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Showing 13 to 24 of 339 entries
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Islet cell tumours.

Cancer imaging : the official publication of the International Cancer Imaging Society

Reznek R.
PMID: 18211851
Cancer Imaging. 2003 Sep 30;4(1):1-4. doi: 10.1102/1470-7330.2003.0023.

Islet cells tumours are a range of rare neoplasms of neuroendocrine origin arising in or close to the pancreas. The normal islet cells of Langerhans in the pancreas contain B-cells (which secrete insulin), A-cells (which secrete glucagon), D-cells (which...

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Reznek R. Islet cell tumours. Cancer Imaging. 2003;4(1):1-4doi: 10.1102/1470-7330.2003.0023.
Reznek, R. (2003). Islet cell tumours. Cancer imaging : the official publication of the International Cancer Imaging Society, 4(1), 1-4. https://doi.org/10.1102/1470-7330.2003.0023
Reznek, R. "Islet cell tumours." Cancer imaging : the official publication of the International Cancer Imaging Society vol. 4,1 (2003): 1-4. doi: https://doi.org/10.1102/1470-7330.2003.0023
Reznek R. Islet cell tumours. Cancer Imaging. 2003 Sep 30;4(1):1-4. doi: 10.1102/1470-7330.2003.0023. PMID: 18211851; PMCID: PMC1434518.
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Actin reorganization is involved in vasoactive intestinal peptide induced human mast cells priming to fraktalkine-induced chemotaxis.

International journal of general medicine

El-Shazly AE.
PMID: 20428403
Int J Gen Med. 2008 Nov 30;1:27-31. doi: 10.2147/ijgm.s3759.

We recently reported a novel neuro-immuno co-operation between vasoactive intestinal peptide (VIP) and fraktalkine (FKN) in recruiting human mast cells to the asthmatic airway that provided a classical example of priming effect on mast cells migratory function, but the...

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El-Shazly AE. Actin reorganization is involved in vasoactive intestinal peptide induced human mast cells priming to fraktalkine-induced chemotaxis. Int J Gen Med. 2008;1:27-31doi: 10.2147/ijgm.s3759.
El-Shazly, A. E. (2008). Actin reorganization is involved in vasoactive intestinal peptide induced human mast cells priming to fraktalkine-induced chemotaxis. International journal of general medicine, 127-31. https://doi.org/10.2147/ijgm.s3759
El-Shazly, Amr E. "Actin reorganization is involved in vasoactive intestinal peptide induced human mast cells priming to fraktalkine-induced chemotaxis." International journal of general medicine vol. 1 (2008): 27-31. doi: https://doi.org/10.2147/ijgm.s3759
El-Shazly AE. Actin reorganization is involved in vasoactive intestinal peptide induced human mast cells priming to fraktalkine-induced chemotaxis. Int J Gen Med. 2008 Nov 30;1:27-31. doi: 10.2147/ijgm.s3759. PMID: 20428403; PMCID: PMC2840542.
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PKC modulation of inhibitory coupling of angiotensin II receptors with adenylate cyclase in lactotroph cells.

Molecular and cellular neurosciences

Audinot V, Rasolonjanahary R, Bertrand P, Priam M, Kordon C, Enjalbert A.
PMID: 19912854
Mol Cell Neurosci. 1992 Apr;3(2):145-51. doi: 10.1016/1044-7431(92)90018-w.

Angiotensin II (AII) and thyreoliberin (TRH) have recently been shown to stimulate intracellular cAMP formation in rat lactotroph cells, in addition to their already documented coupling to phospholipase C. The effect on intracellular cAMP is unaffected by pertussis toxin...

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Audinot V, Rasolonjanahary R, Bertrand P, et al. PKC modulation of inhibitory coupling of angiotensin II receptors with adenylate cyclase in lactotroph cells. Mol Cell Neurosci. 1992;3(2):145-51doi: 10.1016/1044-7431(92)90018-w.
Audinot, V., Rasolonjanahary, R., Bertrand, P., Priam, M., Kordon, C., & Enjalbert, A. (1992). PKC modulation of inhibitory coupling of angiotensin II receptors with adenylate cyclase in lactotroph cells. Molecular and cellular neurosciences, 3(2), 145-51. https://doi.org/10.1016/1044-7431(92)90018-w
Audinot, V, et al. "PKC modulation of inhibitory coupling of angiotensin II receptors with adenylate cyclase in lactotroph cells." Molecular and cellular neurosciences vol. 3,2 (1992): 145-51. doi: https://doi.org/10.1016/1044-7431(92)90018-w
Audinot V, Rasolonjanahary R, Bertrand P, Priam M, Kordon C, Enjalbert A. PKC modulation of inhibitory coupling of angiotensin II receptors with adenylate cyclase in lactotroph cells. Mol Cell Neurosci. 1992 Apr;3(2):145-51. doi: 10.1016/1044-7431(92)90018-w. PMID: 19912854.
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mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum.

Frontiers in neuroscience

Foong JP, Bornstein JC.
PMID: 20582273
Front Neurosci. 2009 May 29;3:46. doi: 10.3389/neuro.21.001.2009. eCollection 2009.

Vasoactive intestinal peptide (VIP) immunoreactive secretomotor neurons in the submucous plexus are involved in mediating bacterial toxin-induced hypersecretion leading to diarrhoea. VIP neurons become hyperexcitable after the mucosa is exposed to cholera toxin, which suggests that the manipulation of...

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Foong JP, Bornstein JC. mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum. Front Neurosci. 2009;3:46doi: 10.3389/neuro.21.001.2009.
Foong, J. P., & Bornstein, J. C. (2009). mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum. Frontiers in neuroscience, 346. https://doi.org/10.3389/neuro.21.001.2009
Foong, Jaime Pei Pei, and Bornstein, Joel C. "mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum." Frontiers in neuroscience vol. 3 (2009): 46. doi: https://doi.org/10.3389/neuro.21.001.2009
Foong JP, Bornstein JC. mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum. Front Neurosci. 2009 May 29;3:46. doi: 10.3389/neuro.21.001.2009. eCollection 2009. PMID: 20582273; PMCID: PMC2695390.
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Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease.

Neurochemistry international

De Laet MH, Dassonville M, Steyaert H, Vanderveken E, Lotstra F, Vierendeels G, Przedborski S, Vaudry H, Vanderhaeghen JJ.
PMID: 20504409
Neurochem Int. 1989;14(2):135-41. doi: 10.1016/0197-0186(89)90113-7.

Ganglionic and aganglionic full-thickness samples, at 4 levels of the colon of 26 infants with Hirschsprung's disease, were studied by immunohistochemistry. In the distal part of the aganglionic bowel, we observe a decrease of substance P and vasoactive intestinal...

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De Laet MH, Dassonville M, Steyaert H, et al. Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease. Neurochem Int. 1989;14(2):135-41doi: 10.1016/0197-0186(89)90113-7.
De Laet, M. H., Dassonville, M., Steyaert, H., Vanderveken, E., Lotstra, F., Vierendeels, G., Przedborski, S., Vaudry, H., & Vanderhaeghen, J. J. (1989). Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease. Neurochemistry international, 14(2), 135-41. https://doi.org/10.1016/0197-0186(89)90113-7
De Laet, M H, et al. "Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease." Neurochemistry international vol. 14,2 (1989): 135-41. doi: https://doi.org/10.1016/0197-0186(89)90113-7
De Laet MH, Dassonville M, Steyaert H, Vanderveken E, Lotstra F, Vierendeels G, Przedborski S, Vaudry H, Vanderhaeghen JJ. Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease. Neurochem Int. 1989;14(2):135-41. doi: 10.1016/0197-0186(89)90113-7. PMID: 20504409.
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Vasoactive-intestinal-Peptide (vip) modulates the growth fraction of epithelial skin cells.

International journal of oncology

Wollina U, Bonnekoh B, Mahrle G.
PMID: 21584504
Int J Oncol. 1992 Jun;1(1):17-24. doi: 10.3892/ijo.1.1.17.

Using the human keratinocyte cell line HaCaT, modifications of the growth fraction due to vasoactive intestinal peptide (VIP) were determined by immunostaining with monoclonal antibody Ki67. In addition, the expression of VIP receptor and epidermal growth factor (EGF) receptor...

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Wollina U, Bonnekoh B, Mahrle G. Vasoactive-intestinal-Peptide (vip) modulates the growth fraction of epithelial skin cells. Int J Oncol. 1992;1(1):17-24doi: 10.3892/ijo.1.1.17.
Wollina, U., Bonnekoh, B., & Mahrle, G. (1992). Vasoactive-intestinal-Peptide (vip) modulates the growth fraction of epithelial skin cells. International journal of oncology, 1(1), 17-24. https://doi.org/10.3892/ijo.1.1.17
Wollina, U, et al. "Vasoactive-intestinal-Peptide (vip) modulates the growth fraction of epithelial skin cells." International journal of oncology vol. 1,1 (1992): 17-24. doi: https://doi.org/10.3892/ijo.1.1.17
Wollina U, Bonnekoh B, Mahrle G. Vasoactive-intestinal-Peptide (vip) modulates the growth fraction of epithelial skin cells. Int J Oncol. 1992 Jun;1(1):17-24. doi: 10.3892/ijo.1.1.17. PMID: 21584504.
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Evidence for the presence of cholinergic muscarinic receptors negatively linked to adenylate cyclase in the iris-ciliary body.

Neurochemistry international

Tobin AB, Osborne NN.
PMID: 20501312
Neurochem Int. 1988;13(4):517-23. doi: 10.1016/0197-0186(88)90083-6.

Adenylate cyclase activity can be stimulated in the rabbit iris-ciliary body directly by forskolin or through receptor-mediated mechanisms by vasoactive intestinal peptide (VIP) and the ?-adrenoreceptor agonists isoproterenol and salbutamol. Increases in the level of c-AMP observed following application...

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Tobin AB, Osborne NN. Evidence for the presence of cholinergic muscarinic receptors negatively linked to adenylate cyclase in the iris-ciliary body. Neurochem Int. 1988;13(4):517-23doi: 10.1016/0197-0186(88)90083-6.
Tobin, A. B., & Osborne, N. N. (1988). Evidence for the presence of cholinergic muscarinic receptors negatively linked to adenylate cyclase in the iris-ciliary body. Neurochemistry international, 13(4), 517-23. https://doi.org/10.1016/0197-0186(88)90083-6
Tobin, A B, and Osborne, N N. "Evidence for the presence of cholinergic muscarinic receptors negatively linked to adenylate cyclase in the iris-ciliary body." Neurochemistry international vol. 13,4 (1988): 517-23. doi: https://doi.org/10.1016/0197-0186(88)90083-6
Tobin AB, Osborne NN. Evidence for the presence of cholinergic muscarinic receptors negatively linked to adenylate cyclase in the iris-ciliary body. Neurochem Int. 1988;13(4):517-23. doi: 10.1016/0197-0186(88)90083-6. PMID: 20501312.
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Modulation of IgA synthesis by neuroendocrine peptides.

Trends in endocrinology and metabolism: TEM

Hermann GE, Sue O'Dorisio M.
PMID: 18411168
Trends Endocrinol Metab. 1991 Mar-Apr;2(2):68-72. doi: 10.1016/1043-2760(91)90043-m.

Prolactin, estrogen, and progesterone together induce proliferation of the mammary glands with a coincident increase in the IgA-secreting plasma cells in the tissue. Of these three hormones, Prolactin is the most effective single peptide in stimulating IgA production. Vasoactive...

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Hermann GE, Sue O'Dorisio M. Modulation of IgA synthesis by neuroendocrine peptides. Trends Endocrinol Metab. 1991;2(2):68-72doi: 10.1016/1043-2760(91)90043-m.
Hermann, G. E., & Sue O'Dorisio, M. (1991). Modulation of IgA synthesis by neuroendocrine peptides. Trends in endocrinology and metabolism: TEM, 2(2), 68-72. https://doi.org/10.1016/1043-2760(91)90043-m
Hermann, G E, and Sue O'Dorisio, M. "Modulation of IgA synthesis by neuroendocrine peptides." Trends in endocrinology and metabolism: TEM vol. 2,2 (1991): 68-72. doi: https://doi.org/10.1016/1043-2760(91)90043-m
Hermann GE, Sue O'Dorisio M. Modulation of IgA synthesis by neuroendocrine peptides. Trends Endocrinol Metab. 1991 Mar-Apr;2(2):68-72. doi: 10.1016/1043-2760(91)90043-m. PMID: 18411168.
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Agonist-induced GPCR shedding from the ciliary surface is dependent on ESCRT-III and VPS4.

Current biology : CB

Soetedjo L, Jin H.
PMID: 24530064
Curr Biol. 2014 Mar 03;24(5):509-18. doi: 10.1016/j.cub.2014.01.010. Epub 2014 Feb 13.

BACKGROUND: Membrane trafficking of G protein-coupled receptors (GPCRs) is crucial for temporal and spatial control of cell-surface GPCR signaling. Receptor internalization is a well-documented method cells use for regulating a wide variety of GPCRs following their exposure to agonists.RESULTS:...

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Soetedjo L, Jin H. Agonist-induced GPCR shedding from the ciliary surface is dependent on ESCRT-III and VPS4. Curr Biol. 2014;24(5):509-18doi: 10.1016/j.cub.2014.01.010.
Soetedjo, L., & Jin, H. (2014). Agonist-induced GPCR shedding from the ciliary surface is dependent on ESCRT-III and VPS4. Current biology : CB, 24(5), 509-18. https://doi.org/10.1016/j.cub.2014.01.010
Soetedjo, Livana, and Jin, Hua. "Agonist-induced GPCR shedding from the ciliary surface is dependent on ESCRT-III and VPS4." Current biology : CB vol. 24,5 (2014): 509-18. doi: https://doi.org/10.1016/j.cub.2014.01.010
Soetedjo L, Jin H. Agonist-induced GPCR shedding from the ciliary surface is dependent on ESCRT-III and VPS4. Curr Biol. 2014 Mar 03;24(5):509-18. doi: 10.1016/j.cub.2014.01.010. Epub 2014 Feb 13. PMID: 24530064.
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Gut hormones in cyclostomes.

Fish physiology and biochemistry

Van Noorden S.
PMID: 24220989
Fish Physiol Biochem. 1990 Sep;8(5):399-408. doi: 10.1007/BF00003371.

The current state of knowledge about regulatory peptides in endocrine cells and nerves of the alimentary canal of lampreys and hagfishes is reviewed. Cyclostomes have a wide range of peptides similar immunochemically to those of higher vertebrates. They include,...

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Van Noorden S. Gut hormones in cyclostomes. Fish Physiol Biochem. 1990;8(5):399-408doi: 10.1007/BF00003371.
Van Noorden, S. (1990). Gut hormones in cyclostomes. Fish physiology and biochemistry, 8(5), 399-408. https://doi.org/10.1007/BF00003371
Van Noorden, S. "Gut hormones in cyclostomes." Fish physiology and biochemistry vol. 8,5 (1990): 399-408. doi: https://doi.org/10.1007/BF00003371
Van Noorden S. Gut hormones in cyclostomes. Fish Physiol Biochem. 1990 Sep;8(5):399-408. doi: 10.1007/BF00003371. PMID: 24220989.
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Activation of PAC1 Receptors in Rat Cerebellar Granule Cells Stimulates Both Calcium Mobilization from Intracellular Stores and Calcium Influx through N-Type Calcium Channels.

Frontiers in endocrinology

Basille-Dugay M, Vaudry H, Fournier A, Gonzalez B, Vaudry D.
PMID: 23675369
Front Endocrinol (Lausanne). 2013 May 10;4:56. doi: 10.3389/fendo.2013.00056. eCollection 2013.

High concentrations of pituitary adenylate cyclase-activating polypeptide (PACAP) and a high density of PACAP binding sites have been detected in the developing rat cerebellum. In particular, PACAP receptors are actively expressed in immature granule cells, where they activate both...

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Basille-Dugay M, Vaudry H, Fournier A, et al. Activation of PAC1 Receptors in Rat Cerebellar Granule Cells Stimulates Both Calcium Mobilization from Intracellular Stores and Calcium Influx through N-Type Calcium Channels. Front Endocrinol (Lausanne). 2013;4:56doi: 10.3389/fendo.2013.00056.
Basille-Dugay, M., Vaudry, H., Fournier, A., Gonzalez, B., & Vaudry, D. (2013). Activation of PAC1 Receptors in Rat Cerebellar Granule Cells Stimulates Both Calcium Mobilization from Intracellular Stores and Calcium Influx through N-Type Calcium Channels. Frontiers in endocrinology, 456. https://doi.org/10.3389/fendo.2013.00056
Basille-Dugay, Magali, et al. "Activation of PAC1 Receptors in Rat Cerebellar Granule Cells Stimulates Both Calcium Mobilization from Intracellular Stores and Calcium Influx through N-Type Calcium Channels." Frontiers in endocrinology vol. 4 (2013): 56. doi: https://doi.org/10.3389/fendo.2013.00056
Basille-Dugay M, Vaudry H, Fournier A, Gonzalez B, Vaudry D. Activation of PAC1 Receptors in Rat Cerebellar Granule Cells Stimulates Both Calcium Mobilization from Intracellular Stores and Calcium Influx through N-Type Calcium Channels. Front Endocrinol (Lausanne). 2013 May 10;4:56. doi: 10.3389/fendo.2013.00056. eCollection 2013. PMID: 23675369; PMCID: PMC3650316.
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Pituitary adenylate cyclase-activating polypeptide prevents contrast-induced nephropathy in a novel mouse model.

Physiological reports

Khan AM, Maderdrut JL, Li M, Toliver HL, Coy DH, Simon EE, Batuman V.
PMID: 24400164
Physiol Rep. 2013 Nov;1(6):e00163. doi: 10.1002/phy2.163. Epub 2013 Nov 19.

We determined whether pituitary adenylate cyclase-activating polypeptide 38 (PACAP38) prevents contrast-induced nephropathy using human renal proximal tubule epithelial (HK-2) cells and homozygous endothelial nitric oxide synthase-deficient (eNOS(-/-)) mice as a novel in vivo model. Cultured HK-2 cells were pretreated...

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Khan AM, Maderdrut JL, Li M, et al. Pituitary adenylate cyclase-activating polypeptide prevents contrast-induced nephropathy in a novel mouse model. Physiol Rep. 2013;1(6):e00163doi: 10.1002/phy2.163.
Khan, A. M., Maderdrut, J. L., Li, M., Toliver, H. L., Coy, D. H., Simon, E. E., & Batuman, V. (2013). Pituitary adenylate cyclase-activating polypeptide prevents contrast-induced nephropathy in a novel mouse model. Physiological reports, 1(6), e00163. https://doi.org/10.1002/phy2.163
Khan, Altaf-M, et al. "Pituitary adenylate cyclase-activating polypeptide prevents contrast-induced nephropathy in a novel mouse model." Physiological reports vol. 1,6 (2013): e00163. doi: https://doi.org/10.1002/phy2.163
Khan AM, Maderdrut JL, Li M, Toliver HL, Coy DH, Simon EE, Batuman V. Pituitary adenylate cyclase-activating polypeptide prevents contrast-induced nephropathy in a novel mouse model. Physiol Rep. 2013 Nov;1(6):e00163. doi: 10.1002/phy2.163. Epub 2013 Nov 19. PMID: 24400164; PMCID: PMC3871477.
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Showing 13 to 24 of 339 entries