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Screening and Identification of Potential iNOS Inhibitors to Curtail Cervical Cancer Progression: an In Silico Drug Repurposing Approach.

Applied biochemistry and biotechnology

Poleboyina PK, Rampogu S, Doneti R, Pasha A, Poleboyina SM, Bhanothu S, Pasumarthi D, S D A, Kumbhakar D, Lee KW, Pawar SC.
PMID: 34705247
Appl Biochem Biotechnol. 2022 Jan;194(1):570-586. doi: 10.1007/s12010-021-03718-2. Epub 2021 Oct 27.

Cervical cancer is the second most common cause of cancer deaths in women worldwide and remains the main reason of mortality among women of reproductive age in developing countries. Nitric oxide is involved in several physiological functions inclusive of...

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Poleboyina PK, Rampogu S, Doneti R, et al. Screening and Identification of Potential iNOS Inhibitors to Curtail Cervical Cancer Progression: an In Silico Drug Repurposing Approach. Appl Biochem Biotechnol. 2021;194(1):570-586doi: 10.1007/s12010-021-03718-2.
Poleboyina, P. K., Rampogu, S., Doneti, R., Pasha, A., Poleboyina, S. M., Bhanothu, S., Pasumarthi, D., S D, A., Kumbhakar, D., Lee, K. W., & Pawar, S. C. (2022). Screening and Identification of Potential iNOS Inhibitors to Curtail Cervical Cancer Progression: an In Silico Drug Repurposing Approach. Applied biochemistry and biotechnology, 194(1), 570-586. https://doi.org/10.1007/s12010-021-03718-2
Poleboyina, Pavan Kumar, et al. "Screening and Identification of Potential iNOS Inhibitors to Curtail Cervical Cancer Progression: an In Silico Drug Repurposing Approach." Applied biochemistry and biotechnology vol. 194,1 (2022): 570-586. doi: https://doi.org/10.1007/s12010-021-03718-2
Poleboyina PK, Rampogu S, Doneti R, Pasha A, Poleboyina SM, Bhanothu S, Pasumarthi D, S D A, Kumbhakar D, Lee KW, Pawar SC. Screening and Identification of Potential iNOS Inhibitors to Curtail Cervical Cancer Progression: an In Silico Drug Repurposing Approach. Appl Biochem Biotechnol. 2022 Jan;194(1):570-586. doi: 10.1007/s12010-021-03718-2. Epub 2021 Oct 27. PMID: 34705247.
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The role of nitric oxide in renovascular hypertension: from the pathophysiology to the treatment.

Naunyn-Schmiedeberg's archives of pharmacology

Pereira BP, do Vale GT, Ceron CS.
PMID: 34994823
Naunyn Schmiedebergs Arch Pharmacol. 2022 Feb;395(2):121-131. doi: 10.1007/s00210-021-02186-z. Epub 2022 Jan 07.

Renovascular hypertension is one of the most relevant causes of secondary hypertension, mostly caused by atherosclerotic renovascular stenosis or fibromuscular dysplasia. The increase in angiotensin II production, oxidative stress, and formation of peroxynitrite promotes the decrease in nitric oxide...

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Pereira BP, do Vale GT, Ceron CS. The role of nitric oxide in renovascular hypertension: from the pathophysiology to the treatment. Naunyn Schmiedebergs Arch Pharmacol. 2022;395(2):121-131doi: 10.1007/s00210-021-02186-z.
Pereira, B. P., do Vale, G. T., & Ceron, C. S. (2022). The role of nitric oxide in renovascular hypertension: from the pathophysiology to the treatment. Naunyn-Schmiedeberg's archives of pharmacology, 395(2), 121-131. https://doi.org/10.1007/s00210-021-02186-z
Pereira, Bruna Pinheiro, et al. "The role of nitric oxide in renovascular hypertension: from the pathophysiology to the treatment." Naunyn-Schmiedeberg's archives of pharmacology vol. 395,2 (2022): 121-131. doi: https://doi.org/10.1007/s00210-021-02186-z
Pereira BP, do Vale GT, Ceron CS. The role of nitric oxide in renovascular hypertension: from the pathophysiology to the treatment. Naunyn Schmiedebergs Arch Pharmacol. 2022 Feb;395(2):121-131. doi: 10.1007/s00210-021-02186-z. Epub 2022 Jan 07. PMID: 34994823.
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Strategic Modification of Gut Microbiota through Oral Bacteriotherapy Influences Hypoxia Inducible Factor-1α: Therapeutic Implication in Alzheimer's Disease.

International journal of molecular sciences

Bonfili L, Gong C, Lombardi F, Cifone MG, Eleuteri AM.
PMID: 35008786
Int J Mol Sci. 2021 Dec 29;23(1). doi: 10.3390/ijms23010357.

Dysbiosis contributes to Alzheimer's disease (AD) pathogenesis, and oral bacteriotherapy represents a promising preventative and therapeutic opportunity to remodel gut microbiota and to delay AD onset and progression by reducing neuroinflammation and amyloid and tau proteins aggregation. Specifically, SLAB51...

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Bonfili L, Gong C, Lombardi F, et al. Strategic Modification of Gut Microbiota through Oral Bacteriotherapy Influences Hypoxia Inducible Factor-1α: Therapeutic Implication in Alzheimer's Disease. Int J Mol Sci. 2021;23(1)doi: 10.3390/ijms23010357.
Bonfili, L., Gong, C., Lombardi, F., Cifone, M. G., & Eleuteri, A. M. (2021). Strategic Modification of Gut Microbiota through Oral Bacteriotherapy Influences Hypoxia Inducible Factor-1α: Therapeutic Implication in Alzheimer's Disease. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010357
Bonfili, Laura, et al. "Strategic Modification of Gut Microbiota through Oral Bacteriotherapy Influences Hypoxia Inducible Factor-1α: Therapeutic Implication in Alzheimer's Disease." International journal of molecular sciences vol. 23,1 (2021). doi: https://doi.org/10.3390/ijms23010357
Bonfili L, Gong C, Lombardi F, Cifone MG, Eleuteri AM. Strategic Modification of Gut Microbiota through Oral Bacteriotherapy Influences Hypoxia Inducible Factor-1α: Therapeutic Implication in Alzheimer's Disease. Int J Mol Sci. 2021 Dec 29;23(1). doi: 10.3390/ijms23010357. PMID: 35008786.
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Inflammatory Mechanisms Contributing to Endothelial Dysfunction.

Biomedicines

Theofilis P, Sagris M, Oikonomou E, Antonopoulos AS, Siasos G, Tsioufis C, Tousoulis D.
PMID: 34356845
Biomedicines. 2021 Jul 06;9(7). doi: 10.3390/biomedicines9070781.

Maintenance of endothelial cell integrity is an important component of human health and disease since the endothelium can perform various functions including regulation of vascular tone, control of hemostasis and thrombosis, cellular adhesion, smooth muscle cell proliferation, and vascular...

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Theofilis P, Sagris M, Oikonomou E, et al. Inflammatory Mechanisms Contributing to Endothelial Dysfunction. Biomedicines. 2021;9(7)doi: 10.3390/biomedicines9070781.
Theofilis, P., Sagris, M., Oikonomou, E., Antonopoulos, A. S., Siasos, G., Tsioufis, C., & Tousoulis, D. (2021). Inflammatory Mechanisms Contributing to Endothelial Dysfunction. Biomedicines, 9(7), . https://doi.org/10.3390/biomedicines9070781
Theofilis, Panagiotis, et al. "Inflammatory Mechanisms Contributing to Endothelial Dysfunction." Biomedicines vol. 9,7 (2021). doi: https://doi.org/10.3390/biomedicines9070781
Theofilis P, Sagris M, Oikonomou E, Antonopoulos AS, Siasos G, Tsioufis C, Tousoulis D. Inflammatory Mechanisms Contributing to Endothelial Dysfunction. Biomedicines. 2021 Jul 06;9(7). doi: 10.3390/biomedicines9070781. PMID: 34356845; PMCID: PMC8301477.
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Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease.

Frontiers in pharmacology

Caruso G, Grasso M, Fidilio A, Torrisi SA, Musso N, Geraci F, Tropea MR, Privitera A, Tascedda F, Puzzo D, Salomone S, Drago F, Leggio GM, Caraci F.
PMID: 35002742
Front Pharmacol. 2021 Dec 24;12:809541. doi: 10.3389/fphar.2021.809541. eCollection 2021.

Depression is a risk factor for the development of Alzheimer's disease (AD). A neurobiological and clinical continuum exists between AD and depression, with neuroinflammation and oxidative stress being involved in both diseases. Second-generation antidepressants, in particular selective serotonin reuptake...

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Caruso G, Grasso M, Fidilio A, et al. Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease. Front Pharmacol. 2021;12:809541doi: 10.3389/fphar.2021.809541.
Caruso, G., Grasso, M., Fidilio, A., Torrisi, S. A., Musso, N., Geraci, F., Tropea, M. R., Privitera, A., Tascedda, F., Puzzo, D., Salomone, S., Drago, F., Leggio, G. M., & Caraci, F. (2021). Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease. Frontiers in pharmacology, 12809541. https://doi.org/10.3389/fphar.2021.809541
Caruso, Giuseppe, et al. "Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease." Frontiers in pharmacology vol. 12 (2021): 809541. doi: https://doi.org/10.3389/fphar.2021.809541
Caruso G, Grasso M, Fidilio A, Torrisi SA, Musso N, Geraci F, Tropea MR, Privitera A, Tascedda F, Puzzo D, Salomone S, Drago F, Leggio GM, Caraci F. Antioxidant Activity of Fluoxetine and Vortioxetine in a Non-Transgenic Animal Model of Alzheimer's Disease. Front Pharmacol. 2021 Dec 24;12:809541. doi: 10.3389/fphar.2021.809541. eCollection 2021. PMID: 35002742; PMCID: PMC8740153.
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An updated systematic review and meta-analysis of the effect of statins on asymmetric dimethylarginine.

Nitric oxide : biology and chemistry

Zinellu A, Mangoni AA.
PMID: 35032642
Nitric Oxide. 2022 Jan 12; doi: 10.1016/j.niox.2022.01.001. Epub 2022 Jan 12.

OBJECTIVES: We conducted an updated systematic review and meta-analysis of the effect of statins on serum or plasma concentrations of the endogenous inhibitor of endothelial nitric oxide synthase, asymmetric NMETHODS: A systematic literature search was conducted in the electronic...

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Zinellu A, Mangoni AA. An updated systematic review and meta-analysis of the effect of statins on asymmetric dimethylarginine. Nitric Oxide. 2022;doi: 10.1016/j.niox.2022.01.001.
Zinellu, A., & Mangoni, A. A. (2022). An updated systematic review and meta-analysis of the effect of statins on asymmetric dimethylarginine. Nitric oxide : biology and chemistry, . https://doi.org/10.1016/j.niox.2022.01.001
Zinellu, Angelo, and Mangoni, Arduino A. "An updated systematic review and meta-analysis of the effect of statins on asymmetric dimethylarginine." Nitric oxide : biology and chemistry vol. (2022). doi: https://doi.org/10.1016/j.niox.2022.01.001
Zinellu A, Mangoni AA. An updated systematic review and meta-analysis of the effect of statins on asymmetric dimethylarginine. Nitric Oxide. 2022 Jan 12; doi: 10.1016/j.niox.2022.01.001. Epub 2022 Jan 12. PMID: 35032642.
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Crocin Improves Endothelial Mitochondrial Dysfunction .

Frontiers in cell and developmental biology

Li X, Liu Y, Cao A, Li C, Wang L, Wu Q, Li X, Lv X, Zhu J, Chun H, Laba C, Du X, Zhang Y, Yang H.
PMID: 33777959
Front Cell Dev Biol. 2021 Mar 12;9:651434. doi: 10.3389/fcell.2021.651434. eCollection 2021.

Mitochondrial dysfunction contributes to excessive reactive oxygen species (ROS) generation, which is a dramatic cause to promote endothelial dysfunction in diabetes. It was previously demonstrated that crocin protected the endothelium based on its diverse medicinal properties, but its effect...

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Li X, Liu Y, Cao A, et al. Crocin Improves Endothelial Mitochondrial Dysfunction . Front Cell Dev Biol. 2021;9:651434doi: 10.3389/fcell.2021.651434.
Li, X., Liu, Y., Cao, A., Li, C., Wang, L., Wu, Q., Li, X., Lv, X., Zhu, J., Chun, H., Laba, C., Du, X., Zhang, Y., & Yang, H. (2021). Crocin Improves Endothelial Mitochondrial Dysfunction . Frontiers in cell and developmental biology, 9651434. https://doi.org/10.3389/fcell.2021.651434
Li, Xuemei, et al. "Crocin Improves Endothelial Mitochondrial Dysfunction ." Frontiers in cell and developmental biology vol. 9 (2021): 651434. doi: https://doi.org/10.3389/fcell.2021.651434
Li X, Liu Y, Cao A, Li C, Wang L, Wu Q, Li X, Lv X, Zhu J, Chun H, Laba C, Du X, Zhang Y, Yang H. Crocin Improves Endothelial Mitochondrial Dysfunction . Front Cell Dev Biol. 2021 Mar 12;9:651434. doi: 10.3389/fcell.2021.651434. eCollection 2021. PMID: 33777959; PMCID: PMC7994751.
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Adrenergic Blockade by Nebivolol to Suppress Oral Squamous Cell Carcinoma Growth .

Frontiers in pharmacology

Chen Q, Jiang H, Wang Z, Cai LY, Jiang YC, Xie L, Zhou Y, Zeng X, Ji N, Shen YQ, Chen QM.
PMID: 34456721
Front Pharmacol. 2021 Aug 12;12:691998. doi: 10.3389/fphar.2021.691998. eCollection 2021.

Adrenergic nerve fibers in the tumor microenvironment promote tumor growth and represent a potential target for cancer therapy. However, the effectiveness of targeting adrenergic nerve fibers for oral squamous cell carcinoma (OSCC) therapy needs to be evaluated by preclinical...

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Chen Q, Jiang H, Wang Z, et al. Adrenergic Blockade by Nebivolol to Suppress Oral Squamous Cell Carcinoma Growth . Front Pharmacol. 2021;12:691998doi: 10.3389/fphar.2021.691998.
Chen, Q., Jiang, H., Wang, Z., Cai, L. Y., Jiang, Y. C., Xie, L., Zhou, Y., Zeng, X., Ji, N., Shen, Y. Q., & Chen, Q. M. (2021). Adrenergic Blockade by Nebivolol to Suppress Oral Squamous Cell Carcinoma Growth . Frontiers in pharmacology, 12691998. https://doi.org/10.3389/fphar.2021.691998
Chen, Qian, et al. "Adrenergic Blockade by Nebivolol to Suppress Oral Squamous Cell Carcinoma Growth ." Frontiers in pharmacology vol. 12 (2021): 691998. doi: https://doi.org/10.3389/fphar.2021.691998
Chen Q, Jiang H, Wang Z, Cai LY, Jiang YC, Xie L, Zhou Y, Zeng X, Ji N, Shen YQ, Chen QM. Adrenergic Blockade by Nebivolol to Suppress Oral Squamous Cell Carcinoma Growth . Front Pharmacol. 2021 Aug 12;12:691998. doi: 10.3389/fphar.2021.691998. eCollection 2021. PMID: 34456721; PMCID: PMC8387679.
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Showing 3469 to 3476 of 3476 entries