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Calcium dysregulation increases right ventricular outflow tract arrhythmogenesis in rabbit model of chronic kidney disease.

Journal of cellular and molecular medicine

Huang SY, Chen YC, Kao YH, Lu YY, Lin YK, Higa S, Chen SA, Chen YJ.
PMID: 34761510
J Cell Mol Med. 2021 Dec;25(24):11264-11277. doi: 10.1111/jcmm.17052. Epub 2021 Nov 10.

Chronic kidney disease (CKD) increases the risk of arrhythmia. The right ventricular outflow tract (RVOT) is a crucial site of ventricular tachycardia (VT) origination. We hypothesize that CKD increases RVOT arrhythmogenesis through its effects on calcium dysregulation. We analysed...

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Huang SY, Chen YC, Kao YH, et al. Calcium dysregulation increases right ventricular outflow tract arrhythmogenesis in rabbit model of chronic kidney disease. J Cell Mol Med. 2021;25(24):11264-11277doi: 10.1111/jcmm.17052.
Huang, S. Y., Chen, Y. C., Kao, Y. H., Lu, Y. Y., Lin, Y. K., Higa, S., Chen, S. A., & Chen, Y. J. (2021). Calcium dysregulation increases right ventricular outflow tract arrhythmogenesis in rabbit model of chronic kidney disease. Journal of cellular and molecular medicine, 25(24), 11264-11277. https://doi.org/10.1111/jcmm.17052
Huang, Shih-Yu, et al. "Calcium dysregulation increases right ventricular outflow tract arrhythmogenesis in rabbit model of chronic kidney disease." Journal of cellular and molecular medicine vol. 25,24 (2021): 11264-11277. doi: https://doi.org/10.1111/jcmm.17052
Huang SY, Chen YC, Kao YH, Lu YY, Lin YK, Higa S, Chen SA, Chen YJ. Calcium dysregulation increases right ventricular outflow tract arrhythmogenesis in rabbit model of chronic kidney disease. J Cell Mol Med. 2021 Dec;25(24):11264-11277. doi: 10.1111/jcmm.17052. Epub 2021 Nov 10. PMID: 34761510; PMCID: PMC8650029.
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Oxytocin Protects Against Isoproterenol-Induced Cardiac Hypertrophy by Inhibiting PI3K/AKT Pathway via a lncRNA GAS5/miR-375-3p/KLF4-Dependent Mechanism.

Frontiers in pharmacology

Yang Y, Wang Z, Yao M, Xiong W, Wang J, Fang Y, Yang W, Jiang H, Song N, Liu L, Qian J.
PMID: 34925023
Front Pharmacol. 2021 Dec 03;12:766024. doi: 10.3389/fphar.2021.766024. eCollection 2021.

Cardiac hypertrophy is caused by cardiac volume or pressure overload conditions and ultimately leads to contractile dysfunction and heart failure. Oxytocin (OT), an endocrine nonapeptide, has been identified as a cardiovascular homeostatic hormone with anti-hypertrophic effects. However, the underlying...

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Yang Y, Wang Z, Yao M, et al. Oxytocin Protects Against Isoproterenol-Induced Cardiac Hypertrophy by Inhibiting PI3K/AKT Pathway via a lncRNA GAS5/miR-375-3p/KLF4-Dependent Mechanism. Front Pharmacol. 2021;12:766024doi: 10.3389/fphar.2021.766024.
Yang, Y., Wang, Z., Yao, M., Xiong, W., Wang, J., Fang, Y., Yang, W., Jiang, H., Song, N., Liu, L., & Qian, J. (2021). Oxytocin Protects Against Isoproterenol-Induced Cardiac Hypertrophy by Inhibiting PI3K/AKT Pathway via a lncRNA GAS5/miR-375-3p/KLF4-Dependent Mechanism. Frontiers in pharmacology, 12766024. https://doi.org/10.3389/fphar.2021.766024
Yang, Yuqiao, et al. "Oxytocin Protects Against Isoproterenol-Induced Cardiac Hypertrophy by Inhibiting PI3K/AKT Pathway via a lncRNA GAS5/miR-375-3p/KLF4-Dependent Mechanism." Frontiers in pharmacology vol. 12 (2021): 766024. doi: https://doi.org/10.3389/fphar.2021.766024
Yang Y, Wang Z, Yao M, Xiong W, Wang J, Fang Y, Yang W, Jiang H, Song N, Liu L, Qian J. Oxytocin Protects Against Isoproterenol-Induced Cardiac Hypertrophy by Inhibiting PI3K/AKT Pathway via a lncRNA GAS5/miR-375-3p/KLF4-Dependent Mechanism. Front Pharmacol. 2021 Dec 03;12:766024. doi: 10.3389/fphar.2021.766024. eCollection 2021. PMID: 34925023; PMCID: PMC8678504.
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Myocardial Infarction After High-Dose Catecholamine Application-A Case Report From an Experimental Imaging Study.

Frontiers in cardiovascular medicine

Beyhoff N, Lohr D, Thiele A, Foryst-Ludwig A, Klopfleisch R, Schreiber LM, Kintscher U.
PMID: 33330644
Front Cardiovasc Med. 2020 Nov 19;7:580296. doi: 10.3389/fcvm.2020.580296. eCollection 2020.

Although heart failure following myocardial infarction (MI) represents a major health burden, underlying microstructural and functional changes remain incompletely understood. Here, we report on a case of unexpected MI after treatment with the catecholamine isoproterenol in an experimental imaging...

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Beyhoff N, Lohr D, Thiele A, et al. Myocardial Infarction After High-Dose Catecholamine Application-A Case Report From an Experimental Imaging Study. Front Cardiovasc Med. 2020;7:580296doi: 10.3389/fcvm.2020.580296.
Beyhoff, N., Lohr, D., Thiele, A., Foryst-Ludwig, A., Klopfleisch, R., Schreiber, L. M., & Kintscher, U. (2020). Myocardial Infarction After High-Dose Catecholamine Application-A Case Report From an Experimental Imaging Study. Frontiers in cardiovascular medicine, 7580296. https://doi.org/10.3389/fcvm.2020.580296
Beyhoff, Niklas, et al. "Myocardial Infarction After High-Dose Catecholamine Application-A Case Report From an Experimental Imaging Study." Frontiers in cardiovascular medicine vol. 7 (2020): 580296. doi: https://doi.org/10.3389/fcvm.2020.580296
Beyhoff N, Lohr D, Thiele A, Foryst-Ludwig A, Klopfleisch R, Schreiber LM, Kintscher U. Myocardial Infarction After High-Dose Catecholamine Application-A Case Report From an Experimental Imaging Study. Front Cardiovasc Med. 2020 Nov 19;7:580296. doi: 10.3389/fcvm.2020.580296. eCollection 2020. PMID: 33330644; PMCID: PMC7717942.
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MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling.

Cell biology international

Bi X, Zhang Y, Yu Y, Yuan J, Xu S, Liu F, Ye J, Liu P.
PMID: 34854520
Cell Biol Int. 2021 Dec 02; doi: 10.1002/cbin.11731. Epub 2021 Dec 02.

MicroRNAs (miRNAs) regulate multiple biological processes and participate in various cardiovascular diseases. This study aims to investigate the role of miR-339-5p in cardiomyocyte hypertrophy and the involved mechanism. Neonatal rat cardiomyocytes (NRCMs) were cultured and stimulated with isoproterenol (ISO)....

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Bi X, Zhang Y, Yu Y, et al. MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell Biol Int. 2021;doi: 10.1002/cbin.11731.
Bi, X., Zhang, Y., Yu, Y., Yuan, J., Xu, S., Liu, F., Ye, J., & Liu, P. (2021). MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell biology international, . https://doi.org/10.1002/cbin.11731
Bi, Xueying, et al. "MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling." Cell biology international vol. (2021). doi: https://doi.org/10.1002/cbin.11731
Bi X, Zhang Y, Yu Y, Yuan J, Xu S, Liu F, Ye J, Liu P. MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell Biol Int. 2021 Dec 02; doi: 10.1002/cbin.11731. Epub 2021 Dec 02. PMID: 34854520.
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Comparison of the effects of adenosine, isoproterenol and their combinations on pulmonary transit time in rats using contrast echocardiography.

Medical ultrasonography

Su F, Shi YY, Wang B, Zheng XZ.
PMID: 34379710
Med Ultrason. 2021 Aug 04; doi: 10.11152/mu-3088. Epub 2021 Aug 04.

AIMS: To compare the effects of adenosine (Ade), isoproterenol (Iso) and their combinations on pulmonary transit time (PTT) in rats using contrast echocardiography.MATERIAL AND METHODS: Thirty-two adult Sprague Dawley (SD) rats were divided into four groups (n=8) according the...

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Su F, Shi YY, Wang B, et al. Comparison of the effects of adenosine, isoproterenol and their combinations on pulmonary transit time in rats using contrast echocardiography. Med Ultrason. 2021;doi: 10.11152/mu-3088.
Su, F., Shi, Y. Y., Wang, B., & Zheng, X. Z. (2021). Comparison of the effects of adenosine, isoproterenol and their combinations on pulmonary transit time in rats using contrast echocardiography. Medical ultrasonography, . https://doi.org/10.11152/mu-3088
Su, Feng, et al. "Comparison of the effects of adenosine, isoproterenol and their combinations on pulmonary transit time in rats using contrast echocardiography." Medical ultrasonography vol. (2021). doi: https://doi.org/10.11152/mu-3088
Su F, Shi YY, Wang B, Zheng XZ. Comparison of the effects of adenosine, isoproterenol and their combinations on pulmonary transit time in rats using contrast echocardiography. Med Ultrason. 2021 Aug 04; doi: 10.11152/mu-3088. Epub 2021 Aug 04. PMID: 34379710.
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Segmental analysis by speckle-tracking echocardiography of the left ventricle response to isoproterenol in male and female mice.

PeerJ

Walsh-Wilkinson E, Arsenault M, Couet J.
PMID: 33763310
PeerJ. 2021 Mar 12;9:e11085. doi: 10.7717/peerj.11085. eCollection 2021.

We studied by conventional and speckle-tracking echocardiography, the response of the left ventricle (LV) to a three-week continuous infusion of isoproterenol (Iso), a non-specific beta-adrenergic receptor agonist in male and female C57Bl6/J mice. Before and after Iso (30 mg/kg/day),...

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Walsh-Wilkinson E, Arsenault M, Couet J. Segmental analysis by speckle-tracking echocardiography of the left ventricle response to isoproterenol in male and female mice. PeerJ. 2021;9:e11085doi: 10.7717/peerj.11085.
Walsh-Wilkinson, E., Arsenault, M., & Couet, J. (2021). Segmental analysis by speckle-tracking echocardiography of the left ventricle response to isoproterenol in male and female mice. PeerJ, 9e11085. https://doi.org/10.7717/peerj.11085
Walsh-Wilkinson, Elisabeth, et al. "Segmental analysis by speckle-tracking echocardiography of the left ventricle response to isoproterenol in male and female mice." PeerJ vol. 9 (2021): e11085. doi: https://doi.org/10.7717/peerj.11085
Walsh-Wilkinson E, Arsenault M, Couet J. Segmental analysis by speckle-tracking echocardiography of the left ventricle response to isoproterenol in male and female mice. PeerJ. 2021 Mar 12;9:e11085. doi: 10.7717/peerj.11085. eCollection 2021. PMID: 33763310; PMCID: PMC7958899.
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Showing 721 to 726 of 726 entries