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Osteogenic Effect of ZnO-Mesoporous Glasses Loaded with Osteostatin.

Nanomaterials (Basel, Switzerland)

Pérez R, Sanchez-Salcedo S, Lozano D, Heras C, Esbrit P, Vallet-Regí M, Salinas AJ.
PMID: 30081542
Nanomaterials (Basel). 2018 Aug 04;8(8). doi: 10.3390/nano8080592.

Mesoporous Bioactive Glasses (MBGs) are a family of bioceramics widely investigated for their putative clinical use as scaffolds for bone regeneration. Their outstanding textural properties allow for high bioactivity when compared with other bioactive materials. Moreover, their great pore...

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Pérez R, Sanchez-Salcedo S, Lozano D, et al. Osteogenic Effect of ZnO-Mesoporous Glasses Loaded with Osteostatin. Nanomaterials (Basel). 2018;8(8)doi: 10.3390/nano8080592.
Pérez, R., Sanchez-Salcedo, S., Lozano, D., Heras, C., Esbrit, P., Vallet-Regí, M., & Salinas, A. J. (2018). Osteogenic Effect of ZnO-Mesoporous Glasses Loaded with Osteostatin. Nanomaterials (Basel, Switzerland), 8(8), . https://doi.org/10.3390/nano8080592
Pérez, Rebeca, et al. "Osteogenic Effect of ZnO-Mesoporous Glasses Loaded with Osteostatin." Nanomaterials (Basel, Switzerland) vol. 8,8 (2018). doi: https://doi.org/10.3390/nano8080592
Pérez R, Sanchez-Salcedo S, Lozano D, Heras C, Esbrit P, Vallet-Regí M, Salinas AJ. Osteogenic Effect of ZnO-Mesoporous Glasses Loaded with Osteostatin. Nanomaterials (Basel). 2018 Aug 04;8(8). doi: 10.3390/nano8080592. PMID: 30081542; PMCID: PMC6116259.
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Single-Cell Transcriptomic Analysis Reveals Developmental Relationships and Specific Markers of Mouse Periodontium Cellular Subsets.

Frontiers in dental medicine

Nagata M, Chu AKY, Ono N, Welch JD, Ono W.
PMID: 34966906
Front Dent Med. 2021 Aug;2. doi: 10.3389/fdmed.2021.679937. Epub 2021 Aug 12.

The periodontium is essential for supporting the functionality of the tooth, composed of diversity of mineralized and non-mineralized tissues such as the cementum, the periodontal ligament (PDL) and the alveolar bone. The periodontium is developmentally derived from the dental...

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Nagata M, Chu AKY, Ono N, et al. Single-Cell Transcriptomic Analysis Reveals Developmental Relationships and Specific Markers of Mouse Periodontium Cellular Subsets. Front Dent Med. 2021;2doi: 10.3389/fdmed.2021.679937.
Nagata, M., Chu, A. K. Y., Ono, N., Welch, J. D., & Ono, W. (2021). Single-Cell Transcriptomic Analysis Reveals Developmental Relationships and Specific Markers of Mouse Periodontium Cellular Subsets. Frontiers in dental medicine, 2. https://doi.org/10.3389/fdmed.2021.679937
Nagata, Mizuki, et al. "Single-Cell Transcriptomic Analysis Reveals Developmental Relationships and Specific Markers of Mouse Periodontium Cellular Subsets." Frontiers in dental medicine vol. 2 (2021). doi: https://doi.org/10.3389/fdmed.2021.679937
Nagata M, Chu AKY, Ono N, Welch JD, Ono W. Single-Cell Transcriptomic Analysis Reveals Developmental Relationships and Specific Markers of Mouse Periodontium Cellular Subsets. Front Dent Med. 2021 Aug;2. doi: 10.3389/fdmed.2021.679937. Epub 2021 Aug 12. PMID: 34966906; PMCID: PMC8713353.
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Hypercalcemia of Malignancy Attributed to Cosecretion of PTH and PTHRP in Lung Adenocarcinoma.

AACE clinical case reports

Kroopnick JM, Martinez-Outschoorn U, Tuluc M, Kim CS.
PMID: 34095488
AACE Clin Case Rep. 2021 Jan 19;7(3):200-203. doi: 10.1016/j.aace.2021.01.003. eCollection 2021.

INTRODUCTION: Hypercalcemia of malignancy (HCM) portends a very poor prognosis, and no established guidelines exist regarding its management. Most instances of HCM are due to local osteolysis or secretion of parathyroid hormone related-peptide, while less than 1% of all...

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Kroopnick JM, Martinez-Outschoorn U, Tuluc M, et al. Hypercalcemia of Malignancy Attributed to Cosecretion of PTH and PTHRP in Lung Adenocarcinoma. AACE Clin Case Rep. 2021;7(3):200-203doi: 10.1016/j.aace.2021.01.003.
Kroopnick, J. M., Martinez-Outschoorn, U., Tuluc, M., & Kim, C. S. (2021). Hypercalcemia of Malignancy Attributed to Cosecretion of PTH and PTHRP in Lung Adenocarcinoma. AACE clinical case reports, 7(3), 200-203. https://doi.org/10.1016/j.aace.2021.01.003
Kroopnick, Jeffrey M, et al. "Hypercalcemia of Malignancy Attributed to Cosecretion of PTH and PTHRP in Lung Adenocarcinoma." AACE clinical case reports vol. 7,3 (2021): 200-203. doi: https://doi.org/10.1016/j.aace.2021.01.003
Kroopnick JM, Martinez-Outschoorn U, Tuluc M, Kim CS. Hypercalcemia of Malignancy Attributed to Cosecretion of PTH and PTHRP in Lung Adenocarcinoma. AACE Clin Case Rep. 2021 Jan 19;7(3):200-203. doi: 10.1016/j.aace.2021.01.003. eCollection 2021. PMID: 34095488; PMCID: PMC8165115.
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The hypertrophic chondrocyte: To be or not to be.

Histology and histopathology

Hallett SA, Ono W, Ono N.
PMID: 34137454
Histol Histopathol. 2021 Oct;36(10):1021-1036. doi: 10.14670/HH-18-355. Epub 2021 Jun 17.

Hypertrophic chondrocytes are the master regulators of endochondral ossification; however, their ultimate cell fates cells remain largely elusive due to their transient nature. Historically, hypertrophic chondrocytes have been considered as the terminal state of growth plate chondrocytes, which are...

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Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021;36(10):1021-1036doi: 10.14670/HH-18-355.
Hallett, S. A., Ono, W., & Ono, N. (2021). The hypertrophic chondrocyte: To be or not to be. Histology and histopathology, 36(10), 1021-1036. https://doi.org/10.14670/HH-18-355
Hallett, Shawn A, et al. "The hypertrophic chondrocyte: To be or not to be." Histology and histopathology vol. 36,10 (2021): 1021-1036. doi: https://doi.org/10.14670/HH-18-355
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021 Oct;36(10):1021-1036. doi: 10.14670/HH-18-355. Epub 2021 Jun 17. PMID: 34137454; PMCID: PMC8678381.
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Case Report: Unmasking Hypercalcemia in Patients With Neuroendocrine Neoplasms. Experience From Six Italian Referral Centers.

Frontiers in endocrinology

Giannetta E, Sesti F, Modica R, Grossrubatscher EM, Guarnotta V, Ragni A, Zanata I, Colao A, Faggiano A.
PMID: 34093441
Front Endocrinol (Lausanne). 2021 May 19;12:665698. doi: 10.3389/fendo.2021.665698. eCollection 2021.

BACKGROUND: Hypercalcemia is a common paraneoplastic syndrome which can occur in up to 10% of patients with advanced neoplasms. Paraneoplastic parathyroid hormone-related protein (PTHrP) represents the most frequent cause of this syndrome. In neuroendocrine neoplasms (NENs) paraneoplastic hypercalcemia is...

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Giannetta E, Sesti F, Modica R, et al. Case Report: Unmasking Hypercalcemia in Patients With Neuroendocrine Neoplasms. Experience From Six Italian Referral Centers. Front Endocrinol (Lausanne). 2021;12:665698doi: 10.3389/fendo.2021.665698.
Giannetta, E., Sesti, F., Modica, R., Grossrubatscher, E. M., Guarnotta, V., Ragni, A., Zanata, I., Colao, A., & Faggiano, A. (2021). Case Report: Unmasking Hypercalcemia in Patients With Neuroendocrine Neoplasms. Experience From Six Italian Referral Centers. Frontiers in endocrinology, 12665698. https://doi.org/10.3389/fendo.2021.665698
Giannetta, Elisa, et al. "Case Report: Unmasking Hypercalcemia in Patients With Neuroendocrine Neoplasms. Experience From Six Italian Referral Centers." Frontiers in endocrinology vol. 12 (2021): 665698. doi: https://doi.org/10.3389/fendo.2021.665698
Giannetta E, Sesti F, Modica R, Grossrubatscher EM, Guarnotta V, Ragni A, Zanata I, Colao A, Faggiano A. Case Report: Unmasking Hypercalcemia in Patients With Neuroendocrine Neoplasms. Experience From Six Italian Referral Centers. Front Endocrinol (Lausanne). 2021 May 19;12:665698. doi: 10.3389/fendo.2021.665698. eCollection 2021. PMID: 34093441; PMCID: PMC8170398.
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Can the calcium-regulating hormones counteract the detrimental impact of pro-inflammatory damage-associated molecular patterns in the development of heart failure?.

Journal of investigative medicine : the official publication of the American Federation for Clinical Research

Adamyan SH, Harutyunyan KR, Abrahamyan HT, Khudaverdyan DN, Mkrtchian S, Ter-Markosyan AS.
PMID: 33952612
J Investig Med. 2021 Aug;69(6):1148-1152. doi: 10.1136/jim-2020-001754. Epub 2021 May 05.

Growing evidence suggests an important role of the inflammatory component in heart failure (HF). Recent developments in this field indicate an ambiguous role that innate immunity plays in immune-driven HF. Damaged or stressed cells, cardiomyocytes, in particular, emit damage-associated...

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Adamyan SH, Harutyunyan KR, Abrahamyan HT, et al. Can the calcium-regulating hormones counteract the detrimental impact of pro-inflammatory damage-associated molecular patterns in the development of heart failure?. J Investig Med. 2021;69(6):1148-1152doi: 10.1136/jim-2020-001754.
Adamyan, S. H., Harutyunyan, K. R., Abrahamyan, H. T., Khudaverdyan, D. N., Mkrtchian, S., & Ter-Markosyan, A. S. (2021). Can the calcium-regulating hormones counteract the detrimental impact of pro-inflammatory damage-associated molecular patterns in the development of heart failure?. Journal of investigative medicine : the official publication of the American Federation for Clinical Research, 69(6), 1148-1152. https://doi.org/10.1136/jim-2020-001754
Adamyan, Satenik H, et al. "Can the calcium-regulating hormones counteract the detrimental impact of pro-inflammatory damage-associated molecular patterns in the development of heart failure?." Journal of investigative medicine : the official publication of the American Federation for Clinical Research vol. 69,6 (2021): 1148-1152. doi: https://doi.org/10.1136/jim-2020-001754
Adamyan SH, Harutyunyan KR, Abrahamyan HT, Khudaverdyan DN, Mkrtchian S, Ter-Markosyan AS. Can the calcium-regulating hormones counteract the detrimental impact of pro-inflammatory damage-associated molecular patterns in the development of heart failure?. J Investig Med. 2021 Aug;69(6):1148-1152. doi: 10.1136/jim-2020-001754. Epub 2021 May 05. PMID: 33952612; PMCID: PMC8327405.
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HDAC inhibitors stimulate LIFR when it is repressed by hypoxia or PTHrP in breast cancer.

Journal of bone oncology

Edwards CM, Clements ME, Vecchi LA, Johnson JA, Johnson RW.
PMID: 34934614
J Bone Oncol. 2021 Dec 01;31:100407. doi: 10.1016/j.jbo.2021.100407. eCollection 2021 Dec.

Breast cancer cells frequently disseminate to the bone marrow, where they either induce osteolysis or enter a dormant state. Downregulation of leukemia inhibitory factor receptor (LIFR), a known breast tumor suppressor, enables otherwise dormant MCF7 human breast cancer cells...

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Edwards CM, Clements ME, Vecchi LA, et al. HDAC inhibitors stimulate LIFR when it is repressed by hypoxia or PTHrP in breast cancer. J Bone Oncol. 2021;31:100407doi: 10.1016/j.jbo.2021.100407.
Edwards, C. M., Clements, M. E., Vecchi, L. A., Johnson, J. A., & Johnson, R. W. (2021). HDAC inhibitors stimulate LIFR when it is repressed by hypoxia or PTHrP in breast cancer. Journal of bone oncology, 31100407. https://doi.org/10.1016/j.jbo.2021.100407
Edwards, Courtney M, et al. "HDAC inhibitors stimulate LIFR when it is repressed by hypoxia or PTHrP in breast cancer." Journal of bone oncology vol. 31 (2021): 100407. doi: https://doi.org/10.1016/j.jbo.2021.100407
Edwards CM, Clements ME, Vecchi LA, Johnson JA, Johnson RW. HDAC inhibitors stimulate LIFR when it is repressed by hypoxia or PTHrP in breast cancer. J Bone Oncol. 2021 Dec 01;31:100407. doi: 10.1016/j.jbo.2021.100407. eCollection 2021 Dec. PMID: 34934614; PMCID: PMC8661052.
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The hypertrophic chondrocyte: To be or not to be.

Histology and histopathology

Hallett SA, Ono W, Ono N.
PMID: 34137454
Histol Histopathol. 2021 Jun 17;18355. doi: 10.14670/HH-18-355. Epub 2021 Jun 17.

Hypertrophic chondrocytes are the master regulators of endochondral ossification; however, their ultimate cell fates cells remain largely elusive due to their transient nature. Historically, hypertrophic chondrocytes have been considered as the terminal state of growth plate chondrocytes, which are...

Cite
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021;18355doi: 10.14670/HH-18-355.
Hallett, S. A., Ono, W., & Ono, N. (2021). The hypertrophic chondrocyte: To be or not to be. Histology and histopathology, 18355. https://doi.org/10.14670/HH-18-355
Hallett, Shawn A, et al. "The hypertrophic chondrocyte: To be or not to be." Histology and histopathology vol. (2021): 18355. doi: https://doi.org/10.14670/HH-18-355
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021 Jun 17;18355. doi: 10.14670/HH-18-355. Epub 2021 Jun 17. PMID: 34137454.
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Severe Hypercalcemia as the Initial Presentation of a Neuroendocrine Carcinoma of Unknown Primary Site: A Case Report.

Cureus

Shaka H, Raghavan S.
PMID: 31832292
Cureus. 2019 Nov 05;11(11):e6075. doi: 10.7759/cureus.6075.

Hypercalcemia is a common paraneoplastic syndrome that is associated with many malignancies. Hypercalcemia develops in malignancies through various mechanisms. Parathyroid hormone-related protein (PTHrP) is secreted by malignancy involving the lungs, breast, colorectum, bladder, and, rarely, neuroendocrine tumors. This is...

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Shaka H, Raghavan S. Severe Hypercalcemia as the Initial Presentation of a Neuroendocrine Carcinoma of Unknown Primary Site: A Case Report. Cureus. 2019;11(11):e6075doi: 10.7759/cureus.6075.
Shaka, H., & Raghavan, S. (2019). Severe Hypercalcemia as the Initial Presentation of a Neuroendocrine Carcinoma of Unknown Primary Site: A Case Report. Cureus, 11(11), e6075. https://doi.org/10.7759/cureus.6075
Shaka, Hafeez, and Raghavan, Sairam. "Severe Hypercalcemia as the Initial Presentation of a Neuroendocrine Carcinoma of Unknown Primary Site: A Case Report." Cureus vol. 11,11 (2019): e6075. doi: https://doi.org/10.7759/cureus.6075
Shaka H, Raghavan S. Severe Hypercalcemia as the Initial Presentation of a Neuroendocrine Carcinoma of Unknown Primary Site: A Case Report. Cureus. 2019 Nov 05;11(11):e6075. doi: 10.7759/cureus.6075. PMID: 31832292; PMCID: PMC6892572.
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Calcium Metabolism and Breast Cancer: Echoes of Lactation?.

Current opinion in endocrine and metabolic research

Grinman D, Athonvarungkul D, Wysolmerski J, Jeong J.
PMID: 33299957
Curr Opin Endocr Metab Res. 2020 Dec;15:63-70. doi: 10.1016/j.coemr.2020.11.006. Epub 2020 Nov 21.

Lactation requires a series of adaptations in maternal calcium and bone metabolism to ensure a steady supply of calcium to the lactating mammary gland. The alterations in systemic metabolism are accompanied by alterations in the expression of calcium receptors,...

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Grinman D, Athonvarungkul D, Wysolmerski J, et al. Calcium Metabolism and Breast Cancer: Echoes of Lactation?. Curr Opin Endocr Metab Res. 2020;15:63-70doi: 10.1016/j.coemr.2020.11.006.
Grinman, D., Athonvarungkul, D., Wysolmerski, J., & Jeong, J. (2020). Calcium Metabolism and Breast Cancer: Echoes of Lactation?. Current opinion in endocrine and metabolic research, 1563-70. https://doi.org/10.1016/j.coemr.2020.11.006
Grinman, Diego, et al. "Calcium Metabolism and Breast Cancer: Echoes of Lactation?." Current opinion in endocrine and metabolic research vol. 15 (2020): 63-70. doi: https://doi.org/10.1016/j.coemr.2020.11.006
Grinman D, Athonvarungkul D, Wysolmerski J, Jeong J. Calcium Metabolism and Breast Cancer: Echoes of Lactation?. Curr Opin Endocr Metab Res. 2020 Dec;15:63-70. doi: 10.1016/j.coemr.2020.11.006. Epub 2020 Nov 21. PMID: 33299957; PMCID: PMC7720883.
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The hypertrophic chondrocyte: To be or not to be.

Histology and histopathology

Hallett SA, Ono W, Ono N.
PMID: 34137454
Histol Histopathol. 2021 Jun 17;18355. doi: 10.14670/HH-18-355. Epub 2021 Jun 17.

Hypertrophic chondrocytes are the master regulators of endochondral ossification; however, their ultimate cell fates cells remain largely elusive due to their transient nature. Historically, hypertrophic chondrocytes have been considered as the terminal state of growth plate chondrocytes, which are...

Cite
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021;18355doi: 10.14670/HH-18-355.
Hallett, S. A., Ono, W., & Ono, N. (2021). The hypertrophic chondrocyte: To be or not to be. Histology and histopathology, 18355. https://doi.org/10.14670/HH-18-355
Hallett, Shawn A, et al. "The hypertrophic chondrocyte: To be or not to be." Histology and histopathology vol. (2021): 18355. doi: https://doi.org/10.14670/HH-18-355
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021 Jun 17;18355. doi: 10.14670/HH-18-355. Epub 2021 Jun 17. PMID: 34137454; PMCID: PMC8678381.
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From Good to Bad: The Opposing Effects of PTHrP on Tumor Growth, Dormancy, and Metastasis Throughout Cancer Progression.

Frontiers in oncology

Edwards CM, Johnson RW.
PMID: 33828987
Front Oncol. 2021 Mar 22;11:644303. doi: 10.3389/fonc.2021.644303. eCollection 2021.

Parathyroid hormone related protein (PTHrP) is a multifaceted protein with several biologically active domains that regulate its many roles in normal physiology and human disease. PTHrP causes humoral hypercalcemia of malignancy (HHM) through its endocrine actions and tumor-induced bone...

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Edwards CM, Johnson RW. From Good to Bad: The Opposing Effects of PTHrP on Tumor Growth, Dormancy, and Metastasis Throughout Cancer Progression. Front Oncol. 2021;11:644303doi: 10.3389/fonc.2021.644303.
Edwards, C. M., & Johnson, R. W. (2021). From Good to Bad: The Opposing Effects of PTHrP on Tumor Growth, Dormancy, and Metastasis Throughout Cancer Progression. Frontiers in oncology, 11644303. https://doi.org/10.3389/fonc.2021.644303
Edwards, Courtney M, and Johnson, Rachelle W. "From Good to Bad: The Opposing Effects of PTHrP on Tumor Growth, Dormancy, and Metastasis Throughout Cancer Progression." Frontiers in oncology vol. 11 (2021): 644303. doi: https://doi.org/10.3389/fonc.2021.644303
Edwards CM, Johnson RW. From Good to Bad: The Opposing Effects of PTHrP on Tumor Growth, Dormancy, and Metastasis Throughout Cancer Progression. Front Oncol. 2021 Mar 22;11:644303. doi: 10.3389/fonc.2021.644303. eCollection 2021. PMID: 33828987; PMCID: PMC8019909.
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Showing 13 to 24 of 152 entries