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Showing 13 to 19 of 19 entries
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IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on .

Frontiers in immunology

Barreira-Silva P, Melo-Miranda R, Nobrega C, Roque S, Serre-Miranda C, Borges M, Armada G, de Sá Calçada D, Behar SM, Appelberg R, Correia-Neves M.
PMID: 34987496
Front Immunol. 2021 Dec 20;12:696415. doi: 10.3389/fimmu.2021.696415. eCollection 2021.

Disseminated infection with the high virulence strain of

Cite
Barreira-Silva P, Melo-Miranda R, Nobrega C, et al. IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on . Front Immunol. 2021;12:696415doi: 10.3389/fimmu.2021.696415.
Barreira-Silva, P., Melo-Miranda, R., Nobrega, C., Roque, S., Serre-Miranda, C., Borges, M., Armada, G., de Sá Calçada, D., Behar, S. M., Appelberg, R., & Correia-Neves, M. (2021). IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on . Frontiers in immunology, 12696415. https://doi.org/10.3389/fimmu.2021.696415
Barreira-Silva, Palmira, et al. "IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on ." Frontiers in immunology vol. 12 (2021): 696415. doi: https://doi.org/10.3389/fimmu.2021.696415
Barreira-Silva P, Melo-Miranda R, Nobrega C, Roque S, Serre-Miranda C, Borges M, Armada G, de Sá Calçada D, Behar SM, Appelberg R, Correia-Neves M. IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on . Front Immunol. 2021 Dec 20;12:696415. doi: 10.3389/fimmu.2021.696415. eCollection 2021. PMID: 34987496; PMCID: PMC8721011.
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Endogenous cathelicidin production limits inflammation and protective immunity to Mycobacterium avium in mice.

Immunity, inflammation and disease

Santos JC, Silva-Gomes S, Silva JP, Gama M, Rosa G, Gallo RL, Appelberg R.
PMID: 25400920
Immun Inflamm Dis. 2014 Jun;2(1):1-12. doi: 10.1002/iid3.7. Epub 2013 Oct 31.

The production of antimicrobial peptides, such as the cathelicidins, plays a prominent role in the innate immune response against microbial pathogens. Cathelicidins are widely distributed amongst living organisms, and the antimicrobial peptides generated by proteolysis of the precursor forms...

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Santos JC, Silva-Gomes S, Silva JP, et al. Endogenous cathelicidin production limits inflammation and protective immunity to Mycobacterium avium in mice. Immun Inflamm Dis. 2013;2(1):1-12doi: 10.1002/iid3.7.
Santos, J. C., Silva-Gomes, S., Silva, J. P., Gama, M., Rosa, G., Gallo, R. L., & Appelberg, R. (2014). Endogenous cathelicidin production limits inflammation and protective immunity to Mycobacterium avium in mice. Immunity, inflammation and disease, 2(1), 1-12. https://doi.org/10.1002/iid3.7
Santos, José Carlos, et al. "Endogenous cathelicidin production limits inflammation and protective immunity to Mycobacterium avium in mice." Immunity, inflammation and disease vol. 2,1 (2014): 1-12. doi: https://doi.org/10.1002/iid3.7
Santos JC, Silva-Gomes S, Silva JP, Gama M, Rosa G, Gallo RL, Appelberg R. Endogenous cathelicidin production limits inflammation and protective immunity to Mycobacterium avium in mice. Immun Inflamm Dis. 2014 Jun;2(1):1-12. doi: 10.1002/iid3.7. Epub 2013 Oct 31. PMID: 25400920; PMCID: PMC4220664.
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Nucleoside-modified mRNA vaccines protect IFNAR.

Journal of virology

Appelberg S, John L, Pardi N, Végvári Á, Bereczky S, Ahlén G, Monteil V, Abdurahman S, Mikaeloff F, Beattie M, Tam Y, Sällberg M, Neogi U, Weissman D, Mirazimi A.
PMID: 34817199
J Virol. 2021 Nov 24;JVI0156821. doi: 10.1128/JVI.01568-21. Epub 2021 Nov 24.

Crimean-Congo hemorrhagic fever (CCHF), caused by Crimean-Congo hemorrhagic fever virus (CCHFV), is on the World Health Organizations' list of prioritized diseases and pathogens. With global distribution, high fatality rate and no approved vaccine or effective treatment, CCHF constitutes a...

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Appelberg S, John L, Pardi N, et al. Nucleoside-modified mRNA vaccines protect IFNAR. J Virol. 2021;JVI0156821doi: 10.1128/JVI.01568-21.
Appelberg, S., John, L., Pardi, N., Végvári, �. �., Bereczky, S., Ahlén, G., Monteil, V., Abdurahman, S., Mikaeloff, F., Beattie, M., Tam, Y., Sällberg, M., Neogi, U., Weissman, D., & Mirazimi, A. (2021). Nucleoside-modified mRNA vaccines protect IFNAR. Journal of virology, JVI0156821. https://doi.org/10.1128/JVI.01568-21
Appelberg, Sofia, et al. "Nucleoside-modified mRNA vaccines protect IFNAR." Journal of virology vol. (2021): JVI0156821. doi: https://doi.org/10.1128/JVI.01568-21
Appelberg S, John L, Pardi N, Végvári Á, Bereczky S, Ahlén G, Monteil V, Abdurahman S, Mikaeloff F, Beattie M, Tam Y, Sällberg M, Neogi U, Weissman D, Mirazimi A. Nucleoside-modified mRNA vaccines protect IFNAR. J Virol. 2021 Nov 24;JVI0156821. doi: 10.1128/JVI.01568-21. Epub 2021 Nov 24. PMID: 34817199.
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IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages.

Frontiers in immunology

Cruz A, Ludovico P, Torrado E, Gama JB, Sousa J, Gaifem J, Appelberg R, Rodrigues F, Cooper AM, Pedrosa J, Saraiva M, Castro AG.
PMID: 26483789
Front Immunol. 2015 Sep 30;6:498. doi: 10.3389/fimmu.2015.00498. eCollection 2015.

The fate of infected macrophages is a critical aspect of immunity to mycobacteria. By depriving the pathogen of its intracellular niche, apoptotic death of the infected macrophage has been shown to be an important mechanism to control bacterial growth....

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Cruz A, Ludovico P, Torrado E, et al. IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages. Front Immunol. 2015;6:498doi: 10.3389/fimmu.2015.00498.
Cruz, A., Ludovico, P., Torrado, E., Gama, J. B., Sousa, J., Gaifem, J., Appelberg, R., Rodrigues, F., Cooper, A. M., Pedrosa, J., Saraiva, M., & Castro, A. G. (2015). IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages. Frontiers in immunology, 6498. https://doi.org/10.3389/fimmu.2015.00498
Cruz, Andrea, et al. "IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages." Frontiers in immunology vol. 6 (2015): 498. doi: https://doi.org/10.3389/fimmu.2015.00498
Cruz A, Ludovico P, Torrado E, Gama JB, Sousa J, Gaifem J, Appelberg R, Rodrigues F, Cooper AM, Pedrosa J, Saraiva M, Castro AG. IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages. Front Immunol. 2015 Sep 30;6:498. doi: 10.3389/fimmu.2015.00498. eCollection 2015. PMID: 26483789; PMCID: PMC4588696.
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Gut Microbiota Perturbation in IgA Deficiency Is Influenced by IgA-Autoantibody Status.

Gastroenterology

Moll JM, Myers PN, Zhang C, Eriksen C, Wolf J, Appelberg KS, Lindberg G, Bahl MI, Zhao H, Pan-Hammarström Q, Cai K, Jia H, Borte S, Nielsen HB, Kristiansen K, Brix S, Hammarström L.
PMID: 33662387
Gastroenterology. 2021 Jun;160(7):2423-2434.e5. doi: 10.1053/j.gastro.2021.02.053. Epub 2021 Mar 01.

BACKGROUND & AIMS: IgA exerts its primary function at mucosal surfaces, where it binds microbial antigens to regulate bacterial growth and epithelial attachment. One third of individuals with IgA deficiency (IgAD) suffers from recurrent mucosal infections, possibly related to...

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Moll JM, Myers PN, Zhang C, et al. Gut Microbiota Perturbation in IgA Deficiency Is Influenced by IgA-Autoantibody Status. Gastroenterology. 2021;160(7):2423-2434.e5doi: 10.1053/j.gastro.2021.02.053.
Moll, J. M., Myers, P. N., Zhang, C., Eriksen, C., Wolf, J., Appelberg, K. S., Lindberg, G., Bahl, M. I., Zhao, H., Pan-Hammarström, Q., Cai, K., Jia, H., Borte, S., Nielsen, H. B., Kristiansen, K., Brix, S., & Hammarström, L. (2021). Gut Microbiota Perturbation in IgA Deficiency Is Influenced by IgA-Autoantibody Status. Gastroenterology, 160(7), 2423-2434.e5. https://doi.org/10.1053/j.gastro.2021.02.053
Moll, Janne Marie, et al. "Gut Microbiota Perturbation in IgA Deficiency Is Influenced by IgA-Autoantibody Status." Gastroenterology vol. 160,7 (2021): 2423-2434.e5. doi: https://doi.org/10.1053/j.gastro.2021.02.053
Moll JM, Myers PN, Zhang C, Eriksen C, Wolf J, Appelberg KS, Lindberg G, Bahl MI, Zhao H, Pan-Hammarström Q, Cai K, Jia H, Borte S, Nielsen HB, Kristiansen K, Brix S, Hammarström L. Gut Microbiota Perturbation in IgA Deficiency Is Influenced by IgA-Autoantibody Status. Gastroenterology. 2021 Jun;160(7):2423-2434.e5. doi: 10.1053/j.gastro.2021.02.053. Epub 2021 Mar 01. PMID: 33662387.
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Presence of antibodies to Crimean Congo haemorrhagic fever virus in sheep in Tunisia, North Africa.

Veterinary medicine and science

Khamassi Khbou M, Romdhane R, Bouaicha Zaafouri F, Bouajila M, Sassi L, Appelberg SK, Schulz A, Mirazimi A, Groschup MH, Rekik M, Benzarti M, Gharbi M.
PMID: 34390548
Vet Med Sci. 2021 Nov;7(6):2323-2329. doi: 10.1002/vms3.597. Epub 2021 Aug 14.

Crimean-Congo haemorrhagic fever (CCHF) is an emerging tick-borne disease causing severe and fatal haemorrhagic syndrome in humans. Hyalomma spp. ticks are the primary vectors and sheep are important CCHF virus (CCHFV)-amplifying hosts. In this study, blood samples and ticks...

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Khamassi Khbou M, Romdhane R, Bouaicha Zaafouri F, et al. Presence of antibodies to Crimean Congo haemorrhagic fever virus in sheep in Tunisia, North Africa. Vet Med Sci. 2021;7(6):2323-2329doi: 10.1002/vms3.597.
Khamassi Khbou, M., Romdhane, R., Bouaicha Zaafouri, F., Bouajila, M., Sassi, L., Appelberg, S. K., Schulz, A., Mirazimi, A., Groschup, M. H., Rekik, M., Benzarti, M., & Gharbi, M. (2021). Presence of antibodies to Crimean Congo haemorrhagic fever virus in sheep in Tunisia, North Africa. Veterinary medicine and science, 7(6), 2323-2329. https://doi.org/10.1002/vms3.597
Khamassi Khbou, Médiha, et al. "Presence of antibodies to Crimean Congo haemorrhagic fever virus in sheep in Tunisia, North Africa." Veterinary medicine and science vol. 7,6 (2021): 2323-2329. doi: https://doi.org/10.1002/vms3.597
Khamassi Khbou M, Romdhane R, Bouaicha Zaafouri F, Bouajila M, Sassi L, Appelberg SK, Schulz A, Mirazimi A, Groschup MH, Rekik M, Benzarti M, Gharbi M. Presence of antibodies to Crimean Congo haemorrhagic fever virus in sheep in Tunisia, North Africa. Vet Med Sci. 2021 Nov;7(6):2323-2329. doi: 10.1002/vms3.597. Epub 2021 Aug 14. PMID: 34390548; PMCID: PMC8604105.
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IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on .

Frontiers in immunology

Barreira-Silva P, Melo-Miranda R, Nobrega C, Roque S, Serre-Miranda C, Borges M, Armada G, de Sá Calçada D, Behar SM, Appelberg R, Correia-Neves M.
PMID: 34987496
Front Immunol. 2021 Dec 20;12:696415. doi: 10.3389/fimmu.2021.696415. eCollection 2021.

Disseminated infection with the high virulence strain of

Cite
Barreira-Silva P, Melo-Miranda R, Nobrega C, et al. IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on . Front Immunol. 2021;12:696415doi: 10.3389/fimmu.2021.696415.
Barreira-Silva, P., Melo-Miranda, R., Nobrega, C., Roque, S., Serre-Miranda, C., Borges, M., Armada, G., de Sá Calçada, D., Behar, S. M., Appelberg, R., & Correia-Neves, M. (2021). IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on . Frontiers in immunology, 12696415. https://doi.org/10.3389/fimmu.2021.696415
Barreira-Silva, Palmira, et al. "IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on ." Frontiers in immunology vol. 12 (2021): 696415. doi: https://doi.org/10.3389/fimmu.2021.696415
Barreira-Silva P, Melo-Miranda R, Nobrega C, Roque S, Serre-Miranda C, Borges M, Armada G, de Sá Calçada D, Behar SM, Appelberg R, Correia-Neves M. IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on . Front Immunol. 2021 Dec 20;12:696415. doi: 10.3389/fimmu.2021.696415. eCollection 2021. PMID: 34987496; PMCID: PMC8721011.
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Showing 13 to 19 of 19 entries