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Targeted Quantitative Profiling of GTP-Binding Proteins Associated with Metastasis of Melanoma Cells.

Journal of proteome research

Cai R, Bade D, Liu X, Huang M, Qi TF, Wang Y.
PMID: 34694799
J Proteome Res. 2021 Nov 05;20(11):5189-5195. doi: 10.1021/acs.jproteome.1c00708. Epub 2021 Oct 25.

Metastasis is a major obstacle in the therapeutic intervention of melanoma, and several GTP-binding proteins were found to play important roles in regulating cancer metastasis. To assess systematically the regulatory roles of these proteins in melanoma metastasis, we employed...

Cite
Cai R, Bade D, Liu X, et al. Targeted Quantitative Profiling of GTP-Binding Proteins Associated with Metastasis of Melanoma Cells. J Proteome Res. 2021;20(11):5189-5195doi: 10.1021/acs.jproteome.1c00708.
Cai, R., Bade, D., Liu, X., Huang, M., Qi, T. F., & Wang, Y. (2021). Targeted Quantitative Profiling of GTP-Binding Proteins Associated with Metastasis of Melanoma Cells. Journal of proteome research, 20(11), 5189-5195. https://doi.org/10.1021/acs.jproteome.1c00708
Cai, Rong, et al. "Targeted Quantitative Profiling of GTP-Binding Proteins Associated with Metastasis of Melanoma Cells." Journal of proteome research vol. 20,11 (2021): 5189-5195. doi: https://doi.org/10.1021/acs.jproteome.1c00708
Cai R, Bade D, Liu X, Huang M, Qi TF, Wang Y. Targeted Quantitative Profiling of GTP-Binding Proteins Associated with Metastasis of Melanoma Cells. J Proteome Res. 2021 Nov 05;20(11):5189-5195. doi: 10.1021/acs.jproteome.1c00708. Epub 2021 Oct 25. PMID: 34694799.
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Constitutively Activating GNAS Somatic Mutation in Right Ventricular Outflow Tract Tachycardia.

Circulation. Arrhythmia and electrophysiology

Ip JE, Xu L, Dai J, Steegborn C, Jaffré F, Evans T, Cheung JW, Basson CT, Panaghie G, Krogh-Madsen T, Abbott GW, Lerman BB.
PMID: 34587755
Circ Arrhythm Electrophysiol. 2021 Oct;14(10):e010082. doi: 10.1161/CIRCEP.121.010082. Epub 2021 Sep 30.

[Figure: see text].

Cite
Ip JE, Xu L, Dai J, et al. Constitutively Activating GNAS Somatic Mutation in Right Ventricular Outflow Tract Tachycardia. Circ Arrhythm Electrophysiol. 2021;14(10):e010082doi: 10.1161/CIRCEP.121.010082.
Ip, J. E., Xu, L., Dai, J., Steegborn, C., Jaffré, F., Evans, T., Cheung, J. W., Basson, C. T., Panaghie, G., Krogh-Madsen, T., Abbott, G. W., & Lerman, B. B. (2021). Constitutively Activating GNAS Somatic Mutation in Right Ventricular Outflow Tract Tachycardia. Circulation. Arrhythmia and electrophysiology, 14(10), e010082. https://doi.org/10.1161/CIRCEP.121.010082
Ip, James E, et al. "Constitutively Activating GNAS Somatic Mutation in Right Ventricular Outflow Tract Tachycardia." Circulation. Arrhythmia and electrophysiology vol. 14,10 (2021): e010082. doi: https://doi.org/10.1161/CIRCEP.121.010082
Ip JE, Xu L, Dai J, Steegborn C, Jaffré F, Evans T, Cheung JW, Basson CT, Panaghie G, Krogh-Madsen T, Abbott GW, Lerman BB. Constitutively Activating GNAS Somatic Mutation in Right Ventricular Outflow Tract Tachycardia. Circ Arrhythm Electrophysiol. 2021 Oct;14(10):e010082. doi: 10.1161/CIRCEP.121.010082. Epub 2021 Sep 30. PMID: 34587755; PMCID: PMC8569928.
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Cardiac-specific deletion of FDPS induces cardiac remodeling and dysfunction by enhancing the activity of small GTP-binding proteins.

The Journal of pathology

Wang X, Zhang X, Chen Y, Zhao C, Zhou W, Chen W, Zhang C, Ding K, Li W, Xu H, Lou L, Chu Z, Hu S, Yang J.
PMID: 34467534
J Pathol. 2021 Dec;255(4):438-450. doi: 10.1002/path.5789. Epub 2021 Oct 06.

The mevalonate pathway is essential for cholesterol biosynthesis. Previous studies have suggested that the key enzyme in this pathway, farnesyl diphosphate synthase (FDPS), regulates the cardiovascular system. We used human samples and mice that were deficient in cardiac FDPS...

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Wang X, Zhang X, Chen Y, et al. Cardiac-specific deletion of FDPS induces cardiac remodeling and dysfunction by enhancing the activity of small GTP-binding proteins. J Pathol. 2021;255(4):438-450doi: 10.1002/path.5789.
Wang, X., Zhang, X., Chen, Y., Zhao, C., Zhou, W., Chen, W., Zhang, C., Ding, K., Li, W., Xu, H., Lou, L., Chu, Z., Hu, S., & Yang, J. (2021). Cardiac-specific deletion of FDPS induces cardiac remodeling and dysfunction by enhancing the activity of small GTP-binding proteins. The Journal of pathology, 255(4), 438-450. https://doi.org/10.1002/path.5789
Wang, Xiying, et al. "Cardiac-specific deletion of FDPS induces cardiac remodeling and dysfunction by enhancing the activity of small GTP-binding proteins." The Journal of pathology vol. 255,4 (2021): 438-450. doi: https://doi.org/10.1002/path.5789
Wang X, Zhang X, Chen Y, Zhao C, Zhou W, Chen W, Zhang C, Ding K, Li W, Xu H, Lou L, Chu Z, Hu S, Yang J. Cardiac-specific deletion of FDPS induces cardiac remodeling and dysfunction by enhancing the activity of small GTP-binding proteins. J Pathol. 2021 Dec;255(4):438-450. doi: 10.1002/path.5789. Epub 2021 Oct 06. PMID: 34467534.
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Showing 133 to 135 of 135 entries