Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 13 to 24 of 852 entries
Sorted by: Best Match Show Resources per page
Computational analysis of the electromechanical consequences of short QT syndrome.

Frontiers in physiology

Huang CL.
PMID: 25717305
Front Physiol. 2015 Feb 11;6:44. doi: 10.3389/fphys.2015.00044. eCollection 2015.

No abstract available.

Cite
Huang CL. Computational analysis of the electromechanical consequences of short QT syndrome. Front Physiol. 2015;6:44doi: 10.3389/fphys.2015.00044.
Huang, C. L. (2015). Computational analysis of the electromechanical consequences of short QT syndrome. Frontiers in physiology, 644. https://doi.org/10.3389/fphys.2015.00044
Huang, Christopher L-H. "Computational analysis of the electromechanical consequences of short QT syndrome." Frontiers in physiology vol. 6 (2015): 44. doi: https://doi.org/10.3389/fphys.2015.00044
Huang CL. Computational analysis of the electromechanical consequences of short QT syndrome. Front Physiol. 2015 Feb 11;6:44. doi: 10.3389/fphys.2015.00044. eCollection 2015. PMID: 25717305; PMCID: PMC4324072.
Copy Download .nbib Format: NLM
Effect of lysophosphatidic acid on the immune inflammatory response and the connexin 43 protein in myocardial infarction.

Experimental and therapeutic medicine

Zhang D, Zhang Y, Zhao C, Zhang W, Shao G, Zhang H.
PMID: 27168781
Exp Ther Med. 2016 May;11(5):1617-1624. doi: 10.3892/etm.2016.3132. Epub 2016 Mar 09.

Lysophosphatidic acid (LPA) is an intermediate product of membrane phospholipid metabolism. Recently, LPA has gained attention for its involvement in the pathological processes of certain cardiovascular diseases. The aim of the present study was to clarify the association between...

Cite
Zhang D, Zhang Y, Zhao C, et al. Effect of lysophosphatidic acid on the immune inflammatory response and the connexin 43 protein in myocardial infarction. Exp Ther Med. 2016;11(5):1617-1624doi: 10.3892/etm.2016.3132.
Zhang, D., Zhang, Y., Zhao, C., Zhang, W., Shao, G., & Zhang, H. (2016). Effect of lysophosphatidic acid on the immune inflammatory response and the connexin 43 protein in myocardial infarction. Experimental and therapeutic medicine, 11(5), 1617-1624. https://doi.org/10.3892/etm.2016.3132
Zhang, Duoduo, et al. "Effect of lysophosphatidic acid on the immune inflammatory response and the connexin 43 protein in myocardial infarction." Experimental and therapeutic medicine vol. 11,5 (2016): 1617-1624. doi: https://doi.org/10.3892/etm.2016.3132
Zhang D, Zhang Y, Zhao C, Zhang W, Shao G, Zhang H. Effect of lysophosphatidic acid on the immune inflammatory response and the connexin 43 protein in myocardial infarction. Exp Ther Med. 2016 May;11(5):1617-1624. doi: 10.3892/etm.2016.3132. Epub 2016 Mar 09. PMID: 27168781; PMCID: PMC4840587.
Copy Download .nbib Format: NLM
Volatile anesthetics suppress glucose-stimulated insulin secretion in MIN6 cells by inhibiting glucose-induced activation of hypoxia-inducible factor 1.

PeerJ

Suzuki K, Sato Y, Kai S, Nishi K, Adachi T, Matsuo Y, Hirota K.
PMID: 26713247
PeerJ. 2015 Dec 10;3:e1498. doi: 10.7717/peerj.1498. eCollection 2015.

Proper glycemic control is one of the most important goals in perioperative patient management. Insulin secretion from pancreatic β-cells in response to an increased blood glucose concentration plays the most critical role in glycemic control. Several animal and human...

Cite
Suzuki K, Sato Y, Kai S, et al. Volatile anesthetics suppress glucose-stimulated insulin secretion in MIN6 cells by inhibiting glucose-induced activation of hypoxia-inducible factor 1. PeerJ. 2015;3:e1498doi: 10.7717/peerj.1498.
Suzuki, K., Sato, Y., Kai, S., Nishi, K., Adachi, T., Matsuo, Y., & Hirota, K. (2015). Volatile anesthetics suppress glucose-stimulated insulin secretion in MIN6 cells by inhibiting glucose-induced activation of hypoxia-inducible factor 1. PeerJ, 3e1498. https://doi.org/10.7717/peerj.1498
Suzuki, Kengo, et al. "Volatile anesthetics suppress glucose-stimulated insulin secretion in MIN6 cells by inhibiting glucose-induced activation of hypoxia-inducible factor 1." PeerJ vol. 3 (2015): e1498. doi: https://doi.org/10.7717/peerj.1498
Suzuki K, Sato Y, Kai S, Nishi K, Adachi T, Matsuo Y, Hirota K. Volatile anesthetics suppress glucose-stimulated insulin secretion in MIN6 cells by inhibiting glucose-induced activation of hypoxia-inducible factor 1. PeerJ. 2015 Dec 10;3:e1498. doi: 10.7717/peerj.1498. eCollection 2015. PMID: 26713247; PMCID: PMC4690348.
Copy Download .nbib Format: NLM
Overview of the genetic determinants of primary aldosteronism.

The application of clinical genetics

Al-Salameh A, Cohen R, Desailloud R.
PMID: 24817817
Appl Clin Genet. 2014 Apr 08;7:67-79. doi: 10.2147/TACG.S45620. eCollection 2014.

Primary aldosteronism is the most common cause of secondary hypertension. The syndrome accounts for 10% of all cases of hypertension and is primarily caused by bilateral adrenal hyperplasia or aldosterone-producing adenoma. Over the last few years, the use of...

Cite
Al-Salameh A, Cohen R, Desailloud R. Overview of the genetic determinants of primary aldosteronism. Appl Clin Genet. 2014;7:67-79doi: 10.2147/TACG.S45620.
Al-Salameh, A., Cohen, R., & Desailloud, R. (2014). Overview of the genetic determinants of primary aldosteronism. The application of clinical genetics, 767-79. https://doi.org/10.2147/TACG.S45620
Al-Salameh, Abdallah, et al. "Overview of the genetic determinants of primary aldosteronism." The application of clinical genetics vol. 7 (2014): 67-79. doi: https://doi.org/10.2147/TACG.S45620
Al-Salameh A, Cohen R, Desailloud R. Overview of the genetic determinants of primary aldosteronism. Appl Clin Genet. 2014 Apr 08;7:67-79. doi: 10.2147/TACG.S45620. eCollection 2014. PMID: 24817817; PMCID: PMC4012345.
Copy Download .nbib Format: NLM
Role of endothelium-dependent hyperpolarisation and prostacyclin in diabetes.

The Malaysian journal of medical sciences : MJMS

Mokhtar SS, Rasool AH.
PMID: 26023290
Malays J Med Sci. 2015 Mar-Apr;22(2):8-17.

The endothelium plays a crucial role in maintaining vascular homeostasis by producing several vasodilating factors, including nitric oxide (NO), prostacyclin (PGI2), and endothelium-dependent hyperpolarisation (EDH); however, the balance between endothelial relaxing and contracting factors is disrupted in disease states...

Cite
Mokhtar SS, Rasool AH. Role of endothelium-dependent hyperpolarisation and prostacyclin in diabetes. Malays J Med Sci. 2015;22(2):8-17
Mokhtar, S. S., & Rasool, A. H. (2015). Role of endothelium-dependent hyperpolarisation and prostacyclin in diabetes. The Malaysian journal of medical sciences : MJMS, 22(2), 8-17.
Mokhtar, Siti Safiah, and Rasool, Aida Hanum Ghulam. "Role of endothelium-dependent hyperpolarisation and prostacyclin in diabetes." The Malaysian journal of medical sciences : MJMS vol. 22,2 (2015): 8-17.
Mokhtar SS, Rasool AH. Role of endothelium-dependent hyperpolarisation and prostacyclin in diabetes. Malays J Med Sci. 2015 Mar-Apr;22(2):8-17. PMID: 26023290; PMCID: PMC4438087.
Copy Download .nbib Format: NLM
The Natural Flavone Acacetin Blocks Small Conductance Ca.

Frontiers in pharmacology

Chen KH, Liu H, Sun HY, Jin MW, Xiao GS, Wang Y, Li GR.
PMID: 29081746
Front Pharmacol. 2017 Oct 10;8:716. doi: 10.3389/fphar.2017.00716. eCollection 2017.

The natural flavone acacetin inhibits several voltage-gated potassium currents in atrial myocytes, and has anti-atrial fibrillation (AF) effect in experimental AF models. The present study investigates whether acacetin inhibits the Ca

Cite
Chen KH, Liu H, Sun HY, et al. The Natural Flavone Acacetin Blocks Small Conductance Ca. Front Pharmacol. 2017;8:716doi: 10.3389/fphar.2017.00716.
Chen, K. H., Liu, H., Sun, H. Y., Jin, M. W., Xiao, G. S., Wang, Y., & Li, G. R. (2017). The Natural Flavone Acacetin Blocks Small Conductance Ca. Frontiers in pharmacology, 8716. https://doi.org/10.3389/fphar.2017.00716
Chen, Kui-Hao, et al. "The Natural Flavone Acacetin Blocks Small Conductance Ca." Frontiers in pharmacology vol. 8 (2017): 716. doi: https://doi.org/10.3389/fphar.2017.00716
Chen KH, Liu H, Sun HY, Jin MW, Xiao GS, Wang Y, Li GR. The Natural Flavone Acacetin Blocks Small Conductance Ca. Front Pharmacol. 2017 Oct 10;8:716. doi: 10.3389/fphar.2017.00716. eCollection 2017. PMID: 29081746; PMCID: PMC5646423.
Copy Download .nbib Format: NLM
Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease.

Frontiers in physiology

Tao J, Lan Z, Wang Y, Hei H, Tian L, Pan W, Zhang X, Peng W.
PMID: 27445840
Front Physiol. 2016 Jun 21;7:248. doi: 10.3389/fphys.2016.00248. eCollection 2016.

Large-conductance calcium-activated potassium (BK) channels are currently considered as vital players in a variety of renal physiological processes. In podocytes, BK channels become active in response to stimuli that increase local cytosolic Ca(2+), possibly secondary to activation of slit...

Cite
Tao J, Lan Z, Wang Y, et al. Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease. Front Physiol. 2016;7:248doi: 10.3389/fphys.2016.00248.
Tao, J., Lan, Z., Wang, Y., Hei, H., Tian, L., Pan, W., Zhang, X., & Peng, W. (2016). Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease. Frontiers in physiology, 7248. https://doi.org/10.3389/fphys.2016.00248
Tao, Jie, et al. "Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease." Frontiers in physiology vol. 7 (2016): 248. doi: https://doi.org/10.3389/fphys.2016.00248
Tao J, Lan Z, Wang Y, Hei H, Tian L, Pan W, Zhang X, Peng W. Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease. Front Physiol. 2016 Jun 21;7:248. doi: 10.3389/fphys.2016.00248. eCollection 2016. PMID: 27445840; PMCID: PMC4915313.
Copy Download .nbib Format: NLM
hERG S4-S5 linker acts as a voltage-dependent ligand that binds to the activation gate and locks it in a closed state.

Scientific reports

Malak OA, Es-Salah-Lamoureux Z, Loussouarn G.
PMID: 28273916
Sci Rep. 2017 Mar 02;7(1):113. doi: 10.1038/s41598-017-00155-2.

Delayed-rectifier potassium channels (hERG and KCNQ1) play a major role in cardiac repolarization. These channels are formed by a tetrameric pore (S5-S6) surrounded by four voltage sensor domains (S1-S4). Coupling between voltage sensor domains and the pore activation gate...

Cite
Malak OA, Es-Salah-Lamoureux Z, Loussouarn G. hERG S4-S5 linker acts as a voltage-dependent ligand that binds to the activation gate and locks it in a closed state. Sci Rep. 2017;7(1):113doi: 10.1038/s41598-017-00155-2.
Malak, O. A., Es-Salah-Lamoureux, Z., & Loussouarn, G. (2017). hERG S4-S5 linker acts as a voltage-dependent ligand that binds to the activation gate and locks it in a closed state. Scientific reports, 7(1), 113. https://doi.org/10.1038/s41598-017-00155-2
Malak, Olfat A, et al. "hERG S4-S5 linker acts as a voltage-dependent ligand that binds to the activation gate and locks it in a closed state." Scientific reports vol. 7,1 (2017): 113. doi: https://doi.org/10.1038/s41598-017-00155-2
Malak OA, Es-Salah-Lamoureux Z, Loussouarn G. hERG S4-S5 linker acts as a voltage-dependent ligand that binds to the activation gate and locks it in a closed state. Sci Rep. 2017 Mar 02;7(1):113. doi: 10.1038/s41598-017-00155-2. PMID: 28273916; PMCID: PMC5427910.
Copy Download .nbib Format: NLM
In vitro effects of extracellular hypercapnic acidification on the reactivity of rat aorta: Rat aorta vasodilation during hypercapnic acidification.

Nitric oxide : biology and chemistry

de Nadai TR, de Nadai MN, Cassiano Silveira AP, Celotto AC, Albuquerque AA, de Carvalho MT, Scarpelini S, Rodrigues AJ, Evora PR.
PMID: 26358824
Nitric Oxide. 2015 Nov 15;50:79-87. doi: 10.1016/j.niox.2015.09.001. Epub 2015 Sep 07.

The mechanisms by which pH influences vascular tone are not entirely understood, but evidence suggests that the endothelium is involved. Here, we aimed to study the in vitro vascular responses induced by extracellular hypercapnic acidification (HA), as well as...

Cite
de Nadai TR, de Nadai MN, Cassiano Silveira AP, et al. In vitro effects of extracellular hypercapnic acidification on the reactivity of rat aorta: Rat aorta vasodilation during hypercapnic acidification. Nitric Oxide. 2015;50:79-87doi: 10.1016/j.niox.2015.09.001.
de Nadai, T. R., de Nadai, M. N., Cassiano Silveira, A. P., Celotto, A. C., Albuquerque, A. A., de Carvalho, M. T., Scarpelini, S., Rodrigues, A. J., & Evora, P. R. (2015). In vitro effects of extracellular hypercapnic acidification on the reactivity of rat aorta: Rat aorta vasodilation during hypercapnic acidification. Nitric oxide : biology and chemistry, 5079-87. https://doi.org/10.1016/j.niox.2015.09.001
de Nadai, Tales Rubens, et al. "In vitro effects of extracellular hypercapnic acidification on the reactivity of rat aorta: Rat aorta vasodilation during hypercapnic acidification." Nitric oxide : biology and chemistry vol. 50 (2015): 79-87. doi: https://doi.org/10.1016/j.niox.2015.09.001
de Nadai TR, de Nadai MN, Cassiano Silveira AP, Celotto AC, Albuquerque AA, de Carvalho MT, Scarpelini S, Rodrigues AJ, Evora PR. In vitro effects of extracellular hypercapnic acidification on the reactivity of rat aorta: Rat aorta vasodilation during hypercapnic acidification. Nitric Oxide. 2015 Nov 15;50:79-87. doi: 10.1016/j.niox.2015.09.001. Epub 2015 Sep 07. PMID: 26358824.
Copy Download .nbib Format: NLM
Cross-talk and regulation between glutamate and GABAB receptors.

Frontiers in cellular neuroscience

Kantamneni S.
PMID: 25914625
Front Cell Neurosci. 2015 Apr 10;9:135. doi: 10.3389/fncel.2015.00135. eCollection 2015.

Brain function depends on co-ordinated transmission of signals from both excitatory and inhibitory neurotransmitters acting upon target neurons. NMDA, AMPA and mGluR receptors are the major subclasses of glutamate receptors that are involved in excitatory transmission at synapses, mechanisms...

Cite
Kantamneni S. Cross-talk and regulation between glutamate and GABAB receptors. Front Cell Neurosci. 2015;9:135doi: 10.3389/fncel.2015.00135.
Kantamneni, S. (2015). Cross-talk and regulation between glutamate and GABAB receptors. Frontiers in cellular neuroscience, 9135. https://doi.org/10.3389/fncel.2015.00135
Kantamneni, Sriharsha. "Cross-talk and regulation between glutamate and GABAB receptors." Frontiers in cellular neuroscience vol. 9 (2015): 135. doi: https://doi.org/10.3389/fncel.2015.00135
Kantamneni S. Cross-talk and regulation between glutamate and GABAB receptors. Front Cell Neurosci. 2015 Apr 10;9:135. doi: 10.3389/fncel.2015.00135. eCollection 2015. PMID: 25914625; PMCID: PMC4392697.
Copy Download .nbib Format: NLM
ATP-dependent potassium channels are implicated in simvastatin pretreatment-induced inhibition of apoptotic cell death after renal ischemia/reperfusion injury.

Medical journal of the Islamic Republic of Iran

Dowlatshahi K, Ajami M, Pazoki-Toroudi H, Hajimiresmaiel SJ.
PMID: 26157709
Med J Islam Repub Iran. 2015 Mar 14;29:191. eCollection 2015.

BACKGROUND: Simvastatin is a widely used medication in cardiac care. Here we evaluate the role of ATP sensitive potassium (KATP) channels in simvastatin induced renal protection after renal ischemia/reperfusion (I/R) injury.METHODS: A total of 81 male Wistar rats, were...

Cite
Dowlatshahi K, Ajami M, Pazoki-Toroudi H, et al. ATP-dependent potassium channels are implicated in simvastatin pretreatment-induced inhibition of apoptotic cell death after renal ischemia/reperfusion injury. Med J Islam Repub Iran. 2015;29:191
Dowlatshahi, K., Ajami, M., Pazoki-Toroudi, H., & Hajimiresmaiel, S. J. (2015). ATP-dependent potassium channels are implicated in simvastatin pretreatment-induced inhibition of apoptotic cell death after renal ischemia/reperfusion injury. Medical journal of the Islamic Republic of Iran, 29191.
Dowlatshahi, Kamran, et al. "ATP-dependent potassium channels are implicated in simvastatin pretreatment-induced inhibition of apoptotic cell death after renal ischemia/reperfusion injury." Medical journal of the Islamic Republic of Iran vol. 29 (2015): 191.
Dowlatshahi K, Ajami M, Pazoki-Toroudi H, Hajimiresmaiel SJ. ATP-dependent potassium channels are implicated in simvastatin pretreatment-induced inhibition of apoptotic cell death after renal ischemia/reperfusion injury. Med J Islam Repub Iran. 2015 Mar 14;29:191. eCollection 2015. PMID: 26157709; PMCID: PMC4476217.
Copy Download .nbib Format: NLM
Conduction block in myelinated axons induced by high-frequency (kHz) non-symmetric biphasic stimulation.

Frontiers in computational neuroscience

Zhao S, Yang G, Wang J, Roppolo JR, de Groat WC, Tai C.
PMID: 26217217
Front Comput Neurosci. 2015 Jul 06;9:86. doi: 10.3389/fncom.2015.00086. eCollection 2015.

This study used the Frankenhaeuser-Huxley axonal model to analyze the effects of non-symmetric waveforms on conduction block of myelinated axons induced by high-frequency (10-300 kHz) biphasic electrical stimulation. The results predict a monotonic relationship between block threshold and stimulation...

Cite
Zhao S, Yang G, Wang J, et al. Conduction block in myelinated axons induced by high-frequency (kHz) non-symmetric biphasic stimulation. Front Comput Neurosci. 2015;9:86doi: 10.3389/fncom.2015.00086.
Zhao, S., Yang, G., Wang, J., Roppolo, J. R., de Groat, W. C., & Tai, C. (2015). Conduction block in myelinated axons induced by high-frequency (kHz) non-symmetric biphasic stimulation. Frontiers in computational neuroscience, 986. https://doi.org/10.3389/fncom.2015.00086
Zhao, Shouguo, et al. "Conduction block in myelinated axons induced by high-frequency (kHz) non-symmetric biphasic stimulation." Frontiers in computational neuroscience vol. 9 (2015): 86. doi: https://doi.org/10.3389/fncom.2015.00086
Zhao S, Yang G, Wang J, Roppolo JR, de Groat WC, Tai C. Conduction block in myelinated axons induced by high-frequency (kHz) non-symmetric biphasic stimulation. Front Comput Neurosci. 2015 Jul 06;9:86. doi: 10.3389/fncom.2015.00086. eCollection 2015. PMID: 26217217; PMCID: PMC4491630.
Copy Download .nbib Format: NLM
Showing 13 to 24 of 852 entries