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Overexpression of Isoforms of Nitric Oxide Synthase 1 Adaptor Protein, Encoded by a Risk Gene for Schizophrenia, Alters Actin Dynamics and Synaptic Function.

Frontiers in cellular neuroscience

Hernandez K, Swiatkowski P, Patel MV, Liang C, Dudzinski NR, Brzustowicz LM, Firestein BL.
PMID: 26869880
Front Cell Neurosci. 2016 Feb 02;10:6. doi: 10.3389/fncel.2016.00006. eCollection 2016.

Proper communication between neurons depends upon appropriate patterning of dendrites and correct distribution and structure of spines. Schizophrenia is a neuropsychiatric disorder characterized by alterations in dendrite branching and spine density. Nitric oxide synthase 1 adaptor protein (NOS1AP), a...

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Hernandez K, Swiatkowski P, Patel MV, et al. Overexpression of Isoforms of Nitric Oxide Synthase 1 Adaptor Protein, Encoded by a Risk Gene for Schizophrenia, Alters Actin Dynamics and Synaptic Function. Front Cell Neurosci. 2016;10:6doi: 10.3389/fncel.2016.00006.
Hernandez, K., Swiatkowski, P., Patel, M. V., Liang, C., Dudzinski, N. R., Brzustowicz, L. M., & Firestein, B. L. (2016). Overexpression of Isoforms of Nitric Oxide Synthase 1 Adaptor Protein, Encoded by a Risk Gene for Schizophrenia, Alters Actin Dynamics and Synaptic Function. Frontiers in cellular neuroscience, 106. https://doi.org/10.3389/fncel.2016.00006
Hernandez, Kristina, et al. "Overexpression of Isoforms of Nitric Oxide Synthase 1 Adaptor Protein, Encoded by a Risk Gene for Schizophrenia, Alters Actin Dynamics and Synaptic Function." Frontiers in cellular neuroscience vol. 10 (2016): 6. doi: https://doi.org/10.3389/fncel.2016.00006
Hernandez K, Swiatkowski P, Patel MV, Liang C, Dudzinski NR, Brzustowicz LM, Firestein BL. Overexpression of Isoforms of Nitric Oxide Synthase 1 Adaptor Protein, Encoded by a Risk Gene for Schizophrenia, Alters Actin Dynamics and Synaptic Function. Front Cell Neurosci. 2016 Feb 02;10:6. doi: 10.3389/fncel.2016.00006. eCollection 2016. PMID: 26869880; PMCID: PMC4735351.
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β2-adrenergic stress evaluation of coronary endothelial-dependent vasodilator function in mice using (11)C-acetate micro-PET imaging of myocardial blood flow and oxidative metabolism.

EJNMMI research

Croteau E, Renaud JM, Archer C, Klein R, DaSilva JN, Ruddy TD, Beanlands RS, deKemp RA.
PMID: 25621195
EJNMMI Res. 2014 Dec 16;4(1):68. doi: 10.1186/s13550-014-0068-9. eCollection 2014 Dec.

BACKGROUND: Endothelial dysfunction is associated with vascular risk factors such as dyslipidemia, hypertension, and diabetes, leading to coronary atherosclerosis. Sympathetic stress using cold-pressor testing (CPT) has been used to measure coronary endothelial function in humans with positron emission tomography...

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Croteau E, Renaud JM, Archer C, et al. β2-adrenergic stress evaluation of coronary endothelial-dependent vasodilator function in mice using (11)C-acetate micro-PET imaging of myocardial blood flow and oxidative metabolism. EJNMMI Res. 2014;4(1):68doi: 10.1186/s13550-014-0068-9.
Croteau, E., Renaud, J. M., Archer, C., Klein, R., DaSilva, J. N., Ruddy, T. D., Beanlands, R. S., & deKemp, R. A. (2014). β2-adrenergic stress evaluation of coronary endothelial-dependent vasodilator function in mice using (11)C-acetate micro-PET imaging of myocardial blood flow and oxidative metabolism. EJNMMI research, 4(1), 68. https://doi.org/10.1186/s13550-014-0068-9
Croteau, Etienne, et al. "β2-adrenergic stress evaluation of coronary endothelial-dependent vasodilator function in mice using (11)C-acetate micro-PET imaging of myocardial blood flow and oxidative metabolism." EJNMMI research vol. 4,1 (2014): 68. doi: https://doi.org/10.1186/s13550-014-0068-9
Croteau E, Renaud JM, Archer C, Klein R, DaSilva JN, Ruddy TD, Beanlands RS, deKemp RA. β2-adrenergic stress evaluation of coronary endothelial-dependent vasodilator function in mice using (11)C-acetate micro-PET imaging of myocardial blood flow and oxidative metabolism. EJNMMI Res. 2014 Dec 16;4(1):68. doi: 10.1186/s13550-014-0068-9. eCollection 2014 Dec. PMID: 25621195; PMCID: PMC4293492.
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Functional Effects of Alagebrium (ALT-711)-Isolated Rat Carotid Artery.

The Eurasian journal of medicine

Toprak C, Sirmagul B, Yigitaslan S.
PMID: 29123442
Eurasian J Med. 2017 Oct;49(3):188-192. doi: 10.5152/eurasianjmed.2017.17046.

OBJECTIVE: In our study, the effects of glycosylated protein cross-link breaker, alagebrium was investigated on isolated rat carotid artery using myography. Alagebrium showed vasodilator effect on carotid artery rings; particularly, this effect was significantly increased in endothelium-intact rings.MATERIALS AND...

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Toprak C, Sirmagul B, Yigitaslan S. Functional Effects of Alagebrium (ALT-711)-Isolated Rat Carotid Artery. Eurasian J Med. 2017;49(3):188-192doi: 10.5152/eurasianjmed.2017.17046.
Toprak, C., Sirmagul, B., & Yigitaslan, S. (2017). Functional Effects of Alagebrium (ALT-711)-Isolated Rat Carotid Artery. The Eurasian journal of medicine, 49(3), 188-192. https://doi.org/10.5152/eurasianjmed.2017.17046
Toprak, Cigdem, et al. "Functional Effects of Alagebrium (ALT-711)-Isolated Rat Carotid Artery." The Eurasian journal of medicine vol. 49,3 (2017): 188-192. doi: https://doi.org/10.5152/eurasianjmed.2017.17046
Toprak C, Sirmagul B, Yigitaslan S. Functional Effects of Alagebrium (ALT-711)-Isolated Rat Carotid Artery. Eurasian J Med. 2017 Oct;49(3):188-192. doi: 10.5152/eurasianjmed.2017.17046. PMID: 29123442; PMCID: PMC5665628.
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Pre-gestational overweight in guinea pig sows induces fetal vascular dysfunction and increased rate of large and small fetuses.

Journal of developmental origins of health and disease

Krause BJ, Herrera EA, Díaz-López FA, Farías M, Uauy R, Casanello P.
PMID: 26490762
J Dev Orig Health Dis. 2016 Jun;7(3):237-243. doi: 10.1017/S2040174415007266. Epub 2015 Oct 22.

In humans, obesity before and during pregnancy is associated with both fetal macrosomia and growth restriction, and long-term cardiovascular risk in the offspring. We aimed to determine whether overweighted pregnant guinea pig sows results in an increased fetal weight...

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Krause BJ, Herrera EA, Díaz-López FA, et al. Pre-gestational overweight in guinea pig sows induces fetal vascular dysfunction and increased rate of large and small fetuses. J Dev Orig Health Dis. 2015;7(3):237-243doi: 10.1017/S2040174415007266.
Krause, B. J., Herrera, E. A., Díaz-López, F. A., Farías, M., Uauy, R., & Casanello, P. (2016). Pre-gestational overweight in guinea pig sows induces fetal vascular dysfunction and increased rate of large and small fetuses. Journal of developmental origins of health and disease, 7(3), 237-243. https://doi.org/10.1017/S2040174415007266
Krause, B J, et al. "Pre-gestational overweight in guinea pig sows induces fetal vascular dysfunction and increased rate of large and small fetuses." Journal of developmental origins of health and disease vol. 7,3 (2016): 237-243. doi: https://doi.org/10.1017/S2040174415007266
Krause BJ, Herrera EA, Díaz-López FA, Farías M, Uauy R, Casanello P. Pre-gestational overweight in guinea pig sows induces fetal vascular dysfunction and increased rate of large and small fetuses. J Dev Orig Health Dis. 2016 Jun;7(3):237-243. doi: 10.1017/S2040174415007266. Epub 2015 Oct 22. PMID: 26490762.
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Effects of Benzo(e)pyrene on Reactive Oxygen/Nitrogen Species and Inflammatory Cytokines Induction in Human RPE Cells and Attenuation by Mitochondrial-involved Mechanism.

Journal of ophthalmic & vision research

Estrago-Franco MF, Moustafa MT, Riazi-Esfahani M, Sapkal AU, Piche-Lopez R, Patil AJ, Sharma A, Falatoonzadeh P, Chwa M, Luczy-Bachman G, Kuppermann BD, Kenney MC.
PMID: 27994808
J Ophthalmic Vis Res. 2016 Oct-Dec;11(4):385-393. doi: 10.4103/2008-322X.194091.

PURPOSE: To identify inhibitors that could effectively lower reactive oxygen/nitrogen species (ROS/RNS), complement and inflammatory cytokine levels induced by Benzo(e)pyrene [B(e)p], an element of cigarette smoke, in human retinal pigment epithelial cells (ARPE-19) METHODS: ARPE-19 cells were treated for...

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Estrago-Franco MF, Moustafa MT, Riazi-Esfahani M, et al. Effects of Benzo(e)pyrene on Reactive Oxygen/Nitrogen Species and Inflammatory Cytokines Induction in Human RPE Cells and Attenuation by Mitochondrial-involved Mechanism. J Ophthalmic Vis Res. 2016;11(4):385-393doi: 10.4103/2008-322X.194091.
Estrago-Franco, M. F., Moustafa, M. T., Riazi-Esfahani, M., Sapkal, A. U., Piche-Lopez, R., Patil, A. J., Sharma, A., Falatoonzadeh, P., Chwa, M., Luczy-Bachman, G., Kuppermann, B. D., & Kenney, M. C. (2016). Effects of Benzo(e)pyrene on Reactive Oxygen/Nitrogen Species and Inflammatory Cytokines Induction in Human RPE Cells and Attenuation by Mitochondrial-involved Mechanism. Journal of ophthalmic & vision research, 11(4), 385-393. https://doi.org/10.4103/2008-322X.194091
Estrago-Franco, M Fernanda, et al. "Effects of Benzo(e)pyrene on Reactive Oxygen/Nitrogen Species and Inflammatory Cytokines Induction in Human RPE Cells and Attenuation by Mitochondrial-involved Mechanism." Journal of ophthalmic & vision research vol. 11,4 (2016): 385-393. doi: https://doi.org/10.4103/2008-322X.194091
Estrago-Franco MF, Moustafa MT, Riazi-Esfahani M, Sapkal AU, Piche-Lopez R, Patil AJ, Sharma A, Falatoonzadeh P, Chwa M, Luczy-Bachman G, Kuppermann BD, Kenney MC. Effects of Benzo(e)pyrene on Reactive Oxygen/Nitrogen Species and Inflammatory Cytokines Induction in Human RPE Cells and Attenuation by Mitochondrial-involved Mechanism. J Ophthalmic Vis Res. 2016 Oct-Dec;11(4):385-393. doi: 10.4103/2008-322X.194091. PMID: 27994808; PMCID: PMC5139551.
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Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain.

Frontiers in aging neuroscience

Thangavel R, Kempuraj D, Zaheer S, Raikwar S, Ahmed ME, Selvakumar GP, Iyer SS, Zaheer A.
PMID: 28572767
Front Aging Neurosci. 2017 May 18;9:150. doi: 10.3389/fnagi.2017.00150. eCollection 2017.

Alzheimer's disease (AD) is characterized by the presence of neuropathological lesions containing amyloid plaques (APs) and neurofibrillary tangles (NFTs). AD is associated with mitochondrial dysfunctions, neuroinflammation and neurodegeneration in the brain. We have previously demonstrated enhanced expression of the...

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Thangavel R, Kempuraj D, Zaheer S, et al. Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain. Front Aging Neurosci. 2017;9:150doi: 10.3389/fnagi.2017.00150.
Thangavel, R., Kempuraj, D., Zaheer, S., Raikwar, S., Ahmed, M. E., Selvakumar, G. P., Iyer, S. S., & Zaheer, A. (2017). Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain. Frontiers in aging neuroscience, 9150. https://doi.org/10.3389/fnagi.2017.00150
Thangavel, Ramasamy, et al. "Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain." Frontiers in aging neuroscience vol. 9 (2017): 150. doi: https://doi.org/10.3389/fnagi.2017.00150
Thangavel R, Kempuraj D, Zaheer S, Raikwar S, Ahmed ME, Selvakumar GP, Iyer SS, Zaheer A. Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain. Front Aging Neurosci. 2017 May 18;9:150. doi: 10.3389/fnagi.2017.00150. eCollection 2017. PMID: 28572767; PMCID: PMC5435744.
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Anti-inflammatory Effects of KOTMIN13: A Mixed Herbal Medicine in LPS-stimulated RAW 264.7 Cells and Mouse Edema Models.

Pharmacognosy magazine

Lee E, Kim SG, Park NY, Park HH, Jeong KT, Choi J, Lee IH, Lee H, Lee E.
PMID: 28539710
Pharmacogn Mag. 2017 Apr-Jun;13(50):216-221. doi: 10.4103/0973-1296.204548. Epub 2017 Apr 18.

BACKGROUND: A Korean herbal medicine, KOTMIN13, composed of MATERIALS AND METHODS: To evaluate the anti-inflammatory activity of KOTMIN13 for RESULTS: KOTMIN13 reduced the release of inflammatory mediators [nitric oxide, prostaglandin E2, interleukin (IL)-1β, and IL-6] and the protein expression...

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Lee E, Kim SG, Park NY, et al. Anti-inflammatory Effects of KOTMIN13: A Mixed Herbal Medicine in LPS-stimulated RAW 264.7 Cells and Mouse Edema Models. Pharmacogn Mag. 2017;13(50):216-221doi: 10.4103/0973-1296.204548.
Lee, E., Kim, S. G., Park, N. Y., Park, H. H., Jeong, K. T., Choi, J., Lee, I. H., Lee, H., & Lee, E. (2017). Anti-inflammatory Effects of KOTMIN13: A Mixed Herbal Medicine in LPS-stimulated RAW 264.7 Cells and Mouse Edema Models. Pharmacognosy magazine, 13(50), 216-221. https://doi.org/10.4103/0973-1296.204548
Lee, Eujin, et al. "Anti-inflammatory Effects of KOTMIN13: A Mixed Herbal Medicine in LPS-stimulated RAW 264.7 Cells and Mouse Edema Models." Pharmacognosy magazine vol. 13,50 (2017): 216-221. doi: https://doi.org/10.4103/0973-1296.204548
Lee E, Kim SG, Park NY, Park HH, Jeong KT, Choi J, Lee IH, Lee H, Lee E. Anti-inflammatory Effects of KOTMIN13: A Mixed Herbal Medicine in LPS-stimulated RAW 264.7 Cells and Mouse Edema Models. Pharmacogn Mag. 2017 Apr-Jun;13(50):216-221. doi: 10.4103/0973-1296.204548. Epub 2017 Apr 18. PMID: 28539710; PMCID: PMC5421415.
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Peroxisome proliferator-activated receptor γ (PPARγ) mediates the protective effect of quercetin against myocardial ischemia-reperfusion injury via suppressing the NF-κB pathway.

American journal of translational research

Liu X, Yu Z, Huang X, Gao Y, Wang X, Gu J, Xue S.
PMID: 28077993
Am J Transl Res. 2016 Dec 15;8(12):5169-5186. eCollection 2016.

Quercetin plays an important role in myocardial ischemia and reperfusion injury (IRI). However, the underlying mechanism for the protective effect of quercetin is largely unclear. In this study, we explored the protected effects of quercetin against myocardial IRI and...

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Liu X, Yu Z, Huang X, et al. Peroxisome proliferator-activated receptor γ (PPARγ) mediates the protective effect of quercetin against myocardial ischemia-reperfusion injury via suppressing the NF-κB pathway. Am J Transl Res. 2016;8(12):5169-5186
Liu, X., Yu, Z., Huang, X., Gao, Y., Wang, X., Gu, J., & Xue, S. (2016). Peroxisome proliferator-activated receptor γ (PPARγ) mediates the protective effect of quercetin against myocardial ischemia-reperfusion injury via suppressing the NF-κB pathway. American journal of translational research, 8(12), 5169-5186.
Liu, Xinyu, et al. "Peroxisome proliferator-activated receptor γ (PPARγ) mediates the protective effect of quercetin against myocardial ischemia-reperfusion injury via suppressing the NF-κB pathway." American journal of translational research vol. 8,12 (2016): 5169-5186.
Liu X, Yu Z, Huang X, Gao Y, Wang X, Gu J, Xue S. Peroxisome proliferator-activated receptor γ (PPARγ) mediates the protective effect of quercetin against myocardial ischemia-reperfusion injury via suppressing the NF-κB pathway. Am J Transl Res. 2016 Dec 15;8(12):5169-5186. eCollection 2016. PMID: 28077993; PMCID: PMC5209473.
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Tocotrienol alleviates inflammation and oxidative stress in a rat model of spinal cord injury via suppression of transforming growth factor-β.

Experimental and therapeutic medicine

Xun C, Mamat M, Guo H, Mamati P, Sheng J, Zhang J, Xu T, Liang W, Cao R, Sheng W.
PMID: 28672950
Exp Ther Med. 2017 Jul;14(1):431-438. doi: 10.3892/etm.2017.4505. Epub 2017 May 23.

In recent years accumulating evidence has indicated that tocotrienol exhibits an oxidation resistance function, decreased cholesterol function, inhibits cancer function and has unique physiological functions, including anti-inflammatory, anti-apoptotic and anti-oxidative properties. The present study investigated the effect of tocotrienols...

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Xun C, Mamat M, Guo H, et al. Tocotrienol alleviates inflammation and oxidative stress in a rat model of spinal cord injury via suppression of transforming growth factor-β. Exp Ther Med. 2017;14(1):431-438doi: 10.3892/etm.2017.4505.
Xun, C., Mamat, M., Guo, H., Mamati, P., Sheng, J., Zhang, J., Xu, T., Liang, W., Cao, R., & Sheng, W. (2017). Tocotrienol alleviates inflammation and oxidative stress in a rat model of spinal cord injury via suppression of transforming growth factor-β. Experimental and therapeutic medicine, 14(1), 431-438. https://doi.org/10.3892/etm.2017.4505
Xun, Chuanhui, et al. "Tocotrienol alleviates inflammation and oxidative stress in a rat model of spinal cord injury via suppression of transforming growth factor-β." Experimental and therapeutic medicine vol. 14,1 (2017): 431-438. doi: https://doi.org/10.3892/etm.2017.4505
Xun C, Mamat M, Guo H, Mamati P, Sheng J, Zhang J, Xu T, Liang W, Cao R, Sheng W. Tocotrienol alleviates inflammation and oxidative stress in a rat model of spinal cord injury via suppression of transforming growth factor-β. Exp Ther Med. 2017 Jul;14(1):431-438. doi: 10.3892/etm.2017.4505. Epub 2017 May 23. PMID: 28672950; PMCID: PMC5488605.
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Involvement of receptor-interacting protein 140 in palmitate-stimulated macrophage infiltration of pancreatic beta cells.

Experimental and therapeutic medicine

Zou R, Xue J, Huang Q, Dai Z, Xu Y.
PMID: 28672957
Exp Ther Med. 2017 Jul;14(1):483-494. doi: 10.3892/etm.2017.4544. Epub 2017 Jun 02.

Receptor-interacting protein 140 (RIP140) in macrophages stimulates the nuclear factor-κB subunit RelA to activate tumor necrosis factor (TNF)-α and interleukin (IL)-6 transcription. However, under lipotoxic conditions, the involvement of RIP140 in the infiltration of beta cells by macrophages remains...

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Zou R, Xue J, Huang Q, et al. Involvement of receptor-interacting protein 140 in palmitate-stimulated macrophage infiltration of pancreatic beta cells. Exp Ther Med. 2017;14(1):483-494doi: 10.3892/etm.2017.4544.
Zou, R., Xue, J., Huang, Q., Dai, Z., & Xu, Y. (2017). Involvement of receptor-interacting protein 140 in palmitate-stimulated macrophage infiltration of pancreatic beta cells. Experimental and therapeutic medicine, 14(1), 483-494. https://doi.org/10.3892/etm.2017.4544
Zou, Runmei, et al. "Involvement of receptor-interacting protein 140 in palmitate-stimulated macrophage infiltration of pancreatic beta cells." Experimental and therapeutic medicine vol. 14,1 (2017): 483-494. doi: https://doi.org/10.3892/etm.2017.4544
Zou R, Xue J, Huang Q, Dai Z, Xu Y. Involvement of receptor-interacting protein 140 in palmitate-stimulated macrophage infiltration of pancreatic beta cells. Exp Ther Med. 2017 Jul;14(1):483-494. doi: 10.3892/etm.2017.4544. Epub 2017 Jun 02. PMID: 28672957; PMCID: PMC5488400.
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Nitric oxide in gingival crevicular fluid and nitric oxide synthase expression in the gingiva of patients with sickle cell disease.

Turkish journal of haematology : official journal of Turkish Society of Haematology

Güzeldemir E, Uslu Toygar H, Bal N, Anarat R, Boğa C.
PMID: 27264125
Turk J Haematol. 2011 Jun 05;28(2):115-24. doi: 10.5152/tjh.2011.27.

OBJECTIVE: This study aimed to biochemically measure the production of nitric oxide in gingival crevicular fluid and immunohistochemically measure the expression of inducible nitric oxide synthase in the gingiva of patients with sickle cell disease. Additionally, we aimed to...

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Güzeldemir E, Uslu Toygar H, Bal N, et al. Orak hücre anemili hastaların dişetlerindeki nitrik oksit sentetaz ekspresyonu ve dişeti oluğu sıvılarındaki nitrik oksit düzeyinin değerlendirilmesi. Nitric oxide in gingival crevicular fluid and nitric oxide synthase expression in the gingiva of patients with sickle cell disease. Turk J Haematol. 2011;28(2):115-24doi: 10.5152/tjh.2011.27.
Güzeldemir, E., Uslu Toygar, H., Bal, N., Anarat, R., & Boğa, C. (2011). Orak hücre anemili hastaların dişetlerindeki nitrik oksit sentetaz ekspresyonu ve dişeti oluğu sıvılarındaki nitrik oksit düzeyinin değerlendirilmesi. Nitric oxide in gingival crevicular fluid and nitric oxide synthase expression in the gingiva of patients with sickle cell disease. Turkish journal of haematology : official journal of Turkish Society of Haematology, 28(2), 115-24. https://doi.org/10.5152/tjh.2011.27
Güzeldemir, Esra, et al. "Orak hücre anemili hastaların dişetlerindeki nitrik oksit sentetaz ekspresyonu ve dişeti oluğu sıvılarındaki nitrik oksit düzeyinin değerlendirilmesi." Nitric oxide in gingival crevicular fluid and nitric oxide synthase expression in the gingiva of patients with sickle cell disease. Turkish journal of haematology : official journal of Turkish Society of Haematology vol. 28,2 (2011): 115-24. doi: https://doi.org/10.5152/tjh.2011.27
Güzeldemir E, Uslu Toygar H, Bal N, Anarat R, Boğa C. Orak hücre anemili hastaların dişetlerindeki nitrik oksit sentetaz ekspresyonu ve dişeti oluğu sıvılarındaki nitrik oksit düzeyinin değerlendirilmesi. Nitric oxide in gingival crevicular fluid and nitric oxide synthase expression in the gingiva of patients with sickle cell disease. Turk J Haematol. 2011 Jun 05;28(2):115-24. doi: 10.5152/tjh.2011.27. PMID: 27264125.
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Exogenous erythropoietin aggravates retinal neovascularizationin a murine model of proliferative retinopathy.

Turkish journal of medical sciences

Yang N, Zhang W, He T, Xing Y.
PMID: 29152948
Turk J Med Sci. 2017 Nov 13;47(5):1642-1650. doi: 10.3906/sag-1609-49.

Background/aim: Erythropoietin (EPO) has been proven recently to be a critical mediator in retinal neovascularization (RNV). Previous studies have indicated that the use of recombinant human EPO (rEPO) is a high risk factor in the development of retinopathy of...

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Yang N, Zhang W, He T, et al. Exogenous erythropoietin aggravates retinal neovascularizationin a murine model of proliferative retinopathy. Turk J Med Sci. 2017;47(5):1642-1650doi: 10.3906/sag-1609-49.
Yang, N., Zhang, W., He, T., & Xing, Y. (2017). Exogenous erythropoietin aggravates retinal neovascularizationin a murine model of proliferative retinopathy. Turkish journal of medical sciences, 47(5), 1642-1650. https://doi.org/10.3906/sag-1609-49
Yang, Ning, et al. "Exogenous erythropoietin aggravates retinal neovascularizationin a murine model of proliferative retinopathy." Turkish journal of medical sciences vol. 47,5 (2017): 1642-1650. doi: https://doi.org/10.3906/sag-1609-49
Yang N, Zhang W, He T, Xing Y. Exogenous erythropoietin aggravates retinal neovascularizationin a murine model of proliferative retinopathy. Turk J Med Sci. 2017 Nov 13;47(5):1642-1650. doi: 10.3906/sag-1609-49. PMID: 29152948.
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