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Showing 25 to 36 of 1115 entries
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Association of OPN overexpression with tumor stage, differentiation, metastasis and tumor progression in human laryngeal squamous cell carcinoma.

International journal of clinical and experimental medicine

Chen J, Zhu C, He Z, Geng M, Li G, Tao X, Zhang F.
PMID: 26221249
Int J Clin Exp Med. 2015 May 15;8(5):7116-24. eCollection 2015.

BACKGROUND: Osteopontin (OPN) is overexpressed in many human tumors and involved in promotion of cancer cells by regulating various facets of tumor progression such as cell proliferation, invasion and metastasis. To understand roles of OPN in tumor progression of...

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Chen J, Zhu C, He Z, et al. Association of OPN overexpression with tumor stage, differentiation, metastasis and tumor progression in human laryngeal squamous cell carcinoma. Int J Clin Exp Med. 2015;8(5):7116-24
Chen, J., Zhu, C., He, Z., Geng, M., Li, G., Tao, X., & Zhang, F. (2015). Association of OPN overexpression with tumor stage, differentiation, metastasis and tumor progression in human laryngeal squamous cell carcinoma. International journal of clinical and experimental medicine, 8(5), 7116-24.
Chen, Jianqiu, et al. "Association of OPN overexpression with tumor stage, differentiation, metastasis and tumor progression in human laryngeal squamous cell carcinoma." International journal of clinical and experimental medicine vol. 8,5 (2015): 7116-24.
Chen J, Zhu C, He Z, Geng M, Li G, Tao X, Zhang F. Association of OPN overexpression with tumor stage, differentiation, metastasis and tumor progression in human laryngeal squamous cell carcinoma. Int J Clin Exp Med. 2015 May 15;8(5):7116-24. eCollection 2015. PMID: 26221249; PMCID: PMC4509194.
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The role of Randall plaques on kidney stone formation.

Translational andrology and urology

Chung HJ.
PMID: 26816774
Transl Androl Urol. 2014 Sep;3(3):251-4. doi: 10.3978/j.issn.2223-4683.2014.07.03.

Randall's plaque is microscopically a plaque of calcium deposited in the interstitial tissue of the renal papilla. These plaques are thought to serve as a nidus for urinary stone formation. Large amounts of Randall's plaque are unique to idiopathic...

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Chung HJ. The role of Randall plaques on kidney stone formation. Transl Androl Urol. 2014;3(3):251-4doi: 10.3978/j.issn.2223-4683.2014.07.03.
Chung, H. J. (2014). The role of Randall plaques on kidney stone formation. Translational andrology and urology, 3(3), 251-4. https://doi.org/10.3978/j.issn.2223-4683.2014.07.03
Chung, Hsiao-Jen. "The role of Randall plaques on kidney stone formation." Translational andrology and urology vol. 3,3 (2014): 251-4. doi: https://doi.org/10.3978/j.issn.2223-4683.2014.07.03
Chung HJ. The role of Randall plaques on kidney stone formation. Transl Androl Urol. 2014 Sep;3(3):251-4. doi: 10.3978/j.issn.2223-4683.2014.07.03. PMID: 26816774; PMCID: PMC4708577.
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Association between promoter polymorphisms of OPN gene and cancer risk: a meta-analysis.

OncoTargets and therapy

Liu J, He C, Yuan Q, Wang Z, Xing C, Yuan Y.
PMID: 26766914
Onco Targets Ther. 2015 Dec 30;9:87-97. doi: 10.2147/OTT.S94606. eCollection 2016.

BACKGROUND: Results of the association between polymorphisms of osteopontin (OPN) gene promoter region and risk of cancer were inconclusive. The aim of this meta-analysis was to elucidate whether OPN promoter polymorphisms were associated with cancer risk.METHODS: Electronic databases including...

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Liu J, He C, Yuan Q, et al. Association between promoter polymorphisms of OPN gene and cancer risk: a meta-analysis. Onco Targets Ther. 2015;9:87-97doi: 10.2147/OTT.S94606.
Liu, J., He, C., Yuan, Q., Wang, Z., Xing, C., & Yuan, Y. (2016). Association between promoter polymorphisms of OPN gene and cancer risk: a meta-analysis. OncoTargets and therapy, 987-97. https://doi.org/10.2147/OTT.S94606
Liu, Jingwei, et al. "Association between promoter polymorphisms of OPN gene and cancer risk: a meta-analysis." OncoTargets and therapy vol. 9 (2016): 87-97. doi: https://doi.org/10.2147/OTT.S94606
Liu J, He C, Yuan Q, Wang Z, Xing C, Yuan Y. Association between promoter polymorphisms of OPN gene and cancer risk: a meta-analysis. Onco Targets Ther. 2015 Dec 30;9:87-97. doi: 10.2147/OTT.S94606. eCollection 2016. PMID: 26766914; PMCID: PMC4699542.
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Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin.

Genes & nutrition

Catalán V, Gómez-Ambrosi J, Rodríguez A, Ramírez B, Valentí V, Moncada R, Silva C, Salvador J, Frühbeck G.
PMID: 25869413
Genes Nutr. 2015 May;10(3):460. doi: 10.1007/s12263-015-0460-8. Epub 2015 Apr 14.

Inflammation is a critical contributor to the pathogenesis of metabolic disorders with adipose tissue being crucial in the inflammatory response by releasing multiple adipokines with either pro- or anti-inflammatory activities with potential functions as metabolic regulators. Peripheral blood mononuclear...

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Catalán V, Gómez-Ambrosi J, Rodríguez A, et al. Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin. Genes Nutr. 2015;10(3):460doi: 10.1007/s12263-015-0460-8.
Catalán, V., Gómez-Ambrosi, J., Rodríguez, A., Ramírez, B., Valentí, V., Moncada, R., Silva, C., Salvador, J., & Frühbeck, G. (2015). Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin. Genes & nutrition, 10(3), 460. https://doi.org/10.1007/s12263-015-0460-8
Catalán, Victoria, et al. "Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin." Genes & nutrition vol. 10,3 (2015): 460. doi: https://doi.org/10.1007/s12263-015-0460-8
Catalán V, Gómez-Ambrosi J, Rodríguez A, Ramírez B, Valentí V, Moncada R, Silva C, Salvador J, Frühbeck G. Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin. Genes Nutr. 2015 May;10(3):460. doi: 10.1007/s12263-015-0460-8. Epub 2015 Apr 14. PMID: 25869413; PMCID: PMC4395559.
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The ins and outs of osteopontin.

Oncoimmunology

Chiodoni C, Sangaletti S, Tripodo C, Colombo MP.
PMID: 25949895
Oncoimmunology. 2015 Apr 02;4(3):e978711. doi: 10.4161/2162402X.2014.978711. eCollection 2015 Mar.

The continuous remodeling of progressing tumors demands non-physiologic production of extracellular matrix (ECM) proteins. Among them, osteopontin (OPN) has been largely involved in tumor progression and metastasis. We have recently discovered a new mechanism for OPN in the metastatic...

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Chiodoni C, Sangaletti S, Tripodo C, et al. The ins and outs of osteopontin. Oncoimmunology. 2015;4(3):e978711doi: 10.4161/2162402X.2014.978711.
Chiodoni, C., Sangaletti, S., Tripodo, C., & Colombo, M. P. (2015). The ins and outs of osteopontin. Oncoimmunology, 4(3), e978711. https://doi.org/10.4161/2162402X.2014.978711
Chiodoni, Claudia, et al. "The ins and outs of osteopontin." Oncoimmunology vol. 4,3 (2015): e978711. doi: https://doi.org/10.4161/2162402X.2014.978711
Chiodoni C, Sangaletti S, Tripodo C, Colombo MP. The ins and outs of osteopontin. Oncoimmunology. 2015 Apr 02;4(3):e978711. doi: 10.4161/2162402X.2014.978711. eCollection 2015 Mar. PMID: 25949895; PMCID: PMC4404798.
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Effect of osteopontin on the mRNA expression of ADAMTS4 and ADAMTS5 in chondrocytes from patients with knee osteoarthritis.

Experimental and therapeutic medicine

Gao SG, Zeng C, Song Y, Tian J, Cheng C, Yang T, Li H, Zhang FJ, Lei GH.
PMID: 26136925
Exp Ther Med. 2015 May;9(5):1979-1983. doi: 10.3892/etm.2015.2310. Epub 2015 Feb 24.

Previous studies have demonstrated that osteopontin (OPN) levels are elevated in the synovial fluid and articular cartilage, and are associated with the severity of knee osteoarthritis (OA). However, the role of OPN in the pathogenesis of OA has yet...

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Gao SG, Zeng C, Song Y, et al. Effect of osteopontin on the mRNA expression of ADAMTS4 and ADAMTS5 in chondrocytes from patients with knee osteoarthritis. Exp Ther Med. 2015;9(5):1979-1983doi: 10.3892/etm.2015.2310.
Gao, S. G., Zeng, C., Song, Y., Tian, J., Cheng, C., Yang, T., Li, H., Zhang, F. J., & Lei, G. H. (2015). Effect of osteopontin on the mRNA expression of ADAMTS4 and ADAMTS5 in chondrocytes from patients with knee osteoarthritis. Experimental and therapeutic medicine, 9(5), 1979-1983. https://doi.org/10.3892/etm.2015.2310
Gao, Shu-Guang, et al. "Effect of osteopontin on the mRNA expression of ADAMTS4 and ADAMTS5 in chondrocytes from patients with knee osteoarthritis." Experimental and therapeutic medicine vol. 9,5 (2015): 1979-1983. doi: https://doi.org/10.3892/etm.2015.2310
Gao SG, Zeng C, Song Y, Tian J, Cheng C, Yang T, Li H, Zhang FJ, Lei GH. Effect of osteopontin on the mRNA expression of ADAMTS4 and ADAMTS5 in chondrocytes from patients with knee osteoarthritis. Exp Ther Med. 2015 May;9(5):1979-1983. doi: 10.3892/etm.2015.2310. Epub 2015 Feb 24. PMID: 26136925; PMCID: PMC4471803.
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Genetic variations in the osteopontin promoters T-443C and G-156GG increase carotid intima-media thickness.

International journal of general medicine

Yueniwati Y, Yurina V, Sobah N, Rahayu E.
PMID: 27274305
Int J Gen Med. 2016 May 12;9:117-22. doi: 10.2147/IJGM.S94537. eCollection 2016.

Carotid intima-media thickness (CIMT) is a clear predictor of atherosclerosis. The increase of CIMT is affected by mutations in the osteopontin (OPN) promoters. The purpose of this study was to examine genetic variations in OPN promoters T-443C and G-156GG,...

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Yueniwati Y, Yurina V, Sobah N, et al. Genetic variations in the osteopontin promoters T-443C and G-156GG increase carotid intima-media thickness. Int J Gen Med. 2016;9:117-22doi: 10.2147/IJGM.S94537.
Yueniwati, Y., Yurina, V., Sobah, N., & Rahayu, E. (2016). Genetic variations in the osteopontin promoters T-443C and G-156GG increase carotid intima-media thickness. International journal of general medicine, 9117-22. https://doi.org/10.2147/IJGM.S94537
Yueniwati, Yuyun, et al. "Genetic variations in the osteopontin promoters T-443C and G-156GG increase carotid intima-media thickness." International journal of general medicine vol. 9 (2016): 117-22. doi: https://doi.org/10.2147/IJGM.S94537
Yueniwati Y, Yurina V, Sobah N, Rahayu E. Genetic variations in the osteopontin promoters T-443C and G-156GG increase carotid intima-media thickness. Int J Gen Med. 2016 May 12;9:117-22. doi: 10.2147/IJGM.S94537. eCollection 2016. PMID: 27274305; PMCID: PMC4869651.
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The expression of osteopontin with condylar remodeling in growing rats.

Clinical orthodontics and research

Sugiyama H, Imada M, Sasaki A, Ishino Y, Kawata T, Tanne K.
PMID: 11683808
Clin Orthod Res. 2001 Nov;4(4):194-9. doi: 10.1034/j.1600-0544.2001.40403.x.

It is suggested that osteopontin may promote osteoclast binding to resorptive sites by interacting with the alphavbeta3 receptor on osteoclasts. However, the role of osteopontin in functional remodeling of bony structures remains unclear. The present study was conducted to...

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Sugiyama H, Imada M, Sasaki A, et al. The expression of osteopontin with condylar remodeling in growing rats. Clin Orthod Res. 2001;4(4):194-9doi: 10.1034/j.1600-0544.2001.40403.x.
Sugiyama, H., Imada, M., Sasaki, A., Ishino, Y., Kawata, T., & Tanne, K. (2001). The expression of osteopontin with condylar remodeling in growing rats. Clinical orthodontics and research, 4(4), 194-9. https://doi.org/10.1034/j.1600-0544.2001.40403.x
Sugiyama, H, et al. "The expression of osteopontin with condylar remodeling in growing rats." Clinical orthodontics and research vol. 4,4 (2001): 194-9. doi: https://doi.org/10.1034/j.1600-0544.2001.40403.x
Sugiyama H, Imada M, Sasaki A, Ishino Y, Kawata T, Tanne K. The expression of osteopontin with condylar remodeling in growing rats. Clin Orthod Res. 2001 Nov;4(4):194-9. doi: 10.1034/j.1600-0544.2001.40403.x. PMID: 11683808.
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Angiotensin II: a regulator of inflammation during renal disease?.

International journal of immunopathology and pharmacology

Antus B, Exton MS, Rosivall L.
PMID: 12622886
Int J Immunopathol Pharmacol. 2001 Jan-Apr;14(1):25-30. doi: 10.1177/039463200101400105.

It has been recently recognized that besides its vasoactive actions Angiotensin II (Ang II) exerts various immunomodulatory effects that may contribute to renal injury and to the progression of renal disease. Consistent with this concept, Ang II facilitates macrophage...

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Antus B, Exton MS, Rosivall L. Angiotensin II: a regulator of inflammation during renal disease?. Int J Immunopathol Pharmacol. 2001;14(1):25-30doi: 10.1177/039463200101400105.
Antus, B., Exton, M. S., & Rosivall, L. (2001). Angiotensin II: a regulator of inflammation during renal disease?. International journal of immunopathology and pharmacology, 14(1), 25-30. https://doi.org/10.1177/039463200101400105
Antus, B, et al. "Angiotensin II: a regulator of inflammation during renal disease?." International journal of immunopathology and pharmacology vol. 14,1 (2001): 25-30. doi: https://doi.org/10.1177/039463200101400105
Antus B, Exton MS, Rosivall L. Angiotensin II: a regulator of inflammation during renal disease?. Int J Immunopathol Pharmacol. 2001 Jan-Apr;14(1):25-30. doi: 10.1177/039463200101400105. PMID: 12622886.
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Isolation of progenitor cells from cord blood using adhesion matrices.

Cytotechnology

Maurice S, Srouji S, Livne E.
PMID: 19003027
Cytotechnology. 2007 Jun;54(2):121-33. doi: 10.1007/s10616-007-9077-0. Epub 2007 Jun 30.

The aim of this study was to develop optimal conditions for selective adhesion and isolation of mesenchymal progenitor cells (MPCs) from cord blood and to determine their potential for osteogenic differentiation. Mononuclear cells (MNCs) were isolated by Ficoll-Paque gradient...

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Maurice S, Srouji S, Livne E. Isolation of progenitor cells from cord blood using adhesion matrices. Cytotechnology. 2007;54(2):121-33doi: 10.1007/s10616-007-9077-0.
Maurice, S., Srouji, S., & Livne, E. (2007). Isolation of progenitor cells from cord blood using adhesion matrices. Cytotechnology, 54(2), 121-33. https://doi.org/10.1007/s10616-007-9077-0
Maurice, Sarah, et al. "Isolation of progenitor cells from cord blood using adhesion matrices." Cytotechnology vol. 54,2 (2007): 121-33. doi: https://doi.org/10.1007/s10616-007-9077-0
Maurice S, Srouji S, Livne E. Isolation of progenitor cells from cord blood using adhesion matrices. Cytotechnology. 2007 Jun;54(2):121-33. doi: 10.1007/s10616-007-9077-0. Epub 2007 Jun 30. PMID: 19003027; PMCID: PMC2267502.
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[Twy mouse and ossification of the posterior longitudinal ligament of the spine (OPLL)].

Clinical calcium

Yamazaki M, Okawa A, Moriya H.
PMID: 15775405
Clin Calcium. 2002 Aug;12(8):1109-13. doi: CliCa020811091113.

Spinal hyperostotic lesions in twy mouse resemble to those observed in human ossification of posterior longitudinal ligament of the spine (OPLL). To investigate pathogenic mechanisms of OPLL, studies have been performed on the spinal lesions of twy mouse. Molecular...

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Yamazaki M, Okawa A, Moriya H. [Twy mouse and ossification of the posterior longitudinal ligament of the spine (OPLL)]. Clin Calcium. 2002;12(8):1109-13doi: CliCa020811091113.
Yamazaki, M., Okawa, A., & Moriya, H. (2002). [Twy mouse and ossification of the posterior longitudinal ligament of the spine (OPLL)]. Clinical calcium, 12(8), 1109-13. https://doi.org/CliCa020811091113
Yamazaki, Masashi, et al. "[Twy mouse and ossification of the posterior longitudinal ligament of the spine (OPLL)]." Clinical calcium vol. 12,8 (2002): 1109-13. doi: https://doi.org/CliCa020811091113
Yamazaki M, Okawa A, Moriya H. [Twy mouse and ossification of the posterior longitudinal ligament of the spine (OPLL)]. Clin Calcium. 2002 Aug;12(8):1109-13. doi: CliCa020811091113. Japanese. PMID: 15775405.
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[A gene involved in angiogenesis and bone -osteopontin].

Clinical calcium

Noda M, Tsuji K, Morinobu M, Ishijima M, Nifuji A.
PMID: 15775315
Clin Calcium. 2002 Mar;12(3):363-7. doi: CliCa0203363367.

Osteopontin is expressed in bone and stored in hard tissues while it promoted survival of endothelial cells and enhances angiogenesis towards bone in ectopic implantation experiments. As regulation of bone remodeling in osteoporosis and other metabolic bone diseases is...

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Noda M, Tsuji K, Morinobu M, et al. [A gene involved in angiogenesis and bone -osteopontin]. Clin Calcium. 2002;12(3):363-7doi: CliCa0203363367.
Noda, M., Tsuji, K., Morinobu, M., Ishijima, M., & Nifuji, A. (2002). [A gene involved in angiogenesis and bone -osteopontin]. Clinical calcium, 12(3), 363-7. https://doi.org/CliCa0203363367
Noda, Masaki, et al. "[A gene involved in angiogenesis and bone -osteopontin]." Clinical calcium vol. 12,3 (2002): 363-7. doi: https://doi.org/CliCa0203363367
Noda M, Tsuji K, Morinobu M, Ishijima M, Nifuji A. [A gene involved in angiogenesis and bone -osteopontin]. Clin Calcium. 2002 Mar;12(3):363-7. doi: CliCa0203363367. Japanese. PMID: 15775315.
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Showing 25 to 36 of 1115 entries