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Showing 13 to 24 of 42 entries
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5-HT 1D receptors mediate SKF 99101H-induced hypothermia in the guinea pig.

Journal of psychopharmacology (Oxford, England)

Hatcher JP, Slade PD, Roberts C, Hagan JJ.
PMID: 22297763
J Psychopharmacol. 1995 Jan;9(3):234-41. doi: 10.1177/026988119500900306.

The selective, brain penetrant, 5-HT(1D) receptor agonist SKF 99101H (10-30 mg/kg i.p.) caused a dose-related fall in rectal temperature in guinea pigs which lasted longer than 2 h. Sumatriptan (1.0-100 mg/kg i.p.), a selective 5-HT(1D) agonist which does not...

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Hatcher JP, Slade PD, Roberts C, et al. 5-HT 1D receptors mediate SKF 99101H-induced hypothermia in the guinea pig. J Psychopharmacol. 1995;9(3):234-41doi: 10.1177/026988119500900306.
Hatcher, J. P., Slade, P. D., Roberts, C., & Hagan, J. J. (1995). 5-HT 1D receptors mediate SKF 99101H-induced hypothermia in the guinea pig. Journal of psychopharmacology (Oxford, England), 9(3), 234-41. https://doi.org/10.1177/026988119500900306
Hatcher, J P, et al. "5-HT 1D receptors mediate SKF 99101H-induced hypothermia in the guinea pig." Journal of psychopharmacology (Oxford, England) vol. 9,3 (1995): 234-41. doi: https://doi.org/10.1177/026988119500900306
Hatcher JP, Slade PD, Roberts C, Hagan JJ. 5-HT 1D receptors mediate SKF 99101H-induced hypothermia in the guinea pig. J Psychopharmacol. 1995 Jan;9(3):234-41. doi: 10.1177/026988119500900306. PMID: 22297763.
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Methylphenidate improves prefrontal cortical cognitive function through alpha2 adrenoceptor and dopamine D1 receptor actions: Relevance to therapeutic effects in Attention Deficit Hyperactivity Disorder.

Behavioral and brain functions : BBF

Arnsten AF, Dudley AG.
PMID: 15916700
Behav Brain Funct. 2005 Apr 22;1(1):2. doi: 10.1186/1744-9081-1-2.

BACKGROUND: Methylphenidate (MPH) is the classic treatment for Attention Deficit Hyperactivity Disorder (ADHD), yet the mechanisms underlying its therapeutic actions remain unclear. Recent studies have identified an oral, MPH dose regimen which when given to rats produces drug plasma...

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Arnsten AF, Dudley AG. Methylphenidate improves prefrontal cortical cognitive function through alpha2 adrenoceptor and dopamine D1 receptor actions: Relevance to therapeutic effects in Attention Deficit Hyperactivity Disorder. Behav Brain Funct. 2005;1(1):2doi: 10.1186/1744-9081-1-2.
Arnsten, A. F., & Dudley, A. G. (2005). Methylphenidate improves prefrontal cortical cognitive function through alpha2 adrenoceptor and dopamine D1 receptor actions: Relevance to therapeutic effects in Attention Deficit Hyperactivity Disorder. Behavioral and brain functions : BBF, 1(1), 2. https://doi.org/10.1186/1744-9081-1-2
Arnsten, Amy Ft, and Dudley, Anne G. "Methylphenidate improves prefrontal cortical cognitive function through alpha2 adrenoceptor and dopamine D1 receptor actions: Relevance to therapeutic effects in Attention Deficit Hyperactivity Disorder." Behavioral and brain functions : BBF vol. 1,1 (2005): 2. doi: https://doi.org/10.1186/1744-9081-1-2
Arnsten AF, Dudley AG. Methylphenidate improves prefrontal cortical cognitive function through alpha2 adrenoceptor and dopamine D1 receptor actions: Relevance to therapeutic effects in Attention Deficit Hyperactivity Disorder. Behav Brain Funct. 2005 Apr 22;1(1):2. doi: 10.1186/1744-9081-1-2. PMID: 15916700; PMCID: PMC1143775.
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Dose-dependent discriminative stimulus properties of 8-OH-DPAT.

Behavioural pharmacology

Ybema CE, Slangen JL, Olivier B, Mos J.
PMID: 11224230
Behav Pharmacol. 1993 Dec;4(6):610-624.

Separate groups of rats were trained to discriminate either 0.1mg/kg (low dose; L) or 2.5mg/kg (high dose; H) of 8-OH-DPAT from saline, in a standard operant task. Both cues were found to be dose, time and route dependent and...

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Ybema CE, Slangen JL, Olivier B, et al. Dose-dependent discriminative stimulus properties of 8-OH-DPAT. Behav Pharmacol. 1993;4(6):610-624
Ybema, C. E., Slangen, J. L., Olivier, B., & Mos, J. (1993). Dose-dependent discriminative stimulus properties of 8-OH-DPAT. Behavioural pharmacology, 4(6), 610-624.
Ybema, C.E., et al. "Dose-dependent discriminative stimulus properties of 8-OH-DPAT." Behavioural pharmacology vol. 4,6 (1993): 610-624.
Ybema CE, Slangen JL, Olivier B, Mos J. Dose-dependent discriminative stimulus properties of 8-OH-DPAT. Behav Pharmacol. 1993 Dec;4(6):610-624. PMID: 11224230.
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Electrophysiologic characterization of the antinociceptive actions of S18616, a novel and potent alpha 2-adrenoceptor agonist, after acute and persistent pain states.

The journal of pain

Suzuki R, Green GM, Millan MJ, Dickenson AH.
PMID: 14622778
J Pain. 2002 Jun;3(3):234-43. doi: 10.1054/jpai.2002.123651.

alpha (2)-Adrenoceptor (AR) agonists are active in behavioral models of persistent pain involving tissue and nerve damage. We evaluated the spinal effect of a novel, potent, and selective alpha (2)-AR agonist, [7,8](2-chlorobenzo)-2-amino-1-aza-3-oxa[4,5]spirodeca-1,7-diene (S18616), on the responses of dorsal horn...

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Suzuki R, Green GM, Millan MJ, et al. Electrophysiologic characterization of the antinociceptive actions of S18616, a novel and potent alpha 2-adrenoceptor agonist, after acute and persistent pain states. J Pain. 2002;3(3):234-43doi: 10.1054/jpai.2002.123651.
Suzuki, R., Green, G. M., Millan, M. J., & Dickenson, A. H. (2002). Electrophysiologic characterization of the antinociceptive actions of S18616, a novel and potent alpha 2-adrenoceptor agonist, after acute and persistent pain states. The journal of pain, 3(3), 234-43. https://doi.org/10.1054/jpai.2002.123651
Suzuki, Rie, et al. "Electrophysiologic characterization of the antinociceptive actions of S18616, a novel and potent alpha 2-adrenoceptor agonist, after acute and persistent pain states." The journal of pain vol. 3,3 (2002): 234-43. doi: https://doi.org/10.1054/jpai.2002.123651
Suzuki R, Green GM, Millan MJ, Dickenson AH. Electrophysiologic characterization of the antinociceptive actions of S18616, a novel and potent alpha 2-adrenoceptor agonist, after acute and persistent pain states. J Pain. 2002 Jun;3(3):234-43. doi: 10.1054/jpai.2002.123651. PMID: 14622778.
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Prenatal IV Cocaine: Alterations in Auditory Information Processing.

Frontiers in psychiatry

Mactutus CF, Harrod SB, Hord LL, Moran LM, Booze RM.
PMID: 21747770
Front Psychiatry. 2011 Jun 28;2:38. doi: 10.3389/fpsyt.2011.00038. eCollection 2011.

One clue regarding the basis of cocaine-induced deficits in attentional processing is provided by the clinical findings of changes in the infants' startle response; observations buttressed by neurophysiological evidence of alterations in brainstem transmission time. Using the IV route...

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Mactutus CF, Harrod SB, Hord LL, et al. Prenatal IV Cocaine: Alterations in Auditory Information Processing. Front Psychiatry. 2011;2:38doi: 10.3389/fpsyt.2011.00038.
Mactutus, C. F., Harrod, S. B., Hord, L. L., Moran, L. M., & Booze, R. M. (2011). Prenatal IV Cocaine: Alterations in Auditory Information Processing. Frontiers in psychiatry, 238. https://doi.org/10.3389/fpsyt.2011.00038
Mactutus, Charles F, et al. "Prenatal IV Cocaine: Alterations in Auditory Information Processing." Frontiers in psychiatry vol. 2 (2011): 38. doi: https://doi.org/10.3389/fpsyt.2011.00038
Mactutus CF, Harrod SB, Hord LL, Moran LM, Booze RM. Prenatal IV Cocaine: Alterations in Auditory Information Processing. Front Psychiatry. 2011 Jun 28;2:38. doi: 10.3389/fpsyt.2011.00038. eCollection 2011. PMID: 21747770; PMCID: PMC3128243.
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Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains.

ASN neuro

Zeng F, Fan Y, Brown RW, Drew Gill W, Price JB, Jones TC, Zhu MY.
PMID: 34812056
ASN Neuro. 2021 Jan-Dec;13:17590914211055064. doi: 10.1177/17590914211055064.

This study investigated the effects of the pharmacological manipulation of noradrenergic activities on dopaminergic phenotypes in aged rats. Results showed that the administration of L-threo-3,4-dihydroxyphenylserine (L-DOPS) for 21 days significantly increased the expression of tyrosine hydroxylase (TH) and dopamine...

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Zeng F, Fan Y, Brown RW, et al. Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains. ASN Neuro. 2021;13:17590914211055064doi: 10.1177/17590914211055064.
Zeng, F., Fan, Y., Brown, R. W., Drew Gill, W., Price, J. B., Jones, T. C., & Zhu, M. Y. (2021). Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains. ASN neuro, 1317590914211055064. https://doi.org/10.1177/17590914211055064
Zeng, Fei, et al. "Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains." ASN neuro vol. 13 (2021): 17590914211055064. doi: https://doi.org/10.1177/17590914211055064
Zeng F, Fan Y, Brown RW, Drew Gill W, Price JB, Jones TC, Zhu MY. Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains. ASN Neuro. 2021 Jan-Dec;13:17590914211055064. doi: 10.1177/17590914211055064. PMID: 34812056; PMCID: PMC8613899.
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Effects of imidazoline agents in a rat conditioned place preference model of addiction.

Naunyn-Schmiedeberg's archives of pharmacology

Şorodoc V, Rusu-Zota G, Nechita P, Moraru C, Manole OM.
PMID: 34997272
Naunyn Schmiedebergs Arch Pharmacol. 2022 Jan 08; doi: 10.1007/s00210-021-02194-z. Epub 2022 Jan 08.

Agmatine (AG), idazoxan (IDZ), and efaroxan (EFR) are imidazoline receptor ligands with beneficial effects in central nervous system disorders. The present study aimed to evaluate the interaction between AG, IDZ, and EFR with an opiate, tramadol (TR), in a...

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Şorodoc V, Rusu-Zota G, Nechita P, et al. Effects of imidazoline agents in a rat conditioned place preference model of addiction. Naunyn Schmiedebergs Arch Pharmacol. 2022;doi: 10.1007/s00210-021-02194-z.
Şorodoc, V., Rusu-Zota, G., Nechita, P., Moraru, C., & Manole, O. M. (2022). Effects of imidazoline agents in a rat conditioned place preference model of addiction. Naunyn-Schmiedeberg's archives of pharmacology, . https://doi.org/10.1007/s00210-021-02194-z
Şorodoc, V, et al. "Effects of imidazoline agents in a rat conditioned place preference model of addiction." Naunyn-Schmiedeberg's archives of pharmacology vol. (2022). doi: https://doi.org/10.1007/s00210-021-02194-z
Şorodoc V, Rusu-Zota G, Nechita P, Moraru C, Manole OM. Effects of imidazoline agents in a rat conditioned place preference model of addiction. Naunyn Schmiedebergs Arch Pharmacol. 2022 Jan 08; doi: 10.1007/s00210-021-02194-z. Epub 2022 Jan 08. PMID: 34997272.
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The Impact of α-Adrenoceptors in the Regulation of the Hypotonicity-Induced Increase in Duodenal Mucosal Permeability In Vivo.

Pharmaceutics

Sedin J, Dahlgren D, Sjöblom M, Nylander O.
PMID: 34959377
Pharmaceutics. 2021 Dec 06;13(12). doi: 10.3390/pharmaceutics13122096.

The duodenal mucosa is regularly exposed to a low osmolality, and recent experiments suggest that hypotonicity increases mucosal permeability in an osmolality-dependent manner. The aim was to examine whether the sympathetic nervous system, via action on α-adrenoceptors, affects the...

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Sedin J, Dahlgren D, Sjöblom M, et al. The Impact of α-Adrenoceptors in the Regulation of the Hypotonicity-Induced Increase in Duodenal Mucosal Permeability In Vivo. Pharmaceutics. 2021;13(12)doi: 10.3390/pharmaceutics13122096.
Sedin, J., Dahlgren, D., Sjöblom, M., & Nylander, O. (2021). The Impact of α-Adrenoceptors in the Regulation of the Hypotonicity-Induced Increase in Duodenal Mucosal Permeability In Vivo. Pharmaceutics, 13(12), . https://doi.org/10.3390/pharmaceutics13122096
Sedin, John, et al. "The Impact of α-Adrenoceptors in the Regulation of the Hypotonicity-Induced Increase in Duodenal Mucosal Permeability In Vivo." Pharmaceutics vol. 13,12 (2021). doi: https://doi.org/10.3390/pharmaceutics13122096
Sedin J, Dahlgren D, Sjöblom M, Nylander O. The Impact of α-Adrenoceptors in the Regulation of the Hypotonicity-Induced Increase in Duodenal Mucosal Permeability In Vivo. Pharmaceutics. 2021 Dec 06;13(12). doi: 10.3390/pharmaceutics13122096. PMID: 34959377.
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Different effects of yohimbine and idazoxan in the light-dark choice procedure.

Behavioural pharmacology

Chapouthier G, Jacquot F, Save E, Venault P, Sara S.
PMID: 11175431
Behav Pharmacol. 1990;1(5):459-461.

In a light-dark choice situation, the alpha(2) adrenoceptor antagonists yohimbine and idazoxan had different effects: idazoxan decreased time spent in the fit box, but yohimbine did not. The effects of idazoxan were not blocked by the alpha(2) adrenoceptor agonist...

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Chapouthier G, Jacquot F, Save E, et al. Different effects of yohimbine and idazoxan in the light-dark choice procedure. Behav Pharmacol. 1990;1(5):459-461
Chapouthier, G., Jacquot, F., Save, E., Venault, P., & Sara, S. (1990). Different effects of yohimbine and idazoxan in the light-dark choice procedure. Behavioural pharmacology, 1(5), 459-461.
Chapouthier, G., et al. "Different effects of yohimbine and idazoxan in the light-dark choice procedure." Behavioural pharmacology vol. 1,5 (1990): 459-461.
Chapouthier G, Jacquot F, Save E, Venault P, Sara S. Different effects of yohimbine and idazoxan in the light-dark choice procedure. Behav Pharmacol. 1990;1(5):459-461. PMID: 11175431.
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Modification of naloxone-precipitated withdrawal symptoms in mice by drugs acting on alpha(2)-adrenoceptors.

Behavioural pharmacology

Sharif SI, El-Kadi AO.
PMID: 11224426
Behav Pharmacol. 1996 Aug;7(4):334-340. doi: 10.1097/00008877-199608000-00004.

Modification of naloxone-precipitated withdrawal symptoms by drugs acting on alpha-adrenoceptors was investigated in morphine-dependent mice. Clonidine (0.05-1mg/kg) attenuated most withdrawal symptoms, but potentiated withdrawal hypothermia. Jumping was attenuated by doses of clonidine up to 0.3mg/kg, but markedly potentiated by...

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Sharif SI, El-Kadi AO. Modification of naloxone-precipitated withdrawal symptoms in mice by drugs acting on alpha(2)-adrenoceptors. Behav Pharmacol. 1996;7(4):334-340doi: 10.1097/00008877-199608000-00004.
Sharif, S. I., & El-Kadi, A. O. (1996). Modification of naloxone-precipitated withdrawal symptoms in mice by drugs acting on alpha(2)-adrenoceptors. Behavioural pharmacology, 7(4), 334-340. https://doi.org/10.1097/00008877-199608000-00004
Sharif, S.I., and El-Kadi, A.O.S.. "Modification of naloxone-precipitated withdrawal symptoms in mice by drugs acting on alpha(2)-adrenoceptors." Behavioural pharmacology vol. 7,4 (1996): 334-340. doi: https://doi.org/10.1097/00008877-199608000-00004
Sharif SI, El-Kadi AO. Modification of naloxone-precipitated withdrawal symptoms in mice by drugs acting on alpha(2)-adrenoceptors. Behav Pharmacol. 1996 Aug;7(4):334-340. doi: 10.1097/00008877-199608000-00004. PMID: 11224426.
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Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains.

ASN neuro

Zeng F, Fan Y, Brown RW, Drew Gill W, Price JB, Jones TC, Zhu MY.
PMID: 34812056
ASN Neuro. 2021 Jan-Dec;13:17590914211055064. doi: 10.1177/17590914211055064.

This study investigated the effects of the pharmacological manipulation of noradrenergic activities on dopaminergic phenotypes in aged rats. Results showed that the administration of L-threo-3,4-dihydroxyphenylserine (L-DOPS) for 21 days significantly increased the expression of tyrosine hydroxylase (TH) and dopamine...

Cite
Zeng F, Fan Y, Brown RW, et al. Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains. ASN Neuro. 2021;13:17590914211055064doi: 10.1177/17590914211055064.
Zeng, F., Fan, Y., Brown, R. W., Drew Gill, W., Price, J. B., Jones, T. C., & Zhu, M. Y. (2021). Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains. ASN neuro, 1317590914211055064. https://doi.org/10.1177/17590914211055064
Zeng, Fei, et al. "Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains." ASN neuro vol. 13 (2021): 17590914211055064. doi: https://doi.org/10.1177/17590914211055064
Zeng F, Fan Y, Brown RW, Drew Gill W, Price JB, Jones TC, Zhu MY. Effects of Manipulation of Noradrenergic Activities on the Expression of Dopaminergic Phenotypes in Aged Rat Brains. ASN Neuro. 2021 Jan-Dec;13:17590914211055064. doi: 10.1177/17590914211055064. PMID: 34812056; PMCID: PMC8613899.
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Neuroprotective effects of receptor imidazoline 2 and its endogenous ligand agmatine.

Neuroscience bulletin

Qiu WW, Zheng RY.
PMID: 17704848
Neurosci Bull. 2006 May;22(3):187-91.

Receptor imidazoline 2 (I₂) is one of the imidazoline receptors with high affinity for [³H]-idazoxan. Receptor I₂, being classified into I(₂A) and I(₂B) subtypes, is mainly localized to the outer membrane of mitochondria in liver, kidney and brain. Receptor...

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Qiu WW, Zheng RY. Neuroprotective effects of receptor imidazoline 2 and its endogenous ligand agmatine. Neurosci Bull. 2006;22(3):187-91
Qiu, W. W., & Zheng, R. Y. (2006). Neuroprotective effects of receptor imidazoline 2 and its endogenous ligand agmatine. Neuroscience bulletin, 22(3), 187-91.
Qiu, Wei-Wen, and Zheng, Rong-Yuan. "Neuroprotective effects of receptor imidazoline 2 and its endogenous ligand agmatine." Neuroscience bulletin vol. 22,3 (2006): 187-91.
Qiu WW, Zheng RY. Neuroprotective effects of receptor imidazoline 2 and its endogenous ligand agmatine. Neurosci Bull. 2006 May;22(3):187-91. PMID: 17704848.
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Showing 13 to 24 of 42 entries