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Showing 13 to 24 of 181 entries
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Cellular assessment of muscle in COPD: case studies of two males.

International journal of general medicine

Green HJ, Bombardier E, Burnett ME, D'Arsigny CL, Iqbal S, Webb KA, Ouyang J, O'Donnell DE.
PMID: 20360908
Int J Gen Med. 2009 Dec 29;2:227-42. doi: 10.2147/ijgm.s5981.

The objective of this paper is to provide an overview of the recent developments in muscle physiology and biochemistry in general, and with respect to chronic obstructive pulmonary disease (COPD) specifically. As a way of illustration, we have presented...

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Green HJ, Bombardier E, Burnett ME, et al. Cellular assessment of muscle in COPD: case studies of two males. Int J Gen Med. 2009;2:227-42doi: 10.2147/ijgm.s5981.
Green, H. J., Bombardier, E., Burnett, M. E., D'Arsigny, C. L., Iqbal, S., Webb, K. A., Ouyang, J., & O'Donnell, D. E. (2009). Cellular assessment of muscle in COPD: case studies of two males. International journal of general medicine, 2227-42. https://doi.org/10.2147/ijgm.s5981
Green, Howard J, et al. "Cellular assessment of muscle in COPD: case studies of two males." International journal of general medicine vol. 2 (2009): 227-42. doi: https://doi.org/10.2147/ijgm.s5981
Green HJ, Bombardier E, Burnett ME, D'Arsigny CL, Iqbal S, Webb KA, Ouyang J, O'Donnell DE. Cellular assessment of muscle in COPD: case studies of two males. Int J Gen Med. 2009 Dec 29;2:227-42. doi: 10.2147/ijgm.s5981. PMID: 20360908; PMCID: PMC2840564.
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Use of perfused isolated muscle, as studied by (31)P NMR, to investigate metabolism and post-mortem changes.

Meat science

Miri A, Foucat L, Renou JP, Rodet L, Talmant A, Monin G, Kozak-Reiss G, Gascard JP, Bérenger G.
PMID: 22059403
Meat Sci. 1991;30(4):327-36. doi: 10.1016/0309-1740(91)90041-N.

An experimental system was designed to study as independently as possible the effects of various in-vivo or post-mortem factors susceptible to influence muscle metabolism. This system was made up of an NMR probe, a physiological stimulator, a perfusion system...

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Miri A, Foucat L, Renou JP, et al. Use of perfused isolated muscle, as studied by (31)P NMR, to investigate metabolism and post-mortem changes. Meat Sci. 1991;30(4):327-36doi: 10.1016/0309-1740(91)90041-N.
Miri, A., Foucat, L., Renou, J. P., Rodet, L., Talmant, A., Monin, G., Kozak-Reiss, G., Gascard, J. P., & Bérenger, G. (1991). Use of perfused isolated muscle, as studied by (31)P NMR, to investigate metabolism and post-mortem changes. Meat science, 30(4), 327-36. https://doi.org/10.1016/0309-1740(91)90041-N
Miri, A, et al. "Use of perfused isolated muscle, as studied by (31)P NMR, to investigate metabolism and post-mortem changes." Meat science vol. 30,4 (1991): 327-36. doi: https://doi.org/10.1016/0309-1740(91)90041-N
Miri A, Foucat L, Renou JP, Rodet L, Talmant A, Monin G, Kozak-Reiss G, Gascard JP, Bérenger G. Use of perfused isolated muscle, as studied by (31)P NMR, to investigate metabolism and post-mortem changes. Meat Sci. 1991;30(4):327-36. doi: 10.1016/0309-1740(91)90041-N. PMID: 22059403.
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Cytoskeleton and regulation of mitochondrial function: the role of beta-tubulin II.

Frontiers in physiology

Kuznetsov AV, Javadov S, Guzun R, Grimm M, Saks V.
PMID: 23630499
Front Physiol. 2013 Apr 22;4:82. doi: 10.3389/fphys.2013.00082. eCollection 2013.

The control of mitochondrial function is a cardinal issue in the field of cardiac bioenergetics, and the analysis of mitochondrial regulations is central to basic research and in the diagnosis of many diseases. Interaction between cytoskeletal proteins and mitochondria...

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Kuznetsov AV, Javadov S, Guzun R, et al. Cytoskeleton and regulation of mitochondrial function: the role of beta-tubulin II. Front Physiol. 2013;4:82doi: 10.3389/fphys.2013.00082.
Kuznetsov, A. V., Javadov, S., Guzun, R., Grimm, M., & Saks, V. (2013). Cytoskeleton and regulation of mitochondrial function: the role of beta-tubulin II. Frontiers in physiology, 482. https://doi.org/10.3389/fphys.2013.00082
Kuznetsov, Andrey V, et al. "Cytoskeleton and regulation of mitochondrial function: the role of beta-tubulin II." Frontiers in physiology vol. 4 (2013): 82. doi: https://doi.org/10.3389/fphys.2013.00082
Kuznetsov AV, Javadov S, Guzun R, Grimm M, Saks V. Cytoskeleton and regulation of mitochondrial function: the role of beta-tubulin II. Front Physiol. 2013 Apr 22;4:82. doi: 10.3389/fphys.2013.00082. eCollection 2013. PMID: 23630499; PMCID: PMC3631707.
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Resistance Exercise Training in McArdle Disease: Myth or Reality?.

Case reports in neurological medicine

Pietrusz A, Scalco RS, Quinlivan R.
PMID: 30363996
Case Rep Neurol Med. 2018 Sep 30;2018:9658251. doi: 10.1155/2018/9658251. eCollection 2018.

McArdle disease is a metabolic myopathy mainly characterised by symptom onset during physical activities or isometric muscle contraction. Resistance (also termed strength) training is a type of physical exercise focusing on the use of resistance (e.g., lifting weights) to...

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Pietrusz A, Scalco RS, Quinlivan R. Resistance Exercise Training in McArdle Disease: Myth or Reality?. Case Rep Neurol Med. 2018;2018:9658251doi: 10.1155/2018/9658251.
Pietrusz, A., Scalco, R. S., & Quinlivan, R. (2018). Resistance Exercise Training in McArdle Disease: Myth or Reality?. Case reports in neurological medicine, 20189658251. https://doi.org/10.1155/2018/9658251
Pietrusz, Aleksandra, et al. "Resistance Exercise Training in McArdle Disease: Myth or Reality?." Case reports in neurological medicine vol. 2018 (2018): 9658251. doi: https://doi.org/10.1155/2018/9658251
Pietrusz A, Scalco RS, Quinlivan R. Resistance Exercise Training in McArdle Disease: Myth or Reality?. Case Rep Neurol Med. 2018 Sep 30;2018:9658251. doi: 10.1155/2018/9658251. eCollection 2018. PMID: 30363996; PMCID: PMC6186374.
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Frontal Metabolite Concentration Deficits in Opiate Dependence Relate to Substance Use, Cognition, and Self-Regulation.

Journal of addiction research & therapy

Murray DE, Durazzo TC, Schmidt TP, Abé C, Guydish J, Meyerhoff DJ.
PMID: 27695638
J Addict Res Ther. 2016 Aug;7(4). doi: 10.4172/2155-6105.1000286. Epub 2016 Jul 15.

OBJECTIVE: Proton magnetic resonance spectroscopy (1H MRS) in opiate dependence showed abnormalities in neuronal viability and glutamate concentration in the anterior cingulate cortex (ACC). Metabolite levels in dorsolateral prefrontal cortex (DLPFC) or orbitofrontal cortex (OFC) and their neuropsychological correlates...

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Murray DE, Durazzo TC, Schmidt TP, et al. Frontal Metabolite Concentration Deficits in Opiate Dependence Relate to Substance Use, Cognition, and Self-Regulation. J Addict Res Ther. 2016;7(4)doi: 10.4172/2155-6105.1000286.
Murray, D. E., Durazzo, T. C., Schmidt, T. P., Abé, C., Guydish, J., & Meyerhoff, D. J. (2016). Frontal Metabolite Concentration Deficits in Opiate Dependence Relate to Substance Use, Cognition, and Self-Regulation. Journal of addiction research & therapy, 7(4), . https://doi.org/10.4172/2155-6105.1000286
Murray, Donna E, et al. "Frontal Metabolite Concentration Deficits in Opiate Dependence Relate to Substance Use, Cognition, and Self-Regulation." Journal of addiction research & therapy vol. 7,4 (2016). doi: https://doi.org/10.4172/2155-6105.1000286
Murray DE, Durazzo TC, Schmidt TP, Abé C, Guydish J, Meyerhoff DJ. Frontal Metabolite Concentration Deficits in Opiate Dependence Relate to Substance Use, Cognition, and Self-Regulation. J Addict Res Ther. 2016 Aug;7(4). doi: 10.4172/2155-6105.1000286. Epub 2016 Jul 15. PMID: 27695638; PMCID: PMC5042152.
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Creatine kinase in ischemic and inflammatory disorders.

Clinical and translational medicine

Kitzenberg D, Colgan SP, Glover LE.
PMID: 27527620
Clin Transl Med. 2016 Dec;5(1):31. doi: 10.1186/s40169-016-0114-5. Epub 2016 Aug 15.

The creatine/phosphocreatine pathway plays a conserved and central role in energy metabolism. Compartmentalization of specific creatine kinase enzymes permits buffering of local high energy phosphates in a thermodynamically favorable manner, enabling both rapid energy storage and energy transfer within...

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Kitzenberg D, Colgan SP, Glover LE. Creatine kinase in ischemic and inflammatory disorders. Clin Transl Med. 2016;5(1):31doi: 10.1186/s40169-016-0114-5.
Kitzenberg, D., Colgan, S. P., & Glover, L. E. (2016). Creatine kinase in ischemic and inflammatory disorders. Clinical and translational medicine, 5(1), 31. https://doi.org/10.1186/s40169-016-0114-5
Kitzenberg, David, et al. "Creatine kinase in ischemic and inflammatory disorders." Clinical and translational medicine vol. 5,1 (2016): 31. doi: https://doi.org/10.1186/s40169-016-0114-5
Kitzenberg D, Colgan SP, Glover LE. Creatine kinase in ischemic and inflammatory disorders. Clin Transl Med. 2016 Dec;5(1):31. doi: 10.1186/s40169-016-0114-5. Epub 2016 Aug 15. PMID: 27527620; PMCID: PMC4987751.
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Effects of physical activity and inactivity on muscle fatigue.

Frontiers in physiology

Bogdanis GC.
PMID: 22629249
Front Physiol. 2012 May 18;3:142. doi: 10.3389/fphys.2012.00142. eCollection 2012.

The aim of this review was to examine the mechanisms by which physical activity and inactivity modify muscle fatigue. It is well known that acute or chronic increases in physical activity result in structural, metabolic, hormonal, neural, and molecular...

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Bogdanis GC. Effects of physical activity and inactivity on muscle fatigue. Front Physiol. 2012;3:142doi: 10.3389/fphys.2012.00142.
Bogdanis, G. C. (2012). Effects of physical activity and inactivity on muscle fatigue. Frontiers in physiology, 3142. https://doi.org/10.3389/fphys.2012.00142
Bogdanis, Gregory C. "Effects of physical activity and inactivity on muscle fatigue." Frontiers in physiology vol. 3 (2012): 142. doi: https://doi.org/10.3389/fphys.2012.00142
Bogdanis GC. Effects of physical activity and inactivity on muscle fatigue. Front Physiol. 2012 May 18;3:142. doi: 10.3389/fphys.2012.00142. eCollection 2012. PMID: 22629249; PMCID: PMC3355468.
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Tumour-specific metabolic adaptation to acidosis is coupled to epigenetic stability in osteosarcoma cells.

American journal of cancer research

Chano T, Avnet S, Kusuzaki K, Bonuccelli G, Sonveaux P, Rotili D, Mai A, Baldini N.
PMID: 27186436
Am J Cancer Res. 2016 Mar 15;6(4):859-75. eCollection 2016.

The glycolytic-based metabolism of cancers promotes an acidic microenvironment that is responsible for increased aggressiveness. However, the effects of acidosis on tumour metabolism have been almost unexplored. By using capillary electrophoresis with time-of-flight mass spectrometry, we observed a significant...

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Chano T, Avnet S, Kusuzaki K, et al. Tumour-specific metabolic adaptation to acidosis is coupled to epigenetic stability in osteosarcoma cells. Am J Cancer Res. 2016;6(4):859-75
Chano, T., Avnet, S., Kusuzaki, K., Bonuccelli, G., Sonveaux, P., Rotili, D., Mai, A., & Baldini, N. (2016). Tumour-specific metabolic adaptation to acidosis is coupled to epigenetic stability in osteosarcoma cells. American journal of cancer research, 6(4), 859-75.
Chano, Tokuhiro, et al. "Tumour-specific metabolic adaptation to acidosis is coupled to epigenetic stability in osteosarcoma cells." American journal of cancer research vol. 6,4 (2016): 859-75.
Chano T, Avnet S, Kusuzaki K, Bonuccelli G, Sonveaux P, Rotili D, Mai A, Baldini N. Tumour-specific metabolic adaptation to acidosis is coupled to epigenetic stability in osteosarcoma cells. Am J Cancer Res. 2016 Mar 15;6(4):859-75. eCollection 2016. PMID: 27186436; PMCID: PMC4859889.
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Exercise inhibits tumor growth and central carbon metabolism in patient-derived xenograft models of colorectal cancer.

Cancer & metabolism

Lu M, Sanderson SM, Zessin A, Ashcraft KA, Jones LW, Dewhirst MW, Locasale JW, Hsu DS.
PMID: 30473788
Cancer Metab. 2018 Nov 15;6:14. doi: 10.1186/s40170-018-0190-7. eCollection 2018.

BACKGROUND: While self-reported exercise is associated with a reduction in the risk of recurrence in colorectal cancer, the molecular mechanisms underpinning this relationship are unknown. Furthermore, the effect of exercise on intratumoral metabolic processes has not been investigated in...

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Lu M, Sanderson SM, Zessin A, et al. Exercise inhibits tumor growth and central carbon metabolism in patient-derived xenograft models of colorectal cancer. Cancer Metab. 2018;6:14doi: 10.1186/s40170-018-0190-7.
Lu, M., Sanderson, S. M., Zessin, A., Ashcraft, K. A., Jones, L. W., Dewhirst, M. W., Locasale, J. W., & Hsu, D. S. (2018). Exercise inhibits tumor growth and central carbon metabolism in patient-derived xenograft models of colorectal cancer. Cancer & metabolism, 614. https://doi.org/10.1186/s40170-018-0190-7
Lu, Min, et al. "Exercise inhibits tumor growth and central carbon metabolism in patient-derived xenograft models of colorectal cancer." Cancer & metabolism vol. 6 (2018): 14. doi: https://doi.org/10.1186/s40170-018-0190-7
Lu M, Sanderson SM, Zessin A, Ashcraft KA, Jones LW, Dewhirst MW, Locasale JW, Hsu DS. Exercise inhibits tumor growth and central carbon metabolism in patient-derived xenograft models of colorectal cancer. Cancer Metab. 2018 Nov 15;6:14. doi: 10.1186/s40170-018-0190-7. eCollection 2018. PMID: 30473788; PMCID: PMC6237036.
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Altered Cortical GABA in Female Veterans with Suicidal Behavior: Sex Differences and Clinical Correlates.

Chronic stress (Thousand Oaks, Calif.)

Prescot A, Sheth C, Legarreta M, Renshaw PF, McGlade E, Yurgelun-Todd D.
PMID: 29756082
Chronic Stress (Thousand Oaks). 2018 Jan-Dec;2. doi: 10.1177/2470547018768771. Epub 2018 Apr 06.

BACKGROUND: Suicide is a public health concern in the civilian and veteran populations. Stressful life events are precipitating factors for suicide. The neurochemical underpinnings of the association between stress/trauma and suicide risk are unclear, especially in regards to sex...

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Prescot A, Sheth C, Legarreta M, et al. Altered Cortical GABA in Female Veterans with Suicidal Behavior: Sex Differences and Clinical Correlates. Chronic Stress (Thousand Oaks). 2018;2doi: 10.1177/2470547018768771.
Prescot, A., Sheth, C., Legarreta, M., Renshaw, P. F., McGlade, E., & Yurgelun-Todd, D. (2018). Altered Cortical GABA in Female Veterans with Suicidal Behavior: Sex Differences and Clinical Correlates. Chronic stress (Thousand Oaks, Calif.), 2. https://doi.org/10.1177/2470547018768771
Prescot, Andrew, et al. "Altered Cortical GABA in Female Veterans with Suicidal Behavior: Sex Differences and Clinical Correlates." Chronic stress (Thousand Oaks, Calif.) vol. 2 (2018). doi: https://doi.org/10.1177/2470547018768771
Prescot A, Sheth C, Legarreta M, Renshaw PF, McGlade E, Yurgelun-Todd D. Altered Cortical GABA in Female Veterans with Suicidal Behavior: Sex Differences and Clinical Correlates. Chronic Stress (Thousand Oaks). 2018 Jan-Dec;2. doi: 10.1177/2470547018768771. Epub 2018 Apr 06. PMID: 29756082; PMCID: PMC5947869.
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Psychosocial stress promotes food intake and enhances the neuroenergetic level in men.

Stress (Amsterdam, Netherlands)

Kistenmacher A, Goetsch J, Ullmann D, Wardzinski EK, Melchert UH, Jauch-Chara K, Oltmanns KM.
PMID: 29969341
Stress. 2018 Nov;21(6):538-547. doi: 10.1080/10253890.2018.1485645. Epub 2018 Jul 03.

Psychosocial stress may lead to increased food consumption and overweight. In turn, obesity is related to reduced brain energy content. We hypothesized that psychosocial stress influencing food intake may alter the neuroenergetic status in the human brain. We tested...

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Kistenmacher A, Goetsch J, Ullmann D, et al. Psychosocial stress promotes food intake and enhances the neuroenergetic level in men. Stress. 2018;21(6):538-547doi: 10.1080/10253890.2018.1485645.
Kistenmacher, A., Goetsch, J., Ullmann, D., Wardzinski, E. K., Melchert, U. H., Jauch-Chara, K., & Oltmanns, K. M. (2018). Psychosocial stress promotes food intake and enhances the neuroenergetic level in men. Stress (Amsterdam, Netherlands), 21(6), 538-547. https://doi.org/10.1080/10253890.2018.1485645
Kistenmacher, Alina, et al. "Psychosocial stress promotes food intake and enhances the neuroenergetic level in men." Stress (Amsterdam, Netherlands) vol. 21,6 (2018): 538-547. doi: https://doi.org/10.1080/10253890.2018.1485645
Kistenmacher A, Goetsch J, Ullmann D, Wardzinski EK, Melchert UH, Jauch-Chara K, Oltmanns KM. Psychosocial stress promotes food intake and enhances the neuroenergetic level in men. Stress. 2018 Nov;21(6):538-547. doi: 10.1080/10253890.2018.1485645. Epub 2018 Jul 03. PMID: 29969341.
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4-hydroxytamoxifen does not deteriorate cardiac function in cardiomyocyte-specific MerCreMer transgenic mice.

Basic research in cardiology

Heinen A, Gödecke S, Flögel U, Miklos D, Bottermann K, Spychala A, Gödecke A.
PMID: 33544211
Basic Res Cardiol. 2021 Feb 05;116(1):8. doi: 10.1007/s00395-020-00841-9.

Conditional, cell-type-specific transgenic mouse lines are of high value in cardiovascular research. A standard tool for cardiomyocyte-restricted DNA editing is the αMHC-MerCreMer/loxP system. However, there is an ongoing debate on the occurrence of cardiac side effects caused by unspecific...

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Heinen A, Gödecke S, Flögel U, et al. 4-hydroxytamoxifen does not deteriorate cardiac function in cardiomyocyte-specific MerCreMer transgenic mice. Basic Res Cardiol. 2021;116(1):8doi: 10.1007/s00395-020-00841-9.
Heinen, A., Gödecke, S., Flögel, U., Miklos, D., Bottermann, K., Spychala, A., & Gödecke, A. (2021). 4-hydroxytamoxifen does not deteriorate cardiac function in cardiomyocyte-specific MerCreMer transgenic mice. Basic research in cardiology, 116(1), 8. https://doi.org/10.1007/s00395-020-00841-9
Heinen, Andre, et al. "4-hydroxytamoxifen does not deteriorate cardiac function in cardiomyocyte-specific MerCreMer transgenic mice." Basic research in cardiology vol. 116,1 (2021): 8. doi: https://doi.org/10.1007/s00395-020-00841-9
Heinen A, Gödecke S, Flögel U, Miklos D, Bottermann K, Spychala A, Gödecke A. 4-hydroxytamoxifen does not deteriorate cardiac function in cardiomyocyte-specific MerCreMer transgenic mice. Basic Res Cardiol. 2021 Feb 05;116(1):8. doi: 10.1007/s00395-020-00841-9. PMID: 33544211; PMCID: PMC7864833.
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Showing 13 to 24 of 181 entries