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Vasorelaxation to the Nitroxyl Donor Isopropylamine NONOate in Resistance Arteries Does Not Require Perivascular Calcitonin Gene-Related Peptide.

Hypertension (Dallas, Tex. : 1979)

Pinkney AMH, Lemmey HAL, Dora KA, Garland CJ.
PMID: 28760938
Hypertension. 2017 Jul 31; doi: 10.1161/HYPERTENSIONAHA.117.09737. Epub 2017 Jul 31.

Nitroxyl (HNO) donors offer considerable therapeutic potential for the treatment of hypertension-related cardiovascular disorders, particularly heart failure, as they combine an inotropic action with peripheral vasodilation. Angeli's salt is the only HNO donor whose mechanism has been studied in...

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Pinkney AMH, Lemmey HAL, Dora KA, et al. Vasorelaxation to the Nitroxyl Donor Isopropylamine NONOate in Resistance Arteries Does Not Require Perivascular Calcitonin Gene-Related Peptide. Hypertension. 2017;doi: 10.1161/HYPERTENSIONAHA.117.09737.
Pinkney, A. M. H., Lemmey, H. A. L., Dora, K. A., & Garland, C. J. (2017). Vasorelaxation to the Nitroxyl Donor Isopropylamine NONOate in Resistance Arteries Does Not Require Perivascular Calcitonin Gene-Related Peptide. Hypertension (Dallas, Tex. : 1979), . https://doi.org/10.1161/HYPERTENSIONAHA.117.09737
Pinkney, Alice M H, et al. "Vasorelaxation to the Nitroxyl Donor Isopropylamine NONOate in Resistance Arteries Does Not Require Perivascular Calcitonin Gene-Related Peptide." Hypertension (Dallas, Tex. : 1979) vol. (2017). doi: https://doi.org/10.1161/HYPERTENSIONAHA.117.09737
Pinkney AMH, Lemmey HAL, Dora KA, Garland CJ. Vasorelaxation to the Nitroxyl Donor Isopropylamine NONOate in Resistance Arteries Does Not Require Perivascular Calcitonin Gene-Related Peptide. Hypertension. 2017 Jul 31; doi: 10.1161/HYPERTENSIONAHA.117.09737. Epub 2017 Jul 31. PMID: 28760938.
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8-pCPT-cGMP prevents mitochondrial depolarization and improves the outcome of steatotic partial liver transplantation.

International journal of physiology, pathophysiology and pharmacology

Liu Q, Rehman H, Krishnasamy Y, Lemasters JJ, Zhong Z.
PMID: 28694919
Int J Physiol Pathophysiol Pharmacol. 2017 Jun 15;9(3):69-83. eCollection 2017.

Permeant cGMP analogs prevent the mitochondria permeability transition (MPT)

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Liu Q, Rehman H, Krishnasamy Y, et al. 8-pCPT-cGMP prevents mitochondrial depolarization and improves the outcome of steatotic partial liver transplantation. Int J Physiol Pathophysiol Pharmacol. 2017;9(3):69-83
Liu, Q., Rehman, H., Krishnasamy, Y., Lemasters, J. J., & Zhong, Z. (2017). 8-pCPT-cGMP prevents mitochondrial depolarization and improves the outcome of steatotic partial liver transplantation. International journal of physiology, pathophysiology and pharmacology, 9(3), 69-83.
Liu, Qinlong, et al. "8-pCPT-cGMP prevents mitochondrial depolarization and improves the outcome of steatotic partial liver transplantation." International journal of physiology, pathophysiology and pharmacology vol. 9,3 (2017): 69-83.
Liu Q, Rehman H, Krishnasamy Y, Lemasters JJ, Zhong Z. 8-pCPT-cGMP prevents mitochondrial depolarization and improves the outcome of steatotic partial liver transplantation. Int J Physiol Pathophysiol Pharmacol. 2017 Jun 15;9(3):69-83. eCollection 2017. PMID: 28694919; PMCID: PMC5498880.
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Arabidopsis AtCNGC10 rescues potassium channel mutants of E. coli, yeast and Arabidopsis and is regulated by calcium/calmodulin and cyclic GMP in E. coli.

Functional plant biology : FPB

Li X, Borsics T, Harrington HM, Christopher DA.
PMID: 32689163
Funct Plant Biol. 2005 Aug;32(7):643-653. doi: 10.1071/FP04233.

We have isolated and characterised AtCNGC10, one of the 20 members of the family of cyclic nucleotide (CN)-gated and calmodulin (CaM)-regulated channels (CNGCs) from Arabidopsis thaliana (L.) Heynh. AtCNGC10 bound CaM in a C-terminal subregion that contains a basic...

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Li X, Borsics T, Harrington HM, et al. Arabidopsis AtCNGC10 rescues potassium channel mutants of E. coli, yeast and Arabidopsis and is regulated by calcium/calmodulin and cyclic GMP in E. coli. Funct Plant Biol. 2005;32(7):643-653doi: 10.1071/FP04233.
Li, X., Borsics, T., Harrington, H. M., & Christopher, D. A. (2005). Arabidopsis AtCNGC10 rescues potassium channel mutants of E. coli, yeast and Arabidopsis and is regulated by calcium/calmodulin and cyclic GMP in E. coli. Functional plant biology : FPB, 32(7), 643-653. https://doi.org/10.1071/FP04233
Li, Xinli, et al. "Arabidopsis AtCNGC10 rescues potassium channel mutants of E. coli, yeast and Arabidopsis and is regulated by calcium/calmodulin and cyclic GMP in E. coli." Functional plant biology : FPB vol. 32,7 (2005): 643-653. doi: https://doi.org/10.1071/FP04233
Li X, Borsics T, Harrington HM, Christopher DA. Arabidopsis AtCNGC10 rescues potassium channel mutants of E. coli, yeast and Arabidopsis and is regulated by calcium/calmodulin and cyclic GMP in E. coli. Funct Plant Biol. 2005 Aug;32(7):643-653. doi: 10.1071/FP04233. PMID: 32689163.
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Sildenafil Transiently Delays Early Alveolar Healing of Tooth Extraction Sockets.

Clinics in surgery

Orchard E, Green W, Nair RP, Abreo F, Sunavala-Dossabhoy G.
PMID: 29930993
Clin Surg. 2017 May;2. Epub 2017 May 03.

Bone is a unique tissue that has the ability to repair itself and return to full function. Bone regeneration is a well synchronized biological process that recapitulates embryonic bone development. The establishment of a functional vascular supply has been...

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Orchard E, Green W, Nair RP, et al. Sildenafil Transiently Delays Early Alveolar Healing of Tooth Extraction Sockets. Clin Surg. 2017;2
Orchard, E., Green, W., Nair, R. P., Abreo, F., & Sunavala-Dossabhoy, G. (2017). Sildenafil Transiently Delays Early Alveolar Healing of Tooth Extraction Sockets. Clinics in surgery, 2.
Orchard, Elysse, et al. "Sildenafil Transiently Delays Early Alveolar Healing of Tooth Extraction Sockets." Clinics in surgery vol. 2 (2017).
Orchard E, Green W, Nair RP, Abreo F, Sunavala-Dossabhoy G. Sildenafil Transiently Delays Early Alveolar Healing of Tooth Extraction Sockets. Clin Surg. 2017 May;2. Epub 2017 May 03. PMID: 29930993; PMCID: PMC6006518.
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Gasdermin D in macrophages restrains colitis by controlling cGAS-mediated inflammation.

Science advances

Ma C, Yang D, Wang B, Wu C, Wu Y, Li S, Liu X, Lassen K, Dai L, Yang S.
PMID: 32671214
Sci Adv. 2020 May 20;6(21):eaaz6717. doi: 10.1126/sciadv.aaz6717. eCollection 2020 May.

The functional relevance and mechanistic basis of the effects of the pyroptosis executioner Gasdermin D (GSDMD) on colitis remain unclear. In this study, we observed that GSDMD protein was activated during intestinal inflammation in a model of chemically induced...

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Ma C, Yang D, Wang B, et al. Gasdermin D in macrophages restrains colitis by controlling cGAS-mediated inflammation. Sci Adv. 2020;6(21):eaaz6717doi: 10.1126/sciadv.aaz6717.
Ma, C., Yang, D., Wang, B., Wu, C., Wu, Y., Li, S., Liu, X., Lassen, K., Dai, L., & Yang, S. (2020). Gasdermin D in macrophages restrains colitis by controlling cGAS-mediated inflammation. Science advances, 6(21), eaaz6717. https://doi.org/10.1126/sciadv.aaz6717
Ma, Chunmei, et al. "Gasdermin D in macrophages restrains colitis by controlling cGAS-mediated inflammation." Science advances vol. 6,21 (2020): eaaz6717. doi: https://doi.org/10.1126/sciadv.aaz6717
Ma C, Yang D, Wang B, Wu C, Wu Y, Li S, Liu X, Lassen K, Dai L, Yang S. Gasdermin D in macrophages restrains colitis by controlling cGAS-mediated inflammation. Sci Adv. 2020 May 20;6(21):eaaz6717. doi: 10.1126/sciadv.aaz6717. eCollection 2020 May. PMID: 32671214; PMCID: PMC7314554.
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CGAS is a micronucleophagy receptor for the clearance of micronuclei.

Autophagy

Zhao M, Wang F, Wu J, Cheng Y, Cao Y, Wu X, Ma M, Tang F, Liu Z, Liu H, Ge B.
PMID: 33752561
Autophagy. 2021 Dec;17(12):3976-3991. doi: 10.1080/15548627.2021.1899440. Epub 2021 Mar 22.

Micronuclei are constantly considered as a marker of genome instability and very recently found to be a trigger of innate immune responses. An increased frequency of micronuclei is associated with many diseases, but the mechanism underlying the regulation of...

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Zhao M, Wang F, Wu J, et al. CGAS is a micronucleophagy receptor for the clearance of micronuclei. Autophagy. 2021;17(12):3976-3991doi: 10.1080/15548627.2021.1899440.
Zhao, M., Wang, F., Wu, J., Cheng, Y., Cao, Y., Wu, X., Ma, M., Tang, F., Liu, Z., Liu, H., & Ge, B. (2021). CGAS is a micronucleophagy receptor for the clearance of micronuclei. Autophagy, 17(12), 3976-3991. https://doi.org/10.1080/15548627.2021.1899440
Zhao, Mengmeng, et al. "CGAS is a micronucleophagy receptor for the clearance of micronuclei." Autophagy vol. 17,12 (2021): 3976-3991. doi: https://doi.org/10.1080/15548627.2021.1899440
Zhao M, Wang F, Wu J, Cheng Y, Cao Y, Wu X, Ma M, Tang F, Liu Z, Liu H, Ge B. CGAS is a micronucleophagy receptor for the clearance of micronuclei. Autophagy. 2021 Dec;17(12):3976-3991. doi: 10.1080/15548627.2021.1899440. Epub 2021 Mar 22. PMID: 33752561.
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CGAS is a micronucleophagy receptor for the clearance of micronuclei.

Autophagy

Zhao M, Wang F, Wu J, Cheng Y, Cao Y, Wu X, Ma M, Tang F, Liu Z, Liu H, Ge B.
PMID: 33752561
Autophagy. 2021 Dec;17(12):3976-3991. doi: 10.1080/15548627.2021.1899440. Epub 2021 Mar 22.

Micronuclei are constantly considered as a marker of genome instability and very recently found to be a trigger of innate immune responses. An increased frequency of micronuclei is associated with many diseases, but the mechanism underlying the regulation of...

Cite
Zhao M, Wang F, Wu J, et al. CGAS is a micronucleophagy receptor for the clearance of micronuclei. Autophagy. 2021;17(12):3976-3991doi: 10.1080/15548627.2021.1899440.
Zhao, M., Wang, F., Wu, J., Cheng, Y., Cao, Y., Wu, X., Ma, M., Tang, F., Liu, Z., Liu, H., & Ge, B. (2021). CGAS is a micronucleophagy receptor for the clearance of micronuclei. Autophagy, 17(12), 3976-3991. https://doi.org/10.1080/15548627.2021.1899440
Zhao, Mengmeng, et al. "CGAS is a micronucleophagy receptor for the clearance of micronuclei." Autophagy vol. 17,12 (2021): 3976-3991. doi: https://doi.org/10.1080/15548627.2021.1899440
Zhao M, Wang F, Wu J, Cheng Y, Cao Y, Wu X, Ma M, Tang F, Liu Z, Liu H, Ge B. CGAS is a micronucleophagy receptor for the clearance of micronuclei. Autophagy. 2021 Dec;17(12):3976-3991. doi: 10.1080/15548627.2021.1899440. Epub 2021 Mar 22. PMID: 33752561.
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Endogenous retroviruses promote homeostatic and inflammatory responses to the microbiota.

Cell

Lima-Junior DS, Krishnamurthy SR, Bouladoux N, Collins N, Han SJ, Chen EY, Constantinides MG, Link VM, Lim AI, Enamorado M, Cataisson C, Gil L, Rao I, Farley TK, Koroleva G, Attig J, Yuspa SH, Fischbach MA, Kassiotis G, Belkaid Y.
PMID: 34166614
Cell. 2021 Jul 08;184(14):3794-3811.e19. doi: 10.1016/j.cell.2021.05.020. Epub 2021 Jun 23.

The microbiota plays a fundamental role in regulating host immunity. However, the processes involved in the initiation and regulation of immunity to the microbiota remain largely unknown. Here, we show that the skin microbiota promotes the discrete expression of...

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Lima-Junior DS, Krishnamurthy SR, Bouladoux N, et al. Endogenous retroviruses promote homeostatic and inflammatory responses to the microbiota. Cell. 2021;184(14):3794-3811.e19doi: 10.1016/j.cell.2021.05.020.
Lima-Junior, D. S., Krishnamurthy, S. R., Bouladoux, N., Collins, N., Han, S. J., Chen, E. Y., Constantinides, M. G., Link, V. M., Lim, A. I., Enamorado, M., Cataisson, C., Gil, L., Rao, I., Farley, T. K., Koroleva, G., Attig, J., Yuspa, S. H., Fischbach, M. A., Kassiotis, G., & Belkaid, Y. (2021). Endogenous retroviruses promote homeostatic and inflammatory responses to the microbiota. Cell, 184(14), 3794-3811.e19. https://doi.org/10.1016/j.cell.2021.05.020
Lima-Junior, Djalma S, et al. "Endogenous retroviruses promote homeostatic and inflammatory responses to the microbiota." Cell vol. 184,14 (2021): 3794-3811.e19. doi: https://doi.org/10.1016/j.cell.2021.05.020
Lima-Junior DS, Krishnamurthy SR, Bouladoux N, Collins N, Han SJ, Chen EY, Constantinides MG, Link VM, Lim AI, Enamorado M, Cataisson C, Gil L, Rao I, Farley TK, Koroleva G, Attig J, Yuspa SH, Fischbach MA, Kassiotis G, Belkaid Y. Endogenous retroviruses promote homeostatic and inflammatory responses to the microbiota. Cell. 2021 Jul 08;184(14):3794-3811.e19. doi: 10.1016/j.cell.2021.05.020. Epub 2021 Jun 23. PMID: 34166614; PMCID: PMC8381240.
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Enhanced cGAS-STING-dependent interferon signaling associated with mutations in ATAD3A.

The Journal of experimental medicine

Lepelley A, Della Mina E, Van Nieuwenhove E, Waumans L, Fraitag S, Rice GI, Dhir A, Frémond ML, Rodero MP, Seabra L, Carter E, Bodemer C, Buhas D, Callewaert B, de Lonlay P, De Somer L, Dyment DA, Faes F, Grove L, Holden S, Hully M, Kurian MA, McMillan HJ, Suetens K, Tyynismaa H, Chhun S, Wai T, Wouters C, Bader-Meunier B, Crow YJ.
PMID: 34387651
J Exp Med. 2021 Oct 04;218(10). doi: 10.1084/jem.20201560. Epub 2021 Aug 13.

Mitochondrial DNA (mtDNA) has been suggested to drive immune system activation, but the induction of interferon signaling by mtDNA has not been demonstrated in a Mendelian mitochondrial disease. We initially ascertained two patients, one with a purely neurological phenotype...

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Lepelley A, Della Mina E, Van Nieuwenhove E, et al. Enhanced cGAS-STING-dependent interferon signaling associated with mutations in ATAD3A. J Exp Med. 2021;218(10)doi: 10.1084/jem.20201560.
Lepelley, A., Della Mina, E., Van Nieuwenhove, E., Waumans, L., Fraitag, S., Rice, G. I., Dhir, A., Frémond, M. L., Rodero, M. P., Seabra, L., Carter, E., Bodemer, C., Buhas, D., Callewaert, B., de Lonlay, P., De Somer, L., Dyment, D. A., Faes, F., Grove, L., Holden, S., Hully, M., Kurian, M. A., McMillan, H. J., Suetens, K., Tyynismaa, H., Chhun, S., Wai, T., Wouters, C., Bader-Meunier, B., & Crow, Y. J. (2021). Enhanced cGAS-STING-dependent interferon signaling associated with mutations in ATAD3A. The Journal of experimental medicine, 218(10), . https://doi.org/10.1084/jem.20201560
Lepelley, Alice, et al. "Enhanced cGAS-STING-dependent interferon signaling associated with mutations in ATAD3A." The Journal of experimental medicine vol. 218,10 (2021). doi: https://doi.org/10.1084/jem.20201560
Lepelley A, Della Mina E, Van Nieuwenhove E, Waumans L, Fraitag S, Rice GI, Dhir A, Frémond ML, Rodero MP, Seabra L, Carter E, Bodemer C, Buhas D, Callewaert B, de Lonlay P, De Somer L, Dyment DA, Faes F, Grove L, Holden S, Hully M, Kurian MA, McMillan HJ, Suetens K, Tyynismaa H, Chhun S, Wai T, Wouters C, Bader-Meunier B, Crow YJ. Enhanced cGAS-STING-dependent interferon signaling associated with mutations in ATAD3A. J Exp Med. 2021 Oct 04;218(10). doi: 10.1084/jem.20201560. Epub 2021 Aug 13. PMID: 34387651; PMCID: PMC8374862.
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Activation of caspase-1 is mediated by stimulation of interferon genes and NLR family pyrin domain containing 3 in monocytes of active systemic lupus erythematosus.

Clinical and experimental rheumatology

Inokuchi S, Mitoma H, Kawano S, Ayano M, Kimoto Y, Akahoshi M, Arinobu Y, Akashi K, Horiuchi T, Niiro H.
PMID: 33769266
Clin Exp Rheumatol. 2021 Mar 22; Epub 2021 Mar 22.

OBJECTIVES: Emerging evidence has shown the importance of inflammasome activation in the progression of autoimmune diseases. In this study, we aimed to identify the main cell types activating inflammasome in autoimmune diseases and to clarify the intracellular pathway of...

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Inokuchi S, Mitoma H, Kawano S, et al. Activation of caspase-1 is mediated by stimulation of interferon genes and NLR family pyrin domain containing 3 in monocytes of active systemic lupus erythematosus. Clin Exp Rheumatol. 2021;
Inokuchi, S., Mitoma, H., Kawano, S., Ayano, M., Kimoto, Y., Akahoshi, M., Arinobu, Y., Akashi, K., Horiuchi, T., & Niiro, H. (2021). Activation of caspase-1 is mediated by stimulation of interferon genes and NLR family pyrin domain containing 3 in monocytes of active systemic lupus erythematosus. Clinical and experimental rheumatology, .
Inokuchi, Shoichiro, et al. "Activation of caspase-1 is mediated by stimulation of interferon genes and NLR family pyrin domain containing 3 in monocytes of active systemic lupus erythematosus." Clinical and experimental rheumatology vol. (2021).
Inokuchi S, Mitoma H, Kawano S, Ayano M, Kimoto Y, Akahoshi M, Arinobu Y, Akashi K, Horiuchi T, Niiro H. Activation of caspase-1 is mediated by stimulation of interferon genes and NLR family pyrin domain containing 3 in monocytes of active systemic lupus erythematosus. Clin Exp Rheumatol. 2021 Mar 22; Epub 2021 Mar 22. PMID: 33769266.
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Treatment of Cardiovascular Dysfunction With PDE5-Inhibitors - Temperature Dependent Effects on Transport and Metabolism of cAMP and cGMP.

Frontiers in physiology

Selli AL, Kuzmiszyn AK, Smaglyukova N, Kondratiev TV, Fuskevåg OM, Lyså RA, Ravna AW, Tveita T, Sager G, Dietrichs ES.
PMID: 34393818
Front Physiol. 2021 Jul 30;12:695779. doi: 10.3389/fphys.2021.695779. eCollection 2021.

INTRODUCTION: Cardiovascular dysfunction is a potentially lethal complication of hypothermia. Due to a knowledge gap, pharmacological interventions are not recommended at core temperatures below 30°C. Yet, further cooling is induced in surgical procedures and survival of accidental hypothermia is...

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Selli AL, Kuzmiszyn AK, Smaglyukova N, et al. Treatment of Cardiovascular Dysfunction With PDE5-Inhibitors - Temperature Dependent Effects on Transport and Metabolism of cAMP and cGMP. Front Physiol. 2021;12:695779doi: 10.3389/fphys.2021.695779.
Selli, A. L., Kuzmiszyn, A. K., Smaglyukova, N., Kondratiev, T. V., Fuskevåg, O. M., Lyså, R. A., Ravna, A. W., Tveita, T., Sager, G., & Dietrichs, E. S. (2021). Treatment of Cardiovascular Dysfunction With PDE5-Inhibitors - Temperature Dependent Effects on Transport and Metabolism of cAMP and cGMP. Frontiers in physiology, 12695779. https://doi.org/10.3389/fphys.2021.695779
Selli, Anders L, et al. "Treatment of Cardiovascular Dysfunction With PDE5-Inhibitors - Temperature Dependent Effects on Transport and Metabolism of cAMP and cGMP." Frontiers in physiology vol. 12 (2021): 695779. doi: https://doi.org/10.3389/fphys.2021.695779
Selli AL, Kuzmiszyn AK, Smaglyukova N, Kondratiev TV, Fuskevåg OM, Lyså RA, Ravna AW, Tveita T, Sager G, Dietrichs ES. Treatment of Cardiovascular Dysfunction With PDE5-Inhibitors - Temperature Dependent Effects on Transport and Metabolism of cAMP and cGMP. Front Physiol. 2021 Jul 30;12:695779. doi: 10.3389/fphys.2021.695779. eCollection 2021. PMID: 34393818; PMCID: PMC8361756.
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Statins act as transient type I interferon inhibitors to enable the antitumor activity of modified vaccinia Ankara viral vectors.

Journal for immunotherapy of cancer

Tenesaca S, Vasquez M, Alvarez M, Otano I, Fernandez-Sendin M, Di Trani CA, Ardaiz N, Gomar C, Bella A, Aranda F, Medina-Echeverz J, Melero I, Berraondo P.
PMID: 34321273
J Immunother Cancer. 2021 Jul;9(7). doi: 10.1136/jitc-2020-001587.

BACKGROUND: Modified vaccinia virus Ankara (MVA) are genetically engineered non-replicating viral vectors. Intratumoral administration of MVA induces a cyclic GMP-AMP synthase-mediated type I interferon (IFN) response and the production of high levels of the transgenes engineered into the viral...

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Tenesaca S, Vasquez M, Alvarez M, et al. Statins act as transient type I interferon inhibitors to enable the antitumor activity of modified vaccinia Ankara viral vectors. J Immunother Cancer. 2021;9(7)doi: 10.1136/jitc-2020-001587.
Tenesaca, S., Vasquez, M., Alvarez, M., Otano, I., Fernandez-Sendin, M., Di Trani, C. A., Ardaiz, N., Gomar, C., Bella, A., Aranda, F., Medina-Echeverz, J., Melero, I., & Berraondo, P. (2021). Statins act as transient type I interferon inhibitors to enable the antitumor activity of modified vaccinia Ankara viral vectors. Journal for immunotherapy of cancer, 9(7), . https://doi.org/10.1136/jitc-2020-001587
Tenesaca, Shirley, et al. "Statins act as transient type I interferon inhibitors to enable the antitumor activity of modified vaccinia Ankara viral vectors." Journal for immunotherapy of cancer vol. 9,7 (2021). doi: https://doi.org/10.1136/jitc-2020-001587
Tenesaca S, Vasquez M, Alvarez M, Otano I, Fernandez-Sendin M, Di Trani CA, Ardaiz N, Gomar C, Bella A, Aranda F, Medina-Echeverz J, Melero I, Berraondo P. Statins act as transient type I interferon inhibitors to enable the antitumor activity of modified vaccinia Ankara viral vectors. J Immunother Cancer. 2021 Jul;9(7). doi: 10.1136/jitc-2020-001587. PMID: 34321273; PMCID: PMC8320251.
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