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Molecular dynamics simulations of membrane proteins.

Biophysical reviews

Baştuğ T, Kuyucak S.
PMID: 28510077
Biophys Rev. 2012 Sep;4(3):271-282. doi: 10.1007/s12551-012-0084-9. Epub 2012 Sep 01.

Membrane proteins control the traffic across cell membranes and thereby play an essential role in cell function from transport of various solutes to immune response via molecular recognition. Because it is very difficult to determine the structures of membrane...

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Baştuğ T, Kuyucak S. Molecular dynamics simulations of membrane proteins. Biophys Rev. 2012;4(3):271-282doi: 10.1007/s12551-012-0084-9.
Baştuğ, T., & Kuyucak, S. (2012). Molecular dynamics simulations of membrane proteins. Biophysical reviews, 4(3), 271-282. https://doi.org/10.1007/s12551-012-0084-9
Baştuğ, Turgut, and Kuyucak, Serdar. "Molecular dynamics simulations of membrane proteins." Biophysical reviews vol. 4,3 (2012): 271-282. doi: https://doi.org/10.1007/s12551-012-0084-9
Baştuğ T, Kuyucak S. Molecular dynamics simulations of membrane proteins. Biophys Rev. 2012 Sep;4(3):271-282. doi: 10.1007/s12551-012-0084-9. Epub 2012 Sep 01. PMID: 28510077; PMCID: PMC5418402.
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Antiepileptic Drugs Impair Shortening of Isolated Cardiomyocytes.

Frontiers in neurology

Hulbert J, Elger CE, Meyer R, Surges R.
PMID: 28421035
Front Neurol. 2017 Apr 03;8:133. doi: 10.3389/fneur.2017.00133. eCollection 2017.

BACKGROUND: Most antiepileptic drugs (AEDs) inhibit seizure generation by acting on voltage-dependent ion channels. Voltage-dependent sodium and calcium channels are commonly expressed in brain and heart, suggesting that AEDs may have considerable cardiodepressive effects, thereby facilitating sudden cardiac death...

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Hulbert J, Elger CE, Meyer R, et al. Antiepileptic Drugs Impair Shortening of Isolated Cardiomyocytes. Front Neurol. 2017;8:133doi: 10.3389/fneur.2017.00133.
Hulbert, J., Elger, C. E., Meyer, R., & Surges, R. (2017). Antiepileptic Drugs Impair Shortening of Isolated Cardiomyocytes. Frontiers in neurology, 8133. https://doi.org/10.3389/fneur.2017.00133
Hulbert, Johanna, et al. "Antiepileptic Drugs Impair Shortening of Isolated Cardiomyocytes." Frontiers in neurology vol. 8 (2017): 133. doi: https://doi.org/10.3389/fneur.2017.00133
Hulbert J, Elger CE, Meyer R, Surges R. Antiepileptic Drugs Impair Shortening of Isolated Cardiomyocytes. Front Neurol. 2017 Apr 03;8:133. doi: 10.3389/fneur.2017.00133. eCollection 2017. PMID: 28421035; PMCID: PMC5376916.
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The Jackprot Simulation Couples Mutation Rate with Natural Selection to Illustrate How Protein Evolution Is Not Random.

Evolution

Paz-Y-Miño C G, Espinosa A, Bai CY.
PMID: 26973733
Evolution (N Y). 2011 Sep;4(3):502-514. doi: 10.1007/s12052-011-0329-2. Epub 2011 Mar 24.

Protein evolution is not a random process. Views which attribute randomness to molecular change, deleterious nature to single-gene mutations, insufficient geological time, or population size for molecular improvements to occur, or invoke "design creationism" to account for complexity in...

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Paz-Y-Miño C G, Espinosa A, Bai CY. The Jackprot Simulation Couples Mutation Rate with Natural Selection to Illustrate How Protein Evolution Is Not Random. Evolution (N Y). 2011;4(3):502-514doi: 10.1007/s12052-011-0329-2.
Paz-Y-Miño C, G., Espinosa, A., & Bai, C. Y. (2011). The Jackprot Simulation Couples Mutation Rate with Natural Selection to Illustrate How Protein Evolution Is Not Random. Evolution, 4(3), 502-514. https://doi.org/10.1007/s12052-011-0329-2
Paz-Y-Miño C, Guillermo, et al. "The Jackprot Simulation Couples Mutation Rate with Natural Selection to Illustrate How Protein Evolution Is Not Random." Evolution vol. 4,3 (2011): 502-514. doi: https://doi.org/10.1007/s12052-011-0329-2
Paz-Y-Miño C G, Espinosa A, Bai CY. The Jackprot Simulation Couples Mutation Rate with Natural Selection to Illustrate How Protein Evolution Is Not Random. Evolution (N Y). 2011 Sep;4(3):502-514. doi: 10.1007/s12052-011-0329-2. Epub 2011 Mar 24. PMID: 26973733; PMCID: PMC4785801.
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Therapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug Discovery.

Frontiers in pharmacology

Imbrici P, Liantonio A, Camerino GM, De Bellis M, Camerino C, Mele A, Giustino A, Pierno S, De Luca A, Tricarico D, Desaphy JF, Conte D.
PMID: 27242528
Front Pharmacol. 2016 May 10;7:121. doi: 10.3389/fphar.2016.00121. eCollection 2016.

In the human genome more than 400 genes encode ion channels, which are transmembrane proteins mediating ion fluxes across membranes. Being expressed in all cell types, they are involved in almost all physiological processes, including sense perception, neurotransmission, muscle...

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Imbrici P, Liantonio A, Camerino GM, et al. Therapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug Discovery. Front Pharmacol. 2016;7:121doi: 10.3389/fphar.2016.00121.
Imbrici, P., Liantonio, A., Camerino, G. M., De Bellis, M., Camerino, C., Mele, A., Giustino, A., Pierno, S., De Luca, A., Tricarico, D., Desaphy, J. F., & Conte, D. (2016). Therapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug Discovery. Frontiers in pharmacology, 7121. https://doi.org/10.3389/fphar.2016.00121
Imbrici, Paola, et al. "Therapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug Discovery." Frontiers in pharmacology vol. 7 (2016): 121. doi: https://doi.org/10.3389/fphar.2016.00121
Imbrici P, Liantonio A, Camerino GM, De Bellis M, Camerino C, Mele A, Giustino A, Pierno S, De Luca A, Tricarico D, Desaphy JF, Conte D. Therapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug Discovery. Front Pharmacol. 2016 May 10;7:121. doi: 10.3389/fphar.2016.00121. eCollection 2016. PMID: 27242528; PMCID: PMC4861771.
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Acupuncture for visceral pain: neural substrates and potential mechanisms.

Evidence-based complementary and alternative medicine : eCAM

Chen S, Wang S, Rong P, Wang J, Qiao L, Feng X, Liu J, Zhang J.
PMID: 25614752
Evid Based Complement Alternat Med. 2014;2014:609594. doi: 10.1155/2014/609594. Epub 2014 Dec 29.

Visceral pain is the most common form of pain caused by varied diseases and a major reason for patients to seek medical consultation. Despite much advances, the pathophysiological mechanism is still poorly understood comparing with its somatic counterpart and,...

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Chen S, Wang S, Rong P, et al. Acupuncture for visceral pain: neural substrates and potential mechanisms. Evid Based Complement Alternat Med. 2014;2014:609594doi: 10.1155/2014/609594.
Chen, S., Wang, S., Rong, P., Wang, J., Qiao, L., Feng, X., Liu, J., & Zhang, J. (2014). Acupuncture for visceral pain: neural substrates and potential mechanisms. Evidence-based complementary and alternative medicine : eCAM, 2014609594. https://doi.org/10.1155/2014/609594
Chen, Shuping, et al. "Acupuncture for visceral pain: neural substrates and potential mechanisms." Evidence-based complementary and alternative medicine : eCAM vol. 2014 (2014): 609594. doi: https://doi.org/10.1155/2014/609594
Chen S, Wang S, Rong P, Wang J, Qiao L, Feng X, Liu J, Zhang J. Acupuncture for visceral pain: neural substrates and potential mechanisms. Evid Based Complement Alternat Med. 2014;2014:609594. doi: 10.1155/2014/609594. Epub 2014 Dec 29. PMID: 25614752; PMCID: PMC4295157.
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KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing.

Archives of plastic surgery

Shim JH, Lim JW, Kim BK, Park SJ, Kim SW, Choi TH.
PMID: 25606484
Arch Plast Surg. 2015 Jan;42(1):11-9. doi: 10.5999/aps.2015.42.1.11. Epub 2015 Jan 14.

BACKGROUND: Wound healing is an interaction of a complex signaling cascade of cellular events, including inflammation, proliferation, and maturation. K(+) channels modulate the mitogen-activated protein kinase (MAPK) signaling pathway. Here, we investigated whether K(+) channel-activated MAPK signaling directs collagen...

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Shim JH, Lim JW, Kim BK, et al. KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing. Arch Plast Surg. 2015;42(1):11-9doi: 10.5999/aps.2015.42.1.11.
Shim, J. H., Lim, J. W., Kim, B. K., Park, S. J., Kim, S. W., & Choi, T. H. (2015). KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing. Archives of plastic surgery, 42(1), 11-9. https://doi.org/10.5999/aps.2015.42.1.11
Shim, Jung Hee, et al. "KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing." Archives of plastic surgery vol. 42,1 (2015): 11-9. doi: https://doi.org/10.5999/aps.2015.42.1.11
Shim JH, Lim JW, Kim BK, Park SJ, Kim SW, Choi TH. KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing. Arch Plast Surg. 2015 Jan;42(1):11-9. doi: 10.5999/aps.2015.42.1.11. Epub 2015 Jan 14. PMID: 25606484; PMCID: PMC4297800.
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A Toolkit for Orthogonal and in vivo Optical Manipulation of Ionotropic Glutamate Receptors.

Frontiers in molecular neuroscience

Levitz J, Popescu AT, Reiner A, Isacoff EY.
PMID: 26869877
Front Mol Neurosci. 2016 Feb 02;9:2. doi: 10.3389/fnmol.2016.00002. eCollection 2016.

The ability to optically manipulate specific neuronal signaling proteins with genetic precision paves the way for the dissection of their roles in brain function, behavior, and disease. Chemical optogenetic control with photoswitchable tethered ligands (PTLs) enables rapid, reversible and...

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Levitz J, Popescu AT, Reiner A, et al. A Toolkit for Orthogonal and in vivo Optical Manipulation of Ionotropic Glutamate Receptors. Front Mol Neurosci. 2016;9:2doi: 10.3389/fnmol.2016.00002.
Levitz, J., Popescu, A. T., Reiner, A., & Isacoff, E. Y. (2016). A Toolkit for Orthogonal and in vivo Optical Manipulation of Ionotropic Glutamate Receptors. Frontiers in molecular neuroscience, 92. https://doi.org/10.3389/fnmol.2016.00002
Levitz, Joshua, et al. "A Toolkit for Orthogonal and in vivo Optical Manipulation of Ionotropic Glutamate Receptors." Frontiers in molecular neuroscience vol. 9 (2016): 2. doi: https://doi.org/10.3389/fnmol.2016.00002
Levitz J, Popescu AT, Reiner A, Isacoff EY. A Toolkit for Orthogonal and in vivo Optical Manipulation of Ionotropic Glutamate Receptors. Front Mol Neurosci. 2016 Feb 02;9:2. doi: 10.3389/fnmol.2016.00002. eCollection 2016. PMID: 26869877; PMCID: PMC4735401.
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Mechanosensitive Ion Channels in Cardiovascular Physiology.

Experimental and clinical cardiology

Teng J, Loukin S, Kung C.
PMID: 26778915
Exp Clin Cardiol. 2014;20(10):6550-6560. Epub 2014 Oct 29.

EC coupling is subjected to a mechanical feedback, which originates from physical force-sensing ion channels in the pericardium and elsewhere. Reviewed here are the most recent developments that greatly advanced our understanding of these mechanosensitive (MS) channels, including TRPs...

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Teng J, Loukin S, Kung C. Mechanosensitive Ion Channels in Cardiovascular Physiology. Exp Clin Cardiol. 2014;20(10):6550-6560
Teng, J., Loukin, S., & Kung, C. (2014). Mechanosensitive Ion Channels in Cardiovascular Physiology. Experimental and clinical cardiology, 20(10), 6550-6560.
Teng, Jinfeng, et al. "Mechanosensitive Ion Channels in Cardiovascular Physiology." Experimental and clinical cardiology vol. 20,10 (2014): 6550-6560.
Teng J, Loukin S, Kung C. Mechanosensitive Ion Channels in Cardiovascular Physiology. Exp Clin Cardiol. 2014;20(10):6550-6560. Epub 2014 Oct 29. PMID: 26778915; PMCID: PMC4713040.
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Differences in Left Versus Right Ventricular Electrophysiological Properties in Cardiac Dysfunction and Arrhythmogenesis.

Arrhythmia & electrophysiology review

Molina CE, Heijman J, Dobrev D.
PMID: 27403288
Arrhythm Electrophysiol Rev. 2016 May;5(1):14-9. doi: 10.15420/aer.2016.8.2.

A wide range of ion channels, transporters, signaling pathways and tissue structure at a microscopic and macroscopic scale regulate the electrophysiological activity of the heart. Each region of the heart has optimised these properties based on its specific role...

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Molina CE, Heijman J, Dobrev D. Differences in Left Versus Right Ventricular Electrophysiological Properties in Cardiac Dysfunction and Arrhythmogenesis. Arrhythm Electrophysiol Rev. 2016;5(1):14-9doi: 10.15420/aer.2016.8.2.
Molina, C. E., Heijman, J., & Dobrev, D. (2016). Differences in Left Versus Right Ventricular Electrophysiological Properties in Cardiac Dysfunction and Arrhythmogenesis. Arrhythmia & electrophysiology review, 5(1), 14-9. https://doi.org/10.15420/aer.2016.8.2
Molina, Cristina E, et al. "Differences in Left Versus Right Ventricular Electrophysiological Properties in Cardiac Dysfunction and Arrhythmogenesis." Arrhythmia & electrophysiology review vol. 5,1 (2016): 14-9. doi: https://doi.org/10.15420/aer.2016.8.2
Molina CE, Heijman J, Dobrev D. Differences in Left Versus Right Ventricular Electrophysiological Properties in Cardiac Dysfunction and Arrhythmogenesis. Arrhythm Electrophysiol Rev. 2016 May;5(1):14-9. doi: 10.15420/aer.2016.8.2. PMID: 27403288; PMCID: PMC4939305.
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Gene Network Construction from Microarray Data Identifies a Key Network Module and Several Candidate Hub Genes in Age-Associated Spatial Learning Impairment.

Frontiers in systems neuroscience

Uddin R, Singh SM.
PMID: 29066959
Front Syst Neurosci. 2017 Oct 10;11:75. doi: 10.3389/fnsys.2017.00075. eCollection 2017.

As humans age many suffer from a decrease in normal brain functions including spatial learning impairments. This study aimed to better understand the molecular mechanisms in age-associated spatial learning impairment (ASLI). We used a mathematical modeling approach implemented in...

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Uddin R, Singh SM. Gene Network Construction from Microarray Data Identifies a Key Network Module and Several Candidate Hub Genes in Age-Associated Spatial Learning Impairment. Front Syst Neurosci. 2017;11:75doi: 10.3389/fnsys.2017.00075.
Uddin, R., & Singh, S. M. (2017). Gene Network Construction from Microarray Data Identifies a Key Network Module and Several Candidate Hub Genes in Age-Associated Spatial Learning Impairment. Frontiers in systems neuroscience, 1175. https://doi.org/10.3389/fnsys.2017.00075
Uddin, Raihan, and Singh, Shiva M. "Gene Network Construction from Microarray Data Identifies a Key Network Module and Several Candidate Hub Genes in Age-Associated Spatial Learning Impairment." Frontiers in systems neuroscience vol. 11 (2017): 75. doi: https://doi.org/10.3389/fnsys.2017.00075
Uddin R, Singh SM. Gene Network Construction from Microarray Data Identifies a Key Network Module and Several Candidate Hub Genes in Age-Associated Spatial Learning Impairment. Front Syst Neurosci. 2017 Oct 10;11:75. doi: 10.3389/fnsys.2017.00075. eCollection 2017. PMID: 29066959; PMCID: PMC5641338.
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Chloride: essential micronutrient and multifunctional beneficial ion.

Journal of experimental botany

Raven JA.
PMID: 28040799
J Exp Bot. 2017 Jan;38(3):359-367. doi: 10.1093/jxb/erw421. Epub 2016 Dec 30.

Cl− is an essential micronutrient for oxygenic photolithotrophs. About half of global primary productivity is carried out by oxygenic photolithotrophs exposed to saline waters with Cl− concentrations orders of magnitude higher than that needed to satisfy the micronutrient requirement....

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Raven JA. Chloride: essential micronutrient and multifunctional beneficial ion. J Exp Bot. 2016;38(3):359-367doi: 10.1093/jxb/erw421.
Raven, J. A. (2017). Chloride: essential micronutrient and multifunctional beneficial ion. Journal of experimental botany, 38(3), 359-367. https://doi.org/10.1093/jxb/erw421
Raven, John A. "Chloride: essential micronutrient and multifunctional beneficial ion." Journal of experimental botany vol. 38,3 (2017): 359-367. doi: https://doi.org/10.1093/jxb/erw421
Raven JA. Chloride: essential micronutrient and multifunctional beneficial ion. J Exp Bot. 2017 Jan;38(3):359-367. doi: 10.1093/jxb/erw421. Epub 2016 Dec 30. PMID: 28040799.
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Planar Bilayer Measurements of Alamethicin and Gramicidin Reconstituted in Biomimetic Block Copolymers.

Langmuir : the ACS journal of surfaces and colloids

Martin M, Dubbs T, Fried JR.
PMID: 28040902
Langmuir. 2017 Feb 07;33(5):1171-1179. doi: 10.1021/acs.langmuir.6b03309. Epub 2017 Jan 24.

This work explores methods for forming and characterizing biomimetic planar membranes composed of amphiphilic block copolymers. The membranes are diblocks and triblocks with hydrophilic blocks of poly(2-methyl-2-oxazoline) (PMOXA) and hydrophobic blocks of poly(dimethylsiloxane) (PDMS). Experiments with the lipid diphytanoyl...

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Martin M, Dubbs T, Fried JR. Planar Bilayer Measurements of Alamethicin and Gramicidin Reconstituted in Biomimetic Block Copolymers. Langmuir. 2017;33(5):1171-1179doi: 10.1021/acs.langmuir.6b03309.
Martin, M., Dubbs, T., & Fried, J. R. (2017). Planar Bilayer Measurements of Alamethicin and Gramicidin Reconstituted in Biomimetic Block Copolymers. Langmuir : the ACS journal of surfaces and colloids, 33(5), 1171-1179. https://doi.org/10.1021/acs.langmuir.6b03309
Martin, Michael, et al. "Planar Bilayer Measurements of Alamethicin and Gramicidin Reconstituted in Biomimetic Block Copolymers." Langmuir : the ACS journal of surfaces and colloids vol. 33,5 (2017): 1171-1179. doi: https://doi.org/10.1021/acs.langmuir.6b03309
Martin M, Dubbs T, Fried JR. Planar Bilayer Measurements of Alamethicin and Gramicidin Reconstituted in Biomimetic Block Copolymers. Langmuir. 2017 Feb 07;33(5):1171-1179. doi: 10.1021/acs.langmuir.6b03309. Epub 2017 Jan 24. PMID: 28040902.
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Showing 13 to 24 of 3221 entries