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Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation.

Transfusion medicine and hemotherapy : offizielles Organ der Deutschen Gesellschaft fur Transfusionsmedizin und Immunhamatologie

Kzhyshkowska J, Gudima A, Moganti K, Gratchev A, Orekhov A.
PMID: 27226789
Transfus Med Hemother. 2016 Mar;43(2):66-77. doi: 10.1159/000444943. Epub 2016 Mar 15.

Low-grade chronic inflammation underlies the development of the most dangerous cardiometabolic disorders including type 2 diabetes and its vascular complications. In contrast to acute inflammation induced by bacteria and viruses, chronic inflammation can be driven by abnormal reaction to...

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Kzhyshkowska J, Gudima A, Moganti K, et al. Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation. Transfus Med Hemother. 2016;43(2):66-77doi: 10.1159/000444943.
Kzhyshkowska, J., Gudima, A., Moganti, K., Gratchev, A., & Orekhov, A. (2016). Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation. Transfusion medicine and hemotherapy : offizielles Organ der Deutschen Gesellschaft fur Transfusionsmedizin und Immunhamatologie, 43(2), 66-77. https://doi.org/10.1159/000444943
Kzhyshkowska, Julia, et al. "Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation." Transfusion medicine and hemotherapy : offizielles Organ der Deutschen Gesellschaft fur Transfusionsmedizin und Immunhamatologie vol. 43,2 (2016): 66-77. doi: https://doi.org/10.1159/000444943
Kzhyshkowska J, Gudima A, Moganti K, Gratchev A, Orekhov A. Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation. Transfus Med Hemother. 2016 Mar;43(2):66-77. doi: 10.1159/000444943. Epub 2016 Mar 15. PMID: 27226789; PMCID: PMC4872050.
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Long-Term Effects of Prenatal Exposure to Undernutrition on Cannabinoid Receptor-Related Behaviors: Sex and Tissue-Specific Alterations in the mRNA Expression of Cannabinoid Receptors and Lipid Metabolic Regulators.

Frontiers in behavioral neuroscience

Ramírez-López MT, Arco R, Decara J, Vázquez M, Rivera P, Blanco RN, Alén F, Gómez de Heras R, Suárez J, Rodríguez de Fonseca F.
PMID: 28082878
Front Behav Neurosci. 2016 Dec 27;10:241. doi: 10.3389/fnbeh.2016.00241. eCollection 2016.

Maternal malnutrition causes long-lasting alterations in feeding behavior and energy homeostasis in offspring. It is still unknown whether both, the endocannabinoid (eCB) machinery and the lipid metabolism are implicated in long-term adaptive responses to fetal reprogramming caused by maternal...

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Ramírez-López MT, Arco R, Decara J, et al. Long-Term Effects of Prenatal Exposure to Undernutrition on Cannabinoid Receptor-Related Behaviors: Sex and Tissue-Specific Alterations in the mRNA Expression of Cannabinoid Receptors and Lipid Metabolic Regulators. Front Behav Neurosci. 2016;10:241doi: 10.3389/fnbeh.2016.00241.
Ramírez-López, M. T., Arco, R., Decara, J., Vázquez, M., Rivera, P., Blanco, R. N., Alén, F., Gómez de Heras, R., Suárez, J., & Rodríguez de Fonseca, F. (2016). Long-Term Effects of Prenatal Exposure to Undernutrition on Cannabinoid Receptor-Related Behaviors: Sex and Tissue-Specific Alterations in the mRNA Expression of Cannabinoid Receptors and Lipid Metabolic Regulators. Frontiers in behavioral neuroscience, 10241. https://doi.org/10.3389/fnbeh.2016.00241
Ramírez-López, María T, et al. "Long-Term Effects of Prenatal Exposure to Undernutrition on Cannabinoid Receptor-Related Behaviors: Sex and Tissue-Specific Alterations in the mRNA Expression of Cannabinoid Receptors and Lipid Metabolic Regulators." Frontiers in behavioral neuroscience vol. 10 (2016): 241. doi: https://doi.org/10.3389/fnbeh.2016.00241
Ramírez-López MT, Arco R, Decara J, Vázquez M, Rivera P, Blanco RN, Alén F, Gómez de Heras R, Suárez J, Rodríguez de Fonseca F. Long-Term Effects of Prenatal Exposure to Undernutrition on Cannabinoid Receptor-Related Behaviors: Sex and Tissue-Specific Alterations in the mRNA Expression of Cannabinoid Receptors and Lipid Metabolic Regulators. Front Behav Neurosci. 2016 Dec 27;10:241. doi: 10.3389/fnbeh.2016.00241. eCollection 2016. PMID: 28082878; PMCID: PMC5187359.
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Soluble cluster of differentiation 36 concentrations are not associated with cardiovascular risk factors in middle-aged subjects.

Biomedical reports

Alkhatatbeh MJ, Ayoub NM, Mhaidat NM, Saadeh NA, Lincz LF.
PMID: 27123261
Biomed Rep. 2016 May;4(5):642-648. doi: 10.3892/br.2016.622. Epub 2016 Mar 03.

Cluster of differentiation 36 (CD36) is involved in the development of atherosclerosis by enhancing macrophage endocytosis of oxidized low-density lipoproteins and foam cell formation. Soluble CD36 (sCD36) was found to be elevated in type 2 diabetic patients and possibly...

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Alkhatatbeh MJ, Ayoub NM, Mhaidat NM, et al. Soluble cluster of differentiation 36 concentrations are not associated with cardiovascular risk factors in middle-aged subjects. Biomed Rep. 2016;4(5):642-648doi: 10.3892/br.2016.622.
Alkhatatbeh, M. J., Ayoub, N. M., Mhaidat, N. M., Saadeh, N. A., & Lincz, L. F. (2016). Soluble cluster of differentiation 36 concentrations are not associated with cardiovascular risk factors in middle-aged subjects. Biomedical reports, 4(5), 642-648. https://doi.org/10.3892/br.2016.622
Alkhatatbeh, Mohammad J, et al. "Soluble cluster of differentiation 36 concentrations are not associated with cardiovascular risk factors in middle-aged subjects." Biomedical reports vol. 4,5 (2016): 642-648. doi: https://doi.org/10.3892/br.2016.622
Alkhatatbeh MJ, Ayoub NM, Mhaidat NM, Saadeh NA, Lincz LF. Soluble cluster of differentiation 36 concentrations are not associated with cardiovascular risk factors in middle-aged subjects. Biomed Rep. 2016 May;4(5):642-648. doi: 10.3892/br.2016.622. Epub 2016 Mar 03. PMID: 27123261; PMCID: PMC4840581.
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The prevalence of hypertension and its relationship with demographic factors, biochemical, and anthropometric indicators: A population-based study.

ARYA atherosclerosis

Khajedaluee M, Hassannia T, Rezaee A, Ziadi M, Dadgarmoghaddam M.
PMID: 28607565
ARYA Atheroscler. 2016 Nov;12(6):259-265.

BACKGROUND: Hypertension (HTN) is an important public health challenge worldwide. The prevalence of HTN varies across countries. It is necessary to obtain valid information about the prevalence of chronic condition like HTN and its predictors in different societies. Hence,...

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Khajedaluee M, Hassannia T, Rezaee A, et al. The prevalence of hypertension and its relationship with demographic factors, biochemical, and anthropometric indicators: A population-based study. ARYA Atheroscler. 2016;12(6):259-265
Khajedaluee, M., Hassannia, T., Rezaee, A., Ziadi, M., & Dadgarmoghaddam, M. (2016). The prevalence of hypertension and its relationship with demographic factors, biochemical, and anthropometric indicators: A population-based study. ARYA atherosclerosis, 12(6), 259-265.
Khajedaluee, Mohammad, et al. "The prevalence of hypertension and its relationship with demographic factors, biochemical, and anthropometric indicators: A population-based study." ARYA atherosclerosis vol. 12,6 (2016): 259-265.
Khajedaluee M, Hassannia T, Rezaee A, Ziadi M, Dadgarmoghaddam M. The prevalence of hypertension and its relationship with demographic factors, biochemical, and anthropometric indicators: A population-based study. ARYA Atheroscler. 2016 Nov;12(6):259-265. PMID: 28607565; PMCID: PMC5455324.
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Modeling Small-Angle X-ray Scattering Data for Low-Density Lipoproteins: Insights into the Fatty Core Packing and Phase Transition.

ACS nano

Maric S, Lind TK, Lyngsø J, Cárdenas M, Pedersen JS.
PMID: 28048943
ACS Nano. 2017 Jan 24;11(1):1080-1090. doi: 10.1021/acsnano.6b08089. Epub 2017 Jan 10.

Atherosclerosis and its clinical consequences are the leading cause of death in the western hemisphere. While many studies throughout the last decades have aimed at understanding the disease, the clinical markers in use today still fail to accurately predict...

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Maric S, Lind TK, Lyngsø J, et al. Modeling Small-Angle X-ray Scattering Data for Low-Density Lipoproteins: Insights into the Fatty Core Packing and Phase Transition. ACS Nano. 2017;11(1):1080-1090doi: 10.1021/acsnano.6b08089.
Maric, S., Lind, T. K., Lyngsø, J., Cárdenas, M., & Pedersen, J. S. (2017). Modeling Small-Angle X-ray Scattering Data for Low-Density Lipoproteins: Insights into the Fatty Core Packing and Phase Transition. ACS nano, 11(1), 1080-1090. https://doi.org/10.1021/acsnano.6b08089
Maric, Selma, et al. "Modeling Small-Angle X-ray Scattering Data for Low-Density Lipoproteins: Insights into the Fatty Core Packing and Phase Transition." ACS nano vol. 11,1 (2017): 1080-1090. doi: https://doi.org/10.1021/acsnano.6b08089
Maric S, Lind TK, Lyngsø J, Cárdenas M, Pedersen JS. Modeling Small-Angle X-ray Scattering Data for Low-Density Lipoproteins: Insights into the Fatty Core Packing and Phase Transition. ACS Nano. 2017 Jan 24;11(1):1080-1090. doi: 10.1021/acsnano.6b08089. Epub 2017 Jan 10. PMID: 28048943.
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Chlamydial Lipoproteins Stimulate Toll-Like Receptors 1/2 Mediated Inflammatory Responses through MyD88-Dependent Pathway.

Frontiers in microbiology

Wang Y, Liu Q, Chen D, Guan J, Ma L, Zhong G, Shu H, Wu X.
PMID: 28184217
Front Microbiol. 2017 Jan 26;8:78. doi: 10.3389/fmicb.2017.00078. eCollection 2017.

No abstract available.

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Wang Y, Liu Q, Chen D, et al. Chlamydial Lipoproteins Stimulate Toll-Like Receptors 1/2 Mediated Inflammatory Responses through MyD88-Dependent Pathway. Front Microbiol. 2017;8:78doi: 10.3389/fmicb.2017.00078.
Wang, Y., Liu, Q., Chen, D., Guan, J., Ma, L., Zhong, G., Shu, H., & Wu, X. (2017). Chlamydial Lipoproteins Stimulate Toll-Like Receptors 1/2 Mediated Inflammatory Responses through MyD88-Dependent Pathway. Frontiers in microbiology, 878. https://doi.org/10.3389/fmicb.2017.00078
Wang, Yong, et al. "Chlamydial Lipoproteins Stimulate Toll-Like Receptors 1/2 Mediated Inflammatory Responses through MyD88-Dependent Pathway." Frontiers in microbiology vol. 8 (2017): 78. doi: https://doi.org/10.3389/fmicb.2017.00078
Wang Y, Liu Q, Chen D, Guan J, Ma L, Zhong G, Shu H, Wu X. Chlamydial Lipoproteins Stimulate Toll-Like Receptors 1/2 Mediated Inflammatory Responses through MyD88-Dependent Pathway. Front Microbiol. 2017 Jan 26;8:78. doi: 10.3389/fmicb.2017.00078. eCollection 2017. PMID: 28184217; PMCID: PMC5266682.
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Correction: Lipoproteins comprise at least 10 different classes in rats, each of which contains a unique set of proteins as the primary component.

PloS one

Konishi T, Takahashi Y.
PMID: 29518154
PLoS One. 2018 Mar 08;13(3):e0194258. doi: 10.1371/journal.pone.0194258. eCollection 2018.

[This corrects the article DOI: 10.1371/journal.pone.0192955.].

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Konishi T, Takahashi Y. Correction: Lipoproteins comprise at least 10 different classes in rats, each of which contains a unique set of proteins as the primary component. PLoS One. 2018;13(3):e0194258doi: 10.1371/journal.pone.0194258.
Konishi, T., & Takahashi, Y. (2018). Correction: Lipoproteins comprise at least 10 different classes in rats, each of which contains a unique set of proteins as the primary component. PloS one, 13(3), e0194258. https://doi.org/10.1371/journal.pone.0194258
Konishi, Tomokazu, and Takahashi, Yoko. "Correction: Lipoproteins comprise at least 10 different classes in rats, each of which contains a unique set of proteins as the primary component." PloS one vol. 13,3 (2018): e0194258. doi: https://doi.org/10.1371/journal.pone.0194258
Konishi T, Takahashi Y. Correction: Lipoproteins comprise at least 10 different classes in rats, each of which contains a unique set of proteins as the primary component. PLoS One. 2018 Mar 08;13(3):e0194258. doi: 10.1371/journal.pone.0194258. eCollection 2018. PMID: 29518154; PMCID: PMC5843342.
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Lipoprotein assembly and function in an evolutionary perspective.

Biomolecular concepts

Van der Horst DJ, Rodenburg KW.
PMID: 25961995
Biomol Concepts. 2010 Aug 01;1(2):165-83. doi: 10.1515/bmc.2010.012.

Circulatory fat transport in animals relies on members of the large lipid transfer protein (LLTP) superfamily, including mammalian apolipoprotein B (apoB) and insect apolipophorin II/I (apoLp-II/I). ApoB and apoLp-II/I, constituting the structural (non-exchangeable) basis for the assembly of various...

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Van der Horst DJ, Rodenburg KW. Lipoprotein assembly and function in an evolutionary perspective. Biomol Concepts. 2010;1(2):165-83doi: 10.1515/bmc.2010.012.
Van der Horst, D. J., & Rodenburg, K. W. (2010). Lipoprotein assembly and function in an evolutionary perspective. Biomolecular concepts, 1(2), 165-83. https://doi.org/10.1515/bmc.2010.012
Van der Horst, Dick J, and Rodenburg, Kees W. "Lipoprotein assembly and function in an evolutionary perspective." Biomolecular concepts vol. 1,2 (2010): 165-83. doi: https://doi.org/10.1515/bmc.2010.012
Van der Horst DJ, Rodenburg KW. Lipoprotein assembly and function in an evolutionary perspective. Biomol Concepts. 2010 Aug 01;1(2):165-83. doi: 10.1515/bmc.2010.012. PMID: 25961995.
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Lipoprotein effects of incretin analogs and dipeptidyl peptidase 4 inhibitors.

Clinical lipidology

Zhong J, Maiseyeu A, Rajagopalan S.
PMID: 26005496
Clin Lipidol. 2015;10(1):103-112. doi: 10.2217/clp.14.59.

Elevated post-prandial lipoprotein levels are common in patients with type 2 diabetes. Post-prandial lipoprotein alterations in type 2 diabetics are widely believed to drive inflammation and are considered a major risk factor for cardiovascular disease in diabetic patients. The...

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Zhong J, Maiseyeu A, Rajagopalan S. Lipoprotein effects of incretin analogs and dipeptidyl peptidase 4 inhibitors. Clin Lipidol. 2015;10(1):103-112doi: 10.2217/clp.14.59.
Zhong, J., Maiseyeu, A., & Rajagopalan, S. (2015). Lipoprotein effects of incretin analogs and dipeptidyl peptidase 4 inhibitors. Clinical lipidology, 10(1), 103-112. https://doi.org/10.2217/clp.14.59
Zhong, Jixin, et al. "Lipoprotein effects of incretin analogs and dipeptidyl peptidase 4 inhibitors." Clinical lipidology vol. 10,1 (2015): 103-112. doi: https://doi.org/10.2217/clp.14.59
Zhong J, Maiseyeu A, Rajagopalan S. Lipoprotein effects of incretin analogs and dipeptidyl peptidase 4 inhibitors. Clin Lipidol. 2015;10(1):103-112. doi: 10.2217/clp.14.59. PMID: 26005496; PMCID: PMC4437760.
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The effect of a low-fat, plant-based lifestyle intervention (CHIP) on serum HDL levels and the implications for metabolic syndrome status - a cohort study.

Nutrition & metabolism

Kent L, Morton D, Rankin P, Ward E, Grant R, Gobble J, Diehl H.
PMID: 24283215
Nutr Metab (Lond). 2013 Oct 01;10(1):58. doi: 10.1186/1743-7075-10-58.

BACKGROUND: Low levels of high-density lipoproteins (HDL) are considered an important risk factor for cardiovascular disease and constitute one of the criteria for the Metabolic Syndrome (MetS). Lifestyle interventions promoting a low-fat, plant-based eating pattern appear to paradoxically reduce...

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Kent L, Morton D, Rankin P, et al. The effect of a low-fat, plant-based lifestyle intervention (CHIP) on serum HDL levels and the implications for metabolic syndrome status - a cohort study. Nutr Metab (Lond). 2013;10(1):58doi: 10.1186/1743-7075-10-58.
Kent, L., Morton, D., Rankin, P., Ward, E., Grant, R., Gobble, J., & Diehl, H. (2013). The effect of a low-fat, plant-based lifestyle intervention (CHIP) on serum HDL levels and the implications for metabolic syndrome status - a cohort study. Nutrition & metabolism, 10(1), 58. https://doi.org/10.1186/1743-7075-10-58
Kent, Lillian, et al. "The effect of a low-fat, plant-based lifestyle intervention (CHIP) on serum HDL levels and the implications for metabolic syndrome status - a cohort study." Nutrition & metabolism vol. 10,1 (2013): 58. doi: https://doi.org/10.1186/1743-7075-10-58
Kent L, Morton D, Rankin P, Ward E, Grant R, Gobble J, Diehl H. The effect of a low-fat, plant-based lifestyle intervention (CHIP) on serum HDL levels and the implications for metabolic syndrome status - a cohort study. Nutr Metab (Lond). 2013 Oct 01;10(1):58. doi: 10.1186/1743-7075-10-58. PMID: 24283215; PMCID: PMC3849684.
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Studies of single-walled carbon nanotubes-induced hepatotoxicity by NMR-based metabonomics of rat blood plasma and liver extracts.

Nanoscale research letters

Lin B, Zhang H, Lin Z, Fang Y, Tian L, Yang H, Yan J, Liu H, Zhang W, Xi Z.
PMID: 23680025
Nanoscale Res Lett. 2013 May 16;8(1):236. doi: 10.1186/1556-276X-8-236.

The toxicological effects of single-walled carbon nanotubes (SWCNTs) were investigated after intratracheal instillation in male Wistar rats over a 15-day period using metabonomic analysis of 1H (nuclear magnetic resonance) NMR spectra of blood plasma and liver tissue extracts. Concurrent...

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Lin B, Zhang H, Lin Z, et al. Studies of single-walled carbon nanotubes-induced hepatotoxicity by NMR-based metabonomics of rat blood plasma and liver extracts. Nanoscale Res Lett. 2013;8(1):236doi: 10.1186/1556-276X-8-236.
Lin, B., Zhang, H., Lin, Z., Fang, Y., Tian, L., Yang, H., Yan, J., Liu, H., Zhang, W., & Xi, Z. (2013). Studies of single-walled carbon nanotubes-induced hepatotoxicity by NMR-based metabonomics of rat blood plasma and liver extracts. Nanoscale research letters, 8(1), 236. https://doi.org/10.1186/1556-276X-8-236
Lin, Bencheng, et al. "Studies of single-walled carbon nanotubes-induced hepatotoxicity by NMR-based metabonomics of rat blood plasma and liver extracts." Nanoscale research letters vol. 8,1 (2013): 236. doi: https://doi.org/10.1186/1556-276X-8-236
Lin B, Zhang H, Lin Z, Fang Y, Tian L, Yang H, Yan J, Liu H, Zhang W, Xi Z. Studies of single-walled carbon nanotubes-induced hepatotoxicity by NMR-based metabonomics of rat blood plasma and liver extracts. Nanoscale Res Lett. 2013 May 16;8(1):236. doi: 10.1186/1556-276X-8-236. PMID: 23680025; PMCID: PMC3664573.
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Genetics of lipid traits: Genome-wide approaches yield new biology and clues to causality in coronary artery disease.

Biochimica et biophysica acta

Khetarpal SA, Rader DJ.
PMID: 24931102
Biochim Biophys Acta. 2014 Oct;1842(10):2010-2020. doi: 10.1016/j.bbadis.2014.06.007. Epub 2014 Jun 12.

A wealth of novel lipid loci have been identified through a variety of approaches focused on common and low-frequency variation and collaborative metaanalyses in multiethnic populations. Despite progress in identification of loci, the task of determining causal variants remains...

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Khetarpal SA, Rader DJ. Genetics of lipid traits: Genome-wide approaches yield new biology and clues to causality in coronary artery disease. Biochim Biophys Acta. 2014;1842(10):2010-2020doi: 10.1016/j.bbadis.2014.06.007.
Khetarpal, S. A., & Rader, D. J. (2014). Genetics of lipid traits: Genome-wide approaches yield new biology and clues to causality in coronary artery disease. Biochimica et biophysica acta, 1842(10), 2010-2020. https://doi.org/10.1016/j.bbadis.2014.06.007
Khetarpal, Sumeet A, and Rader, Daniel J. "Genetics of lipid traits: Genome-wide approaches yield new biology and clues to causality in coronary artery disease." Biochimica et biophysica acta vol. 1842,10 (2014): 2010-2020. doi: https://doi.org/10.1016/j.bbadis.2014.06.007
Khetarpal SA, Rader DJ. Genetics of lipid traits: Genome-wide approaches yield new biology and clues to causality in coronary artery disease. Biochim Biophys Acta. 2014 Oct;1842(10):2010-2020. doi: 10.1016/j.bbadis.2014.06.007. Epub 2014 Jun 12. PMID: 24931102.
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Showing 13 to 24 of 2458 entries