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Showing 145 to 152 of 152 entries
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A Case of Ovarian Clear Cell Carcinoma Simultaneously Producing Parathyroid Hormone-related Protein and Granulocyte Colony-Stimulating Factor.

World journal of oncology

Futagami M, Yokoyama Y, Wakui M, Taniguchi R, Higuchi T, Mizunuma H.
PMID: 29147194
World J Oncol. 2010 Jun;1(3):138-141. doi: 10.4021/wjon2010.06.214w. Epub 2010 May 19.

We describe the first report of an ovarian clear cell carcinoma simultaneously producing parathyroid hormone-related protein (PTHrP) and granulocyte colony-stimulating factor (G-CSF). A 64-year-old woman complained of general fatigue, loss of appetite, nausea, vomiting and constipation. The results of...

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Futagami M, Yokoyama Y, Wakui M, et al. A Case of Ovarian Clear Cell Carcinoma Simultaneously Producing Parathyroid Hormone-related Protein and Granulocyte Colony-Stimulating Factor. World J Oncol. 2010;1(3):138-141doi: 10.4021/wjon2010.06.214w.
Futagami, M., Yokoyama, Y., Wakui, M., Taniguchi, R., Higuchi, T., & Mizunuma, H. (2010). A Case of Ovarian Clear Cell Carcinoma Simultaneously Producing Parathyroid Hormone-related Protein and Granulocyte Colony-Stimulating Factor. World journal of oncology, 1(3), 138-141. https://doi.org/10.4021/wjon2010.06.214w
Futagami, Masayuki, et al. "A Case of Ovarian Clear Cell Carcinoma Simultaneously Producing Parathyroid Hormone-related Protein and Granulocyte Colony-Stimulating Factor." World journal of oncology vol. 1,3 (2010): 138-141. doi: https://doi.org/10.4021/wjon2010.06.214w
Futagami M, Yokoyama Y, Wakui M, Taniguchi R, Higuchi T, Mizunuma H. A Case of Ovarian Clear Cell Carcinoma Simultaneously Producing Parathyroid Hormone-related Protein and Granulocyte Colony-Stimulating Factor. World J Oncol. 2010 Jun;1(3):138-141. doi: 10.4021/wjon2010.06.214w. Epub 2010 May 19. PMID: 29147194; PMCID: PMC5649938.
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The hypertrophic chondrocyte: To be or not to be.

Histology and histopathology

Hallett SA, Ono W, Ono N.
PMID: 34137454
Histol Histopathol. 2021 Oct;36(10):1021-1036. doi: 10.14670/HH-18-355. Epub 2021 Jun 17.

Hypertrophic chondrocytes are the master regulators of endochondral ossification; however, their ultimate cell fates cells remain largely elusive due to their transient nature. Historically, hypertrophic chondrocytes have been considered as the terminal state of growth plate chondrocytes, which are...

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Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021;36(10):1021-1036doi: 10.14670/HH-18-355.
Hallett, S. A., Ono, W., & Ono, N. (2021). The hypertrophic chondrocyte: To be or not to be. Histology and histopathology, 36(10), 1021-1036. https://doi.org/10.14670/HH-18-355
Hallett, Shawn A, et al. "The hypertrophic chondrocyte: To be or not to be." Histology and histopathology vol. 36,10 (2021): 1021-1036. doi: https://doi.org/10.14670/HH-18-355
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021 Oct;36(10):1021-1036. doi: 10.14670/HH-18-355. Epub 2021 Jun 17. PMID: 34137454; PMCID: PMC8678381.
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Association of PTHrP levels in CSF with Alzheimer's disease biomarkers.

Clinical mass spectrometry (Del Mar, Calif.)

Kushnir MM, Michno W, Rockwood AL, Blennow K, Strathmann FG, Hanrieder J.
PMID: 34917769
Clin Mass Spectrom. 2018 Oct 09;14:124-129. doi: 10.1016/j.clinms.2018.10.001. eCollection 2019 Nov.

BACKGROUND: Parathyroid hormone-related protein (PTHrP) is involved in intracellular calcium regulation, neural cell proliferation and synaptic transmission. To date, no studies have been performed to evaluate the potential of PTHrP concentrations in cerebrospinal fluid (CSF) as a biomarker of...

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Kushnir MM, Michno W, Rockwood AL, et al. Association of PTHrP levels in CSF with Alzheimer's disease biomarkers. Clin Mass Spectrom. 2018;14:124-129doi: 10.1016/j.clinms.2018.10.001.
Kushnir, M. M., Michno, W., Rockwood, A. L., Blennow, K., Strathmann, F. G., & Hanrieder, J. (2019). Association of PTHrP levels in CSF with Alzheimer's disease biomarkers. Clinical mass spectrometry (Del Mar, Calif.), 14124-129. https://doi.org/10.1016/j.clinms.2018.10.001
Kushnir, Mark M, et al. "Association of PTHrP levels in CSF with Alzheimer's disease biomarkers." Clinical mass spectrometry (Del Mar, Calif.) vol. 14 (2019): 124-129. doi: https://doi.org/10.1016/j.clinms.2018.10.001
Kushnir MM, Michno W, Rockwood AL, Blennow K, Strathmann FG, Hanrieder J. Association of PTHrP levels in CSF with Alzheimer's disease biomarkers. Clin Mass Spectrom. 2018 Oct 09;14:124-129. doi: 10.1016/j.clinms.2018.10.001. eCollection 2019 Nov. PMID: 34917769; PMCID: PMC8669458.
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Hypercalcaemia, Renal Dysfunction, Anaemia, Bone Disease (CRAB Criteria): A Case of Lymphoma.

European journal of case reports in internal medicine

Singh B, Gogia P, Kaur P, Guragai N, Maroules M.
PMID: 33585332
Eur J Case Rep Intern Med. 2020 Dec 21;7(12):002140. doi: 10.12890/2020_002140. eCollection 2020.

Calcium elevation, Renal dysfunction, Anaemia and Bone disease (CRAB criteria) are usually seen in multiple myeloma (MM). We report a unique case of lymphoma with all the features of CRAB criteria. We describe a 59-year-old patient who presented with...

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Singh B, Gogia P, Kaur P, et al. Hypercalcaemia, Renal Dysfunction, Anaemia, Bone Disease (CRAB Criteria): A Case of Lymphoma. Eur J Case Rep Intern Med. 2020;7(12):002140doi: 10.12890/2020_002140.
Singh, B., Gogia, P., Kaur, P., Guragai, N., & Maroules, M. (2020). Hypercalcaemia, Renal Dysfunction, Anaemia, Bone Disease (CRAB Criteria): A Case of Lymphoma. European journal of case reports in internal medicine, 7(12), 002140. https://doi.org/10.12890/2020_002140
Singh, Balraj, et al. "Hypercalcaemia, Renal Dysfunction, Anaemia, Bone Disease (CRAB Criteria): A Case of Lymphoma." European journal of case reports in internal medicine vol. 7,12 (2020): 002140. doi: https://doi.org/10.12890/2020_002140
Singh B, Gogia P, Kaur P, Guragai N, Maroules M. Hypercalcaemia, Renal Dysfunction, Anaemia, Bone Disease (CRAB Criteria): A Case of Lymphoma. Eur J Case Rep Intern Med. 2020 Dec 21;7(12):002140. doi: 10.12890/2020_002140. eCollection 2020. PMID: 33585332; PMCID: PMC7875599.
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MiR-556-5p modulates migration, invasion, and epithelial-mesenchymal transition in breast cancer cells via targeting PTHrP.

Journal of molecular histology

Zhou R, Luo Z, Yin G, Yu L, Zhong H.
PMID: 35000027
J Mol Histol. 2022 Jan 09; doi: 10.1007/s10735-021-10056-4. Epub 2022 Jan 09.

Breast cancer bone metastases may block normal bone remodeling and promote bone degradation, during which several signaling pathways and small non-coding miRNAs might all play a role. miRNAs and target mRNAs that might be associated with breast cancer bone...

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Zhou R, Luo Z, Yin G, et al. MiR-556-5p modulates migration, invasion, and epithelial-mesenchymal transition in breast cancer cells via targeting PTHrP. J Mol Histol. 2022;doi: 10.1007/s10735-021-10056-4.
Zhou, R., Luo, Z., Yin, G., Yu, L., & Zhong, H. (2022). MiR-556-5p modulates migration, invasion, and epithelial-mesenchymal transition in breast cancer cells via targeting PTHrP. Journal of molecular histology, . https://doi.org/10.1007/s10735-021-10056-4
Zhou, Rongjun, et al. "MiR-556-5p modulates migration, invasion, and epithelial-mesenchymal transition in breast cancer cells via targeting PTHrP." Journal of molecular histology vol. (2022). doi: https://doi.org/10.1007/s10735-021-10056-4
Zhou R, Luo Z, Yin G, Yu L, Zhong H. MiR-556-5p modulates migration, invasion, and epithelial-mesenchymal transition in breast cancer cells via targeting PTHrP. J Mol Histol. 2022 Jan 09; doi: 10.1007/s10735-021-10056-4. Epub 2022 Jan 09. PMID: 35000027.
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Bone Metastatic Breast Cancer: Advances in Cell Signaling and Autophagy Related Mechanisms.

Cancers

Othman A, Winogradzki M, Lee L, Tandon M, Blank A, Pratap J.
PMID: 34503118
Cancers (Basel). 2021 Aug 26;13(17). doi: 10.3390/cancers13174310.

Bone metastasis is a frequent complication of breast cancer with nearly 70% of metastatic breast cancer patients developing bone metastasis during the course of their disease. The bone represents a dynamic microenvironment which provides a fertile soil for disseminated...

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Othman A, Winogradzki M, Lee L, et al. Bone Metastatic Breast Cancer: Advances in Cell Signaling and Autophagy Related Mechanisms. Cancers (Basel). 2021;13(17)doi: 10.3390/cancers13174310.
Othman, A., Winogradzki, M., Lee, L., Tandon, M., Blank, A., & Pratap, J. (2021). Bone Metastatic Breast Cancer: Advances in Cell Signaling and Autophagy Related Mechanisms. Cancers, 13(17), . https://doi.org/10.3390/cancers13174310
Othman, Ahmad, et al. "Bone Metastatic Breast Cancer: Advances in Cell Signaling and Autophagy Related Mechanisms." Cancers vol. 13,17 (2021). doi: https://doi.org/10.3390/cancers13174310
Othman A, Winogradzki M, Lee L, Tandon M, Blank A, Pratap J. Bone Metastatic Breast Cancer: Advances in Cell Signaling and Autophagy Related Mechanisms. Cancers (Basel). 2021 Aug 26;13(17). doi: 10.3390/cancers13174310. PMID: 34503118; PMCID: PMC8431094.
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The thermogenic activity of adjacent adipocytes fuels the progression of ccRCC and compromises anti-tumor therapeutic efficacy.

Cell metabolism

Wei G, Sun H, Dong K, Hu L, Wang Q, Zhuang Q, Zhu Y, Zhang X, Shao Y, Tang H, Li Z, Chen S, Lu J, Wang Y, Gan X, Zhong TP, Gui D, Hu X, Wang L, Liu J.
PMID: 34508696
Cell Metab. 2021 Oct 05;33(10):2021-2039.e8. doi: 10.1016/j.cmet.2021.08.012. Epub 2021 Sep 10.

Clear cell renal cell carcinoma (ccRCC) preferentially invades into perinephric adipose tissue (PAT), a process associated with poor prognosis. However, the detailed mechanisms underlying this interaction remain elusive. Here, we describe a bi-directional communication between ccRCC cells and the...

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Wei G, Sun H, Dong K, et al. The thermogenic activity of adjacent adipocytes fuels the progression of ccRCC and compromises anti-tumor therapeutic efficacy. Cell Metab. 2021;33(10):2021-2039.e8doi: 10.1016/j.cmet.2021.08.012.
Wei, G., Sun, H., Dong, K., Hu, L., Wang, Q., Zhuang, Q., Zhu, Y., Zhang, X., Shao, Y., Tang, H., Li, Z., Chen, S., Lu, J., Wang, Y., Gan, X., Zhong, T. P., Gui, D., Hu, X., Wang, L., & Liu, J. (2021). The thermogenic activity of adjacent adipocytes fuels the progression of ccRCC and compromises anti-tumor therapeutic efficacy. Cell metabolism, 33(10), 2021-2039.e8. https://doi.org/10.1016/j.cmet.2021.08.012
Wei, Gang, et al. "The thermogenic activity of adjacent adipocytes fuels the progression of ccRCC and compromises anti-tumor therapeutic efficacy." Cell metabolism vol. 33,10 (2021): 2021-2039.e8. doi: https://doi.org/10.1016/j.cmet.2021.08.012
Wei G, Sun H, Dong K, Hu L, Wang Q, Zhuang Q, Zhu Y, Zhang X, Shao Y, Tang H, Li Z, Chen S, Lu J, Wang Y, Gan X, Zhong TP, Gui D, Hu X, Wang L, Liu J. The thermogenic activity of adjacent adipocytes fuels the progression of ccRCC and compromises anti-tumor therapeutic efficacy. Cell Metab. 2021 Oct 05;33(10):2021-2039.e8. doi: 10.1016/j.cmet.2021.08.012. Epub 2021 Sep 10. PMID: 34508696.
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The hypertrophic chondrocyte: To be or not to be.

Histology and histopathology

Hallett SA, Ono W, Ono N.
PMID: 34137454
Histol Histopathol. 2021 Jun 17;18355. doi: 10.14670/HH-18-355. Epub 2021 Jun 17.

Hypertrophic chondrocytes are the master regulators of endochondral ossification; however, their ultimate cell fates cells remain largely elusive due to their transient nature. Historically, hypertrophic chondrocytes have been considered as the terminal state of growth plate chondrocytes, which are...

Cite
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021;18355doi: 10.14670/HH-18-355.
Hallett, S. A., Ono, W., & Ono, N. (2021). The hypertrophic chondrocyte: To be or not to be. Histology and histopathology, 18355. https://doi.org/10.14670/HH-18-355
Hallett, Shawn A, et al. "The hypertrophic chondrocyte: To be or not to be." Histology and histopathology vol. (2021): 18355. doi: https://doi.org/10.14670/HH-18-355
Hallett SA, Ono W, Ono N. The hypertrophic chondrocyte: To be or not to be. Histol Histopathol. 2021 Jun 17;18355. doi: 10.14670/HH-18-355. Epub 2021 Jun 17. PMID: 34137454; PMCID: PMC8678381.
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Showing 145 to 152 of 152 entries