensitization-Free Dimethyl Fumarate Prodrug, Isosorbide Di-(Methyl Fumarate), Provides a Topical Treatment Candidate for Psoriasis. JID Innov. 2021;1(4):100040doi: 10.1016/j.xjidi.2021.100040.
Bojanowski, K., Ibeji, C. U., Singh, P., Swindell, W. R., & Chaudhuri, R. K. (2021). A Sensitization-Free Dimethyl Fumarate Prodrug, Isosorbide Di-(Methyl Fumarate), Provides a Topical Treatment Candidate for Psoriasis. JID innovations, 1(4), 100040. https://doi.org/10.1016/j.xjidi.2021.100040
Bojanowski, Krzysztof, et al. "A Sensitization-Free Dimethyl Fumarate Prodrug, Isosorbide Di-(Methyl Fumarate), Provides a Topical Treatment Candidate for Psoriasis." JID innovations vol. 1,4 (2021): 100040. doi: https://doi.org/10.1016/j.xjidi.2021.100040
Bojanowski K, Ibeji CU, Singh P, Swindell WR, Chaudhuri RK. A Sensitization-Free Dimethyl Fumarate Prodrug, Isosorbide Di-(Methyl Fumarate), Provides a Topical Treatment Candidate for Psoriasis. JID Innov. 2021 Jul 08;1(4):100040. doi: 10.1016/j.xjidi.2021.100040. eCollection 2021 Dec. PMID: 34909741; PMCID: PMC8659395.
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Hyperglycemia Induces Trained Immunity in Macrophages and Their Precursors and Promotes Atherosclerosis.

Circulation

Edgar L, Akbar N, Braithwaite AT, Krausgruber T, Gallart-Ayala H, Bailey J, Corbin AL, Khoyratty TE, Chai JT, Alkhalil M, Rendeiro AF, Ziberna K, Arya R, Cahill TJ, Bock C, Laurencikiene J, Crabtree MJ, Lemieux ME, Riksen NP, Netea MG, Wheelock CE, Channon KM, Rydén M, Udalova IA, Carnicer R, Choudhury RP.
PMID: 34255973
Circulation. 2021 Sep 21;144(12):961-982. doi: 10.1161/CIRCULATIONAHA.120.046464. Epub 2021 Jul 13.

BACKGROUND: Cardiovascular risk in diabetes remains elevated despite glucose-lowering therapies. We hypothesized that hyperglycemia induces trained immunity in macrophages, promoting persistent proatherogenic characteristics.METHODS: Bone marrow-derived macrophages from control mice and mice with diabetes were grown in physiological glucose (5...

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Edgar L, Akbar N, Braithwaite AT, et al. Hyperglycemia Induces Trained Immunity in Macrophages and Their Precursors and Promotes Atherosclerosis. Circulation. 2021;144(12):961-982doi: 10.1161/CIRCULATIONAHA.120.046464.
Edgar, L., Akbar, N., Braithwaite, A. T., Krausgruber, T., Gallart-Ayala, H., Bailey, J., Corbin, A. L., Khoyratty, T. E., Chai, J. T., Alkhalil, M., Rendeiro, A. F., Ziberna, K., Arya, R., Cahill, T. J., Bock, C., Laurencikiene, J., Crabtree, M. J., Lemieux, M. E., Riksen, N. P., Netea, M. G., Wheelock, C. E., Channon, K. M., Rydén, M., Udalova, I. A., Carnicer, R., & Choudhury, R. P. (2021). Hyperglycemia Induces Trained Immunity in Macrophages and Their Precursors and Promotes Atherosclerosis. Circulation, 144(12), 961-982. https://doi.org/10.1161/CIRCULATIONAHA.120.046464
Edgar, Laurienne, et al. "Hyperglycemia Induces Trained Immunity in Macrophages and Their Precursors and Promotes Atherosclerosis." Circulation vol. 144,12 (2021): 961-982. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.046464
Edgar L, Akbar N, Braithwaite AT, Krausgruber T, Gallart-Ayala H, Bailey J, Corbin AL, Khoyratty TE, Chai JT, Alkhalil M, Rendeiro AF, Ziberna K, Arya R, Cahill TJ, Bock C, Laurencikiene J, Crabtree MJ, Lemieux ME, Riksen NP, Netea MG, Wheelock CE, Channon KM, Rydén M, Udalova IA, Carnicer R, Choudhury RP. Hyperglycemia Induces Trained Immunity in Macrophages and Their Precursors and Promotes Atherosclerosis. Circulation. 2021 Sep 21;144(12):961-982. doi: 10.1161/CIRCULATIONAHA.120.046464. Epub 2021 Jul 13. PMID: 34255973; PMCID: PMC8448412.
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Efficacy of mupirocin, neomycin and octenidine for nasal Staphylococcus aureus decolonisation: a retrospective cohort study.

Antimicrobial resistance and infection control

Allport J, Choudhury R, Bruce-Wootton P, Reed M, Tate D, Malviya A.
PMID: 35012641
Antimicrob Resist Infect Control. 2022 Jan 10;11(1):5. doi: 10.1186/s13756-021-01043-1.

BACKGROUND: Periprosthetic joint infection (PJI) causes significant morbidity. Methicillin sensitive Staphylococcus aureus (MSSA) is the most frequent organism, and the majority are endogenous. Decolonisation reduces PJIs but there is a paucity of evidence comparing treatments. Aims; compare 3 nasal...

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Allport J, Choudhury R, Bruce-Wootton P, et al. Efficacy of mupirocin, neomycin and octenidine for nasal Staphylococcus aureus decolonisation: a retrospective cohort study. Antimicrob Resist Infect Control. 2022;11(1):5doi: 10.1186/s13756-021-01043-1.
Allport, J., Choudhury, R., Bruce-Wootton, P., Reed, M., Tate, D., & Malviya, A. (2022). Efficacy of mupirocin, neomycin and octenidine for nasal Staphylococcus aureus decolonisation: a retrospective cohort study. Antimicrobial resistance and infection control, 11(1), 5. https://doi.org/10.1186/s13756-021-01043-1
Allport, J, et al. "Efficacy of mupirocin, neomycin and octenidine for nasal Staphylococcus aureus decolonisation: a retrospective cohort study." Antimicrobial resistance and infection control vol. 11,1 (2022): 5. doi: https://doi.org/10.1186/s13756-021-01043-1
Allport J, Choudhury R, Bruce-Wootton P, Reed M, Tate D, Malviya A. Efficacy of mupirocin, neomycin and octenidine for nasal Staphylococcus aureus decolonisation: a retrospective cohort study. Antimicrob Resist Infect Control. 2022 Jan 10;11(1):5. doi: 10.1186/s13756-021-01043-1. PMID: 35012641; PMCID: PMC8744346.
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Showing 661 to 663 of 663 entries