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Accuracy of mutational signature software on correlated signatures.

Scientific reports

Wu Y, Chua EHZ, Ng AWT, Boot A, Rozen SG.
PMID: 35013428
Sci Rep. 2022 Jan 10;12(1):390. doi: 10.1038/s41598-021-04207-6.

Mutational signatures are characteristic patterns of mutations generated by exogenous mutagens or by endogenous mutational processes. Mutational signatures are important for research into DNA damage and repair, aging, cancer biology, genetic toxicology, and epidemiology. Unsupervised learning can infer mutational...

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Wu Y, Chua EHZ, Ng AWT, et al. Accuracy of mutational signature software on correlated signatures. Sci Rep. 2022;12(1):390doi: 10.1038/s41598-021-04207-6.
Wu, Y., Chua, E. H. Z., Ng, A. W. T., Boot, A., & Rozen, S. G. (2022). Accuracy of mutational signature software on correlated signatures. Scientific reports, 12(1), 390. https://doi.org/10.1038/s41598-021-04207-6
Wu, Yang, et al. "Accuracy of mutational signature software on correlated signatures." Scientific reports vol. 12,1 (2022): 390. doi: https://doi.org/10.1038/s41598-021-04207-6
Wu Y, Chua EHZ, Ng AWT, Boot A, Rozen SG. Accuracy of mutational signature software on correlated signatures. Sci Rep. 2022 Jan 10;12(1):390. doi: 10.1038/s41598-021-04207-6. PMID: 35013428.
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[No title available]

ZooKeys

Mutanen M, Huemer P, Autto J, Karsholt O, Kaila L.
PMID: 33223909
Zookeys. 2020 Nov 12;992:157-181. doi: 10.3897/zookeys.992.53975. eCollection 2020.

No abstract available.

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Mutanen M, Huemer P, Autto J, et al. . Zookeys. 2020;992:157-181doi: 10.3897/zookeys.992.53975.
Mutanen, M., Huemer, P., Autto, J., Karsholt, O., & Kaila, L. (2020). . ZooKeys, 992157-181. https://doi.org/10.3897/zookeys.992.53975
Mutanen, Marko, et al. "." ZooKeys vol. 992 (2020): 157-181. doi: https://doi.org/10.3897/zookeys.992.53975
Mutanen M, Huemer P, Autto J, Karsholt O, Kaila L. . Zookeys. 2020 Nov 12;992:157-181. doi: 10.3897/zookeys.992.53975. eCollection 2020. PMID: 33223909; PMCID: PMC7677292.
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InMut-finder: a software tool for insertion identification in mutagenesis using Nanopore long reads.

BMC genomics

Song R, Wang Z, Wang H, Zhang H, Wang X, Nguyen H, Holding D, Yu B, Clemente T, Jia S, Zhang C.
PMID: 34923956
BMC Genomics. 2021 Dec 19;22(1):908. doi: 10.1186/s12864-021-08206-9.

BACKGROUND: Biological mutagens (such as transposon) with sequences inserted, play a crucial role to link observed phenotype and genotype in reverse genetic studies. For this reason, accurate and efficient software tools for identifying insertion sites based on the analysis...

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Song R, Wang Z, Wang H, et al. InMut-finder: a software tool for insertion identification in mutagenesis using Nanopore long reads. BMC Genomics. 2021;22(1):908doi: 10.1186/s12864-021-08206-9.
Song, R., Wang, Z., Wang, H., Zhang, H., Wang, X., Nguyen, H., Holding, D., Yu, B., Clemente, T., Jia, S., & Zhang, C. (2021). InMut-finder: a software tool for insertion identification in mutagenesis using Nanopore long reads. BMC genomics, 22(1), 908. https://doi.org/10.1186/s12864-021-08206-9
Song, Rui, et al. "InMut-finder: a software tool for insertion identification in mutagenesis using Nanopore long reads." BMC genomics vol. 22,1 (2021): 908. doi: https://doi.org/10.1186/s12864-021-08206-9
Song R, Wang Z, Wang H, Zhang H, Wang X, Nguyen H, Holding D, Yu B, Clemente T, Jia S, Zhang C. InMut-finder: a software tool for insertion identification in mutagenesis using Nanopore long reads. BMC Genomics. 2021 Dec 19;22(1):908. doi: 10.1186/s12864-021-08206-9. PMID: 34923956.
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Availability of Emergency Drugs and Essential Equipment in Intensive Care Units in Hospitals of Ethiopia: A Multicenter Cross-Sectional Study.

Open access emergency medicine : OAEM

Hunie M, Desse T, Fenta E, Teshome D, Gelaw M, Gashaw A.
PMID: 33293877
Open Access Emerg Med. 2020 Dec 01;12:435-440. doi: 10.2147/OAEM.S285695. eCollection 2020.

PURPOSE: Emergency drugs and essential equipment are important to successfully manage patients in the intensive care unit (ICUs). The absence of these emergency drugs and essential equipment might result in mortality and morbidity which is more compounded in resource-limited...

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Hunie M, Desse T, Fenta E, et al. Availability of Emergency Drugs and Essential Equipment in Intensive Care Units in Hospitals of Ethiopia: A Multicenter Cross-Sectional Study. Open Access Emerg Med. 2020;12:435-440doi: 10.2147/OAEM.S285695.
Hunie, M., Desse, T., Fenta, E., Teshome, D., Gelaw, M., & Gashaw, A. (2020). Availability of Emergency Drugs and Essential Equipment in Intensive Care Units in Hospitals of Ethiopia: A Multicenter Cross-Sectional Study. Open access emergency medicine : OAEM, 12435-440. https://doi.org/10.2147/OAEM.S285695
Hunie, Metages, et al. "Availability of Emergency Drugs and Essential Equipment in Intensive Care Units in Hospitals of Ethiopia: A Multicenter Cross-Sectional Study." Open access emergency medicine : OAEM vol. 12 (2020): 435-440. doi: https://doi.org/10.2147/OAEM.S285695
Hunie M, Desse T, Fenta E, Teshome D, Gelaw M, Gashaw A. Availability of Emergency Drugs and Essential Equipment in Intensive Care Units in Hospitals of Ethiopia: A Multicenter Cross-Sectional Study. Open Access Emerg Med. 2020 Dec 01;12:435-440. doi: 10.2147/OAEM.S285695. eCollection 2020. PMID: 33293877; PMCID: PMC7719042.
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Inhibition of PFKFB3 Hampers the Progression of Atherosclerosis and Promotes Plaque Stability.

Frontiers in cell and developmental biology

Poels K, Schnitzler JG, Waissi F, Levels JHM, Stroes ESG, Daemen MJAP, Lutgens E, Pennekamp AM, De Kleijn DPV, Seijkens TTP, Kroon J.
PMID: 33282864
Front Cell Dev Biol. 2020 Nov 12;8:581641. doi: 10.3389/fcell.2020.581641. eCollection 2020.

AIMS: 6-phosphofructo-2-kinase/fructose-2,6-biphosphatase (PFKFB)3-mediated glycolysis is pivotal in driving macrophage- and endothelial cell activation and thereby inflammation. Once activated, these cells play a crucial role in the progression of atherosclerosis. Here, we analyzed the expression of PFKFB3 in human atherosclerotic...

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Poels K, Schnitzler JG, Waissi F, et al. Inhibition of PFKFB3 Hampers the Progression of Atherosclerosis and Promotes Plaque Stability. Front Cell Dev Biol. 2020;8:581641doi: 10.3389/fcell.2020.581641.
Poels, K., Schnitzler, J. G., Waissi, F., Levels, J. H. M., Stroes, E. S. G., Daemen, M. J. A. P., Lutgens, E., Pennekamp, A. M., De Kleijn, D. P. V., Seijkens, T. T. P., & Kroon, J. (2020). Inhibition of PFKFB3 Hampers the Progression of Atherosclerosis and Promotes Plaque Stability. Frontiers in cell and developmental biology, 8581641. https://doi.org/10.3389/fcell.2020.581641
Poels, Kikkie, et al. "Inhibition of PFKFB3 Hampers the Progression of Atherosclerosis and Promotes Plaque Stability." Frontiers in cell and developmental biology vol. 8 (2020): 581641. doi: https://doi.org/10.3389/fcell.2020.581641
Poels K, Schnitzler JG, Waissi F, Levels JHM, Stroes ESG, Daemen MJAP, Lutgens E, Pennekamp AM, De Kleijn DPV, Seijkens TTP, Kroon J. Inhibition of PFKFB3 Hampers the Progression of Atherosclerosis and Promotes Plaque Stability. Front Cell Dev Biol. 2020 Nov 12;8:581641. doi: 10.3389/fcell.2020.581641. eCollection 2020. PMID: 33282864; PMCID: PMC7688893.
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DNA Methylation-An Epigenetic Mark in Mutagen-Treated .

Plants (Basel, Switzerland)

Bara AW, Braszewska A, Kwasniewska J.
PMID: 34371611
Plants (Basel). 2021 Jul 09;10(7). doi: 10.3390/plants10071408.

The chromatin structure is significantly influenced by some epigenetic modifications including DNA methylation. The nuclear organization plays an essential role in the cell response to external stresses including mutagens. We present an analysis of the correlation between epigenetic modifications...

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Bara AW, Braszewska A, Kwasniewska J. DNA Methylation-An Epigenetic Mark in Mutagen-Treated . Plants (Basel). 2021;10(7)doi: 10.3390/plants10071408.
Bara, A. W., Braszewska, A., & Kwasniewska, J. (2021). DNA Methylation-An Epigenetic Mark in Mutagen-Treated . Plants (Basel, Switzerland), 10(7), . https://doi.org/10.3390/plants10071408
Bara, Adrianna Wiktoria, et al. "DNA Methylation-An Epigenetic Mark in Mutagen-Treated ." Plants (Basel, Switzerland) vol. 10,7 (2021). doi: https://doi.org/10.3390/plants10071408
Bara AW, Braszewska A, Kwasniewska J. DNA Methylation-An Epigenetic Mark in Mutagen-Treated . Plants (Basel). 2021 Jul 09;10(7). doi: 10.3390/plants10071408. PMID: 34371611; PMCID: PMC8309452.
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The influence of organic solvents on estimates of genotoxicity and antigenotoxicity in the SOS chromotest.

Genetics and molecular biology

Quintero N, Stashenko EE, Fuentes JL.
PMID: 22888301
Genet Mol Biol. 2012 Apr;35(2):503-14. doi: 10.1590/S1415-47572012000300018. Epub 2012 Jun 23.

In this work, the toxicity and genotoxicity of organic solvents (acetone, carbon tetrachloride, dichloromethane, dimethylsulfoxide, ethanol, ether and methanol) were studied using the SOS chromotest. The influence of these solvents on the direct genotoxicity induced by the mutagens mitomycin...

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Quintero N, Stashenko EE, Fuentes JL. The influence of organic solvents on estimates of genotoxicity and antigenotoxicity in the SOS chromotest. Genet Mol Biol. 2012;35(2):503-14doi: 10.1590/S1415-47572012000300018.
Quintero, N., Stashenko, E. E., & Fuentes, J. L. (2012). The influence of organic solvents on estimates of genotoxicity and antigenotoxicity in the SOS chromotest. Genetics and molecular biology, 35(2), 503-14. https://doi.org/10.1590/S1415-47572012000300018
Quintero, Nathalia, et al. "The influence of organic solvents on estimates of genotoxicity and antigenotoxicity in the SOS chromotest." Genetics and molecular biology vol. 35,2 (2012): 503-14. doi: https://doi.org/10.1590/S1415-47572012000300018
Quintero N, Stashenko EE, Fuentes JL. The influence of organic solvents on estimates of genotoxicity and antigenotoxicity in the SOS chromotest. Genet Mol Biol. 2012 Apr;35(2):503-14. doi: 10.1590/S1415-47572012000300018. Epub 2012 Jun 23. PMID: 22888301; PMCID: PMC3389540.
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Dnmt3a deficiency in the skin causes focal, canonical DNA hypomethylation and a cellular proliferation phenotype.

Proceedings of the National Academy of Sciences of the United States of America

Chen DY, Ferguson IM, Braun KA, Sutton LA, Helton NM, Ramakrishnan SM, Smith AM, Miller CA, Ley TJ.
PMID: 33846253
Proc Natl Acad Sci U S A. 2021 Apr 20;118(16). doi: 10.1073/pnas.2022760118.

DNA hypomethylation is a feature of epidermal cells from aged and sun-exposed skin, but the mechanisms responsible for this methylation loss are not known. Dnmt3a is the dominant de novo DNA methyltransferase in the skin; while epidermal Dnmt3a deficiency...

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Chen DY, Ferguson IM, Braun KA, et al. Dnmt3a deficiency in the skin causes focal, canonical DNA hypomethylation and a cellular proliferation phenotype. Proc Natl Acad Sci U S A. 2021;118(16)doi: 10.1073/pnas.2022760118.
Chen, D. Y., Ferguson, I. M., Braun, K. A., Sutton, L. A., Helton, N. M., Ramakrishnan, S. M., Smith, A. M., Miller, C. A., & Ley, T. J. (2021). Dnmt3a deficiency in the skin causes focal, canonical DNA hypomethylation and a cellular proliferation phenotype. Proceedings of the National Academy of Sciences of the United States of America, 118(16), . https://doi.org/10.1073/pnas.2022760118
Chen, David Y, et al. "Dnmt3a deficiency in the skin causes focal, canonical DNA hypomethylation and a cellular proliferation phenotype." Proceedings of the National Academy of Sciences of the United States of America vol. 118,16 (2021). doi: https://doi.org/10.1073/pnas.2022760118
Chen DY, Ferguson IM, Braun KA, Sutton LA, Helton NM, Ramakrishnan SM, Smith AM, Miller CA, Ley TJ. Dnmt3a deficiency in the skin causes focal, canonical DNA hypomethylation and a cellular proliferation phenotype. Proc Natl Acad Sci U S A. 2021 Apr 20;118(16). doi: 10.1073/pnas.2022760118. PMID: 33846253; PMCID: PMC8072215.
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Showing 481 to 488 of 488 entries