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Expression of Concern: Protein kinase D activity controls endothelial nitric oxide synthesis. J. Cell Sci. doi: 10.1242/jcs.148601.

Journal of cell science

Aicart-Ramos C, Sánchez-Ruiloba L, Gómez-Parrizas M, Zaragoza C, Iglesias T, Rodríguez-Crespo I.
PMID: 28855382
J Cell Sci. 2017 Oct 01;130(19):3417. doi: 10.1242/jcs.210005. Epub 2017 Aug 30.

No abstract available.

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Aicart-Ramos C, Sánchez-Ruiloba L, Gómez-Parrizas M, et al. Expression of Concern: Protein kinase D activity controls endothelial nitric oxide synthesis. J. Cell Sci. doi: 10.1242/jcs.148601. J Cell Sci. 2017;130(19):3417doi: 10.1242/jcs.210005.
Aicart-Ramos, C., Sánchez-Ruiloba, L., Gómez-Parrizas, M., Zaragoza, C., Iglesias, T., & Rodríguez-Crespo, I. (2017). Expression of Concern: Protein kinase D activity controls endothelial nitric oxide synthesis. J. Cell Sci. doi: 10.1242/jcs.148601. Journal of cell science, 130(19), 3417. https://doi.org/10.1242/jcs.210005
Aicart-Ramos, Clara, et al. "Expression of Concern: Protein kinase D activity controls endothelial nitric oxide synthesis. J. Cell Sci. doi: 10.1242/jcs.148601." Journal of cell science vol. 130,19 (2017): 3417. doi: https://doi.org/10.1242/jcs.210005
Aicart-Ramos C, Sánchez-Ruiloba L, Gómez-Parrizas M, Zaragoza C, Iglesias T, Rodríguez-Crespo I. Expression of Concern: Protein kinase D activity controls endothelial nitric oxide synthesis. J. Cell Sci. doi: 10.1242/jcs.148601. J Cell Sci. 2017 Oct 01;130(19):3417. doi: 10.1242/jcs.210005. Epub 2017 Aug 30. PMID: 28855382.
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Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy.

Pulmonary circulation

Mohamed NA, Davies RP, Lickiss PD, Ahmetaj-Shala B, Reed DM, Gashaw HH, Saleem H, Freeman GR, George PM, Wort SJ, Morales-Cano D, Barreira B, Tetley TD, Chester AH, Yacoub MH, Kirkby NS, Moreno L, Mitchell JA.
PMID: 28447910
Pulm Circ. 2017 Jul-Sep;7(3):643-653. doi: 10.1177/2045893217710224. Epub 2017 Jun 27.

Pulmonary arterial hypertension (PAH) is a progressive and debilitating condition. Despite promoting vasodilation, current drugs have a therapeutic window within which they are limited by systemic side effects. Nanomedicine uses nanoparticles to improve drug delivery and/or reduce side effects....

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Mohamed NA, Davies RP, Lickiss PD, et al. Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy. Pulm Circ. 2017;7(3):643-653doi: 10.1177/2045893217710224.
Mohamed, N. A., Davies, R. P., Lickiss, P. D., Ahmetaj-Shala, B., Reed, D. M., Gashaw, H. H., Saleem, H., Freeman, G. R., George, P. M., Wort, S. J., Morales-Cano, D., Barreira, B., Tetley, T. D., Chester, A. H., Yacoub, M. H., Kirkby, N. S., Moreno, L., & Mitchell, J. A. (2017). Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy. Pulmonary circulation, 7(3), 643-653. https://doi.org/10.1177/2045893217710224
Mohamed, Nura A, et al. "Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy." Pulmonary circulation vol. 7,3 (2017): 643-653. doi: https://doi.org/10.1177/2045893217710224
Mohamed NA, Davies RP, Lickiss PD, Ahmetaj-Shala B, Reed DM, Gashaw HH, Saleem H, Freeman GR, George PM, Wort SJ, Morales-Cano D, Barreira B, Tetley TD, Chester AH, Yacoub MH, Kirkby NS, Moreno L, Mitchell JA. Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy. Pulm Circ. 2017 Jul-Sep;7(3):643-653. doi: 10.1177/2045893217710224. Epub 2017 Jun 27. PMID: 28447910; PMCID: PMC5841901.
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Pre-operative Hepatic Artery Resistive Index is a Non-invasive Predictive Indicator of Prognosis in Biliary Atresia.

Journal of Indian Association of Pediatric Surgeons

Mittal D, Bhatnagar V, Agarwala S, Srinivas M, Jana M, Gupta AK, Das N, Singh MK.
PMID: 28974877
J Indian Assoc Pediatr Surg. 2017 Oct-Dec;22(4):237-241. doi: 10.4103/jiaps.JIAPS_103_17.

AIMS: The aim of this study is to evaluate hepatic artery resistive index (HARI) as a noninvasive prognostic predictor by correlating it with peripheral blood nitric oxide (NO) levels, portal pressure (PP) and histopathological changes in the liver in...

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Mittal D, Bhatnagar V, Agarwala S, et al. Pre-operative Hepatic Artery Resistive Index is a Non-invasive Predictive Indicator of Prognosis in Biliary Atresia. J Indian Assoc Pediatr Surg. 2017;22(4):237-241doi: 10.4103/jiaps.JIAPS_103_17.
Mittal, D., Bhatnagar, V., Agarwala, S., Srinivas, M., Jana, M., Gupta, A. K., Das, N., & Singh, M. K. (2017). Pre-operative Hepatic Artery Resistive Index is a Non-invasive Predictive Indicator of Prognosis in Biliary Atresia. Journal of Indian Association of Pediatric Surgeons, 22(4), 237-241. https://doi.org/10.4103/jiaps.JIAPS_103_17
Mittal, Deepak, et al. "Pre-operative Hepatic Artery Resistive Index is a Non-invasive Predictive Indicator of Prognosis in Biliary Atresia." Journal of Indian Association of Pediatric Surgeons vol. 22,4 (2017): 237-241. doi: https://doi.org/10.4103/jiaps.JIAPS_103_17
Mittal D, Bhatnagar V, Agarwala S, Srinivas M, Jana M, Gupta AK, Das N, Singh MK. Pre-operative Hepatic Artery Resistive Index is a Non-invasive Predictive Indicator of Prognosis in Biliary Atresia. J Indian Assoc Pediatr Surg. 2017 Oct-Dec;22(4):237-241. doi: 10.4103/jiaps.JIAPS_103_17. PMID: 28974877; PMCID: PMC5615899.
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Toxicological Evaluation of Emblica officinalis Fruit Extract and its Anti-inflammatory and Free Radical Scavenging Properties.

Pharmacognosy magazine

Middha SK, Goyal AK, Lokesh P, Yardi V, Mojamdar L, Keni DS, Babu D, Usha T.
PMID: 26929577
Pharmacogn Mag. 2015 Oct;11:S427-33. doi: 10.4103/0973-1296.168982.

BACKGROUND: Emblica officinalis (Euphorbiaceae), popularly known as Indian gooseberry or "Amla" in India, is used in Ayurveda as "rejuvenating herb" since ancient times.OBJECTIVE: This study was carried out to estimate toxicity, anti-inflammatory, and antioxidative activities of the methanolic extract...

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Middha SK, Goyal AK, Lokesh P, et al. Toxicological Evaluation of Emblica officinalis Fruit Extract and its Anti-inflammatory and Free Radical Scavenging Properties. Pharmacogn Mag. 2015;11:S427-33doi: 10.4103/0973-1296.168982.
Middha, S. K., Goyal, A. K., Lokesh, P., Yardi, V., Mojamdar, L., Keni, D. S., Babu, D., & Usha, T. (2015). Toxicological Evaluation of Emblica officinalis Fruit Extract and its Anti-inflammatory and Free Radical Scavenging Properties. Pharmacognosy magazine, 11S427-33. https://doi.org/10.4103/0973-1296.168982
Middha, Sushil Kumar, et al. "Toxicological Evaluation of Emblica officinalis Fruit Extract and its Anti-inflammatory and Free Radical Scavenging Properties." Pharmacognosy magazine vol. 11 (2015): S427-33. doi: https://doi.org/10.4103/0973-1296.168982
Middha SK, Goyal AK, Lokesh P, Yardi V, Mojamdar L, Keni DS, Babu D, Usha T. Toxicological Evaluation of Emblica officinalis Fruit Extract and its Anti-inflammatory and Free Radical Scavenging Properties. Pharmacogn Mag. 2015 Oct;11:S427-33. doi: 10.4103/0973-1296.168982. PMID: 26929577; PMCID: PMC4745213.
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S-Nitrosothiols: chemistry and reactions.

Chemical communications (Cambridge, England)

Zhang C, Biggs TD, Devarie-Baez NO, Shuang S, Dong C, Xian M.
PMID: 28944382
Chem Commun (Camb). 2017 Oct 12;53(82):11266-11277. doi: 10.1039/c7cc06574d.

The formation of S-nitrosothiols (SNO) in protein cysteine residues is an important post-translational modification elicited by nitric oxide (NO). This process is involved in virtually every class of cell signaling and has attracted considerable attention in redox biology. On...

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Zhang C, Biggs TD, Devarie-Baez NO, et al. S-Nitrosothiols: chemistry and reactions. Chem Commun (Camb). 2017;53(82):11266-11277doi: 10.1039/c7cc06574d.
Zhang, C., Biggs, T. D., Devarie-Baez, N. O., Shuang, S., Dong, C., & Xian, M. (2017). S-Nitrosothiols: chemistry and reactions. Chemical communications (Cambridge, England), 53(82), 11266-11277. https://doi.org/10.1039/c7cc06574d
Zhang, Caihong, et al. "S-Nitrosothiols: chemistry and reactions." Chemical communications (Cambridge, England) vol. 53,82 (2017): 11266-11277. doi: https://doi.org/10.1039/c7cc06574d
Zhang C, Biggs TD, Devarie-Baez NO, Shuang S, Dong C, Xian M. S-Nitrosothiols: chemistry and reactions. Chem Commun (Camb). 2017 Oct 12;53(82):11266-11277. doi: 10.1039/c7cc06574d. PMID: 28944382.
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Gaseous Mediators and Mitochondrial Function: The Future of Pharmacologically Induced Suspended Animation?.

Frontiers in physiology

Hartmann C, Nussbaum B, Calzia E, Radermacher P, Wepler M.
PMID: 28974933
Front Physiol. 2017 Sep 19;8:691. doi: 10.3389/fphys.2017.00691. eCollection 2017.

The role of nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H

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Hartmann C, Nussbaum B, Calzia E, et al. Gaseous Mediators and Mitochondrial Function: The Future of Pharmacologically Induced Suspended Animation?. Front Physiol. 2017;8:691doi: 10.3389/fphys.2017.00691.
Hartmann, C., Nussbaum, B., Calzia, E., Radermacher, P., & Wepler, M. (2017). Gaseous Mediators and Mitochondrial Function: The Future of Pharmacologically Induced Suspended Animation?. Frontiers in physiology, 8691. https://doi.org/10.3389/fphys.2017.00691
Hartmann, Clair, et al. "Gaseous Mediators and Mitochondrial Function: The Future of Pharmacologically Induced Suspended Animation?." Frontiers in physiology vol. 8 (2017): 691. doi: https://doi.org/10.3389/fphys.2017.00691
Hartmann C, Nussbaum B, Calzia E, Radermacher P, Wepler M. Gaseous Mediators and Mitochondrial Function: The Future of Pharmacologically Induced Suspended Animation?. Front Physiol. 2017 Sep 19;8:691. doi: 10.3389/fphys.2017.00691. eCollection 2017. PMID: 28974933; PMCID: PMC5610695.
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Effects of N-acetylcysteine (NAC) supplementation in resuscitation fluids on renal microcirculatory oxygenation, inflammation, and function in a rat model of endotoxemia.

Intensive care medicine experimental

Ergin B, Guerci P, Zafrani L, Nocken F, Kandil A, Gurel-Gurevin E, Demirci-Tansel C, Ince C.
PMID: 27671340
Intensive Care Med Exp. 2016 Dec;4(1):29. doi: 10.1186/s40635-016-0106-1. Epub 2016 Sep 26.

BACKGROUND: Modulation of inflammation and oxidative stress appears to limit sepsis-induced damage in experimental models. The kidney is one of the most sensitive organs to injury during septic shock. In this study, we evaluated the effect of N-acetylcysteine (NAC)...

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Ergin B, Guerci P, Zafrani L, et al. Effects of N-acetylcysteine (NAC) supplementation in resuscitation fluids on renal microcirculatory oxygenation, inflammation, and function in a rat model of endotoxemia. Intensive Care Med Exp. 2016;4(1):29doi: 10.1186/s40635-016-0106-1.
Ergin, B., Guerci, P., Zafrani, L., Nocken, F., Kandil, A., Gurel-Gurevin, E., Demirci-Tansel, C., & Ince, C. (2016). Effects of N-acetylcysteine (NAC) supplementation in resuscitation fluids on renal microcirculatory oxygenation, inflammation, and function in a rat model of endotoxemia. Intensive care medicine experimental, 4(1), 29. https://doi.org/10.1186/s40635-016-0106-1
Ergin, Bulent, et al. "Effects of N-acetylcysteine (NAC) supplementation in resuscitation fluids on renal microcirculatory oxygenation, inflammation, and function in a rat model of endotoxemia." Intensive care medicine experimental vol. 4,1 (2016): 29. doi: https://doi.org/10.1186/s40635-016-0106-1
Ergin B, Guerci P, Zafrani L, Nocken F, Kandil A, Gurel-Gurevin E, Demirci-Tansel C, Ince C. Effects of N-acetylcysteine (NAC) supplementation in resuscitation fluids on renal microcirculatory oxygenation, inflammation, and function in a rat model of endotoxemia. Intensive Care Med Exp. 2016 Dec;4(1):29. doi: 10.1186/s40635-016-0106-1. Epub 2016 Sep 26. PMID: 27671340; PMCID: PMC5037099.
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The effects of A2B receptor modulators on vascular endothelial growth factor and nitric oxide axis in chronic cyclosporine nephropathy.

Journal of pharmacology & pharmacotherapeutics

Patel L, Thaker A.
PMID: 26311998
J Pharmacol Pharmacother. 2015 Jul-Sep;6(3):147-53. doi: 10.4103/0976-500X.162014.

INTRODUCTION: To investigate the actions of adenosine A2B receptor modulators on VEGF and NO levels in CsA nephropathy.MATERIALS AND METHODS: Nephropathy was induced by administrating 25 mg/kg (s.c) of CsA for 5 weeks. The VEGF and NO levels were...

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Patel L, Thaker A. The effects of A2B receptor modulators on vascular endothelial growth factor and nitric oxide axis in chronic cyclosporine nephropathy. J Pharmacol Pharmacother. 2015;6(3):147-53doi: 10.4103/0976-500X.162014.
Patel, L., & Thaker, A. (2015). The effects of A2B receptor modulators on vascular endothelial growth factor and nitric oxide axis in chronic cyclosporine nephropathy. Journal of pharmacology & pharmacotherapeutics, 6(3), 147-53. https://doi.org/10.4103/0976-500X.162014
Patel, Leena, and Thaker, Aswin. "The effects of A2B receptor modulators on vascular endothelial growth factor and nitric oxide axis in chronic cyclosporine nephropathy." Journal of pharmacology & pharmacotherapeutics vol. 6,3 (2015): 147-53. doi: https://doi.org/10.4103/0976-500X.162014
Patel L, Thaker A. The effects of A2B receptor modulators on vascular endothelial growth factor and nitric oxide axis in chronic cyclosporine nephropathy. J Pharmacol Pharmacother. 2015 Jul-Sep;6(3):147-53. doi: 10.4103/0976-500X.162014. PMID: 26311998; PMCID: PMC4544136.
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Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus.

Frontiers in cellular neuroscience

Kusek M, Tokarska A, Siwiec M, Gadek-Michalska A, Szewczyk B, Hess G, Tokarski K.
PMID: 28515682
Front Cell Neurosci. 2017 May 03;11:127. doi: 10.3389/fncel.2017.00127. eCollection 2017.

Corticotropin-releasing hormone (CRH)-synthesizing parvocellular neuroendocrine cells (PNCs) of the hypothalamic paraventricular nucleus (PVN) play a key role in the activation of the hypothalamic-pituitary-adrenocortical (HPA) axis. Several studies have demonstrated that synaptic inputs to these cells may undergo stress-related enhancement...

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Kusek M, Tokarska A, Siwiec M, et al. Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Front Cell Neurosci. 2017;11:127doi: 10.3389/fncel.2017.00127.
Kusek, M., Tokarska, A., Siwiec, M., Gadek-Michalska, A., Szewczyk, B., Hess, G., & Tokarski, K. (2017). Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Frontiers in cellular neuroscience, 11127. https://doi.org/10.3389/fncel.2017.00127
Kusek, Magdalena, et al. "Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus." Frontiers in cellular neuroscience vol. 11 (2017): 127. doi: https://doi.org/10.3389/fncel.2017.00127
Kusek M, Tokarska A, Siwiec M, Gadek-Michalska A, Szewczyk B, Hess G, Tokarski K. Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Front Cell Neurosci. 2017 May 03;11:127. doi: 10.3389/fncel.2017.00127. eCollection 2017. PMID: 28515682; PMCID: PMC5413825.
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Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide.

Journal of gastroenterology and hepatology

Kawada N, Seki S, Kuroki T, Inoue M.
PMID: 28976688
J Gastroenterol Hepatol. 1998 Sep;13:S6-S13. doi: 10.1111/jgh.1998.13.s1.6.

We studied the effect of lipopolysaccharide (LPS) on the proliferation of culture-stimulated rat stellate cells. DNA synthesis as determined by [

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Kawada N, Seki S, Kuroki T, et al. Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide. J Gastroenterol Hepatol. 1998;13:S6-S13doi: 10.1111/jgh.1998.13.s1.6.
Kawada, N., Seki, S., Kuroki, T., & Inoue, M. (1998). Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide. Journal of gastroenterology and hepatology, 13S6-S13. https://doi.org/10.1111/jgh.1998.13.s1.6
Kawada, Norifumi, et al. "Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide." Journal of gastroenterology and hepatology vol. 13 (1998): S6-S13. doi: https://doi.org/10.1111/jgh.1998.13.s1.6
Kawada N, Seki S, Kuroki T, Inoue M. Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide. J Gastroenterol Hepatol. 1998 Sep;13:S6-S13. doi: 10.1111/jgh.1998.13.s1.6. PMID: 28976688.
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The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis.

Experimental and therapeutic medicine

Shin SJ, Lee KH, Chung KS, Cheon SY, An HJ.
PMID: 28450936
Exp Ther Med. 2017 Mar;13(3):1025-1031. doi: 10.3892/etm.2017.4088. Epub 2017 Jan 25.

Benign prostatic hyperplasia (BPH) is a pathological condition that affects the majority of men above the age of 50 years. Pharmacological agents are typically used to treat BPH; however, there are currently no pharmacological agents that are able to...

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Shin SJ, Lee KH, Chung KS, et al. The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis. Exp Ther Med. 2017;13(3):1025-1031doi: 10.3892/etm.2017.4088.
Shin, S. J., Lee, K. H., Chung, K. S., Cheon, S. Y., & An, H. J. (2017). The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis. Experimental and therapeutic medicine, 13(3), 1025-1031. https://doi.org/10.3892/etm.2017.4088
Shin, Su-Jin, et al. "The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis." Experimental and therapeutic medicine vol. 13,3 (2017): 1025-1031. doi: https://doi.org/10.3892/etm.2017.4088
Shin SJ, Lee KH, Chung KS, Cheon SY, An HJ. The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis. Exp Ther Med. 2017 Mar;13(3):1025-1031. doi: 10.3892/etm.2017.4088. Epub 2017 Jan 25. PMID: 28450936; PMCID: PMC5403478.
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Effects of foliar application of salicylic acid and nitric oxide in alleviating iron deficiency induced chlorosis of Arachis hypogaea L.

Botanical studies

Kong J, Dong Y, Xu L, Liu S, Bai X.
PMID: 28510913
Bot Stud. 2014 Dec;55(1):9. doi: 10.1186/1999-3110-55-9. Epub 2014 Jan 20.

BACKGROUND: The aim of this experiment was to analyze the alleviation mechanism of exogenous salicylic acid (SA) and sodium nitroprusside (SNP, a nitric oxide donor) on peanut seedlings under Fe deficiency. The effects of SA and SNP on iron...

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Kong J, Dong Y, Xu L, et al. Effects of foliar application of salicylic acid and nitric oxide in alleviating iron deficiency induced chlorosis of Arachis hypogaea L. Bot Stud. 2014;55(1):9doi: 10.1186/1999-3110-55-9.
Kong, J., Dong, Y., Xu, L., Liu, S., & Bai, X. (2014). Effects of foliar application of salicylic acid and nitric oxide in alleviating iron deficiency induced chlorosis of Arachis hypogaea L. Botanical studies, 55(1), 9. https://doi.org/10.1186/1999-3110-55-9
Kong, Jing, et al. "Effects of foliar application of salicylic acid and nitric oxide in alleviating iron deficiency induced chlorosis of Arachis hypogaea L." Botanical studies vol. 55,1 (2014): 9. doi: https://doi.org/10.1186/1999-3110-55-9
Kong J, Dong Y, Xu L, Liu S, Bai X. Effects of foliar application of salicylic acid and nitric oxide in alleviating iron deficiency induced chlorosis of Arachis hypogaea L. Bot Stud. 2014 Dec;55(1):9. doi: 10.1186/1999-3110-55-9. Epub 2014 Jan 20. PMID: 28510913; PMCID: PMC5432746.
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