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Effect of Digoxin on Ventricular Remodeling and Responsiveness of beta-Adrenoceptors in Chronic Volume Overload.

Journal of cardiovascular pharmacology and therapeutics

Sullebarger JT, D'Ambra PM, Clark LC, Thanikarry L, Fontanet HL.
PMID: 10684510
J Cardiovasc Pharmacol Ther. 1998 Oct;3(4):281-290. doi: 10.1177/107424849800300403.

BACKGROUND: Digoxin improves baroreflex function and reduces neurohumoral activation in severe heart failure, but it is uncertain how digoxin affects ventricular remodeling and progression to left ventricular dysfunction. In addition, the effect of digoxin in in vitro beta-adrenoceptor density...

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Sullebarger JT, D'Ambra PM, Clark LC, et al. Effect of Digoxin on Ventricular Remodeling and Responsiveness of beta-Adrenoceptors in Chronic Volume Overload. J Cardiovasc Pharmacol Ther. 1998;3(4):281-290doi: 10.1177/107424849800300403.
Sullebarger, J. T., D'Ambra, P. M., Clark, L. C., Thanikarry, L., & Fontanet, H. L. (1998). Effect of Digoxin on Ventricular Remodeling and Responsiveness of beta-Adrenoceptors in Chronic Volume Overload. Journal of cardiovascular pharmacology and therapeutics, 3(4), 281-290. https://doi.org/10.1177/107424849800300403
Sullebarger, et al. "Effect of Digoxin on Ventricular Remodeling and Responsiveness of beta-Adrenoceptors in Chronic Volume Overload." Journal of cardiovascular pharmacology and therapeutics vol. 3,4 (1998): 281-290. doi: https://doi.org/10.1177/107424849800300403
Sullebarger JT, D'Ambra PM, Clark LC, Thanikarry L, Fontanet HL. Effect of Digoxin on Ventricular Remodeling and Responsiveness of beta-Adrenoceptors in Chronic Volume Overload. J Cardiovasc Pharmacol Ther. 1998 Oct;3(4):281-290. doi: 10.1177/107424849800300403. PMID: 10684510.
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Effect of isoflurane on the beta-adrenergic and endothelium-dependent relaxation of pig cerebral microvessels after cardiopulmonary bypass.

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association

Park KW, Dai HB, Lowenstein E, Stambler A, Sellke FW.
PMID: 17895077
J Stroke Cerebrovasc Dis. 1998 May-Jun;7(3):168-78. doi: 10.1016/s1052-3057(98)80003-7.

We examined the direct vasomotor effect of isoflurane as well as its effect on endothelium-dependent and beta-adrenergic vasodilation of cerebral microcirculation following either normothermic cardiopulmonary bypass (CPB) or profoundly hypothermic CPB with circulatory arrest. Pigs were placed on CPB;...

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Park KW, Dai HB, Lowenstein E, et al. Effect of isoflurane on the beta-adrenergic and endothelium-dependent relaxation of pig cerebral microvessels after cardiopulmonary bypass. J Stroke Cerebrovasc Dis. 1998;7(3):168-78doi: 10.1016/s1052-3057(98)80003-7.
Park, K. W., Dai, H. B., Lowenstein, E., Stambler, A., & Sellke, F. W. (1998). Effect of isoflurane on the beta-adrenergic and endothelium-dependent relaxation of pig cerebral microvessels after cardiopulmonary bypass. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association, 7(3), 168-78. https://doi.org/10.1016/s1052-3057(98)80003-7
Park, K W, et al. "Effect of isoflurane on the beta-adrenergic and endothelium-dependent relaxation of pig cerebral microvessels after cardiopulmonary bypass." Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association vol. 7,3 (1998): 168-78. doi: https://doi.org/10.1016/s1052-3057(98)80003-7
Park KW, Dai HB, Lowenstein E, Stambler A, Sellke FW. Effect of isoflurane on the beta-adrenergic and endothelium-dependent relaxation of pig cerebral microvessels after cardiopulmonary bypass. J Stroke Cerebrovasc Dis. 1998 May-Jun;7(3):168-78. doi: 10.1016/s1052-3057(98)80003-7. PMID: 17895077.
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Identification of a switching model of calcium cycling in isolated rat hearts.

Conference proceedings : ... Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual Conference

Op den Buijs J, Juloski ALj, Ivanics T, Ligeti L, van der Vusse GJ, van Riel NA.
PMID: 17271808
Conf Proc IEEE Eng Med Biol Soc. 2004;2004:841-4. doi: 10.1109/IEMBS.2004.1403289.

So far, the processes involved in regulation of intracellular calcium (Ca/sub i//sup 2+/) in cardiomyocytes have been mainly studied through biochemical and isolated cell analysis. Here, we present a novel technique to model and identify cardiac Ca/sub i//sup 2+/-cycling...

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Op den Buijs J, Juloski ALj, Ivanics T, et al. Identification of a switching model of calcium cycling in isolated rat hearts. Conf Proc IEEE Eng Med Biol Soc. 2004;2004:841-4doi: 10.1109/IEMBS.2004.1403289.
Op den Buijs, J., Juloski, A. L. j., Ivanics, T., Ligeti, L., van der Vusse, G. J., & van Riel, N. A. (2004). Identification of a switching model of calcium cycling in isolated rat hearts. Conference proceedings : ... Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual Conference, 2004841-4. https://doi.org/10.1109/IEMBS.2004.1403289
Op den Buijs, J, et al. "Identification of a switching model of calcium cycling in isolated rat hearts." Conference proceedings : ... Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual Conference vol. 2004 (2004): 841-4. doi: https://doi.org/10.1109/IEMBS.2004.1403289
Op den Buijs J, Juloski ALj, Ivanics T, Ligeti L, van der Vusse GJ, van Riel NA. Identification of a switching model of calcium cycling in isolated rat hearts. Conf Proc IEEE Eng Med Biol Soc. 2004;2004:841-4. doi: 10.1109/IEMBS.2004.1403289. PMID: 17271808.
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Membrane fluidity changes are associated with the antiarrhythmic effects of docosahexaenoic acid in adult rat cardiomyocytes.

The Journal of nutritional biochemistry

Leifert WR, Jahangiri A, McMurchie EJ.
PMID: 15539341
J Nutr Biochem. 2000 Jan;11(1):38-44. doi: 10.1016/s0955-2863(99)00069-8.

Previous studies using neonatal rat cardiomyocytes have reported antiarrhythmic effects of long-chain polyunsaturated fatty acids (PUFAs). In this study, we examined the effects of the n-3 polyunsaturated fatty acid docosahexaenoic acid (DHA) on the spontaneous contractile activity and membrane...

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Leifert WR, Jahangiri A, McMurchie EJ. Membrane fluidity changes are associated with the antiarrhythmic effects of docosahexaenoic acid in adult rat cardiomyocytes. J Nutr Biochem. 2000;11(1):38-44doi: 10.1016/s0955-2863(99)00069-8.
Leifert, W. R., Jahangiri, A., & McMurchie, E. J. (2000). Membrane fluidity changes are associated with the antiarrhythmic effects of docosahexaenoic acid in adult rat cardiomyocytes. The Journal of nutritional biochemistry, 11(1), 38-44. https://doi.org/10.1016/s0955-2863(99)00069-8
Leifert, W R, et al. "Membrane fluidity changes are associated with the antiarrhythmic effects of docosahexaenoic acid in adult rat cardiomyocytes." The Journal of nutritional biochemistry vol. 11,1 (2000): 38-44. doi: https://doi.org/10.1016/s0955-2863(99)00069-8
Leifert WR, Jahangiri A, McMurchie EJ. Membrane fluidity changes are associated with the antiarrhythmic effects of docosahexaenoic acid in adult rat cardiomyocytes. J Nutr Biochem. 2000 Jan;11(1):38-44. doi: 10.1016/s0955-2863(99)00069-8. PMID: 15539341.
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Differences in Cyclic AMP Changes after Stimulation by Prostaglandins and Isoproterenol in Lymphocyte Subpopulations.

The Journal of clinical investigation

Bach MA.
PMID: 16695962
J Clin Invest. 1975 May;55(5):1074-81. doi: 10.1172/JCI108008.

Various lymphocyte populations have been studied for their content in cyclic adenosine 3',5'-monophosphate (cAMP) before and after stimulation by isoproterenol and prostaglandin E(1) (PGE(1)). Basal cAMP levels vary among lymphocytes according to their origin: peripheral blood lymphocytes show high...

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Bach MA. Differences in Cyclic AMP Changes after Stimulation by Prostaglandins and Isoproterenol in Lymphocyte Subpopulations. J Clin Invest. 1975;55(5):1074-81doi: 10.1172/JCI108008.
Bach, M. A. (1975). Differences in Cyclic AMP Changes after Stimulation by Prostaglandins and Isoproterenol in Lymphocyte Subpopulations. The Journal of clinical investigation, 55(5), 1074-81. https://doi.org/10.1172/JCI108008
Bach, M A. "Differences in Cyclic AMP Changes after Stimulation by Prostaglandins and Isoproterenol in Lymphocyte Subpopulations." The Journal of clinical investigation vol. 55,5 (1975): 1074-81. doi: https://doi.org/10.1172/JCI108008
Bach MA. Differences in Cyclic AMP Changes after Stimulation by Prostaglandins and Isoproterenol in Lymphocyte Subpopulations. J Clin Invest. 1975 May;55(5):1074-81. doi: 10.1172/JCI108008. PMID: 16695962; PMCID: PMC301854.
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Conformation changes of albumin in its interaction with physiologically active compounds as studied by quasi-elastic light scattering spectroscopy and ultrasonic method.

Talanta

Hushcha TO, Luik AI, Naboka YN.
PMID: 18968085
Talanta. 2000 Oct 02;53(1):29-34. doi: 10.1016/s0039-9140(00)00454-9.

The effect of pH and binding of ten physiologically active compounds (PAC) on conformational organization of human serum albumin (HSA) in aqueous solutions has been studied using two different methods. The hydrodynamic sizes of albumin globule and its subunits...

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Hushcha TO, Luik AI, Naboka YN. Conformation changes of albumin in its interaction with physiologically active compounds as studied by quasi-elastic light scattering spectroscopy and ultrasonic method. Talanta. 2000;53(1):29-34doi: 10.1016/s0039-9140(00)00454-9.
Hushcha, T. O., Luik, A. I., & Naboka, Y. N. (2000). Conformation changes of albumin in its interaction with physiologically active compounds as studied by quasi-elastic light scattering spectroscopy and ultrasonic method. Talanta, 53(1), 29-34. https://doi.org/10.1016/s0039-9140(00)00454-9
Hushcha, T O, et al. "Conformation changes of albumin in its interaction with physiologically active compounds as studied by quasi-elastic light scattering spectroscopy and ultrasonic method." Talanta vol. 53,1 (2000): 29-34. doi: https://doi.org/10.1016/s0039-9140(00)00454-9
Hushcha TO, Luik AI, Naboka YN. Conformation changes of albumin in its interaction with physiologically active compounds as studied by quasi-elastic light scattering spectroscopy and ultrasonic method. Talanta. 2000 Oct 02;53(1):29-34. doi: 10.1016/s0039-9140(00)00454-9. PMID: 18968085.
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Platelet-derived NO slows thrombus growth on a collagen type III surface.

Thrombosis journal

Williams RH, Nollert MU.
PMID: 15544706
Thromb J. 2004 Nov 15;2(1):11. doi: 10.1186/1477-9560-2-11.

Nitric oxide (NO) is a free radical that plays an important role in modulating platelet adhesion and aggregation. Platelets are a source of vascular NO, but since erythrocytes avidly scavenge NO, the functional significance of platelet-derived NO is not...

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Williams RH, Nollert MU. Platelet-derived NO slows thrombus growth on a collagen type III surface. Thromb J. 2004;2(1):11doi: 10.1186/1477-9560-2-11.
Williams, R. H., & Nollert, M. U. (2004). Platelet-derived NO slows thrombus growth on a collagen type III surface. Thrombosis journal, 2(1), 11. https://doi.org/10.1186/1477-9560-2-11
Williams, Robert H, and Nollert, Matthias U. "Platelet-derived NO slows thrombus growth on a collagen type III surface." Thrombosis journal vol. 2,1 (2004): 11. doi: https://doi.org/10.1186/1477-9560-2-11
Williams RH, Nollert MU. Platelet-derived NO slows thrombus growth on a collagen type III surface. Thromb J. 2004 Nov 15;2(1):11. doi: 10.1186/1477-9560-2-11. PMID: 15544706; PMCID: PMC535342.
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Alterations of cardiac and lymphocyte beta- adrenoceptors in rat with chronic heart failure.

Science in China. Series C, Life sciences

Zhang P, Han Q, Zhang Y, Xu K, Tian B, Lü Z, Guo J, Chen M.
PMID: 18726306
Sci China C Life Sci. 1997 Apr;40(2):113-21. doi: 10.1007/BF02882038.

The alterations of cardiac and lymphocyte beta-adrenoceptors were observed in the rats with chronic heart failure produced by constriction of both abdominal aorta and renal artery. The results showed that beta(1)-adrenoceptor density and mRNA levels were increased, whereas these...

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Zhang P, Han Q, Zhang Y, et al. Alterations of cardiac and lymphocyte beta- adrenoceptors in rat with chronic heart failure. Sci China C Life Sci. 1997;40(2):113-21doi: 10.1007/BF02882038.
Zhang, P., Han, Q., Zhang, Y., Xu, K., Tian, B., Lü, Z., Guo, J., & Chen, M. (1997). Alterations of cardiac and lymphocyte beta- adrenoceptors in rat with chronic heart failure. Science in China. Series C, Life sciences, 40(2), 113-21. https://doi.org/10.1007/BF02882038
Zhang, P, et al. "Alterations of cardiac and lymphocyte beta- adrenoceptors in rat with chronic heart failure." Science in China. Series C, Life sciences vol. 40,2 (1997): 113-21. doi: https://doi.org/10.1007/BF02882038
Zhang P, Han Q, Zhang Y, Xu K, Tian B, Lü Z, Guo J, Chen M. Alterations of cardiac and lymphocyte beta- adrenoceptors in rat with chronic heart failure. Sci China C Life Sci. 1997 Apr;40(2):113-21. doi: 10.1007/BF02882038. PMID: 18726306.
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Beta-adrenergic receptor activation in immortalized human urothelial cells stimulates inflammatory responses by PKA-independent mechanisms.

Cell communication and signaling : CCS

Harmon EB, Porter JM, Porter JE.
PMID: 16091148
Cell Commun Signal. 2005 Aug 09;3:10. doi: 10.1186/1478-811X-3-10.

BACKGROUND: Interstitial cystitis (IC) is a debilitating disease characterized by chronic inflammation of the urinary bladder, yet specific cellular mechanisms of inflammation in IC are largely unknown. Multiple lines of evidence suggest that beta-adrenergic receptor (AR) signaling is increased...

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Harmon EB, Porter JM, Porter JE. Beta-adrenergic receptor activation in immortalized human urothelial cells stimulates inflammatory responses by PKA-independent mechanisms. Cell Commun Signal. 2005;3:10doi: 10.1186/1478-811X-3-10.
Harmon, E. B., Porter, J. M., & Porter, J. E. (2005). Beta-adrenergic receptor activation in immortalized human urothelial cells stimulates inflammatory responses by PKA-independent mechanisms. Cell communication and signaling : CCS, 310. https://doi.org/10.1186/1478-811X-3-10
Harmon, Erin B, et al. "Beta-adrenergic receptor activation in immortalized human urothelial cells stimulates inflammatory responses by PKA-independent mechanisms." Cell communication and signaling : CCS vol. 3 (2005): 10. doi: https://doi.org/10.1186/1478-811X-3-10
Harmon EB, Porter JM, Porter JE. Beta-adrenergic receptor activation in immortalized human urothelial cells stimulates inflammatory responses by PKA-independent mechanisms. Cell Commun Signal. 2005 Aug 09;3:10. doi: 10.1186/1478-811X-3-10. PMID: 16091148; PMCID: PMC1198236.
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Parallel decrease of adenylyl cyclase activity and beta1-adrenoceptor density in brain cortex of aging mice.

Archives of gerontology and geriatrics

Viticchi C, Piantanelli L.
PMID: 15374344
Arch Gerontol Geriatr. 1993 Mar-Apr;16(2):141-8. doi: 10.1016/0167-4943(93)90005-3.

Beta-adrenergic receptors (betaARs) and adenylyl cyclase (AC) activity have been found to undergo progressive alterations in the brain cortex of aging mice. In particular, betaAR changes are in charge of the beta1 subpopulation (beta1AR), beta2-adrenoceptors (beta2ARs) showing no age-related...

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Viticchi C, Piantanelli L. Parallel decrease of adenylyl cyclase activity and beta1-adrenoceptor density in brain cortex of aging mice. Arch Gerontol Geriatr. 1993;16(2):141-8doi: 10.1016/0167-4943(93)90005-3.
Viticchi, C., & Piantanelli, L. (1993). Parallel decrease of adenylyl cyclase activity and beta1-adrenoceptor density in brain cortex of aging mice. Archives of gerontology and geriatrics, 16(2), 141-8. https://doi.org/10.1016/0167-4943(93)90005-3
Viticchi, C, and Piantanelli, L. "Parallel decrease of adenylyl cyclase activity and beta1-adrenoceptor density in brain cortex of aging mice." Archives of gerontology and geriatrics vol. 16,2 (1993): 141-8. doi: https://doi.org/10.1016/0167-4943(93)90005-3
Viticchi C, Piantanelli L. Parallel decrease of adenylyl cyclase activity and beta1-adrenoceptor density in brain cortex of aging mice. Arch Gerontol Geriatr. 1993 Mar-Apr;16(2):141-8. doi: 10.1016/0167-4943(93)90005-3. PMID: 15374344.
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beta-adrenergic modulation of in vivo long-term potentiation in area CA1 and its role in spatial learning in rats.

Science in China. Series C, Life sciences

Ji J, Zhang X, Li B.
PMID: 18758717
Sci China C Life Sci. 2003 Dec;46(6):605-14. doi: 10.1360/02yc0243.

Activation of beta-adrenoceptors in area CA1 of the hippocampus facilitates in vitro long-term potentiation (LTP) in this region. However, it is unclear if in vivo LTP in area CA1 and hippocampus-dependent learning are subjected to beta-adrenergic regulation. To address...

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Ji J, Zhang X, Li B. beta-adrenergic modulation of in vivo long-term potentiation in area CA1 and its role in spatial learning in rats. Sci China C Life Sci. 2003;46(6):605-14doi: 10.1360/02yc0243.
Ji, J., Zhang, X., & Li, B. (2003). beta-adrenergic modulation of in vivo long-term potentiation in area CA1 and its role in spatial learning in rats. Science in China. Series C, Life sciences, 46(6), 605-14. https://doi.org/10.1360/02yc0243
Ji, Jinzhao, et al. "beta-adrenergic modulation of in vivo long-term potentiation in area CA1 and its role in spatial learning in rats." Science in China. Series C, Life sciences vol. 46,6 (2003): 605-14. doi: https://doi.org/10.1360/02yc0243
Ji J, Zhang X, Li B. beta-adrenergic modulation of in vivo long-term potentiation in area CA1 and its role in spatial learning in rats. Sci China C Life Sci. 2003 Dec;46(6):605-14. doi: 10.1360/02yc0243. PMID: 18758717.
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'Oxygen toxicity' induced by isoproterenol oxidation on living cells. An in vitro prokaryotic model.

Archives of gerontology and geriatrics

Giunta S, Galeazzi L, Groppa G.
PMID: 15374515
Arch Gerontol Geriatr. 1990 Mar-Apr;10(2):207-16. doi: 10.1016/0167-4943(90)90020-7.

Oxygen radical damage is a relevant problem in gerontological research. It has been implicated both in the aging process itself and in aging-related pathologies. Oxygen radicals from catecholamines seem to play an important role in central nervous system and...

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Giunta S, Galeazzi L, Groppa G. 'Oxygen toxicity' induced by isoproterenol oxidation on living cells. An in vitro prokaryotic model. Arch Gerontol Geriatr. 1990;10(2):207-16doi: 10.1016/0167-4943(90)90020-7.
Giunta, S., Galeazzi, L., & Groppa, G. (1990). 'Oxygen toxicity' induced by isoproterenol oxidation on living cells. An in vitro prokaryotic model. Archives of gerontology and geriatrics, 10(2), 207-16. https://doi.org/10.1016/0167-4943(90)90020-7
Giunta, S, et al. "'Oxygen toxicity' induced by isoproterenol oxidation on living cells. An in vitro prokaryotic model." Archives of gerontology and geriatrics vol. 10,2 (1990): 207-16. doi: https://doi.org/10.1016/0167-4943(90)90020-7
Giunta S, Galeazzi L, Groppa G. 'Oxygen toxicity' induced by isoproterenol oxidation on living cells. An in vitro prokaryotic model. Arch Gerontol Geriatr. 1990 Mar-Apr;10(2):207-16. doi: 10.1016/0167-4943(90)90020-7. PMID: 15374515.
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Showing 25 to 36 of 726 entries