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Showing 13 to 24 of 1049 entries
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Plasticity of fibroblasts demonstrated by tissue-specific and function-related proteome profiling.

Clinical proteomics

Slany A, Meshcheryakova A, Beer A, Ankersmit HJ, Paulitschke V, Gerner C.
PMID: 26029019
Clin Proteomics. 2014 Nov 21;11(1):41. doi: 10.1186/1559-0275-11-41. eCollection 2014.

BACKGROUND: Fibroblasts are mesenchymal stromal cells which occur in all tissue types. While their main function is related to ECM production and physical support, they are also important players in wound healing, and have further been recognized to be...

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Slany A, Meshcheryakova A, Beer A, et al. Plasticity of fibroblasts demonstrated by tissue-specific and function-related proteome profiling. Clin Proteomics. 2014;11(1):41doi: 10.1186/1559-0275-11-41.
Slany, A., Meshcheryakova, A., Beer, A., Ankersmit, H. J., Paulitschke, V., & Gerner, C. (2014). Plasticity of fibroblasts demonstrated by tissue-specific and function-related proteome profiling. Clinical proteomics, 11(1), 41. https://doi.org/10.1186/1559-0275-11-41
Slany, Astrid, et al. "Plasticity of fibroblasts demonstrated by tissue-specific and function-related proteome profiling." Clinical proteomics vol. 11,1 (2014): 41. doi: https://doi.org/10.1186/1559-0275-11-41
Slany A, Meshcheryakova A, Beer A, Ankersmit HJ, Paulitschke V, Gerner C. Plasticity of fibroblasts demonstrated by tissue-specific and function-related proteome profiling. Clin Proteomics. 2014 Nov 21;11(1):41. doi: 10.1186/1559-0275-11-41. eCollection 2014. PMID: 26029019; PMCID: PMC4448269.
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Interleukin-19 can enhance angiogenesis by Macrophage Polarization.

Macrophage

Gabunia K, Autieri MV.
PMID: 26029742
Macrophage (Houst). 2015;2(1):e562. doi: 10.14800/macrophage.562.

Hypoxia in ischemic limbs typically initiates angiogenic and inflammatory factors to promote angiogenesis in attempt to restore perfusion, and revascularization involves multiple cell types and systems. Macrophage display phenotype plasticity, and can polarize in response to local and systemic...

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Gabunia K, Autieri MV. Interleukin-19 can enhance angiogenesis by Macrophage Polarization. Macrophage (Houst). 2015;2(1):e562doi: 10.14800/macrophage.562.
Gabunia, K., & Autieri, M. V. (2015). Interleukin-19 can enhance angiogenesis by Macrophage Polarization. Macrophage, 2(1), e562. https://doi.org/10.14800/macrophage.562
Gabunia, Khatuna, and Autieri, Michael V. "Interleukin-19 can enhance angiogenesis by Macrophage Polarization." Macrophage vol. 2,1 (2015): e562. doi: https://doi.org/10.14800/macrophage.562
Gabunia K, Autieri MV. Interleukin-19 can enhance angiogenesis by Macrophage Polarization. Macrophage (Houst). 2015;2(1):e562. doi: 10.14800/macrophage.562. PMID: 26029742; PMCID: PMC4447484.
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The diagnostic accuracy of acute phase proteins and proinflammatory cytokines in sheep with pneumonic pasteurellosis.

PeerJ

El-Deeb WM, Elmoslemany AM.
PMID: 27547520
PeerJ. 2016 Jul 19;4:e2161. doi: 10.7717/peerj.2161. eCollection 2016.

The goal of this study was to assess the diagnostic accuracy of acute phase proteins and proinflammatory cytokines in sheep with pneumonic pasteurellosis. Blood samples were collected from 56 sheep (36 naturally infected with Pasteurella multocida and 20 healthy...

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El-Deeb WM, Elmoslemany AM. The diagnostic accuracy of acute phase proteins and proinflammatory cytokines in sheep with pneumonic pasteurellosis. PeerJ. 2016;4:e2161doi: 10.7717/peerj.2161.
El-Deeb, W. M., & Elmoslemany, A. M. (2016). The diagnostic accuracy of acute phase proteins and proinflammatory cytokines in sheep with pneumonic pasteurellosis. PeerJ, 4e2161. https://doi.org/10.7717/peerj.2161
El-Deeb, Wael M, and Elmoslemany, Ahmed M. "The diagnostic accuracy of acute phase proteins and proinflammatory cytokines in sheep with pneumonic pasteurellosis." PeerJ vol. 4 (2016): e2161. doi: https://doi.org/10.7717/peerj.2161
El-Deeb WM, Elmoslemany AM. The diagnostic accuracy of acute phase proteins and proinflammatory cytokines in sheep with pneumonic pasteurellosis. PeerJ. 2016 Jul 19;4:e2161. doi: 10.7717/peerj.2161. eCollection 2016. PMID: 27547520; PMCID: PMC4957991.
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The Effect of Long-Term Exercise on the Production of Osteoclastogenic and Antiosteoclastogenic Cytokines by Peripheral Blood Mononuclear Cells and on Serum Markers of Bone Metabolism.

Journal of osteoporosis

Smith JK, Dykes R, Chi DS.
PMID: 27642534
J Osteoporos. 2016;2016:5925380. doi: 10.1155/2016/5925380. Epub 2016 Aug 24.

Although it is recognized that the mechanical stresses associated with physical activity augment bone mineral density and improve bone quality, our understanding of how exercise modulates bone homeostasis at the molecular level is lacking. In a before and after...

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Smith JK, Dykes R, Chi DS. The Effect of Long-Term Exercise on the Production of Osteoclastogenic and Antiosteoclastogenic Cytokines by Peripheral Blood Mononuclear Cells and on Serum Markers of Bone Metabolism. J Osteoporos. 2016;2016:5925380doi: 10.1155/2016/5925380.
Smith, J. K., Dykes, R., & Chi, D. S. (2016). The Effect of Long-Term Exercise on the Production of Osteoclastogenic and Antiosteoclastogenic Cytokines by Peripheral Blood Mononuclear Cells and on Serum Markers of Bone Metabolism. Journal of osteoporosis, 20165925380. https://doi.org/10.1155/2016/5925380
Smith, J Kelly, et al. "The Effect of Long-Term Exercise on the Production of Osteoclastogenic and Antiosteoclastogenic Cytokines by Peripheral Blood Mononuclear Cells and on Serum Markers of Bone Metabolism." Journal of osteoporosis vol. 2016 (2016): 5925380. doi: https://doi.org/10.1155/2016/5925380
Smith JK, Dykes R, Chi DS. The Effect of Long-Term Exercise on the Production of Osteoclastogenic and Antiosteoclastogenic Cytokines by Peripheral Blood Mononuclear Cells and on Serum Markers of Bone Metabolism. J Osteoporos. 2016;2016:5925380. doi: 10.1155/2016/5925380. Epub 2016 Aug 24. PMID: 27642534; PMCID: PMC5013218.
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New Interleukins in Psoriasis and Psoriatic Arthritis Patients: The Possible Roles of Interleukin-33 to Interleukin-38 in Disease Activities and Bone Erosions.

Dermatology (Basel, Switzerland)

Li J, Liu L, Rui W, Li X, Xuan D, Zheng S, Yu Y, Zhang J, Kong N, Zhu X, Zou H, Wan W, Xue Y.
PMID: 28490011
Dermatology. 2017;233(1):37-46. doi: 10.1159/000471798. Epub 2017 May 10.

OBJECTIVES: New interleukins (ILs), especially members of IL-1 and IL-12 families, have recently been reported to be involved in the development and regulation of autoimmune and inflammatory diseases. In this study, we aimed to explore the impact of these...

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Li J, Liu L, Rui W, et al. New Interleukins in Psoriasis and Psoriatic Arthritis Patients: The Possible Roles of Interleukin-33 to Interleukin-38 in Disease Activities and Bone Erosions. Dermatology. 2017;233(1):37-46doi: 10.1159/000471798.
Li, J., Liu, L., Rui, W., Li, X., Xuan, D., Zheng, S., Yu, Y., Zhang, J., Kong, N., Zhu, X., Zou, H., Wan, W., & Xue, Y. (2017). New Interleukins in Psoriasis and Psoriatic Arthritis Patients: The Possible Roles of Interleukin-33 to Interleukin-38 in Disease Activities and Bone Erosions. Dermatology (Basel, Switzerland), 233(1), 37-46. https://doi.org/10.1159/000471798
Li, Jiang, et al. "New Interleukins in Psoriasis and Psoriatic Arthritis Patients: The Possible Roles of Interleukin-33 to Interleukin-38 in Disease Activities and Bone Erosions." Dermatology (Basel, Switzerland) vol. 233,1 (2017): 37-46. doi: https://doi.org/10.1159/000471798
Li J, Liu L, Rui W, Li X, Xuan D, Zheng S, Yu Y, Zhang J, Kong N, Zhu X, Zou H, Wan W, Xue Y. New Interleukins in Psoriasis and Psoriatic Arthritis Patients: The Possible Roles of Interleukin-33 to Interleukin-38 in Disease Activities and Bone Erosions. Dermatology. 2017;233(1):37-46. doi: 10.1159/000471798. Epub 2017 May 10. PMID: 28490011.
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The Role of Interleukin-18, Oxidative Stress and Metabolic Syndrome in Alzheimer's Disease.

Journal of clinical medicine

Ojala JO, Sutinen EM.
PMID: 28531131
J Clin Med. 2017 May 21;6(5). doi: 10.3390/jcm6050055.

The role of interleukins (ILs) and oxidative stress (OS) in precipitating neurodegenerative diseases including sporadic Alzheimer's disease (AD), requires further clarification. In addition to neuropathological hallmarks-extracellular neuritic amyloid-β (Aβ) plaques, neurofibrillary tangles (NFT) containing hyperphosphorylated tau and neuronal loss-chronic...

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Ojala JO, Sutinen EM. The Role of Interleukin-18, Oxidative Stress and Metabolic Syndrome in Alzheimer's Disease. J Clin Med. 2017;6(5)doi: 10.3390/jcm6050055.
Ojala, J. O., & Sutinen, E. M. (2017). The Role of Interleukin-18, Oxidative Stress and Metabolic Syndrome in Alzheimer's Disease. Journal of clinical medicine, 6(5), . https://doi.org/10.3390/jcm6050055
Ojala, Johanna O, and Sutinen, Elina M. "The Role of Interleukin-18, Oxidative Stress and Metabolic Syndrome in Alzheimer's Disease." Journal of clinical medicine vol. 6,5 (2017). doi: https://doi.org/10.3390/jcm6050055
Ojala JO, Sutinen EM. The Role of Interleukin-18, Oxidative Stress and Metabolic Syndrome in Alzheimer's Disease. J Clin Med. 2017 May 21;6(5). doi: 10.3390/jcm6050055. PMID: 28531131; PMCID: PMC5447946.
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Haptoglobin gene polymorphisms and interleukin-6 and -8 levels in patients with sickle cell anemia.

Revista brasileira de hematologia e hemoterapia

Pierrot-Gallo BS, Vicari P, Matsuda SS, Adegoke SA, Mecabo G, Figueiredo MS.
PMID: 26408368
Rev Bras Hematol Hemoter. 2015 Sep-Oct;37(5):329-35. doi: 10.1016/j.bjhh.2015.07.006. Epub 2015 Jul 31.

BACKGROUND: Haptoglobin genotypes, and interleukin-6 and -8 participate in the pathophysiology of sickle cell anemia. The expression of cytokines is regulated by genetic mechanisms however the effect of haptoglobin polymorphisms on these cytokines is not fully understood. This study...

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Pierrot-Gallo BS, Vicari P, Matsuda SS, et al. Haptoglobin gene polymorphisms and interleukin-6 and -8 levels in patients with sickle cell anemia. Rev Bras Hematol Hemoter. 2015;37(5):329-35doi: 10.1016/j.bjhh.2015.07.006.
Pierrot-Gallo, B. S., Vicari, P., Matsuda, S. S., Adegoke, S. A., Mecabo, G., & Figueiredo, M. S. (2015). Haptoglobin gene polymorphisms and interleukin-6 and -8 levels in patients with sickle cell anemia. Revista brasileira de hematologia e hemoterapia, 37(5), 329-35. https://doi.org/10.1016/j.bjhh.2015.07.006
Pierrot-Gallo, Bruna Spinella, et al. "Haptoglobin gene polymorphisms and interleukin-6 and -8 levels in patients with sickle cell anemia." Revista brasileira de hematologia e hemoterapia vol. 37,5 (2015): 329-35. doi: https://doi.org/10.1016/j.bjhh.2015.07.006
Pierrot-Gallo BS, Vicari P, Matsuda SS, Adegoke SA, Mecabo G, Figueiredo MS. Haptoglobin gene polymorphisms and interleukin-6 and -8 levels in patients with sickle cell anemia. Rev Bras Hematol Hemoter. 2015 Sep-Oct;37(5):329-35. doi: 10.1016/j.bjhh.2015.07.006. Epub 2015 Jul 31. PMID: 26408368; PMCID: PMC4685105.
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Interleukin-3: Promises and Perspectives.

Hematology (Amsterdam, Netherlands)

Mangi MH, Newland AC.
PMID: 27416283
Hematology. 1998;3(1):55-66. doi: 10.1080/10245332.1998.11752123.

Interleukin-3 (IL-3) is a multipotent hematopoietic growth factor produced by activated T-cells, monocytes/macrophages and stroma cells. Human IL-3 gene is located on chromosome 5 near segment 5q31. The high affinity receptor for human IL-3 is composed of alpha and...

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Mangi MH, Newland AC. Interleukin-3: Promises and Perspectives. Hematology. 1998;3(1):55-66doi: 10.1080/10245332.1998.11752123.
Mangi, M. H., & Newland, A. C. (1998). Interleukin-3: Promises and Perspectives. Hematology (Amsterdam, Netherlands), 3(1), 55-66. https://doi.org/10.1080/10245332.1998.11752123
Mangi, M H, and Newland, A C. "Interleukin-3: Promises and Perspectives." Hematology (Amsterdam, Netherlands) vol. 3,1 (1998): 55-66. doi: https://doi.org/10.1080/10245332.1998.11752123
Mangi MH, Newland AC. Interleukin-3: Promises and Perspectives. Hematology. 1998;3(1):55-66. doi: 10.1080/10245332.1998.11752123. PMID: 27416283.
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Serum Levels of Tumor Necrosis Factor-α and Interleukins in Children with Congenital Heart Disease.

The journal of Tehran Heart Center

Noori NM, Shahramian I, Teimouri A, Keyvani B, Mahjoubifard M.
PMID: 28469687
J Tehran Heart Cent. 2017 Jan;12(1):15-22.

No abstract available.

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Noori NM, Shahramian I, Teimouri A, et al. Serum Levels of Tumor Necrosis Factor-α and Interleukins in Children with Congenital Heart Disease. J Tehran Heart Cent. 2017;12(1):15-22
Noori, N. M., Shahramian, I., Teimouri, A., Keyvani, B., & Mahjoubifard, M. (2017). Serum Levels of Tumor Necrosis Factor-α and Interleukins in Children with Congenital Heart Disease. The journal of Tehran Heart Center, 12(1), 15-22.
Noori, Noor Mohammand, et al. "Serum Levels of Tumor Necrosis Factor-α and Interleukins in Children with Congenital Heart Disease." The journal of Tehran Heart Center vol. 12,1 (2017): 15-22.
Noori NM, Shahramian I, Teimouri A, Keyvani B, Mahjoubifard M. Serum Levels of Tumor Necrosis Factor-α and Interleukins in Children with Congenital Heart Disease. J Tehran Heart Cent. 2017 Jan;12(1):15-22. PMID: 28469687; PMCID: PMC5409944.
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Swertianlarin, an Herbal Agent Derived from Swertia mussotii Franch, Attenuates Liver Injury, Inflammation, and Cholestasis in Common Bile Duct-Ligated Rats.

Evidence-based complementary and alternative medicine : eCAM

Zhang L, Cheng Y, Du X, Chen S, Feng X, Gao Y, Li S, Liu L, Yang M, Chen L, Peng Z, Yang Y, Luo W, Wang R, Chen W, Chai J.
PMID: 26273316
Evid Based Complement Alternat Med. 2015;2015:948376. doi: 10.1155/2015/948376. Epub 2015 Jul 27.

Swertianlarin is an herbal agent abundantly distributed in Swertia mussotii Franch, a Chinese traditional herb used for treatment of jaundice. To study the therapeutic effect of swertianlarin on cholestasis, liver injury, serum proinflammatory cytokines, and bile salt concentrations were...

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Zhang L, Cheng Y, Du X, et al. Swertianlarin, an Herbal Agent Derived from Swertia mussotii Franch, Attenuates Liver Injury, Inflammation, and Cholestasis in Common Bile Duct-Ligated Rats. Evid Based Complement Alternat Med. 2015;2015:948376doi: 10.1155/2015/948376.
Zhang, L., Cheng, Y., Du, X., Chen, S., Feng, X., Gao, Y., Li, S., Liu, L., Yang, M., Chen, L., Peng, Z., Yang, Y., Luo, W., Wang, R., Chen, W., & Chai, J. (2015). Swertianlarin, an Herbal Agent Derived from Swertia mussotii Franch, Attenuates Liver Injury, Inflammation, and Cholestasis in Common Bile Duct-Ligated Rats. Evidence-based complementary and alternative medicine : eCAM, 2015948376. https://doi.org/10.1155/2015/948376
Zhang, Liangjun, et al. "Swertianlarin, an Herbal Agent Derived from Swertia mussotii Franch, Attenuates Liver Injury, Inflammation, and Cholestasis in Common Bile Duct-Ligated Rats." Evidence-based complementary and alternative medicine : eCAM vol. 2015 (2015): 948376. doi: https://doi.org/10.1155/2015/948376
Zhang L, Cheng Y, Du X, Chen S, Feng X, Gao Y, Li S, Liu L, Yang M, Chen L, Peng Z, Yang Y, Luo W, Wang R, Chen W, Chai J. Swertianlarin, an Herbal Agent Derived from Swertia mussotii Franch, Attenuates Liver Injury, Inflammation, and Cholestasis in Common Bile Duct-Ligated Rats. Evid Based Complement Alternat Med. 2015;2015:948376. doi: 10.1155/2015/948376. Epub 2015 Jul 27. PMID: 26273316; PMCID: PMC4530240.
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Inflammatory bowel disease-related arthritis - clinical evaluation and possible role of cytokines.

Reumatologia

Dmowska-Chalaba J, Kontny E.
PMID: 27407253
Reumatologia. 2015;53(5):236-42. doi: 10.5114/reum.2015.55824. Epub 2015 Dec 08.

OBJECTIVES: In inflammatory bowel disease (IBD), characterized by chronic mucosal inflammation, rheumatic abnormalities ranging from arthralgia to spondyloarthritis (SpA) are the most common extraintestinal manifestations. The pathogenesis of IBD-related arthritis is unclear. In this study, we search for clinical...

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Dmowska-Chalaba J, Kontny E. Inflammatory bowel disease-related arthritis - clinical evaluation and possible role of cytokines. Reumatologia. 2015;53(5):236-42doi: 10.5114/reum.2015.55824.
Dmowska-Chalaba, J., & Kontny, E. (2015). Inflammatory bowel disease-related arthritis - clinical evaluation and possible role of cytokines. Reumatologia, 53(5), 236-42. https://doi.org/10.5114/reum.2015.55824
Dmowska-Chalaba, Joanna, and Kontny, Ewa. "Inflammatory bowel disease-related arthritis - clinical evaluation and possible role of cytokines." Reumatologia vol. 53,5 (2015): 236-42. doi: https://doi.org/10.5114/reum.2015.55824
Dmowska-Chalaba J, Kontny E. Inflammatory bowel disease-related arthritis - clinical evaluation and possible role of cytokines. Reumatologia. 2015;53(5):236-42. doi: 10.5114/reum.2015.55824. Epub 2015 Dec 08. PMID: 27407253; PMCID: PMC4847320.
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Hypoxia increases the expression of enamel genes and cytokines in an ameloblast-derived cell line.

European journal of oral sciences

Sidaly R, Landin MA, Suo Z, Snead ML, Lyngstadaas SP, Reseland JE.
PMID: 26193448
Eur J Oral Sci. 2015 Oct;123(5):335-340. doi: 10.1111/eos.12201. Epub 2015 Jul 20.

The aim of this study was to investigate the effect of hypoxic conditions on the expression of enamel genes and on the secretion of alkaline phosphatase (ALP), lactate dehydrogenase (LDH), cytokines, and interleukins by an ameloblast-derived cell line. Murine...

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Sidaly R, Landin MA, Suo Z, et al. Hypoxia increases the expression of enamel genes and cytokines in an ameloblast-derived cell line. Eur J Oral Sci. 2015;123(5):335-340doi: 10.1111/eos.12201.
Sidaly, R., Landin, M. A., Suo, Z., Snead, M. L., Lyngstadaas, S. P., & Reseland, J. E. (2015). Hypoxia increases the expression of enamel genes and cytokines in an ameloblast-derived cell line. European journal of oral sciences, 123(5), 335-340. https://doi.org/10.1111/eos.12201
Sidaly, Rivan, et al. "Hypoxia increases the expression of enamel genes and cytokines in an ameloblast-derived cell line." European journal of oral sciences vol. 123,5 (2015): 335-340. doi: https://doi.org/10.1111/eos.12201
Sidaly R, Landin MA, Suo Z, Snead ML, Lyngstadaas SP, Reseland JE. Hypoxia increases the expression of enamel genes and cytokines in an ameloblast-derived cell line. Eur J Oral Sci. 2015 Oct;123(5):335-340. doi: 10.1111/eos.12201. Epub 2015 Jul 20. PMID: 26193448.
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Showing 13 to 24 of 1049 entries