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Showing 25 to 36 of 42 entries
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The immediate future for the medical treatment of atrial fibrillation.

Expert opinion on emerging drugs

Pedersen OD, Brendorp B, Køber L, Torp-Pedersen C.
PMID: 15989549
Expert Opin Emerg Drugs. 2002 Oct;7(2):259-68. doi: 10.1517/14728214.7.2.259.

Atrial fibrillation is the most commonly sustained cardiac arrhythmia and a common reason for mortality and morbidity. Atrial fibrillation causes disease for three reasons: i) the ventricular rate is often high, which leads to symptoms ranging from discomfort to...

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Pedersen OD, Brendorp B, Køber L, et al. The immediate future for the medical treatment of atrial fibrillation. Expert Opin Emerg Drugs. 2002;7(2):259-68doi: 10.1517/14728214.7.2.259.
Pedersen, O. D., Brendorp, B., Køber, L., & Torp-Pedersen, C. (2002). The immediate future for the medical treatment of atrial fibrillation. Expert opinion on emerging drugs, 7(2), 259-68. https://doi.org/10.1517/14728214.7.2.259
Pedersen, Ole Dyg, et al. "The immediate future for the medical treatment of atrial fibrillation." Expert opinion on emerging drugs vol. 7,2 (2002): 259-68. doi: https://doi.org/10.1517/14728214.7.2.259
Pedersen OD, Brendorp B, Køber L, Torp-Pedersen C. The immediate future for the medical treatment of atrial fibrillation. Expert Opin Emerg Drugs. 2002 Oct;7(2):259-68. doi: 10.1517/14728214.7.2.259. PMID: 15989549.
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Neither a nitric oxide donor nor potassium channel blockage inhibit RBC mechanical damage induced by a roller pump.

The open biomedical engineering journal

Ulker P, Meiselman HJ, Baskurt OK.
PMID: 19662112
Open Biomed Eng J. 2008 Apr 01;2:17-21. doi: 10.2174/1874120700802010017.

Red blood cells (RBC) are exposed to various levels of shear stresses when they are exposed to artificial flow environments, such as extracorporeal flow circuits and hemodialysis equipment. This mechanical trauma affects RBC and the resulting effect is determined...

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Ulker P, Meiselman HJ, Baskurt OK. Neither a nitric oxide donor nor potassium channel blockage inhibit RBC mechanical damage induced by a roller pump. Open Biomed Eng J. 2008;2:17-21doi: 10.2174/1874120700802010017.
Ulker, P., Meiselman, H. J., & Baskurt, O. K. (2008). Neither a nitric oxide donor nor potassium channel blockage inhibit RBC mechanical damage induced by a roller pump. The open biomedical engineering journal, 217-21. https://doi.org/10.2174/1874120700802010017
Ulker, Pinar, et al. "Neither a nitric oxide donor nor potassium channel blockage inhibit RBC mechanical damage induced by a roller pump." The open biomedical engineering journal vol. 2 (2008): 17-21. doi: https://doi.org/10.2174/1874120700802010017
Ulker P, Meiselman HJ, Baskurt OK. Neither a nitric oxide donor nor potassium channel blockage inhibit RBC mechanical damage induced by a roller pump. Open Biomed Eng J. 2008 Apr 01;2:17-21. doi: 10.2174/1874120700802010017. PMID: 19662112; PMCID: PMC2701065.
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Diagnosis and treatment of vertigo and dizziness.

Deutsches Arzteblatt international

Strupp M, Brandt T.
PMID: 19629221
Dtsch Arztebl Int. 2008 Mar;105(10):173-80. doi: 10.3238/arztebl.2008.0173. Epub 2008 Mar 07.

INTRODUCTION: Vertigo is not a separate disease process, but a multisensory and sensorimotor syndrome with various etiologies and pathogeneses. It is among the commonest symptoms presented to doctors, with a lifetime prevalence of around 20% to 30%. Patients have...

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Strupp M, Brandt T. Diagnosis and treatment of vertigo and dizziness. Dtsch Arztebl Int. 2008;105(10):173-80doi: 10.3238/arztebl.2008.0173.
Strupp, M., & Brandt, T. (2008). Diagnosis and treatment of vertigo and dizziness. Deutsches Arzteblatt international, 105(10), 173-80. https://doi.org/10.3238/arztebl.2008.0173
Strupp, Michael, and Brandt, Thomas. "Diagnosis and treatment of vertigo and dizziness." Deutsches Arzteblatt international vol. 105,10 (2008): 173-80. doi: https://doi.org/10.3238/arztebl.2008.0173
Strupp M, Brandt T. Diagnosis and treatment of vertigo and dizziness. Dtsch Arztebl Int. 2008 Mar;105(10):173-80. doi: 10.3238/arztebl.2008.0173. Epub 2008 Mar 07. PMID: 19629221; PMCID: PMC2696792.
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Ablation of outflow tract ventricular tachycardia.

Current treatment options in cardiovascular medicine

Liang JJ, Han Y, Frankel DS.
PMID: 25665821
Curr Treat Options Cardiovasc Med. 2015 Feb;17(2):363. doi: 10.1007/s11936-014-0363-9.

OPINION STATEMENT: Ventricular tachycardia in patients with structurally normal hearts is most frequently due to adenosine-sensitive, triggered activity. The most common sites of origin are the right and left ventricular outflow tracts. Patients may present with symptoms such as...

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Liang JJ, Han Y, Frankel DS. Ablation of outflow tract ventricular tachycardia. Curr Treat Options Cardiovasc Med. 2015;17(2):363doi: 10.1007/s11936-014-0363-9.
Liang, J. J., Han, Y., & Frankel, D. S. (2015). Ablation of outflow tract ventricular tachycardia. Current treatment options in cardiovascular medicine, 17(2), 363. https://doi.org/10.1007/s11936-014-0363-9
Liang, Jackson J, et al. "Ablation of outflow tract ventricular tachycardia." Current treatment options in cardiovascular medicine vol. 17,2 (2015): 363. doi: https://doi.org/10.1007/s11936-014-0363-9
Liang JJ, Han Y, Frankel DS. Ablation of outflow tract ventricular tachycardia. Curr Treat Options Cardiovasc Med. 2015 Feb;17(2):363. doi: 10.1007/s11936-014-0363-9. PMID: 25665821.
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Advances in Antiarrhythmic Drug Therapy: New and Emerging Therapies.

Cardiac electrophysiology clinics

Pinter A, Dorian P.
PMID: 28770804
Card Electrophysiol Clin. 2010 Sep;2(3):471-478. doi: 10.1016/j.ccep.2010.06.005.

Despite major advances in the nonpharmacologic therapy for arrhythmias in the past decades, there is still a substantial role for antiarrhythmic drugs especially in the treatment of atrial fibrillation and ventricular tachycardia, the most effective of which is amiodarone....

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Pinter A, Dorian P. Advances in Antiarrhythmic Drug Therapy: New and Emerging Therapies. Card Electrophysiol Clin. 2010;2(3):471-478doi: 10.1016/j.ccep.2010.06.005.
Pinter, A., & Dorian, P. (2010). Advances in Antiarrhythmic Drug Therapy: New and Emerging Therapies. Cardiac electrophysiology clinics, 2(3), 471-478. https://doi.org/10.1016/j.ccep.2010.06.005
Pinter, Arnold, and Dorian, Paul. "Advances in Antiarrhythmic Drug Therapy: New and Emerging Therapies." Cardiac electrophysiology clinics vol. 2,3 (2010): 471-478. doi: https://doi.org/10.1016/j.ccep.2010.06.005
Pinter A, Dorian P. Advances in Antiarrhythmic Drug Therapy: New and Emerging Therapies. Card Electrophysiol Clin. 2010 Sep;2(3):471-478. doi: 10.1016/j.ccep.2010.06.005. PMID: 28770804.
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Outpatient intravenous sotalol load to replace 3-day admission oral sotalol load.

HeartRhythm case reports

Von Bergen NH, Beshish AG, Maginot KR.
PMID: 31341782
HeartRhythm Case Rep. 2019 Apr 24;5(7):382-383. doi: 10.1016/j.hrcr.2019.04.005. eCollection 2019 Jul.

No abstract available.

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Von Bergen NH, Beshish AG, Maginot KR. Outpatient intravenous sotalol load to replace 3-day admission oral sotalol load. HeartRhythm Case Rep. 2019;5(7):382-383doi: 10.1016/j.hrcr.2019.04.005.
Von Bergen, N. H., Beshish, A. G., & Maginot, K. R. (2019). Outpatient intravenous sotalol load to replace 3-day admission oral sotalol load. HeartRhythm case reports, 5(7), 382-383. https://doi.org/10.1016/j.hrcr.2019.04.005
Von Bergen, Nicholas H, et al. "Outpatient intravenous sotalol load to replace 3-day admission oral sotalol load." HeartRhythm case reports vol. 5,7 (2019): 382-383. doi: https://doi.org/10.1016/j.hrcr.2019.04.005
Von Bergen NH, Beshish AG, Maginot KR. Outpatient intravenous sotalol load to replace 3-day admission oral sotalol load. HeartRhythm Case Rep. 2019 Apr 24;5(7):382-383. doi: 10.1016/j.hrcr.2019.04.005. eCollection 2019 Jul. PMID: 31341782; PMCID: PMC6630186.
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Quinazolinone dimers as a potential new class of safer Kv1 inhibitors: Overcoming hERG, sodium and calcium channel affinities.

Bioorganic chemistry

Emam AM, Peigneur S, Depuydt AS, Ibrahim SM, Lopes Pinheiro-Junior E, El-Sadek M, Hendrickx L, Tytgat J, Kothayer H.
PMID: 34416509
Bioorg Chem. 2021 Oct;115:105264. doi: 10.1016/j.bioorg.2021.105264. Epub 2021 Aug 10.

The discovery of more selective and safer voltage-gated potassium channel blockers is an extremely demanding approach. Designing selective Kv1.5 inhibitors is very challenging as only limited data is available on this target due to a lacking crystal structure for...

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Emam AM, Peigneur S, Depuydt AS, et al. Quinazolinone dimers as a potential new class of safer Kv1 inhibitors: Overcoming hERG, sodium and calcium channel affinities. Bioorg Chem. 2021;115:105264doi: 10.1016/j.bioorg.2021.105264.
Emam, A. M., Peigneur, S., Depuydt, A. S., Ibrahim, S. M., Lopes Pinheiro-Junior, E., El-Sadek, M., Hendrickx, L., Tytgat, J., & Kothayer, H. (2021). Quinazolinone dimers as a potential new class of safer Kv1 inhibitors: Overcoming hERG, sodium and calcium channel affinities. Bioorganic chemistry, 115105264. https://doi.org/10.1016/j.bioorg.2021.105264
Emam, Aya M, et al. "Quinazolinone dimers as a potential new class of safer Kv1 inhibitors: Overcoming hERG, sodium and calcium channel affinities." Bioorganic chemistry vol. 115 (2021): 105264. doi: https://doi.org/10.1016/j.bioorg.2021.105264
Emam AM, Peigneur S, Depuydt AS, Ibrahim SM, Lopes Pinheiro-Junior E, El-Sadek M, Hendrickx L, Tytgat J, Kothayer H. Quinazolinone dimers as a potential new class of safer Kv1 inhibitors: Overcoming hERG, sodium and calcium channel affinities. Bioorg Chem. 2021 Oct;115:105264. doi: 10.1016/j.bioorg.2021.105264. Epub 2021 Aug 10. PMID: 34416509.
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Multiple Sclerosis: Symptomatic Treatment.

Current treatment options in neurology

Bever CT.
PMID: 11096711
Curr Treat Options Neurol. 1999 Jul;1(3):221-238. doi: 10.1007/s11940-999-0005-9.

Therapy for multiple sclerosis (MS) that prevents exacerbation of the disease and slows the progression of disability has not diminished the importance of treating symptoms. Because the new agents are not curative and rarely reverse existing deficits, many patients...

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Bever CT. Multiple Sclerosis: Symptomatic Treatment. Curr Treat Options Neurol. 1999;1(3):221-238doi: 10.1007/s11940-999-0005-9.
Bever, C. T. (1999). Multiple Sclerosis: Symptomatic Treatment. Current treatment options in neurology, 1(3), 221-238. https://doi.org/10.1007/s11940-999-0005-9
Bever, C T. "Multiple Sclerosis: Symptomatic Treatment." Current treatment options in neurology vol. 1,3 (1999): 221-238. doi: https://doi.org/10.1007/s11940-999-0005-9
Bever CT. Multiple Sclerosis: Symptomatic Treatment. Curr Treat Options Neurol. 1999 Jul;1(3):221-238. doi: 10.1007/s11940-999-0005-9. PMID: 11096711.
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The inhibitory effects of hydrogen sulfide on pacemaker activity of interstitial cells of cajal from mouse small intestine.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Parajuli SP, Choi S, Lee J, Kim YD, Park CG, Kim MY, Kim HI, Yeum CH, Jun JY.
PMID: 20473379
Korean J Physiol Pharmacol. 2010 Apr;14(2):83-9. doi: 10.4196/kjpp.2010.14.2.83. Epub 2010 Apr 30.

In this study, we studied whether hydrogen sulfide (H(2)S) has an effect on the pacemaker activity of interstitial cells of Cajal (ICC), in the small intestine of mice. The actions of H(2)S on pacemaker activity were investigated using whole-cell...

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Parajuli SP, Choi S, Lee J, et al. The inhibitory effects of hydrogen sulfide on pacemaker activity of interstitial cells of cajal from mouse small intestine. Korean J Physiol Pharmacol. 2010;14(2):83-9doi: 10.4196/kjpp.2010.14.2.83.
Parajuli, S. P., Choi, S., Lee, J., Kim, Y. D., Park, C. G., Kim, M. Y., Kim, H. I., Yeum, C. H., & Jun, J. Y. (2010). The inhibitory effects of hydrogen sulfide on pacemaker activity of interstitial cells of cajal from mouse small intestine. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 14(2), 83-9. https://doi.org/10.4196/kjpp.2010.14.2.83
Parajuli, Shankar Prasad, et al. "The inhibitory effects of hydrogen sulfide on pacemaker activity of interstitial cells of cajal from mouse small intestine." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 14,2 (2010): 83-9. doi: https://doi.org/10.4196/kjpp.2010.14.2.83
Parajuli SP, Choi S, Lee J, Kim YD, Park CG, Kim MY, Kim HI, Yeum CH, Jun JY. The inhibitory effects of hydrogen sulfide on pacemaker activity of interstitial cells of cajal from mouse small intestine. Korean J Physiol Pharmacol. 2010 Apr;14(2):83-9. doi: 10.4196/kjpp.2010.14.2.83. Epub 2010 Apr 30. PMID: 20473379; PMCID: PMC2869455.
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Inhibitory effects of endomorphin-2 on excitatory synaptic transmission and the neuronal excitability of sacral parasympathetic preganglionic neurons in young rats.

Frontiers in cellular neuroscience

Chen YB, Huang FS, Fen B, Yin JB, Wang W, Li YQ.
PMID: 26074773
Front Cell Neurosci. 2015 May 28;9:206. doi: 10.3389/fncel.2015.00206. eCollection 2015.

The function of the urinary bladder is partly controlled by parasympathetic preganglionic neurons (PPNs) of the sacral parasympathetic nucleus (SPN). Our recent work demonstrated that endomorphin-2 (EM-2)-immunoreactive (IR) terminals form synapses with μ-opioid receptor (MOR)-expressing PPNs in the rat...

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Chen YB, Huang FS, Fen B, et al. Inhibitory effects of endomorphin-2 on excitatory synaptic transmission and the neuronal excitability of sacral parasympathetic preganglionic neurons in young rats. Front Cell Neurosci. 2015;9:206doi: 10.3389/fncel.2015.00206.
Chen, Y. B., Huang, F. S., Fen, B., Yin, J. B., Wang, W., & Li, Y. Q. (2015). Inhibitory effects of endomorphin-2 on excitatory synaptic transmission and the neuronal excitability of sacral parasympathetic preganglionic neurons in young rats. Frontiers in cellular neuroscience, 9206. https://doi.org/10.3389/fncel.2015.00206
Chen, Ying-Biao, et al. "Inhibitory effects of endomorphin-2 on excitatory synaptic transmission and the neuronal excitability of sacral parasympathetic preganglionic neurons in young rats." Frontiers in cellular neuroscience vol. 9 (2015): 206. doi: https://doi.org/10.3389/fncel.2015.00206
Chen YB, Huang FS, Fen B, Yin JB, Wang W, Li YQ. Inhibitory effects of endomorphin-2 on excitatory synaptic transmission and the neuronal excitability of sacral parasympathetic preganglionic neurons in young rats. Front Cell Neurosci. 2015 May 28;9:206. doi: 10.3389/fncel.2015.00206. eCollection 2015. PMID: 26074773; PMCID: PMC4446531.
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Pharmacophore mapping based inhibitor selection and molecular interaction studies for identification of potential drugs on calcium activated potassium channel blockers, tamulotoxin.

Pharmacognosy magazine

Kumar RB, Suresh MX.
PMID: 23772102
Pharmacogn Mag. 2013 Apr;9(34):89-95. doi: 10.4103/0973-1296.111239.

BACKGROUND: Tamulotoxin (TmTx) from Buthus tamulus was found to be a highly venomous toxin which accelerates the neurotransmitter release that directly affects the cardiovascular tissues and the respiratory system leading to death. TmTx from red Indian scorpion is a...

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Kumar RB, Suresh MX. Pharmacophore mapping based inhibitor selection and molecular interaction studies for identification of potential drugs on calcium activated potassium channel blockers, tamulotoxin. Pharmacogn Mag. 2013;9(34):89-95doi: 10.4103/0973-1296.111239.
Kumar, R. B., & Suresh, M. X. (2013). Pharmacophore mapping based inhibitor selection and molecular interaction studies for identification of potential drugs on calcium activated potassium channel blockers, tamulotoxin. Pharmacognosy magazine, 9(34), 89-95. https://doi.org/10.4103/0973-1296.111239
Kumar, R Barani, and Suresh, M Xavier. "Pharmacophore mapping based inhibitor selection and molecular interaction studies for identification of potential drugs on calcium activated potassium channel blockers, tamulotoxin." Pharmacognosy magazine vol. 9,34 (2013): 89-95. doi: https://doi.org/10.4103/0973-1296.111239
Kumar RB, Suresh MX. Pharmacophore mapping based inhibitor selection and molecular interaction studies for identification of potential drugs on calcium activated potassium channel blockers, tamulotoxin. Pharmacogn Mag. 2013 Apr;9(34):89-95. doi: 10.4103/0973-1296.111239. PMID: 23772102; PMCID: PMC3680861.
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Identification of a novel potassium channel (GiK) as a potential drug target in .

PeerJ

Palomo-Ligas L, Gutiérrez-Gutiérrez F, Ochoa-Maganda VY, Cortés-Zárate R, Charles-Niño CL, Castillo-Romero A.
PMID: 30834181
PeerJ. 2019 Feb 27;7:e6430. doi: 10.7717/peerj.6430. eCollection 2019.

BACKGROUND: The protozoan METHODS: In this work, we identify and characterize a new potassium channel, GiK, in the genome of RESULTS: The GiK sequence showed 24-50% identity and 50-90% positivity with 21 different types of potassium channels. The quality...

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Palomo-Ligas L, Gutiérrez-Gutiérrez F, Ochoa-Maganda VY, et al. Identification of a novel potassium channel (GiK) as a potential drug target in . PeerJ. 2019;7:e6430doi: 10.7717/peerj.6430.
Palomo-Ligas, L., Gutiérrez-Gutiérrez, F., Ochoa-Maganda, V. Y., Cortés-Zárate, R., Charles-Niño, C. L., & Castillo-Romero, A. (2019). Identification of a novel potassium channel (GiK) as a potential drug target in . PeerJ, 7e6430. https://doi.org/10.7717/peerj.6430
Palomo-Ligas, Lissethe, et al. "Identification of a novel potassium channel (GiK) as a potential drug target in ." PeerJ vol. 7 (2019): e6430. doi: https://doi.org/10.7717/peerj.6430
Palomo-Ligas L, Gutiérrez-Gutiérrez F, Ochoa-Maganda VY, Cortés-Zárate R, Charles-Niño CL, Castillo-Romero A. Identification of a novel potassium channel (GiK) as a potential drug target in . PeerJ. 2019 Feb 27;7:e6430. doi: 10.7717/peerj.6430. eCollection 2019. PMID: 30834181; PMCID: PMC6397635.
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Showing 25 to 36 of 42 entries