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The effect of cold stress on ganglioside fatty acid composition and ganglioside-bound sialic acid content of rat brain subcellular fractions.

Journal of thermal biology

Zakharova IO, Avrova NF.
PMID: 11240228
J Therm Biol. 2001 Jun;26(3):215-222. doi: 10.1016/s0306-4565(00)00045-0.

The effect of cold stress on the ganglioside fatty acid composition and sialic acid content of brain subcellular fractions and homogenate of rats was studied, the animals were kept in a cold room with 12h light-dark cycles at 3...

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Zakharova IO, Avrova NF. The effect of cold stress on ganglioside fatty acid composition and ganglioside-bound sialic acid content of rat brain subcellular fractions. J Therm Biol. 2001;26(3):215-222doi: 10.1016/s0306-4565(00)00045-0.
Zakharova, I. O., & Avrova, N. F. (2001). The effect of cold stress on ganglioside fatty acid composition and ganglioside-bound sialic acid content of rat brain subcellular fractions. Journal of thermal biology, 26(3), 215-222. https://doi.org/10.1016/s0306-4565(00)00045-0
Zakharova, I O., and Avrova, N F.. "The effect of cold stress on ganglioside fatty acid composition and ganglioside-bound sialic acid content of rat brain subcellular fractions." Journal of thermal biology vol. 26,3 (2001): 215-222. doi: https://doi.org/10.1016/s0306-4565(00)00045-0
Zakharova IO, Avrova NF. The effect of cold stress on ganglioside fatty acid composition and ganglioside-bound sialic acid content of rat brain subcellular fractions. J Therm Biol. 2001 Jun;26(3):215-222. doi: 10.1016/s0306-4565(00)00045-0. PMID: 11240228.
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The participation of phospholipids in the interaction of leucocidin and the cell membrane of the polymorphonuclear leucocyte.

The Biochemical journal

Woodin AM, Wieneke AA.
PMID: 16742527
Biochem J. 1967 Dec;105(3):1029-38. doi: 10.1042/bj1051029.

1. The interaction of the two components of leucocidin with various lipids has been studied by sedimentation, flotation, light-scattering and changes in the biological activity of leucocidin. 2. Phosphatidylserine, phosphatidylcholine, diphosphoinositide, triphosphoinositide and phosphatidic acid, but not phosphatidylethanolamine, lysophosphatidylcholine,...

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Woodin AM, Wieneke AA. The participation of phospholipids in the interaction of leucocidin and the cell membrane of the polymorphonuclear leucocyte. Biochem J. 1967;105(3):1029-38doi: 10.1042/bj1051029.
Woodin, A. M., & Wieneke, A. A. (1967). The participation of phospholipids in the interaction of leucocidin and the cell membrane of the polymorphonuclear leucocyte. The Biochemical journal, 105(3), 1029-38. https://doi.org/10.1042/bj1051029
Woodin, A M, and Wieneke, A A. "The participation of phospholipids in the interaction of leucocidin and the cell membrane of the polymorphonuclear leucocyte." The Biochemical journal vol. 105,3 (1967): 1029-38. doi: https://doi.org/10.1042/bj1051029
Woodin AM, Wieneke AA. The participation of phospholipids in the interaction of leucocidin and the cell membrane of the polymorphonuclear leucocyte. Biochem J. 1967 Dec;105(3):1029-38. doi: 10.1042/bj1051029. PMID: 16742527; PMCID: PMC1198422.
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Partial purification and characterization of hemolysin from a psychrotrophic kanagawa-positive marine Vibrio.

Applied and environmental microbiology

Peters S, Baross JA, Morita RY.
PMID: 16345927
Appl Environ Microbiol. 1982 Jan;43(1):39-49. doi: 10.1128/aem.43.1.39-49.1982.

Psychrotrophic Kanagawa-positive marine vibrios were isolated from soft-shelled clams (Mya arenaria) collected in Yaquina Bay, Oreg. The 235 vibrio isolates obtained were screened for Gram reaction and morphology, Kanagawa reaction on Wagastsuma agar, and response to selected biochemical tests....

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Peters S, Baross JA, Morita RY. Partial purification and characterization of hemolysin from a psychrotrophic kanagawa-positive marine Vibrio. Appl Environ Microbiol. 1982;43(1):39-49doi: 10.1128/aem.43.1.39-49.1982.
Peters, S., Baross, J. A., & Morita, R. Y. (1982). Partial purification and characterization of hemolysin from a psychrotrophic kanagawa-positive marine Vibrio. Applied and environmental microbiology, 43(1), 39-49. https://doi.org/10.1128/aem.43.1.39-49.1982
Peters, S, et al. "Partial purification and characterization of hemolysin from a psychrotrophic kanagawa-positive marine Vibrio." Applied and environmental microbiology vol. 43,1 (1982): 39-49. doi: https://doi.org/10.1128/aem.43.1.39-49.1982
Peters S, Baross JA, Morita RY. Partial purification and characterization of hemolysin from a psychrotrophic kanagawa-positive marine Vibrio. Appl Environ Microbiol. 1982 Jan;43(1):39-49. doi: 10.1128/aem.43.1.39-49.1982. PMID: 16345927; PMCID: PMC241778.
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Ganglioside syndrome, a new autoimmune neurologic disorder, experimentally induced with brain gangliosides.

Neuroscience letters

Nagai Y, Momoi T, Saito M, Mitsuzawa E, Ohtani S.
PMID: 19604825
Neurosci Lett. 1976 Apr;2(2):107-11. doi: 10.1016/0304-3940(76)90033-1.

Rabbits intensively immunized with total bovine brain gangliosides or a certain molecular species of gangliosides, such as GD(1a) and GM(1), respectively, in complete Freud's adjuvant developed a variety of neurologic symptoms and signs which closely resembled experimental allergic encephalomyelitis,...

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Nagai Y, Momoi T, Saito M, et al. Ganglioside syndrome, a new autoimmune neurologic disorder, experimentally induced with brain gangliosides. Neurosci Lett. 1976;2(2):107-11doi: 10.1016/0304-3940(76)90033-1.
Nagai, Y., Momoi, T., Saito, M., Mitsuzawa, E., & Ohtani, S. (1976). Ganglioside syndrome, a new autoimmune neurologic disorder, experimentally induced with brain gangliosides. Neuroscience letters, 2(2), 107-11. https://doi.org/10.1016/0304-3940(76)90033-1
Nagai, Y, et al. "Ganglioside syndrome, a new autoimmune neurologic disorder, experimentally induced with brain gangliosides." Neuroscience letters vol. 2,2 (1976): 107-11. doi: https://doi.org/10.1016/0304-3940(76)90033-1
Nagai Y, Momoi T, Saito M, Mitsuzawa E, Ohtani S. Ganglioside syndrome, a new autoimmune neurologic disorder, experimentally induced with brain gangliosides. Neurosci Lett. 1976 Apr;2(2):107-11. doi: 10.1016/0304-3940(76)90033-1. PMID: 19604825.
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Interactive effects of insulin, IGF-I and GMl ganglioside on growth of PC12 cells.

Restorative neurology and neuroscience

Yates AJ, Conley J.
PMID: 21551654
Restor Neurol Neurosci. 1992 Jan 01;4(1):51-4. doi: 10.3233/RNN-1992-4107.

PC12 cells grow in serum-free medium (SFM) if high doses of insulin (5-50 μg/ml), or combined insulin (5 μg/ml) and insulin-like growth factor-I (3 ng/ml) are added daily. Ganglioside GMl counteracts these effects in a dose-responsive fashion between 10...

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Yates AJ, Conley J. Interactive effects of insulin, IGF-I and GMl ganglioside on growth of PC12 cells. Restor Neurol Neurosci. 1992;4(1):51-4doi: 10.3233/RNN-1992-4107.
Yates, A. J., & Conley, J. (1992). Interactive effects of insulin, IGF-I and GMl ganglioside on growth of PC12 cells. Restorative neurology and neuroscience, 4(1), 51-4. https://doi.org/10.3233/RNN-1992-4107
Yates, A J, and Conley, J. "Interactive effects of insulin, IGF-I and GMl ganglioside on growth of PC12 cells." Restorative neurology and neuroscience vol. 4,1 (1992): 51-4. doi: https://doi.org/10.3233/RNN-1992-4107
Yates AJ, Conley J. Interactive effects of insulin, IGF-I and GMl ganglioside on growth of PC12 cells. Restor Neurol Neurosci. 1992 Jan 01;4(1):51-4. doi: 10.3233/RNN-1992-4107. PMID: 21551654.
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Anti-ganglioside anti-idiotypic vaccination: more than molecular mimicry.

Frontiers in oncology

Vázquez AM, Rodrèguez-Zhurbenko N, López AM.
PMID: 23181219
Front Oncol. 2012 Nov 20;2:170. doi: 10.3389/fonc.2012.00170. eCollection 2012.

Surgery, chemotherapy, and radiation therapy are standard modalities for cancer treatment, but the effectiveness of these treatments has reached a plateau. Thus, other strategies are being explored to combine with the current treatment paradigms in order to reach better...

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Vázquez AM, Rodrèguez-Zhurbenko N, López AM. Anti-ganglioside anti-idiotypic vaccination: more than molecular mimicry. Front Oncol. 2012;2:170doi: 10.3389/fonc.2012.00170.
Vázquez, A. M., Rodrèguez-Zhurbenko, N., & López, A. M. (2012). Anti-ganglioside anti-idiotypic vaccination: more than molecular mimicry. Frontiers in oncology, 2170. https://doi.org/10.3389/fonc.2012.00170
Vázquez, Ana M H, et al. "Anti-ganglioside anti-idiotypic vaccination: more than molecular mimicry." Frontiers in oncology vol. 2 (2012): 170. doi: https://doi.org/10.3389/fonc.2012.00170
Vázquez AM, Rodrèguez-Zhurbenko N, López AM. Anti-ganglioside anti-idiotypic vaccination: more than molecular mimicry. Front Oncol. 2012 Nov 20;2:170. doi: 10.3389/fonc.2012.00170. eCollection 2012. PMID: 23181219; PMCID: PMC3501824.
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Immune response to racotumomab in a child with relapsed neuroblastoma.

Frontiers in oncology

Sampor C, Guthmann MD, Scursoni A, Cacciavillano W, Torbidoni A, Galluzzo L, Camarero S, Lopez J, de Dávila MT, Fainboim L, Chantada GL.
PMID: 23267436
Front Oncol. 2012 Dec 20;2:195. doi: 10.3389/fonc.2012.00195. eCollection 2012.

Immunotherapy targeting ganglioside antigens is a powerful tool for the treatment of high risk neuroblastoma. However, only treatment with anti-GD2 antibodies has been used in clinical practice and other options may be pursued. We report the use of racotumomab,...

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Sampor C, Guthmann MD, Scursoni A, et al. Immune response to racotumomab in a child with relapsed neuroblastoma. Front Oncol. 2012;2:195doi: 10.3389/fonc.2012.00195.
Sampor, C., Guthmann, M. D., Scursoni, A., Cacciavillano, W., Torbidoni, A., Galluzzo, L., Camarero, S., Lopez, J., de Dávila, M. T., Fainboim, L., & Chantada, G. L. (2012). Immune response to racotumomab in a child with relapsed neuroblastoma. Frontiers in oncology, 2195. https://doi.org/10.3389/fonc.2012.00195
Sampor, C, et al. "Immune response to racotumomab in a child with relapsed neuroblastoma." Frontiers in oncology vol. 2 (2012): 195. doi: https://doi.org/10.3389/fonc.2012.00195
Sampor C, Guthmann MD, Scursoni A, Cacciavillano W, Torbidoni A, Galluzzo L, Camarero S, Lopez J, de Dávila MT, Fainboim L, Chantada GL. Immune response to racotumomab in a child with relapsed neuroblastoma. Front Oncol. 2012 Dec 20;2:195. doi: 10.3389/fonc.2012.00195. eCollection 2012. PMID: 23267436; PMCID: PMC3526767.
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Catalytic preference of Salmonella typhimurium LT2 sialidase for N-acetylneuraminic acid residues over N-glycolylneuraminic acid residues.

FEBS open bio

Minami A, Ishibashi S, Ikeda K, Ishitsubo E, Hori T, Tokiwa H, Taguchi R, Ieno D, Otsubo T, Matsuda Y, Sai S, Inada M, Suzuki T.
PMID: 23772399
FEBS Open Bio. 2013 May 29;3:231-6. doi: 10.1016/j.fob.2013.05.002. Print 2013.

In a comparison of sialidase activities toward N-acetylneuraminic acid (Neu5Ac) and N-glycolylneuraminic acid (Neu5Gc), we found that Salmonella typhimurium LT2 sialidase (STSA) hardly cleaved 4-methylumbelliferyl Neu5Gc (4MU-Neu5Gc). The k cat/K m value of STSA for 4MU-Neu5Gc was found to...

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Minami A, Ishibashi S, Ikeda K, et al. Catalytic preference of Salmonella typhimurium LT2 sialidase for N-acetylneuraminic acid residues over N-glycolylneuraminic acid residues. FEBS Open Bio. 2013;3:231-6doi: 10.1016/j.fob.2013.05.002.
Minami, A., Ishibashi, S., Ikeda, K., Ishitsubo, E., Hori, T., Tokiwa, H., Taguchi, R., Ieno, D., Otsubo, T., Matsuda, Y., Sai, S., Inada, M., & Suzuki, T. (2013). Catalytic preference of Salmonella typhimurium LT2 sialidase for N-acetylneuraminic acid residues over N-glycolylneuraminic acid residues. FEBS open bio, 3231-6. https://doi.org/10.1016/j.fob.2013.05.002
Minami, Akira, et al. "Catalytic preference of Salmonella typhimurium LT2 sialidase for N-acetylneuraminic acid residues over N-glycolylneuraminic acid residues." FEBS open bio vol. 3 (2013): 231-6. doi: https://doi.org/10.1016/j.fob.2013.05.002
Minami A, Ishibashi S, Ikeda K, Ishitsubo E, Hori T, Tokiwa H, Taguchi R, Ieno D, Otsubo T, Matsuda Y, Sai S, Inada M, Suzuki T. Catalytic preference of Salmonella typhimurium LT2 sialidase for N-acetylneuraminic acid residues over N-glycolylneuraminic acid residues. FEBS Open Bio. 2013 May 29;3:231-6. doi: 10.1016/j.fob.2013.05.002. Print 2013. PMID: 23772399; PMCID: PMC3678298.
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An update on pathobiologic roles of anti-glycan antibodies in Guillain-Barré syndrome.

F1000 biology reports

Sheikh KA, Zhang G.
PMID: 20948812
F1000 Biol Rep. 2010 Mar 25;2. doi: 10.3410/B2-21.

Anti-glycan antibodies directed against gangliosides are now considered the major immune effectors that induce damage to intact nerve fibers in some variants of the monophasic neuropathic disorders that comprise Guillain-Barré syndrome. Recent experimental studies elucidating the complexity of anti-glycan...

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Sheikh KA, Zhang G. An update on pathobiologic roles of anti-glycan antibodies in Guillain-Barré syndrome. F1000 Biol Rep. 2010;2doi: 10.3410/B2-21.
Sheikh, K. A., & Zhang, G. (2010). An update on pathobiologic roles of anti-glycan antibodies in Guillain-Barré syndrome. F1000 biology reports, 2. https://doi.org/10.3410/B2-21
Sheikh, Kazim A, and Zhang, Gang. "An update on pathobiologic roles of anti-glycan antibodies in Guillain-Barré syndrome." F1000 biology reports vol. 2 (2010). doi: https://doi.org/10.3410/B2-21
Sheikh KA, Zhang G. An update on pathobiologic roles of anti-glycan antibodies in Guillain-Barré syndrome. F1000 Biol Rep. 2010 Mar 25;2. doi: 10.3410/B2-21. PMID: 20948812; PMCID: PMC2948347.
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An update in guillain-barré syndrome.

Autoimmune diseases

Winer JB.
PMID: 24511391
Autoimmune Dis. 2014;2014:793024. doi: 10.1155/2014/793024. Epub 2014 Jan 06.

Guillain-Barré syndrome (GBS) was first described in 1916 (Guillain G, 1916) and is approaching its 100th anniversary. Our knowledge of the syndrome has hugely expanded since that time. Once originally considered to be only demyelinating in pathology we now...

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Winer JB. An update in guillain-barré syndrome. Autoimmune Dis. 2014;2014:793024doi: 10.1155/2014/793024.
Winer, J. B. (2014). An update in guillain-barré syndrome. Autoimmune diseases, 2014793024. https://doi.org/10.1155/2014/793024
Winer, J B. "An update in guillain-barré syndrome." Autoimmune diseases vol. 2014 (2014): 793024. doi: https://doi.org/10.1155/2014/793024
Winer JB. An update in guillain-barré syndrome. Autoimmune Dis. 2014;2014:793024. doi: 10.1155/2014/793024. Epub 2014 Jan 06. PMID: 24511391; PMCID: PMC3910670.
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Gangliosides and glycolipids in neurodegenerative disorders.

Advances in neurobiology

Schneider JS.
PMID: 25151391
Adv Neurobiol. 2014;9:449-61. doi: 10.1007/978-1-4939-1154-7_20.

Glycolipids and gangliosides play important roles in maintaining the functional integrity of the nervous system. However, surprisingly little is known about how glycolipids and gangliosides in particular participate in various neurodegenerative processes. For example, it has been known for...

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Schneider JS. Gangliosides and glycolipids in neurodegenerative disorders. Adv Neurobiol. 2014;9:449-61doi: 10.1007/978-1-4939-1154-7_20.
Schneider, J. S. (2014). Gangliosides and glycolipids in neurodegenerative disorders. Advances in neurobiology, 9449-61. https://doi.org/10.1007/978-1-4939-1154-7_20
Schneider, J S. "Gangliosides and glycolipids in neurodegenerative disorders." Advances in neurobiology vol. 9 (2014): 449-61. doi: https://doi.org/10.1007/978-1-4939-1154-7_20
Schneider JS. Gangliosides and glycolipids in neurodegenerative disorders. Adv Neurobiol. 2014;9:449-61. doi: 10.1007/978-1-4939-1154-7_20. PMID: 25151391.
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Effect of vaccination with N-glycolyl GM3/VSSP vaccine by subcutaneous injection in patients with advanced cutaneous melanoma.

Cancer management and research

Osorio M, Gracia E, Reigosa E, Hernandez J, de la Torre A, Saurez G, Perez K, Viada C, Cepeda M, Carr A, Avila Y, Rodríguez M, Fernandez LE.
PMID: 23055778
Cancer Manag Res. 2012;4:341-5. doi: 10.2147/CMAR.S22617. Epub 2012 Oct 05.

NeuGc-containing gangliosides have been described in melanoma cells and are an attractive target for cancer immunotherapy because they are minimally or not expressed in normal human tissues. Melanoma patients treated with a vaccine based on N-glycolyl gangliosides have shown...

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Osorio M, Gracia E, Reigosa E, et al. Effect of vaccination with N-glycolyl GM3/VSSP vaccine by subcutaneous injection in patients with advanced cutaneous melanoma. Cancer Manag Res. 2012;4:341-5doi: 10.2147/CMAR.S22617.
Osorio, M., Gracia, E., Reigosa, E., Hernandez, J., de la Torre, A., Saurez, G., Perez, K., Viada, C., Cepeda, M., Carr, A., Avila, Y., Rodríguez, M., & Fernandez, L. E. (2012). Effect of vaccination with N-glycolyl GM3/VSSP vaccine by subcutaneous injection in patients with advanced cutaneous melanoma. Cancer management and research, 4341-5. https://doi.org/10.2147/CMAR.S22617
Osorio, Marta, et al. "Effect of vaccination with N-glycolyl GM3/VSSP vaccine by subcutaneous injection in patients with advanced cutaneous melanoma." Cancer management and research vol. 4 (2012): 341-5. doi: https://doi.org/10.2147/CMAR.S22617
Osorio M, Gracia E, Reigosa E, Hernandez J, de la Torre A, Saurez G, Perez K, Viada C, Cepeda M, Carr A, Avila Y, Rodríguez M, Fernandez LE. Effect of vaccination with N-glycolyl GM3/VSSP vaccine by subcutaneous injection in patients with advanced cutaneous melanoma. Cancer Manag Res. 2012;4:341-5. doi: 10.2147/CMAR.S22617. Epub 2012 Oct 05. PMID: 23055778; PMCID: PMC3468021.
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Showing 13 to 24 of 325 entries