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Showing 205 to 211 of 211 entries
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Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1.

Pharmacological research

Eid BG, Alhakamy NA, Fahmy UA, Ahmed OAA, Md S, Abdel-Naim AB, Caruso G, Caraci F.
PMID: 34801680
Pharmacol Res. 2021 Nov 19;175:105993. doi: 10.1016/j.phrs.2021.105993. Epub 2021 Nov 19.

A dysregulation of the wound healing process can lead to the development of various intractable ulcers or excessive scar formation. Therefore it is essential to identify novel pharmacological strategies to promote wound healing and restore the mechanical integrity of...

Cite
Eid BG, Alhakamy NA, Fahmy UA, et al. Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1. Pharmacol Res. 2021;175:105993doi: 10.1016/j.phrs.2021.105993.
Eid, B. G., Alhakamy, N. A., Fahmy, U. A., Ahmed, O. A. A., Md, S., Abdel-Naim, A. B., Caruso, G., & Caraci, F. (2021). Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1. Pharmacological research, 175105993. https://doi.org/10.1016/j.phrs.2021.105993
Eid, Basma G, et al. "Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1." Pharmacological research vol. 175 (2021): 105993. doi: https://doi.org/10.1016/j.phrs.2021.105993
Eid BG, Alhakamy NA, Fahmy UA, Ahmed OAA, Md S, Abdel-Naim AB, Caruso G, Caraci F. Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1. Pharmacol Res. 2021 Nov 19;175:105993. doi: 10.1016/j.phrs.2021.105993. Epub 2021 Nov 19. PMID: 34801680.
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The Multimodal MOPr/DOPr Agonist LP2 Reduces Allodynia in Chronic Constriction Injured Rats by Rescue of TGF-β1 Signalling.

Frontiers in pharmacology

Fidilio A, Grasso M, Turnaturi R, Caruso G, Spitale FM, Vicario N, Parenti R, Spoto S, Musso N, Marrazzo A, Chiechio S, Caraci F, Pasquinucci L, Parenti C.
PMID: 34690781
Front Pharmacol. 2021 Oct 06;12:749365. doi: 10.3389/fphar.2021.749365. eCollection 2021.

Neuropathic pain is one of the most disabling forms of chronic pain and it is characterized by hyperalgesia and allodynia linked to an aberrant processing of pain transmission and to neuroinflammation. Transforming growth factor-β1 (TGF-β1) is an anti-inflammatory cytokine,...

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Fidilio A, Grasso M, Turnaturi R, et al. The Multimodal MOPr/DOPr Agonist LP2 Reduces Allodynia in Chronic Constriction Injured Rats by Rescue of TGF-β1 Signalling. Front Pharmacol. 2021;12:749365doi: 10.3389/fphar.2021.749365.
Fidilio, A., Grasso, M., Turnaturi, R., Caruso, G., Spitale, F. M., Vicario, N., Parenti, R., Spoto, S., Musso, N., Marrazzo, A., Chiechio, S., Caraci, F., Pasquinucci, L., & Parenti, C. (2021). The Multimodal MOPr/DOPr Agonist LP2 Reduces Allodynia in Chronic Constriction Injured Rats by Rescue of TGF-β1 Signalling. Frontiers in pharmacology, 12749365. https://doi.org/10.3389/fphar.2021.749365
Fidilio, Annamaria, et al. "The Multimodal MOPr/DOPr Agonist LP2 Reduces Allodynia in Chronic Constriction Injured Rats by Rescue of TGF-β1 Signalling." Frontiers in pharmacology vol. 12 (2021): 749365. doi: https://doi.org/10.3389/fphar.2021.749365
Fidilio A, Grasso M, Turnaturi R, Caruso G, Spitale FM, Vicario N, Parenti R, Spoto S, Musso N, Marrazzo A, Chiechio S, Caraci F, Pasquinucci L, Parenti C. The Multimodal MOPr/DOPr Agonist LP2 Reduces Allodynia in Chronic Constriction Injured Rats by Rescue of TGF-β1 Signalling. Front Pharmacol. 2021 Oct 06;12:749365. doi: 10.3389/fphar.2021.749365. eCollection 2021. PMID: 34690781; PMCID: PMC8526862.
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Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1.

Pharmacological research

Eid BG, Alhakamy NA, Fahmy UA, Ahmed OAA, Md S, Abdel-Naim AB, Caruso G, Caraci F.
PMID: 34801680
Pharmacol Res. 2022 Jan;175:105993. doi: 10.1016/j.phrs.2021.105993. Epub 2021 Nov 19.

A dysregulation of the wound healing process can lead to the development of various intractable ulcers or excessive scar formation. Therefore it is essential to identify novel pharmacological strategies to promote wound healing and restore the mechanical integrity of...

Cite
Eid BG, Alhakamy NA, Fahmy UA, et al. Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1. Pharmacol Res. 2021;175:105993doi: 10.1016/j.phrs.2021.105993.
Eid, B. G., Alhakamy, N. A., Fahmy, U. A., Ahmed, O. A. A., Md, S., Abdel-Naim, A. B., Caruso, G., & Caraci, F. (2022). Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1. Pharmacological research, 175105993. https://doi.org/10.1016/j.phrs.2021.105993
Eid, Basma G, et al. "Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1." Pharmacological research vol. 175 (2022): 105993. doi: https://doi.org/10.1016/j.phrs.2021.105993
Eid BG, Alhakamy NA, Fahmy UA, Ahmed OAA, Md S, Abdel-Naim AB, Caruso G, Caraci F. Melittin and diclofenac synergistically promote wound healing in a pathway involving TGF-β1. Pharmacol Res. 2022 Jan;175:105993. doi: 10.1016/j.phrs.2021.105993. Epub 2021 Nov 19. PMID: 34801680.
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Vitamin K - sources, physiological role, kinetics, deficiency, detection, therapeutic use, and toxicity.

Nutrition reviews

Mladěnka P, Macáková K, Kujovská Krčmová L, Javorská L, Mrštná K, Carazo A, Protti M, Remião F, Nováková L.
PMID: 34472618
Nutr Rev. 2021 Sep 02; doi: 10.1093/nutrit/nuab061. Epub 2021 Sep 02.

Vitamin K is traditionally connected with blood coagulation, since it is needed for the posttranslational modification of 7 proteins involved in this cascade. However, it is also involved in the maturation of another 11 or 12 proteins that play...

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Mladěnka P, Macáková K, Kujovská Krčmová L, et al. Vitamin K - sources, physiological role, kinetics, deficiency, detection, therapeutic use, and toxicity. Nutr Rev. 2021;doi: 10.1093/nutrit/nuab061.
Mladěnka, P., Macáková, K., Kujovská Krčmová, L., Javorská, L., Mrštná, K., Carazo, A., Protti, M., Remião, F., Nováková, L. (2021). Vitamin K - sources, physiological role, kinetics, deficiency, detection, therapeutic use, and toxicity. Nutrition reviews, . https://doi.org/10.1093/nutrit/nuab061
Mladěnka, Přemysl, et al. "Vitamin K - sources, physiological role, kinetics, deficiency, detection, therapeutic use, and toxicity." Nutrition reviews vol. (2021). doi: https://doi.org/10.1093/nutrit/nuab061
Mladěnka P, Macáková K, Kujovská Krčmová L, Javorská L, Mrštná K, Carazo A, Protti M, Remião F, Nováková L. Vitamin K - sources, physiological role, kinetics, deficiency, detection, therapeutic use, and toxicity. Nutr Rev. 2021 Sep 02; doi: 10.1093/nutrit/nuab061. Epub 2021 Sep 02. PMID: 34472618.
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Updating the Canadian Hemophilia Outcomes-Kids' Life Assessment Tool (CHO-KLAT) in the era of extended half-life clotting factor concentrates.

Research and practice in thrombosis and haemostasis

Price VE, Dover S, Blanchette VS, Klaassen RJ, Belletrutti M, Bruce AAK, Chan AK, Wakefield C, Carcao M, Bouskill V, Young NL.
PMID: 33870026
Res Pract Thromb Haemost. 2021 Mar 27;5(3):403-411. doi: 10.1002/rth2.12498. eCollection 2021 Mar.

INTRODUCTION: The purpose of this study was to review and update the content of the Canadian Hemophilia Outcomes-Kids' Life Assessment Tool version 2.0 (CHO-KLAT), in the context of extended half-life (EHL) factor concentrates (FCs) and to establish the validity...

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Price VE, Dover S, Blanchette VS, et al. Updating the Canadian Hemophilia Outcomes-Kids' Life Assessment Tool (CHO-KLAT) in the era of extended half-life clotting factor concentrates. Res Pract Thromb Haemost. 2021;5(3):403-411doi: 10.1002/rth2.12498.
Price, V. E., Dover, S., Blanchette, V. S., Klaassen, R. J., Belletrutti, M., Bruce, A. A. K., Chan, A. K., Wakefield, C., Carcao, M., Bouskill, V., & Young, N. L. (2021). Updating the Canadian Hemophilia Outcomes-Kids' Life Assessment Tool (CHO-KLAT) in the era of extended half-life clotting factor concentrates. Research and practice in thrombosis and haemostasis, 5(3), 403-411. https://doi.org/10.1002/rth2.12498
Price, Victoria E, et al. "Updating the Canadian Hemophilia Outcomes-Kids' Life Assessment Tool (CHO-KLAT) in the era of extended half-life clotting factor concentrates." Research and practice in thrombosis and haemostasis vol. 5,3 (2021): 403-411. doi: https://doi.org/10.1002/rth2.12498
Price VE, Dover S, Blanchette VS, Klaassen RJ, Belletrutti M, Bruce AAK, Chan AK, Wakefield C, Carcao M, Bouskill V, Young NL. Updating the Canadian Hemophilia Outcomes-Kids' Life Assessment Tool (CHO-KLAT) in the era of extended half-life clotting factor concentrates. Res Pract Thromb Haemost. 2021 Mar 27;5(3):403-411. doi: 10.1002/rth2.12498. eCollection 2021 Mar. PMID: 33870026; PMCID: PMC8035800.
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Excess weight gain in the paediatric bleeding disorders population: Impact of the COVID-19 Pandemic.

Haemophilia : the official journal of the World Federation of Hemophilia

Malcolmson C, Tetzlaff D, Maguire B, Wakefield C, Carcao M, Bouskill V.
PMID: 34761485
Haemophilia. 2022 Jan;28(1):91-96. doi: 10.1111/hae.14449. Epub 2021 Nov 10.

INTRODUCTION: The COVID-19 pandemic has resulted in lifestyle changes for children. The aim of this study was to evaluate the impact of the pandemic on weight/BMI in children with severe bleeding disorders.METHODS: We conducted a retrospective review of patients...

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Malcolmson C, Tetzlaff D, Maguire B, et al. Excess weight gain in the paediatric bleeding disorders population: Impact of the COVID-19 Pandemic. Haemophilia. 2021;28(1):91-96doi: 10.1111/hae.14449.
Malcolmson, C., Tetzlaff, D., Maguire, B., Wakefield, C., Carcao, M., & Bouskill, V. (2022). Excess weight gain in the paediatric bleeding disorders population: Impact of the COVID-19 Pandemic. Haemophilia : the official journal of the World Federation of Hemophilia, 28(1), 91-96. https://doi.org/10.1111/hae.14449
Malcolmson, Caroline, et al. "Excess weight gain in the paediatric bleeding disorders population: Impact of the COVID-19 Pandemic." Haemophilia : the official journal of the World Federation of Hemophilia vol. 28,1 (2022): 91-96. doi: https://doi.org/10.1111/hae.14449
Malcolmson C, Tetzlaff D, Maguire B, Wakefield C, Carcao M, Bouskill V. Excess weight gain in the paediatric bleeding disorders population: Impact of the COVID-19 Pandemic. Haemophilia. 2022 Jan;28(1):91-96. doi: 10.1111/hae.14449. Epub 2021 Nov 10. PMID: 34761485; PMCID: PMC8653224.
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Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease.

Frontiers in pharmacology

Caruso G, Grasso M, Fidilio A, Torrisi SA, Musso N, Geraci F, Tropea MR, Privitera A, Tascedda F, Puzzo D, Salomone S, Drago F, Leggio GM, Caraci F.
PMID: 35002742
Front Pharmacol. 2021 Dec 24;12:809541. doi: 10.3389/fphar.2021.809541. eCollection 2021.

Depression is a risk factor for the development of Alzheimer's disease (AD). A neurobiological and clinical continuum exists between AD and depression, with neuroinflammation and oxidative stress being involved in both diseases. Second-generation antidepressants, in particular selective serotonin reuptake...

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Caruso G, Grasso M, Fidilio A, et al. Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease. Front Pharmacol. 2021;12:809541doi: 10.3389/fphar.2021.809541.
Caruso, G., Grasso, M., Fidilio, A., Torrisi, S. A., Musso, N., Geraci, F., Tropea, M. R., Privitera, A., Tascedda, F., Puzzo, D., Salomone, S., Drago, F., Leggio, G. M., & Caraci, F. (2021). Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease. Frontiers in pharmacology, 12809541. https://doi.org/10.3389/fphar.2021.809541
Caruso, Giuseppe, et al. "Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease." Frontiers in pharmacology vol. 12 (2021): 809541. doi: https://doi.org/10.3389/fphar.2021.809541
Caruso G, Grasso M, Fidilio A, Torrisi SA, Musso N, Geraci F, Tropea MR, Privitera A, Tascedda F, Puzzo D, Salomone S, Drago F, Leggio GM, Caraci F. Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease. Front Pharmacol. 2021 Dec 24;12:809541. doi: 10.3389/fphar.2021.809541. eCollection 2021. PMID: 35002742; PMCID: PMC8740153.
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Showing 205 to 211 of 211 entries