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Showing 25 to 36 of 44 entries
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Physicobiochemical Characteristics and Chondrogenic Differentiation of Bone Marrow Mesenchymal Stem Cells (hBM-MSCs) in Biodegradable Porous Sponge Bovine Cartilage Scaffold.

International journal of biomaterials

Utomo DN, Mahyudin F, Wardhana TH, Purwati P, Brahmana F, Gusti AWR.
PMID: 30805001
Int J Biomater. 2019 Jan 20;2019:8356872. doi: 10.1155/2019/8356872. eCollection 2019.

Tissue engineering had been believed to overcome the limitation of cartilage lesions treatment. Nowadays the studies focus on using mesenchymal stem cells in scaffold. A biodegradable porous sponge bovine cartilage scaffold is expected to have the physicobiochemical characterization to...

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Utomo DN, Mahyudin F, Wardhana TH, et al. Physicobiochemical Characteristics and Chondrogenic Differentiation of Bone Marrow Mesenchymal Stem Cells (hBM-MSCs) in Biodegradable Porous Sponge Bovine Cartilage Scaffold. Int J Biomater. 2019;2019:8356872doi: 10.1155/2019/8356872.
Utomo, D. N., Mahyudin, F., Wardhana, T. H., Purwati, P., Brahmana, F., & Gusti, A. W. R. (2019). Physicobiochemical Characteristics and Chondrogenic Differentiation of Bone Marrow Mesenchymal Stem Cells (hBM-MSCs) in Biodegradable Porous Sponge Bovine Cartilage Scaffold. International journal of biomaterials, 20198356872. https://doi.org/10.1155/2019/8356872
Utomo, Dwikora Novembri, et al. "Physicobiochemical Characteristics and Chondrogenic Differentiation of Bone Marrow Mesenchymal Stem Cells (hBM-MSCs) in Biodegradable Porous Sponge Bovine Cartilage Scaffold." International journal of biomaterials vol. 2019 (2019): 8356872. doi: https://doi.org/10.1155/2019/8356872
Utomo DN, Mahyudin F, Wardhana TH, Purwati P, Brahmana F, Gusti AWR. Physicobiochemical Characteristics and Chondrogenic Differentiation of Bone Marrow Mesenchymal Stem Cells (hBM-MSCs) in Biodegradable Porous Sponge Bovine Cartilage Scaffold. Int J Biomater. 2019 Jan 20;2019:8356872. doi: 10.1155/2019/8356872. eCollection 2019. PMID: 30805001; PMCID: PMC6360592.
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Encapsulating TGF-β1 Inhibitory Peptides P17 and P144 as a Promising Strategy to Facilitate Their Dissolution and to Improve Their Functionalization.

Pharmaceutics

Hanafy NAN, Fabregat I, Leporatti S, Kemary ME.
PMID: 32370293
Pharmaceutics. 2020 May 02;12(5). doi: 10.3390/pharmaceutics12050421.

No abstract available.

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Hanafy NAN, Fabregat I, Leporatti S, et al. Encapsulating TGF-β1 Inhibitory Peptides P17 and P144 as a Promising Strategy to Facilitate Their Dissolution and to Improve Their Functionalization. Pharmaceutics. 2020;12(5)doi: 10.3390/pharmaceutics12050421.
Hanafy, N. A. N., Fabregat, I., Leporatti, S., & Kemary, M. E. (2020). Encapsulating TGF-β1 Inhibitory Peptides P17 and P144 as a Promising Strategy to Facilitate Their Dissolution and to Improve Their Functionalization. Pharmaceutics, 12(5), . https://doi.org/10.3390/pharmaceutics12050421
Hanafy, Nemany A N, et al. "Encapsulating TGF-β1 Inhibitory Peptides P17 and P144 as a Promising Strategy to Facilitate Their Dissolution and to Improve Their Functionalization." Pharmaceutics vol. 12,5 (2020). doi: https://doi.org/10.3390/pharmaceutics12050421
Hanafy NAN, Fabregat I, Leporatti S, Kemary ME. Encapsulating TGF-β1 Inhibitory Peptides P17 and P144 as a Promising Strategy to Facilitate Their Dissolution and to Improve Their Functionalization. Pharmaceutics. 2020 May 02;12(5). doi: 10.3390/pharmaceutics12050421. PMID: 32370293; PMCID: PMC7284799.
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Transforming growth factor-beta family members are regulated during induced luteolysis in cattle.

Animal reproduction

Haas CS, Rovani MT, Ilha GF, Bertolin K, Ferst JG, Bridi A, Bordignon V, Duggavathi R, Antoniazzi AQ, Gonçalves PBD, Gasperin BG.
PMID: 32368260
Anim Reprod. 2019 Nov 18;16(4):829-837. doi: 10.21451/1984-3143-AR2018-0146.

The transforming growth factors beta (TGFβ) are local factors produced by ovarian cells which, after binding to their receptors, regulate follicular deviation and ovulation. However, their regulation and function during corpus luteum (CL) regression has been poorly investigated. The...

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Haas CS, Rovani MT, Ilha GF, et al. Transforming growth factor-beta family members are regulated during induced luteolysis in cattle. Anim Reprod. 2019;16(4):829-837doi: 10.21451/1984-3143-AR2018-0146.
Haas, C. S., Rovani, M. T., Ilha, G. F., Bertolin, K., Ferst, J. G., Bridi, A., Bordignon, V., Duggavathi, R., Antoniazzi, A. Q., Gonçalves, P. B. D., & Gasperin, B. G. (2019). Transforming growth factor-beta family members are regulated during induced luteolysis in cattle. Animal reproduction, 16(4), 829-837. https://doi.org/10.21451/1984-3143-AR2018-0146
Haas, Cristina Sangoi, et al. "Transforming growth factor-beta family members are regulated during induced luteolysis in cattle." Animal reproduction vol. 16,4 (2019): 829-837. doi: https://doi.org/10.21451/1984-3143-AR2018-0146
Haas CS, Rovani MT, Ilha GF, Bertolin K, Ferst JG, Bridi A, Bordignon V, Duggavathi R, Antoniazzi AQ, Gonçalves PBD, Gasperin BG. Transforming growth factor-beta family members are regulated during induced luteolysis in cattle. Anim Reprod. 2019 Nov 18;16(4):829-837. doi: 10.21451/1984-3143-AR2018-0146. PMID: 32368260; PMCID: PMC7189511.
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Crosstalk Between Inflammatory Signaling and Methylation in Cancer.

Frontiers in cell and developmental biology

Das D, Karthik N, Taneja R.
PMID: 34901003
Front Cell Dev Biol. 2021 Nov 24;9:756458. doi: 10.3389/fcell.2021.756458. eCollection 2021.

Inflammation is an intricate immune response against infection and tissue damage. While the initial immune response is important for preventing tumorigenesis, chronic inflammation is implicated in cancer pathogenesis. It has been linked to various stages of tumor development including...

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Das D, Karthik N, Taneja R. Crosstalk Between Inflammatory Signaling and Methylation in Cancer. Front Cell Dev Biol. 2021;9:756458doi: 10.3389/fcell.2021.756458.
Das, D., Karthik, N., & Taneja, R. (2021). Crosstalk Between Inflammatory Signaling and Methylation in Cancer. Frontiers in cell and developmental biology, 9756458. https://doi.org/10.3389/fcell.2021.756458
Das, Dipanwita, et al. "Crosstalk Between Inflammatory Signaling and Methylation in Cancer." Frontiers in cell and developmental biology vol. 9 (2021): 756458. doi: https://doi.org/10.3389/fcell.2021.756458
Das D, Karthik N, Taneja R. Crosstalk Between Inflammatory Signaling and Methylation in Cancer. Front Cell Dev Biol. 2021 Nov 24;9:756458. doi: 10.3389/fcell.2021.756458. eCollection 2021. PMID: 34901003; PMCID: PMC8652226.
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Cerebrospinal fluid predictors of shunt-dependent hydrocephalus after hemorrhagic stroke: a systematic review and meta-analysis.

Neurosurgical review

Yang YC, Liu SH, Hsu YH, Wu YL, Chu PT, Lin PC.
PMID: 35015193
Neurosurg Rev. 2022 Jan 11; doi: 10.1007/s10143-022-01731-5. Epub 2022 Jan 11.

Hydrocephalus is a common complication of hemorrhagic stroke and has been reported to contribute to poor neurological outcomes. Herein, we aimed to investigate the validity of cerebrospinal fluid (CSF) data in predicting shunt-dependent hydrocephalus (SDHC) in patients with hemorrhagic...

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Yang YC, Liu SH, Hsu YH, et al. Cerebrospinal fluid predictors of shunt-dependent hydrocephalus after hemorrhagic stroke: a systematic review and meta-analysis. Neurosurg Rev. 2022;doi: 10.1007/s10143-022-01731-5.
Yang, Y. C., Liu, S. H., Hsu, Y. H., Wu, Y. L., Chu, P. T., & Lin, P. C. (2022). Cerebrospinal fluid predictors of shunt-dependent hydrocephalus after hemorrhagic stroke: a systematic review and meta-analysis. Neurosurgical review, . https://doi.org/10.1007/s10143-022-01731-5
Yang, Yao-Chung, et al. "Cerebrospinal fluid predictors of shunt-dependent hydrocephalus after hemorrhagic stroke: a systematic review and meta-analysis." Neurosurgical review vol. (2022). doi: https://doi.org/10.1007/s10143-022-01731-5
Yang YC, Liu SH, Hsu YH, Wu YL, Chu PT, Lin PC. Cerebrospinal fluid predictors of shunt-dependent hydrocephalus after hemorrhagic stroke: a systematic review and meta-analysis. Neurosurg Rev. 2022 Jan 11; doi: 10.1007/s10143-022-01731-5. Epub 2022 Jan 11. PMID: 35015193.
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Fibrin and Transforming Growth Factor Alpha Affect Prostatic Smooth Muscle Cell's Phenotype and Motility.

Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada

Osório DA, Consonni SR, Dos Santos AM, Carvalho HF.
PMID: 33691831
Microsc Microanal. 2021 Mar 11;1-8. doi: 10.1017/S1431927621000209. Epub 2021 Mar 11.

Smooth muscle cells (SMCs) are dynamic and transition from a contractile to a synthetic phenotype under different circumstances. Plasma factors (fibrin and transforming growth factors, TGFs) are possible components affecting SMCs differentiation and behavior. Thus, the objective of this...

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Osório DA, Consonni SR, Dos Santos AM, et al. Fibrin and Transforming Growth Factor Alpha Affect Prostatic Smooth Muscle Cell's Phenotype and Motility. Microsc Microanal. 2021;1-8doi: 10.1017/S1431927621000209.
Osório, D. A., Consonni, S. R., Dos Santos, A. M., & Carvalho, H. F. (2021). Fibrin and Transforming Growth Factor Alpha Affect Prostatic Smooth Muscle Cell's Phenotype and Motility. Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada, 1-8. https://doi.org/10.1017/S1431927621000209
Osório, Daniel A, et al. "Fibrin and Transforming Growth Factor Alpha Affect Prostatic Smooth Muscle Cell's Phenotype and Motility." Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada vol. (2021): 1-8. doi: https://doi.org/10.1017/S1431927621000209
Osório DA, Consonni SR, Dos Santos AM, Carvalho HF. Fibrin and Transforming Growth Factor Alpha Affect Prostatic Smooth Muscle Cell's Phenotype and Motility. Microsc Microanal. 2021 Mar 11;1-8. doi: 10.1017/S1431927621000209. Epub 2021 Mar 11. PMID: 33691831.
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[Myostatin - a modern understanding of the physiological role and significance in the development of age-associated diseases.].

Advances in gerontology = Uspekhi gerontologii

Gazdanova AA, Kukes VG, Parfenova OK, Sidorov NG, Perkov AV, Solovieva SA, Ryazantceva OV, Lenkova NI.
PMID: 34998007
Adv Gerontol. 2021;34(5):701-706.

Myostatin is a protein belonging to the myokine class, the family of transforming growth factors β (TGF-β). The review article, based on the analysis of literature data, shows the key role of myostatin in the development of senile sarcopenia...

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Gazdanova AA, Kukes VG, Parfenova OK, et al. [Myostatin - a modern understanding of the physiological role and significance in the development of age-associated diseases.]. Adv Gerontol. 2021;34(5):701-706
Gazdanova, A. A., Kukes, V. G., Parfenova, O. K., Sidorov, N. G., Perkov, A. V., Solovieva, S. A., Ryazantceva, O. V., & Lenkova, N. I. (2021). [Myostatin - a modern understanding of the physiological role and significance in the development of age-associated diseases.]. Advances in gerontology = Uspekhi gerontologii, 34(5), 701-706.
Gazdanova, A A, et al. "[Myostatin - a modern understanding of the physiological role and significance in the development of age-associated diseases.]." Advances in gerontology = Uspekhi gerontologii vol. 34,5 (2021): 701-706.
Gazdanova AA, Kukes VG, Parfenova OK, Sidorov NG, Perkov AV, Solovieva SA, Ryazantceva OV, Lenkova NI. [Myostatin - a modern understanding of the physiological role and significance in the development of age-associated diseases.]. Adv Gerontol. 2021;34(5):701-706. Russian. PMID: 34998007.
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Interplay of retinal determination gene network with TGF-β signaling pathway in epithelial-mesenchymal transition.

Stem cell investigation

Liu Y, Kong D, Wu H, Yuan X, Xu H, Zhang C, Wu G, Wu K.
PMID: 27358880
Stem Cell Investig. 2015 Jun 09;2:12. doi: 10.3978/j.issn.2306-9759.2015.05.03. eCollection 2015.

As a fundamental event in the generation of tissues and organs during embryogenesis, the epithelial-mesenchymal transition (EMT) has also been implicated in cancer progression by its ability to alter the plasticity of epithelial cells to acquire invasive properties. Evidence...

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Liu Y, Kong D, Wu H, et al. Interplay of retinal determination gene network with TGF-β signaling pathway in epithelial-mesenchymal transition. Stem Cell Investig. 2015;2:12doi: 10.3978/j.issn.2306-9759.2015.05.03.
Liu, Y., Kong, D., Wu, H., Yuan, X., Xu, H., Zhang, C., Wu, G., & Wu, K. (2015). Interplay of retinal determination gene network with TGF-β signaling pathway in epithelial-mesenchymal transition. Stem cell investigation, 212. https://doi.org/10.3978/j.issn.2306-9759.2015.05.03
Liu, Yu, et al. "Interplay of retinal determination gene network with TGF-β signaling pathway in epithelial-mesenchymal transition." Stem cell investigation vol. 2 (2015): 12. doi: https://doi.org/10.3978/j.issn.2306-9759.2015.05.03
Liu Y, Kong D, Wu H, Yuan X, Xu H, Zhang C, Wu G, Wu K. Interplay of retinal determination gene network with TGF-β signaling pathway in epithelial-mesenchymal transition. Stem Cell Investig. 2015 Jun 09;2:12. doi: 10.3978/j.issn.2306-9759.2015.05.03. eCollection 2015. PMID: 27358880; PMCID: PMC4923650.
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Evolving therapies in the treatment of hepatocellular carcinoma.

Biologics : targets & therapy

Spangenberg HC, Thimme R, Blum HE.
PMID: 19707376
Biologics. 2008 Sep;2(3):453-62. doi: 10.2147/btt.s3254.

Hepatocellular carcinoma (HCC) is one of the most common malignant tumors worldwide. The major etiologies and risk factors for HCC development are well defined and some of the steps involved in hepatocarcinogenesis have been elucidated in recent years. Therapeutic...

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Spangenberg HC, Thimme R, Blum HE. Evolving therapies in the treatment of hepatocellular carcinoma. Biologics. 2008;2(3):453-62doi: 10.2147/btt.s3254.
Spangenberg, H. C., Thimme, R., & Blum, H. E. (2008). Evolving therapies in the treatment of hepatocellular carcinoma. Biologics : targets & therapy, 2(3), 453-62. https://doi.org/10.2147/btt.s3254
Spangenberg, Hans Christian, et al. "Evolving therapies in the treatment of hepatocellular carcinoma." Biologics : targets & therapy vol. 2,3 (2008): 453-62. doi: https://doi.org/10.2147/btt.s3254
Spangenberg HC, Thimme R, Blum HE. Evolving therapies in the treatment of hepatocellular carcinoma. Biologics. 2008 Sep;2(3):453-62. doi: 10.2147/btt.s3254. PMID: 19707376; PMCID: PMC2721397.
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Differential response to transforming growth factors-beta(1) and beta(2): connective tissue deposition in animal models.

Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society

Ogawa Y, Gerhardt CO, Ksander GA.
PMID: 17147660
Wound Repair Regen. 1995 Oct-Dec;3(4):485-91. doi: 10.1046/j.1524-475X.1995.30413.x.

The ability of transforming growth factor (TGF)-beta(1) and TGF-beta(2) to promote connective tissue deposition were compared in different animal models. A single subcutaneous injection of TGF-beta(2) in collagen/heparin gel carrier promoted markedly more extensive development of connective tissue than...

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Ogawa Y, Gerhardt CO, Ksander GA. Differential response to transforming growth factors-beta(1) and beta(2): connective tissue deposition in animal models. Wound Repair Regen. 1995;3(4):485-91doi: 10.1046/j.1524-475X.1995.30413.x.
Ogawa, Y., Gerhardt, C. O., & Ksander, G. A. (1995). Differential response to transforming growth factors-beta(1) and beta(2): connective tissue deposition in animal models. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society, 3(4), 485-91. https://doi.org/10.1046/j.1524-475X.1995.30413.x
Ogawa, Y, et al. "Differential response to transforming growth factors-beta(1) and beta(2): connective tissue deposition in animal models." Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society vol. 3,4 (1995): 485-91. doi: https://doi.org/10.1046/j.1524-475X.1995.30413.x
Ogawa Y, Gerhardt CO, Ksander GA. Differential response to transforming growth factors-beta(1) and beta(2): connective tissue deposition in animal models. Wound Repair Regen. 1995 Oct-Dec;3(4):485-91. doi: 10.1046/j.1524-475X.1995.30413.x. PMID: 17147660.
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Platelet-rich plasma: quantitative assessment of growth factor levels and comparative analysis of activated and inactivated groups.

Archives of plastic surgery

Lee JW, Kwon OH, Kim TK, Cho YK, Choi KY, Chung HY, Cho BC, Yang JD, Shin JH.
PMID: 24086805
Arch Plast Surg. 2013 Sep;40(5):530-5. doi: 10.5999/aps.2013.40.5.530. Epub 2013 Sep 13.

BACKGROUND: Platelet-rich plasma (PRP) has more concentrated platelets than normal plasma (approximately 150-400×10(3) cell/dL). Platelets excrete several growth factors and cytokines that are associated with the healing and regeneration process. However, even though PRP is widely used, the mechanism...

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Lee JW, Kwon OH, Kim TK, et al. Platelet-rich plasma: quantitative assessment of growth factor levels and comparative analysis of activated and inactivated groups. Arch Plast Surg. 2013;40(5):530-5doi: 10.5999/aps.2013.40.5.530.
Lee, J. W., Kwon, O. H., Kim, T. K., Cho, Y. K., Choi, K. Y., Chung, H. Y., Cho, B. C., Yang, J. D., & Shin, J. H. (2013). Platelet-rich plasma: quantitative assessment of growth factor levels and comparative analysis of activated and inactivated groups. Archives of plastic surgery, 40(5), 530-5. https://doi.org/10.5999/aps.2013.40.5.530
Lee, Jeong Woo, et al. "Platelet-rich plasma: quantitative assessment of growth factor levels and comparative analysis of activated and inactivated groups." Archives of plastic surgery vol. 40,5 (2013): 530-5. doi: https://doi.org/10.5999/aps.2013.40.5.530
Lee JW, Kwon OH, Kim TK, Cho YK, Choi KY, Chung HY, Cho BC, Yang JD, Shin JH. Platelet-rich plasma: quantitative assessment of growth factor levels and comparative analysis of activated and inactivated groups. Arch Plast Surg. 2013 Sep;40(5):530-5. doi: 10.5999/aps.2013.40.5.530. Epub 2013 Sep 13. PMID: 24086805; PMCID: PMC3785585.
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Effect of Myricetin on Primary Open-angle Glaucoma.

Translational neuroscience

Yang Q, Li Y, Luo L.
PMID: 30473883
Transl Neurosci. 2018 Nov 12;9:132-141. doi: 10.1515/tnsci-2018-0020. eCollection 2018.

BACKGROUND: Primary open angle glaucoma (POAG) is the most common form of glaucoma, with a multifactorial etiology that results in retinal ganglion cell death and loss of vision. In this study, we assessed the effects of myricetin on the...

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Yang Q, Li Y, Luo L. Effect of Myricetin on Primary Open-angle Glaucoma. Transl Neurosci. 2018;9:132-141doi: 10.1515/tnsci-2018-0020.
Yang, Q., Li, Y., & Luo, L. (2018). Effect of Myricetin on Primary Open-angle Glaucoma. Translational neuroscience, 9132-141. https://doi.org/10.1515/tnsci-2018-0020
Yang, Qing, et al. "Effect of Myricetin on Primary Open-angle Glaucoma." Translational neuroscience vol. 9 (2018): 132-141. doi: https://doi.org/10.1515/tnsci-2018-0020
Yang Q, Li Y, Luo L. Effect of Myricetin on Primary Open-angle Glaucoma. Transl Neurosci. 2018 Nov 12;9:132-141. doi: 10.1515/tnsci-2018-0020. eCollection 2018. PMID: 30473883; PMCID: PMC6234474.
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Showing 25 to 36 of 44 entries