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The two faces of Janus kinases and their respective STATs in mammary gland development and cancer.

Journal of carcinogenesis

Wagner KU, Schmidt JW.
PMID: 22279417
J Carcinog. 2011;10:32. doi: 10.4103/1477-3163.90677. Epub 2011 Dec 08.

Since its discovery as "just another kinase" more than twenty years ago, the family of JAK tyrosine kinases and their respective Signal Transducers and Activators of Transcription (STATs) has been a center of attention in the areas of signal...

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Wagner KU, Schmidt JW. The two faces of Janus kinases and their respective STATs in mammary gland development and cancer. J Carcinog. 2011;10:32doi: 10.4103/1477-3163.90677.
Wagner, K. U., & Schmidt, J. W. (2011). The two faces of Janus kinases and their respective STATs in mammary gland development and cancer. Journal of carcinogenesis, 1032. https://doi.org/10.4103/1477-3163.90677
Wagner, Kay-Uwe, and Schmidt, Jeffrey W. "The two faces of Janus kinases and their respective STATs in mammary gland development and cancer." Journal of carcinogenesis vol. 10 (2011): 32. doi: https://doi.org/10.4103/1477-3163.90677
Wagner KU, Schmidt JW. The two faces of Janus kinases and their respective STATs in mammary gland development and cancer. J Carcinog. 2011;10:32. doi: 10.4103/1477-3163.90677. Epub 2011 Dec 08. PMID: 22279417; PMCID: PMC3262999.
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Comparison of different methods for detecting epidermal growth factor receptor mutations in peripheral blood and tumor tissue of non-small cell lung cancer as a predictor of response to gefitinib.

OncoTargets and therapy

Xu F, Wu J, Xue C, Zhao Y, Jiang W, Lin L, Wu X, Lu Y, Bai H, Xu J, Zhu G, Zhang L.
PMID: 23251095
Onco Targets Ther. 2012;5:439-47. doi: 10.2147/OTT.S37289. Epub 2012 Dec 12.

BACKGROUND: Previous studies have reported that epidermal growth factor receptor (EGFR) mutation in tumor tissue and peripheral blood can predict the response to EGFR tyrosine kinase inhibitor (TKI) in non-small cell lung cancer (NSCLC). However, the heterogeneity of the...

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Xu F, Wu J, Xue C, et al. Comparison of different methods for detecting epidermal growth factor receptor mutations in peripheral blood and tumor tissue of non-small cell lung cancer as a predictor of response to gefitinib. Onco Targets Ther. 2012;5:439-47doi: 10.2147/OTT.S37289.
Xu, F., Wu, J., Xue, C., Zhao, Y., Jiang, W., Lin, L., Wu, X., Lu, Y., Bai, H., Xu, J., Zhu, G., & Zhang, L. (2012). Comparison of different methods for detecting epidermal growth factor receptor mutations in peripheral blood and tumor tissue of non-small cell lung cancer as a predictor of response to gefitinib. OncoTargets and therapy, 5439-47. https://doi.org/10.2147/OTT.S37289
Xu, Fei, et al. "Comparison of different methods for detecting epidermal growth factor receptor mutations in peripheral blood and tumor tissue of non-small cell lung cancer as a predictor of response to gefitinib." OncoTargets and therapy vol. 5 (2012): 439-47. doi: https://doi.org/10.2147/OTT.S37289
Xu F, Wu J, Xue C, Zhao Y, Jiang W, Lin L, Wu X, Lu Y, Bai H, Xu J, Zhu G, Zhang L. Comparison of different methods for detecting epidermal growth factor receptor mutations in peripheral blood and tumor tissue of non-small cell lung cancer as a predictor of response to gefitinib. Onco Targets Ther. 2012;5:439-47. doi: 10.2147/OTT.S37289. Epub 2012 Dec 12. PMID: 23251095; PMCID: PMC3525047.
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Enhanced efficacy of human brain-derived neural stem cells by transplantation of cell aggregates in a rat model of Parkinson's disease.

Journal of Korean Neurosurgical Society

Shin ES, Hwang O, Hwang YS, Suh JK, Chun YI, Jeon SR.
PMID: 25535514
J Korean Neurosurg Soc. 2014 Nov;56(5):383-9. doi: 10.3340/jkns.2014.56.5.383. Epub 2014 Nov 30.

OBJECTIVE: Neural tissue transplantation has been a promising strategy for the treatment of Parkinson's disease (PD). However, transplantation has the disadvantages of low-cell survival and/or development of dyskinesia. Transplantation of cell aggregates has the potential to overcome these problems,...

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Shin ES, Hwang O, Hwang YS, et al. Enhanced efficacy of human brain-derived neural stem cells by transplantation of cell aggregates in a rat model of Parkinson's disease. J Korean Neurosurg Soc. 2014;56(5):383-9doi: 10.3340/jkns.2014.56.5.383.
Shin, E. S., Hwang, O., Hwang, Y. S., Suh, J. K., Chun, Y. I., & Jeon, S. R. (2014). Enhanced efficacy of human brain-derived neural stem cells by transplantation of cell aggregates in a rat model of Parkinson's disease. Journal of Korean Neurosurgical Society, 56(5), 383-9. https://doi.org/10.3340/jkns.2014.56.5.383
Shin, Eun Sil, et al. "Enhanced efficacy of human brain-derived neural stem cells by transplantation of cell aggregates in a rat model of Parkinson's disease." Journal of Korean Neurosurgical Society vol. 56,5 (2014): 383-9. doi: https://doi.org/10.3340/jkns.2014.56.5.383
Shin ES, Hwang O, Hwang YS, Suh JK, Chun YI, Jeon SR. Enhanced efficacy of human brain-derived neural stem cells by transplantation of cell aggregates in a rat model of Parkinson's disease. J Korean Neurosurg Soc. 2014 Nov;56(5):383-9. doi: 10.3340/jkns.2014.56.5.383. Epub 2014 Nov 30. PMID: 25535514; PMCID: PMC4272995.
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Sexual dimorphism in the hypophysiotropic tyrosine hydroxylase-positive neurons in the preoptic area of the teleost, Clarias batrachus.

Biology of sex differences

Saha S, Patil S, Singh U, Singh O, Singru PS.
PMID: 26557978
Biol Sex Differ. 2015 Nov 09;6:23. doi: 10.1186/s13293-015-0042-x. eCollection 2015.

BACKGROUND: Dopamine (DA) neurons in the anteroventral periventricular nucleus (AVPV) in the preoptic area (POA) of mammals express estrogen receptors, regulate luteinizing hormone (LH) secretion, and show distinct sexual dimorphism. In teleosts, hypophysiotropic DA neurons of the nucleus preopticus...

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Saha S, Patil S, Singh U, et al. Sexual dimorphism in the hypophysiotropic tyrosine hydroxylase-positive neurons in the preoptic area of the teleost, Clarias batrachus. Biol Sex Differ. 2015;6:23doi: 10.1186/s13293-015-0042-x.
Saha, S., Patil, S., Singh, U., Singh, O., & Singru, P. S. (2015). Sexual dimorphism in the hypophysiotropic tyrosine hydroxylase-positive neurons in the preoptic area of the teleost, Clarias batrachus. Biology of sex differences, 623. https://doi.org/10.1186/s13293-015-0042-x
Saha, Soham, et al. "Sexual dimorphism in the hypophysiotropic tyrosine hydroxylase-positive neurons in the preoptic area of the teleost, Clarias batrachus." Biology of sex differences vol. 6 (2015): 23. doi: https://doi.org/10.1186/s13293-015-0042-x
Saha S, Patil S, Singh U, Singh O, Singru PS. Sexual dimorphism in the hypophysiotropic tyrosine hydroxylase-positive neurons in the preoptic area of the teleost, Clarias batrachus. Biol Sex Differ. 2015 Nov 09;6:23. doi: 10.1186/s13293-015-0042-x. eCollection 2015. PMID: 26557978; PMCID: PMC4640419.
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Molecular pathways associated with aggressiveness of papillary thyroid cancer.

Current genomics

Benvenga S, Koch CA.
PMID: 24955023
Curr Genomics. 2014 Jun;15(3):162-70. doi: 10.2174/1389202915999140404100958.

The most common thyroid malignancy is papillary thyroid cancer (PTC). Mortality rates from PTC mainly depend on its aggressiveness. Geno- and phenotyping of aggressive PTC has advanced our understanding of treatment failures and of potential future therapies. Unraveling molecular...

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Benvenga S, Koch CA. Molecular pathways associated with aggressiveness of papillary thyroid cancer. Curr Genomics. 2014;15(3):162-70doi: 10.2174/1389202915999140404100958.
Benvenga, S., & Koch, C. A. (2014). Molecular pathways associated with aggressiveness of papillary thyroid cancer. Current genomics, 15(3), 162-70. https://doi.org/10.2174/1389202915999140404100958
Benvenga, Salvatore, and Koch, Christian A. "Molecular pathways associated with aggressiveness of papillary thyroid cancer." Current genomics vol. 15,3 (2014): 162-70. doi: https://doi.org/10.2174/1389202915999140404100958
Benvenga S, Koch CA. Molecular pathways associated with aggressiveness of papillary thyroid cancer. Curr Genomics. 2014 Jun;15(3):162-70. doi: 10.2174/1389202915999140404100958. PMID: 24955023; PMCID: PMC4064555.
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Serum proteomic study on EGFR-TKIs target treatment for patients with NSCLC.

OncoTargets and therapy

Wu X, Liang W, Hou X, Lin Z, Zhao H, Huang Y, Fang W, Zhao Y, Wu J, Yang Y, Xue C, Hu Z, Zhang J, Zhang J, Ma Y, Zhou T, Qin T, Zhang L.
PMID: 24204163
Onco Targets Ther. 2013 Oct 21;6:1481-91. doi: 10.2147/OTT.S51887. eCollection 2013.

BACKGROUND: Although epidermal growth factor receptor (EGFR)-tyrosine kinase inhibitors (TKIs) are widely used for EGFR mutated non-small-cell lung cancer (NSCLC) patients, tumor sample availability and heterogeneity of the tumor remain challenging for physicians' selection of these patients. Here, we...

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Wu X, Liang W, Hou X, et al. Serum proteomic study on EGFR-TKIs target treatment for patients with NSCLC. Onco Targets Ther. 2013;6:1481-91doi: 10.2147/OTT.S51887.
Wu, X., Liang, W., Hou, X., Lin, Z., Zhao, H., Huang, Y., Fang, W., Zhao, Y., Wu, J., Yang, Y., Xue, C., Hu, Z., Zhang, J., Zhang, J., Ma, Y., Zhou, T., Qin, T., & Zhang, L. (2013). Serum proteomic study on EGFR-TKIs target treatment for patients with NSCLC. OncoTargets and therapy, 61481-91. https://doi.org/10.2147/OTT.S51887
Wu, Xuan, et al. "Serum proteomic study on EGFR-TKIs target treatment for patients with NSCLC." OncoTargets and therapy vol. 6 (2013): 1481-91. doi: https://doi.org/10.2147/OTT.S51887
Wu X, Liang W, Hou X, Lin Z, Zhao H, Huang Y, Fang W, Zhao Y, Wu J, Yang Y, Xue C, Hu Z, Zhang J, Zhang J, Ma Y, Zhou T, Qin T, Zhang L. Serum proteomic study on EGFR-TKIs target treatment for patients with NSCLC. Onco Targets Ther. 2013 Oct 21;6:1481-91. doi: 10.2147/OTT.S51887. eCollection 2013. PMID: 24204163; PMCID: PMC3818102.
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EGFR-TKI resistance in NSCLC patients: mechanisms and strategies.

American journal of cancer research

Lin Y, Wang X, Jin H.
PMID: 25232485
Am J Cancer Res. 2014 Sep 06;4(5):411-35. eCollection 2014.

The epidermal growth factor receptor (EGFR) is a kind of receptor tyrosine kinase (RTK) that plays a critical role in the initiation and development of malignant tumors via modulating downstream signaling pathways. In non-small cell lung cancer (NSCLC), the...

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Lin Y, Wang X, Jin H. EGFR-TKI resistance in NSCLC patients: mechanisms and strategies. Am J Cancer Res. 2014;4(5):411-35
Lin, Y., Wang, X., & Jin, H. (2014). EGFR-TKI resistance in NSCLC patients: mechanisms and strategies. American journal of cancer research, 4(5), 411-35.
Lin, Yuxin, et al. "EGFR-TKI resistance in NSCLC patients: mechanisms and strategies." American journal of cancer research vol. 4,5 (2014): 411-35.
Lin Y, Wang X, Jin H. EGFR-TKI resistance in NSCLC patients: mechanisms and strategies. Am J Cancer Res. 2014 Sep 06;4(5):411-35. eCollection 2014. PMID: 25232485; PMCID: PMC4163608.
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Imbalance of the Nerve Growth Factor and Its Precursor: Implication in Diabetic Retinopathy.

Journal of clinical & experimental ophthalmology

Mohamed R, El-Remessy AB.
PMID: 26807305
J Clin Exp Ophthalmol. 2015 Oct;6(5). doi: 10.4172/2155-9570.1000483. Epub 2015 Oct 25.

Diabetic retinopathy is the leading cause of blindness in working age in US and worldwide. Neurotrophins including nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), neurotrophin-3 (NT-3) and neurotrophin-4 (NT-4) are known to be essential for growth, differentiation and...

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Mohamed R, El-Remessy AB. Imbalance of the Nerve Growth Factor and Its Precursor: Implication in Diabetic Retinopathy. J Clin Exp Ophthalmol. 2015;6(5)doi: 10.4172/2155-9570.1000483.
Mohamed, R., & El-Remessy, A. B. (2015). Imbalance of the Nerve Growth Factor and Its Precursor: Implication in Diabetic Retinopathy. Journal of clinical & experimental ophthalmology, 6(5), . https://doi.org/10.4172/2155-9570.1000483
Mohamed, Riyaz, and El-Remessy, Azza B. "Imbalance of the Nerve Growth Factor and Its Precursor: Implication in Diabetic Retinopathy." Journal of clinical & experimental ophthalmology vol. 6,5 (2015). doi: https://doi.org/10.4172/2155-9570.1000483
Mohamed R, El-Remessy AB. Imbalance of the Nerve Growth Factor and Its Precursor: Implication in Diabetic Retinopathy. J Clin Exp Ophthalmol. 2015 Oct;6(5). doi: 10.4172/2155-9570.1000483. Epub 2015 Oct 25. PMID: 26807305; PMCID: PMC4721560.
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Treadmill exercise alleviates nigrostriatal dopaminergic loss of neurons and fibers in rotenone-induced Parkinson rats.

Journal of exercise rehabilitation

Shin MS, Kim TW, Lee JM, Ji ES, Lim BV.
PMID: 28349030
J Exerc Rehabil. 2017 Feb 28;13(1):30-35. doi: 10.12965/jer.1734906.453. eCollection 2017 Feb.

Parkinson disease is one of the common brain diseases caused by dopaminergic neuronal loss in the substantia nigra and dopaminergic fiber loss in the striatum. In the present study, the effects of treadmill exercise on motor performance, dopaminergic loss...

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Shin MS, Kim TW, Lee JM, et al. Treadmill exercise alleviates nigrostriatal dopaminergic loss of neurons and fibers in rotenone-induced Parkinson rats. J Exerc Rehabil. 2017;13(1):30-35doi: 10.12965/jer.1734906.453.
Shin, M. S., Kim, T. W., Lee, J. M., Ji, E. S., & Lim, B. V. (2017). Treadmill exercise alleviates nigrostriatal dopaminergic loss of neurons and fibers in rotenone-induced Parkinson rats. Journal of exercise rehabilitation, 13(1), 30-35. https://doi.org/10.12965/jer.1734906.453
Shin, Mal-Soon, et al. "Treadmill exercise alleviates nigrostriatal dopaminergic loss of neurons and fibers in rotenone-induced Parkinson rats." Journal of exercise rehabilitation vol. 13,1 (2017): 30-35. doi: https://doi.org/10.12965/jer.1734906.453
Shin MS, Kim TW, Lee JM, Ji ES, Lim BV. Treadmill exercise alleviates nigrostriatal dopaminergic loss of neurons and fibers in rotenone-induced Parkinson rats. J Exerc Rehabil. 2017 Feb 28;13(1):30-35. doi: 10.12965/jer.1734906.453. eCollection 2017 Feb. PMID: 28349030; PMCID: PMC5331996.
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Effect of thaw temperatures in reducing enzyme activity in human thyroid tissues.

Biopreservation and biobanking

Kokkat TJ, McGarvey D, Lovecchio LC, LiVolsi VA.
PMID: 24836631
Biopreserv Biobank. 2011 Dec;9(4):349-54. doi: 10.1089/bio.2011.0026. Epub 2011 Nov 10.

An identified impediment to the advancement of science in the field of proteomics is the deterioration of proteins in tissue upon removal of the tissue from its natural state. To reduce this degradation, human tissues are frozen and stored...

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Kokkat TJ, McGarvey D, Lovecchio LC, et al. Effect of thaw temperatures in reducing enzyme activity in human thyroid tissues. Biopreserv Biobank. 2011;9(4):349-54doi: 10.1089/bio.2011.0026.
Kokkat, T. J., McGarvey, D., Lovecchio, L. C., & LiVolsi, V. A. (2011). Effect of thaw temperatures in reducing enzyme activity in human thyroid tissues. Biopreservation and biobanking, 9(4), 349-54. https://doi.org/10.1089/bio.2011.0026
Kokkat, Theresa J, et al. "Effect of thaw temperatures in reducing enzyme activity in human thyroid tissues." Biopreservation and biobanking vol. 9,4 (2011): 349-54. doi: https://doi.org/10.1089/bio.2011.0026
Kokkat TJ, McGarvey D, Lovecchio LC, LiVolsi VA. Effect of thaw temperatures in reducing enzyme activity in human thyroid tissues. Biopreserv Biobank. 2011 Dec;9(4):349-54. doi: 10.1089/bio.2011.0026. Epub 2011 Nov 10. PMID: 24836631.
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Amino acids inhibit kynurenic acid formation via suppression of kynurenine uptake or kynurenic acid synthesis in rat brain in vitro.

SpringerPlus

Sekine A, Okamoto M, Kanatani Y, Sano M, Shibata K, Fukuwatari T.
PMID: 25674503
Springerplus. 2015 Feb 01;4:48. doi: 10.1186/s40064-015-0826-9. eCollection 2015.

The tryptophan metabolite, kynurenic acid (KYNA), is a preferential antagonist of the α7 nicotinic acetylcholine receptor at endogenous brain concentrations. Recent studies have suggested that increase of brain KYNA levels is involved in psychiatric disorders such as schizophrenia and...

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Sekine A, Okamoto M, Kanatani Y, et al. Amino acids inhibit kynurenic acid formation via suppression of kynurenine uptake or kynurenic acid synthesis in rat brain in vitro. Springerplus. 2015;4:48doi: 10.1186/s40064-015-0826-9.
Sekine, A., Okamoto, M., Kanatani, Y., Sano, M., Shibata, K., & Fukuwatari, T. (2015). Amino acids inhibit kynurenic acid formation via suppression of kynurenine uptake or kynurenic acid synthesis in rat brain in vitro. SpringerPlus, 448. https://doi.org/10.1186/s40064-015-0826-9
Sekine, Airi, et al. "Amino acids inhibit kynurenic acid formation via suppression of kynurenine uptake or kynurenic acid synthesis in rat brain in vitro." SpringerPlus vol. 4 (2015): 48. doi: https://doi.org/10.1186/s40064-015-0826-9
Sekine A, Okamoto M, Kanatani Y, Sano M, Shibata K, Fukuwatari T. Amino acids inhibit kynurenic acid formation via suppression of kynurenine uptake or kynurenic acid synthesis in rat brain in vitro. Springerplus. 2015 Feb 01;4:48. doi: 10.1186/s40064-015-0826-9. eCollection 2015. PMID: 25674503; PMCID: PMC4318830.
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RRx-001-Induced Tumor Necrosis and Immune Cell Infiltration in an EGFR Mutation-Positive NSCLC with Resistance to EGFR Tyrosine Kinase Inhibitors: A Case Report.

Case reports in oncology

Brzezniak C, Schmitz BA, Peterson PG, Degesys A, Oronsky BT, Scicinski JJ, Caroen SZ, Carter CA.
PMID: 26933418
Case Rep Oncol. 2016 Jan 15;9(1):45-50. doi: 10.1159/000443605. eCollection 2016.

We present the case of a 49-year-old male with metastatic epidermal growth factor receptor (EGFR) mutation-positive adenocarcinoma of the lung that continues to outlive stage IV diagnosis of non-small cell lung cancer after treatment with RRx-001, an experimental anticancer...

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Brzezniak C, Schmitz BA, Peterson PG, et al. RRx-001-Induced Tumor Necrosis and Immune Cell Infiltration in an EGFR Mutation-Positive NSCLC with Resistance to EGFR Tyrosine Kinase Inhibitors: A Case Report. Case Rep Oncol. 2016;9(1):45-50doi: 10.1159/000443605.
Brzezniak, C., Schmitz, B. A., Peterson, P. G., Degesys, A., Oronsky, B. T., Scicinski, J. J., Caroen, S. Z., & Carter, C. A. (2016). RRx-001-Induced Tumor Necrosis and Immune Cell Infiltration in an EGFR Mutation-Positive NSCLC with Resistance to EGFR Tyrosine Kinase Inhibitors: A Case Report. Case reports in oncology, 9(1), 45-50. https://doi.org/10.1159/000443605
Brzezniak, Christina, et al. "RRx-001-Induced Tumor Necrosis and Immune Cell Infiltration in an EGFR Mutation-Positive NSCLC with Resistance to EGFR Tyrosine Kinase Inhibitors: A Case Report." Case reports in oncology vol. 9,1 (2016): 45-50. doi: https://doi.org/10.1159/000443605
Brzezniak C, Schmitz BA, Peterson PG, Degesys A, Oronsky BT, Scicinski JJ, Caroen SZ, Carter CA. RRx-001-Induced Tumor Necrosis and Immune Cell Infiltration in an EGFR Mutation-Positive NSCLC with Resistance to EGFR Tyrosine Kinase Inhibitors: A Case Report. Case Rep Oncol. 2016 Jan 15;9(1):45-50. doi: 10.1159/000443605. eCollection 2016. PMID: 26933418; PMCID: PMC4748757.
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Showing 13 to 24 of 16586 entries