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Showing 13 to 20 of 20 entries
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Alterations of colonic contractility in an interleukin-10 knockout mouse model of inflammatory bowel disease.

Journal of neurogastroenterology and motility

Park JH, Kwon JG, Kim SJ, Song DK, Lee SG, Kim ES, Kim ES, Cho KB, Jang BI, Kim DH, Sin JI, Kim TW, Song IH, Park KS.
PMID: 25537671
J Neurogastroenterol Motil. 2015 Jan 01;21(1):51-61. doi: 10.5056/jnm14008.

BACKGROUND/AIMS: Inflammatory bowel disease is commonly accompanied by colonic dysmotility and causes changes in intestinal smooth muscle contractility. In this study, colonic smooth muscle contractility in a chronic inflammatory condition was investigated using smooth muscle tissues prepared from interleukin-10...

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Park JH, Kwon JG, Kim SJ, et al. Alterations of colonic contractility in an interleukin-10 knockout mouse model of inflammatory bowel disease. J Neurogastroenterol Motil. 2015;21(1):51-61doi: 10.5056/jnm14008.
Park, J. H., Kwon, J. G., Kim, S. J., Song, D. K., Lee, S. G., Kim, E. S., Kim, E. S., Cho, K. B., Jang, B. I., Kim, D. H., Sin, J. I., Kim, T. W., Song, I. H., & Park, K. S. (2015). Alterations of colonic contractility in an interleukin-10 knockout mouse model of inflammatory bowel disease. Journal of neurogastroenterology and motility, 21(1), 51-61. https://doi.org/10.5056/jnm14008
Park, Jae Hyung, et al. "Alterations of colonic contractility in an interleukin-10 knockout mouse model of inflammatory bowel disease." Journal of neurogastroenterology and motility vol. 21,1 (2015): 51-61. doi: https://doi.org/10.5056/jnm14008
Park JH, Kwon JG, Kim SJ, Song DK, Lee SG, Kim ES, Kim ES, Cho KB, Jang BI, Kim DH, Sin JI, Kim TW, Song IH, Park KS. Alterations of colonic contractility in an interleukin-10 knockout mouse model of inflammatory bowel disease. J Neurogastroenterol Motil. 2015 Jan 01;21(1):51-61. doi: 10.5056/jnm14008. PMID: 25537671; PMCID: PMC4288096.
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Ginsenoside Re inhibits pacemaker potentials via adenosine triphosphate-sensitive potassium channels and the cyclic guanosine monophosphate/nitric oxide-dependent pathway in cultured interstitial cells of Cajal from mouse small intestine.

Journal of ginseng research

Hong NR, Park HS, Ahn TS, Kim HJ, Ha KT, Kim BJ.
PMID: 26869823
J Ginseng Res. 2015 Oct;39(4):314-21. doi: 10.1016/j.jgr.2015.02.004. Epub 2015 Mar 06.

BACKGROUND: Ginseng belongs to the genus Panax. Its main active ingredients are the ginsenosides. Interstitial cells of Cajal (ICCs) are the pacemaker cells of the gastrointestinal (GI) tract. To understand the effects of ginsenoside Re (GRe) on GI motility,...

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Hong NR, Park HS, Ahn TS, et al. Ginsenoside Re inhibits pacemaker potentials via adenosine triphosphate-sensitive potassium channels and the cyclic guanosine monophosphate/nitric oxide-dependent pathway in cultured interstitial cells of Cajal from mouse small intestine. J Ginseng Res. 2015;39(4):314-21doi: 10.1016/j.jgr.2015.02.004.
Hong, N. R., Park, H. S., Ahn, T. S., Kim, H. J., Ha, K. T., & Kim, B. J. (2015). Ginsenoside Re inhibits pacemaker potentials via adenosine triphosphate-sensitive potassium channels and the cyclic guanosine monophosphate/nitric oxide-dependent pathway in cultured interstitial cells of Cajal from mouse small intestine. Journal of ginseng research, 39(4), 314-21. https://doi.org/10.1016/j.jgr.2015.02.004
Hong, Noo Ri, et al. "Ginsenoside Re inhibits pacemaker potentials via adenosine triphosphate-sensitive potassium channels and the cyclic guanosine monophosphate/nitric oxide-dependent pathway in cultured interstitial cells of Cajal from mouse small intestine." Journal of ginseng research vol. 39,4 (2015): 314-21. doi: https://doi.org/10.1016/j.jgr.2015.02.004
Hong NR, Park HS, Ahn TS, Kim HJ, Ha KT, Kim BJ. Ginsenoside Re inhibits pacemaker potentials via adenosine triphosphate-sensitive potassium channels and the cyclic guanosine monophosphate/nitric oxide-dependent pathway in cultured interstitial cells of Cajal from mouse small intestine. J Ginseng Res. 2015 Oct;39(4):314-21. doi: 10.1016/j.jgr.2015.02.004. Epub 2015 Mar 06. PMID: 26869823; PMCID: PMC4593795.
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Role of HO/CO in the Control of Peripheral Circulation in Humans.

International journal of hypertension

Sacerdoti D, Mania D, Pesce P, Gaiani S, Gatta A, Bolognesi M.
PMID: 22500215
Int J Hypertens. 2012;2012:236180. doi: 10.1155/2012/236180. Epub 2012 Mar 04.

Experimental studies show that the heme oxygenase/carbon monoxide system (HO/CO) plays an important role in the homeostasis of circulation and in the pathophysiology of hypertension. No data are available on its role in the control of peripheral circulation in...

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Sacerdoti D, Mania D, Pesce P, et al. Role of HO/CO in the Control of Peripheral Circulation in Humans. Int J Hypertens. 2012;2012:236180doi: 10.1155/2012/236180.
Sacerdoti, D., Mania, D., Pesce, P., Gaiani, S., Gatta, A., & Bolognesi, M. (2012). Role of HO/CO in the Control of Peripheral Circulation in Humans. International journal of hypertension, 2012236180. https://doi.org/10.1155/2012/236180
Sacerdoti, David, et al. "Role of HO/CO in the Control of Peripheral Circulation in Humans." International journal of hypertension vol. 2012 (2012): 236180. doi: https://doi.org/10.1155/2012/236180
Sacerdoti D, Mania D, Pesce P, Gaiani S, Gatta A, Bolognesi M. Role of HO/CO in the Control of Peripheral Circulation in Humans. Int J Hypertens. 2012;2012:236180. doi: 10.1155/2012/236180. Epub 2012 Mar 04. PMID: 22500215; PMCID: PMC3303686.
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Role of nitric oxide and cGMP in the modulation of vascular contraction induced by angiotensin II and Bay K8644 during ischemia/reperfusion.

Experimental and therapeutic medicine

Szadujkis-Szadurska K, Grzesk G, Szadujkis-Szadurski L, Gajdus M, Matusiak G.
PMID: 23407814
Exp Ther Med. 2013 Feb;5(2):616-620. doi: 10.3892/etm.2012.846. Epub 2012 Nov 30.

Vascular smooth muscle tone changes under the influence of numerous contracting and relaxing factors. The purpose of the present study was to determine the modulating effect of ischemia and reperfusion (I/R) on contraction triggered by angiotensin II (ANG II)...

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Szadujkis-Szadurska K, Grzesk G, Szadujkis-Szadurski L, et al. Role of nitric oxide and cGMP in the modulation of vascular contraction induced by angiotensin II and Bay K8644 during ischemia/reperfusion. Exp Ther Med. 2012;5(2):616-620doi: 10.3892/etm.2012.846.
Szadujkis-Szadurska, K., Grzesk, G., Szadujkis-Szadurski, L., Gajdus, M., & Matusiak, G. (2013). Role of nitric oxide and cGMP in the modulation of vascular contraction induced by angiotensin II and Bay K8644 during ischemia/reperfusion. Experimental and therapeutic medicine, 5(2), 616-620. https://doi.org/10.3892/etm.2012.846
Szadujkis-Szadurska, Katarzyna, et al. "Role of nitric oxide and cGMP in the modulation of vascular contraction induced by angiotensin II and Bay K8644 during ischemia/reperfusion." Experimental and therapeutic medicine vol. 5,2 (2013): 616-620. doi: https://doi.org/10.3892/etm.2012.846
Szadujkis-Szadurska K, Grzesk G, Szadujkis-Szadurski L, Gajdus M, Matusiak G. Role of nitric oxide and cGMP in the modulation of vascular contraction induced by angiotensin II and Bay K8644 during ischemia/reperfusion. Exp Ther Med. 2013 Feb;5(2):616-620. doi: 10.3892/etm.2012.846. Epub 2012 Nov 30. PMID: 23407814; PMCID: PMC3570230.
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Effects of lubiprostone on pacemaker activity of interstitial cells of cajal from the mouse colon.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Jiao HY, Kim DH, Ki JS, Ryu KH, Choi S, Jun JY.
PMID: 25177167
Korean J Physiol Pharmacol. 2014 Aug;18(4):341-6. doi: 10.4196/kjpp.2014.18.4.341. Epub 2014 Aug 13.

Lubiprostone is a chloride (Cl(-)) channel activator derived from prostaglandin E1 and used for managing constipation. In addition, lubiprostone affects the activity of gastrointestinal smooth muscles. Interstitial cells of Cajal (ICCs) are pacemaker cells that generate slow-wave activity in...

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Jiao HY, Kim DH, Ki JS, et al. Effects of lubiprostone on pacemaker activity of interstitial cells of cajal from the mouse colon. Korean J Physiol Pharmacol. 2014;18(4):341-6doi: 10.4196/kjpp.2014.18.4.341.
Jiao, H. Y., Kim, D. H., Ki, J. S., Ryu, K. H., Choi, S., & Jun, J. Y. (2014). Effects of lubiprostone on pacemaker activity of interstitial cells of cajal from the mouse colon. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 18(4), 341-6. https://doi.org/10.4196/kjpp.2014.18.4.341
Jiao, Han-Yi, et al. "Effects of lubiprostone on pacemaker activity of interstitial cells of cajal from the mouse colon." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 18,4 (2014): 341-6. doi: https://doi.org/10.4196/kjpp.2014.18.4.341
Jiao HY, Kim DH, Ki JS, Ryu KH, Choi S, Jun JY. Effects of lubiprostone on pacemaker activity of interstitial cells of cajal from the mouse colon. Korean J Physiol Pharmacol. 2014 Aug;18(4):341-6. doi: 10.4196/kjpp.2014.18.4.341. Epub 2014 Aug 13. PMID: 25177167; PMCID: PMC4146637.
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Naringenin inhibits pacemaking activity in interstitial cells of Cajal from murine small intestine.

Integrative medicine research

Kim HJ, Kim BJ.
PMID: 28664138
Integr Med Res. 2017 Jun;6(2):149-155. doi: 10.1016/j.imr.2017.02.001. Epub 2017 Feb 16.

BACKGROUND: Naringenin (NRG) is a common dietary polyphenolic constituent of fruits. NRG has diverse pharmacological activities, and is used in traditional medicine to treat various diseases including gastrointestinal (GI) disorders. Interstitial cells of Cajal (ICCs) are pacemaker cells of...

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Kim HJ, Kim BJ. Naringenin inhibits pacemaking activity in interstitial cells of Cajal from murine small intestine. Integr Med Res. 2017;6(2):149-155doi: 10.1016/j.imr.2017.02.001.
Kim, H. J., & Kim, B. J. (2017). Naringenin inhibits pacemaking activity in interstitial cells of Cajal from murine small intestine. Integrative medicine research, 6(2), 149-155. https://doi.org/10.1016/j.imr.2017.02.001
Kim, Hyun Jung, and Kim, Byung Joo. "Naringenin inhibits pacemaking activity in interstitial cells of Cajal from murine small intestine." Integrative medicine research vol. 6,2 (2017): 149-155. doi: https://doi.org/10.1016/j.imr.2017.02.001
Kim HJ, Kim BJ. Naringenin inhibits pacemaking activity in interstitial cells of Cajal from murine small intestine. Integr Med Res. 2017 Jun;6(2):149-155. doi: 10.1016/j.imr.2017.02.001. Epub 2017 Feb 16. PMID: 28664138; PMCID: PMC5478266.
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A Role for Nitric Oxide in Long-term Potentiation.

The European journal of neuroscience

Bon C, Böhme GA, Doble A, Stutzmann JM, Blanchard JC.
PMID: 12106350
Eur J Neurosci. 1992;4(5):420-424. doi: 10.1111/j.1460-9568.1992.tb00891.x.

Nitric oxide production in the cerebellum and induction of long-term potentiation (LTP) in the hippocampus have some characteristics in common: both phenomena are induced by activation of N-methyl-d-aspartate receptors and both are highly dependent on calcium-mediated processes. Here we...

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Bon C, Böhme GA, Doble A, et al. A Role for Nitric Oxide in Long-term Potentiation. Eur J Neurosci. 1992;4(5):420-424doi: 10.1111/j.1460-9568.1992.tb00891.x.
Bon, C., Böhme, G. A., Doble, A., Stutzmann, J. M., & Blanchard, J. C. (1992). A Role for Nitric Oxide in Long-term Potentiation. The European journal of neuroscience, 4(5), 420-424. https://doi.org/10.1111/j.1460-9568.1992.tb00891.x
Bon, Christelle, et al. "A Role for Nitric Oxide in Long-term Potentiation." The European journal of neuroscience vol. 4,5 (1992): 420-424. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00891.x
Bon C, Böhme GA, Doble A, Stutzmann JM, Blanchard JC. A Role for Nitric Oxide in Long-term Potentiation. Eur J Neurosci. 1992;4(5):420-424. doi: 10.1111/j.1460-9568.1992.tb00891.x. PMID: 12106350.
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Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model.

World neurosurgery

Nishida S, Kawauchi S, Toyooka T, Kumagai K, Takeuchi S, Sato S, Kondo A, Wada K, Mori K.
PMID: 34500101
World Neurosurg. 2021 Nov;155:e704-e715. doi: 10.1016/j.wneu.2021.08.130. Epub 2021 Sep 06.

OBJECTIVE: Cortical spreading depolarization (CSD), cortical spreading ischemia (CSI), and early brain injury are involved in the occurrence of delayed brain ischemia after subarachnoid hemorrhage (SAH). We tested whether local application of magnesium (Mg) sulfate solution suppressed CSD and...

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Nishida S, Kawauchi S, Toyooka T, et al. Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model. World Neurosurg. 2021;155:e704-e715doi: 10.1016/j.wneu.2021.08.130.
Nishida, S., Kawauchi, S., Toyooka, T., Kumagai, K., Takeuchi, S., Sato, S., Kondo, A., Wada, K., & Mori, K. (2021). Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model. World neurosurgery, 155e704-e715. https://doi.org/10.1016/j.wneu.2021.08.130
Nishida, Sho, et al. "Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model." World neurosurgery vol. 155 (2021): e704-e715. doi: https://doi.org/10.1016/j.wneu.2021.08.130
Nishida S, Kawauchi S, Toyooka T, Kumagai K, Takeuchi S, Sato S, Kondo A, Wada K, Mori K. Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model. World Neurosurg. 2021 Nov;155:e704-e715. doi: 10.1016/j.wneu.2021.08.130. Epub 2021 Sep 06. PMID: 34500101.
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Showing 13 to 20 of 20 entries