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Showing 25 to 36 of 1801 entries
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GDF11 decreases bone mass by stimulating osteoclastogenesis and inhibiting osteoblast differentiation.

Nature communications

Liu W, Zhou L, Zhou C, Zhang S, Jing J, Xie L, Sun N, Duan X, Jing W, Liang X, Zhao H, Ye L, Chen Q, Yuan Q.
PMID: 27653144
Nat Commun. 2016 Sep 22;7:12794. doi: 10.1038/ncomms12794.

Osteoporosis is an age-related disease that affects millions of people. Growth differentiation factor 11 (GDF11) is a secreted member of the transforming growth factor beta (TGF-β) superfamily. Deletion of Gdf11 has been shown to result in a skeletal anterior-posterior...

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Liu W, Zhou L, Zhou C, et al. GDF11 decreases bone mass by stimulating osteoclastogenesis and inhibiting osteoblast differentiation. Nat Commun. 2016;7:12794doi: 10.1038/ncomms12794.
Liu, W., Zhou, L., Zhou, C., Zhang, S., Jing, J., Xie, L., Sun, N., Duan, X., Jing, W., Liang, X., Zhao, H., Ye, L., Chen, Q., & Yuan, Q. (2016). GDF11 decreases bone mass by stimulating osteoclastogenesis and inhibiting osteoblast differentiation. Nature communications, 712794. https://doi.org/10.1038/ncomms12794
Liu, Weiqing, et al. "GDF11 decreases bone mass by stimulating osteoclastogenesis and inhibiting osteoblast differentiation." Nature communications vol. 7 (2016): 12794. doi: https://doi.org/10.1038/ncomms12794
Liu W, Zhou L, Zhou C, Zhang S, Jing J, Xie L, Sun N, Duan X, Jing W, Liang X, Zhao H, Ye L, Chen Q, Yuan Q. GDF11 decreases bone mass by stimulating osteoclastogenesis and inhibiting osteoblast differentiation. Nat Commun. 2016 Sep 22;7:12794. doi: 10.1038/ncomms12794. PMID: 27653144; PMCID: PMC5036163.
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BMP-2-induced bone formation and neural inflammation.

Journal of orthopaedics

Nguyen V, Meyers CA, Yan N, Agarwal S, Levi B, James AW.
PMID: 28367006
J Orthop. 2017 Mar 20;14(2):252-256. doi: 10.1016/j.jor.2017.03.003. eCollection 2017 Jun.

Bone morphogenetic protein-2 (BMP-2), a potent osteoinductive cytokine from the transforming growth factor beta (TGF-β) family, is currently the most commonly used protein-based bone graft substitute. Although clinical use of BMP-2 has significantly increased in recent years, its prominence...

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Nguyen V, Meyers CA, Yan N, et al. BMP-2-induced bone formation and neural inflammation. J Orthop. 2017;14(2):252-256doi: 10.1016/j.jor.2017.03.003.
Nguyen, V., Meyers, C. A., Yan, N., Agarwal, S., Levi, B., & James, A. W. (2017). BMP-2-induced bone formation and neural inflammation. Journal of orthopaedics, 14(2), 252-256. https://doi.org/10.1016/j.jor.2017.03.003
Nguyen, Vi, et al. "BMP-2-induced bone formation and neural inflammation." Journal of orthopaedics vol. 14,2 (2017): 252-256. doi: https://doi.org/10.1016/j.jor.2017.03.003
Nguyen V, Meyers CA, Yan N, Agarwal S, Levi B, James AW. BMP-2-induced bone formation and neural inflammation. J Orthop. 2017 Mar 20;14(2):252-256. doi: 10.1016/j.jor.2017.03.003. eCollection 2017 Jun. PMID: 28367006; PMCID: PMC5362134.
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Changes in interleukin-17 and transforming growth factor beta 1 levels in serum and bronchoalveolar lavage fluid and their clinical significance among children with asthma.

Translational pediatrics

Jiang K, Chen HB, Wang Y, Lin JH, Hu Y, Fang YR.
PMID: 26835311
Transl Pediatr. 2013 Oct;2(4):154-9. doi: 10.3978/j.issn.2224-4336.2013.10.03.

OBJECTIVES: To investigate the changes in the levels of interleukin-17 (IL-17) and transforming growth factor beta 1 (TGF-β1) in serum and bronchoalveolar lavage fluid (BALF) and their clinical significance among children with asthma.METHODS: A total of 56 children with...

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Jiang K, Chen HB, Wang Y, et al. Changes in interleukin-17 and transforming growth factor beta 1 levels in serum and bronchoalveolar lavage fluid and their clinical significance among children with asthma. Transl Pediatr. 2013;2(4):154-9doi: 10.3978/j.issn.2224-4336.2013.10.03.
Jiang, K., Chen, H. B., Wang, Y., Lin, J. H., Hu, Y., & Fang, Y. R. (2013). Changes in interleukin-17 and transforming growth factor beta 1 levels in serum and bronchoalveolar lavage fluid and their clinical significance among children with asthma. Translational pediatrics, 2(4), 154-9. https://doi.org/10.3978/j.issn.2224-4336.2013.10.03
Jiang, Kun, et al. "Changes in interleukin-17 and transforming growth factor beta 1 levels in serum and bronchoalveolar lavage fluid and their clinical significance among children with asthma." Translational pediatrics vol. 2,4 (2013): 154-9. doi: https://doi.org/10.3978/j.issn.2224-4336.2013.10.03
Jiang K, Chen HB, Wang Y, Lin JH, Hu Y, Fang YR. Changes in interleukin-17 and transforming growth factor beta 1 levels in serum and bronchoalveolar lavage fluid and their clinical significance among children with asthma. Transl Pediatr. 2013 Oct;2(4):154-9. doi: 10.3978/j.issn.2224-4336.2013.10.03. PMID: 26835311; PMCID: PMC4729083.
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RUNX3 defines disease behavior in pancreatic ductal adenocarcinoma.

Molecular & cellular oncology

Whittle MC, Hingorani SR.
PMID: 27308599
Mol Cell Oncol. 2015 Jul 29;3(2):e1076588. doi: 10.1080/23723556.2015.1076588. eCollection 2016 Mar.

Runt-related transcription factor 3 (RUNX3) functions downstream of transforming growth factor beta (TGFβ) and plays dual roles in pancreas cancer by both suppressing (by inhibiting proliferation) and promoting (by enhancing migratory and metastatic capacity) disease progression. Consideration of the...

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Whittle MC, Hingorani SR. RUNX3 defines disease behavior in pancreatic ductal adenocarcinoma. Mol Cell Oncol. 2015;3(2):e1076588doi: 10.1080/23723556.2015.1076588.
Whittle, M. C., & Hingorani, S. R. (2016). RUNX3 defines disease behavior in pancreatic ductal adenocarcinoma. Molecular & cellular oncology, 3(2), e1076588. https://doi.org/10.1080/23723556.2015.1076588
Whittle, Martin C, and Hingorani, Sunil R. "RUNX3 defines disease behavior in pancreatic ductal adenocarcinoma." Molecular & cellular oncology vol. 3,2 (2016): e1076588. doi: https://doi.org/10.1080/23723556.2015.1076588
Whittle MC, Hingorani SR. RUNX3 defines disease behavior in pancreatic ductal adenocarcinoma. Mol Cell Oncol. 2015 Jul 29;3(2):e1076588. doi: 10.1080/23723556.2015.1076588. eCollection 2016 Mar. PMID: 27308599; PMCID: PMC4905377.
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Non-unions treated with bone morphogenic protein 7: introducing the quantitative measurement of human serum cytokine levels as promising tool in evaluation of adjunct non-union therapy.

Journal of inflammation (London, England)

Moghaddam A, Breier L, Haubruck P, Bender D, Biglari B, Wentzensen A, Zimmermann G.
PMID: 26807043
J Inflamm (Lond). 2016 Jan 22;13:3. doi: 10.1186/s12950-016-0111-x. eCollection 2016.

BACKGROUND: In this study we sought to determine if application of bone morphogenic protein 7 (BMP-7) promotes physiological bone healing of non-unions and to investigate if serum cytokine analysis may serve as a promising tool in the analysis of...

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Moghaddam A, Breier L, Haubruck P, et al. Non-unions treated with bone morphogenic protein 7: introducing the quantitative measurement of human serum cytokine levels as promising tool in evaluation of adjunct non-union therapy. J Inflamm (Lond). 2016;13:3doi: 10.1186/s12950-016-0111-x.
Moghaddam, A., Breier, L., Haubruck, P., Bender, D., Biglari, B., Wentzensen, A., & Zimmermann, G. (2016). Non-unions treated with bone morphogenic protein 7: introducing the quantitative measurement of human serum cytokine levels as promising tool in evaluation of adjunct non-union therapy. Journal of inflammation (London, England), 133. https://doi.org/10.1186/s12950-016-0111-x
Moghaddam, Arash, et al. "Non-unions treated with bone morphogenic protein 7: introducing the quantitative measurement of human serum cytokine levels as promising tool in evaluation of adjunct non-union therapy." Journal of inflammation (London, England) vol. 13 (2016): 3. doi: https://doi.org/10.1186/s12950-016-0111-x
Moghaddam A, Breier L, Haubruck P, Bender D, Biglari B, Wentzensen A, Zimmermann G. Non-unions treated with bone morphogenic protein 7: introducing the quantitative measurement of human serum cytokine levels as promising tool in evaluation of adjunct non-union therapy. J Inflamm (Lond). 2016 Jan 22;13:3. doi: 10.1186/s12950-016-0111-x. eCollection 2016. PMID: 26807043; PMCID: PMC4724145.
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Expression of Cytokines Interleukin-2, Interleukin-4, Interleukin-10 and Transforming Growth Factor β in Gastric Adenocarcinoma Biopsies Obtained from Mexican Patients.

Asian Pacific journal of cancer prevention : APJCP

Diaz Orea MA, Muñoz Perez V, Gómez Conde E, Castellanos Sánchez VO, Gonzalez Lopez R, Flores Alonso JC, Cárdenas ME, Galicia AL, Mendoza A.
PMID: 28350427
Asian Pac J Cancer Prev. 2017 Feb 01;18(2):577-582. doi: 10.22034/APJCP.2017.18.2.577.

Objective: In this study, expression of Interleukin-2, Interleukin-4, Interleukin-10 and transforming growth factor beta in diffuse and intestinal type gastric cancers from Mexican patients was assessed for use as markers of malignancy. Methods: A total of 30 biopsies from...

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Diaz Orea MA, Muñoz Perez V, Gómez Conde E, et al. Expression of Cytokines Interleukin-2, Interleukin-4, Interleukin-10 and Transforming Growth Factor β in Gastric Adenocarcinoma Biopsies Obtained from Mexican Patients. Asian Pac J Cancer Prev. 2017;18(2):577-582doi: 10.22034/APJCP.2017.18.2.577.
Diaz Orea, M. A., Muñoz Perez, V., Gómez Conde, E., Castellanos Sánchez, V. O., Gonzalez Lopez, R., Flores Alonso, J. C., Cárdenas, M. E., Galicia, A. L., & Mendoza, A. (2017). Expression of Cytokines Interleukin-2, Interleukin-4, Interleukin-10 and Transforming Growth Factor β in Gastric Adenocarcinoma Biopsies Obtained from Mexican Patients. Asian Pacific journal of cancer prevention : APJCP, 18(2), 577-582. https://doi.org/10.22034/APJCP.2017.18.2.577
Diaz Orea, Maria Alicia, et al. "Expression of Cytokines Interleukin-2, Interleukin-4, Interleukin-10 and Transforming Growth Factor β in Gastric Adenocarcinoma Biopsies Obtained from Mexican Patients." Asian Pacific journal of cancer prevention : APJCP vol. 18,2 (2017): 577-582. doi: https://doi.org/10.22034/APJCP.2017.18.2.577
Diaz Orea MA, Muñoz Perez V, Gómez Conde E, Castellanos Sánchez VO, Gonzalez Lopez R, Flores Alonso JC, Cárdenas ME, Galicia AL, Mendoza A. Expression of Cytokines Interleukin-2, Interleukin-4, Interleukin-10 and Transforming Growth Factor β in Gastric Adenocarcinoma Biopsies Obtained from Mexican Patients. Asian Pac J Cancer Prev. 2017 Feb 01;18(2):577-582. doi: 10.22034/APJCP.2017.18.2.577. PMID: 28350427; PMCID: PMC5454761.
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Cytokine Gene Polymorphisms in Childhood Dilated Cardiomyopathy: Interferon- gamma, Tumor Necrosis Factor-alpha and Transforming Growth Factor - beta 1 Genes Are Associated with the Disease in Turkish Patients.

Iranian journal of pediatrics

Balci SO, Col-Araz N, Baspinar O, Sever T, Balat A, Pehlivan S.
PMID: 24800026
Iran J Pediatr. 2013 Oct;23(5):603-4.

No abstract available.

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Balci SO, Col-Araz N, Baspinar O, et al. Cytokine Gene Polymorphisms in Childhood Dilated Cardiomyopathy: Interferon- gamma, Tumor Necrosis Factor-alpha and Transforming Growth Factor - beta 1 Genes Are Associated with the Disease in Turkish Patients. Iran J Pediatr. 2013;23(5):603-4
Balci, S. O., Col-Araz, N., Baspinar, O., Sever, T., Balat, A., & Pehlivan, S. (2013). Cytokine Gene Polymorphisms in Childhood Dilated Cardiomyopathy: Interferon- gamma, Tumor Necrosis Factor-alpha and Transforming Growth Factor - beta 1 Genes Are Associated with the Disease in Turkish Patients. Iranian journal of pediatrics, 23(5), 603-4.
Balci, Sibel Oguzkan, et al. "Cytokine Gene Polymorphisms in Childhood Dilated Cardiomyopathy: Interferon- gamma, Tumor Necrosis Factor-alpha and Transforming Growth Factor - beta 1 Genes Are Associated with the Disease in Turkish Patients." Iranian journal of pediatrics vol. 23,5 (2013): 603-4.
Balci SO, Col-Araz N, Baspinar O, Sever T, Balat A, Pehlivan S. Cytokine Gene Polymorphisms in Childhood Dilated Cardiomyopathy: Interferon- gamma, Tumor Necrosis Factor-alpha and Transforming Growth Factor - beta 1 Genes Are Associated with the Disease in Turkish Patients. Iran J Pediatr. 2013 Oct;23(5):603-4. PMID: 24800026; PMCID: PMC4006515.
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Recombinant Human Decorin Inhibits TGF-b1 Induced Contraction of Collagen Lattice by Keloid Fibroblasts.

Wounds : a compendium of clinical research and practice

Zhang Z, Garron T, Li XJ, Liu Y, Zhang X, Li YY, Xu WS.
PMID: 25903025
Wounds. 2009 Feb;21(2):47-56.

While wound contraction plays an important role in healing, it may lead to excessive scar formation and pathological wound contracture in extreme conditions. To date, the key regulator of wound contraction and keloid formation is transforming growth factor-beta (TGF-b1)....

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Zhang Z, Garron T, Li XJ, et al. Recombinant Human Decorin Inhibits TGF-b1 Induced Contraction of Collagen Lattice by Keloid Fibroblasts. Wounds. 2009;21(2):47-56
Zhang, Z., Garron, T., Li, X. J., Liu, Y., Zhang, X., Li, Y. Y., & Xu, W. S. (2009). Recombinant Human Decorin Inhibits TGF-b1 Induced Contraction of Collagen Lattice by Keloid Fibroblasts. Wounds : a compendium of clinical research and practice, 21(2), 47-56.
Zhang, Zhi, et al. "Recombinant Human Decorin Inhibits TGF-b1 Induced Contraction of Collagen Lattice by Keloid Fibroblasts." Wounds : a compendium of clinical research and practice vol. 21,2 (2009): 47-56.
Zhang Z, Garron T, Li XJ, Liu Y, Zhang X, Li YY, Xu WS. Recombinant Human Decorin Inhibits TGF-b1 Induced Contraction of Collagen Lattice by Keloid Fibroblasts. Wounds. 2009 Feb;21(2):47-56. PMID: 25903025.
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Inhibition of TGF-β signaling with halofuginone can enhance the antitumor effect of irradiation in Lewis lung cancer.

OncoTargets and therapy

Lin R, Yi S, Gong L, Liu W, Wang P, Liu N, Zhao L, Wang P.
PMID: 26664138
Onco Targets Ther. 2015 Dec 02;8:3549-59. doi: 10.2147/OTT.S92518. eCollection 2015.

PURPOSE: It was reported that halofuginone has inhibitory effects on transforming growth factor-beta (TGF-β) signaling pathway. The study was aimed to: 1) evaluate the antitumor effects of halofuginone in combination with radiation therapy; and 2) preliminarily explore the possible...

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Lin R, Yi S, Gong L, et al. Inhibition of TGF-β signaling with halofuginone can enhance the antitumor effect of irradiation in Lewis lung cancer. Onco Targets Ther. 2015;8:3549-59doi: 10.2147/OTT.S92518.
Lin, R., Yi, S., Gong, L., Liu, W., Wang, P., Liu, N., Zhao, L., & Wang, P. (2015). Inhibition of TGF-β signaling with halofuginone can enhance the antitumor effect of irradiation in Lewis lung cancer. OncoTargets and therapy, 83549-59. https://doi.org/10.2147/OTT.S92518
Lin, Runlong, et al. "Inhibition of TGF-β signaling with halofuginone can enhance the antitumor effect of irradiation in Lewis lung cancer." OncoTargets and therapy vol. 8 (2015): 3549-59. doi: https://doi.org/10.2147/OTT.S92518
Lin R, Yi S, Gong L, Liu W, Wang P, Liu N, Zhao L, Wang P. Inhibition of TGF-β signaling with halofuginone can enhance the antitumor effect of irradiation in Lewis lung cancer. Onco Targets Ther. 2015 Dec 02;8:3549-59. doi: 10.2147/OTT.S92518. eCollection 2015. PMID: 26664138; PMCID: PMC4671802.
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Commentary: Bone morphogenetic protein's contribution to pulmonary artery hypertension: Should this raise concern for patients undergoing spinal fusions with bone morphogenetic protein?.

Surgical neurology international

Epstein NE.
PMID: 25593780
Surg Neurol Int. 2014 Dec 30;5:S570-3. doi: 10.4103/2152-7806.148050. eCollection 2014.

BACKGROUND: Congenital pulmonary artery hypertension (PAH) has been clinically correlated in 70-80% of cases with mutations at the bone morphogenetic protein receptor 2 (BMPR2) genetic site. However, there is also clinical and basic science/laboratory literature indicating a dose-response relationship...

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Epstein NE. Commentary: Bone morphogenetic protein's contribution to pulmonary artery hypertension: Should this raise concern for patients undergoing spinal fusions with bone morphogenetic protein?. Surg Neurol Int. 2014;5:S570-3doi: 10.4103/2152-7806.148050.
Epstein, N. E. (2014). Commentary: Bone morphogenetic protein's contribution to pulmonary artery hypertension: Should this raise concern for patients undergoing spinal fusions with bone morphogenetic protein?. Surgical neurology international, 5S570-3. https://doi.org/10.4103/2152-7806.148050
Epstein, Nancy E. "Commentary: Bone morphogenetic protein's contribution to pulmonary artery hypertension: Should this raise concern for patients undergoing spinal fusions with bone morphogenetic protein?." Surgical neurology international vol. 5 (2014): S570-3. doi: https://doi.org/10.4103/2152-7806.148050
Epstein NE. Commentary: Bone morphogenetic protein's contribution to pulmonary artery hypertension: Should this raise concern for patients undergoing spinal fusions with bone morphogenetic protein?. Surg Neurol Int. 2014 Dec 30;5:S570-3. doi: 10.4103/2152-7806.148050. eCollection 2014. PMID: 25593780; PMCID: PMC4287904.
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Platelet-Rich Gel Supernatants Stimulate the Release of Anti-Inflammatory Proteins on Culture Media of Normal Equine Synovial Membrane Explants.

Veterinary medicine international

Ríos DL, López C, Carmona JU.
PMID: 26090267
Vet Med Int. 2015;2015:547052. doi: 10.1155/2015/547052. Epub 2015 May 18.

The aims were as follows: (1) to evaluate the effects at 48 and 96 h of two concentrations (25 and 50%) of leukocyte and platelet-rich gel (L-PRG) and pure PRG (P-PRG) supernatants on the production/degradation in normal equine synovial...

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Ríos DL, López C, Carmona JU. Platelet-Rich Gel Supernatants Stimulate the Release of Anti-Inflammatory Proteins on Culture Media of Normal Equine Synovial Membrane Explants. Vet Med Int. 2015;2015:547052doi: 10.1155/2015/547052.
Ríos, D. L., López, C., & Carmona, J. U. (2015). Platelet-Rich Gel Supernatants Stimulate the Release of Anti-Inflammatory Proteins on Culture Media of Normal Equine Synovial Membrane Explants. Veterinary medicine international, 2015547052. https://doi.org/10.1155/2015/547052
Ríos, Diana L, et al. "Platelet-Rich Gel Supernatants Stimulate the Release of Anti-Inflammatory Proteins on Culture Media of Normal Equine Synovial Membrane Explants." Veterinary medicine international vol. 2015 (2015): 547052. doi: https://doi.org/10.1155/2015/547052
Ríos DL, López C, Carmona JU. Platelet-Rich Gel Supernatants Stimulate the Release of Anti-Inflammatory Proteins on Culture Media of Normal Equine Synovial Membrane Explants. Vet Med Int. 2015;2015:547052. doi: 10.1155/2015/547052. Epub 2015 May 18. PMID: 26090267; PMCID: PMC4451761.
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Effects of retinoic acid on proliferation, phenotype and expression of cyclin-dependent kinase inhibitors in TGF-beta1-stimulated rat hepatic stellate cells.

World journal of gastroenterology

Huang GC, Zhang JS, Zhang YE.
PMID: 11819702
World J Gastroenterol. 2000 Dec;6(6):819-823. doi: 10.3748/wjg.v6.i6.819.

AIM:To study the molecular mechanisms of retinoic acid (RA)on prolix-feration and expression of cyclin-dependent kinase inhibitors (CKI), i.e.p16, p21 and p27 in cultured rat hepatic stellate cells (HSC) stimulated with transforming growth factor beta 1 (TGF-beta1).METHODS:HSC were isolated from...

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Huang GC, Zhang JS, Zhang YE. Effects of retinoic acid on proliferation, phenotype and expression of cyclin-dependent kinase inhibitors in TGF-beta1-stimulated rat hepatic stellate cells. World J Gastroenterol. 2000;6(6):819-823doi: 10.3748/wjg.v6.i6.819.
Huang, G. C., Zhang, J. S., & Zhang, Y. E. (2000). Effects of retinoic acid on proliferation, phenotype and expression of cyclin-dependent kinase inhibitors in TGF-beta1-stimulated rat hepatic stellate cells. World journal of gastroenterology, 6(6), 819-823. https://doi.org/10.3748/wjg.v6.i6.819
Huang, Guang-Cun, et al. "Effects of retinoic acid on proliferation, phenotype and expression of cyclin-dependent kinase inhibitors in TGF-beta1-stimulated rat hepatic stellate cells." World journal of gastroenterology vol. 6,6 (2000): 819-823. doi: https://doi.org/10.3748/wjg.v6.i6.819
Huang GC, Zhang JS, Zhang YE. Effects of retinoic acid on proliferation, phenotype and expression of cyclin-dependent kinase inhibitors in TGF-beta1-stimulated rat hepatic stellate cells. World J Gastroenterol. 2000 Dec;6(6):819-823. doi: 10.3748/wjg.v6.i6.819. PMID: 11819702; PMCID: PMC4728268.
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Showing 25 to 36 of 1801 entries