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Case Report: Propranolol Therapy for Infantile Tremor Syndrome in a Child With Vitamin B12 Deficiency.

Frontiers in pediatrics

Cyr A, Frehlick R, Stammers D, Crone M.
PMID: 34900874
Front Pediatr. 2021 Nov 24;9:774747. doi: 10.3389/fped.2021.774747. eCollection 2021.

Vitamin B12 deficiency in childhood presents with a wide variety of symptoms including anemia, failure to thrive and developmental delays. It is a diagnostic consideration in children who are exclusively breastfed or have minimal solid intake, especially if their...

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Cyr A, Frehlick R, Stammers D, et al. Case Report: Propranolol Therapy for Infantile Tremor Syndrome in a Child With Vitamin B12 Deficiency. Front Pediatr. 2021;9:774747doi: 10.3389/fped.2021.774747.
Cyr, A., Frehlick, R., Stammers, D., & Crone, M. (2021). Case Report: Propranolol Therapy for Infantile Tremor Syndrome in a Child With Vitamin B12 Deficiency. Frontiers in pediatrics, 9774747. https://doi.org/10.3389/fped.2021.774747
Cyr, Amélie, et al. "Case Report: Propranolol Therapy for Infantile Tremor Syndrome in a Child With Vitamin B12 Deficiency." Frontiers in pediatrics vol. 9 (2021): 774747. doi: https://doi.org/10.3389/fped.2021.774747
Cyr A, Frehlick R, Stammers D, Crone M. Case Report: Propranolol Therapy for Infantile Tremor Syndrome in a Child With Vitamin B12 Deficiency. Front Pediatr. 2021 Nov 24;9:774747. doi: 10.3389/fped.2021.774747. eCollection 2021. PMID: 34900874; PMCID: PMC8652196.
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15,16-dihydrotanshinone I inhibits EOMA cells proliferation by interfering in posttranscriptional processing of hypoxia-inducible factor 1.

International journal of medical sciences

Duan P, Huang Y, Chen K, Cheng C, Wu Z, Wu Y.
PMID: 34400891
Int J Med Sci. 2021 Jul 11;18(14):3214-3223. doi: 10.7150/ijms.60774. eCollection 2021.

Infantile hemangioma (IH), which threatens the physical and mental health of patients, is the most common benign tumor in infants. Previously, we found that 15,16-dihydrotanshinone I (DHTS) was significantly more effective at inhibiting hemangioma proliferation in vitro and in...

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Duan P, Huang Y, Chen K, et al. 15,16-dihydrotanshinone I inhibits EOMA cells proliferation by interfering in posttranscriptional processing of hypoxia-inducible factor 1. Int J Med Sci. 2021;18(14):3214-3223doi: 10.7150/ijms.60774.
Duan, P., Huang, Y., Chen, K., Cheng, C., Wu, Z., & Wu, Y. (2021). 15,16-dihydrotanshinone I inhibits EOMA cells proliferation by interfering in posttranscriptional processing of hypoxia-inducible factor 1. International journal of medical sciences, 18(14), 3214-3223. https://doi.org/10.7150/ijms.60774
Duan, Peiwen, et al. "15,16-dihydrotanshinone I inhibits EOMA cells proliferation by interfering in posttranscriptional processing of hypoxia-inducible factor 1." International journal of medical sciences vol. 18,14 (2021): 3214-3223. doi: https://doi.org/10.7150/ijms.60774
Duan P, Huang Y, Chen K, Cheng C, Wu Z, Wu Y. 15,16-dihydrotanshinone I inhibits EOMA cells proliferation by interfering in posttranscriptional processing of hypoxia-inducible factor 1. Int J Med Sci. 2021 Jul 11;18(14):3214-3223. doi: 10.7150/ijms.60774. eCollection 2021. PMID: 34400891; PMCID: PMC8364454.
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Cerebral lactate uptake during exercise is driven by the increased arterial lactate concentration.

Journal of applied physiology (Bethesda, Md. : 1985)

Siebenmann C, Sørensen H, Bonne TC, Zaar M, Aachmann-Andersen NJ, Nordsborg NB, Nielsen HB, Secher NH, Lundby C, Rasmussen P.
PMID: 34734784
J Appl Physiol (1985). 2021 Dec 01;131(6):1824-1830. doi: 10.1152/japplphysiol.00505.2021. Epub 2021 Nov 04.

Exercise facilitates cerebral lactate uptake, likely by increasing arterial lactate concentration and hence the diffusion gradient across the blood-brain barrier. However, nonspecific β-adrenergic blockade by propranolol has previously reduced the arterio-jugular venous lactate difference (AV

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Siebenmann C, Sørensen H, Bonne TC, et al. Cerebral lactate uptake during exercise is driven by the increased arterial lactate concentration. J Appl Physiol (1985). 2021;131(6):1824-1830doi: 10.1152/japplphysiol.00505.2021.
Siebenmann, C., Sørensen, H., Bonne, T. C., Zaar, M., Aachmann-Andersen, N. J., Nordsborg, N. B., Nielsen, H. B., Secher, N. H., Lundby, C., & Rasmussen, P. (2021). Cerebral lactate uptake during exercise is driven by the increased arterial lactate concentration. Journal of applied physiology (Bethesda, Md. : 1985), 131(6), 1824-1830. https://doi.org/10.1152/japplphysiol.00505.2021
Siebenmann, Christoph, et al. "Cerebral lactate uptake during exercise is driven by the increased arterial lactate concentration." Journal of applied physiology (Bethesda, Md. : 1985) vol. 131,6 (2021): 1824-1830. doi: https://doi.org/10.1152/japplphysiol.00505.2021
Siebenmann C, Sørensen H, Bonne TC, Zaar M, Aachmann-Andersen NJ, Nordsborg NB, Nielsen HB, Secher NH, Lundby C, Rasmussen P. Cerebral lactate uptake during exercise is driven by the increased arterial lactate concentration. J Appl Physiol (1985). 2021 Dec 01;131(6):1824-1830. doi: 10.1152/japplphysiol.00505.2021. Epub 2021 Nov 04. PMID: 34734784.
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beta-Adrenergic involvement in acquisition vs. extinction of a classically conditioned eye blink response in rabbits.

Brain research

Gould TJ.
PMID: 9473661
Brain Res. 1998 Jan 05;780(1):172-5.

beta-adrenergic involvement in classical conditioning and extinction was investigated. Five groups of rabbits (saline, 1, 3, 5 or 10 mg kg-1 of the beta-adrenergic antagonist propranolol) were trained with a tone conditioned stimulus (CS) paired with a corneal air...

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Gould TJ. beta-Adrenergic involvement in acquisition vs. extinction of a classically conditioned eye blink response in rabbits. Brain Res. 1998;780(1):172-5
Gould, T. J. (1998). beta-Adrenergic involvement in acquisition vs. extinction of a classically conditioned eye blink response in rabbits. Brain research, 780(1), 172-5.
Gould. "beta-Adrenergic involvement in acquisition vs. extinction of a classically conditioned eye blink response in rabbits." Brain research vol. 780,1 (1998): 172-5.
Gould TJ. beta-Adrenergic involvement in acquisition vs. extinction of a classically conditioned eye blink response in rabbits. Brain Res. 1998 Jan 05;780(1):172-5. PMID: 9473661.
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Awake Microsurgical Resection for Optochiasmatic Cavernous Malformation.

World neurosurgery

Alejandro SA, Serrato-Avila JL, Paganelli SL, Chang Mulato JE, Vela Rojas EJ, Viera Neves AP, de Souza Coelho D, Silva da Costa MD, Dória-Netto HL, Campos Filho JM, Chaddad-Neto F.
PMID: 34687930
World Neurosurg. 2022 Jan;157:159. doi: 10.1016/j.wneu.2021.10.104. Epub 2021 Oct 21.

Cavernous malformations (CM) affect approximately 0.5% of the population, with only a limited portion being located in the optic nerve and chiasma. The clinical presentation is determined by their locations. In the optochiasmatic CM, the acute visual disturbance is...

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Alejandro SA, Serrato-Avila JL, Paganelli SL, et al. Awake Microsurgical Resection for Optochiasmatic Cavernous Malformation. World Neurosurg. 2021;157:159doi: 10.1016/j.wneu.2021.10.104.
Alejandro, S. A., Serrato-Avila, J. L., Paganelli, S. L., Chang Mulato, J. E., Vela Rojas, E. J., Viera Neves, A. P., de Souza Coelho, D., Silva da Costa, M. D., Dória-Netto, H. L., Campos Filho, J. M., & Chaddad-Neto, F. (2022). Awake Microsurgical Resection for Optochiasmatic Cavernous Malformation. World neurosurgery, 157159. https://doi.org/10.1016/j.wneu.2021.10.104
Alejandro, Sebastián Aníbal, et al. "Awake Microsurgical Resection for Optochiasmatic Cavernous Malformation." World neurosurgery vol. 157 (2022): 159. doi: https://doi.org/10.1016/j.wneu.2021.10.104
Alejandro SA, Serrato-Avila JL, Paganelli SL, Chang Mulato JE, Vela Rojas EJ, Viera Neves AP, de Souza Coelho D, Silva da Costa MD, Dória-Netto HL, Campos Filho JM, Chaddad-Neto F. Awake Microsurgical Resection for Optochiasmatic Cavernous Malformation. World Neurosurg. 2022 Jan;157:159. doi: 10.1016/j.wneu.2021.10.104. Epub 2021 Oct 21. PMID: 34687930.
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The Prevalence of Beta-Blocker Use Among Medical Students at King Saud bin Abdulaziz University for Health Sciences in Jeddah, Saudi Arabia: A Cross-Sectional Study.

Cureus

Alkhatabi R, Alowfi J, Arshad L, Khan MA.
PMID: 33329949
Cureus. 2020 Nov 11;12(11):e11450. doi: 10.7759/cureus.11450.

Objectives The aim of this study was to evaluate the prevalence and patterns of beta-blocker usage among medical students. Reasons for the use and demographic factors influencing their consumption were also evaluated. Methods This was an observational cross-sectional study...

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Alkhatabi R, Alowfi J, Arshad L, et al. The Prevalence of Beta-Blocker Use Among Medical Students at King Saud bin Abdulaziz University for Health Sciences in Jeddah, Saudi Arabia: A Cross-Sectional Study. Cureus. 2020;12(11):e11450doi: 10.7759/cureus.11450.
Alkhatabi, R., Alowfi, J., Arshad, L., & Khan, M. A. (2020). The Prevalence of Beta-Blocker Use Among Medical Students at King Saud bin Abdulaziz University for Health Sciences in Jeddah, Saudi Arabia: A Cross-Sectional Study. Cureus, 12(11), e11450. https://doi.org/10.7759/cureus.11450
Alkhatabi, Rawan, et al. "The Prevalence of Beta-Blocker Use Among Medical Students at King Saud bin Abdulaziz University for Health Sciences in Jeddah, Saudi Arabia: A Cross-Sectional Study." Cureus vol. 12,11 (2020): e11450. doi: https://doi.org/10.7759/cureus.11450
Alkhatabi R, Alowfi J, Arshad L, Khan MA. The Prevalence of Beta-Blocker Use Among Medical Students at King Saud bin Abdulaziz University for Health Sciences in Jeddah, Saudi Arabia: A Cross-Sectional Study. Cureus. 2020 Nov 11;12(11):e11450. doi: 10.7759/cureus.11450. PMID: 33329949; PMCID: PMC7733765.
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Peripartum management of patient with long QT3 after successful implantable cardioverter defibrillator device discharge resulting in device failure: a case report.

European heart journal. Case reports

Lee MJ, Monteil DC, Spooner MT.
PMID: 34926986
Eur Heart J Case Rep. 2021 Nov 30;5(12):ytab487. doi: 10.1093/ehjcr/ytab487. eCollection 2021 Dec.

BACKGROUND: Long QT3 syndrome type 3 (LQT3) is a gain of function mutation of the SCN5A gene that is inherited in an autosomal dominant fashion. Long QT3 syndrome type 3 results in an increase in arrhythmic events during rest,...

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Lee MJ, Monteil DC, Spooner MT. Peripartum management of patient with long QT3 after successful implantable cardioverter defibrillator device discharge resulting in device failure: a case report. Eur Heart J Case Rep. 2021;5(12):ytab487doi: 10.1093/ehjcr/ytab487.
Lee, M. J., Monteil, D. C., & Spooner, M. T. (2021). Peripartum management of patient with long QT3 after successful implantable cardioverter defibrillator device discharge resulting in device failure: a case report. European heart journal. Case reports, 5(12), ytab487. https://doi.org/10.1093/ehjcr/ytab487
Lee, Melissa J, et al. "Peripartum management of patient with long QT3 after successful implantable cardioverter defibrillator device discharge resulting in device failure: a case report." European heart journal. Case reports vol. 5,12 (2021): ytab487. doi: https://doi.org/10.1093/ehjcr/ytab487
Lee MJ, Monteil DC, Spooner MT. Peripartum management of patient with long QT3 after successful implantable cardioverter defibrillator device discharge resulting in device failure: a case report. Eur Heart J Case Rep. 2021 Nov 30;5(12):ytab487. doi: 10.1093/ehjcr/ytab487. eCollection 2021 Dec. PMID: 34926986; PMCID: PMC8672658.
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Manipulation of vocal communication and anxiety through pharmacologic modulation of norepinephrine in the Pink1-/- rat model of Parkinson disease.

Behavioural brain research

Hoffmeister JD, Kelm-Nelson CA, Ciucci MR.
PMID: 34755639
Behav Brain Res. 2022 Feb 10;418:113642. doi: 10.1016/j.bbr.2021.113642. Epub 2021 Oct 28.

Vocal deficits and anxiety are common, co-occurring, and interacting signs of Parkinson Disease (PD) that have a devastating impact on quality of life. Both manifest early in the disease process. Unlike hallmark motor signs of PD, neither respond adequately...

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Hoffmeister JD, Kelm-Nelson CA, Ciucci MR. Manipulation of vocal communication and anxiety through pharmacologic modulation of norepinephrine in the Pink1-/- rat model of Parkinson disease. Behav Brain Res. 2021;418:113642doi: 10.1016/j.bbr.2021.113642.
Hoffmeister, J. D., Kelm-Nelson, C. A., & Ciucci, M. R. (2022). Manipulation of vocal communication and anxiety through pharmacologic modulation of norepinephrine in the Pink1-/- rat model of Parkinson disease. Behavioural brain research, 418113642. https://doi.org/10.1016/j.bbr.2021.113642
Hoffmeister, Jesse D, et al. "Manipulation of vocal communication and anxiety through pharmacologic modulation of norepinephrine in the Pink1-/- rat model of Parkinson disease." Behavioural brain research vol. 418 (2022): 113642. doi: https://doi.org/10.1016/j.bbr.2021.113642
Hoffmeister JD, Kelm-Nelson CA, Ciucci MR. Manipulation of vocal communication and anxiety through pharmacologic modulation of norepinephrine in the Pink1-/- rat model of Parkinson disease. Behav Brain Res. 2022 Feb 10;418:113642. doi: 10.1016/j.bbr.2021.113642. Epub 2021 Oct 28. PMID: 34755639; PMCID: PMC8671235.
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A Novel In Vitro Membrane Permeability Methodology Using Three-dimensional Caco-2 Tubules in a Microphysiological System Which Better Mimics In Vivo Physiological Conditions.

Journal of pharmaceutical sciences

Hagiwara Y, Kumagai H, Ouwerkerk N, Gijzen L, Annida R, Bokkers M, van Vught R, Yoshinari K, Katakawa Y, Motonaga K, Tajiri T.
PMID: 34838780
J Pharm Sci. 2022 Jan;111(1):214-224. doi: 10.1016/j.xphs.2021.11.016. Epub 2021 Nov 25.

The aim of this study was to develop an in vitro drug permeability methodology which mimics the gastrointestinal environment more accurately than conventional 2D methodologies through a three-dimensional (3D) Caco-2 tubules using a microphysiological system. Such a system offers...

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Hagiwara Y, Kumagai H, Ouwerkerk N, et al. A Novel In Vitro Membrane Permeability Methodology Using Three-dimensional Caco-2 Tubules in a Microphysiological System Which Better Mimics In Vivo Physiological Conditions. J Pharm Sci. 2021;111(1):214-224doi: 10.1016/j.xphs.2021.11.016.
Hagiwara, Y., Kumagai, H., Ouwerkerk, N., Gijzen, L., Annida, R., Bokkers, M., van Vught, R., Yoshinari, K., Katakawa, Y., Motonaga, K., & Tajiri, T. (2022). A Novel In Vitro Membrane Permeability Methodology Using Three-dimensional Caco-2 Tubules in a Microphysiological System Which Better Mimics In Vivo Physiological Conditions. Journal of pharmaceutical sciences, 111(1), 214-224. https://doi.org/10.1016/j.xphs.2021.11.016
Hagiwara, Yuki, et al. "A Novel In Vitro Membrane Permeability Methodology Using Three-dimensional Caco-2 Tubules in a Microphysiological System Which Better Mimics In Vivo Physiological Conditions." Journal of pharmaceutical sciences vol. 111,1 (2022): 214-224. doi: https://doi.org/10.1016/j.xphs.2021.11.016
Hagiwara Y, Kumagai H, Ouwerkerk N, Gijzen L, Annida R, Bokkers M, van Vught R, Yoshinari K, Katakawa Y, Motonaga K, Tajiri T. A Novel In Vitro Membrane Permeability Methodology Using Three-dimensional Caco-2 Tubules in a Microphysiological System Which Better Mimics In Vivo Physiological Conditions. J Pharm Sci. 2022 Jan;111(1):214-224. doi: 10.1016/j.xphs.2021.11.016. Epub 2021 Nov 25. PMID: 34838780.
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Propranolol Relieves L-Dopa-Induced Dyskinesia in Parkinsonian Mice.

Brain sciences

Shi Z, Bamford IJ, McKinley JW, Devi SPS, Vahedipour A, Bamford NS.
PMID: 33255421
Brain Sci. 2020 Nov 24;10(12). doi: 10.3390/brainsci10120903.

BACKGROUND: Parkinsonism is caused by dopamine (DA) insufficiency and results in a hypokinetic movement disorder. Treatment with L-Dopa can restore DA availability and improve motor function, but patients can develop L-Dopa-induced dyskinesia (LID), a secondary hyperkinetic movement disorder. The...

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Shi Z, Bamford IJ, McKinley JW, et al. Propranolol Relieves L-Dopa-Induced Dyskinesia in Parkinsonian Mice. Brain Sci. 2020;10(12)doi: 10.3390/brainsci10120903.
Shi, Z., Bamford, I. J., McKinley, J. W., Devi, S. P. S., Vahedipour, A., & Bamford, N. S. (2020). Propranolol Relieves L-Dopa-Induced Dyskinesia in Parkinsonian Mice. Brain sciences, 10(12), . https://doi.org/10.3390/brainsci10120903
Shi, Ziqing, et al. "Propranolol Relieves L-Dopa-Induced Dyskinesia in Parkinsonian Mice." Brain sciences vol. 10,12 (2020). doi: https://doi.org/10.3390/brainsci10120903
Shi Z, Bamford IJ, McKinley JW, Devi SPS, Vahedipour A, Bamford NS. Propranolol Relieves L-Dopa-Induced Dyskinesia in Parkinsonian Mice. Brain Sci. 2020 Nov 24;10(12). doi: 10.3390/brainsci10120903. PMID: 33255421; PMCID: PMC7760026.
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Propranolol inhibits infantile hemangioma by regulating the miR-424/vascular endothelial growth factor-A (VEGFA) axis.

Translational pediatrics

Wu ZB, Shi SL, Pan FJ, Li L, Chen HY.
PMID: 34430434
Transl Pediatr. 2021 Jul;10(7):1867-1876. doi: 10.21037/tp-21-244.

BACKGROUND: Infantile hemangioma (IHA) is the most common tumor in infancy. We aimed to explore the effect of propranolol on the expression of microRNA (miR)-424 in IHA tissues and XPTS-1 cells, as well as its molecular mechanism of inhibiting...

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Wu ZB, Shi SL, Pan FJ, et al. Propranolol inhibits infantile hemangioma by regulating the miR-424/vascular endothelial growth factor-A (VEGFA) axis. Transl Pediatr. 2021;10(7):1867-1876doi: 10.21037/tp-21-244.
Wu, Z. B., Shi, S. L., Pan, F. J., Li, L., & Chen, H. Y. (2021). Propranolol inhibits infantile hemangioma by regulating the miR-424/vascular endothelial growth factor-A (VEGFA) axis. Translational pediatrics, 10(7), 1867-1876. https://doi.org/10.21037/tp-21-244
Wu, Zhen-Bo, et al. "Propranolol inhibits infantile hemangioma by regulating the miR-424/vascular endothelial growth factor-A (VEGFA) axis." Translational pediatrics vol. 10,7 (2021): 1867-1876. doi: https://doi.org/10.21037/tp-21-244
Wu ZB, Shi SL, Pan FJ, Li L, Chen HY. Propranolol inhibits infantile hemangioma by regulating the miR-424/vascular endothelial growth factor-A (VEGFA) axis. Transl Pediatr. 2021 Jul;10(7):1867-1876. doi: 10.21037/tp-21-244. PMID: 34430434; PMCID: PMC8349971.
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