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Antioxidant, anti-inflammatory, and anti-apoptotic effects of crocin against doxorubicin-induced myocardial toxicity in rats.

Environmental science and pollution research international

Abdulkareem Aljumaily SA, Demir M, Elbe H, Yigitturk G, Bicer Y, Altinoz E.
PMID: 34322808
Environ Sci Pollut Res Int. 2021 Dec;28(46):65802-65813. doi: 10.1007/s11356-021-15409-w. Epub 2021 Jul 28.

Doxorubicin (DOX) is a well-known chemotherapeutic drug for most malignancies including breast cancer and leukemia whilst the usage of DOX is limited owing to its cardiotoxicity. In the present study, we aimed to investigate the effects of crocin on...

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Abdulkareem Aljumaily SA, Demir M, Elbe H, et al. Antioxidant, anti-inflammatory, and anti-apoptotic effects of crocin against doxorubicin-induced myocardial toxicity in rats. Environ Sci Pollut Res Int. 2021;28(46):65802-65813doi: 10.1007/s11356-021-15409-w.
Abdulkareem Aljumaily, S. A., Demir, M., Elbe, H., Yigitturk, G., Bicer, Y., & Altinoz, E. (2021). Antioxidant, anti-inflammatory, and anti-apoptotic effects of crocin against doxorubicin-induced myocardial toxicity in rats. Environmental science and pollution research international, 28(46), 65802-65813. https://doi.org/10.1007/s11356-021-15409-w
Abdulkareem Aljumaily, Sara Asaad, et al. "Antioxidant, anti-inflammatory, and anti-apoptotic effects of crocin against doxorubicin-induced myocardial toxicity in rats." Environmental science and pollution research international vol. 28,46 (2021): 65802-65813. doi: https://doi.org/10.1007/s11356-021-15409-w
Abdulkareem Aljumaily SA, Demir M, Elbe H, Yigitturk G, Bicer Y, Altinoz E. Antioxidant, anti-inflammatory, and anti-apoptotic effects of crocin against doxorubicin-induced myocardial toxicity in rats. Environ Sci Pollut Res Int. 2021 Dec;28(46):65802-65813. doi: 10.1007/s11356-021-15409-w. Epub 2021 Jul 28. PMID: 34322808.
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Mode of action and adverse effects of lipid lowering drugs.

Drugs of today (Barcelona, Spain : 1998)

Eghdamian B, Ghose K.
PMID: 14743263
Drugs Today (Barc). 1998 Nov;34(11):943-56. doi: 10.1358/dot.1998.34.11.487479.

Serum lipids, cholesterol and triglycerides are incorporated into hydrophilic lipoproteins, which include chylomicrons, very low density lipoproteins (VLDL), intermediate density lipoproteins (IDL), low density lipoproteins (LDL) and high density lipoproteins (HDL). An elevated level of these lipoproteins, except for...

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Eghdamian B, Ghose K. Mode of action and adverse effects of lipid lowering drugs. Drugs Today (Barc). 1998;34(11):943-56doi: 10.1358/dot.1998.34.11.487479.
Eghdamian, B., & Ghose, K. (1998). Mode of action and adverse effects of lipid lowering drugs. Drugs of today (Barcelona, Spain : 1998), 34(11), 943-56. https://doi.org/10.1358/dot.1998.34.11.487479
Eghdamian, B, and Ghose, K. "Mode of action and adverse effects of lipid lowering drugs." Drugs of today (Barcelona, Spain : 1998) vol. 34,11 (1998): 943-56. doi: https://doi.org/10.1358/dot.1998.34.11.487479
Eghdamian B, Ghose K. Mode of action and adverse effects of lipid lowering drugs. Drugs Today (Barc). 1998 Nov;34(11):943-56. doi: 10.1358/dot.1998.34.11.487479. PMID: 14743263.
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ApoC-III and visceral adipose tissue contribute to paradoxically normal triglyceride levels in insulin-resistant African-American women.

Nutrition & metabolism

Sumner AE, Furtado JD, Courville AB, Ricks M, Younger-Coleman N, Tulloch-Reid MK, Sacks FM.
PMID: 24365086
Nutr Metab (Lond). 2013 Dec 23;10(1):73. doi: 10.1186/1743-7075-10-73.

BACKGROUND: African-Americans are more insulin-resistant than whites but have lower triglyceride (TG) concentrations. The metabolic basis for this is unknown. Our goal was to determine in a cross-sectional study the effect of insulin resistance, visceral adipose tissue (VAT) and...

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Sumner AE, Furtado JD, Courville AB, et al. ApoC-III and visceral adipose tissue contribute to paradoxically normal triglyceride levels in insulin-resistant African-American women. Nutr Metab (Lond). 2013;10(1):73doi: 10.1186/1743-7075-10-73.
Sumner, A. E., Furtado, J. D., Courville, A. B., Ricks, M., Younger-Coleman, N., Tulloch-Reid, M. K., & Sacks, F. M. (2013). ApoC-III and visceral adipose tissue contribute to paradoxically normal triglyceride levels in insulin-resistant African-American women. Nutrition & metabolism, 10(1), 73. https://doi.org/10.1186/1743-7075-10-73
Sumner, Anne E, et al. "ApoC-III and visceral adipose tissue contribute to paradoxically normal triglyceride levels in insulin-resistant African-American women." Nutrition & metabolism vol. 10,1 (2013): 73. doi: https://doi.org/10.1186/1743-7075-10-73
Sumner AE, Furtado JD, Courville AB, Ricks M, Younger-Coleman N, Tulloch-Reid MK, Sacks FM. ApoC-III and visceral adipose tissue contribute to paradoxically normal triglyceride levels in insulin-resistant African-American women. Nutr Metab (Lond). 2013 Dec 23;10(1):73. doi: 10.1186/1743-7075-10-73. PMID: 24365086; PMCID: PMC3878037.
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Effects of Three Medicinal Plants Extracts in Experimental Diabetes: Antioxidant Enzymes Activities and Plasma Lipids Profiles inComparison with Metformin.

Iranian journal of pharmaceutical research : IJPR

Fehresti Sani M, Montasser Kouhsari S, Moradabadi L.
PMID: 24250517
Iran J Pharm Res. 2012;11(3):897-903.

In the present study we aimed to evaluate the effects of methanolic extracts of the bulbs of Garlic (Allium sativum L., Alliaceae) and Persian shallot (Allium ascalonicum L., Alliaceae ) and leaves of Sage (Salvia officinalis L., Lamiaceae ),...

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Fehresti Sani M, Montasser Kouhsari S, Moradabadi L. Effects of Three Medicinal Plants Extracts in Experimental Diabetes: Antioxidant Enzymes Activities and Plasma Lipids Profiles inComparison with Metformin. Iran J Pharm Res. 2012;11(3):897-903
Fehresti Sani, M., Montasser Kouhsari, S., & Moradabadi, L. (2012). Effects of Three Medicinal Plants Extracts in Experimental Diabetes: Antioxidant Enzymes Activities and Plasma Lipids Profiles inComparison with Metformin. Iranian journal of pharmaceutical research : IJPR, 11(3), 897-903.
Fehresti Sani, Mohammad, et al. "Effects of Three Medicinal Plants Extracts in Experimental Diabetes: Antioxidant Enzymes Activities and Plasma Lipids Profiles inComparison with Metformin." Iranian journal of pharmaceutical research : IJPR vol. 11,3 (2012): 897-903.
Fehresti Sani M, Montasser Kouhsari S, Moradabadi L. Effects of Three Medicinal Plants Extracts in Experimental Diabetes: Antioxidant Enzymes Activities and Plasma Lipids Profiles inComparison with Metformin. Iran J Pharm Res. 2012;11(3):897-903. PMID: 24250517; PMCID: PMC3813132.
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Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism.

Molecular metabolism

Jäger J, Greiner V, Strzoda D, Seibert O, Niopek K, Sijmonsma TP, Schäfer M, Jones A, De Guia R, Martignoni M, Dallinga-Thie GM, Diaz MB, Hofmann TG, Herzig S.
PMID: 24634828
Mol Metab. 2014 Jan 08;3(2):155-66. doi: 10.1016/j.molmet.2013.12.007. eCollection 2014 Apr.

Disturbances in lipid homeostasis are hallmarks of severe metabolic disorders and their long-term complications, including obesity, diabetes, and atherosclerosis. Whereas elevation of triglyceride (TG)-rich very-low-density lipoproteins (VLDL) has been identified as a risk factor for cardiovascular complications, high-density lipoprotein...

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Jäger J, Greiner V, Strzoda D, et al. Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism. Mol Metab. 2014;3(2):155-66doi: 10.1016/j.molmet.2013.12.007.
Jäger, J., Greiner, V., Strzoda, D., Seibert, O., Niopek, K., Sijmonsma, T. P., Schäfer, M., Jones, A., De Guia, R., Martignoni, M., Dallinga-Thie, G. M., Diaz, M. B., Hofmann, T. G., & Herzig, S. (2014). Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism. Molecular metabolism, 3(2), 155-66. https://doi.org/10.1016/j.molmet.2013.12.007
Jäger, Julia, et al. "Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism." Molecular metabolism vol. 3,2 (2014): 155-66. doi: https://doi.org/10.1016/j.molmet.2013.12.007
Jäger J, Greiner V, Strzoda D, Seibert O, Niopek K, Sijmonsma TP, Schäfer M, Jones A, De Guia R, Martignoni M, Dallinga-Thie GM, Diaz MB, Hofmann TG, Herzig S. Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism. Mol Metab. 2014 Jan 08;3(2):155-66. doi: 10.1016/j.molmet.2013.12.007. eCollection 2014 Apr. PMID: 24634828; PMCID: PMC3953693.
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Why Do Men Accumulate Abdominal Visceral Fat?.

Frontiers in physiology

Nauli AM, Matin S.
PMID: 31866877
Front Physiol. 2019 Dec 05;10:1486. doi: 10.3389/fphys.2019.01486. eCollection 2019.

Men have a higher tendency to accumulate abdominal visceral fat compared to pre-menopausal women. The accumulation of abdominal visceral fat in men, which is a strong independent predictor of mortality, is mainly due to the higher dietary fat uptake...

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Nauli AM, Matin S. Why Do Men Accumulate Abdominal Visceral Fat?. Front Physiol. 2019;10:1486doi: 10.3389/fphys.2019.01486.
Nauli, A. M., & Matin, S. (2019). Why Do Men Accumulate Abdominal Visceral Fat?. Frontiers in physiology, 101486. https://doi.org/10.3389/fphys.2019.01486
Nauli, Andromeda M, and Matin, Sahar. "Why Do Men Accumulate Abdominal Visceral Fat?." Frontiers in physiology vol. 10 (2019): 1486. doi: https://doi.org/10.3389/fphys.2019.01486
Nauli AM, Matin S. Why Do Men Accumulate Abdominal Visceral Fat?. Front Physiol. 2019 Dec 05;10:1486. doi: 10.3389/fphys.2019.01486. eCollection 2019. PMID: 31866877; PMCID: PMC6906176.
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New therapeutic horizons for plasma phospholipid transfer protein (PLTP): Targeting endotoxemia, infection and sepsis.

Pharmacology & therapeutics

Gautier T, Deckert V, Nguyen M, Desrumaux C, Masson D, Lagrost L.
PMID: 34974028
Pharmacol Ther. 2021 Dec 30;236:108105. doi: 10.1016/j.pharmthera.2021.108105. Epub 2021 Dec 30.

Phospholipid Transfer Protein (PLTP) transfers amphiphilic lipids between circulating lipoproteins and between lipoproteins, cells and tissues. Indeed, PLTP is a major determinant of the plasma levels, turnover and functionality of the main lipoprotein classes: very low-density lipoproteins (VLDL), low-density...

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Gautier T, Deckert V, Nguyen M, et al. New therapeutic horizons for plasma phospholipid transfer protein (PLTP): Targeting endotoxemia, infection and sepsis. Pharmacol Ther. 2021;236:108105doi: 10.1016/j.pharmthera.2021.108105.
Gautier, T., Deckert, V., Nguyen, M., Desrumaux, C., Masson, D., & Lagrost, L. (2021). New therapeutic horizons for plasma phospholipid transfer protein (PLTP): Targeting endotoxemia, infection and sepsis. Pharmacology & therapeutics, 236108105. https://doi.org/10.1016/j.pharmthera.2021.108105
Gautier, Thomas, et al. "New therapeutic horizons for plasma phospholipid transfer protein (PLTP): Targeting endotoxemia, infection and sepsis." Pharmacology & therapeutics vol. 236 (2021): 108105. doi: https://doi.org/10.1016/j.pharmthera.2021.108105
Gautier T, Deckert V, Nguyen M, Desrumaux C, Masson D, Lagrost L. New therapeutic horizons for plasma phospholipid transfer protein (PLTP): Targeting endotoxemia, infection and sepsis. Pharmacol Ther. 2021 Dec 30;236:108105. doi: 10.1016/j.pharmthera.2021.108105. Epub 2021 Dec 30. PMID: 34974028.
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Mitochondria-rough-ER contacts in the liver regulate systemic lipid homeostasis.

Cell reports

Anastasia I, Ilacqua N, Raimondi A, Lemieux P, Ghandehari-Alavijeh R, Faure G, Mekhedov SL, Williams KJ, Caicci F, Valle G, Giacomello M, Quiroga AD, Lehner R, Miksis MJ, Toth K, de Aguiar Vallim TQ, Koonin EV, Scorrano L, Pellegrini L.
PMID: 33730569
Cell Rep. 2021 Mar 16;34(11):108873. doi: 10.1016/j.celrep.2021.108873.

Contacts between organelles create microdomains that play major roles in regulating key intracellular activities and signaling pathways, but whether they also regulate systemic functions remains unknown. Here, we report the ultrastructural organization and dynamics of the inter-organellar contact established...

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Anastasia I, Ilacqua N, Raimondi A, et al. Mitochondria-rough-ER contacts in the liver regulate systemic lipid homeostasis. Cell Rep. 2021;34(11):108873doi: 10.1016/j.celrep.2021.108873.
Anastasia, I., Ilacqua, N., Raimondi, A., Lemieux, P., Ghandehari-Alavijeh, R., Faure, G., Mekhedov, S. L., Williams, K. J., Caicci, F., Valle, G., Giacomello, M., Quiroga, A. D., Lehner, R., Miksis, M. J., Toth, K., de Aguiar Vallim, T. Q., Koonin, E. V., Scorrano, L., & Pellegrini, L. (2021). Mitochondria-rough-ER contacts in the liver regulate systemic lipid homeostasis. Cell reports, 34(11), 108873. https://doi.org/10.1016/j.celrep.2021.108873
Anastasia, Irene, et al. "Mitochondria-rough-ER contacts in the liver regulate systemic lipid homeostasis." Cell reports vol. 34,11 (2021): 108873. doi: https://doi.org/10.1016/j.celrep.2021.108873
Anastasia I, Ilacqua N, Raimondi A, Lemieux P, Ghandehari-Alavijeh R, Faure G, Mekhedov SL, Williams KJ, Caicci F, Valle G, Giacomello M, Quiroga AD, Lehner R, Miksis MJ, Toth K, de Aguiar Vallim TQ, Koonin EV, Scorrano L, Pellegrini L. Mitochondria-rough-ER contacts in the liver regulate systemic lipid homeostasis. Cell Rep. 2021 Mar 16;34(11):108873. doi: 10.1016/j.celrep.2021.108873. PMID: 33730569.
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The Effects of Turnip (.

Animals : an open access journal from MDPI

Eghbaldost-Jadid R, Nosrati M, Rasouli B, Seidavi A, Phillips CJC.
PMID: 33803631
Animals (Basel). 2021 Mar 18;11(3). doi: 10.3390/ani11030867.

There are concerns about inclusion of antibiotics in the feed of broiler chickens, because of the development of antibiotic resistance, leading to a search for alternative feed additives. Turnip extract is known to have antibacterial properties but has not...

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Eghbaldost-Jadid R, Nosrati M, Rasouli B, et al. The Effects of Turnip (. Animals (Basel). 2021;11(3)doi: 10.3390/ani11030867.
Eghbaldost-Jadid, R., Nosrati, M., Rasouli, B., Seidavi, A., & Phillips, C. J. C. (2021). The Effects of Turnip (. Animals : an open access journal from MDPI, 11(3), . https://doi.org/10.3390/ani11030867
Eghbaldost-Jadid, Reza, et al. "The Effects of Turnip (." Animals : an open access journal from MDPI vol. 11,3 (2021). doi: https://doi.org/10.3390/ani11030867
Eghbaldost-Jadid R, Nosrati M, Rasouli B, Seidavi A, Phillips CJC. The Effects of Turnip (. Animals (Basel). 2021 Mar 18;11(3). doi: 10.3390/ani11030867. PMID: 33803631; PMCID: PMC8003009.
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Triglyceride-Rich Lipoproteins: Their Role in Atherosclerosis.

Revista de investigacion clinica; organo del Hospital de Enfermedades de la Nutricion

Castillo-Núñez Y, Morales-Villegas E, Aguilar-Salinas CA.
PMID: 34759386
Rev Invest Clin. 2021 Nov 10; doi: 10.24875/RIC.21000416. Epub 2021 Nov 10.

The term "triglyceride-rich lipoproteins" (TRLs) includes chylomicrons and their remnants, very low-density lipoproteins (VLDL), and intermediate-density lipoproteins (IDL). In this manuscript, the mechanisms by which atherogenic TRLs contribute to the formation of atheroma plaques are reviewed. Cholesterol from TRLs...

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Castillo-Núñez Y, Morales-Villegas E, Aguilar-Salinas CA. Triglyceride-Rich Lipoproteins: Their Role in Atherosclerosis. Rev Invest Clin. 2021;doi: 10.24875/RIC.21000416.
Castillo-Núñez, Y., Morales-Villegas, E., & Aguilar-Salinas, C. A. (2021). Triglyceride-Rich Lipoproteins: Their Role in Atherosclerosis. Revista de investigacion clinica; organo del Hospital de Enfermedades de la Nutricion, . https://doi.org/10.24875/RIC.21000416
Castillo-Núñez, Yulino, et al. "Triglyceride-Rich Lipoproteins: Their Role in Atherosclerosis." Revista de investigacion clinica; organo del Hospital de Enfermedades de la Nutricion vol. (2021). doi: https://doi.org/10.24875/RIC.21000416
Castillo-Núñez Y, Morales-Villegas E, Aguilar-Salinas CA. Triglyceride-Rich Lipoproteins: Their Role in Atherosclerosis. Rev Invest Clin. 2021 Nov 10; doi: 10.24875/RIC.21000416. Epub 2021 Nov 10. PMID: 34759386.
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Apolipoprotein E Content of Very Low-density Lipoprotein Limits Lipoprotein Lipase-mediated Triglyceride Hydrolysis.

Journal of lipid research

Whitacre BE, Howles P, Street S, Morris J, Swertfeger D, Davidson WS.
PMID: 34863862
J Lipid Res. 2021 Dec 01;100157. doi: 10.1016/j.jlr.2021.100157. Epub 2021 Dec 01.

High levels of circulating triglycerides (TG), or hypertriglyceridemia, is a key component of metabolic diseases such as type-II diabetes, metabolic syndrome, and cardiovascular disease. As TGs are carried by lipoproteins in plasma, hypertriglyceridemia can result from overproduction or lack...

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Whitacre BE, Howles P, Street S, et al. Apolipoprotein E Content of Very Low-density Lipoprotein Limits Lipoprotein Lipase-mediated Triglyceride Hydrolysis. J Lipid Res. 2021;100157doi: 10.1016/j.jlr.2021.100157.
Whitacre, B. E., Howles, P., Street, S., Morris, J., Swertfeger, D., & Davidson, W. S. (2021). Apolipoprotein E Content of Very Low-density Lipoprotein Limits Lipoprotein Lipase-mediated Triglyceride Hydrolysis. Journal of lipid research, 100157. https://doi.org/10.1016/j.jlr.2021.100157
Whitacre, Brynne E, et al. "Apolipoprotein E Content of Very Low-density Lipoprotein Limits Lipoprotein Lipase-mediated Triglyceride Hydrolysis." Journal of lipid research vol. (2021): 100157. doi: https://doi.org/10.1016/j.jlr.2021.100157
Whitacre BE, Howles P, Street S, Morris J, Swertfeger D, Davidson WS. Apolipoprotein E Content of Very Low-density Lipoprotein Limits Lipoprotein Lipase-mediated Triglyceride Hydrolysis. J Lipid Res. 2021 Dec 01;100157. doi: 10.1016/j.jlr.2021.100157. Epub 2021 Dec 01. PMID: 34863862.
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Showing 49 to 59 of 59 entries