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Showing 25 to 36 of 152 entries
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Involvement of the Parathyroid Hormone-Related Protein on Changes in the CYP3A Expression in Cancer Cachexia.

Molecular pharmaceutics

Fujita I, Watanabe H, Ikegami K, Imafuku T, Ichimizu S, Chikamatsu M, Kobayashi K, Tanaka R, Yamada K, Maeda H, Maruyama T.
PMID: 34734526
Mol Pharm. 2021 Dec 06;18(12):4322-4330. doi: 10.1021/acs.molpharmaceut.1c00490. Epub 2021 Nov 04.

Parathyroid hormone-related protein (PTHrP), which is secreted from a tumor, contributes to the progression of cachexia, a condition that is observed in half of all cancer patients. Although drug clearance was reported to decrease in patients with cancer cachexia,...

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Fujita I, Watanabe H, Ikegami K, et al. Involvement of the Parathyroid Hormone-Related Protein on Changes in the CYP3A Expression in Cancer Cachexia. Mol Pharm. 2021;18(12):4322-4330doi: 10.1021/acs.molpharmaceut.1c00490.
Fujita, I., Watanabe, H., Ikegami, K., Imafuku, T., Ichimizu, S., Chikamatsu, M., Kobayashi, K., Tanaka, R., Yamada, K., Maeda, H., & Maruyama, T. (2021). Involvement of the Parathyroid Hormone-Related Protein on Changes in the CYP3A Expression in Cancer Cachexia. Molecular pharmaceutics, 18(12), 4322-4330. https://doi.org/10.1021/acs.molpharmaceut.1c00490
Fujita, Issei, et al. "Involvement of the Parathyroid Hormone-Related Protein on Changes in the CYP3A Expression in Cancer Cachexia." Molecular pharmaceutics vol. 18,12 (2021): 4322-4330. doi: https://doi.org/10.1021/acs.molpharmaceut.1c00490
Fujita I, Watanabe H, Ikegami K, Imafuku T, Ichimizu S, Chikamatsu M, Kobayashi K, Tanaka R, Yamada K, Maeda H, Maruyama T. Involvement of the Parathyroid Hormone-Related Protein on Changes in the CYP3A Expression in Cancer Cachexia. Mol Pharm. 2021 Dec 06;18(12):4322-4330. doi: 10.1021/acs.molpharmaceut.1c00490. Epub 2021 Nov 04. PMID: 34734526.
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Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells.

Journal of tissue engineering

Hildreth BE, Hernon KM, Dirksen WP, Leong J, Supsavhad W, Boyaka PN, Rosol TJ, Toribio RE.
PMID: 35003616
J Tissue Eng. 2015 Oct 12;6:2041731415609298. doi: 10.1177/2041731415609298. eCollection 2015.

The N-terminus of parathyroid hormone-related protein regulates bone marrow stromal cell differentiation. We hypothesized that the nuclear localization sequence and C-terminus are involved. MicroRNA and gene expression analyses were performed on bone marrow stromal cells from mice lacking the...

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Hildreth BE, Hernon KM, Dirksen WP, et al. Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells. J Tissue Eng. 2015;6:2041731415609298doi: 10.1177/2041731415609298.
Hildreth, B. E., Hernon, K. M., Dirksen, W. P., Leong, J., Supsavhad, W., Boyaka, P. N., Rosol, T. J., & Toribio, R. E. (2015). Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells. Journal of tissue engineering, 62041731415609298. https://doi.org/10.1177/2041731415609298
Hildreth, Blake E, et al. "Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells." Journal of tissue engineering vol. 6 (2015): 2041731415609298. doi: https://doi.org/10.1177/2041731415609298
Hildreth BE, Hernon KM, Dirksen WP, Leong J, Supsavhad W, Boyaka PN, Rosol TJ, Toribio RE. Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells. J Tissue Eng. 2015 Oct 12;6:2041731415609298. doi: 10.1177/2041731415609298. eCollection 2015. PMID: 35003616; PMCID: PMC8738845.
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CDC5L promotes early chondrocyte differentiation and proliferation by modulating pre-mRNA splicing of SOX9, COL2A1, and WEE1.

The Journal of biological chemistry

Jokoji G, Maeda S, Oishi K, Ijuin T, Nakajima M, Tawaratsumida H, Kawamura I, Tominaga H, Taketomi E, Ikegawa S, Taniguchi N.
PMID: 34298017
J Biol Chem. 2021 Aug;297(2):100994. doi: 10.1016/j.jbc.2021.100994. Epub 2021 Jul 21.

Ossification of the posterior longitudinal ligament (OPLL) of the spine is a common pathological condition that causes intractable myelopathy and radiculopathy, mainly the result of an endochondral ossification-like process. Our previous genome-wide association study identified six susceptibility loci for...

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Jokoji G, Maeda S, Oishi K, et al. CDC5L promotes early chondrocyte differentiation and proliferation by modulating pre-mRNA splicing of SOX9, COL2A1, and WEE1. J Biol Chem. 2021;297(2):100994doi: 10.1016/j.jbc.2021.100994.
Jokoji, G., Maeda, S., Oishi, K., Ijuin, T., Nakajima, M., Tawaratsumida, H., Kawamura, I., Tominaga, H., Taketomi, E., Ikegawa, S., & Taniguchi, N. (2021). CDC5L promotes early chondrocyte differentiation and proliferation by modulating pre-mRNA splicing of SOX9, COL2A1, and WEE1. The Journal of biological chemistry, 297(2), 100994. https://doi.org/10.1016/j.jbc.2021.100994
Jokoji, Go, et al. "CDC5L promotes early chondrocyte differentiation and proliferation by modulating pre-mRNA splicing of SOX9, COL2A1, and WEE1." The Journal of biological chemistry vol. 297,2 (2021): 100994. doi: https://doi.org/10.1016/j.jbc.2021.100994
Jokoji G, Maeda S, Oishi K, Ijuin T, Nakajima M, Tawaratsumida H, Kawamura I, Tominaga H, Taketomi E, Ikegawa S, Taniguchi N. CDC5L promotes early chondrocyte differentiation and proliferation by modulating pre-mRNA splicing of SOX9, COL2A1, and WEE1. J Biol Chem. 2021 Aug;297(2):100994. doi: 10.1016/j.jbc.2021.100994. Epub 2021 Jul 21. PMID: 34298017; PMCID: PMC8363834.
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Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells.

Journal of tissue engineering

Hildreth BE, Hernon KM, Dirksen WP, Leong J, Supsavhad W, Boyaka PN, Rosol TJ, Toribio RE.
PMID: 35003616
J Tissue Eng. 2015 Oct 12;6:2041731415609298. doi: 10.1177/2041731415609298. eCollection 2015.

The N-terminus of parathyroid hormone-related protein regulates bone marrow stromal cell differentiation. We hypothesized that the nuclear localization sequence and C-terminus are involved. MicroRNA and gene expression analyses were performed on bone marrow stromal cells from mice lacking the...

Cite
Hildreth BE, Hernon KM, Dirksen WP, et al. Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells. J Tissue Eng. 2015;6:2041731415609298doi: 10.1177/2041731415609298.
Hildreth, B. E., Hernon, K. M., Dirksen, W. P., Leong, J., Supsavhad, W., Boyaka, P. N., Rosol, T. J., & Toribio, R. E. (2015). Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells. Journal of tissue engineering, 62041731415609298. https://doi.org/10.1177/2041731415609298
Hildreth, Blake E, et al. "Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells." Journal of tissue engineering vol. 6 (2015): 2041731415609298. doi: https://doi.org/10.1177/2041731415609298
Hildreth BE, Hernon KM, Dirksen WP, Leong J, Supsavhad W, Boyaka PN, Rosol TJ, Toribio RE. Deletion of the nuclear localization sequence and C-terminus of parathyroid hormone-related protein decreases osteogenesis and chondrogenesis but increases adipogenesis and myogenesis in murine bone marrow stromal cells. J Tissue Eng. 2015 Oct 12;6:2041731415609298. doi: 10.1177/2041731415609298. eCollection 2015. PMID: 35003616; PMCID: PMC8738845.
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Involvement of parathyroid hormone-related peptide in the aggressive phenotype of colorectal cancer cells.

World journal of gastroenterology

Novoa Díaz MB, Carriere PM, Martín MJ, Calvo N, Gentili C.
PMID: 34887626
World J Gastroenterol. 2021 Nov 07;27(41):7025-7040. doi: 10.3748/wjg.v27.i41.7025.

Colorectal cancer (CRC) remains one of the leading causes of mortality from malignant diseases worldwide. In general terms, CRC presents high heterogeneity due to the influence of different genetic and environmental factors; also, the neoplastic cells are strongly influenced...

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Novoa Díaz MB, Carriere PM, Martín MJ, et al. Involvement of parathyroid hormone-related peptide in the aggressive phenotype of colorectal cancer cells. World J Gastroenterol. 2021;27(41):7025-7040doi: 10.3748/wjg.v27.i41.7025.
Novoa Díaz, M. B., Carriere, P. M., Martín, M. J., Calvo, N., & Gentili, C. (2021). Involvement of parathyroid hormone-related peptide in the aggressive phenotype of colorectal cancer cells. World journal of gastroenterology, 27(41), 7025-7040. https://doi.org/10.3748/wjg.v27.i41.7025
Novoa Díaz, María Belén, et al. "Involvement of parathyroid hormone-related peptide in the aggressive phenotype of colorectal cancer cells." World journal of gastroenterology vol. 27,41 (2021): 7025-7040. doi: https://doi.org/10.3748/wjg.v27.i41.7025
Novoa Díaz MB, Carriere PM, Martín MJ, Calvo N, Gentili C. Involvement of parathyroid hormone-related peptide in the aggressive phenotype of colorectal cancer cells. World J Gastroenterol. 2021 Nov 07;27(41):7025-7040. doi: 10.3748/wjg.v27.i41.7025. PMID: 34887626; PMCID: PMC8613645.
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PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability.

Cancer discovery

Pitarresi JR, Norgard RJ, Chiarella AM, Suzuki K, Bakir B, Sahu V, Li J, Zhao J, Marchand B, Wengyn MD, Hsieh A, Kim IK, Zhang A, Sellin K, Lee V, Takano S, Miyahara Y, Ohtsuka M, Maitra A, Notta F, Kremer R, Stanger BZ, Rustgi AK.
PMID: 33589425
Cancer Discov. 2021 Jul;11(7):1774-1791. doi: 10.1158/2159-8290.CD-20-1098. Epub 2021 Feb 15.

Pancreatic cancer metastasis is a leading cause of cancer-related deaths, yet very little is understood regarding the underlying biology. As a result, targeted therapies to inhibit metastasis are lacking. Here, we report that the parathyroid hormone-related protein (PTHrP encoded...

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Pitarresi JR, Norgard RJ, Chiarella AM, et al. PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. Cancer Discov. 2021;11(7):1774-1791doi: 10.1158/2159-8290.CD-20-1098.
Pitarresi, J. R., Norgard, R. J., Chiarella, A. M., Suzuki, K., Bakir, B., Sahu, V., Li, J., Zhao, J., Marchand, B., Wengyn, M. D., Hsieh, A., Kim, I. K., Zhang, A., Sellin, K., Lee, V., Takano, S., Miyahara, Y., Ohtsuka, M., Maitra, A., Notta, F., Kremer, R., Stanger, B. Z., & Rustgi, A. K. (2021). PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. Cancer discovery, 11(7), 1774-1791. https://doi.org/10.1158/2159-8290.CD-20-1098
Pitarresi, Jason R, et al. "PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability." Cancer discovery vol. 11,7 (2021): 1774-1791. doi: https://doi.org/10.1158/2159-8290.CD-20-1098
Pitarresi JR, Norgard RJ, Chiarella AM, Suzuki K, Bakir B, Sahu V, Li J, Zhao J, Marchand B, Wengyn MD, Hsieh A, Kim IK, Zhang A, Sellin K, Lee V, Takano S, Miyahara Y, Ohtsuka M, Maitra A, Notta F, Kremer R, Stanger BZ, Rustgi AK. PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. Cancer Discov. 2021 Jul;11(7):1774-1791. doi: 10.1158/2159-8290.CD-20-1098. Epub 2021 Feb 15. PMID: 33589425; PMCID: PMC8292165.
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Roles of Parathyroid Hormone-Related Protein (PTHrP) and Its Receptor (PTHR1) in Normal and Tumor Tissues: Focus on Their Roles in Osteosarcoma.

Frontiers in veterinary science

Al-Khan AA, Al Balushi NR, Richardson SJ, Danks JA.
PMID: 33796580
Front Vet Sci. 2021 Mar 16;8:637614. doi: 10.3389/fvets.2021.637614. eCollection 2021.

Osteosarcoma (OS) is the most common primary bone tumor and originates from bone forming mesenchymal cells and primarily affects children and adolescents. The 5-year survival rate for OS is 60 to 65%, with little improvement in prognosis during the...

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Al-Khan AA, Al Balushi NR, Richardson SJ, et al. Roles of Parathyroid Hormone-Related Protein (PTHrP) and Its Receptor (PTHR1) in Normal and Tumor Tissues: Focus on Their Roles in Osteosarcoma. Front Vet Sci. 2021;8:637614doi: 10.3389/fvets.2021.637614.
Al-Khan, A. A., Al Balushi, N. R., Richardson, S. J., & Danks, J. A. (2021). Roles of Parathyroid Hormone-Related Protein (PTHrP) and Its Receptor (PTHR1) in Normal and Tumor Tissues: Focus on Their Roles in Osteosarcoma. Frontiers in veterinary science, 8637614. https://doi.org/10.3389/fvets.2021.637614
Al-Khan, Awf A, et al. "Roles of Parathyroid Hormone-Related Protein (PTHrP) and Its Receptor (PTHR1) in Normal and Tumor Tissues: Focus on Their Roles in Osteosarcoma." Frontiers in veterinary science vol. 8 (2021): 637614. doi: https://doi.org/10.3389/fvets.2021.637614
Al-Khan AA, Al Balushi NR, Richardson SJ, Danks JA. Roles of Parathyroid Hormone-Related Protein (PTHrP) and Its Receptor (PTHR1) in Normal and Tumor Tissues: Focus on Their Roles in Osteosarcoma. Front Vet Sci. 2021 Mar 16;8:637614. doi: 10.3389/fvets.2021.637614. eCollection 2021. PMID: 33796580; PMCID: PMC8008073.
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Metabolic Bone Disease in the Context of Metastatic Neuroendocrine Tumor: Differentiation from Skeletal Metastasis, the Molecular PET-CT Imaging Features, and Exploring the Possible Etiopathologies Including Parathyroid Adenoma (MEN1) and Paraneoplastic Humoral Hypercalcemia of Malignancy Due to PTHrP Hypersecretion.

World journal of nuclear medicine

Ranade R, Basu S.
PMID: 28217023
World J Nucl Med. 2017 Jan-Mar;16(1):62-67. doi: 10.4103/1450-1147.172307.

Three cases of metabolic bone disease in the setting of metastatic neuroendocrine tumor (NET) are illustrated with associated etiopathologies.  One of these cases harbored mixed lesions in the form of vertebral metastasis (biopsy proven) while the other skeletal lesions...

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Ranade R, Basu S. Metabolic Bone Disease in the Context of Metastatic Neuroendocrine Tumor: Differentiation from Skeletal Metastasis, the Molecular PET-CT Imaging Features, and Exploring the Possible Etiopathologies Including Parathyroid Adenoma (MEN1) and Paraneoplastic Humoral Hypercalcemia of Malignancy Due to PTHrP Hypersecretion. World J Nucl Med. 2017;16(1):62-67doi: 10.4103/1450-1147.172307.
Ranade, R., & Basu, S. (2017). Metabolic Bone Disease in the Context of Metastatic Neuroendocrine Tumor: Differentiation from Skeletal Metastasis, the Molecular PET-CT Imaging Features, and Exploring the Possible Etiopathologies Including Parathyroid Adenoma (MEN1) and Paraneoplastic Humoral Hypercalcemia of Malignancy Due to PTHrP Hypersecretion. World journal of nuclear medicine, 16(1), 62-67. https://doi.org/10.4103/1450-1147.172307
Ranade, Rohit, and Basu, Sandip. "Metabolic Bone Disease in the Context of Metastatic Neuroendocrine Tumor: Differentiation from Skeletal Metastasis, the Molecular PET-CT Imaging Features, and Exploring the Possible Etiopathologies Including Parathyroid Adenoma (MEN1) and Paraneoplastic Humoral Hypercalcemia of Malignancy Due to PTHrP Hypersecretion." World journal of nuclear medicine vol. 16,1 (2017): 62-67. doi: https://doi.org/10.4103/1450-1147.172307
Ranade R, Basu S. Metabolic Bone Disease in the Context of Metastatic Neuroendocrine Tumor: Differentiation from Skeletal Metastasis, the Molecular PET-CT Imaging Features, and Exploring the Possible Etiopathologies Including Parathyroid Adenoma (MEN1) and Paraneoplastic Humoral Hypercalcemia of Malignancy Due to PTHrP Hypersecretion. World J Nucl Med. 2017 Jan-Mar;16(1):62-67. doi: 10.4103/1450-1147.172307. PMID: 28217023; PMCID: PMC5314667.
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Hedgehog proteins and parathyroid hormone-related protein are involved in intervertebral disc maturation, degeneration, and calcification.

JOR spine

Bach FC, de Rooij KM, Riemers FM, Snuggs JW, de Jong WAM, Zhang Y, Creemers LB, Chan D, Le Maitre C, Tryfonidou MA.
PMID: 31891120
JOR Spine. 2019 Nov 19;2(4):e1071. doi: 10.1002/jsp2.1071. eCollection 2019 Dec.

Parathyroid hormone-related protein (PTHrP) and hedgehog signaling play an important role in chondrocyte development, (hypertrophic) differentiation, and/or calcification, but their role in intervertebral disc (IVD) degeneration is unknown. Better understanding their involvement may provide therapeutic clues for low back...

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Bach FC, de Rooij KM, Riemers FM, et al. Hedgehog proteins and parathyroid hormone-related protein are involved in intervertebral disc maturation, degeneration, and calcification. JOR Spine. 2019;2(4):e1071doi: 10.1002/jsp2.1071.
Bach, F. C., de Rooij, K. M., Riemers, F. M., Snuggs, J. W., de Jong, W. A. M., Zhang, Y., Creemers, L. B., Chan, D., Le Maitre, C., & Tryfonidou, M. A. (2019). Hedgehog proteins and parathyroid hormone-related protein are involved in intervertebral disc maturation, degeneration, and calcification. JOR spine, 2(4), e1071. https://doi.org/10.1002/jsp2.1071
Bach, Frances C, et al. "Hedgehog proteins and parathyroid hormone-related protein are involved in intervertebral disc maturation, degeneration, and calcification." JOR spine vol. 2,4 (2019): e1071. doi: https://doi.org/10.1002/jsp2.1071
Bach FC, de Rooij KM, Riemers FM, Snuggs JW, de Jong WAM, Zhang Y, Creemers LB, Chan D, Le Maitre C, Tryfonidou MA. Hedgehog proteins and parathyroid hormone-related protein are involved in intervertebral disc maturation, degeneration, and calcification. JOR Spine. 2019 Nov 19;2(4):e1071. doi: 10.1002/jsp2.1071. eCollection 2019 Dec. PMID: 31891120; PMCID: PMC6920702.
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Influence of magnesium and parathyroid hormone on cisplatin-induced nephrotoxicity in esophageal squamous cell carcinoma.

Oncology letters

Konishi H, Fujiwara H, Itoh H, Shiozaki A, Arita T, Kosuga T, Morimura R, Komatsu S, Ichikawa D, Okamoto K, Otsuji E.
PMID: 29391892
Oncol Lett. 2018 Jan;15(1):658-664. doi: 10.3892/ol.2017.7345. Epub 2017 Nov 03.

Magnesium (Mg) supplementation has previously been demonstrated to confer protective effects against nephrotoxicity induced by cisplatin. Parathyroid hormone (PTH) regulates Mg homeostasis. The aim of present study was to determine the protective effects of Mg supplementation against cisplatin-induced nephrotoxicity...

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Konishi H, Fujiwara H, Itoh H, et al. Influence of magnesium and parathyroid hormone on cisplatin-induced nephrotoxicity in esophageal squamous cell carcinoma. Oncol Lett. 2017;15(1):658-664doi: 10.3892/ol.2017.7345.
Konishi, H., Fujiwara, H., Itoh, H., Shiozaki, A., Arita, T., Kosuga, T., Morimura, R., Komatsu, S., Ichikawa, D., Okamoto, K., & Otsuji, E. (2018). Influence of magnesium and parathyroid hormone on cisplatin-induced nephrotoxicity in esophageal squamous cell carcinoma. Oncology letters, 15(1), 658-664. https://doi.org/10.3892/ol.2017.7345
Konishi, Hirotaka, et al. "Influence of magnesium and parathyroid hormone on cisplatin-induced nephrotoxicity in esophageal squamous cell carcinoma." Oncology letters vol. 15,1 (2018): 658-664. doi: https://doi.org/10.3892/ol.2017.7345
Konishi H, Fujiwara H, Itoh H, Shiozaki A, Arita T, Kosuga T, Morimura R, Komatsu S, Ichikawa D, Okamoto K, Otsuji E. Influence of magnesium and parathyroid hormone on cisplatin-induced nephrotoxicity in esophageal squamous cell carcinoma. Oncol Lett. 2018 Jan;15(1):658-664. doi: 10.3892/ol.2017.7345. Epub 2017 Nov 03. PMID: 29391892; PMCID: PMC5769377.
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Parathyroid hormone-related protein exhibits antioxidant features in osteoblastic cells through its N-terminal and osteostatin domains.

Bone & joint research

Portal-Núñez S, Ardura JA, Lozano D, Martínez de Toda I, De la Fuente M, Herrero-Beaumont G, Largo R, Esbrit P.
PMID: 29330344
Bone Joint Res. 2018 Jan;7(1):58-68. doi: 10.1302/2046-3758.71.BJR-2016-0242.R2.

OBJECTIVES: Oxidative stress plays a major role in the onset and progression of involutional osteoporosis. However, classical antioxidants fail to restore osteoblast function. Interestingly, the bone anabolism of parathyroid hormone (PTH) has been shown to be associated with its...

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Portal-Núñez S, Ardura JA, Lozano D, et al. Parathyroid hormone-related protein exhibits antioxidant features in osteoblastic cells through its N-terminal and osteostatin domains. Bone Joint Res. 2018;7(1):58-68doi: 10.1302/2046-3758.71.BJR-2016-0242.R2.
Portal-Núñez, S., Ardura, J. A., Lozano, D., Martínez de Toda, I., De la Fuente, M., Herrero-Beaumont, G., Largo, R., & Esbrit, P. (2018). Parathyroid hormone-related protein exhibits antioxidant features in osteoblastic cells through its N-terminal and osteostatin domains. Bone & joint research, 7(1), 58-68. https://doi.org/10.1302/2046-3758.71.BJR-2016-0242.R2
Portal-Núñez, S, et al. "Parathyroid hormone-related protein exhibits antioxidant features in osteoblastic cells through its N-terminal and osteostatin domains." Bone & joint research vol. 7,1 (2018): 58-68. doi: https://doi.org/10.1302/2046-3758.71.BJR-2016-0242.R2
Portal-Núñez S, Ardura JA, Lozano D, Martínez de Toda I, De la Fuente M, Herrero-Beaumont G, Largo R, Esbrit P. Parathyroid hormone-related protein exhibits antioxidant features in osteoblastic cells through its N-terminal and osteostatin domains. Bone Joint Res. 2018 Jan;7(1):58-68. doi: 10.1302/2046-3758.71.BJR-2016-0242.R2. PMID: 29330344; PMCID: PMC5805825.
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Pancreatic Adenocarcinoma Producing Parathyroid Hormone-Related Protein.

Case reports in oncological medicine

Yamada R, Tanaka K, Inoue H, Sakuno T, Harada T, Yoshizawa N, Miura H, Takeuchi T, Nakamura M, Katsurahara M, Hamada Y, Horiki N, Takei Y.
PMID: 28894614
Case Rep Oncol Med. 2017;2017:5656130. doi: 10.1155/2017/5656130. Epub 2017 Aug 15.

A 48-year-old woman presented to our hospital with a 1-year history of a continuous high fever. She was diagnosed with metastatic pancreatic adenocarcinoma accompanied by leukocytosis without infection. Her serum concentration of granulocyte colony-stimulating factor was highly elevated. Forty-five...

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Yamada R, Tanaka K, Inoue H, et al. Pancreatic Adenocarcinoma Producing Parathyroid Hormone-Related Protein. Case Rep Oncol Med. 2017;2017:5656130doi: 10.1155/2017/5656130.
Yamada, R., Tanaka, K., Inoue, H., Sakuno, T., Harada, T., Yoshizawa, N., Miura, H., Takeuchi, T., Nakamura, M., Katsurahara, M., Hamada, Y., Horiki, N., & Takei, Y. (2017). Pancreatic Adenocarcinoma Producing Parathyroid Hormone-Related Protein. Case reports in oncological medicine, 20175656130. https://doi.org/10.1155/2017/5656130
Yamada, Reiko, et al. "Pancreatic Adenocarcinoma Producing Parathyroid Hormone-Related Protein." Case reports in oncological medicine vol. 2017 (2017): 5656130. doi: https://doi.org/10.1155/2017/5656130
Yamada R, Tanaka K, Inoue H, Sakuno T, Harada T, Yoshizawa N, Miura H, Takeuchi T, Nakamura M, Katsurahara M, Hamada Y, Horiki N, Takei Y. Pancreatic Adenocarcinoma Producing Parathyroid Hormone-Related Protein. Case Rep Oncol Med. 2017;2017:5656130. doi: 10.1155/2017/5656130. Epub 2017 Aug 15. PMID: 28894614; PMCID: PMC5574221.
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