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Methylene blue treatment for cytokine release syndrome-associated vasoplegia following a renal transplant with rATG infusion: A case report and literature review.

Experimental and therapeutic medicine

Denny JT, Burr AT, Balzer F, Tse JT, Denny JE, Chyu D.
PMID: 26136914
Exp Ther Med. 2015 May;9(5):1915-1920. doi: 10.3892/etm.2015.2349. Epub 2015 Mar 12.

Rabbit anti-thymocyte globulin (rATG) is an infusion of polyclonal rabbit-derived antibodies against human thymocyte markers, which can be used to prevent and treat acute rejection following organ transplantation. However, the product monograph issued by the manufacturer (Sanofi Canada) reports...

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Denny JT, Burr AT, Balzer F, et al. Methylene blue treatment for cytokine release syndrome-associated vasoplegia following a renal transplant with rATG infusion: A case report and literature review. Exp Ther Med. 2015;9(5):1915-1920doi: 10.3892/etm.2015.2349.
Denny, J. T., Burr, A. T., Balzer, F., Tse, J. T., Denny, J. E., & Chyu, D. (2015). Methylene blue treatment for cytokine release syndrome-associated vasoplegia following a renal transplant with rATG infusion: A case report and literature review. Experimental and therapeutic medicine, 9(5), 1915-1920. https://doi.org/10.3892/etm.2015.2349
Denny, John T, et al. "Methylene blue treatment for cytokine release syndrome-associated vasoplegia following a renal transplant with rATG infusion: A case report and literature review." Experimental and therapeutic medicine vol. 9,5 (2015): 1915-1920. doi: https://doi.org/10.3892/etm.2015.2349
Denny JT, Burr AT, Balzer F, Tse JT, Denny JE, Chyu D. Methylene blue treatment for cytokine release syndrome-associated vasoplegia following a renal transplant with rATG infusion: A case report and literature review. Exp Ther Med. 2015 May;9(5):1915-1920. doi: 10.3892/etm.2015.2349. Epub 2015 Mar 12. PMID: 26136914; PMCID: PMC4471690.
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Synergistic Inhibition of Tumor Necrosis Factor-Alpha-Stimulated Pro-Inflammatory Cytokine Expression in HaCaT Cells by a Combination of Rapamycin and Mycophenolic Acid.

Annals of dermatology

Kim MY, Lim YY, Kim HM, Park YM, Kang H, Kim BJ.
PMID: 25673929
Ann Dermatol. 2015 Feb;27(1):32-9. doi: 10.5021/ad.2015.27.1.32. Epub 2015 Feb 03.

BACKGROUND: Keratinocytes release various pro-inflammatory cytokines, chemokines, and adhesion molecules such as intercellular adhesion molecule 1 (ICAM-1) in response to cytokines such as tumor necrosis factor (TNF)-α and interferon (IFN)-γ. Rapamycin and mycophenolic acid (MPA) have potent immunosuppressive activity...

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Kim MY, Lim YY, Kim HM, et al. Synergistic Inhibition of Tumor Necrosis Factor-Alpha-Stimulated Pro-Inflammatory Cytokine Expression in HaCaT Cells by a Combination of Rapamycin and Mycophenolic Acid. Ann Dermatol. 2015;27(1):32-9doi: 10.5021/ad.2015.27.1.32.
Kim, M. Y., Lim, Y. Y., Kim, H. M., Park, Y. M., Kang, H., & Kim, B. J. (2015). Synergistic Inhibition of Tumor Necrosis Factor-Alpha-Stimulated Pro-Inflammatory Cytokine Expression in HaCaT Cells by a Combination of Rapamycin and Mycophenolic Acid. Annals of dermatology, 27(1), 32-9. https://doi.org/10.5021/ad.2015.27.1.32
Kim, Min Young, et al. "Synergistic Inhibition of Tumor Necrosis Factor-Alpha-Stimulated Pro-Inflammatory Cytokine Expression in HaCaT Cells by a Combination of Rapamycin and Mycophenolic Acid." Annals of dermatology vol. 27,1 (2015): 32-9. doi: https://doi.org/10.5021/ad.2015.27.1.32
Kim MY, Lim YY, Kim HM, Park YM, Kang H, Kim BJ. Synergistic Inhibition of Tumor Necrosis Factor-Alpha-Stimulated Pro-Inflammatory Cytokine Expression in HaCaT Cells by a Combination of Rapamycin and Mycophenolic Acid. Ann Dermatol. 2015 Feb;27(1):32-9. doi: 10.5021/ad.2015.27.1.32. Epub 2015 Feb 03. PMID: 25673929; PMCID: PMC4323600.
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Improvement of thoracic aortic vasoreactivity by continuous and intermittent exercise in high-fat diet-induced obese rats.

Biomedical reports

Liu H, Yang Z, Hu J, Luo Y, Zhu L, Yang H, Li G.
PMID: 26171160
Biomed Rep. 2015 Jul;3(4):527-532. doi: 10.3892/br.2015.451. Epub 2015 Apr 16.

The aim of the present study was to explore the effects of continuous and intermittent exercise on the thoracic aortic vasoreactivity and free radical metabolism in rats fed with a high-fat diet (HD). Sprague-Dawley (SD) rats were randomly divided...

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Liu H, Yang Z, Hu J, et al. Improvement of thoracic aortic vasoreactivity by continuous and intermittent exercise in high-fat diet-induced obese rats. Biomed Rep. 2015;3(4):527-532doi: 10.3892/br.2015.451.
Liu, H., Yang, Z., Hu, J., Luo, Y., Zhu, L., Yang, H., & Li, G. (2015). Improvement of thoracic aortic vasoreactivity by continuous and intermittent exercise in high-fat diet-induced obese rats. Biomedical reports, 3(4), 527-532. https://doi.org/10.3892/br.2015.451
Liu, Hongpeng, et al. "Improvement of thoracic aortic vasoreactivity by continuous and intermittent exercise in high-fat diet-induced obese rats." Biomedical reports vol. 3,4 (2015): 527-532. doi: https://doi.org/10.3892/br.2015.451
Liu H, Yang Z, Hu J, Luo Y, Zhu L, Yang H, Li G. Improvement of thoracic aortic vasoreactivity by continuous and intermittent exercise in high-fat diet-induced obese rats. Biomed Rep. 2015 Jul;3(4):527-532. doi: 10.3892/br.2015.451. Epub 2015 Apr 16. PMID: 26171160; PMCID: PMC4486789.
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Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages.

Chinese medicine

Peng XX, Zhang SH, Wang XL, Ye TJ, Li H, Yan XF, Wei L, Wu ZP, Hu J, Zou CP, Wang YH, Hu XD.
PMID: 26155304
Chin Med. 2015 Jul 01;10:15. doi: 10.1186/s13020-015-0045-x. eCollection 2015.

BACKGROUND: Panax Notoginseng flower saponins (PNFS) are the main active component of Panax notoginseng (Burk) F. H. Chen flower bud (PNF) and possess significant anti-inflammatory efficacy. This study aims to explore the mechanisms underlying PNFS' antiflammatory action in RAW264.7...

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Peng XX, Zhang SH, Wang XL, et al. Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chin Med. 2015;10:15doi: 10.1186/s13020-015-0045-x.
Peng, X. X., Zhang, S. H., Wang, X. L., Ye, T. J., Li, H., Yan, X. F., Wei, L., Wu, Z. P., Hu, J., Zou, C. P., Wang, Y. H., & Hu, X. D. (2015). Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chinese medicine, 1015. https://doi.org/10.1186/s13020-015-0045-x
Peng, Xiao-Xu, et al. "Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages." Chinese medicine vol. 10 (2015): 15. doi: https://doi.org/10.1186/s13020-015-0045-x
Peng XX, Zhang SH, Wang XL, Ye TJ, Li H, Yan XF, Wei L, Wu ZP, Hu J, Zou CP, Wang YH, Hu XD. Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chin Med. 2015 Jul 01;10:15. doi: 10.1186/s13020-015-0045-x. eCollection 2015. PMID: 26155304; PMCID: PMC4493833.
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Allele-specific transcriptional activity of the variable number of tandem repeats of the inducible nitric oxide synthase gene is associated with idiopathic achalasia.

United European gastroenterology journal

Sarnelli G, Grosso M, Palumbo I, Pesce M, D'Alessandro A, Zaninotto G, Annese V, Petruzzelli R, Izzo P, Sepulveres R, Bruzzese D, Esposito G, Cuomo R.
PMID: 28344787
United European Gastroenterol J. 2017 Mar;5(2):200-207. doi: 10.1177/2050640616648870. Epub 2016 Jul 08.

BACKGROUND: Polymorphisms of genes involved in the regulation of the immune response are risk factors for achalasia, but their contribution to disease pathogenesis is unknown. Nitric oxide is involved both in immune function and inhibitory neurotransmission.OBJECTIVE: The objective of...

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Sarnelli G, Grosso M, Palumbo I, et al. Allele-specific transcriptional activity of the variable number of tandem repeats of the inducible nitric oxide synthase gene is associated with idiopathic achalasia. United European Gastroenterol J. 2016;5(2):200-207doi: 10.1177/2050640616648870.
Sarnelli, G., Grosso, M., Palumbo, I., Pesce, M., D'Alessandro, A., Zaninotto, G., Annese, V., Petruzzelli, R., Izzo, P., Sepulveres, R., Bruzzese, D., Esposito, G., & Cuomo, R. (2017). Allele-specific transcriptional activity of the variable number of tandem repeats of the inducible nitric oxide synthase gene is associated with idiopathic achalasia. United European gastroenterology journal, 5(2), 200-207. https://doi.org/10.1177/2050640616648870
Sarnelli, Giovanni, et al. "Allele-specific transcriptional activity of the variable number of tandem repeats of the inducible nitric oxide synthase gene is associated with idiopathic achalasia." United European gastroenterology journal vol. 5,2 (2017): 200-207. doi: https://doi.org/10.1177/2050640616648870
Sarnelli G, Grosso M, Palumbo I, Pesce M, D'Alessandro A, Zaninotto G, Annese V, Petruzzelli R, Izzo P, Sepulveres R, Bruzzese D, Esposito G, Cuomo R. Allele-specific transcriptional activity of the variable number of tandem repeats of the inducible nitric oxide synthase gene is associated with idiopathic achalasia. United European Gastroenterol J. 2017 Mar;5(2):200-207. doi: 10.1177/2050640616648870. Epub 2016 Jul 08. PMID: 28344787; PMCID: PMC5349359.
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Isolation and in vitro culture of vascular endothelial cells from mice.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Choi S, Kim JA, Kim KC, Suh SH.
PMID: 25605995
Korean J Physiol Pharmacol. 2015 Jan;19(1):35-42. doi: 10.4196/kjpp.2015.19.1.35. Epub 2014 Dec 31.

In cardiovascular disorders, understanding of endothelial cell (EC) function is essential to elucidate the disease mechanism. Although the mouse model has many advantages for in vivo and in vitro research, efficient procedures for the isolation and propagation of primary...

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Choi S, Kim JA, Kim KC, et al. Isolation and in vitro culture of vascular endothelial cells from mice. Korean J Physiol Pharmacol. 2014;19(1):35-42doi: 10.4196/kjpp.2015.19.1.35.
Choi, S., Kim, J. A., Kim, K. C., & Suh, S. H. (2015). Isolation and in vitro culture of vascular endothelial cells from mice. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 19(1), 35-42. https://doi.org/10.4196/kjpp.2015.19.1.35
Choi, Shinkyu, et al. "Isolation and in vitro culture of vascular endothelial cells from mice." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 19,1 (2015): 35-42. doi: https://doi.org/10.4196/kjpp.2015.19.1.35
Choi S, Kim JA, Kim KC, Suh SH. Isolation and in vitro culture of vascular endothelial cells from mice. Korean J Physiol Pharmacol. 2015 Jan;19(1):35-42. doi: 10.4196/kjpp.2015.19.1.35. Epub 2014 Dec 31. PMID: 25605995; PMCID: PMC4297760.
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Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity.

Physiological reports

Ali MI, Chen X, Didion SP.
PMID: 26660551
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12630.

Heterozygous endothelial nitric oxide synthase (eNOS) deficiency is associated with normal endothelium-dependent responses, however, little is known regarding the mechanisms that maintain or impair endothelial function with heterozygous eNOS deficiency. The goals of this study were to (1) determine...

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Ali MI, Chen X, Didion SP. Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12630.
Ali, M. I., Chen, X., & Didion, S. P. (2015). Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12630
Ali, M Irfan, et al. "Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12630
Ali MI, Chen X, Didion SP. Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12630. PMID: 26660551; PMCID: PMC4760452.
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Inhibition of neuronal nitric oxide synthase improves microregional O.

Neurological research

Chi OZ, Rah KH, Barsoum S, Liu X, Weiss HR.
PMID: 26119517
Neurol Res. 2015 Jun 29; doi: 10.1179/1743132815Y.0000000062. Epub 2015 Jun 29.

Objectives Return of regional cerebral blood flow (rCBF) in focal cerebral ischaemia may not ensure proper distribution of blood flow to meet metabolic demand. This study was performed to determine how inhibition of neuronal nitric oxide synthase (NOS) during...

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Chi OZ, Rah KH, Barsoum S, et al. Inhibition of neuronal nitric oxide synthase improves microregional O. Neurol Res. 2015;doi: 10.1179/1743132815Y.0000000062.
Chi, O. Z., Rah, K. H., Barsoum, S., Liu, X., & Weiss, H. R. (2015). Inhibition of neuronal nitric oxide synthase improves microregional O. Neurological research, . https://doi.org/10.1179/1743132815Y.0000000062
Chi, Oak Z, et al. "Inhibition of neuronal nitric oxide synthase improves microregional O." Neurological research vol. (2015). doi: https://doi.org/10.1179/1743132815Y.0000000062
Chi OZ, Rah KH, Barsoum S, Liu X, Weiss HR. Inhibition of neuronal nitric oxide synthase improves microregional O. Neurol Res. 2015 Jun 29; doi: 10.1179/1743132815Y.0000000062. Epub 2015 Jun 29. PMID: 26119517.
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Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity.

Frontiers in immunology

Rodriguez PC, Ochoa AC, Al-Khami AA.
PMID: 28223985
Front Immunol. 2017 Feb 07;8:93. doi: 10.3389/fimmu.2017.00093. eCollection 2017.

Arginine metabolism has been a key catabolic and anabolic process throughout the evolution of the immune response. Accruing evidence indicates that arginine-catabolizing enzymes, mainly nitric oxide synthases and arginases, are closely integrated with the control of immune response under...

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Rodriguez PC, Ochoa AC, Al-Khami AA. Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity. Front Immunol. 2017;8:93doi: 10.3389/fimmu.2017.00093.
Rodriguez, P. C., Ochoa, A. C., & Al-Khami, A. A. (2017). Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity. Frontiers in immunology, 893. https://doi.org/10.3389/fimmu.2017.00093
Rodriguez, Paulo C, et al. "Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity." Frontiers in immunology vol. 8 (2017): 93. doi: https://doi.org/10.3389/fimmu.2017.00093
Rodriguez PC, Ochoa AC, Al-Khami AA. Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity. Front Immunol. 2017 Feb 07;8:93. doi: 10.3389/fimmu.2017.00093. eCollection 2017. PMID: 28223985; PMCID: PMC5293781.
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Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension.

Hepatology international

Rizvi MR, Tauseef M, Shahid M, Babbar R, Fahim M, Sakhuja P, Sarin SK.
PMID: 26201795
Hepatol Int. 2013 Jun;7(2):622-35. doi: 10.1007/s12072-012-9397-9. Epub 2012 Sep 02.

AIMS: To evaluate the participation of nitric oxide (NO) and prostaglandin (PGI2) on hyperdynamic state in endotoxemia-induced portal hypertension (EIP) induced by chronic endotoxemia.METHODS: The portal pressure (PP) and mean arterial pressure (MAP) were recorded, in vivo before and...

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Rizvi MR, Tauseef M, Shahid M, et al. Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension. Hepatol Int. 2012;7(2):622-35doi: 10.1007/s12072-012-9397-9.
Rizvi, M. R., Tauseef, M., Shahid, M., Babbar, R., Fahim, M., Sakhuja, P., & Sarin, S. K. (2013). Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension. Hepatology international, 7(2), 622-35. https://doi.org/10.1007/s12072-012-9397-9
Rizvi, Moattar Raza, et al. "Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension." Hepatology international vol. 7,2 (2013): 622-35. doi: https://doi.org/10.1007/s12072-012-9397-9
Rizvi MR, Tauseef M, Shahid M, Babbar R, Fahim M, Sakhuja P, Sarin SK. Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension. Hepatol Int. 2013 Jun;7(2):622-35. doi: 10.1007/s12072-012-9397-9. Epub 2012 Sep 02. PMID: 26201795.
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Effects of tetrahydrobiopterin oral treatment in hypoxia-induced pulmonary hypertension in rat.

Pulmonary circulation

Francis BN, Hale A, Channon KM, Wilkins MR, Zhao L.
PMID: 25621160
Pulm Circ. 2014 Sep;4(3):462-70. doi: 10.1086/677361.

Endothelial nitric oxide synthase (eNOS) plays a major role in maintaining pulmonary vascular homeostasis. Tetrahydrobiopterin (BH4), an essential cofactor that stabilizes the dimerization of eNOS and balances nitric oxide (NO) and superoxide production, may have therapeutic potential in pulmonary...

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Francis BN, Hale A, Channon KM, et al. Effects of tetrahydrobiopterin oral treatment in hypoxia-induced pulmonary hypertension in rat. Pulm Circ. 2014;4(3):462-70doi: 10.1086/677361.
Francis, B. N., Hale, A., Channon, K. M., Wilkins, M. R., & Zhao, L. (2014). Effects of tetrahydrobiopterin oral treatment in hypoxia-induced pulmonary hypertension in rat. Pulmonary circulation, 4(3), 462-70. https://doi.org/10.1086/677361
Francis, Bahaa N, et al. "Effects of tetrahydrobiopterin oral treatment in hypoxia-induced pulmonary hypertension in rat." Pulmonary circulation vol. 4,3 (2014): 462-70. doi: https://doi.org/10.1086/677361
Francis BN, Hale A, Channon KM, Wilkins MR, Zhao L. Effects of tetrahydrobiopterin oral treatment in hypoxia-induced pulmonary hypertension in rat. Pulm Circ. 2014 Sep;4(3):462-70. doi: 10.1086/677361. PMID: 25621160; PMCID: PMC4278606.
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Chronic Stress Decreases Cerebrovascular Responses During Rat Hindlimb Electrical Stimulation.

Frontiers in neuroscience

Lee S, Kang BM, Shin MK, Min J, Heo C, Lee Y, Baeg E, Suh M.
PMID: 26778944
Front Neurosci. 2015 Dec 23;9:462. doi: 10.3389/fnins.2015.00462. eCollection 2015.

Repeated stress is one of the major risk factors for cerebrovascular disease, including stroke, and vascular dementia. However, the functional alterations in the cerebral hemodynamic response induced by chronic stress have not been clarified. Here, we investigated the in...

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Lee S, Kang BM, Shin MK, et al. Chronic Stress Decreases Cerebrovascular Responses During Rat Hindlimb Electrical Stimulation. Front Neurosci. 2015;9:462doi: 10.3389/fnins.2015.00462.
Lee, S., Kang, B. M., Shin, M. K., Min, J., Heo, C., Lee, Y., Baeg, E., & Suh, M. (2015). Chronic Stress Decreases Cerebrovascular Responses During Rat Hindlimb Electrical Stimulation. Frontiers in neuroscience, 9462. https://doi.org/10.3389/fnins.2015.00462
Lee, Sohee, et al. "Chronic Stress Decreases Cerebrovascular Responses During Rat Hindlimb Electrical Stimulation." Frontiers in neuroscience vol. 9 (2015): 462. doi: https://doi.org/10.3389/fnins.2015.00462
Lee S, Kang BM, Shin MK, Min J, Heo C, Lee Y, Baeg E, Suh M. Chronic Stress Decreases Cerebrovascular Responses During Rat Hindlimb Electrical Stimulation. Front Neurosci. 2015 Dec 23;9:462. doi: 10.3389/fnins.2015.00462. eCollection 2015. PMID: 26778944; PMCID: PMC4688360.
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