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Effect of decompression-induced bubble formation on highly trained divers microvascular function.

Physiological reports

Lambrechts K, Pontier JM, Mazur A, Buzzacott P, Morin J, Wang Q, Theron M, Guerrero F.
PMID: 24400144
Physiol Rep. 2013 Nov;1(6):e00142. doi: 10.1002/phy2.142. Epub 2013 Nov 07.

We previously showed microvascular alteration of both endothelium-dependent and -independent reactivity after a single SCUBA dive. We aimed to study mechanisms involved in this postdive vascular dysfunction. Ten divers each completed three protocols: (1) a SCUBA dive at 400...

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Lambrechts K, Pontier JM, Mazur A, et al. Effect of decompression-induced bubble formation on highly trained divers microvascular function. Physiol Rep. 2013;1(6):e00142doi: 10.1002/phy2.142.
Lambrechts, K., Pontier, J. M., Mazur, A., Buzzacott, P., Morin, J., Wang, Q., Theron, M., & Guerrero, F. (2013). Effect of decompression-induced bubble formation on highly trained divers microvascular function. Physiological reports, 1(6), e00142. https://doi.org/10.1002/phy2.142
Lambrechts, Kate, et al. "Effect of decompression-induced bubble formation on highly trained divers microvascular function." Physiological reports vol. 1,6 (2013): e00142. doi: https://doi.org/10.1002/phy2.142
Lambrechts K, Pontier JM, Mazur A, Buzzacott P, Morin J, Wang Q, Theron M, Guerrero F. Effect of decompression-induced bubble formation on highly trained divers microvascular function. Physiol Rep. 2013 Nov;1(6):e00142. doi: 10.1002/phy2.142. Epub 2013 Nov 07. PMID: 24400144; PMCID: PMC3871457.
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Activation of coagulation and fibrinolysis after off-pump coronary artery bypass grafting with or without endotracheal general anesthesia.

Innovations (Philadelphia, Pa.)

Yachi T, Watanabe G, Tomita S.
PMID: 22437641
Innovations (Phila). 2010 Nov;5(6):444-9. doi: 10.1097/IMI.0b013e3181f8b615.

OBJECTIVE: : In this study, we measured the activity of coagulation and fibrinolysis and clarified the presence of certain differences between off-pump coronary artery bypass grafting (OPCAB) cases and awake off-pump coronary artery bypass grafting (AOCAB) cases to evaluate...

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Yachi T, Watanabe G, Tomita S. Activation of coagulation and fibrinolysis after off-pump coronary artery bypass grafting with or without endotracheal general anesthesia. Innovations (Phila). 2010;5(6):444-9doi: 10.1097/IMI.0b013e3181f8b615.
Yachi, T., Watanabe, G., & Tomita, S. (2010). Activation of coagulation and fibrinolysis after off-pump coronary artery bypass grafting with or without endotracheal general anesthesia. Innovations (Philadelphia, Pa.), 5(6), 444-9. https://doi.org/10.1097/IMI.0b013e3181f8b615
Yachi, Tsuyoshi, et al. "Activation of coagulation and fibrinolysis after off-pump coronary artery bypass grafting with or without endotracheal general anesthesia." Innovations (Philadelphia, Pa.) vol. 5,6 (2010): 444-9. doi: https://doi.org/10.1097/IMI.0b013e3181f8b615
Yachi T, Watanabe G, Tomita S. Activation of coagulation and fibrinolysis after off-pump coronary artery bypass grafting with or without endotracheal general anesthesia. Innovations (Phila). 2010 Nov;5(6):444-9. doi: 10.1097/IMI.0b013e3181f8b615. PMID: 22437641.
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Proteomic analysis of immediate-early response plasma proteins after 70% and 90% partial hepatectomy.

Hepatology research : the official journal of the Japan Society of Hepatology

Kumar S, Zou Y, Bao Q, Wang M, Dai G.
PMID: 23279269
Hepatol Res. 2013 Aug;43(8):876-89. doi: 10.1111/hepr.12030. Epub 2012 Dec 20.

AIM: Partial hepatectomy (PH) induces robust hepatic regenerative and metabolic responses that are considered to be triggered by humoral factors. The aim of the study was to identify plasma protein factors that potentially trigger or reflect the body's immediate-early...

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Kumar S, Zou Y, Bao Q, et al. Proteomic analysis of immediate-early response plasma proteins after 70% and 90% partial hepatectomy. Hepatol Res. 2012;43(8):876-89doi: 10.1111/hepr.12030.
Kumar, S., Zou, Y., Bao, Q., Wang, M., & Dai, G. (2013). Proteomic analysis of immediate-early response plasma proteins after 70% and 90% partial hepatectomy. Hepatology research : the official journal of the Japan Society of Hepatology, 43(8), 876-89. https://doi.org/10.1111/hepr.12030
Kumar, Sudhanshu, et al. "Proteomic analysis of immediate-early response plasma proteins after 70% and 90% partial hepatectomy." Hepatology research : the official journal of the Japan Society of Hepatology vol. 43,8 (2013): 876-89. doi: https://doi.org/10.1111/hepr.12030
Kumar S, Zou Y, Bao Q, Wang M, Dai G. Proteomic analysis of immediate-early response plasma proteins after 70% and 90% partial hepatectomy. Hepatol Res. 2013 Aug;43(8):876-89. doi: 10.1111/hepr.12030. Epub 2012 Dec 20. PMID: 23279269; PMCID: PMC4354878.
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Plasma inorganic pyrophosphate and alkaline phosphatase in patients with pseudoxanthoma elasticum.

Annals of translational medicine

Sánchez-Tévar AM, García-Fernández M, Murcia-Casas B, Rioja-Villodres J, Carrillo JL, Camacho M, Van Gils M, Sánchez-Chaparro MA, Vanakker O, Valdivielso P.
PMID: 32042814
Ann Transl Med. 2019 Dec;7(24):798. doi: 10.21037/atm.2019.12.73.

BACKGROUND: Inorganic pyrophosphate (PPi) plays a major role inhibiting dystrophic calcification. The aim was to analyze levels of PPi in patients having pseudoxanthoma elasticum (PXE), and controls as well as the enzymes who regulate the PPi plasma concentration.METHODS: We...

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Sánchez-Tévar AM, García-Fernández M, Murcia-Casas B, et al. Plasma inorganic pyrophosphate and alkaline phosphatase in patients with pseudoxanthoma elasticum. Ann Transl Med. 2019;7(24):798doi: 10.21037/atm.2019.12.73.
Sánchez-Tévar, A. M., García-Fernández, M., Murcia-Casas, B., Rioja-Villodres, J., Carrillo, J. L., Camacho, M., Van Gils, M., Sánchez-Chaparro, M. A., Vanakker, O., & Valdivielso, P. (2019). Plasma inorganic pyrophosphate and alkaline phosphatase in patients with pseudoxanthoma elasticum. Annals of translational medicine, 7(24), 798. https://doi.org/10.21037/atm.2019.12.73
Sánchez-Tévar, Ana María, et al. "Plasma inorganic pyrophosphate and alkaline phosphatase in patients with pseudoxanthoma elasticum." Annals of translational medicine vol. 7,24 (2019): 798. doi: https://doi.org/10.21037/atm.2019.12.73
Sánchez-Tévar AM, García-Fernández M, Murcia-Casas B, Rioja-Villodres J, Carrillo JL, Camacho M, Van Gils M, Sánchez-Chaparro MA, Vanakker O, Valdivielso P. Plasma inorganic pyrophosphate and alkaline phosphatase in patients with pseudoxanthoma elasticum. Ann Transl Med. 2019 Dec;7(24):798. doi: 10.21037/atm.2019.12.73. PMID: 32042814; PMCID: PMC6989879.
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Pulsed Radiofrequency Neuromodulation Contributes to Activation of Platelet-Rich Plasma in In Vitro Conditions.

Neuromodulation : journal of the International Neuromodulation Society

Michno A, Kirkor Z, Gojtowska E, Suchorzewski M, Śmietańska I, Baścik B.
PMID: 32027438
Neuromodulation. 2020 Feb 06; doi: 10.1111/ner.13105. Epub 2020 Feb 06.

OBJECTIVES: Recent years have brought new developments in interventional chronic pain management, namely regenerative orthopedics utilizing platelet-rich plasma (PRP) as well as further evolution of pulsed radiofrequency neuromodulation (PRF). Both methods have been used separately. Here, we investigated whether...

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Michno A, Kirkor Z, Gojtowska E, et al. Pulsed Radiofrequency Neuromodulation Contributes to Activation of Platelet-Rich Plasma in In Vitro Conditions. Neuromodulation. 2020;doi: 10.1111/ner.13105.
Michno, A., Kirkor, Z., Gojtowska, E., Suchorzewski, M., Śmietańska, I., & Baścik, B. (2020). Pulsed Radiofrequency Neuromodulation Contributes to Activation of Platelet-Rich Plasma in In Vitro Conditions. Neuromodulation : journal of the International Neuromodulation Society, . https://doi.org/10.1111/ner.13105
Michno, Anna, et al. "Pulsed Radiofrequency Neuromodulation Contributes to Activation of Platelet-Rich Plasma in In Vitro Conditions." Neuromodulation : journal of the International Neuromodulation Society vol. (2020). doi: https://doi.org/10.1111/ner.13105
Michno A, Kirkor Z, Gojtowska E, Suchorzewski M, Śmietańska I, Baścik B. Pulsed Radiofrequency Neuromodulation Contributes to Activation of Platelet-Rich Plasma in In Vitro Conditions. Neuromodulation. 2020 Feb 06; doi: 10.1111/ner.13105. Epub 2020 Feb 06. PMID: 32027438.
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Polyphosphate/platelet factor 4 complexes can mediate heparin-independent platelet activation in heparin-induced thrombocytopenia.

Blood advances

Cines DB, Yarovoi SV, Zaitsev SV, Lebedeva T, Rauova L, Poncz M, Arepally GM, Khandelwal S, Stepanova V, Rux AH, Cuker A, Guo C, Ocariza LM, Travers RJ, Smith SA, Kim H, Morrissey JH, Conway EM.
PMID: 29296696
Blood Adv. 2016 Nov 22;1(1):62-74. doi: 10.1182/bloodadvances.2016000877. eCollection 2016 Nov 29.

Heparin-induced thrombocytopenia (HIT) is a thrombotic disorder initiated by antibodies to complexes between platelet factor 4 (PF4) and heparin. The risk of recurrent thromboembolism persists after heparin is cleared and platelet activation leading to release of PF4 has dissipated....

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Cines DB, Yarovoi SV, Zaitsev SV, et al. Polyphosphate/platelet factor 4 complexes can mediate heparin-independent platelet activation in heparin-induced thrombocytopenia. Blood Adv. 2016;1(1):62-74doi: 10.1182/bloodadvances.2016000877.
Cines, D. B., Yarovoi, S. V., Zaitsev, S. V., Lebedeva, T., Rauova, L., Poncz, M., Arepally, G. M., Khandelwal, S., Stepanova, V., Rux, A. H., Cuker, A., Guo, C., Ocariza, L. M., Travers, R. J., Smith, S. A., Kim, H., Morrissey, J. H., & Conway, E. M. (2016). Polyphosphate/platelet factor 4 complexes can mediate heparin-independent platelet activation in heparin-induced thrombocytopenia. Blood advances, 1(1), 62-74. https://doi.org/10.1182/bloodadvances.2016000877
Cines, Douglas B, et al. "Polyphosphate/platelet factor 4 complexes can mediate heparin-independent platelet activation in heparin-induced thrombocytopenia." Blood advances vol. 1,1 (2016): 62-74. doi: https://doi.org/10.1182/bloodadvances.2016000877
Cines DB, Yarovoi SV, Zaitsev SV, Lebedeva T, Rauova L, Poncz M, Arepally GM, Khandelwal S, Stepanova V, Rux AH, Cuker A, Guo C, Ocariza LM, Travers RJ, Smith SA, Kim H, Morrissey JH, Conway EM. Polyphosphate/platelet factor 4 complexes can mediate heparin-independent platelet activation in heparin-induced thrombocytopenia. Blood Adv. 2016 Nov 22;1(1):62-74. doi: 10.1182/bloodadvances.2016000877. eCollection 2016 Nov 29. PMID: 29296696; PMCID: PMC5744055.
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Exploring the Extracellular Regulation of the Tumor Angiogenic Interaction Network Using a Systems Biology Model.

Frontiers in physiology

Li D, Finley SD.
PMID: 31379588
Front Physiol. 2019 Jul 18;10:823. doi: 10.3389/fphys.2019.00823. eCollection 2019.

Tumor angiogenesis is regulated by pro- and anti-angiogenic factors. Anti-angiogenic agents target the interconnected network of angiogenic factors to inhibit neovascularization, which subsequently impedes tumor growth. Due to the complexity of this network, optimizing anti-angiogenic cancer treatments requires detailed...

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Li D, Finley SD. Exploring the Extracellular Regulation of the Tumor Angiogenic Interaction Network Using a Systems Biology Model. Front Physiol. 2019;10:823doi: 10.3389/fphys.2019.00823.
Li, D., & Finley, S. D. (2019). Exploring the Extracellular Regulation of the Tumor Angiogenic Interaction Network Using a Systems Biology Model. Frontiers in physiology, 10823. https://doi.org/10.3389/fphys.2019.00823
Li, Ding, and Finley, Stacey D. "Exploring the Extracellular Regulation of the Tumor Angiogenic Interaction Network Using a Systems Biology Model." Frontiers in physiology vol. 10 (2019): 823. doi: https://doi.org/10.3389/fphys.2019.00823
Li D, Finley SD. Exploring the Extracellular Regulation of the Tumor Angiogenic Interaction Network Using a Systems Biology Model. Front Physiol. 2019 Jul 18;10:823. doi: 10.3389/fphys.2019.00823. eCollection 2019. PMID: 31379588; PMCID: PMC6656929.
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Response to The challenges of diagnosing heparin-induced thrombocytopenia in patients with COVID-19.

Research and practice in thrombosis and haemostasis

Riker RR, May TL, Fraser GL, Gagnon DJ, Bandara M, Zemrak W, Seder DB.
PMID: 32838110
Res Pract Thromb Haemost. 2020 Jul 09; doi: 10.1002/rth2.12417. Epub 2020 Jul 09.

We thank May et al for their comments, expanding the number of reported cases of suspected and confirmed heparin-induced thrombocytopenia (HIT) associated with COVID-19, and reemphasizing the complexity of the prothrombotic state observed (1). We agree that false-positive enzyme...

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Riker RR, May TL, Fraser GL, et al. Response to The challenges of diagnosing heparin-induced thrombocytopenia in patients with COVID-19. Res Pract Thromb Haemost. 2020;doi: 10.1002/rth2.12417.
Riker, R. R., May, T. L., Fraser, G. L., Gagnon, D. J., Bandara, M., Zemrak, W., & Seder, D. B. (2020). Response to The challenges of diagnosing heparin-induced thrombocytopenia in patients with COVID-19. Research and practice in thrombosis and haemostasis, . https://doi.org/10.1002/rth2.12417
Riker, Richard R, et al. "Response to The challenges of diagnosing heparin-induced thrombocytopenia in patients with COVID-19." Research and practice in thrombosis and haemostasis vol. (2020). doi: https://doi.org/10.1002/rth2.12417
Riker RR, May TL, Fraser GL, Gagnon DJ, Bandara M, Zemrak W, Seder DB. Response to The challenges of diagnosing heparin-induced thrombocytopenia in patients with COVID-19. Res Pract Thromb Haemost. 2020 Jul 09; doi: 10.1002/rth2.12417. Epub 2020 Jul 09. PMID: 32838110; PMCID: PMC7361545.
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Scientific premise for the involvement of neutrophil extracellular traps (NETs) in vaccine-induced thrombotic thrombocytopenia (VITT).

Journal of leukocyte biology

Kashir J, Ambia AR, Shafqat A, Sajid MR, AlKattan K, Yaqinuddin A.
PMID: 34467562
J Leukoc Biol. 2021 Sep 01; doi: 10.1002/JLB.5COVR0621-320RR. Epub 2021 Sep 01.

Following on from the devastating spread of COVID-19, a major global priority has been the production, procurement, and distribution of effective vaccines to ensure that the global pandemic reaches an end. However, concerns were raised about worrying side effects,...

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Kashir J, Ambia AR, Shafqat A, et al. Scientific premise for the involvement of neutrophil extracellular traps (NETs) in vaccine-induced thrombotic thrombocytopenia (VITT). J Leukoc Biol. 2021;doi: 10.1002/JLB.5COVR0621-320RR.
Kashir, J., Ambia, A. R., Shafqat, A., Sajid, M. R., AlKattan, K., & Yaqinuddin, A. (2021). Scientific premise for the involvement of neutrophil extracellular traps (NETs) in vaccine-induced thrombotic thrombocytopenia (VITT). Journal of leukocyte biology, . https://doi.org/10.1002/JLB.5COVR0621-320RR
Kashir, Junaid, et al. "Scientific premise for the involvement of neutrophil extracellular traps (NETs) in vaccine-induced thrombotic thrombocytopenia (VITT)." Journal of leukocyte biology vol. (2021). doi: https://doi.org/10.1002/JLB.5COVR0621-320RR
Kashir J, Ambia AR, Shafqat A, Sajid MR, AlKattan K, Yaqinuddin A. Scientific premise for the involvement of neutrophil extracellular traps (NETs) in vaccine-induced thrombotic thrombocytopenia (VITT). J Leukoc Biol. 2021 Sep 01; doi: 10.1002/JLB.5COVR0621-320RR. Epub 2021 Sep 01. PMID: 34467562; PMCID: PMC8667645.
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COVID-19 adenovirus vaccine triggers antibodies against PF4 complexes to activate complement and platelets.

Thrombosis research

Pitkänen HH, Jouppila A, Helin T, Dulipati V, Kotimaa J, Meri S, Kantele A, Jalkanen P, Julkunen I, Lassila R.
PMID: 34768097
Thromb Res. 2021 Dec;208:129-137. doi: 10.1016/j.thromres.2021.10.027. Epub 2021 Nov 06.

BACKGROUND: Vaccine-induced thrombotic thrombocytopenia (VITT) is a rare coagulation disorder reported after administration of COVID-19 adenovirus-vectored vaccines. VITT is mediated by anti-platelet factor 4 (PF4) antibodies activating platelets through the Fcγ-receptor II (FcγRII), and it is associated with strong...

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Pitkänen HH, Jouppila A, Helin T, et al. COVID-19 adenovirus vaccine triggers antibodies against PF4 complexes to activate complement and platelets. Thromb Res. 2021;208:129-137doi: 10.1016/j.thromres.2021.10.027.
Pitkänen, H. H., Jouppila, A., Helin, T., Dulipati, V., Kotimaa, J., Meri, S., Kantele, A., Jalkanen, P., Julkunen, I., & Lassila, R. (2021). COVID-19 adenovirus vaccine triggers antibodies against PF4 complexes to activate complement and platelets. Thrombosis research, 208129-137. https://doi.org/10.1016/j.thromres.2021.10.027
Pitkänen, Hanna H, et al. "COVID-19 adenovirus vaccine triggers antibodies against PF4 complexes to activate complement and platelets." Thrombosis research vol. 208 (2021): 129-137. doi: https://doi.org/10.1016/j.thromres.2021.10.027
Pitkänen HH, Jouppila A, Helin T, Dulipati V, Kotimaa J, Meri S, Kantele A, Jalkanen P, Julkunen I, Lassila R. COVID-19 adenovirus vaccine triggers antibodies against PF4 complexes to activate complement and platelets. Thromb Res. 2021 Dec;208:129-137. doi: 10.1016/j.thromres.2021.10.027. Epub 2021 Nov 06. PMID: 34768097; PMCID: PMC8571998.
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Complement mediates binding and procoagulant effects of ultralarge HIT immune complexes.

Blood

Khandelwal S, Barnes A, Rauova L, Sarkar A, Rux AH, Yarovoi SV, Zaitsev SS, Lambris JD, Myoung SS, Johnson A, Lee GM, Duarte M, Poncz M, Arepally GM, Cines DB.
PMID: 34189574
Blood. 2021 Nov 25;138(21):2106-2116. doi: 10.1182/blood.2020009487.

Heparin-induced thrombocytopenia (HIT) is a prothrombotic disorder mediated by ultra-large immune complexes (ULICs) containing immunoglobulin G (IgG) antibodies to a multivalent antigen composed of platelet factor 4 and heparin. The limitations of current antithrombotic therapy in HIT supports the...

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Khandelwal S, Barnes A, Rauova L, et al. Complement mediates binding and procoagulant effects of ultralarge HIT immune complexes. Blood. 2021;138(21):2106-2116doi: 10.1182/blood.2020009487.
Khandelwal, S., Barnes, A., Rauova, L., Sarkar, A., Rux, A. H., Yarovoi, S. V., Zaitsev, S. S., Lambris, J. D., Myoung, S. S., Johnson, A., Lee, G. M., Duarte, M., Poncz, M., Arepally, G. M., & Cines, D. B. (2021). Complement mediates binding and procoagulant effects of ultralarge HIT immune complexes. Blood, 138(21), 2106-2116. https://doi.org/10.1182/blood.2020009487
Khandelwal, Sanjay, et al. "Complement mediates binding and procoagulant effects of ultralarge HIT immune complexes." Blood vol. 138,21 (2021): 2106-2116. doi: https://doi.org/10.1182/blood.2020009487
Khandelwal S, Barnes A, Rauova L, Sarkar A, Rux AH, Yarovoi SV, Zaitsev SS, Lambris JD, Myoung SS, Johnson A, Lee GM, Duarte M, Poncz M, Arepally GM, Cines DB. Complement mediates binding and procoagulant effects of ultralarge HIT immune complexes. Blood. 2021 Nov 25;138(21):2106-2116. doi: 10.1182/blood.2020009487. PMID: 34189574; PMCID: PMC8617432.
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The roles of platelets in COVID-19-associated coagulopathy and vaccine-induced immune thrombotic thrombocytopenia.

Trends in cardiovascular medicine

Iba T, Levy JH.
PMID: 34455073
Trends Cardiovasc Med. 2021 Aug 27; doi: 10.1016/j.tcm.2021.08.012. Epub 2021 Aug 27.

In coronavirus disease 2019 (COVID-19), multiple thromboinflammatory events contribute to the pathophysiology, including coagulation system activation, suppressed fibrinolysis, vascular endothelial cell injury, and prothrombotic alterations in immune cells such as macrophages and neutrophils. Although thrombocytopenia is not an initial...

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Iba T, Levy JH. The roles of platelets in COVID-19-associated coagulopathy and vaccine-induced immune thrombotic thrombocytopenia. Trends Cardiovasc Med. 2021;doi: 10.1016/j.tcm.2021.08.012.
Iba, T., & Levy, J. H. (2021). The roles of platelets in COVID-19-associated coagulopathy and vaccine-induced immune thrombotic thrombocytopenia. Trends in cardiovascular medicine, . https://doi.org/10.1016/j.tcm.2021.08.012
Iba, Toshiaki, and Levy, Jerrold H. "The roles of platelets in COVID-19-associated coagulopathy and vaccine-induced immune thrombotic thrombocytopenia." Trends in cardiovascular medicine vol. (2021). doi: https://doi.org/10.1016/j.tcm.2021.08.012
Iba T, Levy JH. The roles of platelets in COVID-19-associated coagulopathy and vaccine-induced immune thrombotic thrombocytopenia. Trends Cardiovasc Med. 2021 Aug 27; doi: 10.1016/j.tcm.2021.08.012. Epub 2021 Aug 27. PMID: 34455073; PMCID: PMC8390120.
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Showing 13 to 24 of 257 entries