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Showing 25 to 36 of 148 entries
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SAIL: a new conserved anti-fibrotic lncRNA in the heart.

Basic research in cardiology

Luo S, Zhang M, Wu H, Ding X, Li D, Dong X, Hu X, Su S, Shang W, Wu J, Xiao H, Yang W, Zhang Q, Zhang J, Lu Y, Pan Z.
PMID: 33675440
Basic Res Cardiol. 2021 Mar 06;116(1):15. doi: 10.1007/s00395-021-00854-y.

Long non-coding RNAs (lncRNAs) account for a large proportion of genomic transcripts and are critical regulators in various cardiac diseases. Though lncRNAs have been reported to participate in the process of diverse cardiac diseases, the contribution of lncRNAs in...

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Luo S, Zhang M, Wu H, et al. SAIL: a new conserved anti-fibrotic lncRNA in the heart. Basic Res Cardiol. 2021;116(1):15doi: 10.1007/s00395-021-00854-y.
Luo, S., Zhang, M., Wu, H., Ding, X., Li, D., Dong, X., Hu, X., Su, S., Shang, W., Wu, J., Xiao, H., Yang, W., Zhang, Q., Zhang, J., Lu, Y., & Pan, Z. (2021). SAIL: a new conserved anti-fibrotic lncRNA in the heart. Basic research in cardiology, 116(1), 15. https://doi.org/10.1007/s00395-021-00854-y
Luo, Shenjian, et al. "SAIL: a new conserved anti-fibrotic lncRNA in the heart." Basic research in cardiology vol. 116,1 (2021): 15. doi: https://doi.org/10.1007/s00395-021-00854-y
Luo S, Zhang M, Wu H, Ding X, Li D, Dong X, Hu X, Su S, Shang W, Wu J, Xiao H, Yang W, Zhang Q, Zhang J, Lu Y, Pan Z. SAIL: a new conserved anti-fibrotic lncRNA in the heart. Basic Res Cardiol. 2021 Mar 06;116(1):15. doi: 10.1007/s00395-021-00854-y. PMID: 33675440.
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Attenuated TGFB signalling in macrophages decreases susceptibility to DMBA-induced mammary cancer in mice.

Breast cancer research : BCR

Sun X, Bernhardt SM, Glynn DJ, Hodson LJ, Woolford L, Evdokiou A, Yan C, Du H, Robertson SA, Ingman WV.
PMID: 33761981
Breast Cancer Res. 2021 Mar 24;23(1):39. doi: 10.1186/s13058-021-01417-8.

BACKGROUND: Transforming growth factor beta1 (TGFB1) is a multi-functional cytokine that regulates mammary gland development and cancer progression through endocrine, paracrine and autocrine mechanisms. TGFB1 also plays roles in tumour development and progression, and its increased expression is associated...

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Sun X, Bernhardt SM, Glynn DJ, et al. Attenuated TGFB signalling in macrophages decreases susceptibility to DMBA-induced mammary cancer in mice. Breast Cancer Res. 2021;23(1):39doi: 10.1186/s13058-021-01417-8.
Sun, X., Bernhardt, S. M., Glynn, D. J., Hodson, L. J., Woolford, L., Evdokiou, A., Yan, C., Du, H., Robertson, S. A., & Ingman, W. V. (2021). Attenuated TGFB signalling in macrophages decreases susceptibility to DMBA-induced mammary cancer in mice. Breast cancer research : BCR, 23(1), 39. https://doi.org/10.1186/s13058-021-01417-8
Sun, Xuan, et al. "Attenuated TGFB signalling in macrophages decreases susceptibility to DMBA-induced mammary cancer in mice." Breast cancer research : BCR vol. 23,1 (2021): 39. doi: https://doi.org/10.1186/s13058-021-01417-8
Sun X, Bernhardt SM, Glynn DJ, Hodson LJ, Woolford L, Evdokiou A, Yan C, Du H, Robertson SA, Ingman WV. Attenuated TGFB signalling in macrophages decreases susceptibility to DMBA-induced mammary cancer in mice. Breast Cancer Res. 2021 Mar 24;23(1):39. doi: 10.1186/s13058-021-01417-8. PMID: 33761981; PMCID: PMC7992865.
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Transglutaminase 2 mediates lung inflammation and remodeling by transforming growth factor beta 1 .

Experimental lung research

Kim YC, Kim J, Kim S, Bae B, Kim RL, Jeong EM, Cho SH, Kang HR.
PMID: 34818962
Exp Lung Res. 2021 Dec;47(10):465-475. doi: 10.1080/01902148.2021.1998733. Epub 2021 Nov 24.

Transforming growth factor beta 1 (TGF-β1) induces pulmonary fibrosis by enhancing epithelial apoptosis and affects the enzymatic activity of transglutaminase 2 (TG2). The aim of this study was to determine the role of TG2 in TGF-β1-induced lung remodeling and...

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Kim YC, Kim J, Kim S, et al. Transglutaminase 2 mediates lung inflammation and remodeling by transforming growth factor beta 1 . Exp Lung Res. 2021;47(10):465-475doi: 10.1080/01902148.2021.1998733.
Kim, Y. C., Kim, J., Kim, S., Bae, B., Kim, R. L., Jeong, E. M., Cho, S. H., & Kang, H. R. (2021). Transglutaminase 2 mediates lung inflammation and remodeling by transforming growth factor beta 1 . Experimental lung research, 47(10), 465-475. https://doi.org/10.1080/01902148.2021.1998733
Kim, Young Chan, et al. "Transglutaminase 2 mediates lung inflammation and remodeling by transforming growth factor beta 1 ." Experimental lung research vol. 47,10 (2021): 465-475. doi: https://doi.org/10.1080/01902148.2021.1998733
Kim YC, Kim J, Kim S, Bae B, Kim RL, Jeong EM, Cho SH, Kang HR. Transglutaminase 2 mediates lung inflammation and remodeling by transforming growth factor beta 1 . Exp Lung Res. 2021 Dec;47(10):465-475. doi: 10.1080/01902148.2021.1998733. Epub 2021 Nov 24. PMID: 34818962.
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Combination gene therapy with adenoviral vector-mediated HSV-tk+GCV and IL-12 in an orthotopic mouse model for prostate cancer.

Prostate cancer and prostatic diseases

Nasu Y, Bangma CH, Hull GW, Yang G, Wang J, Shimura S, McCurdy MA, Ebara S, Lee HM, Timme TL, Thompson TC.
PMID: 12497062
Prostate Cancer Prostatic Dis. 2001;4(1):44-55. doi: 10.1038/sj.pcan.4500494.

We previously demonstrated significant therapeutic activities associated with adenoviral vector-mediated Herpes Simplex Virus/thymidine kinase (AdHSV-tk) with ganciclovir (GCV) in situ gene therapy in the RM-1 orthotopic mouse prostate cancer model and interleukin-12 (AdmIL-12) in situ gene therapy in the...

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Nasu Y, Bangma CH, Hull GW, et al. Combination gene therapy with adenoviral vector-mediated HSV-tk+GCV and IL-12 in an orthotopic mouse model for prostate cancer. Prostate Cancer Prostatic Dis. 2001;4(1):44-55doi: 10.1038/sj.pcan.4500494.
Nasu, Y., Bangma, C. H., Hull, G. W., Yang, G., Wang, J., Shimura, S., McCurdy, M. A., Ebara, S., Lee, H. M., Timme, T. L., & Thompson, T. C. (2001). Combination gene therapy with adenoviral vector-mediated HSV-tk+GCV and IL-12 in an orthotopic mouse model for prostate cancer. Prostate cancer and prostatic diseases, 4(1), 44-55. https://doi.org/10.1038/sj.pcan.4500494
Nasu, Y, et al. "Combination gene therapy with adenoviral vector-mediated HSV-tk+GCV and IL-12 in an orthotopic mouse model for prostate cancer." Prostate cancer and prostatic diseases vol. 4,1 (2001): 44-55. doi: https://doi.org/10.1038/sj.pcan.4500494
Nasu Y, Bangma CH, Hull GW, Yang G, Wang J, Shimura S, McCurdy MA, Ebara S, Lee HM, Timme TL, Thompson TC. Combination gene therapy with adenoviral vector-mediated HSV-tk+GCV and IL-12 in an orthotopic mouse model for prostate cancer. Prostate Cancer Prostatic Dis. 2001;4(1):44-55. doi: 10.1038/sj.pcan.4500494. PMID: 12497062.
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Peroxisome Proliferator-Activated Receptor Gamma Agonist Attenuates Liver Fibrosis by Several Fibrogenic Pathways in an Animal Model of Cholestatic Fibrosis.

Pediatric gastroenterology, hepatology & nutrition

Alatas FS, Matsuura T, Pudjiadi AH, Wijaya S, Taguchi T, Anderson MC, Kustas WP, Alfieri JG, Gao F.
PMID: 32704495
Pediatr Gastroenterol Hepatol Nutr. 2020 Jul;23(4):346-355. doi: 10.5223/pghn.2020.23.4.346. Epub 2020 Jul 03.

PURPOSE: Peroxisome proliferator-activated receptor gamma (PPAR-γ) has a key role in hepatic fibrogenesis by virtue of its effect on the hepatic stellate cells (HSCs). Although many studies have shown that PPAR-γ agonists inhibit liver fibrosis, the mechanism remains largely...

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Alatas FS, Matsuura T, Pudjiadi AH, et al. Peroxisome Proliferator-Activated Receptor Gamma Agonist Attenuates Liver Fibrosis by Several Fibrogenic Pathways in an Animal Model of Cholestatic Fibrosis. Pediatr Gastroenterol Hepatol Nutr. 2020;23(4):346-355doi: 10.5223/pghn.2020.23.4.346.
Alatas, F. S., Matsuura, T., Pudjiadi, A. H., Wijaya, S., Taguchi, T., Anderson, M. C., Kustas, W. P., Alfieri, J. G., & Gao, F. (2020). Peroxisome Proliferator-Activated Receptor Gamma Agonist Attenuates Liver Fibrosis by Several Fibrogenic Pathways in an Animal Model of Cholestatic Fibrosis. Pediatric gastroenterology, hepatology & nutrition, 23(4), 346-355. https://doi.org/10.5223/pghn.2020.23.4.346
Alatas, Fatima Safira, et al. "Peroxisome Proliferator-Activated Receptor Gamma Agonist Attenuates Liver Fibrosis by Several Fibrogenic Pathways in an Animal Model of Cholestatic Fibrosis." Pediatric gastroenterology, hepatology & nutrition vol. 23,4 (2020): 346-355. doi: https://doi.org/10.5223/pghn.2020.23.4.346
Alatas FS, Matsuura T, Pudjiadi AH, Wijaya S, Taguchi T, Anderson MC, Kustas WP, Alfieri JG, Gao F. Peroxisome Proliferator-Activated Receptor Gamma Agonist Attenuates Liver Fibrosis by Several Fibrogenic Pathways in an Animal Model of Cholestatic Fibrosis. Pediatr Gastroenterol Hepatol Nutr. 2020 Jul;23(4):346-355. doi: 10.5223/pghn.2020.23.4.346. Epub 2020 Jul 03. PMID: 32704495; PMCID: PMC7354870.
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Empagliflozin Protects Against Proximal Renal Tubular Cell Injury Induced by High Glucose via Regulation of Hypoxia-Inducible Factor 1-Alpha.

Diabetes, metabolic syndrome and obesity : targets and therapy

Ndibalema AR, Kabuye D, Wen S, Li L, Li X, Fan Q.
PMID: 32606855
Diabetes Metab Syndr Obes. 2020 Jun 12;13:1953-1967. doi: 10.2147/DMSO.S243170. eCollection 2020.

BACKGROUND: Evidence from both animal and human studies clearly supports the renal beneficial effects of empagliflozin (emp), a sodium glucose co-transporter 2 (SGLT2) inhibitor, but the mechanism in which it exerts its effect is not well understood. In this...

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Ndibalema AR, Kabuye D, Wen S, et al. Empagliflozin Protects Against Proximal Renal Tubular Cell Injury Induced by High Glucose via Regulation of Hypoxia-Inducible Factor 1-Alpha. Diabetes Metab Syndr Obes. 2020;13:1953-1967doi: 10.2147/DMSO.S243170.
Ndibalema, A. R., Kabuye, D., Wen, S., Li, L., Li, X., & Fan, Q. (2020). Empagliflozin Protects Against Proximal Renal Tubular Cell Injury Induced by High Glucose via Regulation of Hypoxia-Inducible Factor 1-Alpha. Diabetes, metabolic syndrome and obesity : targets and therapy, 131953-1967. https://doi.org/10.2147/DMSO.S243170
Ndibalema, Angelamellisy Revelian, et al. "Empagliflozin Protects Against Proximal Renal Tubular Cell Injury Induced by High Glucose via Regulation of Hypoxia-Inducible Factor 1-Alpha." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 13 (2020): 1953-1967. doi: https://doi.org/10.2147/DMSO.S243170
Ndibalema AR, Kabuye D, Wen S, Li L, Li X, Fan Q. Empagliflozin Protects Against Proximal Renal Tubular Cell Injury Induced by High Glucose via Regulation of Hypoxia-Inducible Factor 1-Alpha. Diabetes Metab Syndr Obes. 2020 Jun 12;13:1953-1967. doi: 10.2147/DMSO.S243170. eCollection 2020. PMID: 32606855; PMCID: PMC7297363.
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Pathways Connecting Late-Life Depression and Dementia.

Frontiers in pharmacology

Linnemann C, Lang UE.
PMID: 32231570
Front Pharmacol. 2020 Mar 13;11:279. doi: 10.3389/fphar.2020.00279. eCollection 2020.

Late-life depression is associated with significant cognitive impairment. Meta-analyses showed that depression is associated with an increased risk for Alzheimer's disease (AD) and it might be an etiological factor for AD. Since late-life depression is often connected with cognitive...

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Linnemann C, Lang UE. Pathways Connecting Late-Life Depression and Dementia. Front Pharmacol. 2020;11:279doi: 10.3389/fphar.2020.00279.
Linnemann, C., & Lang, U. E. (2020). Pathways Connecting Late-Life Depression and Dementia. Frontiers in pharmacology, 11279. https://doi.org/10.3389/fphar.2020.00279
Linnemann, Christoph, and Lang, Undine E. "Pathways Connecting Late-Life Depression and Dementia." Frontiers in pharmacology vol. 11 (2020): 279. doi: https://doi.org/10.3389/fphar.2020.00279
Linnemann C, Lang UE. Pathways Connecting Late-Life Depression and Dementia. Front Pharmacol. 2020 Mar 13;11:279. doi: 10.3389/fphar.2020.00279. eCollection 2020. PMID: 32231570; PMCID: PMC7083108.
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Inhibitors of DNA methylation support TGF-β1-induced .

Journal of periodontal & implant science

Sufaru IG, Beikircher G, Weinhaeusel A, Gruber R.
PMID: 28462005
J Periodontal Implant Sci. 2017 Apr;47(2):66-76. doi: 10.5051/jpis.2017.47.2.66. Epub 2017 Apr 29.

PURPOSE: Oral wound healing requires gingival fibroblasts to respond to local growth factors. Epigenetic silencing through DNA methylation can potentially decrease the responsiveness of gingival fibroblasts to local growth factors. In this study, our aim was to determine whether...

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Sufaru IG, Beikircher G, Weinhaeusel A, et al. Inhibitors of DNA methylation support TGF-β1-induced . J Periodontal Implant Sci. 2017;47(2):66-76doi: 10.5051/jpis.2017.47.2.66.
Sufaru, I. G., Beikircher, G., Weinhaeusel, A., & Gruber, R. (2017). Inhibitors of DNA methylation support TGF-β1-induced . Journal of periodontal & implant science, 47(2), 66-76. https://doi.org/10.5051/jpis.2017.47.2.66
Sufaru, Irina-Georgeta, et al. "Inhibitors of DNA methylation support TGF-β1-induced ." Journal of periodontal & implant science vol. 47,2 (2017): 66-76. doi: https://doi.org/10.5051/jpis.2017.47.2.66
Sufaru IG, Beikircher G, Weinhaeusel A, Gruber R. Inhibitors of DNA methylation support TGF-β1-induced . J Periodontal Implant Sci. 2017 Apr;47(2):66-76. doi: 10.5051/jpis.2017.47.2.66. Epub 2017 Apr 29. PMID: 28462005; PMCID: PMC5410554.
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Latent Transforming Growth Factor-beta1 Functionalised Electrospun Scaffolds Promote Human Cartilage Differentiation: Towards an Engineered Cartilage Construct.

Archives of plastic surgery

Lim EH, Sardinha JP, Myers S, Stevens M.
PMID: 24286039
Arch Plast Surg. 2013 Nov;40(6):676-86. doi: 10.5999/aps.2013.40.6.676. Epub 2013 Nov 08.

BACKGROUND: To overcome the potential drawbacks of a short half-life and dose-related adverse effects of using active transforming growth factor-beta 1 for cartilage engineering, a cell-mediated latent growth factor activation strategy was developed incorporating latent transforming growth factor-β1 (LTGF)...

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Lim EH, Sardinha JP, Myers S, et al. Latent Transforming Growth Factor-beta1 Functionalised Electrospun Scaffolds Promote Human Cartilage Differentiation: Towards an Engineered Cartilage Construct. Arch Plast Surg. 2013;40(6):676-86doi: 10.5999/aps.2013.40.6.676.
Lim, E. H., Sardinha, J. P., Myers, S., & Stevens, M. (2013). Latent Transforming Growth Factor-beta1 Functionalised Electrospun Scaffolds Promote Human Cartilage Differentiation: Towards an Engineered Cartilage Construct. Archives of plastic surgery, 40(6), 676-86. https://doi.org/10.5999/aps.2013.40.6.676
Lim, Erh-Hsuin, et al. "Latent Transforming Growth Factor-beta1 Functionalised Electrospun Scaffolds Promote Human Cartilage Differentiation: Towards an Engineered Cartilage Construct." Archives of plastic surgery vol. 40,6 (2013): 676-86. doi: https://doi.org/10.5999/aps.2013.40.6.676
Lim EH, Sardinha JP, Myers S, Stevens M. Latent Transforming Growth Factor-beta1 Functionalised Electrospun Scaffolds Promote Human Cartilage Differentiation: Towards an Engineered Cartilage Construct. Arch Plast Surg. 2013 Nov;40(6):676-86. doi: 10.5999/aps.2013.40.6.676. Epub 2013 Nov 08. PMID: 24286039; PMCID: PMC3840173.
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Role of caffeic Acid on collagen production in nasal polyp-derived fibroblasts.

Clinical and experimental otorhinolaryngology

Chung SW, Park IH, Hong SM, Cho JS, Moon JH, Kim TH, Lee HM.
PMID: 25436049
Clin Exp Otorhinolaryngol. 2014 Dec;7(4):295-301. doi: 10.3342/ceo.2014.7.4.295. Epub 2014 Nov 14.

OBJECTIVES: Caffeic acids are known to have anti-oxidant, anti-inflammatory, immunomodulatory, and tissue reparative effects. The purposes of this study were to determine the effect of caffeic acid on transforming growth factor (TGF) β1-induced myofibroblast differentiation and collagen production, and...

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Chung SW, Park IH, Hong SM, et al. Role of caffeic Acid on collagen production in nasal polyp-derived fibroblasts. Clin Exp Otorhinolaryngol. 2014;7(4):295-301doi: 10.3342/ceo.2014.7.4.295.
Chung, S. W., Park, I. H., Hong, S. M., Cho, J. S., Moon, J. H., Kim, T. H., & Lee, H. M. (2014). Role of caffeic Acid on collagen production in nasal polyp-derived fibroblasts. Clinical and experimental otorhinolaryngology, 7(4), 295-301. https://doi.org/10.3342/ceo.2014.7.4.295
Chung, Seung Won, et al. "Role of caffeic Acid on collagen production in nasal polyp-derived fibroblasts." Clinical and experimental otorhinolaryngology vol. 7,4 (2014): 295-301. doi: https://doi.org/10.3342/ceo.2014.7.4.295
Chung SW, Park IH, Hong SM, Cho JS, Moon JH, Kim TH, Lee HM. Role of caffeic Acid on collagen production in nasal polyp-derived fibroblasts. Clin Exp Otorhinolaryngol. 2014 Dec;7(4):295-301. doi: 10.3342/ceo.2014.7.4.295. Epub 2014 Nov 14. PMID: 25436049; PMCID: PMC4240487.
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Activation of Tissue Remodeling Precedes Obliterative Bronchiolitis in Lung Transplant Recipients.

Biomarker insights

Ramirez AM, Nunley DR, Rojas M, Roman J.
PMID: 19578518
Biomark Insights. 2008 Jun 06;3:351-359. doi: 10.4137/bmi.s686.

Obliterative bronchiolitis (OB) and Bronchiolitis Obliterans Syndrome (BOS) are frequent complications in the lung transplant recipient, and are the leading cause of mortality after transplantation. The mechanisms responsible for OB remain elusive, but inflammatory and tissue remodeling responses are...

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Ramirez AM, Nunley DR, Rojas M, et al. Activation of Tissue Remodeling Precedes Obliterative Bronchiolitis in Lung Transplant Recipients. Biomark Insights. 2008;3:351-359doi: 10.4137/bmi.s686.
Ramirez, A. M., Nunley, D. R., Rojas, M., & Roman, J. (2008). Activation of Tissue Remodeling Precedes Obliterative Bronchiolitis in Lung Transplant Recipients. Biomarker insights, 3351-359. https://doi.org/10.4137/bmi.s686
Ramirez, Allan M, et al. "Activation of Tissue Remodeling Precedes Obliterative Bronchiolitis in Lung Transplant Recipients." Biomarker insights vol. 3 (2008): 351-359. doi: https://doi.org/10.4137/bmi.s686
Ramirez AM, Nunley DR, Rojas M, Roman J. Activation of Tissue Remodeling Precedes Obliterative Bronchiolitis in Lung Transplant Recipients. Biomark Insights. 2008 Jun 06;3:351-359. doi: 10.4137/bmi.s686. PMID: 19578518; PMCID: PMC2688351.
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Update on Keloid Management: Clinical and Basic Science Advances.

Advances in wound care

Viera MH, Vivas AC, Berman B.
PMID: 24527306
Adv Wound Care (New Rochelle). 2012 Oct;1(5):200-206. doi: 10.1089/wound.2011.0313.

BACKGROUND: Keloids are benign, fibroproliferative lesions that represent abnormal healing resulting in excessive fibrosis. They are composed of mainly type III (early) or type I (late) collagen. Some of the symptoms include pruritus, tenderness, and pain. Often, they are...

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Viera MH, Vivas AC, Berman B. Update on Keloid Management: Clinical and Basic Science Advances. Adv Wound Care (New Rochelle). 2012;1(5):200-206doi: 10.1089/wound.2011.0313.
Viera, M. H., Vivas, A. C., & Berman, B. (2012). Update on Keloid Management: Clinical and Basic Science Advances. Advances in wound care, 1(5), 200-206. https://doi.org/10.1089/wound.2011.0313
Viera, Martha H, et al. "Update on Keloid Management: Clinical and Basic Science Advances." Advances in wound care vol. 1,5 (2012): 200-206. doi: https://doi.org/10.1089/wound.2011.0313
Viera MH, Vivas AC, Berman B. Update on Keloid Management: Clinical and Basic Science Advances. Adv Wound Care (New Rochelle). 2012 Oct;1(5):200-206. doi: 10.1089/wound.2011.0313. PMID: 24527306; PMCID: PMC3839006.
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Showing 25 to 36 of 148 entries